chap 19 arteriosclerosis

End stage of atherosclerosis is ______________ where ________ muscle cell death in the ________ _______ caused by enlarging ________ hinders nutrient diffusion

________________ starts with the ________ _________ being damaged by carbon monoxide, bacteria, or physical factors triggering an __________ __________

LDLS are ___ __________ ____________, these chemicals deliver __________ to ________ ____ in the blood, accumulated LDLS _________ in the _____________ environment damaging neighbor cells and attracting more __________

______ _____ are macrophage and Smooth muscle cells that lose their function due to absorbing an excess of ___________ ____

healthy endothelial cells release chemicals called _____ _________ _________, these chemicals ____________ _____________ from the ________ ____ or arteries to the _____ for disposal in the _____

complicated plaques

________ __________ cell death in the ______ ______ layer caused by enlarging _______ hinders _________ ___________

_______ ________ deteriorate and are replaced with nonelastic ______ _______ and calcium salts

cause of a heart attack

The ______________ process that occurs in fatty _________ in ________ become susceptible to _________, exploding off fragments trigger ______ that cause _______ _______

Fibrous _______ form the accumulated _____ _____ decreasing vessel ________ and increasing ___________ resulting in ______________