Extrapyramydal system, cerebellum
Indicate structure of brain, that does not belong to extrapyramidal system:
1. Caudate nucleus
2. Lentil nucleus
*3. Nucleus of Goll and Burdach
4. Red nucleus
5. Reticular formation of brain trunk
2. Indicate formation, that will not concern to paleostriatic system:
*1. Caudate nucleus
2. Globus pallidus lateral
3. Globus pallidus medial
4. Black substance
5. Red nucleus
3. Indicate formation, which relate to neoeostriatic system:
1. Globus pallidus lateral
2. Red nucleus
3. Globus pallidus medial
4. Black substance
*5. Caudate nucleus
4. Indicate, which of the listed formations will concern to extrapyramidal system of spinal cord:
1. Caudate nucleus
2. Red nucleus
3. Globus pallidus
*4. Y-motoneurons
5. Black substance
5. Indicate physiological function, which is not executed by extrapyramidal system:
1. Myostatic regulation
2. Postural control
3. Regulation of muscular tone
*4. Coordination of movement
5. Realization of automated movements
6. Indicate symptom, characteristic for lesion in neoeostriatic system:
1. Hypomimium
2. Silent monotonous language
3. Muscular hypertension
*4. Hyperkinesis
5. Propulsion
7. Indicate symptom, that is not characteristic for lesion in pallidonigral system:
*1. Hypomimium
2. Hypomyotonia
3. Bradykinesia
4. Silent monotonous language
5. Muscular hypertension behind plastic type
8. Indicate symptom, that are not characteristic for parkinsonism:
1. Silent monotonous language
2. Out of flexors
*3. Hypomyotonia
4. Static tremor
5. Oligokinesia
9. What infringement below does not concern with hyperkinesisnes:
1. Hemiballism
*2. Intention tremor
3. Trochee
4. Athetosis
5. Myoclonia
10. Indicate, what is characteristic of below listed symptoms for lesion of caudate nucleus:
1. Muscular hypertension
*2. Hypomyotonia
3. Hypokinesia
4. Bradykinesia
5. Hypomimium
11. Indicate, what infringement of speech arises at parkinsonism:
1. Mutism
2. Aphasia
3. Scanned language
4. Dysarthrium
*5. Silent, monotonous language
12. Indicate syndrome, that is characteristic for lesion in extrapyramidal system:
1. Vestibular ataxium
2. Oligophrenia
3. Dynamic ataxium
*4. Parkinson
5. Sensitive ataxium
13. Indicate basic function of cerebellum:
1. Realization of voluntary movements
*2. Coordination of movement
3. Postural control
4. Automatic movements
5. Maintenance of expressiveness of mimic reactions
14. Indicate formation, that does not relate with the structure of cerebellum:
1. Legs
*2. Nucleus of Goll and Burdach
3. Worm
4. Hemisphere
5. Dentate body
15. Indicate cerebellar nucleus:
1. Tail nucleus
2. Red, Swalbe
3. Deiters, Bechterev
*4. Fastigial nucleus, gear nucleus
5. Nucleus of Goll and Burdach
16. Indicate, which of the below-mentioned will pass through the middle leg of cerebellum:
1. Dentorubralis
*2. Anterior-pons-cerebellar
3. Rubrospinal
4. Govers
5. Vestibulocerebellar
17. Indicate, which of the below mentioned will pass through bottom leg of cerebellum:
1. Spinocerebellar of Govers
*2. Spinocerebellar of Flexig
3. Frontal bridge
4. Rubrospinal
5. Occipital bridge
18. Indicate ways, that will pass through top leg of cerebellum:
1. Olivocerebellar
2. Reticulocerebellar
3. Ponscerebellar
4. Vestibulocerebellar
*5. Spinocerebellar of Govers
19. Indicate symptom, not characteristic for cerebellar disease:
1. Intention tremor
*2. Muscular hypertension
3. Hypomyotonia
4. Uncertain shaky gait
5. Nistagmus
20. Indicate, what dissonance of language arises at cerebellar disease:
1. Aphasia
2. Nasal language
*3. Scanned
4. Dysphonia
5. Dysarthrium
21. Which of the listed methods for research of function of cerebellum are not used:
1. Finger-nose test
2. Test on diadochokinesia
*3. Test of Barre
4. Calcaneal-knee test
5. Test of Stewart-Holme
22. Indicate, which of the below mentioned are afferent leading ways of communications of cerebellum
*1. Spinal way of Flexig
2. Tectospinal
3. Dentorubralis
4. Vestibulospinal
5. Rubrospinal
23. Indicate, which of the below mentioned are efferent leading ways of communications of cerebellum:
1. Spine-cerebellar Flexig
2. Spinocerebellar of Govers
3. Vestibulocerebellar
*4. Dentorubralis
5. Front-pons-cerebellar
24. Name method of revealing dynamic cerebellar ataxium:
*1. Finger-nose test
2. Station test
3. Test of Barre
4. Test of Budda
5. Liquorodynamic tests
25. Indicate infringement, characterized by lesion of vermis:
*1. Static ataxium
2. Sensitive ataxium
3. Muscular hypertension
4. Bradykinesia
5. Vestibular ataxium
26. Indicate detection method of infringement of statics:
1. Test on diadochokinesia
2. Test on hypermetria
3. Finger-nose test
*4. Stability in pose Romberg
5. Test of Babiski
27.
Name clinical attribute of cortical-frontal ataxium:
1. Deviation at movements aside center
2. Nausea, vomitting
3. Nistagmus
*4. Astasium-abasia
5. Scanned language
28. Indicate biochemical mechanism of development of Parkinson syndrome:
1. Infringement of copper metabolism
2. Infringement of exchange of phenylalanine
*3. Infringement of exchange of dofaminum
4. Infringement of carbohydrate metabolism
5. Infringement of exchange of lipids
29. Indicate syndromes, that are characteristic for lesion of tail and red nuclei, «оЁб®perл© body:
1. Parkinson
*2. Hyperkinetic
3. Non-tactal
4. Central tetraparesis
5. Peripheral tetraparesis
30. On what side of cerebellum infringements at hemilesion and cortex of cerebellar hemispheres are
developed:
1. Ataxium of extremities of opposite focus
*2. Ataxium of extremities on side of focus
3. Ataxium of trunk from opposite side
4. Ataxium of trunk on same side of foucs
5. Bilateral ataxium of extremities
31. Impossibile to walk a straight line, alternately putting right heel of one leg to toes of other, is
connected to:
*1. Cerebellar dysfunction
2. Lesion of parietal share of brain
3. Lesion of temporal share of brain
4. Oculomotor dysfunction
5. Loss of sensitivity in cadences
32. Tremor of hands in calmness, especially when awakening the patient, lesion is usually developed
where:
1. Visual hump
*2. Black substance
3. Caudate nucleus
4. Spinal cord
5. Inside capsule of brain
33. Adiadochokinesia testifies about infringement of:
1. Consecutive movement of fingers
2. Walking from heel to sock
3. Hold back of tremor
*4. Synchronisation of movementsa of arms
5. Coordination of colloquial muscles
34. Indicate medication, that is used for treatment of Parkinson syndrome:
1. Nootropic preparations
2. Sedative mean
*3. Cholinoblockers (anticholinergic)
4. Anticholine esterase preparations
5. Corticosteroids
35. Sharp static ataxium is characteristic with muscular atony and asynergia for lesion of :
1. Bottom legs of cerebellum
2. Middle legs of cerebellum
3. Top legs of cerebellum
*4. Vermis
5. Cerebellar hemispheres
36.
Uncertainty and unsteadiness while walking in darkness and good illumination are characteristic for
ataxium:
*1. Sensitive
2. Vestibular
3. Dynamical cerebellar
4. Static-locomotor cerebellar
5. Cortical (frontal)
37. Functions of cerebellum are all of the below listed, except:
1. Coordination of movement
2. Regulation of muscular tone
3. Synergy of movements
4. Equilibrium of body
*5. Postural tone
38. Indicate, with what choreic hyperkinesis is clinically characterized:
1. Slowness
*2. Speed, unevenness, precipitancy, absence of stereotype
3. Speed, unevenness, precipitancy, stereotype
4. Twitching in muscular groups and in lonely muscles
5. Involuntary tonic reduction of mimic muscles of person
39. Patient has hypomimicry, flexor, shuffling gait, muscular hypertension behind plastic type. Indicate
localization of pathological center:
1. Neoeostriatic system
*2. Pallidonigral system
3. Cerebellum
4. Internal capsule
5. Brain trunk
40. Ten year old child has violent movements of extremities face, trunk.
Indicate localization of pathological center:
1. Pallidonigral system
*2. Neoeostriatic system
3. Cerebellum
4. Fronto-prefrontal site of cerebrum
5. Anterior central convolution
41. Patient with parkinson's disease syndrome. Indicate, how gait is affected:
1. Spastic
2. Spastico-atactic
3. Hemiparesthetical
4. Steppage
* 5. Shuffling, fine steps
42. Due to disease unsteadiness at circulation appeared, intensive tremor at finger-nose and calcanealknee tests, More expressed on the right side, Nistagmus at sight to the right. In pose Romberg patient
falls to the right. Determine localization of lesion:
1. Vermis
2. Lefthand cerebellar hemisphere
*3. Right cerebellar hemisphere
4. Top legs of cerebellum
5. Bottom legs of cerebellum
43. Fast arrhythmic involuntary movements of extremities and trunk are observed in patient.
He grimaces, frequently sticks out tongue. Tone of muscles of extremities is lowered.
Described syndrome is named:
1. Athetosis
2. Myoclonia
*3. Trochee
4. Hemiballism
5. Tic
44. Patient is worried by difficulty while walking. Objectively -- Hypomimium, delay of voluntary
movements, increase of muscular tone of extremities behind plastic type, walks in small steps, shuffling.
Described syndrome is named:
1. Hyperkinetic
*2. Parkinson
3. Non-tactal
4. Alternating
5. Brown-Sechar
45. Patient walks with short steps, body is inclined forward, hands half-bent, face is masklike.
Speech monotonous, silent, calmed down. Stereotyped tremor of fingers of hands of "account of coins
"type is observed. Syndrome is named:
1. Alternating
2. Non-tactal
3. Hyperkinetic
*4. Parkinson
5. Brown-Sechar
46. Patient after having influenza developed violent vermiculations in fingers of bones.
These movements are named:
*1. Athetosis
2. Hemiballism
3. Myoclonia
4. Tic
5. Trochee
47. Patient has sharp headaches, nausea, vomitting. At objective research horizontal nystagmus is
revealed, it is primary at sight to the left, adiadochokinesia at the left, scanned language, unsteadiness
of gait, in pose Romberg falls to the left.
Determine localization of lesion:
1. Top legs of cerebellum
2. Bottom legs of cerebellum
*3. Cerebellar hemisphere at the left
4. Cerebellar hemisphere on the right
5. Vermis
48. Schoolboy began to slovenly, turns head in different directions during lessons, grimace, write letters
of different size.
Objectively -Lowered tone of muscles in extremities, fast arrhythmic involuntary movements of extremities and
trunk are observed.
Described syndrome is named:
1. Athetosis
2. Hemiballism
3. Myoclonia
4. Tic
*5. Trochee
49. Man-40 years old choreolike hyperkinesis, dementia.
Indicated symptoms gradually progress.
Name disease:
1. Sharp cerebral circulatory disturbance
2. Paralysis agitan
3. Paraplegia of Strumpell
*4. Huntington disease
5. Meningoencephalitis
50. 36 yr old man in residual period of encephalitis, arose attacks of myoclonia of muscles of face, and
left hand.
At review in interattack period - somewhat lowered muscular tone in left sided extremities, intensive
tremor, hypermetria of left sided extremities.
Indicate localization of lesion:
*1. Red nucleus on the right
2. Black substance at the left
3. Vermis
4. Lefthand cerebellar hemisphere
5. Dentate body on the right
51. Man 42 years, 3 year back survived head trauma.
Complains on unsteadiness of gait, infringement of movements of right hand and leg.
At review - right-hand pyramidal deficiency behind central type, coordination tests at the left execute
with intention, adiadochokinesia through negative chronotropism at the left, hypomyotonia at the left.
Indicate striked structures:
*1. Anterior central convolution at the left, left side of hemisphere of cerebellum.
2. Anterior central convolution at the left, lefthand frontal -cerebellar way
3. Anterior central convolution on the right, right frontal-cerebellar way
4. Spinocerebellar tracts on the right, rubrospinal at the left
5. Spinocerebellar tracts at the left, rubrospinal on the right
52. Woman 42 years, complains of residual phenomena of trunk encephalitis on complication of gait,
unsteadiness, impossible to stand in vertical position and balance.
At review - light central tetraparesis, bilateral expressed static-locomotor ataxium.
Indicate localization of lesion:
*1. Vermis, overlap of pyramidal route
2. Vermis, cerebellar hemisphere
3. Cerebellar hemisphere, red nuclei
4. Internal capsule on the right, vermis
5. Internal capsule at the left, vermis
53. Patient has shaky gait, on the right missdirect, intensive tremor, adiadochokinesia, horizontal
nystagmus.
Where can lesion be located:
1. Vermis
2. Right cerebral peduncle
3. Lefthand cerebellar hemisphere
*4. Right cerebellar hemisphere
5. Lefthand anterior central convolution
54. Patient is moved with effort, cannot stand because of unsteadiness, falls in pose of Romberg, also
cannot sit. Volume of active movements in extremities is full, force of muscles is 5 points.
Sensitivity (superficial and deep) is kept.
What is damaged?:
1. Weigh cerebellum
2. Back cords of spinal cord
3. Cerebellar hemisphere
*4. Vermis
5. Precentral convolution of the brainof cortex
55. Patient is worried by shaky gait, rotatory vertigo, nausea, cannot stand in Horizontal
position.Nystagmus checks out. Falls In Romberg pose
What is damaged?
1. Vermis
2. All parts of cerebellum
3. Back cords of spinal cord
*4. Vestibular apparatus
5. Cerebellar hemisphere
56. In woman, 42 years, appeared infringement of movements in left hand, general constraint.
Therapist, at which woman was treated from psoriatic polyarthritis, suspected neurological disorder.
Neurologic inspection found:
Hypomimicry, bradykinesia, flexor pose, extrapyramidal hypertonus.
Pathogenetic therapy was ordered
Which medication is most used in this instance:
1. Selegelin
*2Cyclodol
3. Bromocryptine
4.Haloperidol
5.Amantadin