Psychotherapy: Does it Work? Why Does it Work? Bruce E. Wampold, Ph.D., ABPP Department of Counseling Psychology Department of Psychiatry University of Wisconsin-- Madison & Research Institute Modum Bad Psychiatric Center Vikersund NORWAY Does it work? Psychotherapy v. No-tx Eysenck, science, and behaviorism Evidence from RCTs: ◦ Smith and Glass (1977) ◦ Effect size: ◦ g = (mean Tx - mean Control)/SD es = .80 Accounts for 13% of variance in outcomes Average treated person does better than 80% of untreated persons Psychotherapy works NNT = 3 – three patients need to be treated to obtain one additional success Aspirin as a prophylaxis for heart attacks (NNT = 129) Superior to interventions in cardiology, geriatric medicine, asthma, flu vaccine, cataract surgery Comparable to psychopharmacology interventions Enduring and safe Effects in practice comparable to benchmarks created by RCTs Elite club: Medicine and psychotherapy Effect sizes d r % variance nnt Description .2 .10 1.0% 9 small .3 .15 2.2% 6 .4 .20 3.8% 5 .5 .25 5.9% 4 .6 .29 8.3% 4 .7 .33 10.9% 3 .8 .37 13.8% 3 Tx v. No Tx Medium Large How does it work? Treatment Common factors Interaction of specific and common factors– the contextual model Specific Treatment Effects Psychological treatments = built on characteristics found in a variety of treatments, including “the therapeutic alliance, the induction of positive expectancy of change, and remoralization,” but contain important “specific psychological procedures targeted at the psychopathology at hand” (Barlow, 2004, p. 873). Empirically Supported Treatments ◦ Evidence based treatments ◦ 2 trials, > control or = EST, manual, 2 different groups Inference: Specified treatment differences will exist Treatment Differences Treatment intended to be therapeutic ◦ Psychological rationale, trained therapists who have allegiance to tx, no proscription of usual therapeutic actions Null Hypothesis: ◦ All treatment intended to be therapeutic are equally effective Wampold et al. (1997) All direct comparisons across disorders Effects homogeneously distributed about zero– No evidence to reject the null hypothesis Upper bound ◦ ◦ ◦ ◦ d = .2 % variance < 1% NNT = 9 SMALL Effect sizes d r % variance nnt Description .2 .10 1.0% 9 small .3 .15 2.2% 6 .4 .20 3.8% 5 .5 .25 5.9% 4 .6 .29 8.3% 4 .7 .33 10.9% 3 .8 .37 13.8% 3 Tx A v. Tx B Medium Large Depression (see http://www.div12.org/PsychologicalTreatments ESTs: behavioral activation, cognitive therapy, interpersonal therapy, brief dynamic therapy, reminiscence therapy, self-control therapy, social problem solving therapy, self-system therapy, acceptance and commitment therapy, behavioral couple therapy, self/management self-control therapy… and The case of process-experiential therapy Behavioral/cognitive behavioral not superior to verbal therapies intended to be therapeutic Dynamic therapies produce effect sizes comparable to CBT Does CBT work through specific ingredients? CT for Depression (Jacobson et al. 1996) The purpose of this study was to “provide an experimental test of the theory of change put forth by A. T. Beck, A. J. Rush, B. F. Shaw, and G. Emery (1979) to explain the efficacy of cognitive-behavioral therapy (CT) for depression” (p. 295). Complete Cognitive Therapy (CT) ◦ Behavioral activation (monitoring, activity assignment, social skills training) ◦ Dysfunctional thoughts (Monitoring, assessment, reality testing, alternative cognitions, examination of attributional biases, homework) ◦ Core Schema (Identify core beliefs and alternatives, advantages and disadvantages, modification of core beliefs) Activation + modification of dysfunctional thoughts (AT) Behavioral Activation (BA) CT v. AT v. BA Jacobson results “According to the cognitive theory of depression, CT should work significantly better than AT, which in turn, should work significantly better than BA.” BA = AT = CT “These findings run contrary to hypotheses generated by the cognitive model of depression put forth by Beck and his associates (1979), who proposed that direct efforts aimed at modifying negative schema are necessary to maximize treatment outcome and prevent relapse.” Depression placebo responsive… “real disorders” PTSD PE, Stress Inoculation Training v. Supportive Counseling (Foa et al.) PE, SIT scientifically designed treatments PE, SIT > Supportive Counseling Conclusion: ◦ Exposure, cognitive change needed. Supportive Counseling “Patients were taught a general problem-solving technique. Therapists played an indirect and unconditionally supportive role. Homework consisted of the patient’s keeping a diary of daily problems and her attempts at problem solving. Patients were immediately redirected to focus on current daily problems if discussions of the assault occurred.” Belief of therapists delivering Supportive Counseling? But examine another study… PTSD in Adult Female Childhood Sexual Abuse (Completer Sample) Measure Tx A Tx B ES % not ptsd (3 month follow up) 47.1% 82.4% 35.0% 42.1 Clinician PTSD 38.5 47.2 .34 BDI 7.5 10.4 .31 Spielberger TAI 39.4 45.6 .53 TSI Beliefs 2.2 2.4 .39 Dissoc. experiences 7.6 9.4 .24 Cook Hostility 12.9 14.9 .27 Qual of Life 47.1 38.9 .58 PTSD in Adult Female Childhood Sexual Abuse (Intent to treat) Measure % not ptsd Tx A 27.6% Tx B 31.8% Effect size Clinician PTSD BDI Spielberger TAI 53.1 12.9 46.2 47.2 10.8 46.4 -.22 -.18 .02 TSI Beliefs Dissoc. experiences Cook Hostility 2.7 12.4 21.6 2.4 11.5 17.1 -.41 -.09 -.54 Qual of Life 39.5 39.0 .03 PTSD Dropout Rate Tx A Tx B Enrolled 29 22 Completed 17 20 Dropped out 12 2 % dropped out WL chose tx 41% 5/10 dropped 9% 0/9 dropped PTSD “As expected, our hypothesis that Tx A would be more effective than WL received consistent support. There was no effect of either tx on quality of life. Our hypothesis that Tx A would be superior to Tx B received support (at follow-up only). In summary, for women who remained in Tx A, it was highly effective.” PTSD Tx A = CBT, prolonged imaginal exposure, in vivo exposure, cognitive restructuring, breathing retraining ◦ Psychologist therapist, Foa supervisor ◦ Cogent rationale Tx B = PCT (Present-centered treatment) ◦ Rationale: impact of trauma on current functioning, systematic approach to problem solving, manual. ◦ MSW therapists, trained by authors ◦ No cognitive or behavioral components (no exposure) Quality of Life? McDonagh et al. 2005 Present Centered Therapy RCT 1: PCGT v TFGT Scnurr et al. 2003 Vietnam Vets PCGT TFGT No Difference Drop CAPS Total Rate*** severity RCT 2: PCT v CBT McDonagh et al. 2005 Childhood Sexual Abuse PCGT TFGT No Difference Drop CAPS Rate * RCT 3: PFGT v TFGT Classen 2011 Childhood Sexual Abuse/HIV Risk PCGT TFGT No Difference Total HIV risk * Anger/ Irratability** PTSD Severity CAPS B BDI CAPS C PTSD Checklist General Health Q SF-36 Physical SF Mental STAI DES Sexual revictimization Substance Use Risky Sex COOK # of partners STAXI Avoidance QOLI Reexperiencing Interpersonal Problems Depression Dissociation Sexual Concerns Dysfunction Sex Impaired Selfreferences Tension Reduction Posstraumatic Growth Drop Rate Present Centered Therapy 3 Trials Comparable (or better) than Evidencebased Treatment > 2 Research groups Manualized Meets standards for evidence-based treatment (Frost et al., submitted) Consider EMDR ◦ Pseudo science, Mesmerism Resick et al. 2008 PTSD Cognitive Processing Therapy Cognitive therapy only Written Accounts 2hr/wk, 6 weeks (writing 45-60 min) All 3 treatments showed improvement Post Traumatic Diagnostic Scale PTSD Prolonged exposure, CBT, EMDR, hypnotherapy, psychodynamic, trauma desensitization, presentcentered therapy, CBT without exposure No differences among treatments intended to be therapeutic (Benish, Imel, & Wampold, 2008) Other diagnoses ◦ Panic: Panic Control Tx, Psychodynamic (Mildrod et al., 2007) ◦ Alcohol Use Disorders Meta-analysis of all tx, including CBT, MI, AA, etc. No differences (Imel et al., 2008) Children Depression and Anxiety ◦ CBT = non-CBT (when intended to be therapeutic) Spielmans, Pasek, & McFall, 2007 Depression, anxiety, conduct disorder, ADHD ◦ Small differences ◦ Entirely explained by allegiance of researcher Miller, Wampold, & Varhely, 2008 Meta-analysis of studies comparing 2 treatments Meta-analysis of studies comparing 2 treatments 9 comparisons Overall effect not significant Only 1 of 9 statistically significant ◦ Markowitz: HIV Depressed men, IPT > CBT NIMH funded 1992-2009 $11,760,874 (78,848,306 SEK) Value? If not treatment, then…. Common Factors Alliance Bond (i.e., relationship) Agreement on Goals Agreement on Tasks Alliance and outcome correlation Horvath et al. (2011) reviewed 190 studies, > 14,000 patients Correlation of alliance at early session and outcome r = .27 d = .57 > MEDIUM Effect sizes-- Alliance d r % variance nnt Description .2 .10 1.0% 9 small .3 .15 2.2% 6 .4 .20 3.8% 5 .5 .25 5.9% 4 .6 .29 8.3% 4 .7 .33 10.9% 3 .8 .37 13.8% 3 Alliance Medium Large Alliance and outcome correlation Horvath et al. (2011) reviewed 190 studies, > 14,000 patients Correlation of alliance at early session and outcome r = .27 d = .57 > MEDIUM Not confounded by improvement (Klein et al. 2003; Crits-Christoph et al. 2011) Other factors (Flückiger et al., 2012) ◦ CBT v non CBT ◦ Manual driven or not/Specific treatment ◦ Allegiance to alliance Therapist or patient contribution? Psychotherapy Relationships that Work: Norcross Relationships that Work (2011) Factor # Studies # Patients Effect size d Alliance 190 > 14,000 .57 Alliance-Child & Adolescents 29 2630 .39 Alliance-Couple & Family 24 1461 .54 Empathy 59 3599 .63 Goal Consensus, Collaboration 15 1302 .72 Positive regard, affirmation 18 1067 .56 Congruence, genuineness 16 863 .49 Common Factors—Specific Factors Factor # Studies # Patients Effect size d Alliance 190 > 14,000 .57 Alliance-Child & Adolescents 29 2630 .39 Alliance-Couple & Family 24 1461 .54 Empathy 59 3599 .63 Goal Consensus, Collaboration 15 1302 .72 Positive regard, affirmation 18 1067 .56 Congruence, genuineness 16 863 .49 Adherence to specific protocol 28 .04 Rated competence 18 .14 Webb, DeRubeis, & Barber, 2010 NOT SIGNIFICANT Correlations v. RCTs Correlation does imply causation Issues with RCTs ◦ ◦ ◦ ◦ ◦ ◦ Selection and Generalizability Blinding Distinguishability Active ingredients Therapist effects Outcome measures Therapist Effects Definition: Some therapists consistently attain better outcomes than other therapists Not due to contribution of patients Not due to chance Generalizable to the population of therapists Compare to effects for other factors (e.g., treatment differences) Therapist Effects– The Evidence Clinical Trials ◦ Selected, trained, supervised and monitored ◦ 8% of variability due to therapists ◦ Tx differences: At most 1 percent Naturalistic settings ◦ ◦ ◦ ◦ 3% to 17% due to therapists Across age, severity, & diagnosis Possibly not across racial and ethnic groups Cross validated NIMH TDCRP reanalysis Nested Design (CBT and IPT) Well trained therapists, adherence monitored, supervision Elkin: ◦ The treatment conditions being compared in this study are, in actuality, “packages” of particular therapeutic approaches and the therapists who choose to and are chosen to administer them…. The central question… is whether the outcome findings for each of the treatments, and especially for differences between them, might be attributable to the particular therapists participating in the study. $6,000,000 (40,198,715.15 SEK) Random Effects Modeling Therapists considered a random factor Therapists nested within treatments (multilevel model) Final observations, controlling for pretest at patient and therapist level ◦ Kim, Wampold, & Bolt, Psychotherapy Research, 2006 Random Effects Modeling Therapists considered a random factor Therapists nested within treatments (multilevel model) Final observations, controlling for pretest at patient and therapist level Therapist slope fixed and random ◦ Kim, Wampold, & Bolt, Psychotherapy Research, 2006 Greater Severity Greater Severity Variance due to Tx: CBT v IPT Variable Treatment BDI 0% HRSD 0% HSCL-90 0% GAS 0% Therapist Variance due to Tx and Therapists Variable Treatment Therapist BDI 0% 5% - 12% HRSD 0% 7% - 12% HSCL-90 0% 4% - 10% GAS 0% 8% - 10% Note: Elkin et al. (2006) found negligible therapist effects in the same data Psychiatrist Effects– Psychopharmacology Antidepressants: Imipramine v. Placebo 30 minutes, biweekly 3% due to treatment 9% due to therapist Best psychiatrists got better outcome with placebo than worst psychiatrists with imipramine (McKay, Imel & Wamold, 2006) Effect sizes– Therapists Effects d r % variance nnt Description .2 .10 1.0% 9 small .3 .15 2.2% 6 .4 .20 3.8% 5 .5 .25 5.9% 4 .6 .29 8.3% 4 .7 .33 10.9% 3 .8 .37 13.8% 3 Therapists Effects Medium Large Therapists make a difference Characteristics and Actions of Effective Therapists? Consult Beutler (Handbook of Psychotherapy and Behavior Change) ◦ We don’t know ◦ And we don’t care ◦ Education, agriculture, medicine…. And psychotherapy Fundamental unanswered question Beginning to accumulate evidence Btw: therapist effects inflates treatment differences Alliance: Patient v. Therapist Contribution to Alliance Counseling center consortium data OQ pre and post, Alliance 4th session 331 patients, 80 therapists Alliance/outcome correlation .24 3% of variance due to therapists What is correlation of alliance with outcome ◦ Within therapists? ◦ Between therapists? And the results…. Within or between? Better therapist Therapist contribution to alliance is critical Patient contribution to alliance not predictive of outcome Therapist contribution is predictive of outcome Interaction not significant Alliance is not a result of outcome Interpersonal skills Verbal fluency, interpersonal perception, affective modulation and expressiveness, warmth and acceptance, empathy, focus on others Measured with a challenge test ◦ Responses to vignettes Accounts for therapist differences ◦ Anderson, Ogles, Patterson, Lambert, & Vermeersch, D. A. (2009) ◦ Supported in meta-analyses (see Norcross, Psychotherapy Relationships that Work) Conclusions Treatment ◦ Particular treatment not important ◦ Treatment IS important Who delivers the treatment is primary ◦ Therapist who can form alliances with patients ◦ Interpersonal skills AN EVIDENCED-BASED MODEL OF PSYCHOTHERAPY Relationship Elements Real relationship, belongingness, social connection Therapist Trust, Understanding, Expertise Better Quality of Life Creation of expectation through explanation and some form of treatment Patient Symptom Reduction Tasks/Goals Therapeutic Actions Healthy Actions Initial formation of therapeutic bond Humans evolved to discriminate between those who can be trusted and those who cannot 50 ms Context, healing practice Nonverbal Therapist Trust, Understanding, Expertise Patient Real Relationship Transference-free genuine relationship based on realistic perceptions (Gelso, 2009) Social relations = well being Social isolation = pathology Psychotherapy is uniquely ENDURING Real relationship, belongingness, social connection Trust, Understanding, Expertise Better Quality of Life Expectation Expectation influence on well being Placebo effects Created in interpersonal interaction Explanation of disorder Agreement about tasks and goals of Tx Treatment actions Trust, Understanding, Expertise Better Quality of Life Creation of expectation through explanation and some form of treatment Symptom Reduction Specific Actions Indirect Effect Agreement tasks & goals adherence to protocol Healthy actions Need to develop and test protocols Better Quality of Life Trust, Understanding, Expertise Symptom Reduction Tasks/Goals Therapeutic Actions Healthy Actions Conclusions Relationship factors critical ◦ Real relationship ◦ Explanation expectations ◦ Agreement about tasks and goals healthy actions Human evolved to heal through social means Treatment important, but is the particular treatment? IMPROVE QUALITY OF CARE Disseminate Evidence-based Treatments Measure and manage outcomes ◦ Use best therapists ◦ Help poorer therapists improve Provide therapists feedback Provide training ◦ Common fctors ◦ Specific treatments Thank You Bruce E. Wampold, Ph.D., ABPP Patricia L. Wolleat Professor of Counseling Psychology Clinical Professor, Psychiatry University of Wisconsin--Madison Director, Research Institute Modum Bad Psychiatric Center Vikersund, Norway bwampold@wisc.edu