High Risk Newborn Chapter 23 & 24 Mary L. Dunlap MSN Fall 2015 Birth Weight Variations • Appropriate for gestational age (AGA) • Small for gestational age (SGA) • Large for gestational age (LGA) SGA • SGA weight- less than 5lb 8 oz and below the 10th% at term • IUGR- High risk growth does not meet the expected growth pattern and is pathologic SGA Characteristics • Decreased breast tissue • Scaphoid abdomen (sunken) • Wide sutures • Thin umbilical cord • Head larger than body • Wasted appearance to extremities • Reduced fat stores SGA Common Problems • • • • Perinatal asphyxia Hypothermia- lack of brown fat Hypoglycemia- lack of glycogen stores Polycythemia- increase rate of production due to hypoxia • Meconium Aspiration Nursing Management • Initiate early and frequent oral feedings • Monitor for hypoglycemia • IV infusion of 10% dextrose if unable to maintain glucose level. • Weigh daily • Promote rest periods to decrease metabolic requirements • Monitor for Polycythemia LGA • Weight- Larger than 9 lbs. and above the 90th% • Infant can be preterm, term, or postterm LGA Characteristics • • • • Large body-plump full face Body size is proportionate Poor motor skills Difficulty in regulating behavioral state (arouse to quiet alert state) LGA Common Problems • • • • • Birth Trauma Hypoglycemia Polycythemia Hyperbilirubinemia Shoulder dystocia LGA Nursing Management Hypoglycemia • Screen newborn for hypoglycemia • Encourage feedings • IV glucose Hyperbilirubinemia • Hydration • Phototherapy Gestational Age Variations • • • • Preterm newborn Late Preterm Post-term newborn Term newborn Post Term Newborn • Gestation > 42 weeks • Must determine if EDC is truly post term • After 42 weeks placenta loses ability to nourish the fetus Post term Newborn Characteristics • Newborn emaciated • Meconium stained • Hair and nails long • Dry peeling skin • Creases cover soles • Limited vernix and lanugo Nursing Management • Monitor blood glucose levels and treat as required • Initiate feedings as soon as possible • Monitor temperature and respiratory characteristics • Assess for polycythemia and hyperbilirubinemia Preterm Infant • Infant born prior to the completion of the 37th week • Organs immature • Lack physical reserves • Survivability related to weight and gestational age Preterm Infant Causes based on research: • Infection • Maternal or fetal stress • Bleeding • Stretching Immediate Delivery Care • • • • Evaluate prenatal risk factor Rapid assessment Basic equipment Box 23.2 pg. 795 ABC’s of resuscitation Box 23.3 Preterm Infant Respiratory last to mature • Surfactant deficiency-RDS • Unstable chest wall-atelectasis • Immature respiratory centers-apnea • Small passages-obstructions • Unable to clear fluid-TTN Preterm Infant Cardiovascular • Difficulty transitioning from fetal to neonatal circulatory pattern • Congenital anomalies associated with continued fetal circulation • Fragile blood vessels (brain) • Impaired regulation of B/P Preterm Infant Gastrointestinal • Lack neuromuscular coordination suckswallow-breath • Perinatal Hypoxia shunts blood from the gut • Small stomach-compromised metabolic function • Risk for malnutrition -wt. loss Preterm Infant Renal System • Slow glomerular filtration rate • Reduced ability to concentrate urine • Risk: fluid retention, electrolyte imbalance, drug toxicity Preterm Infant Immune system • Deficiency of IgG (trans-placental transfer after 34 wks.) • Impaired ability to produce antibodies • Thin skin- limited protection barrier Preterm Infant Central nervous system • Long term disability due to injury • Immature temperature-regulating center • Susceptible to hypoglycemia Preterm Characteristics • • • • • • Wt less than 5.5lb Head larger than chest Poor muscle tone Minimal fat Thin transparent skin Undescended testicles & minimal scrotal rugae • Prominent labia & clitoris Nursing Assessment • • • • • Review prenatal record for risk factors Head to toe assessment Monitor respiratory effort Monitor V.S. Monitor for hypoglycemia Nursing Management Varies with gestational age • Promote oxygenation • Promote Thermoregulation • Promote optimal nutrition Nursing Management Continue • Prevent infections • Provide stimulation • Pain management Prevention & Management Box23.4 pg. 800 Nonpharmacologic Techniques Box 23.5 pg.801 • Pharmacologic agents Nursing Management Continue • • • • Promote bonding Quite environment Promote parent coping Discharge Planning Box 23.6 pg 803 Nursing Care plan 23.1 pg 792-794 Neonatal Asphyxia • Failure to establish adequate, sustained respirations after birth • Pathophysiology: insufficient oxygen delivery to meet metabolic demands Nursing Assessment • • • • • • Asses for risk factors newborn’s color work of breathing heart rate Temperature Apgar scores Nursing Management • • • • • Immediate resuscitation Continued observation Neutral thermal environment Blood glucose levels Parental support and education Transient Tachypnea Newborn TTN • Mild respiratory condition • Result of delayed or incomplete absorption of fluid from the lungs • Occurs within a few hours of birth • Resolves over 24-72 hour period Transient Tachypnea Newborn TTN Symptoms • Respiratory rate as high as 100-140 • Labored breathing • Grunting nasal flaring • Retractions • Chest x-ray shows lymphatic engorgement ( retained lung fluid) Transient Tachypnea Newborn Nursing Care • Mainly supportive • Monitory VS & O2 Sats • Provide supplemental O2 • IV fluids • Gavage feedings Respiratory Distress Syndrome • RDS result of lung immaturity and surfactant deficiency • Poor gas exchange & ventilation • Seen in preterm newborns • Cesarean births without labor • Infants of diabetic mothers Nursing Assessment Symptoms • Noted at birth or within in a few hours • Expiratory grunting • Nasal flaring • Chest wall retractions • Seesaw respirations • Generalized cyanosis Nursing Assessment Symptoms • Tachypnea- rates above 60 • Fine inspiratory crackles • Tachycardia- rates above 150-180 • Silverman-Anderson index assessment • Chest x-ray- alveolar atelectasis (ground glass pattern) • Lab test done to r/o infection and sepsis Silverman-Anderson Tool RDS Nursing Management Supportive care • Thermoregulation- prevent cold stress • O2 administration • Fluid management • Nutritional support • Surfactant replacement therapy • Monitor VS & O2 sats Meconium Aspiration • Fetus inhales meconium into the lungs while in utero • Meconium blocks the airway preventing exhalation • Meconium irritates the airway making breathing difficult (chemical pneumonia) • Meconium aspiration related to fetal distress during labor. Nursing Assessment Symptoms • Cyanosis • Tachypnea • Course & rhonchi • Labored breathing • Apnea • X-ray patches or streaks of meconium & trapped air Meconium Aspiration Nursing Management • Assess for risk factors prior to delivery • Neutral Thermal environment • Supplemental O2 • Medications • Monitor response to treatment Persistent Pulmonary Hypertension • Marked pulmonary hypertension causing right to left extrapulmonary shunting and hypoxemia • Cause occur idiopathically or as a complication of perinatal asphyxia, meconium aspiration syndrome, congenital heart defects Persistent Pulmonary Hypertension Nursing Assessment • Tachypnea within 12 hours after birth • Marked cyanosis, grunting, and retractions • Systolic ejection murmur • Blood pressure • Oxygen saturation • Echocardiogram Persistent Pulmonary Hypertension Nursing Management • Monitoring of oxygenation, perfusion, and blood pressure • Immediate resuscitation; oxygen therapy • Respiratory support • Medications • Clustering of care • Parental support and education Retinopathy • Developmental abnormality affecting immature blood vessels of the retina • Five stages from mild to severe based on severity, location by zones in the retina, and proportion of retinal circumference • If a newborn is premature, vessels may cease to develop. • ROP typically develops in both eyes due to hyperoxemia (because of assisted ventilation and high oxygen exposure), acidosis, or shock. Retinopathy • Review prenatal history for risk factors (hypertension, substance abuse, preeclampsia, heavy cigarette smoking, or placental insufficiency). • Assess newborn’s gestational age and weight; newborns weighing less than 1,500 grams or born at 28 weeks’ gestation or less are at risk. • Evaluate the newborn’s history for duration of intubation and use of oxygen therapy, intraventricular hemorrhage, and sepsis. Retinopathy • Administer oxygen therapy cautiously— ensure lowest concentration and shortest duration • Assist with scheduling ophthalmic exam; administer mydriatic eye agent 1 hour before appointment • Protect newborn’s eyes from light • Provide support to parents by giving them information and providing details about the condition and follow-up examinations Periventricular-Intraventricular Hemorrhage • Bleeding in the brain due to fragility of cerebral vessels; most common in the first 72 hours after birth; Grades I to V Periventricular-Intraventricular Hemorrhage Nursing Assessment • Possibly no symptoms • Risk factors • Unexplained drop in hematocrit, pallor, poor perfusion, seizures, lethargy, weak suck, high-pitched cry, Hypotonia • Cranial ultrasonography Periventricular-Intraventricular Hemorrhage Nursing Management • Prevention • Correction of anemia, acidosis, hypotension • Flexed contained positioning • Daily head circumferences • Clustering of care; limiting of stimulation • Parental support Necrotizing Entercolitis • Pathologic mechanisms: Bowel ischemia, bacterial flora, and effect of feeding Necrotizing Entercolitis Nursing Assessment • Risk factors (see Box 24.1) • Signs and symptoms: abdominal distention and tenderness, bloody stools, feeding intolerance (bilious vomiting), sepsis, lethargy, apnea, shock • KUB: air in bowel wall; dilated bowel loops Necrotizing Entercolitis Necrotizing Entercolitis Nursing Management • Maintenance of fluid and nutritional status; IV fluids • Bowel rest and antibiotic therapy • Surgery with proximal enterostomy • Supportive care • Family education Infant of Diabetic Mother • High levels of maternal glucose crossing placenta, stimulating increased fetal insulin production leading to somatic fetal growth Infant of Diabetic Mother • Congenital abnormalities- during first trimester due to fluctuations in BS and ketoacidosis • Macrosomia- develops last trimester due to maternal hyperglycemiaexcessive fetal growth • Tight control over glucose levels needed ( less than 105 mg/dl) Infant of Diabetic Mother Common Problems • Congenital Abnormalities • Macrosomia • Birth Trauma • Perinatal Asphyxia • • • • • RDS Hypoglycemia Hyperbilirubinemia Polycythemia Birth trauma Infant of Diabetic Mother Infant Characteristics • Rosy cheeks • Short neck • Wide shoulders • Excessive subcutaneous fat • Distended abdomen Nursing Management • Monitor glucose level q. 3-4 h keep level above 40 mg/dl until stable • Feed q. 2-3 h to create a constant level • IV glucose • Monitor serum bilirubin levels • Maintain neutral thermal environment to prevent cold stress Newborns of Substance-Abusing Mothers • Most common substances: tobacco, alcohol, and marijuana Table 24.4 • Fetal alcohol syndrome: physical and mental disorders appearing at birth and remaining problematic throughout the child’s life Box 24.2 Newborns of Substance-Abusing Mothers • Fetal alcohol spectrum disorders • Alcohol-related birth defects • Neonatal abstinence syndrome: drug dependency acquired in utero manifested by neurologic and physical behaviors Newborns of Substance-Abusing Mothers Nursing Assessment • Maternal history; risk behaviors, toxicology • Newborn behaviors (see Box 24.3); • WITHDRAWAL assessment Birth Trauma Injuries due to the forces of labor and birth • Fractures • Brachial plexus injury • Cranial nerve trauma • Head trauma Table 24.3 Nursing Assessment Risk factors • Physical and neurologic assessment: bruising, bumps, swelling, paralysis, symmetry of structure and function Nursing Management • Supportive • Assessment for resolution or complications • Support and education • Realistic appraisal of situation • Community referral for ongoing followup and care Hyperbilirubinemia • Excess of bilirubin in the bloodelevated bilirubin level > 5mg/dl • Heme from erythrocytes break down forms unconjugated bilirubin • Jaundice • Physiologic • Pathologic Hyperbilirubinemia Causes • Drugs/Medical conditions disrupt conjugation and albumin binding sites • Decreased hepatic function • Increased erythrocyte production • Enzymes in breast milk Hyperbilirubinemia Physiologic • • • • • Develops in 3-4 days after term birth Develops3-5 days after preterm birth Term birth resolves 7 days Preterm birth resolves 9-10 days Unconjugated bilirubin level < 12.9 mg/100 ml Hyperbilirubinemia Pathologic • Present at birth or develops within 24 hrs. • Persists beyond 7 days • Bilirubin > 12.9mg/100 term • Bilirubin > 15mg/100 preterm • Increases > 5mg/100ml in 24hrs Hyperbilirubinemia Nursing Management • Phototherapy- eye shield, turn q2hrs. monitor temperature 3-4 hrs. • Increase feeding to q 2-3 hrs. • Bili level q 6 hrs. Phenylketonuria PKU • Inability to metabolize phenylalanineamino acid found in protein • Affect brain and CNS development • Interferes with the production of melanin, epinephrine & Thyroxine • Both parents must pass the gene on Phenylketonuria PKU Symptoms • Seizures • Irritability • Tremors • Jerking movements arms & legs • Hyperactivity • Unusual hand posturing Phenylketonuria PKU • Diagnosed with PKU screening prior to discharge from hospital Hemolytic Disorders • Hemolytic disease occurs when blood groups of mother and newborn are different • Antibodies are present or formed in response to antigen from fetal blood crossing placenta and entering maternal circulation Hemolytic Disorders • Maternal antibodies of IgG class cross placenta, causing hemolysis of fetal RBCs • Fetal anemia • Neonatal jaundice • Hyperbilirubinemia Hemolytic Disorders Rh incompatibility (isoimmunization) • Only Rh-positive offspring of Rhnegative mother is at risk • If fetus is Rh positive and mother Rh negative, mother forms antibodies against fetal blood cells Hemolytic Disorders ABO incompatibility • Occurs if fetal blood type is A, B, or AB, and maternal type is O • Incompatibility arises because naturally occurring anti-A and anti-B antibodies are transferred across placenta to fetus • Exchange transfusions required occasionally Neonatal Infections Sepsis Bacterial, viral, fungal, Group B Strip Patterns • Early onset or congenital • Nosocomial infection—late onset Neonatal Infection Septicemia • Pneumonia • Bacterial meningitis • Gastroenteritis is sporadic Neonatal Infections TORCH infections • Toxoplasmosis • Gonorrhea • Syphilis • Varicella-zoster • Hepatitis B virus (HBV) • Human immunodeficiency virus (HIV) and acquired immunodeficiency syndrome (AIDS)