Part II “Air-Fluid Levels” seen in bowel obstruction Anti-Acids (Antacids) Prototype: aluminum hydroxide gel (Amphojel) Physical Assessment Inspection Palpation Percussion Auscultation KEY ASSESSMENTS Lab Monitoring Prokinetic Agents: Prototype: metoclopramide (Reglan) Histamine 2 Receptor Agonists Prototype: ranitidine hydrochloride (Zantac) ***Diagnostic Testing Proton Pump Inhibitors) Prototype: omeprazole (Prilosec) Mucosal Barriers Prototype: sucralfate (Carafate) Disease Specific Medications: Care Planning Plan for client adl’s, Monitoring, med admin., Patient education, more…based On Nursing Process: A_D_O_P_I_E ***Preparing for Diagnostic Tests Nursing Skills: NG Tube Insertion Enteral Feedings Nursing Interventions & Evaluation Execute the care plan, evaluate for Efficacy, revise as necessary Gastritis Dumping Syndrome Small & Large Intestines Appendicitis Peritonitis Diverticulitis Ulcerative Colitis Crohn’s Disease Bowel Obstruction Irritable Bowel Syndrome (IBS) Hemorrhoids Polyps Bowel Cancer Inflammation of the gastric mucosa Types: erosive vs. non-erosive Acute vs. Chronic S&S: Abdominal tenderness, bloating, hematesis, melena Diagnostic: EGD with biopsy Management: see GERD Rapid gastric emptying into the small intestines usually occurs after a gastric surgery Types: Early and Late EARLY 30 min after eating Rapid emptying Vertigo Syncope Pallor Diaphoresis Tachycardia palpitations LATE 90 min-3 hr after eating Excessive insulin release Abdominal distention Cramping Nausea Dizziness Diaphoresis confusion Lying down after a meal Eliminate liquids with meals Avoid milk, sweets, or sugars Eat small frequent meals Consume high protein and fat with low to moderate carbohydrate Pectin Oral: slows absorption of carbs Octreotide SQ: blocks gastric and pancreatic hormones Postprandial B L O O D G L U C O S E L E V E L Hypoglycemia Increased blood glucose level increases the release of insulin. Insulin causes the blood glucose levels to go down…. Time----------- “The Somogyi Effect”, a.k.a., “Rebound Effect” Movement Digestion Absorption Movement Absorption Elimination Acute inflammation of veriform appendix Lower right quadrant pain Low grade fever Nausea and vomiting Rebound tenderness @Mc Burney’s point Rosving sign positive Increased WBC Monitor pain (severe rebound tenderness) Monitor bowel sounds (absent) NPO, IVF, NO laxatives or enemas Surgical management: -Open or laparoscopic appendectomy Ultrasound Abdominal x-ray Abdominal CT scan Acute pain Alteration in comfort Risk for injury Knowledge deficit Risk for infection Monitor vital signs Assess bowel sounds Monitor pain Monitor lab values Post operative management: -Vitals signs, bowel sounds, diet resumption, antibiotic therapy as ordered Acute inflammation of the visceral / parietal peritoneum and endothelial lining of abdominal cavity Types: primary and secondary PRIMARY Acute bacterial infection Contamination of peritoneum via vascular system TB (tuberculin infection) Alcoholic cirrhosis Leakage SECONDARY Usually caused by a bacterial invasion in the abdomen Gangrenous bowel Blunt or penetrating trauma Leakage Rigid board like abdomen Abdominal pain/tenderness Distended abdomen Nausea and vomiting Diminished to no bowel sounds No stools or flatus Fever Tachycardia CBC (WBC, H&H) Electrolytes CR (creatinine) & BUN (Blood urea nitrogen) Abdominal x-ray CT scan Peritoneal lavage Surgery Non-surgical: -IV fluids -Broad spectrum antibiotics -Intake and outputs (I&O) -NG (nasogastric) tube -NPO -Pain management Surgical: Optimal treatment Exploratory laparotomy: repair or remove inflamed organ Peritonitis: EMERGENCY / Life Threatening -Symptoms: rigid abd., distended abd., absent bowel sounds, high fever, decreased urine output, hypotension Fluid shifts from extracellular to peritoneal cavity Inflammation of one or more diverticula. Results when diverticulum perforates and a local abscess forms Abdominal pain, tenderness to palpation Elevated temperature >101, may have chills Abdominal guarding, rebound tenderness CT scan Abdominal flat plate EGD DO NOT do barium enema with active untreated diverticulitis Non Surgical: -Broad spectrum antibiotics -Anticholinergics -NPO until clear liquids tolerated -Stop fiber therapy until attack is limited -NO enemas or laxatives Surgical -completed for ruptured peritonitis, fistula formation, bleeding, bowel obstruction, or unresponsive medical management Health teaching: diet, fiber, symptom recognition, activity Post op management: -Monitor colostomy, if present -monitor VS, urine output, wound condition -Psychosocial adjustment to stoma Ulcerative colitis: Chronic inflammatory process affecting mucosal lining of colon or rectum 10-20 liquid stools per day Tenesmus (Straining) Anemia Fatigue LLQ pain/cramping Wt loss CT scans Colonoscopy or Siqmoidoscopy Barium Swallow studies Stools for O&P, occult blood, & C&S Labs: electrolyte panel and CBC Salicylate: -inhibit prostglandins to reduce inflammation Corticosteroids: -Suppress immune system and reduce inflammation Immunomodulators: -reduce steroid use and overrides body immune system Antibiotics: -acute exacerbations prone to infection Anti-diarrheals: -Symptomatic relief of severe diarrhea NPO if symptoms are severe TPN if NPO for extended time Elemental formula Low fiber foods Lactose free products No caffeine, spices, alcohol, or smoking Surgery is curative Total colectomy with permanent ileostomy Total colectomy with continent ileostomy (Kock’s pouch) Pain acute and chronic Fluid volume deficit Alteration in nutrition Nutritional assessment Monitoring fluid and electrolytes Monitor lab values Monitor for complications Monitor weight Psychosocial assessment Post operative care Hemorrhage/perforation Coagulation problems Malabsorption Increase risk for colon cancer Toxic megacolon Inflammatory disease of small intestines, colon, or both (terminal ileum) 5-10 fatty stools per day (steatorrhea) Flatus Malabsorption Weight loss Diffuse bilateral lower quadrant pain Fever with perforation or fistula Fluid, electrolyte and vitamin deficits CBC Electrolyte panels Vitamin & folic acid levels Albumin & nutritional labs Barium studies Colonoscopy Drug Therapy -Salicylate -Corticosteriods -Immunomodulators -Biologic Therapy -Antibiotics (abscess/perforation) TPN for long term use Nutritional supplements Elemental supplements No caffeine or carbonated beverages No ETOH Prebiotics (non-digestive food ingredients) Surgery is NOT a “cure” Repair of fistulas Release of intestinal obstructions Partial resection with primary anastamosis Ileostomy Intestinal obstruction Fistulas Malabsorption syndrome Liver and biliary diseases Kidney stones Arthritis Administering PPN and TPN Provide adequate nutrition: pre-medicate as ordered Assess stools: quality, frequency, amount, and pain issues with stooling Assess vital signs Teach relaxation techniques Education for ileostomy or colostomy for both client and family Reduce or eliminate factors that cause diarrhea and pain Chronic pain management Provide small frequent meals with specific dietary preferences Detailed abdominal assessment SMALL INTESTINES Pain is spasmodic Peristaltic waves Profuse projectile vomiting Feculent odor to emesis “Air-Fluid Levels” in intestinal obstruction LARGE INTESTINES Vague diffuse constant pain Abdominal distention Infrequent vomiting Possible diarrhea MECHANICAL Adhesions Tumors Volvulus Intussusception Fecal impactions Foreign Bodies / Objects NON-MECHANICAL Decreased peristalsis Electrolyte imbalance Inflammatory response Neurogenic disorder Vascular disorder Foreign Body in the Colon Dehydration Perforation Ischemic or strangulated bowel Metabolic acidosis and Alkalosis Chronic disorder of diarrhea and constipation No exact cause known Affects women 3x more then men Possible causes: diet and behavioral (psychological) illness “Manning Criteria:” -abdominal pain relieved by defecation -abdominal distention -sensation of incomplete BM (bowel movement) -Presence of mucus Exacerbation (flare up): -worsening cramps -abdominal pain (LLQ) -diarrhea or constipation -increased pain after eating -nausea with defecation and mealtime CBC Serum albumin Stools for occult blood Sigmoidoscopy Colonscopy Stress Management Diet Therapy: -Avoid lactose products, caffeine, ETOH, sorbitol or fructose -Increase fiber (30-40 gm) -Fluid intake of 8-10 cups per day -meal planning Monitor Drug Therapy -laxatives -diarrheals / antidiarrheals -anticholinergic -tricyclic antidepressants -muscarinic receptor antagonist -antispasmatics -5HT4 (Zelnorm) Swollen or distended veins in rectal region Internal & external Cause: pregnancy, obesity, constipation Symptoms: bleeding, edema, and prolapsed Treatment: cold packs, sitz bath, diet, Tucks ®, topical anesthetics, and surgery “The Jackknife Position” Rectal Surgery Small growths covered with mucosa and attached to the surface of intestines Asymptomatic-bleeding, obstruction, & intussusception Benign vs. malignant Colorectal cancer Colon and rectum=large intestines Molecular changes Metastasize & tissue to blood, lymph, surrounding Purpose for Naso-Gastric Tubes: 1. Decompression 2. Feeding 3. Administration of Medications ***4. Lavage General Golden Rule for Feeding Tubes: Ensure correct placement prior to putting ANYTHING DOWN a TUBE!!! X-Ray Confirmation At 1st looks OK but distal tip NOT SEEN This tube ended up exiting the mid abdomen with the feedings entering the peritoneal cavity Tube feeding formula remaining in contact with gastric acid can result in the precipitation of casein and the subsequent formation of a solid mass around the tube Date & time Reason for insertion Type of tube Size of tube Length of tube Nostril tube inserted Number of attempts required Additional comments Any complications Method of placement confirmation Signature: name & designate of Nurse inserting tube Pharmacological Action Neutralize gastric acid and inactivate pepsin. Evaluation of Medication Effectiveness Mucosal protection may occur by the antacid’s ability to stimulate the production of prostaglandins. Depending on therapeutic intent, effectiveness may be evidenced by: Therapeutic Uses Healing of gastric and duodenal ulcers. Reduced frequency or absence of GERD symptoms. No signs or symptoms of GI bleeding. Treat peptic ulcer disease (PUD) by promoting healing and relieving pain. Symptomatic relief for clients with GERD. Nursing Interventions and Client Education Clients taking tablets should be instructed to chew the tablets thoroughly and then drink at least 8 oz of water or milk. Teach the client to shake liquid formulations to ensure even dispersion of the medication. Compliance is difficult for clients because of the frequency of administration. Administered seven times a day: 1 hr before and 3 hr after meals, and again at bedtime. Teach clients to take all medications at least 1 hr before or after taking an antacid. Back to Concept Map Pharmacological Action Block dopamine and serotonin receptors in the chemoreceptor trigger zone (CTZ), and thereby suppress emesis. Prokinetic agents augment action of acetylcholine which causes an ↑ in upper GI motility. Therapeutic Uses Control postoperative and chemotherapyinduced nausea and vomiting. Prokinetic agents are used to treat GERD. Prokinetic agents are used to treat diabetic gastroparesis. Side Effects / Adverse Effects Extra Pyramidal Symptoms (EPS) Sedation Diarrhea Contraindications / Precautions Contraindicated in clients with GI perforation, GI bleeding, bowel obstruction, and hemorrhage Contraindicated in clients with a seizure disorder due to ↑ risk of seizures Use cautiously in children and older adults due to the ↑ risk for EPS. Nursing Interventions and Client Education Monitor clients for CNS depression and EPS. Can be given orally or intravenously. If dose is < 10 mg, it may be administered undiluted over 2 min. If the dose is > 10 mg, it should be diluted and infused over 15 min. Dilute medication in at least 50 mL of D5W or lactated Ringer’s solution. Evaluation of Medication Effectiveness Control of nausea and vomiting Back to Concept Map Overview Tardive dyskinesia is a disorder that involves involuntary movements, especially of the lower face. Tardive means "delayed" and dyskinesia means "abnormal movement." Symptoms Facial grimacing Jaw swinging Repetitive chewing Tongue thrusting Tardive dyskinesia is a serious side effect that occurs when you take medications called neuroleptics. It occurs most frequently when the medications are taken for a long time, but in some cases it can also occur after you take them for a short amount of time. The drugs that most commonly cause this disorder are older antipsychotic drugs, including: Haloperidol Fluphenazine Trifluoperazine Other drugs, similar to antipsychotic drugs, that can cause tardive dyskinesia include: Cinnarizine Flunarizine (Sibelium) Metoclopramide Prognosis Next Page Causes If diagnosed early, the condition may be reversed by stopping the drug that caused the symptoms. Even if the antipsychotic drugs are stopped, the involuntary movements may become permanent and in some cases may become significantly worse. Pharmacological Action Suppress the secretion of gastric acid by selectively blocking H2 receptors in parietal cells lining the stomach. Treatment of peptic ulcer disease is usually started as an oral dose twice a day until he ulcer is healed, followed by a maintenance dose, which is usually taken once a day at bedtime. Evaluation of Medication Effectiveness Therapeutic Uses Gastric and peptic ulcers, gastroesophageal reflux disease (GERD), and hypersecretory conditions, such as Zollinger-Ellison syndrome. Used in conjunction with antibiotics to treat ulcers caused by H. pylori. No signs or symptoms of GI bleeding. Therapeutic Nursing Interventions and Client Education Healing of gastric and duodenal ulcers. Depending on therapeutic intent, effectiveness may be evidenced by: Reduced frequency or absence of GERD symptoms (e.g., heartburn, bloating, belching). Encourage client to avoid aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs). Ranitidine can be taken with or without food. Back to Concept Map Pharmacological Action Reduce gastric acid secretion by irreversibly inhibiting the enzyme that produces gastric acid. Reduce basal and stimulated acid production. Therapeutic Uses Prescribed for gastric and peptic ulcers, GERD, and hypersecretory conditions (e.g., Zollinger-Ellison syndrome). Precaution: The client should take omeprazole once a day prior to eating. Encourage the client to avoid irritating medications (e.g., ibuprofen and alcohol). Active ulcers should be treated for 4 to 6 weeks. Pantoprazole (Protonix) can be administered to the client intravenously. Monitor the client’s IV site for signs of inflammation (e.g., redness, swelling, local pain) and change the IV site if indicated. Teach clients to notify the primary care provider for any sign of obvious or occult GI bleeding (e.g., coffee ground emesis). Evaluation of Medication Effectiveness Depending on therapeutic intent, effectiveness may be evidenced by: Increases the risk for pneumonia. Omeprazole ↓ gastric acid pH, which promotes bacterial colonization of the stomach and the respiratory tract. Use cautiously in clients at high risk for pneumonia (e.g., clients with COPD). Healing of gastric and duodenal ulcers. Reduced frequency or absence of GERD symptoms (e.g., heartburn, sour stomach). No signs or symptoms of GI bleeding. Other PPI’s: Nursing Interventions and Client Education Do not crush, chew, or break sustained-release capsules. The client may sprinkle the contents of the capsule over food to facilitate swallowing. omeprazole; lansoprazole; rabeprozole; pantoprazole; esomeprazole; Back to Concept Map Pharmacological Action Changes into a viscous substance that adheres to an ulcer; protects ulcer from further injury by acid and pepsin. Viscous substance adheres to the ulcer for up to 6 hr. Sucralfate has no systemic effects. Therapeutic Uses Acute duodenal ulcers and maintenance therapy. Investigational use in gastric ulcers and gastroesophageal reflux disease. (GERD) Nursing Interventions and Client Education Assist the client with the medication regimen. Instruct the client that the medication should be taken on an empty stomach. Instruct the client that sucralfate should be taken four times a day, 1 hr before meals, and again at bedtime. The client can break or dissolve the medication in water, but should not crush or chew the tablet. Encourage the client to complete the course of treatment. Evaluation of Medication Effectiveness Depending on therapeutic intent, effectiveness may be evidenced by: Healing of gastric and duodenal ulcers. No signs or symptoms of GI bleeding. Back to Concept Map Blood Tests Complete Blood Count (CBC c Diff) Stool Tests: Stool for occult blood; (Guiac) Stool for ova & parasites (O&P); Stool for Clostridium difficile (C-Diff) Stool Culture & Sensitivity (C&S) Upper GI Series (UGI) Upper GI Series with Small Bowel Follow- Through (UGI-SBFT) Barium Enema Endoscopy Return to Concept Map http://www.saddleback.edu/alfa/n170/tubefeeding.aspx Tum-E-Vac? Salem Sump Levin Tube (single lumen) Maloney JPEN 2002;26:S34-42 FDA advisory FD&C Blue No. 1 Intermittent gravity Intermittent Via Pump: -continuous (or) cyclic