CESTODOSIS
MONIEZIASIS
Cause:
 Moniezia expansa
 Moniezia benedini
Sensitive animal:
 M. benedini: cattle (main host) & others ruminants
M. expansa: sheep,goat,cattle,& others ruminants
Predilection: Small intestine
Route of infections:
Ingested by cysticercoid g mites
(fam: Oribatidae) together with the grass
Pathogenicity:
- Young animals (< 6 bl) very sensitive.
- The degree of infections depend on the amount of
cysticercoid
- Enteritis
Clinical symptoms:
Its not so clear in general,weakness&thin
Acute: Intoxication because of toxin produced by the
excretion of the adult worm
Mild infections: Gastrointestinal disturbance
(indigesti) & inhibited of the bodies growth
Heavy infections: Anaemia, watery diarrhea, inhibited
of the bodies growth, for the young cattle it must be
fatal
Diagnoses
 Fecal examination  Eggs  or 
 Segment/proglottids
Egg of M. benedini
Control by:
Dichlorophene 300-600 mgs/kgs BW
Yomesan 75 mgs/kgs BW
 Control for the mites, photophobia is the
characteristic of mites.
CYSTICERCOSIS CELLULOSE
CYSTICERCOSIS BOVIS
CAUSE:
Larva Taenia solium / Larva T. saginata
PREDILECTION&HOST:
Striated muscles, such as m.lingualis, inner parts of m.masseter,
muscles of shoulder, muscles of abdomen, diaphragma, mesenterium,
pulmo, cor, ren, eyes, and brain of pig (very sensitive parts), cattle,dog
,cat,sheep,monkey,deer, and human.
ROUTE OF TRANSMISSION :
by ingest the eggs of T. solium / T. saginata
EPIZOOTIOLOGY :
* Zoonotic characteristics
* in Indonesia it be found at: Bali, North Sumatera
(Tapanuli), Tanah Toraja and Papua
CLINICAL SYMPTOM:
Pig :
- Heavy infections : Hipersensitivity of the nostril, repeat
edly rub the nostril on to the wall/floor. Convultion the
tongue, striated muscles
- Mild infection: not so clear
~ Dog :
Similar as rabies
~ Human :
- Cerebral cysticercosis : Disturbances of balanced
Disturbances of visual
Epileptiform
- Opthalmic cysticercosis : Blindness
PATHOLOGYS OF CHANGES :
Oedema inside the predilection
Anaemia
Meningoencephalitis
Choroidae atrophy
DIAGNOSES :
⇨ Observe the clinical symptom: hipersensitivity
at the nostril of pig
⇨ ante-mortem examinations at the tongue of pig
⇨ post-mortem examinations at the site of predilec
tion
⇨ examinations by radiologies
⇨ examinations by serologic: sero-precipitation,
sero-aglutination, allergy test diagnostic = intra
dermal reaction
PREVENTION :
- to break the life cycles/ : hygienis defecations
location for human beeing
- treatments for sufferer taeniasis patients
- Beeing kept the pig intensively & hygienis
- to examinations pork and beef routinely
- Well cooked the pork and beef
- Freezing the meats which contaminated by
cysticercosis at 8 – 10°C for 4 days
- Salted meat with concentration of 20 % for 3–4
wks
- Stamping out all meats infected by cysticercosis
DIPYLLIDIASIS
Cause: Dipyllidium caninum
Predilections&Host:Small intestine dog,cat,
human (seldom)
Transmission: ingested by Cysticercoidg
mites or dog lice
Pathogenesis:
 Inside the small intestine, cause of interference
of the gastrointestinal tract (GIT).
 Heavy infections: enteritis chronics, with colic
Clinical symptoms :
* The suffering dogs walks with dragged his
anus
* They roll on the soil or bitting of the abdomen area
* Convulsion, symptom similar with epilepsy
* Child (human): anorexia, thin,abdominal discomfort,
diarrhea, epigastrium painful,
allergy reactions
Diagnose:
- CS/specifics:suffering walks with dragged his anus
- Proglottids inside the anus has a cucumber seed type
- Fecal examnations  eggs with a morphology as a
package form
Control:
Treatment:
 Arecoline hydrobromide: 0,8 mgs/kgs BW
 Bunamidine hydrochloride (Scolaban):11 mgs/kgs BW
2 x interval 48 hrs
 Dichlorophene (Dicestal):50–100 mgs/kgs BW
 Yomesan (Mansonil) : 50 –100 mgs/kgs BW
Prevention:
- By elimination the flea&dog lice with:
0,1 BHC – Asuntol (Bayer) – Super Killer
DIPHYLLOBOTHRIASIS
CAUSE: Diphyllobothrium latum
PREDILECTION&HOST: Small intestine of
dog,pig,bear, animals which eat fish&human.
ROUTE OF INFECTIONS:By eating raw fish/
uncooked fish and consist of plerocercoid.
PATHOGENESIS & CLINCAL SYMPTOM :
Adult worm inside the small intestine cause of
diarrhea and anaemia perniciosa (deficiency of
vit.B12)
DIAGNOSE :
Fecal examination  eggs
Stages: Eggs  Coracidium  Procercoid 
Plerocercoid (infective stage)
TREATMENT:
- Quinacrine hydrobromide (=mepacrine)
- Yomesan
- Arecoline hydrobromide
- Dichlorophen
PREVENTION :
by cooking fishes before consumed
TAENIASIS
CAUSE : Taenia solium & Taenia saginata
PREDILECTION & HOST:
Small intestine of human
ROUTE OF TRANSMISSION:
Ingest of Cysticercus cellulose or Cysticercus bovis
from pork / beef , because of meat processing is not
completelly accurate
EPIDEMY :
HOST
PARASITE


I.H (pig,cattle)
gravid proglottids exit (motile) from the host ⇨ rupture ⇨
free eggs
Ingest the eggs by i.h & or human ⇨ Cysticercosis
CS/ :
- non specific symptom at abdomen: diarrhea, constipation,
epigastrium painful ⇨ Taeniasis
- neurologyc symptom ⇨ Cysticercosis
AVIAN TAENIASIS
CAUSE:
~ Davainea proglotina
~ Raillietina tetragona
~ Raillietina echinobothrida
~ Raillietina cesticillus
~ Amoebotaenia sphenoides
~ Choanotaenia infundubulum
ROUTE OF TRANSMISSION :
cysticercoid ingest by definitive host
INTERMEDIATE HOST:
⇨ Davainea Proglotina : soil snail (slug) from genus
Limax, Arion, Cepoea and Agrolimax
⇨ Raillietina tetragona : Musca domestica and ant
from genus Tetramorium and Pheidole
⇨ Raillietina echinobothrida : ant : Tetramorium
caespitum and Pheidole vinelandica
⇨ Raillietina cesticillus : Musca domestica and
beetles
⇨Amoebotaenia sphenoides : Earth
worm from genus Eisenia, Pheretina
ocnerodritus, and Allobophora
⇨Choanotaenia infundubulum :
Musca domestica and beetle:
Geotripus, Aphodius, Calathus dan
Tribolium
PATHOGENICITY INFECTION OF AVIAN
CESTODES:
- Pathogenicity of each species be different
- D.proglotina : the tiny size but most pathogen
 penetration very deep at intestine mucosae
 cause of enteritis and often cause of
bleeding for heavy infection.
- R. tetragona and R. echinobothrida also pathogen after
D. proglotina. Young worms penetrated deepest into
the mucosae and sub mucosae of the duodenum 
cause of nodules. Nodules  cavum peritoneum,
consist of tissue necrotic and leucocyte. First stage :
young worm be found hang up inside the lumen of
small intestine. Mature worm be found at a part of
posterior small intestine
- Other species is not dangerous.
CLINICAL SYMPTOM:
⇨ Avian / young bird often infected
⇨ Anorexia, listless, often trifty, weakness and easy to
be fatigue, thin and anemia.
⇨ Heavy infection : cause of death at young animals
⇨ Decrease of eggs production at laying hens
⇨ D proglotina : cause of diarrhea, faeces mixed with
blood, seldom occur of nervous attacks at a part or
totally but not clear
CHANGES OF POST MORTEM:
D. proglotina :
~ Intestine mucosae tickness + bleeding
Degeneration & villies inflammation in the lumen of
intestines +
much of mucous watery & smelt putridness
R. tetragona dan R. echinobothrida :
~ Nodule at the wall of the small intestine
DIAGNOSE:
⇨ Clinical symptom&much occur segment of cestode
⇨ Fecal examination  eggs worm inside the animal
fecal
⇨ Post mortem: enteritis & nodule-2  Raillietina sp.
infections
⇨ Scrapping of intestinal mucosae  D/ D. proglotina :
deep inside the intestinal mucosae.
TREATMENT AND PREVENTION :
~ Panhelmin : mix of levamisole and praziquantel. Each
100 mls consist of 5 grs levamisole and 3,5 grs
praziquantel
Doses : 100 mls panhelmin to be solved into 100 l
water  give into drinking water for 2 days & repeat
after 10 days for 2 days.
PREVENTION:
⇨Elimination beetles/ants/grass hopper
surrounding the farm with insectiside
⇨Rearing the avian, preferable put into the
pen
⇨Elimination the snail with molluscida
⇨Cages Hygiene
⇨Give feed and drinking water at a good
position
- Thank You We hope you know it
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