Orthopedic Principles William Beaumont Hospital Department of Emergency Medicine Fractures in Kids – Salter Harris Classification Injuries to epiphyseal growth plate result from compressive or shearing force The weak cartilaginous growth zone separates before tendons or bones If unsure, get comparison views Type I and V not always evident on x-ray, so immobilize if clinically suspect fracture Salter Harris Classification Mnemonic “ ME ” What Salter-Harris type is this? Type 2 What Salter-Harris type is this? Type 3 What Salter-Harris type is this? Type 3 What Salter-Harris type is this? Type 4 What Salter-Harris type is this? Type 1 Examination Basics Determine the point of maximum tenderness Examine the joint above and below the site of injury Check for joint stability Check the neurovascular status Is the fracture open? Neurovascular compromise requires emergent reduction of the fracture or dislocation Early Ortho consult Signs of compartment syndrome – Five P’s Compartment Syndrome Ischemic injury to muscles and nerves in a closed fascial compartment Caused by edema in a closed compartment Decreased venous return Eventual decreased arterial flow Commonly seen with tibia or forearm fractures Most common lower extremity compartments: anterior > lateral > deep posterior > posterior Most common upper extremity compartment: deep flexor compartment Compartment Syndrome Fracture not necessary Can occur with excessive muscle contractions, crush injury, circumferential burns, prolonged compression (i.e. drug OD) Earliest and most reliable sign is referred pain to the compartment with passive stretch of the ischemic muscle group (i.e. plantar foot flexion causes pain in the anterior leg compartment) Compartment Syndrome - Signs Pain burning, poorly localized, disproportionate to injury pain on active or passive stretch of muscles Paresthesias in distribution of nerves Pallor – late and ominous sign Paralysis Pulselessness – late and ominous sign Compartment Syndrome Diagnosis – hand held device for measuring compartment pressure Pressures > 30mmHg are abnormal Treatment – immediate fasciotomy Associated Nerve Injuries ORTHO INJURY NERVE INJURY Elbow injury Shoulder dislocation Sacral fracture Acetabular fracture Hip dislocation Femoral shaft fracture Knee dislocation median or ulnar axillary cauda equina sciatica femoral peroneal tibial or peroneal Radiographic Evaluation Rule of two's Minimal of 2 views perpendicular to each other when possible Include 2 joints Include 2 limb comparison views 2 sets of X-rays Pre-reduction and post-reduction films; obtain prereduction x-rays unless neurovascular compromise Possible repeat x-ray in 7-10 days for suspected occult fractures (i.e. scaphoid fractures) Radiographic Evaluation Is the fracture intraarticular? – increased risk of subsequent arthritis Are the fragments distracted? Is there a joint dislocation? Treatment Principles The first priority = ABCs Obvious fractures should NOT deter from ABCs. Hypovolemic shock possible secondary to fractures Pelvic fracture – 2 Liters Femur fracture – 1.5 Liters Multiple fractures Worsened by third spacing Treatment Principles For fractures: immobilize the joint proximal and distal For joint injuries: immobilize the affected joint only Reassess neurovascular status after immobilization or manipulation Consider analgesia and/or sedatives prior to attempting reduction Treatment Principles Plaster splinting Circumferential casting rarely done in the ED for an acute fracture evolving edema may lead to compartment syndrome Ice and elevate for 48 hours post injury Healing occurs over 4-10 weeks if properly immobilized Cervical Spine Injuries Etiology: MVC 50% Falls 20% Sports 15% Classified as stable or unstable and by injury mechanism (flexion, extension, rotation, compression) Anterior column – vertebra, discs, and anterior and posterior longitudinal ligaments Posterior column – spinal cord, pedicles, facets, spinous processes, held together by the nuchal and capsular ligaments, and ligamentum flavum Let’s move on to the specifics… Anatomy of the Cervical Spine Navigating the C Spine X-ray Count vertebrae – if you don’t see C7 and the C7-T1 interface the film is inadequate The Key – integrity of the anterior cervical line, posterior cervical line and spinolaminar line Anterior cervical line maintained by anterior longitudinal ligament Posterior cervical line maintained by the posterior longitudinal ligament Spinolaminar line maintained by ligamentum flavum Cervical Spine Xray Unstable Cervical Spine Injuries An unstable C spine injury occurs when there is disruption of the ligaments of the anterior and posterior column elements Chance of spinal cord injury great Unstable Cervical Spine Injuries C1 (Jefferson burst fracture) C2 (Hangman fracture) Odontoid fracture Flexion tear drop fracture Bilateral facet dislocation Jefferson Burst Fracture Burst fracture of C1 ring Mechanism: axial loading force on the occiput Diving into shallow water Falling from a height Lateral displacement of the lateral masses Disruption of the transverse ligament Unstable, but often no neuro deficit because the ring widens when it fractures limiting cord compression Jefferson Burst Fracture Hangman Fracture Mechanism: skull is thrown into extreme hyperextension as a result of abrupt deceleration (i.e. MVC). Bilateral fractures of the pedicles of C2 Spinal cord damage is minimal because the bilateral fractures allow the spinal cord to decompress Unstable fracture Hangman Fracture Odontoid Fracture 15% of all C spine fractures Mechanism: MVC or fall Type 1 – tip fracture Type 2 – base fracture, unstable, most common 60% of odontoid fx Type 3 – thru body of C3 very unstable Odontoid Fracture Flexion Tear Drop Fracture Mechanism: flexion and axial loading forces cause avulsion of anteroinferior portion of vertebral body Involves injury to anterior and posterior longitudinal ligaments creating spinal instability Often associated with spinal cord damage Unstable fracture Flexion Tear Drop Fracture Stable Cervical Spine Injuries Wedge fracture Vertebral body burst fracture Clay Shoveler’s fracture Transverse process fracture Unilateral facet dislocation Wedge Fracture Mechanism: Flexion injury causes a longitudinal pull on the nuchal ligament complex that, because of its strength, usually remains intact. The anterior vertebral body bears most of the force, sustaining simple wedge compression anteriorly without any posterior disruption. The prevertebral soft tissues are swollen. Stable fracture Wedge Fracture Vertebral Burst Fracture Mechanism: Downward compressive force is transmitted to lower levels in the C spine vertebra can shatter outward, causing a burst fracture Disruption of anterior and posterior longitudinal ligaments Posterior protrusion of the fracture may extend into the spinal canal and be associated with anterior cord syndrome Burst fractures require a CT or MRI to document degree of retropulsion Vertebral Burst Fracture Clay Shoveler’s Fracture Avulsion of C6 or C7 spinous process Mechanism: Abrupt flexion of neck combined with muscular contraction of upper body/neck muscles; can also result from a direct blow to neck Seen best on lateral C spine X-ray Stable fracture Clay Shoveler’s Fracture Any questions about the cervical spine? Let’s Move On Anterior Shoulder Dislocations Mechanism: abduction and external rotation with a posterior force (line backer injury) 98% are anterior Signs & Symptoms: squared shoulder, held in abduction/external rotation, anterior shoulder appears full Check axillary nerve function: abduction of arm and sensory sergeant stripe distribution Treatment: closed reduction by hanging weight, scapular manipulation, traction/countertraction Posterior Shoulder Dislocations 2% are posterior Most common cause is from a seizure X-ray – light bulb sign Anterior Shoulder Dislocations Forearm Fractures Monteggia fracture – fracture of the proximal 1/3 ulna with an associated radial head dislocation Galeazzi fracture – fracture distal 1/3 radius with dislocation of the distal radioulnar joint Treatment – urgent Ortho consult for operative repair Monteggia Fracture Galleazzi Fracture Significance of the Fat Pad Sign Anterior fat pad may be seen in normal elbow but usually is a thin strip Posterior fat pad sign indicates occult fracture – in children indicates supracondylar fracture and in adults indicates radial head fracture Pathophysiology – intraarticular hemorrhage or effusion causes distention of synovium making posterior fat pad visible on X-ray Fat Pad Sign normal anterior fat pad abnormal fat pad Wrist Fractures – Colles Fracture Distal radius fracture with dorsal displacement of the distal fragment Mechanism: Swan fall on an outstretched hand neck or dinner fork deformity non-displaced volar splint; displaced/angulated ortho referral Treatment: Colles Fracture Wrist Fractures – Smith’s Fracture Distal radius fracture with volar displacement of the distal fragment Mechanism: fall backwards on outstretched hand, direct blow Treatment: same as Colles Smith’s Fracture Exam of the Injured Hand – Tendon Examination Flexor Digitorum Profundis Flexor Digitorum Superficialis Flex DIP joint against resistance, while blocking MCP and PIP action Flex MCP joint - block other digits With partial tendon laceration - may be able to flex or extend, but will be weak or painful Exam of the Injured Hand Sensory exam Ulnar: tip of little finger Median: tip of middle finger or pad of index finger Radial: 1st dorsal web space Motor exam Ulnar: Spear fingers against resistance, injury causes a claw hand Median: oppose thumb (recurrent branch) injury causes thenar eminence muscles to atrophy giving the hand an “apelike” appearance Radial: extend wrist, injury causes wrist drop Boxer’s Fracture 5th metacarpal fracture Mechanism: punching Be suspicious of any laceration over the knuckles Often the result of fist hitting mouth High incidence of infection, may need antibiotics Treatment: ulnar gutter splint to the PIP joint Boxer’s Fracture Mallet Finger Mechanism: distal tip of finger is forcibly flexed, resulting in rupture or avulsion of the lateral expansions of the extensor hood Diagnosis: unable to extend DIP joint; defect may not be seen for 5-7 days Treatment: splint DIP joint in slight hyperextension for full 6 weeks Mallet Finger Boutonniere Deformity Mechanism: injured central band of the extensor hood Diagnosis: painful, swollen PIP joint tenderness over PIP joint fixed flexion of PIP hyperextension of DIP, unable to extend PIP Treatment: splint only the PIP joint in extension Boutonniere Deformity Carpal Bone Injuries – Scaphoid Fractures Most common carpal bone fracture Mechanism: fall on the outstretched palm Diagnosis: snuff box tenderness or tenderness with axial loading of thumb Treatment: thumb spica with volar splint Complications: Avascular necrosis if not correctly immobilized Non-union, because scaphoid with unique distal origin of blood supply Scaphoid Fractures OK, let’s move down Pelvic Fractures The pelvis is a ring structure, so if see 1 fracture you need to check for another Associated with bladder rupture or membranous urethral injuries Higher incidence with symphysis pubis fracture Retrograde cystourethrogram if: High riding boggy prostate Blood at urethral meatus Common cause of mortality is hemorrhagic shock Open book fracture Displacement of pelvic fracture > 0.5 cm Pelvic Fractures Hip Dislocations 80-90% are posterior high energy injury Mechanism: strike knee on dash, while leg flexed and adducted Presentation: leg flexed, adducted, shortened, and internally rotated with knee resting on opposite thigh Associated injuries: patellar fracture, sciatic nerve (peroneal branch), femoral vessels Treatment: immediate attempt at closed reduction Complications: Avascular necrosis Hip Dislocations Hip Dislocations Hip Fracture Hip Fracture Hip Pain – Differential Diagnosis Referred pain from back or knee Herniated disc Discitis Toxic synovitis, bursitis, tendonitis of hip Septic joint Occult fracture of hip Tumor (lymphoma) DVT or arterial ischemia Osteomyelitis Slipped capital femoral epiphysis Tibial Plateau Fracture High energy injury in younger age group Fall from height MVC Low energy injury due to compressive forces on osteoporotic bones Complications: Popliteal artery injury Lateral condyle fracture Ligamentous injuries Compartment syndrome Tibial Plateau Fractures Knee Dislocation Described by position of tibia in relation to femur Considered high energy injury Anterior dislocation most common and caused by hyperextension of knee Posterior dislocation from direct trauma to flexed knee Initial evaluation may not reveal obvious deformity because of spontaneous reduction so grossly unstable knee treated as if dislocation occurred Knee Dislocations - Complications Vascular injury to popliteal artery Crucial to document DP & DT pulses Highly predictive of arterial injury when diminished or absence Diagnose with arteriogram Early revascularization within 6 hours decreases risk of amputation Peroneal nerve injury Check sensation on dorsum of foot Dorsiflex the ankle Knee Dislocations The End