The shoulder Shallow G-H jtglenoid labrum deepens capsule;also requires strong muscle force to stabilize the jointRTC (rotator cuff muscles) SITS ms. Ligaments of shoulder joint: A-C ligament-sup and inf reinforce the joint capsule and prevent post dislocation of the clavicle G-H ligaments-originate from labrum and attach to lesser tubercle and anat neck (reinforce capsule) sup, mid and inf bands Coracoclavicular lig.- lat(trapezoid) and med(conoid) Both prevent backward mvmt of the scapula and ind they limit scap rotation Acromioclavicular Joint A-C joint capsule Coracoclavicular ligaments Clavicular Ligaments A-C Joint Conoid ligament Common Glenohumeral Problems Rotator cuff tendinitis Rotator cuff tears Bicipital tendinitis, rupture Glenohumeral dislocation/subluxation Labral Tears Frozen shoulder syndrome Arthritis Rotator Cuff Problems Rotator Cuff Impingement Rotator Cuff Tear (RCT) Phase 1 (0 to 6 weeks) Phase 2 (6 to 12 weeks) • Passive range of motion exercises only for almost all tears. • Active-assisted range of motion for very small tears or repairs with exceptionally good tissue • Full passive motion • Begin active-assisted motion • Strengthen intact cuff muscles • Begin to strengthen the muscles that stabilize the shoulder blade Phase 3 (12 to 16 weeks) Phase 4 IV (> 16 weeks) •Passive stretching beyond the patient's own range of motion •Strengthening the repaired cuff muscles •More strengthening of the stabilizers of the shoulder blade •Functional strengthening •Rehabilitation for sports Normal Cuff, Torn Supraspinatus on MRI Bicipital Tendinitis Impingement Shear in bicipital groove Long biceps tendon in intimate with joint capsule. May be impinged beneath acromion, or sheared within bicipital groove. Bony Structures Avascular Necrosis of Humeral Head May be seen with chronic corticosteroid use. (GENTLY handle patients with history of steroid use.) Can lead to total shoulder replacement. Glenohumeral Arthritis Glenohumeral Arthritis Frozen Shoulder Syndrome “Freezing” shoulder “Frozen” shoulder “Thawing” shoulder Freezing Shoulder “Freezing” shoulder Usually starts with inflammatory process, such as impingement syndrome. Subscapularis trigger points limit external rotation, abduction Shoulder becomes painful, then stiff Best opportunity for intervention is here! Frozen Shoulder Capsule undergoes fibrotic changes (“Adhesive capsulitis”) PT intervention alone is of questionable help. May benefit from manipulation under anesthesia, followed by PT care. Thawing Shoulder Shoulder spontaneously becomes less painful, less stiff. If in rehab, take credit for result, but probably little effect from treatment. Nearly all frozen shoulders spontaneously resolve in 6 to 18 months May recur on opposite side Rare in African-Americans Glenohumeral/Scapulothor acic Rhythm Occur in 2:1 ratio GH/ST, but not in constant ratio. GH joint moves first, with stabilized scapula Then, move in 1:1 ratio. Then finish with mostly GH motion FINAL ratio is 2:1 Glenohumeral Dislocation Usually caused by violent abduction/external rotation of humerus. Humerus dislocates in anterior, inferior direction. Causes disruption of anterior labrum (Bankart lesion) If repeated, posterior aspect of humerus strikes labrum, producing indentation in humerus (Hill Sachs lesion.) Superior Labral Tear Anterior and Posterior to Biceps Attachment (SLAP) Biceps tendon Posterior tear Anterior tear Bicipital Tear (Longhead) Scapulothoracic Problems Winging scapula from poor posture, habit. Common in tall, early developing females, swimmers Correlated with G-H problems May be from long thoracic nerve palsy, taking out serratus anterior. Results in inability to raise arm above 120 degrees (ever.) Serratus Anterior Loss Winging 120 degrees abduction Suprascapular Nerve Palsy Suprascaular nerve innervates supra- and infraspinatus. Injury results in selected weakness. What’s the sensory pattern?? Coracoacromial lig- provides roof over the humeral head - acts as a protective arch Scapular movements must be accompanied by shoulder joint movements therefore if you have impairment at G-H joint, must look at scapula Kinematics of shoulder jointscapulohumeral rythym external rotation with abduction scapular plane Muscles-RTC(rotator cuff muscles) SITS supraspinatus-imp to keep head of humerus in glenoid fossa along with other ms. Infra, teres minor, subscap-act to depress head during flexion and abduction-counteract strong deltoid long head of biceps becomes very active in shld flex and abd past 90 Ms. named from areas they originate and insert-grouping as follows: Scapulohumeral:deltoid, supraspinatus, infraspinatus, teres minor, subscapularis, teres major, coracobrachialis Axioscapular:pect minor, trapezius, rhomboids, lev scap, serr ant Axiohumeral: pect major, lat dorsi Deltoid-ant, mid and post portion Origin: ant portion-lateral 1/3rd of clavicle mid-acromion, post-spine of scapula Insertion-deltoid tuberosity of humerus and med rotate, post fibers extend and laterally rotate innervation-axillary (C5,6) supraspinatus: origin-supraspinatus fossa of scapula insertion-greater tubercle of humerus action- stabilizes head of humerus in capsule, assists in abduction-acts as force couple with deltoid to assist with abd innervation-suprascapular (C4,5,6) Infraspinatus-origin-infra fossa insertion-greater tubercle and shld capsule innervaton-suprascap nerve action-ext rotation of shoulder and depression of humeral head and stabilizes head during movement Teres minor-origin-upper lateral border of scapula insertion-greater tub and shoulder capsule action-lat rotation and add of humerus Subscapularis-origin-subscapular fossa insertion-lesser tubercle of humerus and capsule action-int rotation of humerus and works with other ms. Innervation-subscapular (C5-7) Teres major-origin-acillary border of inf angle of scap insertion-med tip of inter groove action-med rotation, adduction and shouler ext Innervation-lower subscapular(C5-7) Axioscapular-pect minor: origin-ribs 3,4,5 and fascia of intercostal ms insert-coracoid process action-elevation and downward rot of scap innervation-medial pect (C8-T1) trapezius-origin-upper from occ protuberance, nuchal line and spinous porcess of C7, middle from spinous process T1-5 and lower from T6-12 insertion- upper from lat clav and Rhomboid major-origin-spinous process T2-5 insertion-vertebral border action-down rotation, elevation and adduction of scap innervation-dorsal scapular (C4-5) rhomboid minor-origin-spinous processes C7-T1 insert-root of spine of scap action-same as major inn-same as major Levator scapula-origin-transverse processes C1-4 insertion-sup med border of scap action- elevation, down rotation and add of scap innervation-dorsal scapular Serratus anterior-origin-upper 8-9 ribs ant surface insertion -medial, inf surf of scap action-up rot, elevation and abduction inn-long thoracic (C5-7) AxiohumeralPectoralis major-origin:clavicle, sternum and cartilage of first 6-7 ribs insert-lat inter. Groove action: med rotation, flexion and horizontal adduction Latissimus dorsi-origin-sp processes of T6-12, last 3 ribs, thoracolumbar fascia and iliac crest insert-inter groove action-med rotation, adduction and ext of shld, ext of L spine, flex of T spine Disorders of PNSneuropraxia-local blockage interfering with conduction , it’s OK above and below-commonly caused by compression-Saturday night palsy-radial nerve or Bell’s palsy, no disruption of axon Axonotmesis-nerve injury characterized by disruption of the axon and myelin sheath but with preservation of supporting CT resulting in axonal degeneration distal to the injury site-the deficit depends on the # of axons neurotmesis- partial or complete severance of a nerve with disruption of axons, myelin sheaths and supporting connective tissue resulting in degeneration of axons distal to the injury site (worst of the 3) Disorders of PNS Erb’s palsy-compression or stretching of upper BP nerve roots (C5,6)-results in “waiter’s tip” sign Klumpke’s paralysis-compression or stretching of lower BP (C8,T1)-results in functionless hand Bursae-fluid filled sac which can be inflammed-bursitis-most common in shoulder-subdeltoid and subacromialleast likely subscapular bursitis Signs-warm, edematous with tenderness over area Pain quality-intense, dull, throbbing all movements painful Tendonitis-inflammation of the tendon RTC tendonitis-supraspinatus most involved-results from overuse, tennis, baseball, carpenters, plumbers-can also be poor blood supply causing scarring or Ca deposits-can bring about tears, bursitis or impingements; local steroids can relieve symptoms but may cause structural wknss of tendon Pain quality-sharp twinges ie. Donning jacket, reaching OH, abd or IR arm Onset-gradual. May sometimes refer to C5-6 dermatome RTC tears-acute, chronic, full, partial thickness tears;<1cm. Small, >5cm. Massive-usually traumatic but may be degenerative pain-not always severe but pt con’t raise arm and has severs atrophy lat and ant deltoid region-may require surgery Adhesive capsulitis-frozen shld.-trauma, disuse, immobilization, RTC lesions pain-dull-severe with activity, pain at Onset-gradual, will see increase activity of upper traps Impingement syndrome-supra, long head biceps, subacro bursa most affected-pt. will exhibit painful arc of motion b/w 70-120 degrees 3 stages: I-edema-athlete or poor posture, young person with no recollection of injury II-fiborsis and tendinitis (20-40 yo)recurrent pain with activity III-bone spurs and tendon ruptureslong history (50-60yo) G-H instability-hum head dislocates through ant capsule, RTC ms. Can be weak Brachial plexus lesions-numbness and burning entire arm, hand, fingers, sensory loss over 2 or more dermatomes, paralysis of arm, may be transcient tenderness over BP with increased symptoms with movement of head to opposite side Thoracic outlet syndrome-often called neurovascular compression-symptoms resulting from injury at upper border of thorax where BP and subclavian a are located-can be caused from a C-rib treatment-postural correction ex to bring back shoulders Brach plex lesions-numbness and burning entire arm, hand, fingers-sensory los over 2 or more derm-paralysis of arm-may be transcient-tenderness over BP with increase symptoms when turning head opp. side Diagnostic testsX-ray-for bony defects, alignment, exostosis (bone spurs), osteophytes and diseases C-T scans-specific for bone MRI-magnetic resonance imaging-soft tissue-no radiation as in X-ray angiography-contrast mat injected into vascular system myelograpy-inject dye into SA space EEG-records brain electrical activity EMG and NVC-see if diseases are neuromuscular in origin arthrogram-injects dye and air-views jt space, cartilage, ligs