Analysis of Reelin signaling and neurodevelopmental trajectory in
primary cultured cortical neurons with RELN deletion identified in
schizophrenia
Aden Wee Chu Yu
INTRODUCTION
RESULTS AND DISCUSSION
Will add background information and definitions
Graph and analysis of results
LITERATURE REVIEW
External information from articles or previous findings
MATERIALS AND METHODS
Materials
CONCLUSION
Conclusion or derivation of trend
Materials and experiment procedure (picture to be included if
possible)
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REFERENCES
Arnaud, L., Ballif, B.A., Cooper, J.A., 2003. Regulation of protein
tyrosine kinase signaling by substrate degradation during brain
development. Mol. Cell Biol. 23, 9293–9302. Beffert, U., Morfini, G.,
Bock, H.H., Reyna, H., Brady, S.T., Herz, J., 2002. Reelin- mediated
signaling locally regulates protein kinase B/Akt and glycogen synthase
kinase 3beta. J. Biol. Chem. 277, 49958–49964. Beffert, U., Weeber,
E.J., Durudas, A., Qiu, S., Masiulis, I., Sweatt, J.D., Li, W.P., Adelmann,
G., Frotscher, M., Hammer, R.E., Herz, J., 2005. Modulation of synaptic
plasticity and memory by Reelin involves differential splicing of the
lipoprotein receptor Apoer2. Neuron 47, 567–579. Benhayon, D.,
Magdaleno, S., Curran, T., 2003. Binding of purified Reelin to ApoER2
and VLDLR mediates tyrosine phosphorylation of Disabled-1. Brain Res
Mol Brain Res 112, 33–45.
ACKNOWLEDGEMENTS
Special thanks to Dr Huang Sinong for her help in this project.
RESEARCH POSTER PRESENTATION
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