BVMCh V Semester Test (Supplementary) 24.08.01 (Memorandum) 2. Discuss the role of lighting programs in the management of commercial laying hens from day old to death. (10) 0-7 days – 23 hours good intensity (25 lux), to find feed (1.5) Rearing 1-18 w – about 8-10 hours, low intensity about 10 lux at bird level (1.5) Early lay 19-30 weeks increase by ½ hour per week to 16 hours (1.5) Late lay maintain to cull at 65 –75 weeks. (1) Rapid increase in light intensity and photoperiod is used with correct body weight to stimulate onset of lay at around 20 weeks. NB birds come into production together and not too early to ensure good peaks and limit weight loss in lay. (3) Used to increase overall production (0.5) Lowered light intensity reduces incidence of vices such as cannibalism (0.5) Never increase light duration during rearing, never decrease light duration during lay (2) Comments on stimulation of the pineal gland by light. (0.5) 2. Define minimum ventilation. (3) The minimum ventilation required to maintain birds at full production potential by ensuring an adequate supply of oxygen and removing waste gases such as CO2, ammonia and excess dust and water vapour. 3. Discuss omphalitis in broiler chicks under the following headings : causative organisms, source of infection, post mortem findings and treatment. (10) Cause : bacterial – E.coli, Pseudomonas general Source – egg or unhealed navels PM – distended abdomen with inflamed and vascularised yolk sac, congestion and peritonitis, inflamed navel Rx – none advised, control by hatchery and farm hygiene 4. List five viruses that are vertically transmitted through the egg in chickens. (5) EDS, AE, leucosis/sarcoma group, reovirus, avian influenza, CAV, reticuloendotheliosis virus 5. List the most important post mortem lesions you would expect to find in broilers infected by virulent Infectious Bursal Disease. (6) Haemorrhages in muscles Haemorrhages in proventriculus Haemorrhages in bursa Oedematous swollen bursa Nephrosis Congested carcass 6. How would you confirm a suspected diagnosis of Newcastle disease? (2) Virus isolation (in eggs) 7. Name the main method of control of EDS 76 in commercial layer flocks. (1) Oil vaccination 8. Name the viral diseases which are specifically associated with a decrease in egg quality and egg shell abnormalities. (3) EDS, IB, ND 9. Discuss Infectious Bronchitis briefly under the following headings: etiology, transmission, symptoms, treatment and prevention. (14) Aetiology: Coronavirus Transmission: Droplet, Not egg transmitted Symptoms: Layers: mild respiratory Severe drop in egg production Egg shell changes High % culls with oophoritis Young chicks and broilers: respiratory infections Reduced appetite Poor growth Prevention and treatment: Treat secondary E coli Evaluate and rectify management Vaccines: live attenuated – H120 Inactivated 10.Which Salmonella is responsible for Fowl Typhoid? Salmonella gallinarum (14) (1) 11. Which organisms are most likely to be involved in “Chronic Respiratory Disease”? (2) MG and E. coli 12. Is Coryza (Haemophilus paragallinarum) an upper or lower respiratory tract infection? Where should samples/swabs be taken from for bacterial culture? (2) Upper respiratory Infraorbital sinuses 13. What advice would you give to a broiler producer experiencing high mortalities due to ascites, to prevent the condition in future flocks? (7) Associated with rapid growth – slow growth by: o reducing feed intake by reducing lighting, especially in 2nd and 3rd week of life o changing from pellets to mash feeding. Associated with respiratory disease – check management esp. ventilation and ammonia levels also resp. pathogens Associated with altitude – aware certain incidence on Highveld is normal Males more susceptible due to rapid growth – difficult to solve. 14.Name two treatments (active ingredients) for roundworms in chickens: (2) Levamisole Piperazine Fenbendazole Ivermectin Hygromycin B 15. How would one confirm a diagnosis of Aspergillosis: (3) Culture - on Saborauds dextrose agar Microscopy – using material = 10% KOH and heat – visualise hyphae Histopathology – hyphae seen 16. a) Indicate which of the 4 most important coccidia are medium pathogenic species and which are high pathogenic species. (4) Medium pathogenic: E. acervulina and E. maxima High pathogenic: E. tenella, E. necatrix b) Compare the clinical symptoms seen in medium pathogenic species and high pathogenic species. (6) Medium pathogenic: Diarhhoea (mucoid) Poor Growth Poor FCR High pathogenic: Listless, ruffled, huddle together Dysentry (haemorrhagic diarrhoea) Mortality 17. Name the fat-soluble vitamins and list 1 of the most important deficiency symptoms/ post mortem signs/ syndromes of each in chickens (4): Vit A: White faeces due to excessive urate formation, nephrosis and renal failure (before skin esions) erosions in mouth and tongue, hyperkeratosis of cornea, oesophagus, eyelids, hyperkeratosis with secondary infection in respiratory system, GIT and ureters, poor appetite and growth, usually 4- 8 weeks. Vitamin D3: Rickets, osteomalacia, fractures, soft shelled eggs, cracked eggs, Vit E: Crazy Chick disease –encephalomalacia, Exudative diathesis, early embryonic mortality, Nutritional muscular dystrophy Vit K: Widespread haemorrhage 18. There are 3 main manure management systems in caged layer hen units. Describe each briefly and indicate how frequently the manure is removed in each of the systems. (6) 1. Manure accumulates under cages and is removed once per week. 2. Manure accumulates on conveyor belts under cages and is removed daily from the house. 3. Deep manure system (hi-rise) – manure removed when the flock is culled.