Chapter 22 Pneumothorax CL GA DD Figure 22-1. Right-side pneumothorax. GA, Gas accumulation; DD, depressed diaphragm; CL, collapsed lung. Inset, Atelectasis, a common secondary anatomic alteration of the lungs. Slide 1 Copyright © 2006 by Mosby, Inc. Anatomic Alterations of the Lungs Slide 2 Lung collapse Atelectasis Chest wall expansion Compression of the great veins and decreased cardiac venous return Copyright © 2006 by Mosby, Inc. Etiology—3 Ways Slide 3 From the lungs through a perforation of the visceral pleura From the surrounding atmosphere through a perforation of the chest wall and parietal pleura or, rarely, through an esophageal fistula or a perforated abdominal viscus From gas-forming microorganisms in an empyema in the pleural space (rare) Copyright © 2006 by Mosby, Inc. Pneumothorax Classifications General Terms Slide 4 Closed pneumothorax Open pneumothorax Tension pneumothorax Copyright © 2006 by Mosby, Inc. Pneumothorax Classifications Based on Origin Slide 5 Traumatic pneumothorax Spontaneous pneumothorax Iatrogenic pneumothorax Copyright © 2006 by Mosby, Inc. Figure 22-3. Closed (tension) pneumothorax produced by a chest wall wound. Slide 6 Copyright © 2006 by Mosby, Inc. Figure 22-4. Pneumothorax produced by a rupture in the visceral pleura that functions as a check valve. Slide 7 Copyright © 2006 by Mosby, Inc. Spontaneous Pneumothorax Slide 8 Copyright © 2006 by Mosby, Inc. Iatrogenic Pneumothorax Slide 9 Copyright © 2006 by Mosby, Inc. Overview of the Cardiopulmonary Clinical Manifestations Associated with PNEUMOTHORAX The following clinical manifestations result from the pathophysiologic mechanisms caused (or activated) by Atelectasis (see Figure 9-7)—the major anatomic alterations of the lungs associated with pneumothorax (see Figure 22-1). Slide 10 Copyright © 2006 by Mosby, Inc. Figure 9-7. Atelectasis clinical scenario. Slide 11 Copyright © 2006 by Mosby, Inc. Clinical Data Obtained at the Patient’s Bedside Vital signs Increased respiratory rate Stimulation of peripheral chemoreceptors Other possible mechanisms • • • • • Slide 12 Decreased lung compliance Activation of the deflation receptors Activation of the irritant receptors Stimulation of the J receptors Pain/anxiety Increased heart rate, cardiac output, blood pressure Copyright © 2006 by Mosby, Inc. Figure 22-5. Venous admixture in pneumothorax. Slide 13 Copyright © 2006 by Mosby, Inc. Clinical Data Obtained at the Patient’s Bedside Slide 14 Cyanosis Chest assessment findings Hyperresonant percussion note over the pneumothorax Diminished breath sounds over the pneumothorax Tracheal shift Displaced heart sounds Increased thoracic volume on the affected side • Particularly in tension pneumothorax Copyright © 2006 by Mosby, Inc. Figure 22-6. Because the ratio of extrapulmonary gas to solid tissue increases in a pneumothorax, hyperresonant percussion notes are produced over the affected area. Slide 15 Copyright © 2006 by Mosby, Inc. Figure 22-7. Breath sounds diminish as gas accumulates in the intrapleural space. Slide 16 Copyright © 2006 by Mosby, Inc. Figure 22-8. As gas accumulates in the intrapleural space, the chest diameter increases on the affected side in a tension pneumothorax. Slide 17 Copyright © 2006 by Mosby, Inc. Clinical Data Obtained from Laboratory Tests and Special Procedures Slide 18 Copyright © 2006 by Mosby, Inc. Pulmonary Function Study: Lung Volume and Capacity Findings VT Slide 19 RV FRC TLC N or VC IC ERV RV/TLC% N Copyright © 2006 by Mosby, Inc. Arterial Blood Gases Small Pneumothorax pH Slide 20 Acute alveolar hyperventilation with hypoxemia PaCO2 HCO3 (Slightly) PaO2 Copyright © 2006 by Mosby, Inc. Time and Progression of Disease Disease Onset Alveolar Hyperventilation 100 90 PaO2 or PaCO2 80 Point at which PaO2 declines enough to stimulate peripheral oxygen receptors 70 60 PaO2 50 40 30 20 10 0 Figure 4-2. PaO2 and PaCO2 trends during acute alveolar hyperventilation. Slide 21 Copyright © 2006 by Mosby, Inc. Arterial Blood Gases Large Pneumothorax Acute ventilatory failure with hypoxemia pH Slide 22 PaCO2 HCO3 (Slightly) PaO2 Copyright © 2006 by Mosby, Inc. Time and Progression of Disease Disease Onset Alveolar Hyperventilation Acute Ventilatory Failure 100 90 Pa02 or PaC02 80 70 Point at which PaO2 declines enough to stimulate peripheral oxygen receptors Point at which disease becomes severe and patient begins to become fatigued 60 50 40 30 20 10 0 Figure 4-7. PaO2 and PaCO2 trends during acute ventilatory failure. Slide 23 Copyright © 2006 by Mosby, Inc. Oxygenation Indices QS/QT DO2 VO2 Normal O2ER Slide 24 C(a-v)O2 (severe) SvO2 Copyright © 2006 by Mosby, Inc. Hemodynamic Indices (Large Pneumothorax) Slide 25 CVP RAP PA PCWP CO SV SVI CI RVSWI LVSWI PVR SVR Copyright © 2006 by Mosby, Inc. Radiologic Findings Chest radiograph Slide 26 Increased translucency Mediastinal shift to unaffected side in tension pneumothorax Depressed diaphragm Lung collapse Atelectasis Copyright © 2006 by Mosby, Inc. Figure 22-9. Left-sided pneumothorax (arrows). Note the shift of the heart and mediastinum to the right away from the tension pneumothorax. Slide 27 Copyright © 2006 by Mosby, Inc. A B Figure 22-10. A, Development of a small tension pneumothorax in the lower part of the right lung (arrow). B, The same pneumothorax 30 minutes later. Note the shift of the heart and mediastinum to the left away from the tension pneumothorax. Also note the depression of the right hemidiaphragm (arrow). Slide 28 Copyright © 2006 by Mosby, Inc. General Management of Pneumothorax >20%—gas should be evacuated Negative pressure—5 to 12 cm H2O Slide 29 Should not exceed negative 12 cm H2O Copyright © 2006 by Mosby, Inc. General Management of Pneumothorax Respiratory care treatment protocols Slide 30 Oxygen therapy protocol Hyperinflation therapy protocol Mechanical ventilation protocol Copyright © 2006 by Mosby, Inc. General Management of Pneumothorax PLEURODESIS Chemical or medication injected into the chest cavity Talc Tetracycline Bleomycin sulfate Produces inflammatory reaction between lungs and inner chest cavity Slide 31 Causes lung to stick to chest cavity Copyright © 2006 by Mosby, Inc. Classroom Discussion Case Study: Pneumothorax Slide 32 Copyright © 2006 by Mosby, Inc.