Sporozoans Phylum Apicomplexa Plasmodium, Gregarina Apicomplexans Heterotrophs: Parasites living in animal cells Cell-piercing structure made of microtubules Reproduce sexually and asexually in host cells Only gametes have flagella Example: Plasmodium (malaria) Phylum Apicomplexa (Sporozoa) No obvious means of locomotion Some have flagellated gametes Class Sporozoea (Sporozoans) All are obligate parasites of animals Gregarina in mealworms Plasmodium causes malaria Complicated life histories involving many hosts Most form oocysts Produce sporozoites following sexual reproduction All life stages are haploid except for 2N zygote Apicomplexa Named for the apical complex Aids cells in penetration of host Covered by pellicle and two plasma membranes Plasmodium Apicomplexa Cause parasitic disease in humans (Plasmodium) Coccidian diseases including: Cryptosporidiosis (Cryptosporidium parvum) Cyclosporiasis (Cyclospora cayetanensis) Isosporiasis (Isospora belli) Malaria Toxoplasmosis (Toxoplasma gondii) Diseases in livestock cause large scale economic losses Eimeria, Babesia, Theileria Apicomplexa Outer covering Thick, triple outer layer which is very flexible, but nearly impervious to biological, and even to most chemical, agents. First is the plasma membrane Next is inner, double membrane made up of flattened alveoli sutured together. Finally, the whole business is reinforced with the cellular equivalent of rebar -- longitudinal bundles of microtubules running from the apical complex back towards the posterior end of the cell. These are cross-linked in some fashion. Apical Complex Set of secretory and cytoskeletal structures that enables the young parasite to enter a host cell. Several components Two apical or polar rings A conoid - an open cone of spiral rod-like elements Polar ring of dense granular material that connects the ends of the longitudinal cytoskeletal microtubules Rhoptries and micronemes; these are modified secretory vesicles. Possess apicoplast (plastid) Apicoplast Organelle derived from endosymbiosis in which the apicomplexan ancestor engulfed a unicellular alga. Contains its own genome, a circular molecule of DNA which encodes ~ 30 proteins a full set of tRNAs plus some other RNAs Sequencing of apicoplast DNA shows it is closely related to chloroplasts (Chlamydomonas) Potential drug target – kill malaria with herbicides! Apicomplexa Apical Complex Rhoptries (rhoptry = sing.) and micronemes Special secretory organelles of the apical complex that produce a large number of proteins Protein functions: Part of the machinery that is used by these parasites both for invasion and to commandeer host functions. Modulate key regulators of the immune response of the host. Responsible for motility, adhesion to host cells, invasion of host cells and establishment of the parasitophorous vacuole inside the host cell. Plasmodium (Malaria) Plasmodium species cause malaria Malaria Malaria is a mosquito-borne disease caused by several members of the genus Plasmodium. People with malaria often experience fever, chills, and flu-like illness. Left untreated, they may develop severe complications and die. Each year 350-500 million cases of malaria occur worldwide, and over one million people die, most of them young children in Africa south of the Sahara. Mosquito = vector - transmits the malaria parasite Plasmodium vivax (a sporozoan) Malaria parasite in red blood cells Parasites breaking out of red blood cells Malaria Humans infected with malaria parasites can develop a wide range of symptoms. These vary from asymptomatic infections (no apparent illness), to the classic symptoms of malaria (fever, chills, sweating, headaches, muscle pains), to severe complications (cerebral malaria, anemia, kidney failure) that can result in death. The severity of the symptoms depends on several factors, species of infecting Plasmodium human's genetic background any acquired immunity to the parasite. Malaria This sometimes fatal disease can be prevented and cured. Bednets, insecticides, and antimalarial drugs are effective tools to fight malaria in areas where it is transmitted. Travelers to a malaria-risk area should avoid mosquito bites and take a preventive antimalarial drug such as chloroquine. Malaria Antimalarial drugs taken for prophylaxis by travelers can delay the appearance of malaria symptoms by weeks or months, long after the traveler has left the malaria-endemic area. can happen particularly with P. vivax and P. ovale, both of which can produce dormant liver stage parasites. Liver stages may reactivate and cause disease months after the infective mosquito bite. This Anopheles mosquito vector Malaria The incubation period in most cases varies from 7 to 30 days. The shorter periods are observed most frequently with P. falciparum and the longer ones with P. malariae. The classical (but rarely observed) malaria attack lasts 6-10 hours. It consists of: a cold stage (sensation of cold, shivering) a hot stage (fever, headaches, vomiting; seizures in young children) and finally a sweating stage (sweats, return to normal temperature, tiredness) Classically (but infrequently observed) the attacks occur every second day with (P. falciparum, P. vivax, and P. ovale) and every third day with (P. malariae). Malaria Each attack starts with shaking chills, usually at mid-day between 11 a.m. to 12 noon, and this lasts from 15 minutes to 1 hour (the cold stage), followed by high grade fever, even reaching above 41oC, which lasts 2 to 6 hours (the hot stage). The cycle corresponds to the toxins released during rupture of the schizont-infected red cells and release of large numbers of merozoites. These toxins reset the hypothalamus gland’s temperature set point – chills help raise body temp to this new point. The hot stage is followed by profuse sweating and the fever gradually subsides over 2-4 hours. Toxoplasmosis Toxoplasma gondii Kinetoplastida Toxoplasmosis Parasite causes eye and brain damage in a baby, if untreated. Acute infection in older children and adults may be without symptoms, cause flu like illness or enlarged lymph glands. Latent parasite occurs very commonly in people infecting approximately a third to a half of all humans. Can cause active disease if a person becomes immune compromised Brain damage and death Toxoplasma gondii Clinical Features: Generally an asymptomatic or mild self-limiting infection. Immunodeficient patients Brain lesions, death Pneumonitis Pregnant women/infant Miscarriage; still births Cerebral palsey; seisures Mental retardation Eye infections; impaired vision Enlarged liver and spleen Gregarines Apicomplexan (Sporozoan) parasites of insects. Recall observations of these parasites in intestine of meal-worm larvae (Tenebrio molitor beetles). Gregarina polymorpha Gregarines Host consumes a sporozoite or sporocyst containing them Sporozoite attaches to host intestinal epithelium with structure called epimerite Transforms into feeding stage called trophozoite Sexual stage begins when two trophozoites (gamonts) of opposite sex associate in syzyge Produces a walled gametocyst which is expelled with the feces. Within cyst the two gamonts divide to form many gametes which fertilize within the cyst to form a zygote called a sporocyst which undergo meiosis to form 1n sporozoites Gregarina oosysts Gregarina in syzyge Cryptosporidium Causes Cryptosporidiosis Obligate intracellular parasite Causes diarrhea Affects humans, cattle, sheep, dogs No effective drug treatment for cryptosporidiosis Antibiotics are contraindicated; supportive care only Cryptosporidium Major outbreaks in Milwaukee in 1993 and 1996 Cause was failure in one of the water treatment pumping stations Sewage No backed up into drinking water required testing for Crypto under EPA stds. Major cities now test for Crypto regularly Cryptosporidium affects humans, dogs, and cattle Cryptosporidium can be a problem in municipal water supplies. Ciliates Subphylum Ciliophora Functional groupings of ciliates Holotrich- uniform ciliation Heterotrich- possess membranes &/or cirri Peritrich- cilia only around the cytostome Colonial-living in colonies Suctorian- a clade of Ciliophora possessing hollow feeding tentacles An assortment of freshwater ciliates- biodiversity! This figure shows 167 species- about 9500 species are known Ciliophora (ciliates) A major clade within the Alveolates. Synapomorphies of ciliates include: Ciliated pellicle and associated kinetodesmata Dimorphic nuclei conjugation Primarily holotrophic- few parasitic. Many can form cyst stage (resting stage). Ciliates Paramecium Paramecium bursaria Stentor coeruleus Vorticella Stylonychia Cirri ciliary organelles used for food handling and locomotion- form membranes or bundles Ciliophora Euplotes a heterotrich ciliate showing complex cirri used for feeding and locomotion Euplotes Spirostomum Didinium http://www.uga.edu/protozoa/portal/images/movies/didinium.mov Balantidiasis Most cases are asymptomatic. Clinical manifestations, when present, include persistent diarrhea, occasionally dysentery, abdominal pain, and weight loss. Symptoms can be severe in debilitated persons. Diagnosis is based on detection of trophozoites in stool specimens or in tissue collected during endoscopy. Repeated stool samples necessary to find trophozoites Treatment: Tetracycline with metronidazole and iodoquinol as alternatives Trophozoites Cyst B. coli trophozoites Ciliophora Ichthiopthirius multifilis (Ich) • Common parasite of freshwater and marine fishes • Trophozoite burrows in skin • Mature troph leaves fish and encysts. • Multiple mitoses produce hundreds of “swarmers” that reinfest fish. Ich trophozoite in fin of freshwater drum M. C. Barnhart Diversity of protists use pseudopodia for movement Most are heterotrophs and use pseudopods to hunt for food Unsure of their place in a phylogenetic tree So we won’t worry about trying to get the newest phylogeny here See organization on the phylogenetic tree in this slide set Ameobozoa (like Chaos and Arcella) and some in the Chromista (heliozoans like Actinosphaeria) Rhizaria Radiolaria and Foraminifera Rhizaria Have thin, hairlike extensions of the cytoplasm called filose pseudopodia Chlorarachniophyta Radiolaria and Foraminifera Ocean plankton that make mineral shells White cliffs of Dover, England Foraminifera used to infer past climates Rhizaria Single-celled heterotrophs with a secreted shell Many openings for pseudopods Actinopoda (heliozoans and radiolarians) Actinopod means “ray foot” Extensions of slender pseudopods Radiolarians Heliozoans – Fresh water habitat Skeletons consist of silica Radiolaria siliceous tests Axopodia with axonemes Test - Note pores Foraminifera (forams) Forams are all marine Named for their porous shells Hardened with calcium carbonate Chalk White Cliffs of Dover Foraminifera Fig. 25-20b, p. 549 Amoebozoa All unicellular Amoeba proteus Amoebae Actinosphaerium Chaos Arcella Amoebae Difflugia Arcella Amoebozoa Pseudopodia feeding locomotion a few also have flagella Anatomy simpler than other protozoa fewer organelles no pellicle may have test (shell) “testate” amoebae secreted or made of debris Free-living, symbionts, parasites Amoebozoa Classification Based on type of pseudopodia Lobopodia: broad, rounded lobes Filopodia: thin, no endoplasm, (branched) Reticulopodia: thin, branched, netlike Actinopodia: long, straight, thin, core of microtubules See page 73 and http://www.biani.unige.ch/msg/Amoeboids/Amoebozoa.html Mycetozoa (Slime Molds) Mycetozoa “fungus animals” They decompose leaf litter like fungi But they are not fungi Use pseudopodia for movement Two distinct groups Plasmodial slime mold Cellular slime mold Myxogastridia (Plasmodial Slime Mold) Most are brightly colored All are heterotrophic Life cycle contains: Feeding stage (mobile) Large mass of unicellular organisms that is not separate by cell walls Fruiting body (not mobile) Plasmodial Slime Mold Dictyostelida: Cellular Slime Mold Feeding stage consists of individual cells When food is scarce form an aggregate that functions as a unit Cellular Slime Mold Infective Amoebas: Entamoeba Amoebiasis caused by Fourth most common protozoan infection in the world Aka amoebic dysentery Entamoeba histolytica Entamoeba histolytica Figure 5.28 Entameoba histolytica Causes amoebic dysentery (diarrhea) and can enter the liver, lungs, and brain Cysts Entameoba histolytica Clinical Features: A wide spectrum from asymptomatic infection ("luminal amebiasis"), to invasive intestinal amebiasis (dysentery, colitis, appendicitis, toxic megacolon, amebomas), to invasive extraintestinal amebiasis (liver abscess, peritonitis, pleuropulmonary abscess, cutaneous and genital amebic lesions). Up to 100K deaths/yr worldwide, second only to malaria and schistosomiasis Naegleria fowleri Naegleria fowleri Clinical Features: Acute primary amebic meningoencephalitis (PAM) is caused by Naegleria fowleri. It presents with severe headache and other meningeal signs, fever, vomiting, and focal neurologic deficits, and progresses rapidly (<10 days) and frequently to coma and death. Acanthamoeba spp. causes mostly subacute or chronic granulomatous amebic encephalitis (GAE), with a clinical picture of headaches, altered mental status, and focal neurologic deficit, which progresses over several weeks to death. Acanthamoeba addition, Acanthamoeba spp. can cause granulomatous skin lesions and, more seriously, keratitis and corneal ulcers following corneal trauma or in association with contact lens use. Non-contact lens users and contact lens users with safe lens care practices can become infected. However, poor contact lens hygiene and exposure to contaminated water may increase the risk among contact lens users. In Opisthokonts Animals and their kin Opisthokonts One group of flagellates, the choanoflagellates, is thought to have given rise to the simplest animals, the sponges. Choanoflagellida Sponge Choanocytes Amoebocytes in the mesohyl