202spinalppt

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Interferences to Rest and Activity
Needs Due to Trauma and
Degenerative Disorders, Spinal
Cord Injuries/Developmental
Alterations, Amputations
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
Lumbosacral Back Pain (Low Back
Pain)

Herniated nucleus pulposus
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
2
Health Promotion and
Maintenance
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Good posture
Proper lifting
Exercise
Ergonomics
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
3
Patient-Centered Collaborative
Care
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Assessment
Diagnostic assessment
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
4
Nonsurgical Management
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Positioning
Drug therapy
Heat therapy
Physical therapy
Weight control
Complementary and alternative therapies
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5
Surgical Management

Minimally invasive surgery:
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Percutaneous lumbar diskectomy
Thermodiskectomy
Laser-assisted laparoscopic lumbar
diskectomy
Conventional open surgical procedures:
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Diskectomy
Laminectomy
Spinal fusion
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6
Postoperative Care
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Prevention and assessment of
complications
Neurologic assessment; vital signs
Patient’s ability to void
Pain control
Wound care
CSF check
Patient positioning and mobility
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7
Community-Based Care
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Home care management
Health teaching
Health care resources
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8
Cervical Neck Pain
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Conservative treatment is the same as
described for back pain except that the
exercises focus on shoulder and neck.
If these treatments do not work, soft collar
may be used at night for a period of no
longer than 10 days.
If conservative treatment is ineffective,
surgery such as an anterior cervical
diskectomy and fusion is commonly
performed.
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
9
Spinal Cord Injuries
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Hyperflexion injury
Hyperextension injury
Axial loading injury or vertical compression
such as those that occur in jumping
Excessive rotation of the head beyond its
range
Penetration injury, such as those wounds
caused by a bullet or a knife
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10
Spinal Cord Injuries (Cont’d)
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11
Spinal Cord Injuries (Cont’d)
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12
Common Spinal Cord Syndromes
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Complete lesion
Anterior cord syndrome
Posterior cord lesion
Brown-Séquard syndrome
Central cord syndrome
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13
Common Spinal Cord Syndromes
(Cont’d)
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14
Anterior Cord Syndrome
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Damage to the anterior portion of both
gray and white matter of the spinal cord
Usually a result of decreased blood supply
Motor function and pain and temperature
lost below the level of the injury
Sensations of touch, position, and
vibration remain intact
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15
Posterior Cord Lesion
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Damage to the posterior gray and white
matter of the spinal cord
Motor function remains intact
Patient experiences loss of vibratory
sense, touch, and position sensation
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16
Brown-Séquard Syndrome
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

Results from penetrating injuries that
cause hemisection of the spinal cord, or
injuries that affect half of the spinal cord.
Motor function, proprioception, vibration,
deep touch sensations are lost on the
same side (ipsilateral) of the body as the
lesion.
Opposite side (contralateral) of the body
sensations of pain, temperature, light
touch are affected.
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17
Central Cord Syndrome
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Lesions of the central portion of the spinal
cord.
Loss of motor function is more pronounced
in the upper extremities than in the lower
extremities.
Varying degrees and patterns of sensation
remain intact.
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18
SCI: Etiology
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Trauma is the leading cause
Incidence/prevalence
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19
Patient with SCI: Initial
Assessment
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First priority is assessment of the patient’s
airway, breathing pattern, and circulation
status
Assessment for indications of intraabdominal hemorrhage or hemorrhage or
bleeding around fracture sites
Assessment of level of consciousness
using Glasgow Coma Scale
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
20
Initial Assessment (Cont’d)
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Establishment of level of injury: tetraplegia,
quadriplegia, quadriparesis, paraplegia,
and paraparesis
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21
Spinal Shock/Spinal Shock
Syndrome
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This condition occurs immediately as a
concussion response to the injury. The
patient has:
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
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Flaccid paralysis
Loss of reflex activity below the level of the
lesion
Usually resolves within 24 hours
Muscle spasticity begins in patients with
cervical or high thoracic injuries
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22
Assessment of Sensory and Motor
Ability
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Hypoesthesia
Hyperesthesia
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23
Cardiovascular and Respiratory
Assessment
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Cardiovascular dysfunction is usually the
result of disruption of the autonomic
nervous system especially if the injury is
above the 6th thoracic vertebra.
Cardiac dysrhythmias may result.
Systolic BP below 90 requires treatment
because lack of perfusion to the spinal
cord could worsen the patient’s condition.
Hypothermia.
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24
Cardiovascular and Respiratory
Assessment (Cont’d)
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Patients with cervical SCI are at risk for
respiratory problems resulting from
immobility or from an interruption of spinal
innervations to the respiratory muscles.
Continued respiratory assessment
including vital capacity and minute volume.
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25
Gastrointestinal and Genitourinary
Assessment
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Assess abdomen for indications of
hemorrhage, distention, or paralytic ileus.
Assess for reflex or hypotonic bowel.
Assess for areflexic bladder, which later
leads to urinary retention.
Assess for neurogenic bladder.
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Other Assessments
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Lower motor neuron assessment
Upper motor neuron assessment
Skin assessment
Heterotrophic ossification assessment
Psychosocial assessment
Laboratory assessment
Imaging assessment
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27
Nonsurgical Management
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Constant assessment
Assess for neurogenic shock. Neurogenic
shock is spinal shock with:
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Bradycardia
Decreased or absent bowel sounds
Warm, dry skin
Hypothermia
Hypotension
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Immobilization for Cervical Injuries
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Fixed skeletal traction to realign the
vertebrae, facilitate bone healing, and
prevent further injury
Halo fixation and cervical tongs
Stryker frame, rotational bed, kinetic
treatment table
Pin site care and monitoring of traction
ropes
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29
Immobilization of Thoracic and
Lumbosacral Injuries
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For patients with thoracic injuries—bedrest
and possible immobilization with a
fiberglass or plastic body cast
For patients with lumbar and sacral
injuries—immobilization of the spine with a
brace or corset worn when the patient is
out of bed; custom-fit thoracic lumbar
sacral orthoses preferred
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
30
Drug Therapy
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Methylprednisolone (controversial)
Dextran
Atropine sulfate
Dopamine hydrochloride
Tizanidine
Intrathecal baclofen
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31
Surgical Management
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Emergency surgery necessary for spinal
cord decompression
Decompressive laminectomy
Spinal fusion
Harrington rods to stabilize thoracic spinal
injuries
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32
Ineffective Airway Clearance and
Breathing Pattern
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Interventions for the patient with spinal
cord injury:
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Airway management is the priority.
Patients with injuries at or above the 6th
thoracic vertebra are especially at risk for
respiratory complications.
Provide measures to maintain airway.
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33
Ineffective Airway Clearance and
Breathing Pattern (Cont’d)
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Assisted coughing, quad cough, cough assist
Use of incentive Spiro meter
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34
Impaired Physical Mobility; SelfCare Deficit
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Interventions include:
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In patients with spinal cord injury, monitor for
risk of pressure ulcers, contractures, and deep
vein thrombosis or pulmonary emboli.
Proper positioning, skin inspection, ROM
exercises, heparin, and graduated
compression stockings.
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35
Impaired Physical Mobility; Self-Care
Deficit (Cont’d)
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Prevent orthostatic hypotension.
Promote self-care.
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Impaired Urinary Elimination;
Constipation
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Interventions include:
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A bladder retraining program
Spastic bladder—manipulating external area
Flaccid bladder—Valsalva maneuver
Encouraging consumption of 2000 to 2500 mL
of fluid daily to prevent urinary tract infection
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Impaired Urinary Elimination;
Constipation (Cont’d)
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
Long-term renal complication
Signs and symptoms of urinary tract
infection not perceived by the patient
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38
Autonomic Dysreflexia
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Commonly seen in patients with upper
spinal cord injury
Severe hypertension
Bradycardia
Severe headache
Nasal stuffiness
Flushing
Treatment
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Establishing a Bowel Retraining
Program
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Consistent time for bowel elimination
High fluid intake
High-fiber diet
Rectal stimulation (with or without
suppositories)
Stool softener medications, as needed
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Impaired Adjustment
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Interventions include:
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Invite patients to ask questions about
significant life changes; reply openly and
honestly.
Encourage patients to discuss their
perceptions of their situation and coping
strategies that can be used.
Begin a patient education program to clarify
misconceptions.
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41
Community-Based Care
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Home care management
Health teaching
Health care resources
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42
Multiple Sclerosis
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Chronic autoimmune disease affecting the
myelin sheath and conduction pathway of
the CNS
Characterized by periods of remission and
exacerbation
Inflammatory response resulting in random
or patchy areas of plaque in the white
matter of the CNS
Elsevier items and derived items © 2010, 2006, 2002 by Saunders, an imprint of Elsevier Inc.
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Multiple Sclerosis (Cont’d)
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Etiology
Genetic risk
Incidence
Prevalence
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Major Types of Multiple Sclerosis
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Relapsing-remitting
Primary progressive
Secondary progressive
Progressive-relapsing
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Patient-Centered Collaborative Care
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Patient history
Physical assessment/clinical
manifestations
Fatigue
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Common Physical Assessment
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Findings include:
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Flexor spasms at night
Intention tremor
Dysmetria
Blurred vision, diplopia, decreased visual
acuity, scotomas, nystagmus
Hypalgesia, numbness, tingling, or burning
Bowel and bladder dysfunction
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47
Assessment
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Psychosocial assessment
Laboratory assessment
Other diagnostic tests
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Drug Therapy
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Therapies include:
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Interferon beta
Monoclonal antibodies
Copaxone
Novantrone
Immunosuppressive therapy
Methylprednisolone
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Drug Therapy (Cont’d)
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Muscle relaxants
Treatment of paresthesia
Treatment of bladder dysfunction
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50
Other Interventions
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Promoting mobility
Managing symptoms
Complementary and alternative therapies
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Community-Based Care
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Home care management
Health teaching
Health care resources
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Guillain-Barré Syndrome
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Demyelination of the peripheral nerves,
progressive motor weakness and sensory
abnormalities
Ascending paralysis
Result of a variety of related immunemediated pathologic processes
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53
Clinical Manifestations
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Muscle weakness and pain have abrupt
onset; cause remains obscure.
Cerebral function or pupillary signs are not
affected.
Cranial nerve involvement.
Autonomic dysfunction.
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Clinical Manifestations (Cont’d)
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Weakness and paresthesia begin in the
lower extremities and progress upward
toward the trunk, arms, and cranial nerves
in ascending GBS.
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Interventions
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Drug therapy
Plasmapheresis
Monitoring respiratory status and
managing the airway
Managing cardiac dysfunction
Improving mobility and preventing
complications of immobility
Managing pain
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Interventions (Cont’d)
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Promoting communication
Providing emotional support
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Plasmapheresis
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Plasmapheresis removes the circulating
antibodies assumed to cause the disease.
Plasma is selectively separated from
whole blood; the blood cells are returned
to the patient without the plasma.
Plasma usually replaces itself, or the
patient is transfused with albumin.
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Myasthenia Gravis
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
Chronic disease characterized by
weakness primarily in muscles innervated
by cranial nerves, as well as in skeletal
and respiratory muscles
Thymoma—encapsulated thymus gland
tumor
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Myasthenia Gravis (Cont’d)
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
Progressive paresis of affected muscle
groups that is partially resolved by resting
Most common symptoms—involvement of
eye muscles, such as ocular palsies,
ptosis, diplopia, weak or incomplete eye
closure
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Tensilon Testing
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Within 30 to 60 sec after injection of
Tensilon, most myasthenic patients show
marked improvement in muscle tone that
lasts 4 to 5 minutes.
Prostigmin is also used.
Cholinergic crisis is due to overmedication.
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61
Tensilon Testing (Cont’d)
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Myasthenic crisis is due to
undermedication.
Atropine sulfate is the antidote for Tensilon
complications.
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Nonsurgical Management
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Respiratory support
Promoting mobility
Drug therapy:

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
Cholinesterase inhibitor drugs
Immunosuppression
Plasmapheresis
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Cholinesterase Inhibitor Drugs
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Drugs include anticholinesterase and
antimyasthenics.
Enhance neuromuscular impulse
transmission by preventing decrease of
ACh by the enzyme ChE.
Administer with food.
Observe drug interactions.
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Emergency Crises
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
Myasthenic crisis—an exacerbation of the
myasthenic symptoms caused by
undermedication with anticholinesterases
Cholinergic crisis—an acute exacerbation
of muscle weakness caused by
overmedication with cholinergic
(anticholinesterase) drugs
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Myasthenic Emergency Crisis
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Tensilon test is performed.
Priority for nursing management is to
maintain adequate respiratory function.
Cholinesterase-inhibiting drugs are
withheld because they increase respiratory
secretions and are usually ineffective for
the first few days after the crisis begins.
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Cholinergic Emergency Crisis
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Anticholinergic drugs are withheld while
the patient is maintained on a ventilator.
Atropine may be given and repeated, if
necessary.
Observe for thickened secretions due to
the drugs.
Improvement is usually rapid after
appropriate drugs have been given.
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Management
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Immunosuppression
Plasmapheresis
Respiratory support
Promoting self-care guidelines
Assisting with communication
Nutritional support
Eye protection
Surgical management usually involving
thymectomy
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Health Teaching
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Factors in exacerbation include infection,
stress, surgery, hard physical exercise,
sedatives, enemas, and strong cathartics.
Avoid overheating, crowds, overeating,
erratic changes in sleeping habits, and
emotional extremes.
Teach warning signs.
Teach importance of compliance.
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Trigeminal Neuralgia
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
Affects trigeminal or fifth cranial nerve
Nonsurgical management of facial pain—
drug therapy
Surgical management—microvascular
decompression, radiofrequency thermal
coagulation, percutaneous balloon
microcompression
Postoperative care—monitoring for
complications
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70
Trigeminal Nerve
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Facial Paralysis or Bell’s Palsy
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
Acute paralysis of seventh cranial nerve
Medical management—prednisone,
analgesics, acyclovir
Protection of the eye
Nutrition
Massage; warm, moist heat; facial
exercises
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Amputations
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Surgical amputation
Traumatic amputation
Levels of amputation
Complications of amputations—
hemorrhage, infection, phantom limb pain,
neuroma, flexion contracture
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Common Levels of Amputation
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Phantom Limb Pain
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
Phantom limb pain is a frequent
complication of amputation.
Patient complains of pain at the site of the
removed body part, most often shortly
after surgery.
Pain is intense burning feeling, crushing
sensation, or cramping.
Some patients feel that the removed body
part is in a distorted position.
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Management of Pain
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
Phantom limb pain must be distinguished
from stump pain because they are
managed differently.
Recognize that this pain is real and
interferes with the amputee’s ADLs.
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Management of Pain (Cont’d)


Opioids are not as effective for phantom
limb pain as they are for residual limb pain.
Other drugs include beta blockers,
antiepileptic drugs, antispasmodics, and IV
infusion of calcitonin.
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Exercise After Amputation
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

ROM to prevent flexion contractures,
particularly of the hip and knee
Trapeze and overhead frame
Firm mattress
Prone position every 3 to 4 hours
Elevation of lower-leg residual limb
controversial
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Stump Care
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Prostheses
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

Devices to help shape and shrink the
residual limb and help patient adapt
Wrapping of elastic bandages
Individual fitting of the prosthesis; special
care
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Complex Regional Pain Syndrome


A poorly understood complex disorder that
includes debilitating pain, atrophy,
autonomic dysfunction, and motor
impairment
Collaborative management—pain relief,
maintaining ROM, endoscopic thoracic
sympathectomy, and psychotherapy
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Cerebral Palsy


Copy and past the URL below to see a
video ppt presentation on CP
http://www.authorstream.com/Presentation
/laqueitaowens23-388993-cerebral-palsypower-point-presentation-education-pptpowerpoint/
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Spina Bifida

The most common, permanent and
disabling birth defect in the United
States
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What are the effects of Spina
Bifida?
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Ongoing medical challenges
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
Full or partial paralysis
Nervous system complications
Bladder and bowel control difficulties
Learning disabilities
Depression
Social and sexual issues
Latex allergy
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
Latex allergy


70% of children and adolescents with Spina
Bifida are sensitive to latex
Items made with latex include:
• Promotional items like stress balls
• Balloons
• Rubber bands
• Rubber erasers
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How does Spina Bifida
happen?

First month of pregnancy


Before most women know they are
pregnant
The spine does not close


Causes an opening or lesion
Damaged nerves may result in varying
degrees of paralysis
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What happens after birth?


Surgery
 Reduces risk of infection
 Further damage to the spinal
cord
Life-long treatment and care
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Can Spina Bifida be prevented?



Exact cause is not known
Medical research confirms link between
folic acid and Spina Bifida*
Women who take 400 mcg of folic acid
every day reduce the risk by up to 70%.


BEFORE becoming pregnant
During first three months of pregnancy
* (and other birth defects)
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What is folic acid?



B-vitamin that builds healthy cells
In most multivitamins
Foods



Leafy green vegetables (broccoli and spinach)
Fruits and juices (orange juice)
Folic acid-fortified breakfast cereals and other
bread and grain products
The surest way to get enough folic acid every day
is to take a vitamin with folic acid.
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