Chapter 10 Pain Copyright 2005 Lippincott Williams & Wilkins Pain An unpleasant sensory and emotional experience associated with actual or potential tissue damage, or described in terms of such damage. International Association for the Study of Pain Copyright 2005 Lippincott Williams & Wilkins Types of Pain Acute Pain Chronic Pain Referred Pain Usually short lived and is associated with muscle strains, tendinitis, contusions, surgery or ligament injuries Pain that persists after noxious stimulus has been removed. Pain that is felt at a site distant from the location of injury or disease. Copyright 2005 Lippincott Williams & Wilkins Physiology of Pain – Source of Pain Microtrauma Macrotrauma A long-standing or recurrent musculoskeletal problem that was not initiated by an acute injury. An immediately noticeable injury involving a sudden, direct, or indirect trauma. Copyright 2005 Lippincott Williams & Wilkins Non-nociceptive Pain Damage to central nociceptive system triggers non-nociceptive activity may elicit pain – non-nociceptive pain (NNP) Copyright 2005 Lippincott Williams & Wilkins Pain Pathway Lowered firing threshold Aberrant muscle activity Receptive threshold expansion CNS Spontaneous discharge Articular dysfunction Ascending afferents Disordered proprioception Non-nociceptive input Nociception Spinothalamic tract Spinal cord A&C fibers Copyright 2005 Lippincott Williams & Wilkins Pain Theory – Gate Control Copyright 2005 Lippincott Williams & Wilkins Examination and Evaluation Use of Questionnaires Assess affective qualities of pain Assess pain intensity Assess psychological aspects of pain Copyright 2005 Lippincott Williams & Wilkins Pain Scales Visual analogue scale (VAS) – Pain intensity McGill Pain Questionnaire (MPQ) – More sensitive, but longer than VAS; three categories (sensory, affective, evaluative) Copyright 2005 Lippincott Williams & Wilkins Disability and Health-Related Quality of Life Scales Quality of Well-Being Scale (QWB) Sickness Impact Profile (SIP) Duke Health Profile (DHP) Short Form-36 (SF-36) The Oswestry Low Back Disability Questionnaire Waddell Disability Index Disability Questionnaire Arthritis Impact Measurement Scales Copyright 2005 Lippincott Williams & Wilkins Therapeutic Exercise Intervention for Pain Acute Combination of medication, gentle exercise, ice (within first 24 hours). Exercise directed at restoring motion, strength, and function. Copyright 2005 Lippincott Williams & Wilkins Exercise Intervention for Chronic Pain Often requires a team approach. Realistic goals and patient education are crucial. Goals may extend beyond treatment of impairments. Exercise is used to inhibit pain, facilitate nonnociceptive input, while addressing impairments and functional limitations. Copyright 2005 Lippincott Williams & Wilkins Activity and Mode Depends on source of pain and results of evaluation. Should focus on awareness and use of proper posture. Incorporation of movement therapies (e.g., Feldenkrais) is helpful in restoring movement patterns. Aerobic exercise (low impact) is helpful for chronic pain. Copyright 2005 Lippincott Williams & Wilkins Dosage Dosage should not increase pain. Sessions may be brief initially to assess response. Frequency is determined by activity type, purpose, and quantity prior to experience of pain = “pain-free dosage.” Functional progression to previous activity levels. Copyright 2005 Lippincott Williams & Wilkins Adjunctive Agents Medications Massage therapy Relaxation techniques Biofeedback Psychological care Acupuncture Heat Cold Transcutaneous electrical stimulation (TENS) Copyright 2005 Lippincott Williams & Wilkins Summary Pain impairment occurs with most musculoskeletal conditions and must be treated as a primary impairment along with any secondary limitations that may result. Nociceptors transmit pain via A&C fibers. Information is processed w/in SC and then ascends via contralateral spinothalamic tract to thalamus. Gate theory – Incoming information from non-pain receptors can close the gate to pain information. Copyright 2005 Lippincott Williams & Wilkins Summary – (cont.) Chronic pain may result from increased sensitization of nociceptors and spinal level changes that perpetuate + feedback loops in the pain-spasm pain cycle. Descending impulses can influence pain perceptions through several mechanisms, including endogenous opiates. Pain can be assessed through direct measurement tools (questionnaires). Copyright 2005 Lippincott Williams & Wilkins Summary – (cont.) Therapeutic exercise is a cornerstone of treatment for chronic pain. TENS, heat, cold, and medications are components of a comprehensive treatment program. Copyright 2005 Lippincott Williams & Wilkins