Nadia Northway Deviation varies with size and or direction of gaze In truth nearly all forms of strabismus are incomitant to a degree but clinically there is usually more than 5o difference before incomitancy is noted. C la ssifica tion C o n ge n ital N e u rog e n ic T h ird n e rve p a lsy F o u rth n erve p a lsy S ixth n e rve p la sy M e cha n ical B ro w n's S ynd ro m e D u a ne 's S yn dro m e Classification Acquired Adults and childhood Neurogenic Third nerve palsy Fourth nerve palsy Sixth nerve plasy Mechanical Brown's Syndrome Duane's Syndrome Rare Myogenic Dysthyroid Eye disease Myasthenia G ravis Vascular affects all nerves equally Head trauma more commonly affects IVth nerve but may affect all Aneurysm most commonly affects IIIrd nerve Neoplasm Unknown Other Diabetes Thyrotoxicosis Hypertension Aneurysm Giant cell arteritis Multiple Sclerosis Myasthenia Gravis History and symptoms External Examination Cover test Motility Ophthalmoscopy Fields Diplopia Abnormal head posture-chin, turn and tilt Acuity Associated symptoms General health Injury Strabismus Lid position Injury- chemosis, oedema Proptosis Pupils Asymmetry Always turn in direction of action of palsied muscle e.g. LMR palsy will turn to right Always move chin in direction of action of palsied muscle e.g. LSR palsy will elevate chin Always tilt to lower eye Small deviation in primary position may indicate very recent onset < 36 hours or mechanical problem In palsy- will be greater when fixing with the affected eye and usually larger size of deviation Know muscle actions Take patients eyes into extremes of gaze Use objective and subjective assessmentcorneal reflexes and CT. Do not rely on pt reporting diplopia since suppression or poor VA may affect results. Hess chart and diplopia chart. RAD SIN- recti adduct and superiors intort Recti muscles pull the eye in the direction of their name in the abducted position Obliques push the eye in the direction opposite to their name in the adducted position Original palsy Overaction of the contralateral synergist Overaction of the ipsilateral antagonist Inhibitional palsy This applies to neurogenic palsy and after all stages of sequelae have occurred concomitancy is achieved IO IO SR SR MR LR LR IR SO SO Overaction of contralateral synergist only Left Brown’s syndrome overaction of right superior rectus is seen Look for smallest field to identify affected eye Look at center circle to determine deviation in primary position Look for area with greatest deflection to identify affected muscles Used to differentiate between SR and SO palsy Muscle sequelae identical In left SO palsy deviation will increase when head tilted to left due to unopposed action of the LIO Complete or partial Rare to find individual muscles affected but Congenital SR palsy quite common May also be multiple muscle involvement including pupil and ciliary body Hypotropia of affected eye and may be slightly exo Chin elevation Can be longstanding -usually have enlarged fusion range and some suppression Hypertropia in primary position Hypotropia in primary position with possible slight eso. Exo deviation Exotropia with hypotropia, ptosis and possible dilation of pupil and accommodation palsy Esotropia which is greater on distance fixation Hypertropia with slight eso , eye also extorted, greater at near Small devation in primary position but hypotropia of affected eye on elevation in adduction May be hypotrpia or hypertropia Infraorbital anaesthesia Chemosis Vertical diplopia Restricted eye movement in upgaze and downgaze Wet phase when muscles swell -myogenic Dry phase when eye movement restrictions become mechanical in characteristics Muscles affected - IR MR SR rarely LR Proptosis or exophthalmos Check Fields Lid retraction and lid lag Mechanical Small deviation in pp Ductions and versions equal Ceasing of movement abrupt Pain Reversal of diplopia Upshoots and downshoots Neurogenic Large deviation in pp Ductions better than versions Gradual failure of movement No pain No upshoots and downshoots Differentiation of mechanical and neurogenic palsy • Mechanical • Muscle sequelae- only overaction of contra syn • Hess chart -pointed field which look squashed • Neurogenic • Full muscle sequelae • Smoother filed on Hess Longstanding AHP - fixed and pt usually unaware No diplopia Enlarged fusion ranges Old photographs Gradual onset of symptoms usually Amblyopia Suppression Newly acquired Pt aware of AHP and uncomfortable Diplopia Sudden onset No enlarged fusion range Differentiate SR and SO palsy • SO • SR • • • • • Exo deviation more typical • AHP- chin elevation • V exo pattern • Greater deviation in distance • Bielchowsky -ve • May have history of ptosis • Diplopia greatest on elevation Eso deviation more typical AHP - chin depression V eso pattern Greater vertical deviation at near • Bielchowsky +ve • Diplopia greatest on depression Sudden onset diplopia Incomitant deviation previously unidentified Uncomfortable head posture Patient has localisation disturbance Patient symptomatic Other signs and symptoms