科技英文寫作練習
Class (
)
Number (
)
Name (
)
1. ...-induced ... : ____________________________________________________
2. ...is coupled to...: __________________________________________________
3. The mechanisms by which ... are not yet fully understood.
______________________________________________________________
______________________________________________________________
4. ...is associated with ...: _______________________________________________
5. ..., suggesting that ...: ________________________________________________
__________________________________________________________________
6. Our results show that ...: ______________________________________________
7. ...-promoting ...: ________________________________________________
8. ... has been linked to ...: ______________________________________________
9. To ... we did a ... study : _____________________________________________
10. We assayed V-ing ...: _______________________________________________
11. When compared with ..., ...: ___________________________________________
__________________________________________________________________
12. ... was most marked among ...: _________________________________________
13. ...-derived ...: ______________________________________________________
14. ... in response to ... respectively: _______________________________________
15. When ..., ...were enhanced: ___________________________________________
_________________________________________________________________
進階科技英文寫作測驗
1. estrogen receptor alpha 或 the estrogen receptor alpha
2. tumor suppressor protein p53 或 the tumor suppressor protein p53
3. They exert opposing effect on cellular proliferation. 或 They exert opposing
effects on cellular proliferation. 或 They exert opposing effects on the cellular
proliferation.
4. As a transcriptional regulator, p53 is ... 或 As transcriptional regulator, p53 is ...
5. P53 is capable of activating various target genes. 或 P53 is capable of the
activating various target genes. 或 P53 is capable of the activating of various
target genes. P53 is capable of activation various target genes. P53 is capable of
activation of various target genes. 或 P53 is capable of the activation of various
target genes.
6. P53 is capable of activating or repressing various target genes. 或 P53 is capable
of activating or repressing the various target genes. 或 P53 is able to activate or
repress various target genes.
7. We have previously reported that ... 或 We previously reported that ... 或 We
have
previous reported that ... 或 We have a previous report that ...
8. ERalpha binds directly to p53. 或 ERalpha is bound directly to p53.
9. ERalpha binds directly to p53, leading to ... 或 ERalpha binds directly to p53,
leads to ...
10. ERalpha binds directly to p53, leading to down-regulation of transcriptional
activation by p53. 或 ERalpha binds directly to p53, leading to the
down-regulation of the transcriptional activation of p53.
11. In addition to transcriptional activation, transcriptional repression plays important
role in diverse biological process. 或 In addition to transcriptional activation,
transcriptional repression plays important roles in diverse biological processes.
12. Transcriptional repression of a subset of target genes by p53 plays ... 或
Transcriptional repression by a subset of target genes of p53 play ...
13. ... in diverse biological processes, such as apoptosis. 或 ... in diverse biological
processes such as the apoptosis.
14. Here, we report that ... 或 Here, we reported that ...
15. Here, we report that ERalpha inhibited p53-mediating transcriptional repression.
或 Here, we report that ERalpha inhibits p53-mediated transcriptional repression.
16. Chromatin immunoprecipitation assays reveal that ... 或 Chromatin
immunoprecipitation assay revealed that ...
17. ... assays reveal that ERalpha interacts in vivo with p53 bound to promoters of
Survivin. 或 ... assays reveal that ERalpha interacted in vivo with p53 binding to
promoters of Survivin.
18. ERalpha interacts in vivo with p53 bound to promoters of Survivin and multidrug
resistance gene 1. 或 ERalpha interacts in vivo with p53 bound to promoters of
Survivin and of multidrug resistance gene 1.
19. ... Survivin and multidrug resistance gene 1, both targets for transcriptional
repression by p53. 或 ... Survivin and multidrug resistance gene 1, both being
targets for transcriptional repression by p53.
20. ERalpha binding to p53 leads to inhibition of p53-mediated transcriptional
regulation of these genes in human cancer cells. 或 ERalpha binds to p53 leading
to inhibition of p53-mediated transcriptional regulation of these gene in human
cancer cell.
Writing test
1. Transcriptional derepression of Survivin by ERalpha is depend on the p53-binding
site on Survivin promoter. 或 Transcriptional derepression of Survivin by
ERalpha is dependent on the p53-binding site on the Survivin promoter.
2. A is dependent on B, consistent with our observation that … 或 A is dependent
on B, which is consistent with our observation that …
3. … our observation that p53 is necessary for ERalpha to access the promoters.
或 … our observation that p53 is necessary to ERalpha for access the promoters.
4. Important, mutagenic conversion of this site for an activation element enabled
ERalpha for repress p53-mediated transcriptional activation. 或 Importantly,
mutagenic conversion of this site to an activation element enabled ERalpha to
repress p53-mediated transcriptional activation.
5. Further, RNA interference-mediated knockdown of ERalpha resulted in reduced
Survivin expression and enhanced the propensity of MCF-7 cells to undergo
apoptosis in response to staurosporine treatment. 或 Furthermore, RNA
interference-mediated knockdown of ERalpha resulted reduced Survivin
expression and enhanced in the propensity of MCF-7 cells for undergoing
apoptosis in response to staurosporine treatment.
6. A resulted in B, an effect that was blocked by exogenous expression of Survivin.
或 A resulted in B, that was an effect blocked by exogenous expression of
Survivin.
7. These results unravel a novel mechanism which ERalpha opposes to p53-mediated
apoptosis in breast cancer cell. 或 These results unravel a novel mechanism by
which ERalpha opposes p53-mediated apoptosis in breast cancer cells.
8. The findings could have translational implications in developing new therapeutic
and prevention strategies. 或 The findings can have translational implication in
developing new therapeutic and prevention strategy.
9. Studies on retinoblastoma have been at the heart of many of the landmark
discoveries in cancer genetics over the past 35 years. 或 Studies of
retinoblastoma have been in the heart of many of the landmark discoveries on
cancer genetics for the past 35 years.
10. However, these advances in the laboratory have had a little effect in the treatment
of children with retinoblastoma. 或 However, these advances in the laboratory
have had little effect on the treatment of children with retinoblastoma.
11. One of the reasons for this has been the lack of preclinical models that
recapitulated the genetic and histopathologic features of human retinoblastoma.
或 One of the reasons of this have been the lack of preclinical models which
recapitulated genetic and histopathologic features of human retinoblastoma.
The Structure of a Thesis or Paper
Title
Abstract
Key words and phrases
The Main Body of the Text
1.
2.
3.
4.
5.
6.
Introduction
Review of literature
Material(s)
Method(s)
Work done (in lab and/or elsewhere)
Result(s) or finding(s)
7. Discussion
8. Conclusion(s) and suggestion(s)
Notes
Bibliography
Appendix or Appendices
*A doctorate dissertation, an MS thesis, a term paper, a journal paper, a conference
paper, etc.—each has its required length, format, and content. Before writing, you
need to know what type of work you are writing and what is the required length,
format, and content.
*The so-called “scientific English” is actually no other than ordinary English, aside
from some scientific terms science workers have to use in communication.