Nurture and Personality Development Jada Milan Yorkville University PSYC 6123 Theories of Personality (20W-3O) Dr. Melissa Moore September 23, 2022 Introduction Nature and Nurture were originally pitted against each other by psychologist Francis Galton, the cousin of Charles Darwin (Cherry, 2019). Aristotle is also known for questioning the origin of traits and behaviours, however he believed that intelligence was gained through experience (McCartney & Tomcho, 2020). In contrast, Plato suggested knowledge was innate (McCartney & Tomcho, 2020). Behaviour and personality derived from genetic inheritance and biological factors is described as nature, while external factors such as, experience, observation and learning, are considered nurture (Zaky, 2015). Biological psychology focuses on the influence of genetics, while behaviourism focuses on the influence of one’s surrounding environment on behaviour (Zaky, 2015). Nativists take a stance that most of our personality and behaviour is the result of inheritance (Zaky, 2015). While at the other end, environmentalists or empiricists assume that at birth one is a blank slate and personality is a result of experience and learning (Zaky, 2015). Many psychologists today, understand that there is a complex relationship between nature and nurture. Epigenetics are radically changing the way we think about personality development. McCartney and Tomcho (2020) describe epigenetics as the following: Epigenetics involves an array of chemical markers and switches that play a role in genetic activity, collectively called an individual’s epigenome. These markers and switches are modified due to experiences (e.g., diet, behaviors, and surroundings), and the edited epigenome can be passed down to future generations. (p.3) Environmental changes do not change DNA; however, they do influence the epigenetic markers that inhibit or enable certain traits. McCartney and Tomcho (2020) suggest that “epigenetics is nature and nurture” (p.3). According to Smoller (2016) “Genes and stress are the two most widely acknowledged contributors to psychopathology” (p.297). Stressful environments have been connected to anxiety, depression, and traumatic stress disorders. Stress and trauma during childhood are risk factors for anxiety disorders (AD) (Smoller, 2016). Bandelow and Michaelis (2015) explain that most AD start in childhood, peak during middle age and decrease with older age (p.331). Nature vs. Nature is no longer a debate, given the advancements in the study of genetics as well as psychology. AD are a result of both biological and environmental factors. This paper will examine the role in which both these factors contribute to AD and argue that nurture is significantly more influential. Review of the nature argument Twin studies have been the primary source of research conducted to determine whether genetics or environment influence personality and behaviour. Identical twins, monozygotic (MZ) typically share %100 of their DNA. If a trait is to be considered heritable then both twins should exhibit the trait equally (Barlow, 2019). Non-identical, dizygotic (DZ) twins share %50 of their DNA, on average. In order to determine whether a trait is heritable or environmental, results of MZ pairs are compared to that of DZ pairs (Shimada‐ Sugimoto, Otowa, & Hettema, 2015). If the similarity of MZ pairs is greater than that of the DZ pairs, this is evidence for genetic influence (Shimada-Sugimoto, et.al, 2015). Studies have found that AD are %30 - %50 heritable (Rapee 2012). “A study of over 20,000 people in the Danish population with a psychiatric record indicated an individual with an anxiety disorder was 6.8 times more likely to have a first-degree relative with another disorder” (Rapee, 2012, p.69). When looking at immediate families, a child with one parent who has an AD was rated 2, whereas if both parents has anxiety, the risk increased to 5 (Rapee, 2012, p.70). This suggests that there is a strong correlation between genetics and AD. When one or both parents have anxiety, children are at a higher risk of developing AD. Nebel-Schwalm and Davis (2013) speculate that environmental influences are responsible for the specific disorder that develops in people who have a general genetic predisposition to mental health disorders. Behavioural genetics suggests that temperament is genetically influenced (Kamran, 2016). “Behavioral geneticists claim that shared family environment has little if any effect on most psychological traits; family resemblances are almost all attributable to shared genes rather than shared environment” (Zaky, 2015, p.2). Conduct issues are the one exception to this assumption (Zaky, 2015). If this is true, then siblings should exhibit similarities in temperament. However, it’s difficult to determine the specific genetic or environmental factors that contribute to personality. hanging in response to the environment” (p.69). Behavioural epigenetics is the study of environmental experiences that trigger biological changes that modify genetic markers (Powledge, 2011). Researchers believe that early childhood experience shape adult behaviour (Powledge, 2011). Through rat studies, they have found that early experiences influence gene changes and disease susceptibility (Powledge, 2011, p.590). These studies suggest that experience contributes to AD through genetic changes. Zaky (2015) suggests that “genetic predisposition and appropriate environmental triggers are required for a mental disorder to develop” (p.1). Barlow (2019) explains that the nature vs. nurture debate is over, “we are each bundles of genes, reacting and changing in response to the environment” (p.69). Nature and nurture are not in opposition, they are intertwined, and both play a role in the etiology of AD. Review of the nurture argument Psychologists such as Freud, speculated that parents are the most critical in shaping children’s personality and behaviour (Barlow, 2019). Freud suggested that worry ensues when a child’s needs are not met, and the pattern of needs not being met causes anxiety (Schultz & Schultz, 2017). Freud theorized that one is constantly in search of balance, and tension is experience when the body is in a state of need (Schultz & Schultz, 2017). This was called ‘instinct’, and he believed it to be an innate characteristic of all human beings. Children are dependent on their parents to satisfy their needs and reduce tension. If their needs are consistently neglected the development of an AD may occur. Alfred Adler theorized that all behaviour is motivated by feelings of inferiority (Schultz & Schultz, 2017). He suggested that neglecting or spoiling children results in an inferiority complex. Children who are spoiled may not know how to manage a challenge or conflict later in life, which results in feelings of inferiority. Whereas children who are neglected may experience feelings of worthlessness which also yields an inferiority complex (Schultz & Schultz, 2017). Adler found that these types of people were unable to cope with life and were most likely to develop mental health issues. He also believed that the mother was the most influential in shaping children's personality. He described social interest as one’s innate ability to socialize with others, and mothers could foster or neglect it (Schultz & Schultz, 2017). “Research using the SIS found that those high in social interest reported less stress, depression, anxiety, and hostility than those low in social interest” (Schultz & Schultz, 2017, p.126). Mothers who nurture social interest will have children with high social interest that are less susceptible to AD. Many psychologists have researched the influence of childhood experiences, however Barlow (2019) suggests that how we remember our childhood also shapes us. Childhood experiences are subjective, and this helps explains why twins exhibit different characteristics later in life. “Different people interpret and respond to the same stimuli in different ways” (Barlow, 2019, p.71). Why would twins respond differently to the same stimuli? Their DNA may be identical; however, their environment may have had variations. For example, parents may treat children differently, or one child may have experienced a traumatic event. These experiences could influence one's genetic markers, which is an example of epigenetics (Barlow, 2019). “Multiple other studies confirm that the same family environments are either objectively different or experienced differently by people in a way that is reliably accounted for (in part) by genes” (Barlow, 2019, p.72). When exposed to the same stimuli, their response may vary based on unique past experiences and their differing genetic markers. Parenting styles are also considered important in determining personality. Adler theorized birth-order and its impact on personality. The relationship between parents and children as well as between siblings has been researched extensively. Only children are more likely to be pampered by their parents, as they are the center of attention (Schultz & Schultz, 2017). First-borns are typically high achievers with attention to detail and are adaptive to change. Adler noted that parenting style changes over time. With the first born, everything is new, and parents may be more neurotic (Schultz & Schultz, 2017). For example, mothers may stress more easily since they are navigating new territory. A second child is more work; however, parents may feel more relaxed having experienced the first child (Schultz & Schultz, 2017). Even though the environment is the same for each child, most siblings have varying personalities. Birth order and the evolution of parenting styles is the reason behind this. Clear stance with support for the chosen side, /25 Nature and nurture may be intertwined; however, nurture is more significant because it can alter nature. As previously discussed, the environment and life experiences can alter genetic markers. One’s life experiences are constantly shaping personality and determine the likelihood of developing a mental illness. Anxiety is a “chronic fear that persists in the absence of any direct threat – common psychological correlate of stress” (Pinel et al., 2017, p.489). Anxiety is a human response that evolved for protective means, however, persistent anxiety without a present threat, can be debilitating (Nebel-Schwalm & Davis, 2013). AD is the most common of all psychiatric disorders with estimates of 14-34 percent of people suffering from an anxiety disorder in their life (Pinel et al., 2017). According to Smoller (2016) “The role of stressful environments and the physiology of stress response systems have been most closely linked to depressive, anxiety, and traumatic stress disorders” (p.297). Parents are the first people who influence a child’s personality development. Children observe and learn from their parent’s behaviour (Schultz & Schultz, 2017). An example of this can be seen in Bandura's Bobo doll experiment. Bandura’s social learning theory states that “aggression is learnt from the environment through observation and imitation” (Zaky, 2015, p.3). Kamran (2016) suggests that peer modeling, culture, media and technology also influence a child’s personality and behaviours (p.107). How one responds to their environment, can determine vulnerability for AD. Bourgeois and Brown (2015) explain that expressing negative emotions in response to stress, is a sign of vulnerability for emotional disorders (p.531). Bourgeois and Brown (2015) discuss how emotional vulnerability arises: General psychological vulnerability that is the product of stressful childhood experiences, such as harsh or unpredictable early environments (e.g., authoritarian or inconsistent parenting styles). This general psychological vulnerability may manifest as a sense of unpredictability or lack of control over life events and emotions. In the context of situational stressors, individuals with both general vulnerabilities are more likely to develop emotional disorders. (p.532) Parenting style contributes to psychological vulnerability. Rapee (2012) suggests that parental overprotection and control is associated with AD. They explain that overprotection can lead to anxiety later in life, while childhood anxiety can lead to parental overprotection (Rapee 2012). Perhaps this is why AD are observed crossgenerationally in family units. Bourgeois and Brown (2015) also explain that “disorder-specific psychological vulnerability develops through learning experiences that emphasize the potential danger of certain situations, objects, or internal experiences (e.g., thoughts, memories, physical sensations)” (p.532). Children learn that certain stimuli are dangerous, such as fire or busy streets. However, parents may pass on fears that do not have a logical cause for fear, such as spiders. Nebel-Schwalm and Davis (2013) explain that theories attempting to explain the development of AD often focus on four pathways: direct learning, indirect learning, biological preparedness pathway, and a non-associative pathway. Direct learning, also known as classical conditioning, refers to fear that is associated with a stimulus (Nebel-Schwalm & Davis, 2013). For example, someone is bitten by a spider, then avoids spiders to avoid the fear they invoke, and a phobia develops. Observational learning is when a fear is learned through the observation of others (Nebel-Schwalm & Davis, 2013). For example, a child witnesses their mother scream at the sight of a mouse. Exposure to a mouse in the future, elicits the same fear response observed in their mother. Negative information transfer is “the idea that one can learn to be anxious by hearing others talking negatively (or anxiously) about a subject” (Nebel-Schwalm & Davis, 2013, p.5 ). This can be observed in overly cautious parenting for example, when a parent makes a big fuss when their child falls and scrapes. Biological preparedness refers to stimuli that risk our lives, such as heights. It has been suggested that biological preparedness explains why certain phobias form more easily than others (Nebel-Schwalm & Davis, 2013). An example of this can be seen when observing how common the fear of spiders is, versus the fear of nonthreatening stimuli such as dolls. Nebel-Schwalm and Davis (2013) explain that biological preparedness theory presumes that a conditioning event has occurred (p.6). Finally, non-associative pathways occur when a direct event cannot be determined as the cause of one’s fear and anxiety (Nebel-Schwalm & Davis, 2013). “For example, researchers studying water phobia found that most people did not know how the fear started” (Nebel-Schwalm & Davis, 2013, p.6).These four factors are dependent on nurturement and can be used to explain the etiology of most AD. Parents have the greatest influence in the development of personality. Parenting style is detrimental to the resilience of a child later in life. Parents who overprotect are preventing their children from developing coping mechanisms which results in children who have a higher chance of experiencing anxiety later in life (Rapee, 2012). In contrast, parents who neglect their children may result in feeling of worthlessness. Adler explained that both ends of the spectrum can result in mental health issues later in life (Schultz & Schutlz, 2017). Through the review of the nurture theory as well as the backing research, there is no doubt that nurture has the most influence over personality development. Conclusion Evidently there are many environmental factors that influence personality. Nurture plays an appreciable role in the development of AD, through parenting, culture, peers and life experiences. Theorists such as, Freud, Adler, and Bandura have all emphasised the importance of social and environmental factors in the development of personality. Heritability also plays a role in the predisposition to AD, and advancements in the scientific field have shown that environmental factors influence changes in genetic markers. Epigenetics is the amalgamation of nature and nurture. The age-old debate is over, and a dialogue around the complex relationship between nature and nurture is underway. This paper reviewed the significant role that nurture plays in the development of personality and anxiety as a mental health disorder. 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