End stage of atherosclerosis is ______________ where ________ muscle cell death in the ________ _______ caused by enlarging ________ hinders nutrient diffusion
arteriosclerosis
smooth
tunica media
plaques
________________ starts with the ________ _________ being damaged by carbon monoxide, bacteria, or physical factors triggering an __________ __________
atherosclerosis
tunica intima
immune response
LDLS are ___ __________ ____________, these chemicals deliver __________ to ________ ____ in the blood, accumulated LDLS _________ in the _____________ environment damaging neighbor cells and attracting more __________
low density lipoproteins
cholesterol
tissue cells
oxidize
inflammatory
macrophages
______ _____ are macrophage and Smooth muscle cells that lose their function due to absorbing an excess of ___________ ____
Foam cells
oxidized fat
healthy endothelial cells release chemicals called _____ _________ _________, these chemicals ____________ _____________ from the ________ ____ or arteries to the _____ for disposal in the _____
high-density lipoproteins
transport cholesterol
tissue cells
liver
bile
complicated plaques
________ __________ cell death in the ______ ______ layer caused by enlarging _______ hinders _________ ___________
_______ ________ deteriorate and are replaced with nonelastic ______ _______ and calcium salts
smooth muscle
tunica media
plaques
nutrient diffusion
Elastic fibers
scar tissue
cause of a heart attack
The ______________ process that occurs in fatty _________ in ________ become susceptible to _________, exploding off fragments trigger ______ that cause _______ _______
inflammatory
plaques
arteries
rupture
clots
heart attacks
Fibrous _______ form the accumulated _____ _____ decreasing vessel ________ and increasing ___________ resulting in ______________
plaques
foam cells
diameter
resistance
hypertension