PTRS 705 MIDTERM STUDY GUIDE PHYSICAL AGENTS TOPICS ACOUSTIC SPECTRUM VS. ELECTROMAGNETIC SPECTRUM Acoustic spectrum includes: ultrasound and extracorporeal shock wave therapy Electromagnetic (visible light) spectrum: RED/LONG λ/LOW FREQUENCY ------------- VIOLET/SHORT λ/HIGH FREQUENCY Characteristics of both spectrums: Their effect is produced when sufficient intense electrical or chemical forces are applied to a material Arndt-Schultz Principle = no reactions or changes can occur in the body tissues if the amount of energy absorbed is insufficient to stimulate the absorbing tissues Radiation = energy traveling through space in multiple forms When radiations come in contact with various biological tissues, the velocity and direction of travel are altered within the various types of tissues CONCEPTS OF REFLECTION, REFRACTION, TRANSMISSION, AND ABSORPTION Reflection = to bounce off Refraction = to change direction Transmission = to penetrate Absorption = to be absorbed CHARACTERISTICS OF WAVELENGTH Wavelength = distance from peak to peak All various types of radiations in the electromagnetic spectrum have a specific wavelength & frequency of vibration that separates one region from another The longest wavelengths tend to have the greatest depths of penetration regardless of their frequencies Frequency = number of oscillations in one second (Hertz, Hz) Velocity = wavelength x frequency (~300 million m/s) Inverse relationship between energy absorption by a tissue & energy penetration to deeper layers Law of Grotthus-Draper = energy that is not absorbed by the superficial tissues will penetrate to deeper tissues FACTORS AFFECTING PENETRATION 1. Tissue density 2. Tissue absorption rate (Grotthus-Draper) 3. Tissue interface – there is always refraction at each interface 4. Angle of physical agent to treatment area – ex. US head should be perpendicular to surface of body Cosine Law = the smaller the angle between the propagating ray & the right angle, the less radiation reflected and the greater absorbed 5. Distance between the source of radiation and the surface of patient Inverse Square Law = the intensity of the radiation striking a particular surface varies inversely with the square of the distance from the source ELECTROMAGNETIC RADIATIONS Longer λ= greater penetration (heat formation) INFRARED, DIATHERMY, E-STIM (beyond red) Shorter λ= produce chemical effects (healing characteristic) UV RAYS, IONIZING RADIATION (beyond violet) As the density of the transmitting medium increases, the velocity of travel significantly decreases as a result of refraction, reflection, or absorption by my the molecules in the medium ACOUSTIC SPECTRUM Mechanical vibrations depend on conduction through molecular collision The denser the medium, the greater the velocity Acoustic waves travel slower than electromagnetic radiation Wavelengths of acoustic waves are considerably shorter than electromagnetic radiation can penetrate deeper because you can manipulate the frequency Good for deep penetration because acoustic waves travel well in homogenous tissues (e.g. fat tissue) US waves can travel 5-13 cm deep POMAS 1. Identify the PHASE of healing (inflammation, proliferation, maturation) 2. Decide on an OBJECTIVE (whether or not to address swelling, pain, strength, etc.) 3. Choose a MODALITY 4. Decide on APPLICATOR 5. Decide on SETTINGS CASCADES OF HEALING THREE PHASES OF HEALING 1. Inflammatory Phase (3-7 days) o Main purpose = DEBRIS REMOVAL o Stimulated due to a hypoxic state in the injured area o Cellular response → Delivery of leukocytes and macrophages and exudate → Localize or dispose of injury by-products → Protective response o Vascular reaction → Vascular spasm → Dilation and stasis → 24-36 hrs (clear the agent) o Chemical mediators → Histamine and leucotaxin – increase cell permeability → Necrosin – phagocytic activity o Platelet function → When collagen is exposed, platelets attach o Clot formation → Thromboplastin is released by the cell → Conversion of prothrombin to thrombin → Conversion of fibrinogen to fibrin → Clots off the injury within 12 hrs and complete by 48 hrs after injury → Symptoms: pain, swelling, redness 2. Proliferative Phase (21-30 days) o Main purposes = FIBROBLASTIC REPAIR and FILLING THE INJURY o Begins within hours of the injury o The hypoxia of inflammation triggers angiogenesis o As the clot breaks down, granulation tissue will appear – fibroblasts, collagen, and capillaries o Fibroblasts secrete extracellular matrix and encourage contraction o Symptoms: pain and tenderness start to decrease, minimal residual swelling 3. Maturation Phase (up to years) o Main purposes = REMODELING and STRENGTHENING o Collagen rearrangement – apply stress to assist with aligning the fibers (Wolff’s Law) o Strength only returns to about 80% original strength o Symptoms: no pain, may have tenderness with end-range, weakness Chronic Inflammation Agent is not eliminated Leukocytes replaced with macrophages, lymphocytes, and plasma cells (metabolically active by-products) Increased fibrogenesis Tissue damage NO TIME FRAME Factors that slow down healing: Extent of injury (microtears due to overuse, macrotears due to acute trauma) Edema Hemorrhage Poor circulation Tissue separation – body heals fastest if the edges of the wound are approximated (unless it’s a deep wound) Muscle spasm Atrophy Corticosteroids – stop inflammation Infections – increase exudates in interstitium Dehydration – need fluid to move waste Aging Malnutrition Modality selection based on phase of healing: Inflammatory Phase – want to reduce swelling and pain o FIRST CHOICE: Cryotherapy – ice, cold pack o Compression o E-stim – increase blood flow, stimulate contraction o US – non-thermal US (pulse) works best in inflammatory phase o LASER – primarily works on chemical properties Proliferative Phase – want to reduce pain, increase ROM/strength o FIRST CHOICE: Thermotherapy – hot pack o Diathermy o E-stim o Compression o US Maturation Phase – want to focus on ROM/strength o Any, but deep heating works best (thermal US and diathermy) – add stress to the tissue too Chronic Inflammation o Cryotherapy o Thermotherapy o Diathermy PHYSIOLOGICAL EFFECT OF INFRARED MODALITIES Increase or decrease circulation (vasodilation/vasoconstriction) Stimulation of cutaneous sensory nerve endings causing analgesic effects Enhance the rate of healing – to return to function TRANSMISSION OF HEAT Conduction = by direct contact of body with heat or cold modality (until equilibrium is reached) Convection = particles (air or water) move across the body, creating a temperature variation Radiation = transfer of heat from a warmer source to a cooler source through a conducting medium such as infrared lamps transferring heat through air Conversion = changing of energy from one form to another MECHANISMS OF HEAT TRANSFER BY MODALITY CONDUCTION CONVECTION RADIATION Ice massage Hot whirlpool Infrared lamps CONVERSION Ultrasound Cold packs Hydrocollator Cold spray Ice immersion Contrast baths Cryo-cuff Cryokinetics Paraffin bath Cold whirlpool *Fluidotherapy (for CRPS pts.) LASER UV light Diathermy *Not an infrared modality INDICATIONS, CONTRAINDICATIONS, EFFECTS OF CRYOTHERAPY General info about cryotherapy: Greatest benefit in ACUTE injury Lowers the temperature in the injured area Reduces the metabolic rate Promotes vasoconstriction Reduces pain, spasms, and spastic conditions Decreases local neural activity Cryotherapy stages: 1. Cool 2. Burning/Stinging 3. Aching 4. Numb INDICATIONS CONTRAINDICATIONS Acute, Chronic pain (cold has a longer λ) Impaired circulation (if you use cryotherapy, make sure to document pulses) Acute swelling Peripheral vascular disease Myofascial trigger points Hypersensitivity to cold Muscle guarding, spasms Skin anesthesia (loss of sensation) Acute muscle/ligament sprain *Open wounds or skin conditions Acute contusion Infections (localization of waste products Bursitis during cold therapy, then when the area Tenosynovitis warms via vasodilatation, the waste Tendinitis products may spread systemically) Delayed Onset Muscle Soreness (DOMS) INDICATIONS, CONTRAINDICATIONS, EFFECTS OF THERMOTHERAPY General info about thermotherapy: Beneficial for SUBACUTE and CHRONIC conditions Raises temperature in the injured area Preferred treatment for pain and discomfort Promotes vasodilatation Increases metabolic rate Increases local neural activity INDICATIONS CONTRAINDICATIONS Subacute, Chronic pain Impaired circulation Subacute, Chronic inflammatory conditions *Peripheral vascular disease Resolution of swelling/edema removal Skin anesthesia (loss of sensation) Myofascial trigger points *Open wounds or skin conditions Muscle guarding, spasms Acute musculoskeletal conditions - Make sure to take extra care with pts. who have a thin Subacute muscle/ligament sprain epidermis Subacute contusion THERAPEUTIC USE OF INFRARED MODALITIES REMEMBER: stimulation can only occur if the amount of energy delivered and absorbed is sufficient (ArndtSchultz) GOAL: to deliver sufficient energy, in the best form & in the safest way, to stimulate the tissues to perform their normal function CPT CODES FOR CRYOTHERAPY AND THERMOTHERAPY CPT MODALITY SERVICE/TIMED 97010 HMP/Cold Pack N/A 97022 Whirlpool/Fluidotherapy Service – no time limit 97018 Paraffin Service – no time limit 97034 Contrast Baths Timed – at least 8 mins 97028 UV light Service – no time limit $$$$$$$$ Bundled – not reimbursed $14 $6 $13 ?? DOCUMENTATION OF INFRARED MODALITIES When documenting the use of a modality, must include: 1. Type of modality 2. Location of treatment 3. Parameters 4. Response to treatment 5. Treatment goal – related to function CHARACTERISTICS OF DIATHERMY Thermal or non-thermal (chemical) effects Depth of penetration deeper than other infrared modalities Categorized as shortwave & microwave (not commercially used) Can heat a large area - ~12 cm deep MICROWAVE (MWS) SHORTWAVE (SWD) 1. Emits electromagnetic energy at a much higher 1. Three specific frequencies & corresponding λ: frequency 915 MHz & 2450 MHz, 12 cm deep - 13.56 MHz, 22 m 2. As frequency INCREASES, the penetration of energy - 27.12 MHz, 11 m (MOST COMMON) in the tissues DECREASES provides a more - 40.68 Mhz, 7 m shallow tissue heating effect 2. Two models: continuous & pulsed (or hybrid) 3. Generate a stronger ELECTRICAL FIELD 3. Generate a stronger MAGNETIC FIELD MOST COMMON DIATHERMY FREQUENCY 27.12 MHz (11 m λ) ELECTROMAGNETIC RESONANCE Tuning = occurs when both the biological tissues (pt. circuit) and oscillating generator (device circuit) are oscillating at the same frequency always going to be some resistance Only with complete resonance can the electromagnetic energy be fully delivered to the tissues (rarely happens) PHYSIOLOGICAL EFFECTS OF DIATHERMY Microwave (MWS) Much more localized heating effect than SWD Shortwave (SWD) Continuous = causes deep heating of soft tissues Pulsed = the delivery of radiation is interrupted for the purpose of inducing non-thermal (chemical) physiological & therapeutic effects INDICATIONS OF DIATHERMY INDICATIONS Most effective modality for heating skeletal muscle OA, RA, or traumatic arthritis Strain or sprain Acute or chronic bursitis Traumatic injury to muscle, ligament, or tendon resulting in functional loss Joint dislocation or subluxation Treatment for a post surgical functional loss Adhesive capsulitis Joint contracture Wound care CPT CODE FOR DIATHERMY CPT MODALITY 97024 Shortwave/Microwave SERVICE/TIMED Service – no time limit $$$$$$ $4.91 *Numbers to Know Hydrocollator (heat) 170 degrees F Hydrocollator (cold) 8 degrees F COMPARISON OF MODALITIES Infrared modalities – based on electromagnetic spectrum Cryotherapy – longer λ, deeper penetration Thermotherapy Diathermy – longer λ than thermotherapy or cryotherapy, greater penetration NOTE: Focus on the temperature change needed for tissue change Cryotherapy benefits occur around 57°F Thermotherapy ideal is 38-40°C (above 45°C is dangerous STOP THE HEAT) 1° change – increases metabolism (hot packs) 2-3° change – helps with pain (hot packs or paraffin) 4° change – vigorous, collagen extension (US or diathermy) DEPTH OF PENETRATION INCREASES AS THE TEMPERATURE CHANGE INCREASES GENERAL TIME FRAMES FOR APPLYING CRYOTHERAPY AND THERMOTHERAPY Heat – 20-30 min Cold – as long as it takes for pt. to get through the 4 stages (depends on body type); typically 10-20 min LASER THERAPY CONCEPTS OF COHERENCE, MONOCHROMACITY, AND COLLIMATION Coherence = same wavelength & in phase Monochromacity = same color (same as wavelength) Collimation = in parallel (minimal divergence) LASER TYPES Main ones used in PT: gas lasers (He:Ne, 632 nm) and diode lasers (GaAS, 904 nm) High power surgical laser produces heat & can cut tissue (>500 W) Low power < 1 mW LASER CLASSIFICATIONS Class I infrared, GaAs Class II all visible lasers CLINICAL APPLICATIONS Largely UNKNOWN Pain (OA, 3/2009) – FDA approved but not clinically tested Wound Healing/Scar Immunological response Connective tissue stimulation (bone) Edema management (lymphedema, 11/2006) (LIGHT) PENETRATION Depth of penetration depends on the type of laser (λ) HeNe laser absorbed in first 2-5 mm (for carpal tunnel , epicondylitis) SUPERFICIAL GaAs laser absorbed in 1-2 cm MORE DEPTH IR lasers can penetrate up to 13 cm DEEPER DOSAGE Measure in Joules : 1 J = 1 W/sec Energy density J/cm2 average power Pulsed or continuous (at same output) – more energy with continuous Depends on: 1. Output of the laser (W) 2. Exposure time (sec) 3. Surface area of the LASER beam (cm2) Formula for how long to irradiate an area: TA = (E/P) X A TA = treatment time for a given area (sec) E = energy in J/cm2 P = average power of the laser (W) A = beam area (cm2) ACUTE CONDITIONS 0.05 – 0.5 J/cm2 CHRONIC CONDITION 0.5 – 3.0 J/cm2 CONTRAINDICATIONS Cancer – 1989 paper by FDA showed that tumors could be activated Do not shine in eyes Pregnancy or children ULTRASOUND ULTRASOUND Inaudible, acoustic vibrations of high frequency that may produce either thermal or nonthermal physiologic effects (18,000 Hz) CHARACTERISTICS OF WAVES Frequency Period Amplitude Intensity Propagation Speed (Velocity) **Energy is transmitted through the vibration of molecules – displaced molecules follow the acoustic wave LONGITUDINAL AND TRANSVERSE WAVES LONGITUDINAL TRANSVERSE Displacement is along the direction in which the wave travels Areas of high density compressions Areas of low density rarefactions Travel in both solids and liquids Ultrasound travels as a longitudinal wave in soft tissue Displacement occurs perpendicular to the direction of the wave Travel only in solids Ultrasound travels as a transverse wave when it contacts bone CONCEPTS OF FREQUENCY, PERIOD, AMPLITUE, INTENSITY, VELOCITY FREQUENCY How many cycles/second (Hz or MHz) Therapeutic US 0.75 - 3 MHz Frequency and shape of the wave are directly related Frequency and depth of penetration are inversely related i.e. the lower the frequency, the deeper the penetration PERIOD Time for one complete cycle Measured in seconds or microseconds AMPLITUDE Magnitude of vibration in a wave Gives indication of attenuation Decrease in energy intensity by: absorption OR dispersion and scattering INTENSITY Measure of the rate at which energy is delivered per unit area (W/cm2) Not to be confused with power, which is the amount of energy in the beam (W) VELOCITY Speed at which sound travels through a medium Denser and rigid materials demonstrate higher velocity: SOLID LIQUID GAS Soft tissue = 1540 m/s Compact bone = 4000 m/s Water = 1540 m/s (same as soft tissue because soft tissue is mainly made up of water) ACOUSTIC IMPEDENCE Determined by the product of the density of the material and speed of the sound within it PIEZOELECTRIC EFFECT Definition: when an alternating electrical current generated at the same frequency as the crystal resonance is passed through the piezoelectric crystal, the crystal will expand and contract or vibrate at the frequency of the electrical oscillation thus generating ultrasound at a desired frequency Direct piezoelectric effect = generation of an electrical voltage across the crystal when it is compressed or expanded Indirect/reverse piezoelectric effect = created when an alternating current moves through the crystal, producing compression or expansion TYPE USED TO GENERATE ULTRASOUND AT A DESIRED FREQUENCY ERA (Effective Radiating Area) total area of surface of transducer that actually produces the sound wave (~75%) CHARACTERISTICS OF ULTRASOUND BEAM Spread = the entire beam measurement in the far field Divergence = measure of one side of beam to central axis in the far field Factors affecting divergence: Size of soundhead - smaller soundhead increased divergence Frequency - smaller frequency increased divergence BNR (Beam Nonuniformity Ratio) Definition: ratio of the maximum point intensity to the average intensity across the transducer surface (ERA) Importance: The lower the BNR, the more uniform the output (less chance of hotspots) The speed and patient compliance during application The FDA requires that BNR be listed on ultrasound devices Allowed = 2:1 – 8:1 (anything above 6:1 or 7:1 significantly increases chance for hotspots) With a higher BNR, the pt. has a higher risk of getting injured (tissue damage) if the intensity is higher than 1.0 W/cm2 CONTINUOUS VERSUS PULSED ULTRASOUND Continuous = the intensity remains constant ( or 100% pulsed) Pulsed = the intensity is periodically interrupted Measured by the DUTY CYCLE 20% pulsed non-thermal effect 50% pulsed some thermal effect 100% pulsed (continuous) non-thermal effect if low intensity (0.1-0.5) is used, otherwise it is thermal DUTY CYCLE EQUATION (DURATION OF PULSE/PULSE PERIOD) X 100 = DUTY CYCLE (ex. 1 msec/5msec x 100 = 20%) INTENSITY MEASURES Spatial-averaged (SA) intensity = intensity averaged over the area of the transducer (ERA) Temporal peak intensity = maximum intensity during the on period with pulsed ultrasound Spatial-averaged temporal peak (SATP) intensity = maximum intensity occurring in time of the spatially averaged intensity (i.e. spatial average during a single pulse) WHAT IS DOCUMENTED Spatial-averaged temporal-averaged (SATA) intensity = average intensity delivered in the ERA during a pulse ****THE OBJECTIVE OF ULTRASOUND**** To provide the LOWEST INTENSITY of ultrasound energy at the HIGHEST FREQUENCY that will transmit the energy to a specific tissue and achieve desired therapeutic effect. PHYSIOLOGICAL EFFECTS, CONTRAINDICATIONS, AND INDICATIONS OF ULTRASOUND Thermal effects: Increase extensibility of collagen fibers Decrease in joint stiffness Reduction of muscle spasms Modulation of pain Increased blood flow Mild inflammatory response to reduce chronic edema Non-thermal effects: Cavitation = expansion and compression of gas-filled bubbles due to pressure changes Stable the bubbles expand & contract in response to regularly repeated pressure changes over many acoustic cycles (ONLY TYPE OF CAVITATION TO BRING ABOUT THERAPEUTIC BENEFIT) Unstable/transient violent large excursions in bubble volume before implosion & collapse occurs after only a few cycles (greater rarefaction than compression) Microstreaming = unidirectional movement of fluids along the boundaries of cell membranes Can produce changes in cell membrane permeability due to high viscous stresses BAD!! If you overstress the cell, it will die INDICATIONS CONTRAINDICATIONS Soft tissue healing (non-thermal) DVTs Pitting edema (thermal) Scar tissue & joint contracture management (thermal) Chronic inflammation (non-thermal) Bone healing (non-thermal) Other treatments - ??? Plantar wart treatment Absorption of calcium deposits Assessing stress fracture Pain reduction Placebo effects Decreased circulation (non-thermal probably OK) Eye Reproductive organs Pregnancy Pacemaker Malignant tumor (thermal can increase blood supply to the tumor) Growth plates (can cause plate to fuse faster) Plastic implants Decreased sensation MECHANISMS TO ACHIEVE EFFECTS Thermal effects Increase tissues to 40-45°C Increase tissues from baseline 1° increased metabolism/healing 2-3° decreased pain/muscle spasm 4° greater collagen extensibility & decreased joint stiffness Non-thermal effects 0.1-0.5 W/cm2; continuous (100%); spatial-averaged temporal-averaged intensity 1.0 W/cm2; 20% pulsed (duty cycle); temporal average intensity Temp change per minute = Desired change of treatment Ex. 0.4 degree = 4 degree ------------------------------ ------------------------------------------- ---------1 minute Duration of treatment 1 minute x X= 10 minutes DIFFERENCE BETWEEN 1 MHz AND 3 MHz 1 MHz for deep tissue penetration (of 5 cm) 3 MHz for shallow tissue penetration (of 2 cm) APPLICATION TECHNIQUES DIRECT APPLICATION Apply gel to skin surface & transducer IMMERSION TECHNIQUE Use for small or irregular areas Use a plastic, ceramic, or rubber basin Move transducer parallel to surface at a distance 0.5-1 cm Intensity should be increased for adequate heating (up to 50%) BLADDER TECHNIQUE If immersion is not suitable Gel should be added to both sides of the balloon/glove INDICATIONS AND TECHNIQUE FOR PHONOPHORESIS Ultrasound is used to enhance delivery of selected medication into tissue Technique: Apply medication directly onto surface Apply gel for ultrasound treatment Can use direct or immersion technique Can use pulsed or continuous Pulsed low spatial-averaged temporal peak intensity (decreased inflammation or pain) Continuous to produce inflammatory response COMBINATIONS OF MODALITIES Ultrasound and hot packs Additive heating effect Creates less dense medium, decreased penetration Ultrasound and cold packs Cold will increase the density of the tissue and improve thermal effects Recent studies have refuted this claim Ultrasound and electrical stimulation ****Well-documented and commonly used CPT CODE FOR ULTRASOUND CPT SERVICE/TIMED 97035 Timed – at least 8 mins $$$$$ $11.80/unit DOCUMENTATION OF ULTRASOUND Record the specific parameters Frequency Spatial-averaged temporal peak intensity Pulsed or continuous Duty cycle (if pulsed) ERA Duration of treatment Number of treatments/week Non-thermal US is beneficial for inflammatory and proliferative phases of healing Thermal US is appropriate for the maturation phase MASSAGE EFFECTS OF MASSAGE PHYSIOLOGICAL Type of effects depends on stroke/technique Inhibitory/relaxing (slow, long, rotational strokes) effleurage, petrissage Excitatory/stimulating (quick, short strokes) percussion, friction, vibration, knuckling Inhibitory to voluntary mm deep tendon pressure or stretch (Charlie horse, cramps) Reflex Stimulation of sensory receptors in the skin & superficial tissues increase body awareness Mechanical Stretching, elongating, or mobilizing techniques Should perform after reflexive techniques REFLEXIVE Red flare or streak, arteriole dilation (BAD) Effects on capillaries in the skin this is determined by the amount of pressure - 1st a white reaction or blanching - 2nd a red reaction or increase in local circulation (if not a local response, can indicate cellulitis) **Keep in mind: massage done to only one area does not affect total body blood flow Red wheal (welt), release of histamines & prostaglandins (BAD) should go away within 24 hrs Decrease in pain (NORMAL) gate control vs. opiate Changes in chemicals in the blood flow (i.e. bicarbonates) German studies Increase of lymph flow lymph system important in tissue nutrition, healing, & immunity by removing metabolic waste & edema MECHANICAL Deep work can be unpleasant & cause soreness On soft tissue Stretching tissue, increase in flexibility & length (passive stretch & traction techniques) Can break up or prevent fibrotic mm (X-fiber technique) fibrotic cording in breast cancer Can increase muscle health through decreasing metabolic acids (lactic, uric), improving tissue nutrition & enhancing waste exchange can aid in ridding fatigue of mm Decreases scar tissue Effects on pulmonary system percussion/tapotement strokes can loosen mucous plugs when combined with postural drainage Local capillary dilation via splanchnic autonomic fibers Relaxation of voluntary muscle Sedation of pain stimuli (gate theory) Increase in skin temperature INDICATIONS For edema of venous/lymph Garage Theory = if there is edema, always massage the proximal area first (because lymph dumps into lymph nodes proximal to the edematic area & you need to clear them first) Promote wound healing getting rid of metabolic byproducts To decrease/prevent adhesions fibrotic scar tissue Pulmonary congestion To relax muscle or whole body CONTRAINDICATIONS Edema from total system failure (i.e. CHF) Acute inflammatory edema Any area known or with suspected clot (DVT) Any site of known or suspected aneurysm Tumors (exception in terminal cases, massage can be used for comfort) Over open lesions, conditions that spread Abnormal abdominal mass Non-union fractures Graft sites PRECAUTIONS Chronic fatigue syndrome – massage could increase fatigue Fracture sites – even ones that have been set Osteoporosis Hypersensitive to touch Diabetes mellitus – decreased sensation Complicated pregnancies High BP Asthma Alcoholism Psychiatric illness – never know how they will respond to initial touch Open lesions on pt. Children with shunts (ventriculoperitoneal) – take care with positioning (head elevated) TERMINOLOGY Effleurage = long stroking, gliding movements following length of muscle (origin insertion) Used to apply lubricant Begins and ends massage Accustoms patient to touch before deeper work PT can listen to hands & search for hot spots, edema, tightness, & other types of injury Petrissage = kneading, compress & release movements used to lift subcutaneous tissue up & off underlying structures Applied uni or bi-manually with open “C” type position of the hand (no open fingers) Breaks up soft tissue tightness Friction = small circular strokes with deep pressure done with the thumb; linear strokes done with sides of hand, finger tips, back of hand, or heel of hand Used to loosen/soften tight muscle tissue breaks up adhesions X-fiber, rolling, wringing Tapotement = percussive strokes using alternating hands in rapid rhythmic motion Practitioners hand should be relaxed Tapping = striking with fingertips (on face) Hacking = striking with ulnar borders of hand Cupping = striking with curved (cupped) hands Pinchment = alternate pinching with thumb and index finger (for localized areas) Vibration = transmitting trembling motion from PT hands onto tissue Use after deep pressure of trigger point increases circulation into the area Used for joints after deep stroking **Flow of movement: EFFLEURAGE PETRISSAGE FRICTION AND/OR TAPOTEMENT AND/OR VIBRATION PETRISSAGE EFFLEURAGE COMPONENTS OF MASSAGE 1. Lubricant 2. Draping 3. Positioning 4. Behavior of PT Remember to have a relaxed demeanor – puts pt. at ease Always be professional – avoid leading with fingertips Always inspect the area for skin condition before beginning Remember: the first touch should be done with respect, addressing the skin, not invading it 5. Preparation Pt. should remove as much clothing as is comfortable – all clothing removed for full body massage 6. Application Body mechanics – table height, hands relaxed, move from the hips and knees, knees bent, deep breathing, avoid hyperextension of thumbs/fingers, do not lock elbows **Once beginning the massage, try not to break contact, gauge pressure carefully, never apply pressure over the spinous processes, try to make transitional movements as smooth as possible CPT CODES 97124 – Massage 97140 – Manual therapy/Trigger point INTRODUCTION TO ELECTRICAL STIMULATION GENERAL INDICATIONS FOR USING E-STIM Supplementation of functional activity Strengthening of muscle (atrophied muscle) Reduction of acute pain Reduction of chronic pain GENERAL CONTRAINDICATIONS FOR USING E-STIM Pts. with an electronic demand-type cardiac pacemaker Over the carotid sinus Internally (may damage mucosal linings) Over the eyes Transcranially or in the upper cervical regions in pt. with history of CVA, TIA, or seizure Transthoracically (careful of heart) Over any area that would have a tendency to hemorrhage Over laryngeal or pharyngeal muscles GENERAL SET-UP FOR E-STIM Clip excessive hair Use an alcohol prep to remove skin oils Apply a coupling agent (like US gel) to the skin under the electrode (unless electrode is pre-gelled) Place electrodes in such a way that the motor end plate is between the electrodes The motor end plate is generally found at the proximal third of the muscle Orient the electrodes in the direction of the muscle fibers For Russian e-stim, place the active electrode directly over the motor point Electrode placement is CRITICAL: “Electrode placement is probably one of the biggest causes of poor results from electrotherapy.” GENERAL INFORMATION ON CURRENT FLOW Current flow can be either unidirectional or bidirectional Types of current flow: Direct o Unidirectional and continuous o One second or longer Alternating o Bidirectional Pulsatile o Unidirectional or bidirectional (alternating polarities) o Interrupted o Milliseconds or less duration PHASE- current flow in ONE direction for a finite period of time (i.e. one polarity) If phase duration is too short, there will be no action potential PULSE- the time from the beginning to the end of an electrical event, even if the waveform leaves and returns to the baseline several times (i.e. can be one or more polarities); may contain one or more phases Pulse duration = length of time the electrical flow is “on” (i.e. pulse width) Pulse rise time = the time it takes to reach peak intensity (i.e. ramp) o Rapid rising pulses cause nerve depolarization o Slow rising pulses cause nerve accommodation and an action potential is not elicited - Good for muscle re-education with assisted contraction with ramping, the shock of the current is reduced, mimicking more of a functional control CHARACTERISTICS OF WAVEFORMS Types of waveforms Square Sine Spike Monophasic vs. Biphasic Phase refers to the number of current flow switches per pulse Monophasic polarity stays the same Biphasic polarity switches two times Polyphasic polarity switches multiple times Symmetrical vs. Asymmetrical Refers to the amount of time and amplitude of each phase of the pulse Symmetry same time and amplitude Asymmetrical either the time or the amplitude are different, or both are different Balanced vs. Unbalanced Refers to the amount of charge being delivered (i.e. the area “under” the waveform) Balanced amount of charge is the same Unbalanced amount of charge is different FREQUENCY- number of pulses per unit of time Measured in pulses per second (pps) or beats per second (bps) for monophasic/pulsatile current (DC); cycles per second (cps or Hz) for biphasic current (AC) DUTY CYCLE- ratio of on time/total cycle time 1:1 – fatigues muscle rapidly 1:5 – less fatigue 1:7 – no fatigue (passive muscle exercise) ELECTRODE PLACEMENT AND SIZE Monopolar – one active electrode (DC) Bipolar – two active electrodes Quadripolar – four active electrodes Place the electrodes such that the motor point is between them (or the active electrode is over the motor point for Russian) Electrode spacing: Make sure there is at least one “electrode width” between each electrode Current density is affected by spacing of the electrodes o Closer together more superficial current density o Farther apart deeper penetration of current density Electrode size: A small electrode can still deliver a lot of current to a localized area will give the greatest effect to the nerve or motor point (use a larger electrode farther from the treatment area for DC current) A large electrode disperses the current over a wider area TERMS OF ELECTRICTY Electrical current = the flow of energy between two points Needs a voltage (electromotive force) and a conductor (material/tissue which allows free flow of energy) Two modality classifications: 1. Hi Volt – greater than 100-150 V 2. Low Volt – less than 100-150 V MOST COMMON Ampere = the amount of current AMPLITUDE- the intensity of the current ; Measured in millivolts (mV) o Associated with the depth of penetration The deeper the penetration, the more muscle fiber recruitment possible Arndt-Schultz Principle – have to have sufficient stimulation to get the potential to its threshold point CLINICAL APPLICATION OF ELECTRICITY Temperature Relationship: an increase in temperature increases resistance to current flow need to either raise the intensity of the e-stim to compensate OR put the hot pack on the pt. after completing e-stim treatment Applicability: preheating the treatment area may increase the comfort of the treatment, but it also increases the resistance and the need for higher output intensities Length of circuit Relationship: greater the cross-sectional area of a path, the less resistance to current flow Applicability: nerves of larger diameter are depolarized before nerves of smaller diameter Material of the circuit Excitable tissues include: nerves, muscle fibers, blood cells, cell membranes Non-excitable tissues include: bone, cartilage, tendons, ligaments CONDUCTION Conductors include: muscle, nerve, loose collagen, tendon, ligament Insulators include: fat, skin Poorest conductor = BONE Nerve conducts 6x faster than muscle, but is surrounded by a fat layer and sheath RESISTANCE (R) Definition: relative opposition to movement of charged particles Skin thickness, level of hydration, skin cream all affect current flow in our clients The gel on the back of the electrode helps to reduce resistance Measure is ohms (Ω) with DC or impedence (Z) with AC Other factors affecting resistance: Material composition Length greater length yields greater resistance Temperature increased temperature = increased resistance For a muscle contraction: lower frequency (20-40 Hz), longer duration (>300 μs) For a sensory stimulation: higher frequency (80-120 Hz), shorter duration (50-300 μs) TRICKS WITH AMPLITUDE MODULATION AND FREQUENCY MODULATION Accommodation = getting used to a feeling and diverting attention from it adjust AM or FM to delay it ELECTRODES Cations = positively charged atoms Anions = negatively charged atoms Cathode = negative terminal, attracts cations Negative pole (cathode) has a high concentration of electrons Anode = positive terminal , attracts anions Positive pole (anode) has a low concentration of electrons ACTION POTENTIALS AND DEPOLARIZATION Each cell membrane has a voltage sensitivity permeability The voltage sensitivity permeability determines resting potential (inside = -70 mV, outside = -90 mV) Changes in the membrane permeability create an action potential resulting in depolarization Depolarization normally occurs at the cathode (concentration of negative ions) Shift in polarity (positive) due to Na+ influx The cell will repolarize and restore its resting potential (K+ efflux) Changes at the NMJ cause the contraction of muscle TYPES OF MUSCLE CONTRACTIONS Described according to the pulse width Twitch = 1 pps (low frequency) Treppe/summation = 10 pps Tetanus = 25-30 pps (most fibers will reach tetany by 50 pps) Initial tetany = 25-30 pps Mid tetany = 30-40 pps Full tetany = >40 pps 300-600 ms pulse width for all of these FIBER TYPES A-alpha Efferent to muscle (extrafusal) fibers A-beta Afferent from discriminative touch, pressure receptors A-delta Afferent from pain and temperature receptors (fast pain) III C Afferent from pain receptors and free nerve endings (slow pain) IV Axon myelination and diameter directly affect depolarization by e-stim o A-alpha fibers are myelinated C fibers are not myelinated o Large diameter: A-alpha ↔ A-beta ↔ A-delta ↔ C : small diameter GENERAL RULES TO REMEMBER FOR MUSCLE CONTRACTION Larger fibers have a lower threshold to depolarization from externally applied electrical current Large fibers offer less resistance to ion movement and therefore conduct action potentials faster Larger fibers will display conduction failure before smaller fibers in the presence of asphyxia/anoxia, compression, and cold more sensitive than smaller fibers to mechanical pressures Smaller fibers will display conduction failure before larger fibers in the presence of anesthetics or toxins PHYSIOLOGIC VS. ELECTRICAL INDUCED MUSCLE CONTRACTION VOLUNTARY Asynchronous firing Varied motor units: less fatigue First, small slow twitch fatigue resistant fibers, then large Graded movement depending on load ELECTRICAL INDUCED Synchronous firing Same motor units respond Large fast twitch fibers All or nothing, depending on intensity FREQUENCY SELECTION FOR DESIRED OUTCOME Pain relief = 100 Hz Muscle contraction = 50-60 Hz Increased circulation (1:1 ratio for on/off time) = 1-50 Hz The higher the frequency (Hz) the more quickly the muscle will fatigue GATE THEORY OF PAIN CONTROL Theory: stimulating bigger nerve fibers will override any signals coming from smaller ones through inhibitory interneurons Since bigger nerves transmit impulses faster, they may elicit high frequency activation of the inhibitory interneurons that “shut down” the firing of pain fibers This is good because our big nerve fibers are touch and our small nerve fibers are achy, dull pain Two ways to close the “gate” on pain: 1. Block pain at the spinal cord level via inhibitory interneurons 2. Change activity at the brainstem level activate the periaqueductal gray or Nucleus Raphae IONTOPHORESIS BASIC PRINCIPLES OF APPLICATION Electrical current causes topically applied ions to migrates TOWARDS the oppositely charged electrode and AWAY FROM the similarly charged electrode Knowledge of a drug’s or ion’s polarity is critical for choosing the correct polarity of the electrode needed to drive the drug into the underlying treatment area DRUG POLARITY DRUG ELECTRODE DISPERSIVE PAD Negative (-) Black (-) = CATHODE Red (+) = ANODE Positive (+) Red (+) = ANODE Black (-) = CATHODE INDICATIONS FOR IONTOPHORESIS Frequently used for lateral epicondylitis, Achilles and patellar tendinitis, ITB syndrome, and CTS General indications: Pain Inflammation Edema Calcium deposits Hyperhydrosis CONTRAINDICATIONS OF IONTOPHORESIS Near venous/arterial thrombosis or thrombophlebitis Over areas of impaired sensation Cardiac pacemakers (or any implant) IONIC CHARACTERISTICS Current type = DC required to provide polarity for drug “push” Cathode: alkaline reaction occurs, NaOH forms Anode: acidic reaction occurs, HCl forms Some commercial electrodes have built in buffering agents to help control ionic reactions MEDICATIONS Dexamethasone (DSP) is the most commonly used steroid to treat inflammation DSP has a (-) polarity and must be placed under the cathode to drive it into the underlying target tissue A referral and medication prescription are required (must be specific) The simultaneous delivery of two medications under one electrode is popular, but is also controversial It is recommended that only one medication be delivered Iontophoresis is current limited, meaning if using two drugs at the same time, ionic competition can occur (if the polarities between the two meds differ) Common medications used: ION SOURCE POLARITY INDICATIONS % Acetate Acetic acid (-) Calcium deposits 2.5-5.0 Dexamethasone DexNa2PO3 (-) Inflammation 0.4 Lidocaine Lidocaine (+) Pain 5 Salicylate NaSal (-) Inflammation 2 Water (+/-) Hyperhydrosis NA Zinc ZnO2 (+) Wounds NA FACTORS AFFECTING DRUG DELIVERY TO TARGET TISSUE Maximum depth of drug ion penetration has not been established Estimated to be 1 cm or less Skin thickness Thickness of subcutaneous tissues Muscle mass **Follow-up with treatment that increases circulation TERMINOLOGY USED IN IONTOPHORESIS Current amplitude applied depends on: Pt. tolerance Polarity of delivery electrode (alkaline reactions more reactive with skin) Size of delivery electrode – large can take up to 4.0 mA Length of treatment APPLICATION OF IONTOPHORESIS Set-up: Clip hair if needed Cleanse skin with alcohol wipe Apply electrode in monopolar arrangement Current type: DC Current amplitude: ≤4.0 mA Treatment duration: 10-40 min Dosage: 40-80 mA•min Polarity: delivery electrode and active ion should be the same Treatment frequency: 24-48 hr separation Total # of treatments: positive results should be obtained within 4-5 treatments CURRENT PARAMETERS USED IN IONTOPHORESIS Any current dosage can be achieved by multiple amplitude-duration combinations DOSAGE AMPLITUDE DURATION 40 mA•min 2 mA 20 min 40 mA•min 3 mA 13.5 min 40 mA•min 4 mA 10 min PRECAUTIONS OF IONTOPHORESIS Monitor the pt. closely during treatment, checking for a local or systemic reaction Minimize skin irritation Size of electrode – current density should not exceed 0.5 mA/cm2 if the cathode is used or 1.0 mA/cm2 if the anode is used CHEMICAL BURNS Most common problem associated with iontophoresis Occur because of the DC current, not the ion used in the drug Skin will appear pink and dry, irritated-looking Allow skin to heal do not apply to the same area and change parameters to prevent future burns Can minimize the potential for burns by decreasing the current density: Increase the size of the electrode Decrease the current intensity Space the electrodes accordingly Change the electrode location between treatments RUSSIAN ELECTRICAL STIMULATION OBJECTIVE OF RUSSIAN Medium frequency goes deeper into the muscles to give a more intense contraction Russian is only for muscle strengthening GOAL: create a tetanic contraction Similar to NMES usage for muscle strengthening CHARACTERISTICS OF RUSSIAN Type of NMES because the muscle is directly stimulated Considered “medium frequency” Polyphasic AC waveform (lots of modulation) Net physiologic effect: Independent of intrinsic number of pulses per burst o Nerve/muscle membrane EACH burst of pulses (Russian) is seen as a SINGLE PULSE o Result = repeated delivery of bursts or beats of pulses induces motor nerve/motor unit depolarizations and tetanic contractions Time modulation of continuous sine or square wave with carrier frequency of 2,500 pps Typical “Russian current” has a burst frequency of 50 bursts/second (50 pulses/burst) The “burst intervals” make the current intensity more tolerable, decrease tissue impedence, and increase the number of motor units activated Fixed interburst and interpulse intervals PARAMETERS OF RUSSIAN Pulse duration: 50-250 μs; may be sine or square wave Intensity: enough for desired muscle contraction without undue discomfort Polarity: irrelevant because it is biphasic Current density: avoid too narrow try to use larger electrodes that are farther apart APPLICATION OF RUSSIAN Inter-electrode distance: not less than diameter of smallest electrode Electrode orientation: “line of pull” Electrode attachment: good coupling Location: electrodes over at least one motor point Sample treatment protocol: 10/50/10 10 seconds on 50 seconds off 10 minute treatment session TRANSCUTANEOUS ELECTRICAL NERVE STIMULATION (TENS) TENS Light weight units 2 channels Variety of features depending on the unit ****GOLD STANDARD for acute pain management**** ELECTRODE LAYOUT Use electrodes of the same size for biphasic/AC or different sizes for monophasic/DC (smaller electrode over treatment area) All electrodes are active so place accordingly: Bipolar – two active electrodes Quadripolar – four active electrodes; can cross channels (IFC) or have them parallel (NMES) GATE CONTROL THEORY (discussed previously) CENTRAL BIASING THEORY Stimulation of small fibers (C or pain fibers) for short time periods Peripheral sites Stimulation of descending neurons Closes the gate at the spinal cord level, affecting the transmission of pain OPIATE PAIN CONTROL THEORY Release of enkephalin and endorphins Endorphins released from pituitary gland and hypothalamus into the CSF CONTRAINDICATIONS OF TENS Cardiac pacemakers History of cardiac arrhythmias Phrenic or urinary bladder stimulators Deep brain stimulators Carotid sinus (scalene triangle) Temporal lobe Eyes Larynx locations Malignancy Superficial skin lesion Pelvic region in pregnant females Anxiety LEVELS OF PAIN INHIBITION LEVEL 1 – periphery (subsensory) LEVEL 2 – occurs in dorsal horn (sensory) LEVEL 3 – involves hormonal system (motor) LEVEL 4 – involves brainstem sites and dorsolateral funiculus (noxious) LEVEL 5 – involves cortical area (placebo) LEVEL 1 – SUBSENSORY LEVEL Microcurrent Electrical Nerve Stimulators (MENS) Can be beneficial for bone healing Peak amplitude below 1 mA, too low to stimulate either nerve or muscle LEVEL 2 – SENSORY LEVEL Conventional TENS most used and most studied (for acute pain) At or above the sensory threshold, but below motor threshold Mechanisms: Direct peripheral block of transmission Central inhibition of original gate-control theory Should not see a twitch in muscle, just “pins and needles” feeling (i.e. no muscle contraction) Indications for Conventional TENS: Any painful musculoskeletal condition Use with ice or heat Post-op management of pain Use when muscle contraction increases pain or is contraindicated except when the pain increases substantially Acute injuries LEVEL 3 – MOTOR LEVEL Low-rate TENS primarily used for subacute and chronic pain Pts. may describe pain as deep and throbbing Not as comfortable as sensory stimulation Visible muscle contraction Response is not immediate, but longer-lasting Mechanisms: Central inhibition of original gate-control theory Opiate Pain Theory (endorphin release) Indications for Motor TENS: Chronic pain Trigger points Muscle guarding FYI’s: Relief from 1-6 hrs Can use Conventional TENS first for rapid onset of relief and then apply low-rate Motor TENS Recommended to place electrodes in an area remote to pain site, but in same or related myotome LEVEL 4 – NOXIOUS LEVEL Hyperstimulation TENS seldom used Electrodes placed over painful site Brief treatment: 30-45 seconds Stimulation lasts for 1-6 hrs Daily treatments may reduce effectiveness (accommodation) Variable parameters OBJECTIVE: pt. to perceive stimulant as noxious Mechanism: Endorphin-mediated (Opiate Pain Theory) release of endogenous opiates Central Biasing Theory LEVEL 5 – PLACEBO LEVEL Well-documented Depends on pt.’s belief in the treatment Placebo effects are common in all treatments BIOFEEDBACK DEFINITION Information provided to the pt. about biologic function Common biofeedback instruments: Plethysmography measures diameter; changes in volume or air (Rx: ED, respiration, relaxation) Electrogoniometry measures joint angle (Rs: ROM) Electromyography measure neuromuscular activity; MOST COMMON (Rx: stiffness, muscle retraining, relaxation) ULTIMATE GOAL: encourage appropriate motor activity BENEFITS OF BIOFEEDBACK Gives the pt. a lot of good, real-time info but forces the pt. to do the work on their own Can be employed during functional tasks Non-invasive Fits better with the mechanisms of use-dependent plasticity and task-specific training than e-stim or bracing DRAWBACKS OF BIOFEEDBACK People can become dependent on feedback need to “wean” pts. off People seem to fall back into their old habits (i.e. poor retention) Research doesn’t strongly support it (difficult to quantify because of its subjectivity) GAIN Definition: ratio of input to output High gain: 1x ↔ 10x ↔ 100x : Low gain Directly related to sensitivity/inversely related to threshold – if you increase the gain, you increase the sensitivity (lower threshold); if you decrease the gain, you decrease the sensitivity (higher threshold) For severe muscle weakness INCREASE THE GAIN For eliciting a bigger contraction or encouraging a maximal contraction DECREASE THE GAIN FREQUENCY Definition: how often activity is measured For minimal muscle activity INCREASE THE SAMPLING FREQUENCY (to pick up any twitches) For muscle spasms DECREASE THE SAMPLING FREQUENCY (to facilitate relaxation) AREA Definition: size of area to be sampled To monitor more muscle units INCREASE THE RECORDING AREA Two ways to modify the area of recording: 1. Size of electrodes 2. Distance between electrodes GENERAL GUIDELINES FOR SUCCESS WITH BIOFEEDBACK The pt. needs to be cognitively aware enough to understand the feedback The pt. should have some volitional (voluntary) control of the muscle Make it functional! NEUROMUSCULAR ELECTRICAL STIMULATION (NMES) FACTS ABOUT NMES Also called Electrical Muscle Stimulation (EMS) Uses a biphasic current Purist Theory: the active electrode should be negative (placed distally) and positive electrode placed proximally POTENTIAL USES FOR NMES Muscle dysfunction due to CNS lesion (CVA, spinal cord, TBI) Post-operative orthopedic – increase strength Disuse atrophy Peripheral nerve injuries ?? CONTRAINDICATIONS AND APPLICATION OF NMES Same as TENS NMES OUTCOMES Muscle re-education Indication: muscle inhibition after surgery or injury (pain automatically causes muscle inhibition) Only a factor if neuromuscular mechanisms are intact – a totally deinervated muscle will NOT work by NMES Increase sensory input to the muscle OBJECTIVE: to reestablish control Edema reduction Indication: to duplicate regular muscle contractions to increase circulation through venous and lymphatics (mimic muscle pump) Use with ice and elevation for best results Contracture management Indication: increased ROM with prolonged stretching Muscle strengthening Indication: increased force production Research: article on NMES for strengthening quadriceps femoris o NMES makes sense compared with doing no exercises but voluntary exercises appear to be more effective in most situations o NMES may only be preferred over voluntary exercise for within-cast muscle training and perhaps in specific situations where voluntary training does not receive sufficient pt. compliance STIMULATION OF DEINERVATED MUSCLE THEORY: prevention of muscle atrophy, edema and venous stasis, and decreased degeneration E-stim has no effect on nerve regeneration Some studies suggest that it may interfere with nerve regeneration by traumatizing the deinervated muscle Not enough evidence to support the use of e-stim of deinervated muscle! FUNCTIONAL ELECTRICAL STIMULATION Uses multiple channel stimulators to recruit muscles in a sequence to facilitate functional movement through a microprocessor Current devices use surface electrodes needle electrodes in research Clinical applications: CVA and SCI MICROCURRENT ELECTRICAL STIMULATION GENERAL INFO ABOUT MICROCURRENT Other names: Low Intensity Stimulation (LIS); formerly known as MENS Objectives: Simulate the “body’s current” Aid healing at the cellular level – for bone and wound healing Characteristics: Very low amplitude currents – most units <1 mA No activation of sensory nerve fibers Waveforms are rectangular, monophasic with periodic reversal of polarity MENS/MES/LIS EFFECTS FOR WOUND HEALING Increases blood flow to wounded areas Increases cell proliferation, migration, and motility Increases DNA synthesis (promoted by polarity) Increases collagen synthesis Increases growth factor receptor levels Enhances electrical potential gradients of stimulated cells MENS/MES/LIS EFFECTS FOR CHRONIC PAIN Approved by the FDA for “the symptomatic relief of chronic intractable pain and as an adjunctive treatment in the management of post-surgical traumatic pain problems” No evidence for the measureable effectiveness of MENS (or any other electrotherapy) below sensory threshold to decrease pain INDICATIONS FOR LOW INTENSITY STIMULATION Wounds, such as skin ulcers Fractures – humerus, tibia, clavicle, radius, ulna, femur, fibula, MTs, MCs Healing post-surgical spinal fusion – Capacitive Coupling ***Bone healing Can be implanted or external Used when healing is delayed – 90 days w/o progress on serial x-ray (HCFA Medicare criteria) or 1 cm gap or less on x-ray (CIGNA) Pts use for extended multiple daily periods Care is prescribed and monitored by physician – not by PT HIGH VOLTAGE PULSED CURRENT (HVPC) HVPC CHARACTERISTICS Twin-peaked, monophasic, pulsed current, DC High electromotive force (voltage): 150-500 V HVPC pulse duration = phase duration of both spikes: 100-200 μs, fixed duration Short stimulation periods: interpulse time is approx. 50x as long as stimulation time Interval between waves (one wave is twin-peaked) can be adjusted i.e. pulse interval from one twin-spike to the next twin-spike As the time decreases and spikes overlap, stronger stimulation is perceived Acid reaction under anode and alkaline reaction under cathode Output polarity constant during stimulation Pulse frequency: 1-200 pps Tissue impedance – HV encounters less capacitance; therefore, less impedance and more comfortable INDICATIONS FOR HVPC Pain Edema Muscle weakness Wound management – GOLD STANDARD CONTRAINDICATIONS OF HVPC Over neoplastic regions Heavy scarring Thick adipose tissue Extreme edema Osteomyelitis - has to be resolved prior to use because HVPC will mask it (with granulation tissue) Anterior cervical area Transthoracic region Transcranial area Pregnancy (over lumbar and abdomen) Hemorrhagic area Electronic implants PHYSIOLOGIC EFFECTS OF HVPC Increased circulation Pain therapy Possibly via gate-control theory or by reducing spasm Muscle weakness research shows its ineffective Dermal wounds Promotes healing by increasing “current of injury”, maintenance of polarity (GOLD STANDARD) Reduces soft-tissue edema Muscle pumping – increased with greater contraction strength and lower frequencies Studies suggest acutely “post-histamine microvessel leakiness” is reduced – retards edema formation (cathode over injury site) Muscle spasm (similar to low-rate TENS) PARAMETERS FOR VARIOUS HVPC USES ***Dermal wound therapy Polarity: anode at/near site (clean dermal wound should be negatively polarized) Frequency: arbitrary 30-200 pps Mode: continuous Amplitude: arbitrary 1-500 V, submotor Duration: arbitrary 10-60 min Pain therapy Frequency: arbitrary 1-200 pps Mode: continuous Amplitude: arbitrary 1-500 V Duration: arbitrary 10-20 min Edema/Spasm/Weakness Produce a tetanic muscle contraction Frequency: arbitrary 30-60 pps Mode: pulsed, on/off 1:5 or 1:3 Amplitude: arbitrary 1-500 V Duration: arbitrary 5-30 min PRECAUTIONS OF HVPC Chemical burn Excessive electrical density o Intensity too high for size of the active electrode o Direct metal contact INTERFERENTIAL ELECTRICAL STIMULATION (IFC) INTERFERENTIAL EFFECTS Physiologic effects: Set to depolarize peripheral sensory or motor fibers Therapeutic effects: Pain, urinary incontinence, blood flow/edema management, spasm reduction INTERFERENTIAL CHARACTERISTICS Two channel stimulators AC current at 2000-5000 Hz Medium frequency decreases impendence at skin Deeper current flow Crossing quadripolar set-up: Currents from one electrode “interfere with” current from the other, resulting in “interference current” with beat frequency equal to the difference between the two channels Each channel has its own frequency Example: Channel 1 = 3050, Channel 2 = 3000 BEAT FREQUENCY = 50 Beat frequency should be selected to correspond to the desired effect over the treatment area Pain = 80-150 bps Edema = 1-10 bps (muscle pump) Muscle rehab/strengthening = 20-50 bps Premodulated IF estim = beat frequency and summation of currents occurs inside the machine (is a homogenous medium vs. body tissues) Methods of delivery: 1. Bipolar: 2 electrodes, 2 medium frequency currents (1 channel) oval shaped field 2. Quadripolar (crossing): 4 electrodes, 2 unmodulated medium frequency sine currents (2 channels) Interference at level of treatment area “4 leaf clover” shaped field 3. Dual (not crossing), bipolar: 4 electrodes, 2 unmodulated medium frequency sine currents (2 channels) Interference area is much smaller Anecdotal decreased spasm 4. Quadripolar with vector scan: enlarge field Current amplitude of 1 circuit varies 50-100% of max, other circuit fixed at one amplitude Creates vector scan (field rotates) circular shaped field of interferential current INTERFERENTIAL DOSAGE Modes of delivery Constant preset beat frequency or set by user Sweep preset beat frequency range Carrier frequency preset by manufacturer Electrode placement – cover desired treatment area If doing IFC for pain, painful area should be located within the 4 electrodes If doing IFC for motor response, motor point should be located within the 4 electrodes OVERVIEW OF E-STIM: WHEN TO USE WHAT FOR STRENGTHENING: (1) NMES (2) IFC – quad set up (large muscle belly) or pre-mod (smaller/focused strengthening) (3) HVPC Russian best to use for athletes strengthening (exaggerated, intense); AC, pulsed, burst FOR PAIN (AND CREATING A MUSCLE CONTRACTION): Acute: Conventional TENS (AC) Acute: IFC (quad set up for large, pre-mod for smaller) Acute: HVPC GOAL FOR ACUTE = initiate gate-control theory Chronic: low-rate (motor) TENS or noxious TENS Chronic: HVPC Chronic: NMES GOAL FOR CHRONIC = fatigue muscle FOR EDEMA: HVPC – pulsed DC NMES – AC, biphasic IFC – interference Ionto (steroid) – DC FOR BONE HEALING: Micro – GOLD STANDARD DC or AC Pulsed US Wound Healing Characteristics of Arterial and Venous Insufficiency Ulcers Arterial Ulcers Venous Ulcers Location Lower 1/3 of leg, toes, web spaces (distal toes, dorsal foot, lateral malleolus) Proximal to the medial malleolus Appearance Smooth edges, well defined; lack granulation tissue; tend to be deep Irregular shape; shallow Pain Severe Mild to moderate Pedal Pulses Diminished or absent Normal Edema Normal Increased Skin Temperature Decreased Normal Tissue Changes Thin and shiny; hair loss; yellow nails Flaking, dry skin; brownish discoloration Miscellaneous Leg elevation increases pain Leg elevation lessens pain The Wagner Ulcer Grade Classification Scale Commonly used as an assessment instruction for the evaluation of diabetic foot ulcers Grade No open lesion but may possess pre-ulcerative lesions; healed ulcers; presence of bony deformity 0 Superficial ulcer not involving subcutaneous tissue 1 Deep ulcer with penetration through the subcutaneous tissue; potentially exposing bone, tendon, ligament, or 2 joint capsule Deep ulcer with osteitis, abscess or osteomyelitis 3 Gangrene of digit 4 Gangrene of foot requiring disarticulation 5 Pressure Ulcer Staging Stage I o An observable pressure related alteration of intact skin whose indicators as compared to an adjacent or opposite area on the body may include changes in skin color, skin temperature, skin stiffness, or sensation Stage II o A partial-thickness skin loss that involves the epidermis and/or dermis. The ulcer is superficial and presents clinically as an abrasion, a blister or a shallow crater. Stage III o A full-thickness skin loss that involves damage or necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia. The ulcer presents clinically as a deep crater with or without undermining adjacent tissue. Stage IV o A full-thickness skin loss with extensive destruction, tissue necrosis or damage to muscle, bone, or supporting structures (e.g. tendon, joint capsule) Bony Prominences Associated with Pressure Injuries Supine Prone Occiput Forehead Anterior portion of Spine of the scapula acromion process Inferior angle of scapula Anterior head of humerus Vertebral spinous processes Medial epicondyle of humerus Posterior iliac crest Sacrum Coccyx Heel Sternum Side-lying Ears Lateral portion of acromion process Lateral head of humerus Lateral epicondyle of humerus ASIS Greater trochanter Patella Dorsum of foot Head of fibula Lateral malleolus Medial malleolus Sitting (chair) Spine of the scapula Vertebral spinous processes Ischial tuberosities Types of Dressings: Hydrocolloids Consist of gel-forming polymers with a strong film or foam adhesive backing. Hydrocolloids absorb exudate by swelling into a gel-like mass. The dressing does not attach to the actual wound itself and is instead anchored to intact skin surrounding the wound. Advantages o Provides a moist environment for wound healing o Enables autolytic debridement o Offers protection from microbial contamination o Provides moderate absorption o Does not requires a secondary dressing o Provides a waterproof surface Disadvantages o May traumatize surrounding intact skin upon removal o May tend to roll in areas of excessive friction o Cannot be used on infected wounds Hydrogels Indications o Hydrogels are commonly used on superficial and partial-thickness wounds (e.g. abrasions, blisters, pressure ulcers) that have minimal drainage. Rather than absorb drainage, hydrogels are moisture retentive. Advantages o Provides a moist environment for wound healing o Enables autolytic debridement o May reduce pressure and diminish pain o Can be used as a coupling agent for ultrasound o Minimally adheres to wound Disadvantages o Potential for dressings to dehydrate o Cannot be used on wounds with significant drainage o Typically requires a secondary dressing Foam Dressings The dressings are hydrophilic at the wound contact surface and are hydrophobic on the outer surface. The dressings allow exudates to be absorbed into the foam through the hydrophilic layer. . Indications o Used to provide protection over partial and full-thickness wounds with varying levels of exudate. They can also be used as secondary dressings over amorphous hydrogels. Advantages o Provides a moist environment for wound healing o Available in adhesive and non-adhesive forms o Provides prophylactic protection and cushioning o Encourages autolytic debridement o Provides moderate absorption Disadvantages o May tend to roll in areas of excessive friction o Adhesive form may traumatize periwound area upon removal o Lack of transparency makes inspection of wound difficult Transparent Film Film dressings are thin membranes. The dressings are permeable to vapor and oxygen, but are mostly impermeable to bacteria and water. Indications o Useful for superficial wounds (scalds, abrasions, lacerations) or partial-thickness wounds with minimal drainage Advantages o Provides a moist environment for wound healing o Enables autolytic debridement o Allows visualization of the wound o Resistant to shearing and frictional forces o Cost effective over time Disadvantages o Excessive accumulation of exudates can result in periwound maceration o Adhesive may traumatize periwound area upon removal o Cannot be used on infected wounds Gauze Indications o Commonly used on infected or non-infected wounds of any size. The dressings can be used for wet-towet, wet-to-moist, or wet-to-dry debridement Advantages o Readily available, cost effective dressings o Can be used alone or in combo with other dressings or topical agents o Can modify number of layers to accommodate for changing wound status o Can be used on infected or uninfected wounds Disadvantages o Has a tendency to adhere to wound bed o Highly permeable and therefore requires frequent dressing changes (prolonged use decreases cost effectiveness) o Increased infection rate compared to occlusive dressings Alginates o Extracted from seaweed. Alginates are highly permeable and non-occlusive. As a result, they require a secondary dressing. o Indications o Alginates are typically used on partial and full-thickness draining wounds such as pressure wounds or venous insufficiency ulcers. Alginates are often used on infected wounds due to the likelihood of excessive drainage o Advantages o High absorptive capacity o Enables autolytic debridement o Offers protection from microbial contamination o Can be used on infected or uninfected wounds o Non-adhering to wound o Disadvantages o May require frequent dressing changes based on level of exudate o Requires a secondary dressing o Cannot be used on wounds with an exposed tendon, joint capsule, or bone Wound Classification: Red-Yellow-Black System Color Wound description Red Pink granulation tissue Yellow Moist yellow slough Black Black, thick eschar firmly adhered Goals Protect wound; maintain moist environment Remove exudate and debris; absorb drainage Debride necrotic tissue Selective Debridement o Involves removing only nonviable tissues from a wound. Selective debridement is most often performed by sharp debridement, enzymatic debridement, and autolytic debridement. o Sharp Debridement o Requires use of scalpel, scissors, and/or forceps to selectively remove devitalized tissues, foreign materials or debris from a wound. Sharp debridement is most often used for wounds with large amounts of thick, adherent, necrotic tissue; however, it is also used in the presence of cellulitis or sepsis. Sharp debridement is the most expedient form of removing necrotic tissue. PTs are permitted to perform sharp debridement in the majority of states. o Enzymatic Debridement o Refers to the topical application of enzymes to the surface of necrotic tissue. Enzymatic debridement can be used on infected and non-infected wounds with necrotic tissue. This type of debridement may be used in wounds that have not responded to autolytic debridement or in conjunction with other debridement techniques. Enzymatic debridement can be slow to establish a clean wound bed and should be discontinued after removal of devitalized tissues in order to avoid damage. o Autolytic Debridement o Refers to using the body’s own mechanisms to remove nonviable tissue. Common methods of autolytic debridement include transparent films, hydrocolloids, hydrogels, and alginates. Autolytic debridement results in a moist wound environment that permits rehydration of the necrotic tissue and eschar and allows enzymes to digest the nonviable tissue. Autolytic debridement can be used with any amount of necrotic tissue and is non-invasive and pain free. Patients and caregivers can be instructed to perform autolytic debridement with relative ease; however, this type of debridement requires a longer period of time for overall wound healing to occur. Autolytic debridement should not be performed on infected wounds. Non-selective Debridement o Involves removing both viable and nonviable tissues from a wound. Non-selective debridement is often termed ‘mechanical’ and is most commonly performed by wet-to-dry dressings, wound irrigation, and hydrotherapy (whirlpool). o Wet-to-dry Dressings o The application of a moistened gauze dressing placed in an area of necrotic tissue. The dressing is then allowed to dry completely and is later removed along with the necrotic tissue that has adhered to the gauze. Wet-to-dry dressings are most often used to debride wounds with moderate amounts of exudate and necrotic tissue. This type of debridement should be used sparingly on wounds with both necrotic and viable tissue since granulation tissue will be traumatized in the process. Removal of dry dressings from granulation tissue may cause bleeding and be extremely painful. o Wound Irrigation o Removes necrotic tissue from the wound bed using pressurized fluid. Pulsatile lavage is an example of wound irrigation that uses a pressured stream of irrigation solution. This type of debridement is most desirable for wounds that are infected or have loose debris. Moist devices permit varying pressure settings and provide suction for removal of the exudate and debris. o Hydrotherapy o Most commonly employed using a whirlpool tank with agitation directed toward a wound that requires debridement. This process results in the softening and loosening of adherent necrotic tissue. PTs must be aware of the side effects of hydrotherapy such as dependent positioning of the lower extremities, systemic effects such as a drop in bp, and maceration of surrounding skin. Wound Terminology o Abrasion: a wound that occurs from the scraping away of the surface layers of the skin, often as a result of trauma o Contusion: an injury in which the skin is not broken. The injury is characterized by pain, swelling, and discoloration o Hematoma: a swelling or mass of blood localized in an organ, space or tissue, usually caused by a break in a blood vessel. o Laceration: a wound or irregular tear of tissues that is often associated with trauma o Penetrating wound: a wound that enters into the interior of an organ or cavity. o Puncture: a wound that is made by a sharp pointed instrument or object by penetrating through the skin into underlying tissues. o Ulcer: a lesion on the surface of the skin or the surface of a mucous membrane, produced by the sloughing of inflammatory, necrotic tissue. Factors Influencing Wound Healing o Age: decreased metabolism in older adults tends to decrease overall rate of wound healing o Illness: compromised medical status such as cardiovascular disease may significantly delay healing. This often results secondary to diminished oxygen and nutrients at the cellular level. o Infection: an infected wound will impact essential activity associated with wound healing including fibroblast activity, collagen synthesis, and phagocytosis. o Lifestyle: regular physical activity results in increased circulation that enhances wound healing. Lifestyle choices such as smoking negatively impacts wound healing by limiting the blood’s oxygen carrying capacity. o Medications: Steroids, anti-inflammatory drugs, heparin, antineoplastic agents, and oral contraceptives. Undesirable physiologic effects include delayed collagen synthesis, reduced blood supply, and decreased tensile strength of connective tissues. Exudate Classifications o Serous: Presents as clear, light color with a thin, watery consistency. Serous exudate is considered to be normal in a healthy healing wound. o Sanguineous: Presents as red with a thin, watery consistency. Sanguineous exudate appears to be red due to the presence of blood or may be brown if allowed to dehydrate. This type of exudate may be indicative of new blood vessel growth or the disruption of blood vessels. o Serosanguineous: Presents as light red or pink color with a thin, watery consistency. Serosanguineous exudate can be normal in a healthy healing wound. o Seropurulent: Presents as opaque, yellow or tan in color with a thin, watery consistency. Seropurulent exudate may be an early warning sign of an impending infection. o Purulent: Presents as yellow or green color with a thick, viscous consistency. Purulent exudate is generally an indicator of wound infection.