Uploaded by VladyslavKurbatskyy

Gillian Todd, Rhena Branch (eds.) - Evidence - Based Treatment for Anxiety Disorder and Depression A Cognitive Behavioral Therapy Compendium-Cambridge University Press (2022)

advertisement
EVIDENCE-BASED TREATMENT FOR ANXIETY
DISORDERS AND DEPRESSION
Written by internationally recognized experts, this comprehensive
CBT clinician’s manual provides disorder-specific chapters and accessible pedagogical features. The cutting-edge research, advanced theory,
and attention to special adaptations make this an appropriate reference text for qualified CBT practitioners, students in postgraduate
CBT courses, and clinical psychology doctorate students. The case
examples demonstrate clinical applications of specific interventions
and explain how to adapt CBT protocols for a range of diverse populations. It strikes a balance between core, theoretical principles, and
protocol-based interventions, simulating the experience of private
supervision from a top expert in the field.
Gillian Todd is a freelance CBT trainer, supervisor, and practitioner.
She is a fellow of the British Association of Behavioural and Cognitive
Psychotherapies (BABCP). Now retired, Gillian was affiliated with the
University of Cambridge and University of East Anglia. She also
coauthored two CBT self-help books and two CBT therapist manuals.
Rhena Branch coauthored CBT for Dummies and is the clinical lead
and director of a mental health charity (Fig Branch, reg no. 1189312).
Now retired from lecturing, Rhena was affiliated with Goldsmith’s
University London, Anglia Ruskin University, and University of East
Anglia.
Evidence-Based Treatment for Anxiety
Disorders and Depression
A COGNITIVE BEHAVIORAL THERAPY
COMPENDIUM
Edited by
Gillian Todd
Rhena Branch
University Printing House, Cambridge CB2 8BS, United Kingdom
One Liberty Plaza, 20th Floor, New York, NY 10006, USA
477 Williamstown Road, Port Melbourne, VIC 3207, Australia
314–321, 3rd Floor, Plot 3, Splendor Forum, Jasola District Centre,
New Delhi – 110025, India
103 Penang Road, #05–06/07, Visioncrest Commercial, Singapore 238467
Cambridge University Press is part of the University of Cambridge.
It furthers the University’s mission by disseminating knowledge in the pursuit of
education, learning, and research at the highest international levels of excellence.
www.cambridge.org
Information on this title: www.cambridge.org/9781108420891
DOI: 10.1017/9781108355605
© Cambridge University Press 2022
This publication is in copyright. Subject to statutory exception
and to the provisions of relevant collective licensing agreements,
no reproduction of any part may take place without the written
permission of Cambridge University Press.
First published 2022
A catalogue record for this publication is available from the British Library.
ISBN 978-1-108-42089-1 Hardback
ISBN 978-1-108-43107-1 Paperback
Cambridge University Press has no responsibility for the persistence or accuracy
of URLs for external or third-party internet websites referred to in this publication
and does not guarantee that any content on such websites is, or will remain,
accurate or appropriate.
Contents
List of Figures
page ix
List of Tables
xi
List of Contributors
1 Introduction
xiii
. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1
Rhena Branch and Gillian Todd
2 The History and Philosophical Underpinnings of CBT:
The State of the Art . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 6
Jodie M. Paget
PART I COGNITIVE BEHAVIORAL THERAPY FOR
ANXIETY DISORDERS
3 CBT for Specific Phobias . . . . . . . . . . . . . . . . . . . . . . . . . . 29
Lina A. Gega and Barry J. D. Wright
4 CBT for Panic Disorder . . . . . . . . . . . . . . . . . . . . . . . . . . . 50
Robin D. Bailey
5 The Cognitive Behavioral Treatment of Agoraphobia
. . . . . . . . . 75
Vijaya Manicavasagar and Derrick Silove
6 Cognitive Behavioral Therapy for Social Anxiety Disorder
. . . . . . 93
Michaela B. Swee, M. Taylor Wilmer, and Richard G. Heimberg
7 CBT for Social Anxiety Disorder
. . . . . . . . . . . . . . . . . . . . . 115
Henrik Nordhal and Adrian Wells
8 CBT for Somatic Symptom Disorder and Illness Anxiety Disorder . . 136
Lillian Reuman and Jonathan S. Abramowitz
9 Cognitive Behavioral Therapy for Generalized Anxiety Disorder:
Targeting Intolerance of Uncertainty . . . . . . . . . . . . . . . . . . . 152
Cara R. Dunkley and Melisa Robichaud
v
vi
Contents
PART II COGNITIVE BEHAVIORAL THERAPY FOR
POSTTRAUMATIC STRESS DISORDER
10 CBT for PTSD . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 177
Lily A. Brown and Edna B. Foa
PART III COGNITIVE BEHAVIORAL THERAPY FOR OBSESSIVECOMPULSIVE DISORDER AND ASSOCIATED DISORDERS
11 Obsessive-Compulsive Disorder: An Updated Cognitive
Behavioral Approach . . . . . . . . . . . . . . . . . . . . . . . . . . . . 197
Oliver Sündermann and David Veale
12 CBT for OCD: Evidence-Based Treatment for Contamination
and Checking . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 222
Elizabeth A. Forrester
13 Obsessive-Compulsive Disorder: Treating Obsessions and
Associated Behavioral Responses . . . . . . . . . . . . . . . . . . . . . 252
Gillian M. Alcolado
14 Other Specified and Unspecified Obsessive-Compulsive and
Related Disorders . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 269
Jan van Niekerk and Kieron O’Connor
15 A Cognitive Behavioral Approach to Body Dysmorphic Disorder:
Assessment, Treatment and New Developments . . . . . . . . . . . . . 295
Oliver Sündermann and David Veale
16 Comprehensive Behavioral Treatment for Trichotillomania
(Hair Pulling Disorder) and Excoriation (Skin Picking) Disorder . . . 316
Charles S. Mansueto
17 Cognitive Behavioral Therapy for Hoarding Disorder
. . . . . . . . . 341
Caitlin A. Stamatis, McKenzie K. Roddy, and Kiara R. Timpano
PART IV COGNITIVE BEHAVIORAL THERAPY FOR DEPRESSION
18 Behavioral Activation . . . . . . . . . . . . . . . . . . . . . . . . . . . . 367
Courtney Colgan, Carl W. Lejuez, and Jessica F. Magidson
19 CBT for Persistent Depressive Disorder
. . . . . . . . . . . . . . . . . 383
Anne Garland
20 Rumination-Focused Cognitive Behavioral Therapy
. . . . . . . . . . 402
Edward R. Watkins
21 Cognitive Behavioral Therapy for Perinatal Depression
Laura E. Sockol
. . . . . . . . 418
Contents
vii
PART V COMPLEXITY AND COMORBIDITY IN ANXIETY
DISORDERS AND DEPRESSION: CASE FORMULATION AND
TREATMENT PLANNING
22 Case Formulation for Complexity and Comorbidity in Anxiety
Disorders and Depression . . . . . . . . . . . . . . . . . . . . . . . . . 461
Simona Stefan and Daniel David
23 Treating Complexity and Comorbidity in Anxiety Disorders
and Depression . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 475
Cory F. Newman
PART VI SPECIALIST APPLICATIONS OF COGNITIVE
BEHAVIORAL THERAPY FOR ANXIETY DISORDERS
AND DEPRESSION
24 The Application of CBT for Adults with Learning Disabilities
. . . . 507
Marc Serfaty, Matt Broadway-Horner, and Angela Hassiotis
25 Cognitive Behavioral Therapy for Children and Adults
with Autism . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 533
Peter E. Langdon
26 Adapting CBT for Treating Anxiety Disorders and Depression
in Adults with ADHD . . . . . . . . . . . . . . . . . . . . . . . . . . . 553
Laura D. Eddy, Laura E. Knouse, and Steven A. Safren
27 Adapting CBT for Children and Adolescents with Anxiety
Disorders and Depression . . . . . . . . . . . . . . . . . . . . . . . . . 573
Taylor N. Stephens, Kimberly Blears, Erin E. Soares, Julia Rogers,
and Eduardo L. Bunge
28 Adapting Cognitive Behavioral Therapy for Older Adults with
Anxiety Disorders and Depression . . . . . . . . . . . . . . . . . . . . 599
Suma P. Chand and George T. Grossberg
29 Cross-Cultural CBT . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 620
Andrew Beck and Michelle Brooks-Ucheaga
PART VII FUTURE DEVELOPMENTS
30 Definitions of Self-Esteem across CBT and REBT Literature . . . . . 643
Jodie Paget, Rhena Branch, and Michael Townend
Appendices
Index
667
681
Figures
2.1 ABC schematic diagram showing the possible interaction among
As, Bs, and Cs
3.1 Three-systems model for specific phobia
3.2 Fear cycle for BII phobia
3.3 CBT cycle for BII phobia
3.4 Habituation and extinction
3.5 Overview of CBT process for specific phobias
4.1 Cognitive model of panic disorder
4.2 A cognitive model of panic with maintenance cycles added
4.3 Psychoeducation and socialization to the model
7.1 A cognitive model of social phobia
7.2 An idiosyncratic SAD case conceptualization for Jenny
9.1 Initial clinical model of GAD for CBT-IU
9.2 Expanded clinical model of GAD for CBT-IU model featuring
negative beliefs about uncertainty
11.1 Eric’s case formulation, demonstrating how his contamination-related
OCD developed and was maintained
12.1 Case formulation
14.1 Cognitive-behavioral formulation of obsessional jealousy
14.2 BFRB model
15.1 Case formulation
16.1 Kaitlyn’s progress in ComB treatment for two BFRBs
17.1 Etiological model of hoarding disorder
17.2 Blank copy of hoarding case conceptualization model
17.3 Ron’s case conceptualization
18.1 Behavioral model of depression
19.1 Case formulation for David
21.1 Rachel’s EPDS scores over the course of therapy
22.1 Stress-diathesis model as case formulation
22.2 The general model of integrative and multimodal CBT/IM-CBT
22.3 The ABC model for specific emotions
22.4 The behavioral ABC model
23.1 Cognitive conceptualization diagram (CCD)
page 16
38
38
39
39
40
52
57
60
116
126
159
164
209
232
273
279
306
335
344
345
349
372
394
448
462
469
470
471
493
ix
x
List of Figures
24.1 Case study 1: Joe. Core belief focus formulation
24.2 Case study 2: Lisa. Belief focus formulation
30.1 Cognitive (bottom-up) model of self-esteem formation
and maintenance
30.2 An affective (top-down) model of self-esteem formation
and maintenance
523
525
647
648
Tables
3.1 Example of phobia-specific CBT assessment questions
page 34
3.2 Behavioral approach task (BAT) examples
37
4.1 Examples of catastrophic misinterpretations and an
alternative explanation
64
4.2 Panic thought record
65
4.3 Behavioral experiment record modeled with SPEAR
70
6.1 Commonly used self-report measures of social anxiety and
related constructs
98
9.1 Examples of challenges to positive beliefs about the usefulness
of worry
162
9.2 Example of behavioral experiment record form
166
10.1 Joyce’s baseline assessment scores
183
10.2 Session content
187
10.3 Joyce’s in vivo hierarchy
190
11.1 A cognitive-behavioral assessment and treatment protocol for OCD
202
11.2 Worksheet for Theory A/Theory B with example of client suffering
from sexual obsessions about children
206
11.3 Assessment measures for OCD
215
12.1 Examples illustrating the range of contamination and
checking concerns
223
12.2 A picture paints a thousand words
235
12.3 Theory A/Theory B
237
12.4 OCD/non-OCD way
238
12.5 OCD/non-/anti-OCD
244
14.1 Overview of reasoning errors in IBT
283
15.1 A cognitive behavioral therapy protocol for BDD
300
15.2 Worksheet for Theory A/Theory B with example of client suffering
from a preoccupation with balding hair
303
15.3 Assessment measures for BDD
310
16.1 Five domains of the ComB model
324
16.2 Weekly self-monitoring form
329
16.3 Intervention ideas list
332
17.1 Example sorting: Rules for Ron
355
xi
xii
List of Tables
19.1 DSM-5 diagnostic criteria for persistent depressive disorder
21.1 Assessment questions addressing characteristics and concerns of
perinatal patients
21.2 Measures of psychological symptoms and related outcomes for
perinatal populations
21.3 Example three-column thought record
25.1 Suggested changes to the process of cognitive behavioral therapy
for people with autism
27.1 Developmental framework to consider: Piaget’s theory of
cognitive development
27.2 Assessment measures
28.1 Age-specific examples of maladaptive avoidance
28.2 Age-specific examples of maladaptive safety behaviors
28.3 Cognitive distortions
28.4 Modifications in CBT for late life depression
28.5 Modifications in CBT for late-life anxiety and depression
29.1 The main differences between culturally adapted and culturally
responsive CBT
30.1 DSM-5 references to self-esteem across disorders
30.2 ICD-10 references to self-esteem and self-worth across disorders
30.3 Definitions of self-esteem across CBT and REBT literature
386
428
431
446
542
576
579
605
605
610
612
612
627
651
652
660
Contributors
JONATHAN S. ABRAMOWITZ, Department of Psychology and Neuroscience,
University of North Carolina at Chapel Hill, USA
GILLIAN M. ALCOLADO, Department of Clinical Health Psychology,
University of Manitoba, Canada
ROBIN D. BAILEY, School of Psychology, Liverpool John Moores University,
Liverpool, UK
ANDREW BECK, East Lancashire Child and Family Service, UK
KIMBERLY BLEARS, Children and Adolescents Psychotherapy and Technology
(CAPT) Research Lab, Palo Alto University, Palo Alto, California, USA
RHENA BRANCH, University of East Anglia, Norwich, UK, formerly affiliated
with Goldsmiths University of London, Anglia Ruskin University and the
University of East Anglia.
MATT BROADWAY-HORNER, University of Chester, UK
MICHELLE BROOKS-UCHEAGA, University of Derby, UK
LILY A. BROWN, Perelman School of Medicine, Department of Psychiatry,
University of Pennsylvania, Philadelphia, USA
EDUARDO L. BUNGE, Children and Adolescents Psychotherapy and
Technology (CAPT) Research Lab, Palo Alto University, Palo Alto,
California, USA
SUMA P. CHAND, Department of Psychiatry and Behavioral Neuroscience, Saint
Louis University School of Medicine, Missouri, USA
COURTNEY COLGAN, Boston College, Counseling Psychology Program, Lynch
School of Education and Human Development, Massachusetts, USA
DANIEL DAVID, Department of Clinical Psychology and Psychotherapy,
International Institute for the Advanced Study of Psychotherapy and Applied
Mental Health, Babes-Bolyia University, Romania
xiii
xiv
List of Contributors
CARA R. DUNKLEY, Department of Psychology, University of British Columbia,
Vancouver, Canada
LAURA D. EDDY, ADHD Clinic at UNC Greensboro, University of North
Carolina Greensboro, Greensboro, USA
EDNA B. FOA, Perelman School of Medicine, Department of Psychiatry,
University of Pennsylvania, Philadelphia, USA
ELIZABETH A. FORRESTER, chartered clinical psychologist in independent
practice, Orpington, UK
ANNE GARLAND, Nottingham Psychotherapy Unit, School of Health Sciences,
Medical School, Queen’s Medical Centre, Nottingham, UK
LINA A. GEGA, Department of Health Sciences and Hull York Medical School,
University of York, UK
GEORGE T. GROSSBERG, Department of Psychiatry and Behavioral
Neuroscience, Saint Louis University School of Medicine, Missouri, USA
ANGELA HASSIOTIS, Faculty of Brain Sciences, Division of Psychiatry,
University College London, UK, and Camden Learning Disability Service,
London, UK
RICHARD G. HEIMBERG, Department of Psychology, Temple University,
Philadelphia, USA
LAURA E. KNOUSE, Department of Psychology, University of Richmond,
Virginia, USA
PETER E. LANGDON, Centre for Educational Development Appraisal and
Research (CEDAR), University of Warwick, UK, and Coventry and
Warwickshire NHS Partnership Trust, UK
CARL W. LEJUEZ, Department of Psychology, University of Connecticut, USA
JESSICA F. MAGIDSON,
Maryland, USA
Department
of
Psychology,
University
of
VIJAYA MANICAVASAGAR, School of Psychiatry, University of New South
Wales, Kensington, Sydney, Australia
CHARLES S. MANSUETO, Behavior Therapy Center of Greater Washington,
Silver Spring, Maryland, USA
CORY F. NEWMAN, Perelman School of Medicine, University of Pennsylvania,
Philadelphia, USA
HENRIK NORDAHL, Department of Psychology, Norwegian University of
Science and Technology, Trondheim, Norway
List of Contributors
KIERON O’CONNOR, Research Center of the Mental Health University Institute
at Montreal, Canada, and Department of Psychiatry, University of Montreal,
Canada
JODIE M. PAGET, Department of Clinical Psychology, University of East Anglia,
Norwich, UK
LILLIAN REUMAN, Department of Psychology and Neuroscience, University of
North Carolina at Chapel Hill, North Carolina, USA
MELISA ROBICHAUD, Vancouver CBT Centre, and Department of Psychology,
University of British Columbia, Vancouver, Canada
McKENZIE K. RODDY, Department of Psychology, University of Miami, USA
JULIA ROGERS, Children and Adolescents Psychotherapy and Technology
(CAPT) Research Lab, Palo Alto University, Palo Alto, California, USA
STEVEN A. SAFREN, Department of Psychology, University of Miami, USA
MARC SERFATY, Department of Epidemiology and Applied Clinical Research,
Division of Psychiatry, University College London, and Priory Hospital, North
London, UK
DERRICK SILOVE, School of Psychiatry, University of New South Wales,
Kensington, Sydney, Australia
ERIN E. SOARES, Children and Adolescents Psychotherapy and Technology
(CAPT) Research Lab, Palo Alto University, Palo Alto, California, USA
LAURA E. SOCKOL, Psychology Department, Davidson College, North
Carolina, USA
CAITLIN A. STAMATIS, Department of Psychology, University of Miami, USA
SIMONA STEFAN, Department of Clinical Psychology and Psychotherapy,
International Institute for the Advanced Study of Psychotherapy and Applied
Mental Health, Babes-Bolyia University, Romania
TAYLOR N. STEPHENS, Children and Adolescents Psychotherapy and
Technology (CAPT) Research Lab, Palo Alto University, Palo Alto,
California USA
OLIVER SÜNDERMANN, Clinical and Health Psychology Centre (CHPC),
Department of Psychology, National University of Singapore, Singapore
MICHAELA B. SWEE, Department of Psychology, Temple University,
Philadelphia, Pennsylvania, USA
KIARA R. TIMPANO, Department of Psychology, University of Miami, USA
xv
xvi
List of Contributors
GILLIAN TODD, University of East Anglia, Norwich, UK, formerly affiliated with
the University of Cambridge and the University of East Anglia.
MICHAEL TOWNEND, Department of Health and Social Care, University of
Derby, UK
JAN VAN NIEKERK, Beechwood Centre, Cambridge, UK, and Cambridge
Clinical Research Centre for Affective Disorders, Addenbrooke’s Hospital,
Cambridge, UK
DAVID VEALE, The Institute of Psychiatry, Psychology and Neurosciences,
King’s College London, UK
EDWARD R. WATKINS, Sir Henry Wellcome Building for Mood Disorders
Research, and University of Exeter, UK
ADRIAN WELLS, School of Psychological Sciences, Faculty of Biology Medicine
and Health, University of Manchester, UK
M. TAYLOR WILMER, Department of Psychology, Temple University,
Philadelphia, USA
BARRY J. D. WRIGHT, Hull York Medical School and Child Oriented Mental
Health Intervention Centre (COMIC), University of York, UK
1
Introduction
Rhena Branch and Gillian Todd
In 2015, it was estimated that in excess of 300 million people globally suffered with
depression (WHO, 2017). Roughly the same number were estimated to suffer from a
range of anxiety disorders. Considering that many people experience comorbidity,
simply adding these figures together to arrive at a total for common mental health
disorders is not sufficient. Depression was ranked as the single largest contributor to
global disability (7.5% of all years lived with a disability in 2015) with anxiety disorders
ranked sixth largest contributor (3.4% of all years lived with a disability in 2015).
Cognitive behavioral therapy (CBT) is a therapeutic tradition that also refers to a
range of interventions sharing the basic premise that psychological and emotional
distress are maintained by cognitive factors. Early pioneers Albert Ellis (Ellis, 1962)
and Aaron Beck (Beck, 1970) posited that maladaptive cognitions including core
beliefs (also referred to as ‘schemas’) about self, others, and the world/future, when
triggered by particular conditions or situations, give rise to related negative automatic
thoughts, associated negative emotions, and behaviors. In essence, CBT holds that
interventions and strategies aimed at changing maladaptive cognitions ultimately
lead to therapeutic change in both emotional distress and dysfunctional behavior.
Since the emergence of CBT, much research has been carried out demonstrating its efficacy with specific anxiety disorders and depression. A plethora of metaanalytic studies, considered to be the gold standard of research methodology, have
examined the effectiveness of CBT for anxiety disorders and depression, quantified
in terms of the effect size, that is, a quantitative measure of statistical significance of
an observed specific treatment effect when compared with other treatments or with
a control condition. The larger the effect size, the greater the difference is between
two groups. Watts et al. (2015) conducted a meta-analysis of randomized controlled
trials (RCTs) that compared CBT with treatment as usual (TAU) in the treatment
of anxiety disorders and depression. The meta-analysis demonstrated a media effect
size favouring CBT over TAU. Cuijpers et al. (2013) in their meta-analysis support
the efficacy of CBT for depression.
The generalizability of delivering evidence-based treatment protocols derived
from RCTs into healthcare settings is often questioned by clinicians in the setting
1
2
Rhena Branch and Gillian Todd
itself. Researchers are often regarded as cherry picking participants for their studies,
recruiting a stable of talented therapists who are rigorously trained and closely
supervised; however, the evidence suggests otherwise. Otte (2011) conducted a
meta-analysis of meta-analyses of CBT for anxiety disorders and concluded that
both placebo-controlled trails and trials within a naturalistic setting supported the
efficacy and effectiveness of CBT as a treatment intervention for anxiety disorders.
Stewart and Chambless’s (2009) meta-analysis of effectiveness studies in real-world
circumstances found CBT to be effective.
In recent years, numerous disorder-specific protocols have been developed to
address specific cognitive and behavioral elements which are key components of a
disorder’s maintenance. As such, disorder-specific protocols demonstrate some
considerable differences in treatment techniques from those typically found in
‘standard’ CBT. Despite these differences in assessment and treatment focus, as
well as the use of distinctive interventions, the core theory and general approach to
treatment remains consistent.
The roll-out of the UK government initiative Improving Access to
Psychological Therapies (IAPT) in 2008, based on the premise that CBT can be
delivered effectively and economically (Layard and Clark, 2014), has increased the
number of trainees learning from a protocol-based curriculum. However, initiatives
to roll out CBT training to the masses, thereby affordably and speedily developing
an army of therapists to provide evidence-based therapy, may have observable
limitations. Brief, rapid-fire, protocol-based training means that most trainees will
learn a maximum of two protocols per common disorder. The majority of these brief
courses include little or no training on core theoretical and philosophical CBT
principles. Moreover, lack of emphasis on basic transdiagnostic skill acquisition,
coupled with a paucity of time to focus on developing and demonstrating overarching clinical competency, leaves newly trained clinicians dependent on dogged
obedience to protocols. CBT theoretically consistent ‘eclecticism’, or deviation from
protocol, is clinically justifiable and is expected among seasoned clinicians with
adequate training. CBT clinicians taught only the bare minimum of protocols on
an intensive course may, therefore, be likely to drift away from CBT altogether
when a single protocol proves insufficient.
It has been observed that therapists easily slip into being eclectic in their
therapeutic stance, offering a pick and mix approach to therapy, which has no
empirical or theoretical basis. Not all patients who think they are having CBT
actually receive the evidence-based version they rightfully expect.
Waller has extensively examined the issue of therapist drift (Waller, 2009).
For a detailed analysis of factors leading to therapist drift, see Waller and
Turner (2016).
In addition to disorder-specific protocols (i.e., obsessive-compulsive disorder
[OCD], generalized anxiety disorder, panic disorder), CBT increasingly comprises a
number of specific approaches for identified populations (i.e., older adults, people
with intellectual disabilities) and ‘third wave’ approaches (i.e., mindfulness-based
CBT, acceptance, and commitment therapy).
Introduction
The aim of this compendium is to provide a framework so clinicians can follow
evidence-based CBT protocols for treating their patients with anxiety disorders and
depression that have proven efficacy. Experts in their field from around the world
have shared their protocols and wisdom to enable the reader to (1) develop a
knowledge base of current research, (2) understand the phenomenology of psychological disorders, (3) learn idiosyncratic additions to the assessment process, (4)
follow a step-by-step guide to treatment, (5) identify and overcome common problems in treatment, and (6) effectively evaluate treatment progress.
While anxiety, depression, and other common mental health disorders are the
main focus of this compendium, some disorders such as OCD have more than one
chapter devoted to them, with more than one model or protocol represented. More
complex and nuanced disorders like OCD, as well as more newly understood
disorders such as body dysmorphic disorder (BDD), require variation in focus with
respect to assessment and treatment. Knowledge of more than one protocol and
recent research findings will prove invaluable to the clinician.
Specialist approaches of CBT focus on the treatment of anxiety disorders and
depression in distinct populations. Based on clinical presentation and need, adaptations to protocols need to be made and implemented flexibly. Leading authorities in
their field have contributed chapters on adapting CBT for common mental health
problems in diverse communities. Chapters on specialist applications of CBT for
older adults and for youth are included. CBT for mental health problems in patients
with neuro-cognitive-biological disorders, including autism spectrum disorder
(ASD), attention-deficit/hyperactivity disorder (ADHD), and identity disorder
(ID), is covered. And a chapter is included on cultural adaptations of CBT where
the authors consider the need for an understanding of ethnicity and cultural
variations.
The clinician will be enlightened in their understanding of the nuances observed
in clinical presentations and will gain knowledge in how to work effectively with
complex and highly comorbid clinical presentations.
A strength of this compendium is its international focus: chapters have been
contributed from around the world, including the United Kingdom, United States,
Canada, Australia, Singapore, Norway, and Romania.
All chapters are consistent with the American Psychiatric Association’s
Diagnostic and statistical manual of mental disorders (5th ed.) (DSM-5).
This compendium assumes a degree of reader knowledge and therefore suits an
intended audience of qualified clinicians. However, it is also likely to be useful for
post-graduate trainees and students.
Book Overview
This compendium is divided into six parts. The state of CBT today is explored and
its historical and philosophical underpinnings are reviewed in Chapter 2, which
examines how CBT has developed, transformed, and, to an extent, migrated from
its early roots (Beck 1976; Ellis, 1962).
3
4
Rhena Branch and Gillian Todd
Part I, includes seven chapters focusing on DSM-5 anxiety disorders, offering
specific models as well as providing transdiagnostic interventions. There are two
protocols offered for the treatment of social anxiety.
Part II is comprised of a chapter on the treatment of posttraumatic stress
disorder (PTSD) by pioneer in the field Edna Foa and specialist Lily Brown.
Part III includes seven chapters dealing with OCD and related disorders,
including BDD, trichotillomania, hoarding, and excoriation disorder.
Part IV features four comprehensive chapters on CBT for depressive disorders,
including behavioral activation (BA) for moderate to severe depression, targeting
depressive rumination, and chapters dealing with the treatment of persistent depression and depression within the perinatal population.
Part V hosts two chapters, the first dealing with case formulation of complex
and comorbid cases and the second discussing the treatment of complexity
and comorbidity.
Part VI consists of six chapters dealing with specialist CBT applications to
distinct patient populations.
We hope readers will agree that the result of the editors’ collaboration with
internationally recognized expert clinicians, trainers, academics, and researchers is a
comprehensive compendium that is highly relevant to all cognitive
behavioral practitioners.
We would like to thank all our esteemed contributors for their hard work
and cooperation.
REFERENCES
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Association.
Beck, A. T. (1970). Cognitive therapy: Nature and relation to behavior therapy. Behavior
Therapy, 1, 184–200.
(1976). Cognitive therapy and the emotional disorders. Penguin Group.
Cuijpers, P., Berking, M., Andersson, G., Quigley, L., Kleiboer, A., & Dobson, K. (2013).
A meta-analysis of cognitive-behavioural therapy for adult depression, alone and in
comparison with other treatments. Canadian Journal of Psychiatry, 58(7), 376–385.
Ellis, A. (1962). Reason and emotion in psychotherapy. Stuart.
(1994). Reason and emotion in psychotherapy: A comprehensive method of treating human
disturbances. Citadel.
Layard, R., & Clark, D. M. (2014). Thrive: The power of evidence-based psychological
therapies. Allen Lane.
Otte, C. (2011). Cognitive behavioral therapy in anxiety disorders: Current state of the
evidence. Dialogues in Clinical Neuroscience 13(4), 413–421.
Stewart, R. E., & Chambless, D. L. (2009). Cognitive-behavioral therapy for adult anxiety
disorders in clinical practice: A meta-analysis of effectiveness studies. Journal of
Consulting and Clinical Psychology, 77(4), 595–606.
Waller, G. (2009). Evidence-based treatment and therapist drift. Behaviour Research and
Therapy, 47(2), 119–127.
Introduction
Waller, G., & Turner, H. (2016). Therapist drive redux: Why well-meaning clinicians fail to
deliver evidence-based therapy, and how to get back on track. Behaviour Research and
Therapy, 77, 129–137.
Watts, S. E., Turnell, A., Kladnitskj, N., Newby, J. M., & Andrews, G. (2015). Treatment-asusual (TAU) is anything but usual: A meta-analysis of CBT versus TAU for anxiety and
depression. Journal of Affective Disorders, 175, 1152–1167.
World Health Organization. Depression and other common mental disorders: Global health
estimates. (2017). World Health Organization. License: CC BY-NC-SA 3.0 IGO.
5
2
The History and Philosophical Underpinnings
of CBT: The State of the Art
Jodie M. Paget
You don’t need to go out and buy pop psychology self-help books. When you read
the classics in the humanities, you become aware that the big ideas have been
around for a long time, despite the fact they are often served up today in modern
psychological explanations of human action as novel and scientific.
—James Stockdale (1995, p. 24)
Cognitive therapies are often attributed to Albert Ellis and Aaron Beck, who
created rational emotive behavior therapy (REBT) and cognitive therapy (CT),
respectively. This chapter discusses the historical development, philosophical underpinnings, and current state of CBT. ‘Third wave’ approaches, and what these have in
common with ‘second wave’ theoretical concepts and protocol-driven CBT, are
discussed alongside possible implications for quality of CBT training (i.e.,
Improving Access to Psychological Therapies [IAPT] training courses in the
United Kingdom). Commonalities between REBT and ‘new developments’ are
highlighted, asking the question of how it is that third wave CBT such as ACT
(Hayes et al., 2012), and dialectical behavior therapy (DBT) (Linehan, 1993), for
example, give little credence to Ellis, when those practicing REBT, or who have an
understanding of it, are keenly aware of the commonalities (Malkinson, 2011).
Furthermore, are concepts such as ‘self-compassion’ (Neff, 2003), ‘clinical perfectionism’ (Egan et al., 2014), and ‘intolerance of uncertainty’ (Dugas and Robichaud,
2007) a reinvention of core aspects of REBT? The idea that some of these new
developments were posited by early theorists will be explored.
First Wave Behavioral Therapies
Classical CBT (REBT and CT) are commonly referred to as ‘second wave’ cognitive
behavior therapies. It is important to note that during the 1950s and 1960s ‘first wave’
behavior therapies were also developing and great efforts were made to have them
empirically validated. Briefly stated, behavior therapy can trace its genesis to the
work of Pavlov (1955), whose research in classical conditioning theory paved the way
6
The History and Philosophical Underpinnings of CBT
for the study of abnormal behavior, in particular, research pertaining to fear acquisition and de-conditioning (Wolpe, 1958). The Institute of Psychiatry and the Maudsley
Hospital in London were home to several pioneering psychologists (such as Cyril
Franks, Hans Eysenck, and Stanley Rachman), who each made significant contributions to behavioral research during World War II and afterwards. In 1956, H. S.
Jones, a clinical psychologist at the Maudsley, wrote, ‘The Application of
Conditioning of Learning Techniques to the Treatment of a Psychiatric Patient’,
considered the first published article on behavior therapy. In 1965, Eysenck and
Rachman wrote the first behavioral therapy textbook, considered one of the single
most significant developments within the field at that time (Marks, 2012).
In 1966 whilst at Middlesex Hospital in London, Dr Victor Meyer applied some
of the techniques he had acquired (and achieved success with) during his time at the
Maudsley, towards treatment of OCD. At that time there was little to offer sufferers
of OCD, but through the application of exposure and response prevention (ERP)
significant improvements were made. Over the next decade the treatment was
refined and made available to outpatients to good effect (see Rachman et al.,
1979). Rachman (2015) provides a detailed history of behavior therapy. He states
that in the 1970s attention to cognitive factors occurred in part due to Bandura’s
(1969, 1977) research into social-cognitive factors, and success was seen in the
treatment of social anxiety and generalized anxiety. Due to the success of these
approaches, behavior therapists became open to incorporating cognitive concepts,
and began using cognitive techniques alongside behavioral components, in part due
to a need to develop an effective therapy for depression. Rachman adds that the
inclusion of behavioral experiments within Beckian CT was reassuring to behavioral
therapists and, in part, paved the way for a cognitive behavioral form of therapy.
The combining of these two approaches was further solidified by Clark’s (1986)
cognitive therapy for panic disorder and Barlow’s (1988) panic control therapy.
Second Wave Cognitive Behavioral Therapies
REBT
In the 1950s Ellis created what is now known as rational emotive behavior therapy
(REBT) and described it as a cognitive-experiential-behavioral system of therapy
that has strong philosophical underpinnings and an active focus on disputing dysfunctional self-defeating beliefs (Ellis, 2001). REBT was the first form of cognitive
behavioral therapy and Ellis’s ABC(DE) model is not only the foundation of
REBT, but also a core theoretical construct of all subsequent versions of CBT
(David et al., 2005). It is well documented that the creation of REBT was born
primarily out of Ellis’s scepticism of the efficiency of psychoanalysis, the therapeutic
orientation Ellis had originally trained in (Ellis, 1962):
I went back to my hobby since the age of 16 . . . the philosophy of the ancients, largely of
Epictetus, Marcus Aurelius, and in the east of Confucius and Lao Tsu and Buddha, and
7
8
Jodie M. Paget
amalgamated it with behavior therapy, which I had used on myself at the age of nineteen
to get myself over my phobia of public speaking and of approaching young females.
(Interview with Ellis in Ellis, 2010)
Ellis overcame his fear of rejection from potential dating partners via philosophically informed interventions. He exposed himself to feared situations, deliberately challenging his pre-existent belief systems and deliberately adopting more
functional ways of thinking and behaving. Ellis gave his first presentation of
REBT, then known as rational therapy, in the late 1950s, and shortly after changed
the name of the approach (due to an accusation of rationalism) to rational emotive
therapy (Yankura and Dryden, 1994). Rationalism deems thought superior to other
senses when determining the validity of knowledge, and that distinct boundaries
exist between mental and physical processes (see DiGiuseppe and Linscott, 1993,
for a detailed review). Whilst it is not surprising for such a misunderstanding to
occur, perhaps partly due to common misunderstandings of the underpinning philosophy of Stoicism, Ellis (1958) states that human emotion and creativity are not
controlled (nor should they be) by reason and intellect alone.
It is evident here that although REBT does stress that if one is to achieve
significant reduction in emotional distress, one must alter the associated belief
system. This does not mean, however, that psychological intervention is limited to
or should only focus on work at a cognitive level. Throughout the 1950s, 1960s, and
1970s REBT grew in popularity, and in 1959 the Albert Ellis Institute was established in New York. In 1993 the second (and most current) rebranding of rational
emotive behavior therapy was undertaken. This was done in part due to Ellis
insisting the ‘between session tasks’ and behavioral work undertaken in therapy
be acknowledged (Yankura & Dryden, 1994).
In 1994, the second edition of Reason and Emotion in Psychotherapy was
published, containing an updated and revised version of Ellis’s conceptual model
of human disturbance, known as the ABC(DE) model (see Figure 2.1). This model
is still widely used within CBT, in both its original and updated forms (Dryden,
2013a; Dryden & Branch, 2008). Simplified versions that may neglect or incorrectly
present core philosophical and theoretical constructs also exist currently (e.g.,
Skinner & Wrycraft, 2014). Yankura and Dryden (1994) state that during the
1980s, REBT had developed an international presence and treatment manuals were
developed for couples therapy (Ellis et al., 1989), individual counselling (Dryden,
1987), and family therapy (Huber & Baruth, 1989). A study conducted by Smith
(1982) found that Ellis was placed between Carl Rogers and Sigmund Freud as the
second most influential psychologist (Aaron Beck was seventh) by a vote of over
400 American psychologists. Despite the undeniable contributions Ellis made to
psychology and psychotherapy, many CBT therapists are unaware of the origins of
CBT, or credit it primarily to Aaron Beck. So, what happened? Why is REBT
regarded as ‘the disturbed child in the family’ (Malkinson, 2011, p. 259)?
REBT Current Status. When Ellis developed his theory of personality (Ellis,
1978b) and put forward a conceptual model that accounted for psychological
The History and Philosophical Underpinnings of CBT
disturbance, it offered an explanation for emotional distress as opposed to advancing specific disorders. This could be considered one of the failings of REBT, in that
unlike CT it has done little to advance understanding of specific psychiatric disorders. This was highlighted by David (2014), who states REBT research tends not
to be related to specific psychiatric conditions and that there is a lack of rigorous
research that is necessary for a therapeutic approach to be considered evidencebased. REBT interventions are primarily aimed at challenging evaluative beliefs,
rather than inferences (i.e., negative automatic thoughts [NATs]), and its interventions are transdiagnostic. Knowing why a psychological intervention works is just as
important as the theory that attempts to explain the phenomena; therefore, welldesigned research aimed at examining the efficacy of REBT is well overdue (David
and Montgomery, 2011).
A criticism of REBT put forward by Padesky and Beck (2003) is that it is it
philosophically based whilst CT is empirically based. Ellis (2005a) argues, however,
that both therapies are philosophically and empirically based, but REBT is more
philosophical and CT currently has more empirical support. When Isaac Newton
felt the apple hit his head, this was empirical evidence that apples fall from trees.
Seeing an apple fall does not explain why it fell, but it does provide validation for
the theory of the law of universal gravitation. Both Ellis (2005a) and Padesky and
Beck (2005) welcome the recommendation that REBT become more empirically
validated, yet there is still a significant lack of well-designed studies to examine its
empirical status. Qualitative studies have shown that REBT is effective for both
clinical and non-clinical populations, that the efficacy of individual and group
therapy is broadly similar, that a higher number of sessions produces greater results,
and that the more well trained the therapist, the better the results (Dryden, 2012a).
A meta-analysis by David et al. (2005) concluded REBT interventions are efficacious for a wide range of conditions, regardless of how interventions were delivered
and the clinical status or mean age of the sample. When analyzing presumed
mechanisms of change, REBT is deemed to be efficacious. However, due to the
diversity of outcomes and reporting and the quality of measures utilized, more
research is needed that includes transdiagnostic and diagnostic categories.
Philosophy of CBT
Stoicism
The philosophical underpinnings of cognitive behavioral therapies can trace their
origins to ancient Greek and Roman Stoic philosophers, such as Marcus Aurelius
and Epictetus, as well as Asian philosophers such as Buddha, Confucius, and Lao
Tsu. Both Ellis and Beck cite Stoicism as a primary philosophical foundation in the
development of their approaches, yet this fact is seldom acknowledged by many
therapists (Robertson, 2018). Stoicism is a school of thought founded by Zeno of
Citium (330 BCE) that existed as a philosophical movement for around 500 years.
Its main assertions are not limited to what we can know, or what is right and wrong,
9
10
Jodie M. Paget
but concerns itself more with how one should live. Whilst not unique in this idea –
the same can be said for Epicureanism – Stoic philosophy insists happiness can be
achieved through virtue alone (Sellars, 2010). Stoicism is commonly, and erroneously, considered to be synonymous with repression of emotion. Stoic philosophy
does not advocate suppression of affect, but rather endeavours to determine what
are natural human feelings and what affective responses one would have if one were
free from false belief. The aim, therefore, is to avoid feeling what is false, as opposed
to eradicating negative affect (Graver, 2007).
Epicureanism
The Stoic concept of faulty judgement and its link to affective distress clearly
mirrors the core theory of REBT: rigid absolutism as a primary cause of human
distress. However, this is not the only Hellenistic school of philosophy that Ellis
deemed informative when it came how one should live. Ellis advocated responsible
hedonism, and suggested limiting short-term indulgences, a key theme in Epicurean
philosophy (Ellis et al., 2009). Epicurus argued that the primary motivators that
underpin almost every action are the pursuit of pleasure and avoidance of pain, and
that providing our beliefs are under control, hedonism need not be a threat to our
freedom (Warren, 2009).
Ellis frequently highlights the philosophic emphasis REBT has over CT. He
proposes humans are natural born philosophers with an innate biological tendency
to be irrational; humans create meaning, have the ability to predict the future via
rational means, and are natural scientists (Yankura & Dryden, 1994). It is the role of
the REBT therapist, therefore, to facilitate profound philosophical change within
the client that will ease both current and future emotional distress (Ellis, 1980). In
addition to the ancient philosophies mentioned, REBT also has current influences,
such as Karl Popper and Bertrand Russell, and aligns itself well with Christian
values, ethical humanism, and a humanistic-existential view of life (Dryden, 2012a).
History of Cognitive Therapy
Similarly to Ellis, Aaron Beck also practiced as a psychoanalyst and had endeavoured to put to empirical testing theories such as depression being the result of
unreleased aggression. He found that dream studies and many other psychoanalytic
concepts did not stand up to empirical testing, yet did fit with cognitive explanations
(Beck, 1991). In 1967 Beck wrote the pioneering and comprehensive text
Depression: Clinical, Experimental and Theoretical Aspects, a first look at cognitive
therapy for depression (Beck, 1967). This text outlined the cognitive model of
depression and its proposed treatment recommendations. The model proposes
three concepts that explain the proposed psychological mechanisms of depression,
these being the cognitive triad, schemas, and cognitive errors. The cognitive triad is
comprised of three cognitive patterns that create an idiosyncratic experience for the
person, these being negative views of self, experiences (world), and the future. The
The History and Philosophical Underpinnings of CBT
lack of motivation common in people with depression is viewed as a consequence of
cognitions that result from these mechanisms. Suicidal ideation is viewed as
an escape strategy, dependency a consequence of views of self as helpless/
ineffective, and psychomotor inhibition a result of assuming all is futile (Beck,
1970; Beck et al., 1979).
Schemas are described as enduring attitudes that influence the content of
cognitions. They screen, code, and evaluate stimuli that impinge on the person,
enabling them to interpret experiences in a meaningful way. Schemas can be
inactive, meaning they are asymptomatic at times, but can be activated and energized by external factors. When schemas are activated in depression, thoughts are
quickly saturated with depressed content, and are more easily evoked by external
triggers (Beck, 1970).
Cognitive distortions, the third concept, are the frequently articulated thoughts
of the depressed individual. These thoughts deviate from logical thinking and can be
classified as paralogical (arbitrary inference, selective abstraction, and overgeneralization), stylistic (exaggeration), or semantic (inexact labelling) (Beck, 1970).
Modification of schemas should therefore lead to changes in how one interprets
and organizes experiences and how one goes about setting and achieving goals
(Beck et al., 1979). In 1991 Beck published a subsequent edition of Cognitive
Therapy and the Emotional Disorders where he cites Ellis’s contributions as a major
development in the field of cognitive therapy and goes on to state that upon
discovering the similarities in techniques and formulations between both
approaches, he gained confidence he was traveling in the right trajectory
(Beck, 1991).
Beck’s relentless pursuit of researching depression and anxiety disorders has
culminated in a variety of validated measurement tools (e.g., Beck Depression
Inventory [BDI-II, Beck et al., 1996], Beck Hopelessness Scale [BHS, Beck and
Steer, 1993a], Beck Anxiety Inventory [BAI, Beck and Steer, 1993b]) used as gold
standards for research, and a sound body of research demonstrating the efficacy of
CBT for common mental health problems. In 1994, Beck and his daughter Judith
Beck set up the Beck Institute for Cognitive Therapy in Philadelphia, which provides training and supervision in CBT. Subsequently, CBT, under its broad heading,
has been found to be efficacious for a wide range of disorders, and research into the
proposed change mechanisms responsible for symptom reduction within disorders is
vast (see Hofmann et al., 2012, for a detailed meta-analysis of CBT efficacy).
Third Wave Cognitive Behavioral Therapy
Having provided a brief overview of first wave behavioral approaches of the
treatment of psychological problems and the influence and contribution these had
on the development of second wave approaches (specifically REBT and CT), this
section outlines the basic principles of acceptance and commitment therapy (ACT;
Hayes et al., 2012), arguably the only form of third wave CBT. The concept of selfcompassion (Neff, 2003) is also discussed (but as to whether these legitimately fall
11
12
Jodie M. Paget
into second wave or third wave therapies is beyond the scope of this chapter).
Briefly put, Hayes et al. (2006) state that metacognitive therapy (MCT; Wells,
2009), dialectical behavior therapy (DBT; Linehan, 1993), and mindfulness-based
cognitive therapy (MBCT; Segal et al., 2001) can be considered third wave
approaches; however, David and Hofmann (2013) claim the respective authors do
not share this view). Discussion of ACT (Hayes et al., 2012) and self-compassion
(Neff, 2003) are included not to provide a detailed account of their developments or
current standing within the field, but rather to discuss similarities in core principles
that have been overlooked, often due to comparisons occurring between CT and
various third wave approaches.
Acceptance and Commitment Therapy (ACT)
Hayes and Hofmann (2017) claim third wave CBT began approximately in 2004 and
was primarily defined by a set of contextual concepts that paid attention to how a
person relates to thoughts and emotions, as opposed to the actual content of
thoughts. They propose that ACT, DBT, and MCT can be defined as third wave
versions of CBT and add that these approaches emphasize emotions, metacognition, and acceptance. In 2012, Hayes et al. outlined the main principles of
ACT, a psychotherapeutic approach based on radical behaviorist principles, in
particular, relational frame theory (RFT) (see Hayes, 2004, for a detailed account).
Despite its close link to first wave approaches, it is referred to as a third wave
modality due to that fact that interventions target the way people relate to thinking
and feeling, rather than direct challenging of specific cognition or experience
(Morris & Oliver, 2012). ACT is based on functional contextualism, a pragmatic
philosophy of science that aims to predict and influence behavior. Biglan and Hayes
(1996) argue behavioral problems may be prevented or ameliorated if behavioral
scientists focus on developing empirically based concepts that enable behavioral
phenomena to be predicted and influenced. A central aim within ACT is to increase
psychological flexibility, defined as enhancing the ability to make contact with the
present moment, based on what is possible in that moment, and to behave in ways
that are in accordance with one’s chosen values. In order to achieve this end,
ACT incorporates mindfulness, acceptance, and cognitive defusion skills (Hayes
et al., 2012).
A key difference often acknowledged between ACT and CBT is ACT’s rejection of cognitive restructuring (CR) as an intervention, due to the assumption it
encourages suppression. This is discussed in detail by Arke and Craske (2008), who
state that the labelling of some thoughts as ‘faulty’ may increase a desire to suppress
such content, and that thought challenging may lead to reduced sense of control;
however, they do conversely state the distress-reducing effects of CR and highlight a
great number of similarities between ACT and CBT (specifically, CT). A common
oversimplification of CR is the identification and labelling of thoughts as ‘faulty’ and
this is often equated to using empirical-based disputing techniques as a primary
intervention. Testing whether a thought (most likely a negative automatic thought
The History and Philosophical Underpinnings of CBT
[NAT]) stands up to empirical challenging is ubiquitous throughout CBT textbooks
(e.g., Greenberger et al., 2015) and to many disorder specific protocols to the
neglect of more advanced disputing methods (see DiGiuseppe, 1991; Ellis, 1993;
Neenan and Dryden, 1999). Arguably, skilled CR necessitates going beyond
empirical-based arguments to challenge cognition (whether via verbal discussion
or thought records); however, sophisticated CR methods are too seldom represented within protocol-driven CBT.
Defusion
Defusion is a core component of ACT which aims to reduce the believability and/or
attachment to thoughts without direct challenging of thought content. Defusion is
intended to counteract cognitive fusion, defined by Hayes et al. (2012) as the
inability to distinguish between awareness and the cognitive narrative of one’s mind,
due to the inextricable binding of thoughts and meaning. David and Hofmann
(2013) claim a fundamental weakness of ACT is the idea that it is possible to have
cognition (thought) without simultaneously passing some kind of judgement (evaluation). Furthermore, they argue this assumption can be counter-argued by the
Spinoza model of mental representation, which lends credence to this criticism
(see Appendix 2.1). This model argues that in order for one to understand, one
simultaneously and automatically attributes a value of true (or false) to a specific
thought. In other words, the idea that it is possible to change dysfunctional cognition
via ACT interventions (acceptance, cognitive defusion, and mindfulness) into ‘neutral’ thoughts is potentially flawed. The view that people automatically, unconsciously, and quickly evaluate cognition and assign a value of true or false to it is
anecdotally endorsed by many REBT and CBT clinicians.
David and Hofmann (2013) propose that taking a look at the differences
between CT and REBT may help in clarifying the issue of whether one can have
cognition and decide what to do with it without simultaneously evaluating it. Briefly
stated, Abelson and Rosenberg (1958) define cold cognition as that which reflects
the process of representation, knowing, and description, whereas hot cognitions are
indicative of the process of an evaluation or appraisal. CT views cold cognitions as a
proximate cause of distress, whereas REBT views them as distal causes and views
hot cognitions as proximate to distress (Hyland & Boduszek, 2012). Hence, a
fundamental research difference between these two approaches is CT’s focus on
cognitive distortions (e.g., selective abstraction, arbitrary influence, absolutistic
thinking; see A. Beck, 1970; J. Beck, 2011) and REBT’s focus on evaluations and
appraisals in the form of irrational beliefs (i.e., rigid demands and their derivatives;
awfulizing, low frustration tolerance, and self/other/world depreciation; Dryden &
Branch, 2008).
REBT is consistent with Lazarus’s (1991) appraisal theory of emotions, which
proposes that despite representational cognitions contributing to appraisal, it is the
process of appraisal itself that gives rise to emotions (Hyland & Boduszek, 2012).
Therefore, ‘neutralizing’ (defusing) of cognition can occur, but probably does so due
13
14
Jodie M. Paget
to changing an underlying evaluation (an irrational belief/evaluative assumption),
meaning ‘defusing’ may occur in part due to a form of CR (David & Hofmann,
2013).
Acceptance
In addition to developing defusion skills, ACT promotes developing acceptance,
defined by Hayes et al. (2012) as an ongoing process – an alternative to avoidance
and a function, rather than a technique. For a discussion of similarities between
CBT (Beckian CT, in particular) and ACT, see Arch and Craske (2008), who claim
CBT may contain elements of acceptance. However, if CBT also included REBT in
such comparisons, it would be hard to ignore that acceptance is a core principle,
explicitly defined and deeply rooted in REBT’s underpinning philosophy. It is
postulated that lack of acknowledgement of CBT’s advocacy of acceptance is in
part due to (1) current literature defining CBT solely as Beckian CT and omitting
REBT and (2) a faulty assumption that REBT and CT hold a shared view on
acceptance and self-esteem. Indeed, Beck’s pursuit to empirically validate CT for
depression occurred whilst Ellis was forcefully promoting attitudes of self, other,
and world acceptance (Ellis, 2005b, 2007).
Ellis (2005b) (and subsequent REBT theorists such as Windy Dryden, Ray
DiGiuseppe, Janet Wolfe, and Paul Hauck, to name a few) consistently promotes
the adoption of acceptance-based beliefs, and of behaving in accordance with new
healthy attitudes (rational beliefs). Though perhaps not as explicit as ACT’s focus
on value-directed action, rational beliefs that are fair, compassionate, and pragmatic
tend to be highly concordant with an individual’s values, thereby promoting valuedriven behavior. Thus, although Ellis and other early theorists have not put specific
emphasis on values per se, it seems reasonable to assume that one’s values are
considered both important and intrinsic to meaningful therapy.
A final note regarding philosophical similarities between ACT and components
of REBT is that both make reference to sharing aspects of Buddhist philosophy.
Hayes (2002) highlights that ACT shares several key notions with Buddhism that
are relevant to human distress, namely, attachment and its role in suffering, and
mindfulness. Ellis indicates that irrational and therefore unaccepting (self/others’/
world) beliefs greatly contribute to distress, and that in order to be less distressed,
one better become less attached to such ideas. He advocates adopting flexible
accepting preferences that reflect strongly held ideals but allow for philosophical
acceptance when life falls short of one’s ideals. In summary, the ACT intervention
of cognitive defusion seems unlikely to occur without some kind of change in the
associated belief system that is, in part, causative of such cognitive content. Whilst it
may be true that CT does not explicitly advocate acceptance, this has been a core
principle of REBT since its inception. Therefore, accuracy would be promoted if
comparative analyses more clearly stated to which school of CBT they referred.
Whilst ACT provides a valued contribution in the continued development of CBT,
it may be worthwhile taking stock of how similar some of the key change methods
The History and Philosophical Underpinnings of CBT
are to REBT, giving due acknowledgement and continuing research into these
mechanisms of change.
Self-Compassion
Neff (2003) claims the concept of self-compassion arose due to problems with the
concept of self-esteem, an ill-defined psychological phenomenon, yet a concept widely
endorsed within CBT literature (see Beck, 2011; Fennell & Jenkins, 2010; Wilding &
Palmer, 2010). Self-compassion differs from self-esteem in that is does not
advocate rating the ‘self’ as good or bad (Neff, 2009). Self-compassion is comprised
of (1) self-kindness, as opposed to harsh self-criticism, (2) common humanity and
one’s connectedness to others, and (3) mindfulness (Neff and Lamb, 2009). Ellis
rejects the concept of self-esteem and instead advocates unconditional self-acceptance
(USA), endorsing principles of self-compassion (Ellis, 2005b).
Encouraging clients to develop unconditional self-acceptance (USA) and unconditional other-acceptance (UOA) is a key aspect of REBT. Essentially, this means
viewing others as fallible, complex, and in a state of flux. When USA and UOA are
implemented via consistent and deliberate practice, this increases one’s interconnectedness (Dryden, 2013b). Therefore, self-kindness has much in common with USA,
and the connectedness to others can be seen within UOA. Mindfulness, however, is as
not seen as a necessary component in USA, but rather a likely by-product that is a
consequence of USA (Ellis, 2005b). For a detailed discussion on the differences
between self-compassion and USA and how these concepts can be integrated, see
Dryden (2013b), who provides a model for how USA and self-compassion can be
utilized concomitantly towards the reduction of psychological distress.
When looking for alternative or innovative ways of working with low selfesteem or distressing emotions such as guilt and shame, CBT therapists who are
trained primarily in protocol-driven CBT might understandably search for new
developments within second or third wave CBT literature. However, a wealth of
literature addressing interventions for emotional problems exists within REBT (e.g.,
anger, Dryden, 1993; guilt, Dryden 1994; shame, Dryden, 1997; jealously, Dryden,
2005; hurt, Dryden, 2007). Self-compassion and REBT have long been bedfellows
and are happy in their relationship.
The (D)Evolution of the ABC Model
When attempting to put forward a conceptual model derived from theory, complex
ideas are often simplified and presented as partial versions of the greater phenomenon. Although models are useful guides, a map of the territory should not be
confused with the territory itself (Korzybski, 1958). In 1962 Ellis put forward a
conceptual model of human disturbance, which was later updated and illustrated
diagrammatically in the second edition of Reason and Emotion (Ellis, 1994). For a
detailed account of earlier models that existed, influenced, and shaped this model, see
Korzybski (1958). This model was simple, yet profound: ‘A’ represents Activating
15
16
Jodie M. Paget
stimulus energy transmitted by external happening
sensory system response
A
activity in sensory area of the brain (sensation
experienced)
activity in sensory association area (sensation
interpreted)
B
cortical area (experience tied in with belief systems
and evaluated)
C
-emotional and behavioral reactions
Figure 2.1. ABC schematic diagram (as cited in Ellis, 1994, p. 84) showing the possible
interaction among As, Bs, and Cs. The forward progression of the event extends from
the top to the bottom of the diagram, and the interactional feedback is indicated by
the arrows.
Events, ‘B’ beliefs systems, and ‘C’ emotional and behavioral consequences (see
Figure 2.1). What is significant about this model is the acknowledgement of its
interactional nature and the proposition that Bs are inherently emotionally laden
(for a full discussion, see Ellis, 1994, p. 87). REBT ‘Bs’ differ greatly from
Meichenbaum’s ‘self-statements’ and Beck’s ‘automatic thoughts’. Beliefs at B are
comprised of evaluations and musts (often omitted in the formulations of
Meichenbaum and Beck). Overt evaluations of good or bad, desirable or undesirable,
are clearly stated at ‘B’, as such overt evaluation renders them emotionally laden.
This point is stressed by Dryden (2012b), who states that in order to correctly
present theory, REBT theorists make a conscious effort to avoid stating ‘B’ causes
‘C’, and will instead use terms such as ‘B’ largely determines ‘C’, or ‘B’ is at the core
of ‘C’. Yet this significant point is too frequently omitted in examples across CBT
literature, resulting at times in reproduction of diluted constructs. Moreover, cognitive consequences are not indicated at ‘C’. Whilst this is true of Ellis’s original
model, the Dryden framework acknowledges that cognition occurs at each part of
the ABC, and specifically that cognitive consequences (of ‘B’) occur at ‘C’ (see
Dryden, 2012b). The omission of cognitive consequences is significant, as this is
what many REBT theorists would label worry or rumination (e.g., ‘I’ll always be
alone, no one will ever like me’), which some CT theorists might label NATs.
When discussing theoretical comparisons between approaches, the use of the
word ‘belief’ can at times be problematic. The word ‘belief’ in some CBT literature
is not always evaluative, as it is in REBT, which can cause theoretical confusion. For
example, in Westbrook et al. (2012, p. 198), they state, ‘If I become very anxious
The History and Philosophical Underpinnings of CBT
I will lose control and go mad.’ Some textbooks may label this a prediction or a
predictive NAT as opposed to a belief. REBT activating events can be internal or
external; past, present, or future; real or imagined (Cristea et al., 2015), meaning the
above prediction, if one is utilizing the ABC, is best placed at A, rather than B. This
may result in different interventions depending on where the CBT therapist places
this level of cognition.
In order for complex psychological concepts to be understood and communicated accurately, it would be ideal that any form of psychotherapy that places itself
under the umbrella of CBT would make consistent use of CBT nomenclature.
A typical example of the dilution of the ABC model can be seen in Skinner and
Wrycraft (2014)’s CBT textbook (recommended for those training within IAPT),
where ‘A’ is an exam, ‘B’ is meaning (I might fail), and ‘C’ is anxiety. ‘I might fail’ is
not overtly evaluative, nor stated clearly as a rule/demand, but rather is a predictive
NAT indicating the possibility or prospect of failing. As such, the prospect of failing
belongs at A, the meaning of which we can infer is negative due to its associated
feelings of anxiety.
Kuyken et al. (2009, p. 176) make use of an ABC model, and refer to it as an
ABC functional analysis. A refers to antecedents, B refers to behaviors, and
C refers to consequences (see Figure 2.2). Cognition – beliefs, in particular – are
not clearly labelled nor are they given indication of where one should place relevant
cognition. In the B (behavior) of their example, they place ‘Thought about all the
work I had not done and how useless I am.’ Whilst thinking is a cognitive behavior,
this statement contains an evaluative label, so some CBT therapists may have
identified the ‘I’m useless’ and would label this a belief. A therapist utilizing this
ABC model would therefore have to put evaluative cognition within a behavioral
heading. Whilst Kuyken et al. (2009) do not refer to the origin of the ABC model in
their text, this example demonstrates how some CBT therapists make use of an
ABC model that differs greatly from how other CBT therapists may utilize an ABC
model, such as Skinner and Wrycraft (2014, see above). Having a B within an
ABC model labelled ‘behavior’ in a CBT textbook demonstrates how the use of
ABC models within CBT varies greatly, arguably creating theoretical confusion.
These points demonstrate that the ABC model is not always represented
accurately or consistently within CBT literature. Consequently, recent REBT developments are not always acknowledged within CBT literature or are presented in a
manner that neglects core theoretical principles. Potential consequences can include
a theoretical minefield across literature and possibly the implementation of superficial interventions that result from theoretical inconsistencies in the presentation of
terminology (for more detail on the definitions of self-esteem within CBT, see
Chapter 30).
Protocol-Driven CBT
Protocol-based CBT or empirically supported treatment (EST) protocols are a set of
specific interventions, used within a set time period for a specific disorder that have
17
18
Jodie M. Paget
been shown to be efficacious. These are often compared to a case-formulation
approach, which arguably provides greater flexibility over treatment, enables work
with more complex presentations, and allows clinicians to make treatment decisions
based on continued assessment and their clinical acumen that stems from a sound
understanding of core theory (Persons, 2006). There is now a wealth of manuals
available to therapists for the treatment of both common and more complex mental
health problems (e.g., PTSD, Foa et al., 2007; social anxiety disorder, Hope et al., 2010).
The efficacy of protocols for specific disorders is beyond this chapter (see Addis et al.,
2006, for a detailed discussion), but what is known is that the efficacy of CBT for
depression appears to be decreasing. A meta-analysis conducted by Johnsen and
Friborg (2015) found CBT is not quite as potent as it once was. It is no great surprise
that they found that patients who received CT for depression from experienced
psychologists achieved better results than those receiving it from student therapists.
They propose the decline in treatment efficacy may in part be due to a misconception
that CBT is easy to learn, and highlight the need for high-quality training and supervision that is conducted by appropriately qualified therapists. In 1979 Beck outlined the
prerequisites for psychotherapists conducting CT for depression and stated that the
aspiring cognitive therapist should be, at first, a competent psychotherapist. He found
fewer than 25% of trainees (consisting of psychiatrists and psychologists) met minimum competency in CT after three months of training in the delivery of CT for
depression (Beck et al., 1979). Considering that many training courses in the United
Kingdom require competency in delivering CBT for depression, in addition to a wide
range of mental health problems within an academic year, it may be worth researching
whether increasing duration of training facilitates a wider range of competencies, to
help ensure the efficacy of CBT remains as high as it once was.
Whilst empirically supported protocols (ESPs) and manuals are useful, patients
rarely fit neatly into one diagnostic criterion. Hayes and Hofmann (2017) claim that
evidence-based practitioners can enhance their efficacy by employing methods from
all generations of CBT and add that there is increased recognition of the importance
of philosophical assumptions to methods of treatment interventions. Moreover, they
state that upon recognition of this, the inter-organizational task force on cognitive
and behavioral psychology doctoral education insists that CBT training include
philosophy of science training. Comorbidity is common, and patients can experience
a great deal of distress that does not have an associated DSM-5 diagnosis (e.g., meet
criteria for a major depressive episode but also struggle with jealously or anger).
CBT training that includes teaching of core theoretical concepts, in addition to
disorder-specific protocols, may go some way to ensuring that CBT therapists are
better equipped to work with the wide variety of presentations they will no
doubt encounter.
Emotional Disorders and Transdiagnostic CBT
In addition to manuals that guide treatment of specific disorders, protocols are
beginning to surface that provide guidance for working with emotional disorders
The History and Philosophical Underpinnings of CBT
and transdiagnostic processes. CT is known for its diagnostic orientation, specifically
for treatment of depression, with only a recent move towards process-oriented or
transdiagnostic CBT (Mansell et al., 2012). Although Beck (1991) outlined some of
the cognitive themes for anger, depression, and sadness, cognitive theory and
interventions for emotional disorders has been refined and further developed since
the early 1990s, primarily by REBT theorists (see DiGiuseppe & Tafrate, 2010;
Dryden, 1993, 1994, 1997; Dryden & Branch, 2008).
REBT is inherently transdiagnostic, and does not introduce specific models to
explain disorders; rather, it indicates what beliefs and derivatives should be the
target for intervention. In generalized anxiety disorder (GAD), for example, beliefs
related to uncertainty are the focus of therapy, as well as awfulizing and low
frustration tolerance (LFT) derivatives (Cristea et al., 2015). ‘Intolerance of uncertainty’ (IOU) is a central feature in Dugas and Robichaud’s (2007) cognitive model
and treatment manual for GAD. They state that GAD sufferers view uncertainty as
stressful and upsetting, see uncertain future events as unfair, attempt to avoid
unexpected events, and believe uncertainty can interfere with one’s ability to
function. Theoretically, these concepts are similar to the REBT ‘demands’ that
would be a target of intervention within this disorder (i.e., ‘I need to know I’ll be
able to support my family’) and associated derivatives (i.e., ‘It’s horrible that I do
not know, I can’t stand not knowing, the uncertainty of life is unfair’).
Both CT and REBT help clients to relieve their ego anxiety (self-deprecation
when one acts or performs less than ideal or is disapproved of ). The concept of
discomfort anxiety, or low frustration tolerance (LFT), however, is specific to REBT,
yet aspects of this concept can be seen within Dugas and Robichaud’s (2007)
intolerance of uncertainty model (IUM) of GAD. IUM is defined as negative beliefs
about uncertainty which impede an individual’s ability to effectively manage situations, resulting in ineffective problem solving and increased worry in an attempt to
prevent negative events from occurring (Bottesi et al., 2016). Discomfort anxiety, or
LFT, is a state that arises when a person decides or finds (1) their comfort is
threatened, (2) they should not have to experience such discomfort, and (3) it is
awful to feel such discomfort, as opposed to being merely uncomfortable (Ellis,
1978a, 1980). Ellis conceptualizes LFT as a key factor of poor life function that leads
to avoidance, stagnation, impulsivity, abandonment of goals, numbing pain in
unhealthy ways, and so on. When discomfort anxiety is evident, the pursuit on
behalf of the REBT therapist is to elicit and dispute beliefs that perpetuate it,
reconstructing beliefs to be in line with developing high frustration tolerance
(HFT), that is, hard to bear versus unbearable, uncomfortable versus intolerable,
unfortunate versus completely awful. Furthermore, Ellis claims that the healthy
individual, one who demonstrates emotional maturity and stability, will be successful in confronting frustration and proficient in the practice of delayed gratification
(responsible hedonism) (Ellis et al., 2009).
Ellis (1980, 1994) makes specific reference to REBT’s focus on helping clients
manage their discomfort anxiety and notes that therapists should also help patients
work on their ‘secondary symptoms of disturbance’ (and tertiary disturbance if
19
20
Jodie M. Paget
necessary), specifically, patient anxieties about anxiety. Thus, there are salient
points of the IUM put forward by Dugas and Robichaud (2007) that share aspects
of Ellis’s concept of discomfort anxiety, a feature that sets it apart from Beckian CT.
‘Clinical perfectionism’ has been identified as a concept within CBT and there
are many CBT manuals available that provide guidance for treatment.
‘Perfectionism’ is ubiquitous in the sense that people experience it to varying
degrees, as opposed to having or not having it, whereas clinical perfectionism can
result in functional impairment due to self-worth being contingent on achieving selfimposed high standards of performance (Egan et al., 2014). Similarly, REBT theory
provides a rationale for interventions aimed at changing rigid rules/demands to
more healthy standards of performance, and refraining from attaching one’s overall
worth to a specific domain (e.g., career). Ellis’s identification of demands is acknowledged by Egan et al. (2014) and they include an example of a completed thought
diary set out in an ABC format (see Appendix 2.2).
Whilst it is positive to see protocols emerging that further develop or refine
CBT interventions, there are aspects of these ‘new developments’ that share much
in common with the psychological interventions first put forward by Ellis. As
previously discussed, it is not uncommon for CBT nomenclature to be used inconsistently with CBT texts. In the example ABC provided by Egan et al. (2014), some
of the cognitions under the ‘beliefs’ section are not evaluative (e.g., ‘the whole
presentation was ruined, I may lose my job’) but are inferences/predictive NATs,
creating theoretically muddy waters. Moreover, it demonstrates how the ABC has
morphed into a format that at times inaccurately displays theoretical principles.
Protocol-Driven CBT in IAPT
Protocols provide therapists with an overview of evidenced-based treatment interventions and a session-by-session therapy plan for specific disorders, thus are well
suited to the psychological services of the UK National Health Service (NHS). In
2008, the English IAPT service began and from its inception was an impressive and
radical initiative aimed at providing government-funded evidence-based psychological treatments for adults with depression and anxiety disorders. The three
defining features of IAPT are: (1) evidence-based psychological therapies, (2)
routine outcome monitoring, and (3) regular outcomes-focused supervision. Its
current aim is to increase services to support 1.9 million people by 2024, an increase
of 380,000 patients (NHS England, 2019). In order to reach these targets, continued
recruitment of trainees is vital as well as the ongoing training, development, and
supervision of those working within the service. Unfortunately, according to the
British Association for Behavioural and Cognitive Psychotherapies (BABCP),
IAPT funding difficulties has resulted in a lack of training places, meaning fewer
therapists, long wait times, and ‘patchy’ provision for those who would benefit from
these services (BABCP, 2018).
Despite IAPT’s good intentions, the cuts imposed by funding Clinical
Commissioning Groups (CCGs) have resulted in session limits set below guidelines
The History and Philosophical Underpinnings of CBT
recommended by the National Institute for Health and Care Excellence (NICE)
(e.g., a maximum of six sessions despite guidelines suggesting 12–20). Roscoe (2019)
states that IAPT trainees describe a number of restrictions imposed on them by the
services in which they work, including a need to rigidly apply disorder-specific
protocols (EST protocols) despite this not always being appropriate, not being
allowed outside the therapy room to undertake in vivo exposures, and active
discouragement from talking about core beliefs due to fear of patient suicides.
Moreover, there is an increasing gap between recommended guidelines and what
services provide, resulting in sub-therapeutic doses of diluted CBT and culminating
in patients returning to therapy for multiple episodes of treatment with different
therapists (perhaps partly due to insufficient number of sessions and/or a lack of topup sessions having been offered from the outset). Roscoe further states that this
situation is likely to strengthen the misconception that CBT is a superficial
treatment.
When protocols are taught in isolation there is a danger that therapists, in
particular, IAPT therapists, are throwing interventions at ‘problems’ because
manuals advise them to, as opposed to using their own clinical judgement based
on a sound understanding of theory. Due to the short duration of courses and the
necessary adherence to IAPT curriculum, there is little time to teach core theoretical concepts in sufficient detail. Swiftly taught protocols might be less problematic if
newly qualified therapists had a sound understanding of core theory, were well
supported within service, and were reviewed for competency regularly and meaningfully by experienced and highly trained CBT supervisors. However, this is too
often not the case as highlighted by Isabel Clarke, a clinical psychologist within the
NHS, who states that it suits managers to regard CBT as a simple, technical fix that
can be applied by any of their existing staff, with minimal training and inadequate
supervision (Clarke, 2018).
The current level of clinical supervision provided by most services to qualified
IAPT therapists is one hour (per week) for 20–24 contact hours (per week).
Assuming this one hour were only used to discuss how to work with the most
difficult presentations (as opposed to being taken up with risk management,
referrals, etc.), does this workload/supervision ratio permit therapists time
to review sessions, and to appropriately prepare for supervision? Are IAPT
therapists consistently supervised by highly qualified, highly experienced, and
well-supervised supervisors? Even if the answer to these questions is ‘yes’, it
seems improbable that one hour per week is a sufficient amount of time to
adequately (and ethically) support fledgling therapists in their continued
clinical development.
The BABCP insists that newly qualified trainees complete one year of clinical
practice before granting full accreditation (BABCP, 2019). Currently, however,
newly qualified IAPT therapists are able to become BABCP-accredited cognitive
behavior psychotherapists, the same level of accreditation given to those who have
studied for many years outside IAPT training courses (i.e., master’s and doctorate
courses). This creates the opportunity for IAPT-trained therapists to leave the NHS
21
22
Jodie M. Paget
fairly quickly after training and set up private practices, despite limited CBT
knowledge, training, and experience. This constitutes a concern for both the reputation of CBT and the NHS staff retention rate.
Considerations
If funding and/or a dire need for qualified therapists imposes brevity of training,
high caseloads, and minimal supervision, then the selection criteria need to be
robust enough to ensure that prospective trainees are well suited to post-graduate
academic study and demonstrate characteristics suitable for a CBT therapist (i.e., a
scientific and curious mind and the ability to apply CBT theory and practice to
themselves).
It is evident that some of the new developments within CBT have evolved from
Ellis’s original model of psychological disturbance. Yet despite the importance and
contribution of REBT, its core principles are all too frequently reproduced inaccurately or are diluted versions of what is a sound psychological principle. In an age
where the need for instant gratification has never been more present, and selfesteem is so fragile one must be given an award just for showing up, perhaps it is
time to focus on increasing one’s discomfort anxiety in addition to fostering healthy
self-esteem. When attempting to advance or build on CBT principles, it might be
sensible to resurrect such early principles in their entirety, give credence to where
they came from, and work on what adaptations need to be made for current
presentations and future developments.
RECOMMENDED READING
David, D. (2014). Rational emotive behavior therapy. Oxford University Press.
Dryden, W. (Ed.). (2012). Cognitive behaviour therapies. Sage.
Ellis, A. (1994). Reason and emotion in psychotherapy: A comprehensive method of treating
human disturbances. Citadel.
Robertson, D. (2018). The philosophy of cognitive-behavioural therapy (CBT): Stoic philosophy as rational and cognitive psychotherapy. Routledge.
REFERENCES
Abelson, R. P., & Rosenberg, M. J. (1958). Symbolic psycho-logic: A model of attitudinal
cognition. Behavioural Science, 3(1), 1–13.
Addis, M. E., Cardemil, E. V., Duncan, B. L., & Miller, S. D. (2006). Does manualization
improve therapy outcomes? In J. C. Norcross, L. E. Beutler, & R. F. Levant (Eds.),
Evidence-based practices in mental health: Debate and dialogue on the fundamental
questions (pp. 131–160). American Psychological Association.
Arch, J. J., & Craske, M. G. (2008). Acceptance and commitment therapy and cognitive
behavioural therapy for anxiety disorders: Different treatments, similar mechanisms?
Clinical Psychology: Science and Practice, 15(4), 263–279.
BABCP. (2018). IAPT therapist funding and stated mental health policy. www.babcp.com/
About/Press/IAPT-therapist-funding-and-stated-mental-health-policy.aspx
(2019). Accreditation. www.babcp.com/Accreditation/Accreditation.aspx
The History and Philosophical Underpinnings of CBT
Bandura, A. (1969). Principles of behaviour modification. Holt, Rinehart and Winston.
(1977). Social learning theory. Prentice-Hall.
Barlow, D. H. (1988). Anxiety and its disorders. Guilford Press.
Beck, A. T. (1967). Depression: Clinical, experimental and theoretical aspects. Hoeber Medical
Division.
(1970). Depression: Causes and treatment. University of Pennsylvania Press.
(1991). Cognitive therapy and the emotional disorders. Penguin.
Beck, A. T., Brown, G. K., & Steer, R. A. (1996). Beck Depression Inventory®-II (BDI®-II).
www.pearsonassessments.com/
Beck, A. T., Rush, J. A., Shaw, B. F., & Emery, G. (1979). Cognitive therapy of depression.
Guilford Press.
Beck, A. T., & Steer, R. A. (1993a). Beck Anxiety Inventory® (BAI®). www.pearsonassess
ments.com/
(1993b). Beck Hopelessness Scale® (BHS®). www.pearsonassessments.com/
Beck, J. (2011). Cognitive behavior therapy: Basics and beyond. Guilford Press.
Biglan, A., & Hayes, S. C. (1996). Should the behavioral sciences become more pragmatic?
The case for functional contextualism in research on human behaviour. Applied and
Preventive Psychology, 5(1), 47–57.
Bottesi, G., Ghisi, M., Carraro, E., Barclay, N., Payne, R., & Freeston, M. H. (2016). Revising
the intolerance of uncertainty model of generalized anxiety disorder: Evidence from UK
and Italian undergraduate samples. Frontiers in Psychology, 7, 1723.
Clark, D. M. (1986). A cognitive approach to panic. Behaviour Research Therapy, 24(4),
461–470.
Clarke, I. (2018). The case for CBT: A practical perspective from the NHS frontline. In D.
Loewenthal & G. Proctor (Eds.), Why not CBT? Against and for CBT revisited (pp.
362–373). PCCS Books.
Cristea, I. A., Stefan, S., David, O., Mogoase, C., & Dobrean, A. (2015). REBT in the
treatment of anxiety disorders in children and adults. Springer International Publishing.
David, D. (2014). Rational emotive behavior therapy. Oxford University Press.
David, D., & Hofmann, S. (2013). Another error of Descartes? Implications for the ‘third
wave’ cognitive-behavioural therapy. Journal of Cognitive and Behavioral
Psychotherapies, 13(1), 111–121.
David, D., & Montgomery, G. H. (2011). The scientific status of psychotherapies: A new
evaluative framework for evidence-based psychosocial interventions. Clinical
Psychology: Science and Practice, 18(2), 89–99.
David, D., Szentagotai, A., Eva, K., & Macavei, B. (2005). A synopsis of rational-emotive
behaviour therapy (REBT): Fundamental and applied research. Journal of RationalEmotive & Cognitive-Behaviour Therapy, 23(3), 175–221.
DiGiuseppe, R. (1991). Comprehensive cognitive disputing in RET. In M. Bernard (Ed.),
Using rational-emotive therapy effectively: A practitioner’s guide (pp. 173–195). Plenum.
DiGiuseppe, R., & Linscott, J. (1993). Philosophical differences among cognitive behavioral
therapists: Rationalism, constructivism, or both? Journal of Cognitive Psychotherapy, 7
(2), 117–130.
DiGiuseppe, R., & Tafrate, R. C. (2010). Understanding anger disorders. Oxford University
Press.
Dryden, W. (1987). Counselling individuals: The rational emotive approach. Taylor and
Francis.
(1993). Overcoming anger: When anger helps and when it hurts. Sheldon.
(1994). Overcoming guilt. Sheldon.
(1997). Overcoming shame. Sheldon Press.
(2005). Overcoming jealously. Sheldon Press.
23
24
Jodie M. Paget
(2007). Overcoming hurt. Sheldon.
(2012a). Rational emotive behaviour therapy. In W. Dryden (Ed.), Cognitive behaviour
therapies (pp. 189–215). Sage.
(2012b). The ‘ABCs’ of REBT I: A preliminary study of errors and confusions in counselling and psychotherapy textbooks. Journal of Rational-Emotive & Cognitive-Behaviour
Therapy, 30(3), 133–172.
(2013a). The ABCs of REBT revisited: Perspectives on conceptualization. Springer.
(2013b). Unconditional self-acceptance and self-compassion. In M. Bernard (Ed.), The
strength of self-acceptance: Theory, practice and research (pp. 107–120). Springer.
Dryden, W., & Branch, R. (2008). The fundamentals of rational emotive behaviour therapy:
A training handbook (2nd ed.). Wiley.
Dugas, M., & Robichaud, M. (2007). Cognitive behavioural treatment for generalised anxiety
disorder: From science to practice. Routledge.
Egan, S. J., Wade, T. D., Shafran, R., & Antony, M. M. (2014). Cognitive behavioural
treatment of perfectionism. Guilford Press.
Ellis, A. (1958). Rational psychotherapy. The Journal of General Psychology, 59(1), 35–49.
(1962). Reason and emotion in psychotherapy. Citadel Press.
(1978a). Discomfort anxiety: A new cognitive-behavioral construct. BMA Audiotapes.
(1978b). Rational-emotive theory. In R. J. Corsini (Ed.), Readings in current personality
theories (pp. 297–311). F. E. Peacock.
(1980). Rational-emotive therapy and cognitive behaviour therapy: Similarities and differences. Cognitive Therapy and Research, 4(4), 325–340.
(1993). Better, deeper, and more enduring brief therapy: The rational emotive behaviour
therapy approach. Routledge.
(1994). Reason and emotion in psychotherapy: A comprehensive method of treating human
disturbances. Citadel.
(2001). Behaviour psychotherapy: Rational and emotive. In N. J. Smelser & P. B. Baltes
(Eds.), International encyclopaedia of the social and behavioural sciences (pp.
1072–1076). Pergamon.
(2005a). Discussion of Christine A. Padesky and Aaron T. Beck, ‘Science and Philosophy:
Comparison of Cognitive Therapy and Rational Emotive Behaviour Therapy’. Journal
of Cognitive Psychotherapy, 19(2), 181–185.
(2005b). The myth of self-esteem: How rational emotive behaviour therapy can change your
life forever. Prometheus Books.
(2007). Unconditionally accepting yourself. Albert Ellis Institute.
(2010). A guide to rational living [Film]. Thinking Allowed Productions.
Ellis, A., Abrams, M., & Abrams, L. (2009). Personality theories: Critical perspectives. Sage.
Ellis, A., Sichel, J. L., Yeager, R. J., Dimattia, D. J., & DiGiuseppe, R. (1989). Rationalemotive couples therapy. Pergamon Press.
Fennell, M., & Jenkins, H. (2010). Low self-esteem. In J. Bennett-Levy et al. (Eds.), Oxford
guide to behaviour experiments in cognitive therapy (pp. 413–430). Oxford University
Press.
Foa, E. B., Hembree, E. A., & Rothbaum, B. O. (2007). Prolonged exposure therapy for
PTSD: Emotional processing of traumatic experiences. Therapist Guide. Oxford
University Press.
Graver, M. R. (2007). Stoicism and emotion. University of Chicago Press.
Greenberger, D., Padesky, C. A., & Beck, A. (2015). Mind over mood: Change how you feel
by changing the way you think (2nd ed.). Guilford Press.
Hayes, S. C. (2002). Buddhism and acceptance and commitment therapy. Cognitive and
Behavioural Practice, 9(1), 58–66.
The History and Philosophical Underpinnings of CBT
(2004). Acceptance and commitment therapy, relational frame theory, and the third wave
of behavioural and cognitive therapies. Behaviour Therapy, 35(4), 639–665.
Hayes, S. C., & Hofmann, S. G. (2017). The third wave of cognitive behavioural therapy and
the rise of process-based care. World Psychiatry, 16(3), 245–246.
Hayes, S. C., Luoma, J. B., Bond, F. W., Masuda, A., & Lillis, J. (2006). Acceptance and
commitment therapy: Model, processes and outcomes. Behaviour Research and Therapy,
44(1), 1–25.
Hayes, S. C., Strosahl, K. D., & Wilson, K. G. (2012). Acceptance and commitment therapy:
The process and practice of mindful change (2nd ed.). Guilford Press.
Hofmann, S. G., Asnaani, A., Vonk, I. J. J., Sawyer, A. T., & Fang, A. (2012). The efficacy of
cognitive behavioural therapy: A review of meta-analyses. Cognitive Therapy and
Research, 36(5), 427–440.
Hope, D. A., Heimberg, R. G., & Turk, C. L. (2010). Managing social anxiety: A cognitive
behavioural therapy approach. Therapist Guide. Oxford University Press.
Huber, C. H., & Baruth, L. G. (1989). Rational-emotive family therapy: A systems perspective.
Springer.
Hyland, P., & Boduszek, D. (2012). Resolving a difference between cognitive therapy and
rational emotive behaviour therapy: Towards the development of an integrated CBT
model of psychopathology. Mental Health Review Journal, 17(2), 104–116.
Johnsen, T. J., & Friborg, O. (2015). The effects of cognitive behavioural therapy as an
anti-depressive treatment is falling: A meta-analysis. Psychological Bulletin, 141(4),
747–768.
Korzybski, A. (1958). Science and sanity: An introduction to non-Aristotelian systems and
general semantic. International Non-Aristotelian Library Publishing Company.
Kuyken, W., Padesky, C. A., & Dudley, R. (2009). Collaborative case conceptualisation:
Working effectively with clients in cognitive-behavioural therapy. Guilford Press.
Lazarus, R. (1991). Emotion and adaptation. Oxford University Press.
Linehan, M. (1993). Cognitive-behavioural treatment of borderline personality disorder.
Guilford Press.
Malkinson, R. (2011). REBT: Past, present and future. Journal of Rational-Emotive &
Cognitive-Behavior Therapy, 29(4), 257–262.
Mansell, W., Carey, T. A., & Tai, S. J. (2012). A transdiagnostic approach to CBT using
method of levels therapy: Distinctive features. Routledge.
Marks, S. (2012). CBT in Britain: Historical and present. In W. Dryden (Ed.), Cognitive
behaviour therapies (pp. 1–24). Sage.
Morris, E., & Oliver, J. (2012). Acceptance and commitment therapy. In W. Dryden (Ed.),
Cognitive behaviour therapies (pp. 70–92). Sage.
Neenan, M., & Dryden, W. (1999). Rational emotive behaviour therapy: Advances in theory
and practice. Whurr.
Neff, K. (2003). Self-compassion: An alternative conceptualization of a healthy attitude
toward oneself. Self and Identity, 2(2), 85–101.
(2009). Self-compassion. In M. R. Leary & R. H. Hoyle (Eds.), Handbook of individual
differences in social behaviour (pp. 561–573). Guilford Press.
Neff, K. D., & Lamb, L. M. (2009). Self-compassion. In S. Lopez (Ed.), The encyclopedia of
positive psychology (pp. 864–867). Blackwell.
NHS England. (2019). Adult improving access to psychological therapies programme. www
.england.nhs.uk/mental-health/adults/iapt/
Padesky, C. A., & Beck, A. T. (2003). Science and philosophy: Comparison of cognitive
therapy and rational emotive behaviour therapy. Journal of Cognitive Psychotherapy,
17(3), 211–224.
25
26
Jodie M. Paget
(2005). Response to Ellis’ discussion of ‘Science and Philosophy: Comparison of Cognitive
Therapy and Rational Emotive Behaviour Therapy’. Journal of Cognitive
Psychotherapy, 19(2), 187–192.
Pavlov, I. P. (1955). Selected works. Foreign Languages Publishing House.
Persons, J. B. (2006). Case formulation-driven psychotherapy. Clinical Psychology: Science
and Practice, 13(2), 167–170.
Rachman, S. (2015). The evolution of behaviour therapy and cognitive behaviour therapy.
Behaviour Research and Therapy, 64, 1–8.
Rachman, S., Cobb, J., Grey, S., McDonald, B., Mawson, D., Sartory, G., & Stern, R. (1979).
The behavioural treatment of obsessional-compulsive disorders, with and without clomipramine. Behaviour Research and Therapy, 17(5), 467–478.
Robertson, D. J. (2018). The philosophy of cognitive therapy (CBT): Stoic philosophy as
rational and cognitive psychotherapy. Routledge.
Roscoe, J. (2019, February). Has IAPT become a bit like Frankenstein’s monster? CBT
Today.
Segal, Z. V., Williams, J. M. G., & Teasdale, J. D. (2001). Mindfulness-based cognitive therapy
for depression: A new approach to preventing relapse. Guilford Press.
Sellars, J. (2010). Stoicism. Acumen.
Skinner, V., & Wrycraft, N. (2014). CBT fundamentals: Theory and cases. McGraw-Hill.
Smith, C. A. (1982). Trends in counselling and psychotherapy. American Psychologists, 37,
802–809.
Stockdale, J. B. (1995). Thoughts of a philosophical fighter pilot. Hoover Institution Press.
Warren, J. (2009). The Cambridge companion to epicureanism. Cambridge University Press.
Wells, A. (2009). Metacognitive therapy for anxiety and depression. Guilford Press.
Westbrook, D., Kennerley, H. & Kirk, J. (2012). An introduction to cognitive behaviour
therapy: Skills and applications. Sage.
Wilding, C., & Palmer, S. (2010). Teach yourself: Beat low self-esteem with CBT. Hodder.
Wolpe, J. (1958). Psychotherapy by reciprocal inhibition. Stanford University Press.
Yankura, J., & Dryden, W. (1994). Albert Ellis (Key Figures in Counselling and
Psychotherapy Series). Sage.
PART ONE
COGNITIVE BEHAVIORAL
THERAPY FOR ANXIETY
DISORDERS
3
CBT for Specific Phobias
Lina A. Gega and Barry J. D. Wright
Introduction to Specific Phobias
Fear takes centre stage in phobias. It is an emotion fuelled by our body’s ‘fight-orflight’ response to a threat, whether it is a sabre-toothed cat in the Ice Age or an
enclosed windowless office in the Digital Age. Our feared consequences may have
evolved – from ‘Will I eat today, or will I be eaten?’ to ‘Will I escape from this
office?’ – but the fear cycle remains the same: we spot a threat, we feel anxious, we
run away from it, or we use tactics to keep ourselves safe, and we learn that we have
escaped once more, till the next time – and the cycle of fear goes on.
Diagnosis
Fear has an important role in keeping us safe, and although fear is here to protect us
from harm and prepare for danger, disproportionally extreme fear can become
harmful because it interferes with people’s lives and with their mental well-being.
For a diagnosis of specific phobia according to the DSM-5 (First et al., 2015) the
following criteria must be met over a minimum period of six months:
1.
2.
3.
4.
Marked fear of a specific object or situation
Exposure to the specific object or situation leading to marked anxiety
Avoidance of the feared object or situation whenever possible
Impact on functioning or daily life.
There are the following five diagnostic subtypes of specific phobias:
1. Living creature type: fear of dogs, snakes, spiders, mice, birds, insects, or other
creatures
2. Natural environment type: fear of phenomena such as water or storms or of
heights
3. Blood-injection-injury type (BII): fear of seeing blood, receiving an injection, or
watching or receiving invasive dental or medical procedures
4. Situational type: fear of flying, tunnels, bridges, elevators, or enclosed spaces
29
30
Lina A. Gega and Barry J. D. Wright
5. Other type: fears that do not fit into one of the other categories, such as fear of
choking or vomiting, of inanimate objects (e.g., buttons), or of specific people
(e.g., clowns, costumed characters).
The Curious Case of Blood-Injury-Injection (BII) Phobia
Blood-injury-injection (BII) phobia is characterized by intense fear when thinking
about or seeing blood, needles, or medical procedures. Individuals tend to avoid any
situation connected with blood, for example, going to the dentist or the doctor,
watching films with scenes of blood or injury. The sight of blood can cause someone
with BII phobia to faint, something which happens very rarely with other types of
phobias. The physiological mechanism behind fainting in BII phobia, known as
vasovagal syncope, is due to the activation of the parasympathetic nervous system
(some theories link this with disgust), which causes the blood pressure to drop and
the pulse to slow down (for a review, see Ayala et al., 2009). This is very different
from other phobias where the heart rate increases. Vasovagal syncope has implications for CBT as it often necessitates the use of applied tension to increase
blood pressure and prevent fainting during treatment (discussed later in the
chapter). Case example 1 gives a typical picture of BII phobia.
Differential Diagnosis
To be certain of a diagnosis of a specific phobia, the fear (or the avoidance
and safety behaviors associated with the fear) must not be better explained by
another condition, especially agoraphobia/panic disorder and social phobia, or
obsessive-compulsive disorder (OCD) and posttraumatic stress disorder (PTSD).
The key differentiator between specific phobias and other conditions is the feared
consequences (e.g., that the worst thing the individual fears may happen
CASE EXAMPLE 1: CHRIS (BII PHOBIA)
Chris is a 47-year-old woman who has been recently diagnosed with multiple sclerosis
(MS) and needs to start on injectable medication, which she will either administer
herself daily or have done weekly by a nurse. She currently refuses the medication
because of an extreme fear of injections: she faints as the needle approaches her arm,
or she gets so anxious that she starts trembling and has muscle spasms. She is worried
about the pain that the injections may cause. She is less worried about watching other
people having injections except at times that it looks painful or when people wince
with a sharp pain. She also avoids seeing the doctor or dentist, and she avoids watching
medical dramas on the TV. On previous occasions when she needed to have an
injection, she managed her fear by distraction, by either holding her breath or digging
her nails into her palms. She pretended that her arm was an orange and that she was
someone else observing this (almost like a feeling of depersonalization). She is more
fearful of having an injection than having blood taken, because she says it is the idea of
having something injected into her body that makes her worried.
CBT for Specific Phobias
CASE EXAMPLE 2: MARY (VOMIT PHOBIA)
Mary is a 20-year-old woman with fear of vomiting. She sought treatment because her
phobia is now restricting her life at the university; for example, she avoids socializing
because her fellow students often get drunk on a night out and vomit. Also, she
cannot eat food from the student canteen as it is served in open booths that she fears
may have been exposed to germs. She avoids watching medical TV series in case
someone vomits. She is vigilant for signs of someone being unwell (e.g., looking
clammy, pale, and complaining of feeling sick). When she and her family or friends
go out to restaurants, she never orders chicken, meat, or dairy products because she
worries about contracting gastroenteritis or getting other bacterial infections. She
washes her hands several times before she eats; if her fingers or anything else touch
her food (e.g., a wrapped sandwich on a plate), then she discards it. She never eats
until she is full when she is in a public place (e.g., she leaves half of her meal at a
restaurant) because she worries that if she vomits, the more food she has in her
stomach, the more horrible it will be; in fact, she has an image in her mind of her
‘projectile vomiting’ on the table and everyone looking at her with disgust. When
asked about what the worst thing is that she fears may happen, she says that it would
be so awful to vomit or watch someone else vomit that she will be traumatized, and
that this is not worth the risk. She remembers when her little brother was violently
sick with gastroenteritis and he nearly choked while vomiting.
in certain situations). Let’s take the example of Mary, who has vomit phobia, in case
example 2.
In case example 2, could the diagnosis for Mary be anything other than vomit
phobia? How could we make sense of her feared consequences to help us exclude
other diagnoses? For example, could it be social anxiety (fear of embarrassment),
health anxiety (fear of becoming ill), PTSD (recalling a traumatic incident), or OCD
(fear of contamination, repeated hand washing, and not touching food)? In Mary’s
case, it is most likely that her fear, avoidance, and safety behaviors are due to vomit
phobia because the overriding feared consequence is the ‘awfulness’ of vomiting
itself. Social anxiety would have been likely if Mary was worried about vomiting
only when she was in a public place, but not worried if she were at home. If Mary’s
fear was about becoming ill with conditions involving vomiting (e.g., cancer, heart
conditions, sexually transmitted infections), then health anxiety might have been an
alternative diagnosis. Finally, if she was worried about contamination from things
other than food (e.g., using public bathrooms, touching doors, sitting on public
transport), then a diagnosis of OCD also could have been plausible.
Background on CBT for Specific Phobias
Behavior therapy, the earliest form of CBT, was introduced when Freudian psychotherapy dominated psychiatry, in the 1950s. While Freudian analysis sought to
31
32
Lina A. Gega and Barry J. D. Wright
probe the unconscious roots of a phobia, as in the case of Little Hans (a boy who
had a fear of horses; Freud, 1909), behavior therapy viewed fear as a learned
association (classical conditioning) between a stimulus (the horse) and a response
(fear). If Little Hans had received behavior therapy at the time, the strategy would
have been to pair the phobic stimulus with a pleasant stimulus (e.g., food) to
‘counter-condition’ the fear response with a more neutral or pleasant response.
This was the basis for ‘systematic desensitization’ (Wolpe, 1961), the first CBT
variant for specific phobias.
Systematic desensitization has evolved into what is now known as ‘exposure
therapy’ (established widely by Marks, 1997), in which an individual confronts their
feared situation until fear subsides over time (‘habituation’). Through repeated experiences of the feared situations, the association between the stimulus and the fear
response weakens until it disappears (‘extinction’). During exposure therapy, the
individual must remain focused on the physical and emotional manifestations of their
fear (e.g., focus on the heart pounding) and must refrain from engaging in ‘safety
behaviors’ to mask or reduce fear (e.g., seeking reassurance or trying to relax).
Exposure therapy does not try to counteract a person’s fear responses, as systematic
desensitization does; instead, fear in exposure therapy is regulated by grading the
feared situations from the least to the most feared, thereby generating a ‘fear hierarchy’. The only meta-analysis available to date of randomized controlled trials on
specific phobia treatments (Wolitzky-Taylor et al., 2008) concluded that exposurebased treatments have large effects relative to no treatment, and they also outperform
placebo conditions and non-exposure treatment. Still, the same meta-analysis found
that non-exposure treatments can have a large effect compared with no treatment.
The advent of cognitive therapy in the 1970s (Beck, 1976) influenced CBT for
phobias by highlighting that exaggerated negative predictions and unhelpful beliefs
about the feared situation fuel fear responses (emotional and physical) and drive safety
behaviors. These predictions and beliefs relate to how likely, catastrophic, or awful the
consequences of a feared situation are expected to be and how limited the individual’s
ability will be to cope with these (‘feared consequences’). Examples of feared consequences in specific phobias relate to coping (‘It will be too much to deal with’), harm (‘I
may choke and die’); emotional consequences (‘I hate being out of control of my
feelings,’ ‘Vomiting is too awful’), sensory consequences (‘The injection will be painful,’
‘I hate the big furry body of spider, it makes me cringe and feels like something is
crawling on me’), and social consequences (‘If I vomit, people will think am disgusting’).
While traditional exposure therapy uses habituation and extinction to reduce
fear, cognitive techniques use exposure-based behavioral experiments (BennettLevy et al., 2004) to test key negative predictions that are central to the maintenance
of the phobia and to generate and reinforce more realistic beliefs about the feared
consequences. These exposure-based behavioral experiments involve some incremental experiences of the feared situation to facilitate new learning and revision of
exaggerated predictions; however, these experiments do not need to be repeated or
prolonged to achieve habituation and extinction as in traditional exposure therapy.
Wolitzky-Taylor et al.’s (2008) meta-analysis of randomized controlled trials on
specific phobia treatments drew on five studies that were available at the time to
CBT for Specific Phobias
compare exposure plus cognitive techniques compared with exposure alone. They
concluded that ‘contrary to prediction, exposure augmented with cognitive procedures did not outperform exposure treatment alone. The overall composite effect size
was not significant, and comparisons of the two treatments for each assessment
domain separately revealed no significant advantage for combining exposure with
cognitive techniques’ (p. 1031).
CBT Assessment for Specific Phobias
Assessment Interview
A good starting point for a phobia-specific CBT assessment is to explore a recent
occasion when an individual experienced extreme fear. The therapist can start by
asking the individual to describe this occasion (what happened, what made them feel
scared) and what their particular emotional and physical responses were. For example,
some people commonly describe fear with expressions such as ‘being terrified’ or
‘freaking out’, whereas others experience ‘disgust’, feel worried or uncomfortable, or
feel like fainting in response to fear. It is important to understand how fear manifests
within an individual and thereafter use their own words while referring to their fear
responses during formulation and treatment. Other than a recent incident, the therapist
can also ask about the first remembered incident or the most severe episode, to form a
picture about the phobia’s development and fluctuations over time.
A clinical assessment should gather information about an individual’s general
mental state, risk factors, serious physical conditions, medication, and social circumstances. A CBT assessment for specific phobias should also gather information
about three important topics that relate to formulation and treatment: (1) triggers,
(2) feared consequences, and (3) avoidance and safety behaviors. Table 3.1 gives
examples of questions to assess each topic. When asking these questions, the
therapist gathers information about not only phobia-specific maintenance factors,
but also contextual factors that may influence the fear response are identified (e.g.,
with whom, where, and when it is better or worse) and any adaptive coping methods
(e.g., time management to cope with stress).
The last part of the assessment is about understanding the impact that the phobia
has on different domains of the individual’s life: studies/work, friends/family, hobbies,
perception of self, aspirations for the future. This will also be a stepping-stone for
setting the end-of-treatment goals, that is, statements about what a person would be
able to do and under what circumstances to know that their phobia has improved.
Questions that can help the therapist elicit such goals are the following:
• What made you decide to come for help now?
• Why is it important for you that you stop being afraid of/worry about . . .?
• What would you like to do in the future that you cannot do now because of your
phobia?
• If you woke up tomorrow and were 100% free of your fear/worry, what would
be the first thing that you like to do?
33
34
Lina A. Gega and Barry J. D. Wright
Table 3.1. Example of phobia-specific CBT assessment questions
Phobia-specific
information
5Ws
What
(triggers)
Example questions
•
•
•
•
•
•
•
•
•
Can you think of a recent incident when you felt really scared of . . . (e.g.,
heights)/worried about . . . (e.g., vomiting)?
Can you think back to the very first time that you felt extreme fear/worry
about . . . ?
Can you remember any time when you had the worst experience of . . . (e.g.,
seeing a spider, having an injection)?
Looking back, can you pick a situation that was particularly frightening/
overwhelming/stays vivid in your mind?
Could you tell me what it was and what makes this situation stand out in your
memory?
Could you tell me more about what happened?
Can you tell me how you felt at the time?
What was the first thing you noticed in your body when you started getting
worried?
What was the most intense feeling that you noticed in your body when you felt
extremely scared?
Where
(better/worse)
Are there any places where:
• the phobia gets better/worse?
• you find it easier to cope with the phobia?
• you feel safer/at ease?
• you feel more at ‘risk’ or ‘exposed’?
With Whom
(better/worse)
•
•
•
Are there any people that make the phobia better/worse?
Who is most helpful when you feel really scared/worried?
How do other people (friends, family) help you with the phobia?
When
(better/worse)
•
•
•
Do you remember any times when your phobia got worse?
Do you remember any times when your phobia got better?
Does your fear of/worry out . . . (e.g., enclosed spaces) change at all over the
course of the day/week/year?
Why
(feared consequences)
•
What is the worst thing you worry may happen if you had to face up to the
things you are most scared of?
What is the worst thing you fear may happen if you stopped yourself from
scanning the environment for the things that make you anxious/scared?
When you are in a situation where you feel anxious/scared, what sort of things
go through your mind?
•
•
Avoidance
•
•
•
•
•
Is there anything that you avoid doing because you are scared of/about . . . or
because it makes you uncomfortable/worried?
What does the phobia stop you from doing?
Are there activities you would like to do or places you would like to go or
people you would like to see, but you can’t because of your phobia/fear?
Is there anything than you don’t do as much as you would like to, because of
fear or worry?
Is there anything you used to do but now have stopped doing because you feel
scared?
CBT for Specific Phobias
35
Table 3.1. (cont.)
Phobia-specific
information
Example questions
•
•
Safety behaviors
•
•
•
•
If you had no fear or worry, what more would you have done differently in the
last week or would do differently each day?
If you woke up tomorrow and you were no longer scared of/about . . . (e.g.,
heights, vomiting), how could you family or friends tell? What would they
notice you doing differently in your day-to-day life?
Is there anything that you do more of/excessively because you are scared of/
worry about . . .?
What do you do when you come across (e.g. a spider)/when you find yourself in
a situation where (e.g., you have to have an injection), in order to cope?
Is there anything that you tell yourself or that you have to do discreetly/in your
head when you are faced with situations that trigger your phobia?
Is there anything that your family/friends do for you to help you cope with your
phobia?
Standardized Assessment Tools
Diagnostic Tools
The specific phobia subsections of the Structured Clinical Interview for DSM-5
(SCID-5; First et al., 2015) and the Anxiety and Related Disorders Interview
Schedule for DSM-5 (ADIS-5; Brown & Barlow, 2014) are considered to be the
gold standard for establishing a diagnosis through a clinical interview. These diagnostic tools can be used as outcome measures before and after treatment to establish whether an individual has ‘recovered’ from their phobia. The tools collect
information about the type of specific phobia (e.g., animal/insect, small spaces,
blood/injection phobias), the level of fear, the degree of avoidance, and the extent
of interference with daily life.
Phobia-Specific Self-Report Questionnaires
Standardized self-report questionnaires can measure treatment outcomes for individual specific phobias, such as the following:
Spider Phobia: Fear of Spiders Questionnaire (FSQ) and Spider Phobia
Questionnaire (SPQ) (Muris & Merckelbach, 1996)
Claustrophobia: Claustrophobia Scale (CS) (Öst et al., 2007), Claustrophobia
Questionnaire (CLQ) (Radomsky et al., 2001)
Fear of Flying: Flight Anxiety Situations (FAS) questionnaire and Flight
Anxiety Modality (FAM) questionnaire (Van Gerwen et al., 1999).
Fear of Heights: Heights Interpretation Questionnaire (HIQ) (Steinman &
Teachman, 2011)
BII Phobia: The Blood-Injection Symptom Scale (BISS) (Page et al., 1997)
36
Lina A. Gega and Barry J. D. Wright
Vomit Phobia: Emetophobia Questionnaire (EmetQ) (Boschen et al., 2013),
Specific Phobia of Vomiting Inventory (SPOVI) (Veale et al., 2013)
Generic Phobia Self-Report Questionnaires: Two seminal self-report questionnaires that assess fear and avoidance (including but not limited to specific
phobias) are the Fear Questionnaire (FQ) (Marks & Mathews, 1979) and the
Fear Survey Schedule (FSS) (Wolpe & Lang, 1964). Since their development
decades ago, they have been through multiple iterations, revisions, adaptations, and validations and have been used in hundreds of studies. A more
recently developed self-report measure, the Phobic Stimuli Response Scales
(PSRS) (Cutshall & Watson, 2004) assesses the cognitive and emotional
factors of five fears: social, animal, physical confinement, bodily harm, and
blood injection; however, like the FQ and the FSS, the PSRS does not focus
on specific phobias and its items do not correspond to the five specific phobia
diagnostic subtypes.
The only outcome measure designed for specific phobias that covers all subtypes is the recently validated Specific Phobia Questionnaire (SPQ) (Ovanessian
et al., 2019). The SPQ assesses the extent of fear and its interference with daily life
for a broad range of objects and situations. It consists of 43 items, each rated using a
five-point Likert scale on two dimensions – level of fear and interference with daily
life – with scores ranging from 0 (no fear/interference) to 4 (extreme fear/interference). The SPQ is scored by combining fear ratings of each specific phobia subtype
to create five different composite scores: animals, natural environment, situational,
blood-injection-injury, and other.
Behavioral Approach Task (BAT)
A behavioral approach task (BAT), also known as behavioral avoidance task/test, is
a measurement technique in which an assessor observes and rates how far an
individual can approach their feared object or enter their feared situation in successive steps. It starts with a simple instruction:
I’d like to understand how you react around [the stimulus, e.g., blood/needle, dog,
vomit]. What I would like you to do is to open the door, go inside, sit in the chair, and
stay until it’s time to leave after several minutes. However, you don’t have to do any
more of this than you want to. You don’t even have to open the door.
The number of steps is the main unit of measurement for the BAT; that is, scores
correspond to the steps that the individual completes, for example, 0 for the first step,
1 for the second, through to 10 for the 11th step. Subjective units of distress (SUDs)
are also included at the start and end of the BAT by asking: ‘Using an 8-point scale
where 0 is none and 8 is the most, how anxious/scared are you right now?’
The BAT has been introduced by Öst et al. (1991) and subsequently has been
widely used with adults (Vernon, 2007) and children (Silverman & Ollendick, 2005).
BATs need to be devised for different phobic stimuli (e.g., a spider phobia BAT is
CBT for Specific Phobias
37
Table 3.2. Behavioral approach task (BAT) examples
Score
BAT 1: Vomit phobia
BAT 2: Height phobia
0
1
2
3
Does not agree to proceed with BAT
Opens door
Enters room (passes through doorway)
Enters approximately ¼ way into room (about 3–4
feet from doorway)
Enters at least ½ way into room (about 6–7 feet
from doorway)
Enters at least 3/4 way into room (about 9–10 feet
from doorway)
Stands/comes within 3 feet of transparent container
with fake vomit
Stands/comes within 6 inches of container
Touches or holds container
Opens container
Sits down beside container for final step (20
seconds)
Does not agree to proceed with BAT
Presses button to call elevator
Enters elevator (passes through doorway)
Allows elevator doors to close while inside
4
5
6
7
8
9
10
Rides elevator to top floor
Exits elevator
Stands more than 6 feet away from railing
Stands more than 3 feet away from railing
Stands within 6 inches of railing
Looks down over railing for < 15 seconds
Looks down over railing without using
hands for 30 seconds
different to a claustrophobia BAT) and may vary in the number of steps included,
the way the steps are constructed (e.g., by grading the stimulus or the time spent
with the stimulus), the way the steps are scored (e.g., from 0 to 1, or from 0 to 100)
and how the instructions are given by the assessor. Table 3.2 illustrates two different
BATs for vomit phobia and for fear of heights.
CBT Formulation for Specific Phobias
Maintenance Cycle
A CBT mini-formulation is a way of demonstrating how a phobia is maintained
through an interaction between thoughts, feelings, and behaviors that are specific
and meaningful to that individual. The three-systems model (Figure 3.1) (introduced by Lang, 1968), although decades old, is a CBT icon that conveys aptly and
parsimoniously the ‘maintenance cycle’ of phobias. It does so by demonstrating the
links between feared consequences (thoughts), avoidance and safety behaviors, and
fear-associated feelings (emotions and physical symptoms). For example, Figure 3.2
illustrates the fear cycle for Chris in case study 1 (BII phobia). The added advantage
of a simple visual model is that it can be readily used to explain CBT to children for
whom specific phobias are common and debilitating, more so than in adults.
Working Hypothesis
The three-systems mini-formulation of a specific phobia is the stepping-stone for
CBT interventions, because it highlights that a change in any one corner of the
triangle can lead to changes in the other two. CBT aims to break the ‘maintenance
38
Lina A. Gega and Barry J. D. Wright
Fear response
(how or what we feel)
Emotions and Physical Symptoms
Behaviors
(what we do or avoid doing because of
fear)
Thoughts
(what we think may happen in a feared
situation)
Figure 3.1. Three-systems model for specific phobia.
Feelings
Feeling worried and scared, muscles tense
and have spasms
Behaviors
Avoid watching medical programs
Avoid doctor check-up
Digging nails in hands
Thoughts
‘I will faint’
‘ It will be very painful’
‘I won’t be able to cope’
Image of needle breaking the
skin
Figure 3.2. Fear cycle for BII phobia.
cycle’ of fear by creating a ‘alternative cycle’ in which avoidance is replaced by
graded experiences of the feared situation, safety behaviors are removed, and
feared consequences are replaced by new ideas about the feared situation. The
aim is to replace extreme fear with more neutral and proportionate emotional and
physical responses. The individual may never feel happy when they find themselves
face-to-face with a situation that they used to dread, but our aim is to replace fear
with boredom, and to change the feeling of dread to a sense of readiness
and confidence.
CBT for Specific Phobias
39
FEAR
Figure 3.3. CBT cycle for BII phobia.
TIME
Figure 3.4. Habituation and extinction.
Figure 3.3 illustrates an alternative cycle for case example 2 (Chris, BII phobia)
which includes confronting feared situations and reinforcing alternatives to her
feared consequences of pain and not being able to cope.
Socialization to the Model
Habituation and extinction are the proposed mechanisms that explain how graded
exposure works (Figure 3.4). Fear is the body’s natural response to excessive
adrenaline production when a person is faced with a situation which is threatening
or is perceived as threatening. Adrenaline prepares the body for a fight-or-flight
response. Fear increases until it reaches a point when the person cannot tolerate it
anymore, so the person runs away or uses safety behaviors to cope; by so doing, fear
goes down rapidly. As soon as the person comes across the same situation again,
fear reappears with the same or greater intensity. If the person remains in this
situation a little longer than usual, and most importantly passes the point when they
would normally run away or use safety behaviors, the excess adrenaline in the body
is gradually depleted and the anxiety symptoms eventually subside. Over repeated
and prolonged exposure to the same or similar situations, the peak fear brought on
by a situation reduces until it is no longer a feared situation.
40
Lina A. Gega and Barry J. D. Wright
Box 3.1 The Principles of Exposure Therapy
• Graded: Start from the easiest task and build up to the most dreaded one with
increments in between.
• Focused: Experience fully the physical and emotional aspects of fear without
trying to mask it or avoid it with safety behaviors.
• Repeated: Carry out the same exposure task until the peak fear decreases (due
to extinction).
• Prolonged: Stay with the fear long enough to allow habituation to take place.
CBT Methods for Specific Phobias
For specific phobias, treatment duration can be shortened by delivering a single threehour session (introduced by Öst, 1989) or with the use of digital media such as virtual
reality (Freeman et al., 2018). Irrespective of the duration of treatment or the form by
which it is delivered, CBT for specific phobias includes constructing a fear hierarchy,
dropping all safety behaviors, exposure practice, and cognitive restructuring. Figure 3.5
gives an overview of the CBT process from assessment to formulation and treatment.
Constructing a Fear Hierarchy
Constructing a fear hierarchy involves making a list of situations that can cause fear/
anxiety and attaching a rating of distress to each of them on a 0–10 Likert scale,
where 0 = not at all distressing and 10 = extremely distressing. The questions asked
during the assessment would have elicited some information to help the therapist
populate the hierarchy; however, the situations in the hierarchy have to be strategically selected to be directly linked to the feared stimulus and the feared consequence, so that they can be engineered and tested during the treatment process. For
example, in spider phobia, ‘sitting in the garden’ or ‘going into the shed’ are not
helpful in the fear hierarchy; instead, statements like ‘looking for spider webs in the
Assessment
Triggers
Feared
Avoidance and
safety behaviors
Impact and goals
Formulation
Treatment
Maintenance cycle
Construct fear
Working hypothesis
Drop safety behaviors
Socialization to model
Exposure practice
Cognitive restructuring
Figure 3.5. Overview of CBT process for specific phobias.
CBT for Specific Phobias
garden and the shed and putting my hand through these webs’ or ‘finding a small
spider in the garden, putting it in a glass jar and holding the jar’ are more specific
and directly linked to the stimulus (spider) and feared consequence (‘touching it’).
An easy way to start constructing a fear hierarchy is by using cards or an
standard-sized piece of paper cut into eighths. Each card describes a task that
involves entering a feared situation/approaching a feared object in various incremental steps. The increments may be of the feared object or of the exposure task.
For example, increments of a spider will be ‘small, big, dead, live, toy, picture’,
whereas increments of a task will be ‘handling the spider, handling a jar with a
spider in it, looking at a spider’. The following is how the relevant hierarchy would
look like (the number in parentheses is the predicted fear score):
1.
2.
3.
4.
5.
6.
7.
8.
9.
10.
Handle small realistic toy spiders. (3)
Look at a dead small spider. (4)
Handle a jar with a small dead spider inside. (4.5)
Handle a jar with a small live spider inside. (5)
Touch a small live spider with a short stick. (6)
Handle a small live spider. (6.5)
Handle a jar with a medium-sized dead spider inside. (7)
Handle a jar with a medium live spider inside. (8)
Touch a medium-sized live spider with a short stick. (9)
Handle a medium-sized live spider. (10)
The following is another example of a fear hierarchy for case study 1, Chris (BII
phobia), starting from the least difficult and building up to the final goal and most
dreaded task, with ratings of predicted anxiety for each task:
1. Handle a syringe without a needle attached and touching my arm with it. (3)
2. Handle a syringe with a needle attached and hold it against the inside of my
arm. (4)
3. Watch a nurse prepare all the injection instruments. (4)
4. Have a tourniquet strapped around my upper arm and watch my vein bulge. (5)
5. Watch a video of someone having an injection. (6.5)
6. Watch someone having an injection live. (7)
7. Give an injection to a ‘medical arm’ (a realistic dummy used by doctors to
practice injections). (8)
8. Have blood taken (needle draws something out rather than putting something
in). (9)
9. Have an injection done to me by a nurse. (9.5)
10. Give an injection to myself. (10)
Dropping Safety Behaviors
In cognitive behavioral therapy, safety behaviors are discouraged because the
individual may attribute the fact that their feared consequences did not happen
41
42
Lina A. Gega and Barry J. D. Wright
(or were not as bad as expected) to the safety-seeking behavior (Salkovskis, 1991),
rather than take it as evidence that their feared consequence was unlikely to happen
(at least to the expected severity) in the first place. In the context of learning
theories, using safety behaviors during exposure may get in the way of habituation
(because the person does not learn to tolerate distress) and of extinction (because
the person does not make new stimulus-response associations) (for a review, see
Blakey & Abramowitz, 2016).
Safety behaviors can be overt (physical, observable actions) or covert (mental
actions, often subtle, that can go unnoticed). Examples of overt safety behaviors
include asking for reassurance or for help from someone we trust, holding on to
someone or something that makes us feel safe, closing our eyes, or looking away.
Examples of covert safety behaviors include imagining that the phobic object is
something different (e.g., instead of our arm receiving an injection, pretending it is
an orange), having an internal dialogue that takes our focus away from the feared
situation (e.g., this is not real, we are dreaming), or counting or reciting things in our
minds to feel safe (e.g., a poem or a prayer).
When it comes to safety behaviors in CBT, it is important do three things: (1)
identify them, (2) understand how they maintain a phobia and how they can
obstruct therapy, and (3) eliminate them before, during, and after exposure.
Sometimes it is difficult for people to give up their safety behaviors. Children are
a good example of this, as they often hold on to objects that make them feel more
comfortable, such as a blanket or a teddy. Adolescents and adults hold on to their
mobile phone instead of a comfort blanket, or they bring along to therapy their
parent or partner instead of a teddy. This may work in the short term but may serve
as an ‘emotional crutch’ without which the individual cannot function.
We have to be careful not to unintentionally collude with safety behaviors; some
therapists may get this wrong by encouraging the graded use of safety behaviors. During
exposure therapy, our aim is to engineer an incremental approach to the feared situation
so that each increment can be completed without any safety behaviors. In case example
3 below (Kevin, fear of heights), a potential fear hierarchy could involve Kevin going up
a staircase, looking out of a window from a high building, or crossing a bridge.
Increments for ‘going up a staircase’ could be for Kevin to climb up one step and then
turn and walk back down, then climb up two steps, turn and walk back down, and so on.
Kevin needs to complete each step (climb up, turn, walk down) until he is ready to move
up. Each step needs to be taken as close to the railings as possible (to counteract the
safety behavior of walking in the middle of the staircase) but without holding on to the
railings (we can suggest that Kevin carries two cups of water while going up the stairs,
which makes it impossible to hold on to the railings). Similarly, Kevin can take one step
closer to a window or across a bridge and describe how far down is the ‘drop’ he can see
(the further he is from the window, the less ‘drop’ he can see), and stay there until his
anxiety subsides. The increment here is the distance travelled towards the window or
across the bridge, but also the sense of ‘drop’ while looking down.
Rachman et al. (2008) introduced the concept of ‘judicious use of safety behaviors’ to overcome obstacles and improve the acceptability and tolerance of exposure
CBT for Specific Phobias
CASE EXAMPLE 3: KEVIN (FEAR OF HEIGHTS)
Kevin is a 19-year-old man with height phobia. He does not use open escalators
while he is in a shopping mall, and although he can use a closed elevator, he will not
go into a glass one. When he has to walk up the stairs, he would either walk in the
centre of the staircase or on the side, holding tightly onto the railings. Kevin avoids
going into any building taller than two floors, and if he has to approach a window, he
never looks down. He refuses to go walking with his family as this involves going
over some hills and bridges in the nearby countryside. When he has to approach any
‘edge’ with a drop beneath it, he freezes and starts shaking. The latest incident
happened at a train station when he would not cross the bridge above the platforms
to get to a train. When he is asked what the worst thing is that he imagines may
happen if he stands at the edge of a cliff or a building or a bridge, he says that he is
worried he will fall. When asked why he will not go into a glass elevator or look out
of a closed window from a tall building, despite having a barrier that protects him
from falling, he says that he feels dizzy and wants to avoid this unpleasant sensation.
therapy. Hood et al. (2010) took this further to suggest that safety behaviors could in
fact enhance exposure therapy because they can expedite progression through the
fear hierarchy and enable people to ‘go the extra mile’ with their final exposure task.
Some authors argue strongly for the identification of and eradication of unhelpful
safety behaviors (Helbig-Lang & Petermann, 2010). A recent high-quality randomized controlled trial in specific phobias did not find any difference between abstaining
completely from safety behaviors or introducing them at first and then systematically
fading them out over the course of multiple exposure sessions (Blakey et al., 2019).
In summary, empirical findings are mixed as to whether safety behaviors are
good, bad, or indifferent when it comes to exposure therapy: their effect seems to
depend on how they are used. Safety behaviors tend to have a positive or neutral
effect when used selectively for some people at the beginning of exposure, but they
are counterproductive when used indiscriminately and throughout exposure. In
practice, it is important to weigh both the positive and negative consequences of
encouraging the use of safety behaviors. As a rule of thumb, safety behaviors should
be dropped at the first opportunity, but their strategic use at the earlier stages of
exposure therapy may be helpful. Sometimes it is helpful to carry out behavioral
experiments to demonstrate that safety behaviors maintain fear and that these
behaviors can become obsolete as the person confirms their fear, as in case example
4 (Susan, claustrophobia and the ‘comforting pendant’).
Exposure Practice
Repeated practice of an exposure task needs to take place as many times as needed
until the task is performed with relative ease. During each exposure task, we
monitor an individual’s distress/fear by asking, ‘On a scale from 0 to 10, how
43
44
Lina A. Gega and Barry J. D. Wright
CASE EXAMPLE 4: SUSAN (CLAUSTROPHOBIA AND THE ‘COMFORTING PENDANT’)
Susan is a 22-year-old woman with claustrophobia. She does not remember how
her fear of being trapped somewhere and suffocating started, but she remembers
that even as a child she would never lock the toilet door, nor would she sleep on
the ‘inside’ of the bed when she had to share a bed with her sister. Her family and
friends know about her phobia and accommodate it; for example, they do not use
elevators but use the stairs instead when they go to a shopping mall; they also let
Susan sit near the door when they are in a new place (e.g., a restaurant) or when
using public transport. Susan wears a pendant with a religious symbol on it that
gives her a sense of protection if she feels trapped. She always has to wear it
when she goes out and has to take public transport. When she feels trapped, she
will hold and stroke the pendant between her fingers. During therapy we discussed what would happen if she forgot her pendant at home or lost it. We also
talked about how she would feel if she managed to stay in an enclosed space
without wearing her pendant. When we constructed the fear hierarchy, we agreed
that Susan would do the first few exposure tasks first with the pendant and then
without it. Before the exposure task, Susan’s anticipatory anxiety was higher
when she did not wear the pendant than when she did. In both occasions,
Susan’s anxiety faded away anyway after the exposure task, with or without the
pendant. Although the pendant helped her start exposure therapy, once we were
able to move up the hierarchy, Susan realized that the pendant did not really
make a difference, so she no longer needed it.
uncomfortable/worried/scared do you feel at the moment?’ Distress and discomfort
are part of exposure, and although they are unpleasant, they are not harmful.
However, discomfort should not be so great as to make exposure practice traumatic
or disheartening if the individual cannot achieve the task and considers they have
failed. The pace of exposure should be negotiated with the individual who carries it
out; tasks are fine-tuned throughout the exposure practice and do not have to
adhere strictly to the fear hierarchy.
An important aspect of exposure is modelling. Once we describe the exposure step (what to do and how), then we show the exposure task without the
individual participating, then we do it together with the individual, and finally we
encourage them to do it independently. Positive reinforcement should be specific
and explicit during each step; for example, not only saying, ‘Well done’, but
saying, ‘Well done for moving two steps forward towards this jar of vomit’ or
‘Well done that in the last 10 minutes you focused on this frame of a videoclip of
someone vomiting.’
It is expected that many individuals will face difficulties when they try to
complete their exposure tasks or will report failure of the exposure to produce the
desired effects. The following are some examples and ways to overcome
these difficulties.
CBT for Specific Phobias
‘I faint when I see blood; how can I do exposure when I am unconscious?’
Applied tension (Ost & Sterner, 1987) (note: different to applied relaxation) aims
to stop blood pressure dropping and to increase heart rate. It is carried out by
tensing groups of large body muscles (arms, legs, stomach) simultaneously for 10–15
seconds at a time and then releasing them gradually. This is repeated up to five
times until faintness subsides. In children it is often difficult to use applied tension
because of the speed with which they faint. In this case, using other strategies to
increase the blood pressure, such as running on the spot while sitting down or doing
star-jumps as the child is standing up are useful ways of keeping the heart pumping
and preventing the blood pressure from dropping.
‘I could look out of a window from a tall building, but I could not go up the
escalator.’ Exposure tasks should have enough variance in them to address all
the feared consequences; for example, fear of heights may relate to escalators
(fear of falling) and looking down a window from a tall building (fear of feeling
dizzy). The individual may use some covert safety behaviours during exposure
which are easy to apply in some tasks but not to others, for example, knowing
that they can quickly move away from the window but not easily once they are
on an escalator.
‘I was comfortable doing one exposure task but could not move up the hierarchy.’
We may need to grade down the task (develop an intermediate task between two
existing tasks in the hierarchy) or choose a different task with similar fear/anxiety
rating or use shaping (successive approximation) to help the individual move
incrementally towards the next step.
Cognitive Restructuring
Exposure-based activities are important learning experiences that can be used to
restructure exaggerated or unhelpful ideas about a feared situation. Behavioral
experiments can be set up in different ways. For example, an experiment may be
open-ended to test what might happen if an individual stopped doing their safety
behaviors. On the other hand, an exposure-based behavioral experiment could have
two or three possible predictions to test which one is likely to happen. Behavioral
experiments should ultimately be designed to target key predictions about coping,
harm, and aversive emotional, sensory, and social feared consequences.
Key questions after successful exposure-based behavioral experiments can help
make experiential learning explicit, correct myths about the feared situation, and
challenge false assumptions about the feared consequences and about the person’s
ability to cope. Examples of such questions are the following:
• What have you learnt from this?
• What does this tell you about how dangerous/painful/awful . . . (dogs, spiders,
heights, injections, etc.) are?
• What does this tell you about yourself when you have to . . . (e.g., go into a lift,
have an injection)?
45
46
Lina A. Gega and Barry J. D. Wright
• Before we started, you thought that you would become very dizzy
and not be able to go on an escalator; how do you explain that you can
now do it?
• What do you expect to happen next time you . . . (e.g., have to go on an escalator)?
In between therapy sessions, and after therapy ends, it is important for the
individual to carry out self-directed exposure-based activities for three reasons: first,
so that the gains from therapy sessions generalize to everyday life and to a variety of
feared situations; second, so that avoidance and safety behaviors do not creep back
in; and third, so that the individual accumulates positive memories of coping
and evidence to reinforce helpful new beliefs about the feared situation. The
activities should be designed with specificity and detail. A suggested format is this:
‘to be able to do (observable, measurable behavior) . . ., when/where/with
whom (what conditions) . . ., without doing . . . (safety behaviors), for . . . (duration),
every . . . (frequency)’.
Self-directed behavioral experiments can be used to lay down new memories of
coping and success. An individual with a specific phobia can easily forget their
positive experiences and selectively attend to evidence that supports their previously held beliefs about the awfulness of their feared situations. To reinforce
memories of positive/coping experiences during behavioral experiments, a useful
strategy is to create ‘self-talk’ flashcards based on these experiences (e.g., ‘I have
done this ten days in a row, I am not looking back’) and by keeping diaries of the
behavioral experiments (e.g., when they took place, what the prediction was, what
actually happened, what the person learnt). Smartphones, tablets, or laptops can be
used to produce idiosyncratic electronic equivalents of flashcards to read, diaries to
make notes in, and self-talk recordings to listen to; this may be less cumbersome and
fit in better with people’s day-to-day habits (e.g., making notes on their tablet or
listening to music and podcasts on their phone), rather than creating paper documents that need to be carried around discreetly, filled in by hand, and kept
somewhere privately.
Conclusion
The application of CBT techniques may vary according to the practitioner’s theoretical orientation, working environment, and level of competence. Practitioners may
offer traditional graded exposure therapy or cognitive behavior therapy via
exposure-based behavioral experiments. Some may offer multiple, individual CBT
sessions; others may offer brief CBT sessions supplemented by self-help materials
(guided self-help); and others may deliver a one-session treatment. Irrespective of
the format that CBT takes, practitioners should draw on their core skills of engaging
an individual, and their family as appropriate, building a trusting working relationship, considering comorbid problems and risks, communicating clearly the treatment process and giving the individual CBT skills and self-confidence to overcome
their fear beyond therapy sessions.
CBT for Specific Phobias
Summary
CBT for specific phobias has its roots in behavior therapy but evolved through
cognitive therapy. We have discussed two key therapeutic techniques for specific
phobias under the CBT umbrella: exposure therapy and cognitive restructuring.
Both techniques aim to help people overcome their fear by changing the way they
think about, and respond to, their feared situation. Cognitive restructuring for
specific phobias focuses on identifying and changing key predictions about coping,
harm, and aversive emotional, sensory, and social consequences associated with
someone’s fear. Exposure therapy focuses on getting used to a feared situation by
letting the fear response subside with time and by weakening the association
between the feared situation and the fear response. Both techniques involve
working through a fear hierarchy and refraining from using safety behaviors.
FURTHER RECOMMENDED READING
Davis, T. E., III., Ollendick, T. H., & Ost, L. G. (2012). Intensive one-session treatment of
specific phobias. Springer-Verlag.
This is the definitive guide on CBT for specific phobias. The first part of the book gives an
extensive background on the phenomenology and assessment of specific phobias; it also gives
an overview of the evidence for various treatments for specific phobias. The second part of
the book focuses on one-session treatment for specific phobias with adults and children. It
gives detailed instructions and treatment plans and materials on how to conduct one-session
treatments. The last part of the book addresses special topics in CBT for specific phobias,
including how to train therapists and assess their competence, how to adapt the treatment for
specialist populations (e.g., people with learning or developmental difficulties), ethical issues,
research evidence, and use of technologies.
Öst, L.-G., & Reuterskiöld, L. (2013). Specific phobias. In S. Gregoris & S. G. Hofmann (Eds.),
CBT for anxiety disorders: A practitioner book (pp. 107–133). Wiley Online Publishing.
This chapter summarizes the state of the art for specific phobias, including diagnosis and
differential diagnosis. It gives an overview of key characteristics of specific phobias, such as
age of onset, gender, focus of fear, neurobiology and physiology, comorbidity, impairment,
risk factors, and treatment response. The chapter also gives a broad overview of the variants
of CBT for specific phobias and discusses evidence of their efficacy. It concludes with a case
example of CBT for a complex specific phobia.
REFERENCES
Abramowitz, J. S., Deacon, B. J., & Whiteside, S. P. H. (2011). Exposure therapy for anxiety:
Principles and practice. Gilford Press
Ayala, E. S., Meuret, A. E., & Ritz, T. (2009). Treatments for blood-injury-injection phobia:
A critical review of current evidence. Journal of Psychiatric Research, 43(15):1235–1242.
Beck, A. T. (1976). Cognitive therapy and the emotional disorders. International Universities
Press.
Bennett-Levy, J., Butler, G., Fennell, M., Hackmann, A., Mueller, M., & Westbrook, D.
(Eds). (2004). Oxford guide to behavioural experiments in cognitive therapy. Oxford
University Press.
47
48
Lina A. Gega and Barry J. D. Wright
Blakey, S. M., & Abramowitz, J. S. (2016). The effects of safety behaviors during exposure
therapy for anxiety: Critical analysis from an inhibitory learning perspective. Clinical
Psychology Review, 49, 1–15.
Blakey, S. M., Abramowitz, J. S., Buchholz, J. L., Jessup, S. C., Jacoby, R. J., Reuman, L., &
Pentel, K. Z. (2019). A randomized controlled trial of the judicious use of safety behaviors during exposure therapy. Behaviour Research and Therapy, 112, 28–35.
Boschen, M. J., Veale, D., Ellison, N., & Reddell, T. (2013). The emetophobia questionnaire
(EmetQ-13): Psychometric validation of a measure of specific phobia of vomiting.
Journal of Anxiety Disorders, 27(7), 670–677.
Brown, T. A., & Barlow, D. H. (2014). Anxiety and related disorders interview schedule for
DSM-5 (ADIS-5): Client interview schedule. Oxford University Press.
Butler, G. (1989). Phobic disorders. In K. Hawton, P. M. Salkovskis, J. Kirk, & D. M. Clark
(Eds.), Cognitive behaviour therapy for psychiatric problems: A practical guide
(pp. 97–128). Oxford University Press.
Cutshall, C., & Watson, D. (2004). The phobic stimuli response scales: A new self-report
measure of fear. Behaviour Research and Therapy, 42, 1193–1201.
Davis, T. E., III, Ollendick, T. H., & Ost, L. G. (2012). Intensive one-session treatment of
specific phobias. Springer-Verlag.
First, M. B., Williams, J. B. W., Karg, R. S., & Spitzer, R. L. (2015). Structured clinical
interview for DSM-5: Research version. American Psychiatric Publishing.
Freeman, D., Haselton, P., Freeman, J., et al. (2018). Automated psychological therapy using
immersive virtual reality for treatment of fear of heights: A single-blind, parallel-group,
randomised controlled trial. Lancet Psychiatry, 5(8), 625–632.
Freud, S. (1909). Analysis of a phobia of a five-year old boy. In Case Histories, vol. 8
(pp. 169–306). Pelican Library.
Helbig-Lang, S., & Petermann, F. (2010). Tolerate or eliminate? A systematic review on the
effects of safety behavior across anxiety disorders. Clinical Psychology: Science and
Practice, 17(3), 218–233.
Hood, L., Fenwick, J., & Butt, J. (2010). A story of scrutiny and fear: Australian midwives'
experiences of an external review of obstetric services, being involved with litigation and
the impact on clinical practice. Midwifery, 26(3), 268–285.
Lang, P. J. (1968). Fear reduction and fear behavior: Problems in treating a construct. In J. M.
Shiler (Ed.), Research in psychotherapy (Vol. 3) (pp. 90–102). American Psychological
Association.
Marks, I. M. (1987). Fears, phobias and rituals. Oxford University Press.
(1997). Behaviour therapy for obsessive-compulsive disorder: a decade of progress.
Canadian Journal of Psychiatry, 42(10), 1021–1027.
Marks, I. M., & Mathews, A. M. (1979). Brief standard self-rating for phobic patients.
Behaviour Research and Therapy, 17, 263–267.
Muris, P., & Merckelbach, H. (1996). A comparison of two spider fear questionnaires. Journal
of Behavior Therapy and Experimental Psychiatry, 27(3), 241–244.
Öst, L. G. (1989). One-session treatment for specific phobias. Behaviour Research and
Therapy, 27(1), 1–7.
Öst, L. G. (2007). The claustrophobia scale: A psychometric evaluation. Behaviour Research
and Therapy, 45(5), 1053–1064.
Öst, L. G., Salkovskis, P. M., & Hellstrom, K. (1991). One-session therapist-directed
exposure vs self-exposure in the treatment of spider phobia. Behavior Therapy, 22(3),
407–422.
Ost, L. G., & Sterner, U. (1987). Applied tension: A specific behavioural method for
treatment of blood phobia. Behaviour Research and Therapy, 25, 25–30.
Ovanessian, M. M., Fairbrother, N., Vorstenbosch, V., McCabe, R. E., Rowa, K., & Antony,
M. M. (2019). Psychometric properties and clinical utility of the specific phobia
CBT for Specific Phobias
questionnaire in an anxiety disorders sample. Journal of Psychopathology and
Behavioral Assessment, 41, 36–52.
Page, A. C., Bennett, K. S., Carter, O., Smith, J., & Woodmore, K. (1997). Assessing a
structure of phobic symptoms elicited by blood and injections. Behaviour Research and
Therapy, 35(5), 457–464.
Rachman, S., Radomsky, A. S., & Shafran, R. (2008). Safety behaviour: A reconsideration.
Behaviour Research and Therapy, 46(2), 163–173.
Radomsky, A. S., Rachman, S., Thordarson, D. S., McIsaac, H. K., & Teachman, B. A.
(2001). The Claustrophobia Questionnaire. Journal of Anxiety Disorders, 15(4), 287–297.
Salkovskis, P. M. (1991). The importance of behaviour in the maintenance of anxiety and
panic: A cognitive account. Behavioural Psychotherapy, 19(1), 6–19.
Silverman, W. K., & Ollendick, T. H. (2005). Evidence-based assessment of anxiety and its
disorders in children and adolescents. Journal of Clinical Child and Adolescent
Psychology, 34(3), 380–411.
Steinman, S., & Teachman, B. (2011). Cognitive processing and acrophobia: Validating the
Heights Interpretation Questionnaire. Journal of Anxiety Disorders, 25, 896–902.
Van Gerwen, L. J., Spinhoven, P., Van Dyck, R., & Diekstra, R. F. W. (1999). Construction
and psychometric characteristics of two self-report questionnaires for the assessment of
fear of flying. Psychological Assessment, 11(2), 146–158.
Veale, D., Ellison, N., Boschen, M. J., Costa, A., Whelan, C., Muccio, F., et al. (2013).
Development of an inventory to measure specific phobia of vomiting (emetophobia).
Cognitive Therapy and Research, 37(3), 595–604.
Vernon, L. L. (2007). Specific phobia. In M. Hersen & J. C. Thomas (Eds.), Handbook of
clinical interviewing with adults. Sage Publications, 247–270.
Wolitzky-Taylor, K. B., Horowitz, J. D., Powers, M. B., & Telch, M. J. (2008). Psychological
approaches in the treatment of specific phobias: A meta-analysis. Clinical Psychology
Review, 28, 1021–1037.
Wolpe, J. (1961). The systematic desensitization of neurosis. Journal of Nervous Mental
Disorder, 132(March), 189–203.
Wolpe, J., & Lang, P. J. (1964). A fear survey schedule for use in behaviour therapy.
Behaviour Research and Therapy, 2, 27–30.
49
4
CBT for Panic Disorder
Robin D. Bailey
Introduction
Panic disorder is an anxiety disorder where people experience recurrent, unexpected panic attacks. According to the DSM-5 (American Psychiatric Association,
2013) panic attacks are defined as a period of intense fear in which a minimum of
four out of 13 defined physiological symptoms are experienced, for example, palpitations, breathlessness, sweating, and shaking. Panic attacks can also be experienced
as ‘unexpected’ or ‘expected’. Unexpected panic attacks appear without an obvious
cause, whilst expected panic attacks are often brought on by external stressors, for
example, taking a test or in response to a phobia.
Although panic attacks are common, to meet diagnostic criteria the individual
needs to have a minimum of one panic attack, which is followed by at least one
month of persistent concern and worry about having more attacks, and/or maladaptive behavior related to the attacks (e.g., cognitive and behavioral avoidance).
Although panic attacks are an essential component of panic disorder, they are
not unique to this disorder. Individuals with a wide range of mental health problems
may experience panic attacks (which may be considered a key criterion of a
disorder), such as the full range of anxiety disorders, including phobia, social anxiety
disorder, and generalized anxiety disorder (Goodwin & Gotlib, 2004); depression
(Goodwin et al., 2004a); psychotic disorders (Goodwin et al., 2004b); personality
disorders (Goodwin et al., 2005); and substance use disorders (Bernstein et al.,
2006). The DSM-5 acknowledges this by making panic attacks a generic symptom
specifier for all DSM-5 disorders.
Before a diagnosis of panic disorder can be made, it is recommended that patients
have a medical examination to rule out organic causes that might better explain their
anxiety symptoms and panic attack. Direct physiological effects of a substance need
to be considered (e.g., excessive alcohol consumption or alcohol withdrawal; medication that has stimulant properties, e.g., corticosteroids, asthma medication, caffeine
and caffeine-based medication; medication discontinuation; illicit drugs such as
amphetamines). Significant medical conditions of the respiratory (e.g., asthma),
50
CBT for Panic Disorder
cardiovascular (e.g., hypertension, cardiac arrhythmia, tachycardia, coronary heart
disease), endocrine (e.g., hyperthyroidism, hypoglycaemia, pregnancy), hepatic
(anaemia), neurological (e.g., epilepsy), and aural (e.g., vertigo, labyrinthitis, otitis
media) systems that might better explain panic symptoms need to be ruled out.
Panic disorder is a common mental health problem, with a cross national
lifetime prevalence of 1.7% (De Jonge et al., 2016). In an international crosscultural sample, lifetime mental health comorbidity was 80.4%, with anxiety and
mood disorders. In primary care settings, panic disorder has been detected in 10.3%
of women and 8.8% of men (King et al., 2008).
Cognitive Behavioral Model of Panic Disorder and Evidence Base
A number of different cognitive and behavioral models of conceptualizing and
treating panic disorder have been developed over the last 35 years; these include
behavioral models based on the principles of classical conditioning (Wolpe &
Rowan, 1988). The key principle of classical conditioning in relation to panic
disorder is the process of paired association: the anxiety symptoms of an uncued
panic attack are paired with a neutral stimulus (e.g., going on a train). Anxiety then
becomes associated with the neutral stimulus. Models based on cognitive, learning,
and biological elements (Barlow, 1988) and expectancy theory (Vroom, 1964) put
the concept of anxiety sensitivity at the forefront of the development of anxiety
disorders. Anxiety sensitivity is based on the theory that bodily sensations arise
from beliefs that physical sensations are harmful (Reiss & McNally, 1985). Recently
mindfulness-based approaches have begun to explore and treat panic disorder
(Beck et al., 1985). The dominant evidence-based CBT model of panic disorder
was developed by Clark (1986), which was based on Beck’s (Beck et al., 1985)
generic cognitive theory of anxiety and the expectancy model of Reiss and McNally
(1985). Wells (1997) revised this model, to give emphasis to the role of safety
behaviors as an additional maintenance factor. Clark’s model (see Figure 4.1)
revolves around a key theme that people with panic disorder ‘catastrophically
misinterpret’ the experience of specific bodily sensations and mental events.
According to the Clark model, a panic attack is triggered by either external or
internal events. External events can be environments associated with the first or subsequent panic attacks (paired associations), for example, supermarkets. Other external
triggers can be hearing or reading words associated with feared symptoms. More often
internal experiences act as more powerful triggers for a panic experience. These could
be common low-level bodily sensations, such as heart palpitations and light-headedness
that arise from normal daily activities (i.e., waking up, exercise, or tiredness). They can
also stem from non-anxiety-related emotional states, such as excitement, anger, and
happiness. When these triggers arise, people with panic disorder will tend to selectively
attend to a particular bodily sensation that is perceived as threatening; for example, a
person may worry, ‘This rapid heartbeat could mean I am having a heart attack’.
Additionally, the fear of having a panic attack (particularly if one is anxious in
anticipation of panic itself ) tends to result in a state of apprehension (‘What if
51
52
Robin D. Bailey
TRIGGER STIMULUS
(internal or external)
Perceived
threat
Interpretation of
sensations as catastrophic
Apprehension
Body
sensations
Figure 4.1. Cognitive model of panic disorder (Clark, 1986).
I . . .?) and further anxiety. This heightened state of anxiety produces a range of
physiological and cognitive symptoms, which the individual will selectively attend to
and monitor closely. These symptoms of panic are perceived as dangerous and
threatening (i.e., indicating imminent physical or psychological harm). When these
symptoms are perceived in a catastrophic way, the patient interprets their physical/
cognitive symptoms as indicative of ‘immediate, impending disaster’ (Clark, 1986;
1988, p. 73). The model assumes that cognition is key to the aetiology and maintenance of panic disorder. Once the symptoms are misinterpreted in this catastrophic
way, a negative feedback cycle occurs whereby anxiety perpetuates further
increases, physical/cognitive symptoms intensify, and conviction in the belief
becomes stronger.
In this vicious cycle, the individual becomes motivated to reduce the perceived
threat and associated anxiety symptomology. This can include further selective
attention to bodily symptoms as a means either to ‘monitor’ the level of threat or
to control or stop the anxiety. This can result in increased intensity of symptoms and
decreased perceived ability to cope with symptoms and sensations.
Ultimately, the patient engages in a range of idiosyncratic safety behaviors
intended to prevent the feared catastrophe from occurring and to regulate the
associated anxious response (Salkovskis, 1991; Wells, 1997). For example, a person
who believes they are going to choke during a panic attack may excessively swallow
to clear their throat, or, as a preventive safety behavior, they may carry a bottle of
water with them at all times. Paradoxically, the employment of such strategies
CBT for Panic Disorder
results in the individual feeling less safe as the use of safety behaviors serves to
exacerbate physical and cognitive symptoms (e.g., excessively forcing swallowing
can mimic the experience of throat constriction), thereby reinforcing the believability of the catastrophic misinterpretation (‘I’m choking’). According to the model,
safety behaviors are also problematic as they both prevent disconfirmation of the
feared catastrophe and become associated with stopping the feared catastrophe
from occurring. Hence, the patient attributes carrying a bottle of water as a preventative strategy for the feared consequence (choking), thereby preventing disconfirmation (they were never going to choke in the first instance).
Evidence for the Model
For any psychological treatment to be effective, it must have a theory which is well
supported. A number of studies over the years have provided evidence for the main
tenets of Clark’s model of panic. Findings have supported the assumption that
catastrophic misinterpretations of bodily symptoms is a key factor in the development and maintenance of panic disorder (Casey et al., 2004; Hoffart, 2016). It has
also been identified that targeting these beliefs in treatment is associated with a
reduction in symptoms, including the frequency of panic attacks and the intensity of
panic symptoms and associated distress (Hoffart et al., 2008; Teachman et al., 2010)
When exploring the role of selective of attention in panic disorder, it has been
found that individuals with panic disorder have an attentional bias towards panicrelated stimuli (Pergamin-Hight et al., 2015; Reinecke et al., 2011). In early tests of
the model an increased use of safety behaviors has been shown to prevent change in
the catastrophic misinterpretations and reduction in levels of anxiety when compared with participants who reduced such behaviors (Salkovskis et al., 1996;
Salkovskis et al., 1999).
CBT is recommended in the NICE guidelines (NICE, 2011) as the first line of
treatment for people with panic disorder. Although other treatments exist (i.e.,
pharmacotherapy, psychodynamic therapy, and supportive therapy), CBT has the
strongest evidence base (Pompoli et al., 2016). In this Cochrane review it should be
noted that, although CBT was deemed superior to other treatments, the authors
conclude the evidence was not unequivocal. When different variations of cognitive
and behavioral therapies were compared, CBT had statistically significant superiority over behavior therapy and cognitive therapy. Evidence also exists that a combination of medication and therapy in the treatment of panic disorder is more
effective than medication alone, both during treatment and at 24 months followup (Furukawa et al., 2007).
Components of Treatment
Theoretical models of psychological disorder, on which treatment is based, should
have a strong evidence base to support the key treatment components used to target
specific aspects of the theory. Within CBT and BT there is a range of techniques and
53
54
Robin D. Bailey
interventions which have been developed to treat panic disorder, including cognitive
restructuring, interoceptive exposure, breathing retraining, muscle relaxation, in vivo
exposure, and virtual reality exposure. What has also emerged is how these specific
interventions, and importantly the combinations and sequence of these interventions,
impact on treatment efficacy. In a component network meta-analysis (Pompoli et al.,
2018), it emerged that a combination of interoceptive exposure and cognitive restructuring produced the most efficacious form of CBT. The combination of these components (behavioral experiments) are used in both Clark’s (1986) and the modified
version of Clark’s treatment protocol for panic (Wells, 1997). Treatments that combine breathing retraining, muscle relaxation, in vivo exposure, and virtual reality
exposure in their treatment protocol are less efficacious. It is also worth noting that
breathing retraining has a specific low impact on positive outcomes in the treatment
of panic, and relaxation is associated with worse outcomes in treatment (Pompoli
et al., 2018). This corresponds with the idea that relaxation and breathing retraining
may act as a safety behavior that paradoxically prevents disconfirmation of the
catastrophic misinterpretation (Wells, 1997). This may explain why CBT may be
superior to BT in the treatment of panic disorder (Pompoli et al., 2016).
Interpersonal Skills and Therapeutic Alliance
It has been shown that patient engagement and perception of the therapeutic
alliance may be a contributing factor to positive outcomes in the treatment of panic
disorder and low attrition rates (Huppert et al., 2014). These findings highlight the
importance of collaboration between the therapist and patient in areas such as goal
setting, understanding the model based on the patient’s own experiences of panic,
accepting treatment rationale, and a sense of working together towards the amelioration of symptoms.
Adherence to the treatment protocol is important in the successful application of
CBT for panic disorder. This is particularly important with treatment-resistant patients,
as evidence suggests that adherence to protocol can decrease with such patients
(Zickgraf, 2016). Treatment resistance may be responded to through re-evaluating
the formulation or exploring beliefs about the patient’s fears regarding exposure-based
behavioral experiments or concerns about reducing safety behaviors. Ultimately,
maintaining adherence to the protocol in a skilled and sensitive manner is key.
Patients are generally seen weekly for between five and 15 sessions. Successfully
treating panic patients in as few as five sessions is best supported with guided selfhelp materials (Clark et al., 1999).
Measurement and Assessment of Panic Disorder
Session 1
Assessment should begin with the completion and review of panic-specific outcomes
measures. A useful measure is the Panic Disorder Severity Scale (PDSS; Shear
CBT for Panic Disorder
et al., 1997), which is both a screen for panic disorder (Houck et al., 2002) and
measure of severity. This measure also captures pertinent information that can
inform and guide the assessment, such as frequency and levels of distress, worry
about reoccurring panic attacks, avoidance, fear of physiological symptoms, and
impact related to the disorder. The PDSS is repeated and reviewed on a weekly
basis since the measure has shown both sensitivity to change and the ability to
capture a reduction in panic symptoms over the full course of CBT treatment
(Barlow et al., 2000; Shear et al., 1997). Other measures that capture aspects of
panic disorder include the Mobility Inventory (Chambless et al., 1985), which
assesses agoraphobic avoidance. The Agoraphobic Cognitions Questionnaire
assesses the frequency and believability of panic-related thoughts (Chambless
et al., 1984).
Additionally, in session belief ratings can and should be used before and after
any intervention, where 0 is ‘do not believe at all’ to 100 ‘completely believe it to
be true’.
Often the physiological effects of a panic attack can be a similar to a range of
other conditions, including asthma, angina, irregular heartbeat, benign paroxysmal
positional vertigo (BPPV), migraine, and thyroid issues. It is important to explore in
the assessment whether the panic is linked to any of these conditions or if the patient
has had their symptoms checked by a general practitioner; if not, such considerations
should be discussed. Patients may have some of these conditions as well as panic
disorder; however, it is not within the scope of this chapter to address comorbidity and
the reader is advised to read research/treatment manuals that deal with treating panic
disorder alongside physical health conditions. Current issues with substance misuse,
such as amphetamines, benzodiazepines, and cocaine, which can be associated with
panic disorder, need to be addressed during the assessment.
Practitioners sometimes find it difficult to differentiate between panic disorder
and health anxiety. In both panic disorder and illness anxiety disorder (IAD),
individuals misinterpret bodily symptoms as the sign of illness threat, attend to
bodily symptoms, and respond with disproportionate levels of fear (Deacon &
Abramowitz, 2008). Equally, individuals with both these conditions have a tendency
to overutilize healthcare services as a means of explaining the symptoms and gaining
medical reassurance (Olatunji et al., 2007).
A good starting point in differentiating between these two disorders is to refer
to diagnostic criteria such as the DSM-5 as a guide. Additionally, a number of
studies have found clear differences between the two disorders (Deacon &
Abramowitz, 2008). In panic disorder, the sudden onset of bodily sensations leads
to the ‘catastrophic misinterpretation’ that is considered imminent, for example,
having a heart attack, whereas with IAD the illness is perceived as more protracted
and existing in the future, with more concern being placed on the future significance
of symptoms (Noyes et al., 1986; Salkovskis & Clark, 1993). Studies have found that
individuals with IAD rate their symptoms as more distressing and tend to have
poorer relationships with their general practitioner (Deacon & Abramowitz, 2008;
Hiller et al., 2005) than patients with panic disorder. Furthermore, with IAD
55
56
Robin D. Bailey
perceived medical symptoms may not be the direct response of anxiety, for example,
someone finding a blemish on their skin, unlike panic disorder where symptoms
directly arise from autonomic arousal (Williams, 2004).
If a patient presents with both panic disorder and IAD, CBT focuses on
targeting the same maintaining factor of catastrophic misinterpretations; however,
IAD may need more specific interventions due to the chronological occurrence of
the perceived illness. (See Chapter 8 for further information on IAD.)
Other areas to explore during the assessment can include health-related attitudes and behavior of significant others (family, colleagues, and doctors), current
alcohol and drug use, previous medical and psychological treatment, personal
strengths, and social and financial circumstances to obtain a wider perspective of
panic disorder.
Assessment and Formulation (Sessions 1 and 2)
Assessment and formulation of panic disorder requires a thorough exploration of
the key components of the model that are specific to the patient’s own experience of
the disorder. This requires having a template of the model when carrying out the
assessment, which can be used for formulation, psychoeducation, and treatment.
The panic model is in essence cross-sectional in nature so a detailed evaluation of
longitudinal factors may be unnecessary, outside the initial onset of panic symptoms,
that is, the first panic attack.
Using the template as a guide (see Figure 4.2), ask the patient to think of a
recent example of panic that is typical of how they experience the disorder. It is
important to do this as it will be fresh in the patient’s memory; it also keeps the
assessment focussed and enables the therapist to walk with the patient through the
model. The use of Socratic questioning is essential as it enables the development of
the therapeutic alliance and helps raise the patient’s insight into the model and how
their panic is being maintained. At the end of the assessment it is important to check
that all the information gleaned is congruent with the patient’s own experience of
their panic. If this is not ascertained, it will have a detrimental effect on both
socialization and treatment.
Assessment Stages Using the Cognitive Model of
Panic Disorder (see Figure 4.1)
Internal/External Trigger. Once you ascertain the situational specific aspects of the
panic attack, begin by focusing on component number 1 in the model (Internal/
External Trigger). The patient may generate a number of triggers; therefore, it is
important to ascertain which of these is most problematic for the patient.
Therapist/Patient Dialogue
Therapist:
Patient:
So when you were walking into the lecture theatre, which
symptom did you notice first?
I just started sweating, my head was racing, I was dizzy.
CBT for Panic Disorder
57
INTERNAL/EXTERNAL 1.
TRIGGER
PERCEIVED THREAT 3.
ANXIETY
2.
MISINTERPRETATION
5.
PHYSICAL/COGNITIVE
SYMPTOMS 4.
AVOIDANCE &
SAFETY BEHAVIORS
(Including Selective
Attention) 6.
Figure 4.2. A cognitive model of panic disorder with maintenance cycles added. From
Wells (1997, p. 102). Copyright 1997 by John Wiley & Sons Limited. Reprinted by permission
Therapist:
Patient:
OK that sounds scary, which of these symptoms concerned
you most?
Feeling dizzy.
Anxiety. Then move to component number 2 in the model, to ascertain the
emotional experiences. Again, it is important to remember that the patient may
have many different ways to describe anxiety, so it is always worth clarifying their
terminology. It also worth ascertaining a baseline rating of intensity for the level of
emotion using a 1–10 Likert scale.
Therapist: And when you noticed the dizziness, how did you feel?
Patient:
I was like, oh no.
Therapist: As a feeling how would you describe ‘oh no’?
Patient:
It was like pure fear.
Therapist: So you were feeling fearful? Scared?
Patient:
Yes, both.
Therapist: On a scale of 0 to 100, how high would you rate your fear, with
0 being none, to 100 being the worst?
Patient:
Erm, about 70%.
58
Robin D. Bailey
Perceived Threat. At component 3 the therapist elicits thoughts that moderate
the relationship between the initial trigger and the emotional response. At this stage
the catastrophic misinterpretation is not elicited, but more the anticipatory thought
about what the patient thought was about to happen.
Therapist:
Patient:
Therapist:
Patient:
OK, so it sounds like when you went into the lecture you noticed
yourself feeling dizzy and as a result you are started to feel
yourself getting fearful and scared. Is that correct?
Yes, it was horrible.
It sounds like it; at that point what was worrying you about these
symptoms starting?
It just didn’t feel right. I just knew something bad was going
to happen.
Physical/Cognitive Symptoms. At component 4 the physical and cognitive
symptoms experienced in a panic attack are assessed. The aim here is to elicit all
the physiological and cognitive symptoms the patient experienced during this panic
episode and further establish which of these caused most concern. This can often
indicate what the feared catastrophe might be.
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
So when you were feeling really anxious, and you thought
something bad was going to happen, did you experience any more
physical sensations?
Yes, my breathing got worse, I was even dizzier, my legs felt
really heavy and wobbly, and then my mind started racing.
OK, so which of these symptoms concerned you most?
At that moment all of them, though the dizziness was scaring the
hell out of me.
So as these symptoms escalated what happened to your anxiety?
It was going through the roof. It was sky high.
Catastrophic Misinterpretation. At component 5 the aim is to elicit the catastrophic misinterpretation. The patient may be able to identify this readily or it may
require additional Socratic exploration. It also worth ascertaining a baseline rating
of believability in the catastrophic misinterpretation.
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
Therapist:
So when you were having these feelings, what was going through
your mind?
I was just really scared and needed to get out.
Ok, so what would have happened if you didn’t get out at
that point?
Something awful.
So something awful like what?
Like . . . erm . . . collapsing.
So what might have made you collapse?
CBT for Panic Disorder
Patient:
Therapist:
Patient:
Therapist:
Patient:
The anxiety was getting so bad I thought it was going to make me
pass out.
So it sounds like you thought the anxiety could have made you
pass out?
Yes, it was awful.
In that moment in time, how much did you believe that thought to
be true, with 0 being not at all to 100 being completely convinced
it would happen?
Oh, definitely 90%.
Safety Behaviors. Initially, it is important to establish any in situ safety behaviors that may be used to prevent the feared catastrophe from occurring or as a
means to control the symptoms of anxiety.
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
Therapist:
Patient:
So, when you were in the lecture theatre is there anything you
tried to do to prevent yourself from passing out?
I just tried to do some breathing exercises that I read
about online.
So what did that involve?
Breathing in and out quickly into a paper bag.
Did this reduce your dizziness?
I am not sure, but it might have helped me not pass out.
How much do you believe doing these techniques stopped you
passing out?
Probably about 80% because I did not pass out.
Did you do anything to try to control your anxiety in that moment
in time?
Well I always have my phone ready and have emergency services
on speed dial.
OK, how does that help reduce your anxiety?
Well, if I know I can contact them before I pass out, then they
may be able to find and help me.
Did this help reduce your anxiety on that day?
It did a little, but I was still scared.
It is also important to assess any behaviors that may be employed as a way to
prevent panic attacks recurring in the future; this can include forms of avoidance.
Therapist:
Patient:
Therapist:
Patient:
So, at other times maybe when you are not having a panic attack
is there anything you do or maybe don’t to prevent another panic
attack occurring?
I have stopped drinking caffeine.
OK, so why is that?
I just do not like the feeling it gives, makes me feel dizzy and
shaky, which scares me. Even the smell has a negative effect.
59
60
Robin D. Bailey
Therapist:
Patient:
So you avoid caffeine, as you feel it may increase your anxiety
and lead to you passing out?
Yes.
Psychoeducation and Socializing to the Model: (Sessions 2 and 3)
Once all the information is gathered after assessment, the therapist should then use
psychoeducation to show how all the components of the model fit together to
explain the patient’s experience of panic. It is imperative that this is carried out
using the patient’s own idiosyncratic experiences of panic, coupled with a clear
connection to the theory expressed in a language that is easily understood by the
patient. It is important this balance is achieved; otherwise, it can result in a theoretical didactic lecture which may have no relevance to the patient and may potentially
rupture the alliance. Alternatively, if it is overly simplistic this may prevent the
patient understanding and grasping the theory.
This process can be achieved through both verbal and visual presentation of the
labelled model with the patient’s own experiences included (see Figure 4.3).
INTERNAL/EXTERNAL
TRIGGER
Lecture
1.
theatre/dizzy
PERCEIVED THREAT
3.
Something bad!
ANXIETY
2.
Fear/scared 70%
MISINTERPRETATION
5.
Going to pass
out! 90%
PHYSICAL/COGNITIVE
SYMPTOMS
4.
Dizzy, racing
mind, wobbly
legs
AVOIDANCE &
SAFETY BEHAVIORS
(Including Selective
Attention) 6.
Breathing
exercises, avoid
coffee
Figure 4.3. Psychoeducation and socialization to the model.
CBT for Panic Disorder
It is always important at this stage (and throughout therapy) to check that what
is presented is congruent and relevant to the patient’s experience; otherwise, this
can lead to a breakdown in alliance and interventions that target the
wrong processes.
The rationale of socializing the patient to the model is to provide patients with a
better understanding of what maintains their panic disorder and how working on
these processes can lead to symptom relief. Psychoeducational interventions help
the patient understand the problem and also increase the patients’ sense of agency,
which can lead to a reduction of catastrophic thoughts and emotions. In panic
disorder, socialization is a powerful therapeutic intervention in its own right.
The key to socializing the patient to the model is to pick important connective
components of the model to illustrate experientially how they cause a
maintaining effect.
In-Session Socialization Exercises
The Effect of Thought on Emotion and Physical Sensations
A way for patients to see how thinking about specific symptoms is linked to
increased anxiety is to get them to complete the paired associates task (Clark,
1988). This activity involves reading a list of words related to physical symptoms,
paired with a catastrophic misinterpretation. The aim of this exercise is to enable the
patient to discover the true cause of the sensations that occur in panic attacks. In the
original version of this task, when patients with panic disorder were presented with a
series of associated word pairs referring to catastrophic interpretation (e.g.,
palpitations-dying), 83% experienced panic symptoms. In a more up-to-date version
of the paired associates task, two categories of paired words are used: 10 panicrelated words and 10 neutral words (De Cort et al., 2013). Neutral words are used to
help patients understand that only panic-specific words elicit anxiety and not
other words.
Before beginning the exercise, it is important to get a rating of the patient’s
anxiety so this can be compared with levels of anxiety post-exercise. It is important
that the patient is not told that this exercise may induce panic, as they may avoid or
disengage from reading. Patients should be encouraged to read the words out loud
and think about the meaning of the words. After completing the exercise, if there is
an increase in anxiety, ask the patient why that might be. In addition, what does that
tell them about the role thinking plays in relation to anxiety and increase in physical
symptoms? The patient’s specific formulation can be used to further explain how
this component of the model maintains anxiety.
Paired Association Words (De Cort et al., 2013)
Headache – Brain tumour
Bookcase – Installation
Shortness of breath – Suffocation
Flowers – Picking
61
62
Robin D. Bailey
Purchase – Shopping
Newspaper – Reading
Dizziness – Fainting
Lemonade – Drinking
Shaking – Lose control
Shaking – Paralysis
Meal – Cooking
Weeds – Pull out
Tingling – Stroke
Rocking chair – Wiggling
Chest pain – Heart attack
Television – News
Nausea – Vomit
Garden table – Summertime
Racing thoughts – Going crazy
The Role of Attention on Thinking and Physical Symptoms
Using the exercise above you can highlight how selective attention plays a role in
panic disorder. Ask the patient how many catastrophic words they can recall from
the list, then ask them to recall how many neutral words they can recall. As patients
with panic disorder tend to display strong attentional biases towards panic-related
stimuli compared with neutral stimuli (Reinecke et al., 2011), they will be more
likely to recall more panicogenic words than neutral words.
Another technique used to highlight the role of selective attention is the body
focus task. This involves asking the patient to monitor a neutral part of their body,
for example, their toes or fingers, to identify as many physical sensations as they can.
After a few minutes ask the patient what they noticed. Once the patient reports
what sensations they noticed, then ask them how aware they were of the same
sensations prior to completing the exercise. This enables the patient to connect that
focusing on symptoms increases both the awareness and intensity of the symptoms.
This experiment can be expanded upon, to highlight how body focusing may
also be an ineffective safety-seeking behavior. Often patients report that if they
focus or ‘keep their eye’ on the symptoms, they can either stop or control them. This
can be tested by asking the patient to repeat the exercise, but this time, whilst
focusing on the sensations, ask them to try to stop or control the sensations by
focusing more intensely on them. This tends to result in an increase in sensations
and a sense of decreased control. Again, help the patient see how safety behaviors
and selective attention can increase and enhance physical sensations, and strengthen
belief in the feared catastrophe.
The Role of Safety Behaviors
The role of safety behaviors in panic disorder can be particularly problematic for
two reasons. First, it maintains and exacerbates symptoms, and second, prevents
CBT for Panic Disorder
disconfirmation of the feared catastrophe. To socialize the patient to this idea the
use of metaphors and allegories maybe useful. The following is an example.
1. Some children (and adults) believe scary monsters exist under the bed. Each
night when they go to bed, they may get anxious and worry that the monster will
get them. To prevent this from happening, they may hold a teddy, sleep with the
light on, cover the gap between the bed and floor, or get someone else to check.
To date no child has been taken by a scary monster. Because of this the child
may believe two things: that if they look under the bed the monster will get
them and that teddies, night lights, and so on keep children safe from monsters.
A question to help the patient make the connection may be: ‘How can the children
discover that their belief is false and that monsters don’t exist?’
One way to highlight further the role of safety behaviors is for the patient to
complete a behavioral experiment whilst engaging in and even increasing their
safety behaviors, to prove that catastrophe does not occur. This principle is explored
later when we look at behavioral experiments.
Treatment
Discussion Techniques (Sessions 3–13)
The aim of treatment is to help the patient to challenge their catastrophic misinterpretation and recognize the benign nature of anxiety symptoms, ultimately becoming less fearful of the unpleasant symptoms. As stated earlier in the chapter, two
optimal CBT techniques can be utilized to achieve this: cognitive restructuring and
exposure to bodily symptoms. Optimal improvements may be obtained when cognitive and exposure techniques are integrated to the modification of specific misinterpretations (Salkovskis et al., 2007).
Cognitive Restructuring
The aim of verbal reattribution techniques is to weaken the patient’s belief in the
likelihood of the catastrophic misinterpretation (CM) occurring and to strengthen
the belief that physical sensations are not inherently dangerous and that it is the
meaning assigned to them that makes them frightening. Completing verbal reattribution prior to behavioral experiments is a beneficial way to help loosen conviction
in the CM. As always in CBT, the aim is to teach the patient how to challenge their
own CM independently.
The following standard principles of verbal reattribution are applied:
1. The belief in the catastrophic misinterpretation is identified and rated.
2. Evidence for and against the catastrophic interpretations is reviewed
and challenged.
3. Alternative cognitive accounts of the patient’s panic attacks are explored.
63
64
Robin D. Bailey
Table 4.1. Panic thought record
Emotion
and % of
intensity
Situation
Going into
lecture and
feeling
dizzy
Anxiety
and fear
70%
Catastrophic
misinterpretation
(CM)
and % of belief
Was going to pass
out. 90%
Evidence
for CM
Evidence
against CM
Alternative
explanation
Felt dizzy.
Was
scared.
Felt hot.
I didn’t pass
out. I have
never passed
out and felt
like this lots
of times.
Feeling these
symptoms
does not lead
to passing out.
To pass out,
blood pressure
needs to drop;
when people
panic, their blood
pressure rises.
Lots of other
people feel
anxious and do
not pass out.
New
belief
rating
40%
4. Rating of belief in the alternative response and re-rating of belief in the
catastrophic interpretation are carried out.
Completion of Cognitive Restructuring for Homework
A panic thought record (see Table 4.1) to illustrate how to complete cognitive
restructuring may be a useful tool to facilitate practicing this technique and enhance
homework practice. Some patients find the use of the paper redundant in this
technological age, so adaptation through electronic means (completion on an electronic device) may be more relevant.
Identification of Catastrophic Misinterpretation. This will have been identified
during assessment; however, it is always worth rechecking with the patient: Is it still
the same or are there other additional beliefs?
Evidence for and Against. At step 2 of cognitive restructuring, the therapist
questions and challenges the evidence for the patient’s catastrophic misinterpretation(s), and substitutes these ideas with more realistic interpretations of the symptom(s). It is quite common for patients with panic disorder to neglect objective
evaluation of CM-supporting thoughts. The following questions may be helpful.
If you have experienced at least 20 panic attacks, what do you think are the
reasons you have never passed out?
If you didn’t believe that you were going to pass out, how anxious would you be?
How does anxiety lead to, for example, having a heart attack? (questioning the
mechanism)
If anxiety caused (stroke/madness/heart attack), how many people in a day
would suffer these illnesses?
How many people do you know who have experienced anxiety but have not had
a heart attack/fainted/had a stroke/gone blind?
CBT for Panic Disorder
65
Table 4.2. Examples of catastrophic misinterpretations and an alternative explanation
Catastrophic
misinterpretation
‘I am going to pass out’
‘I am going to have a
heart attack’
‘I am going to suffocate’
‘I am going blind’
‘I am having a stroke’
‘I’m going crazy’
Alternative explanation
To pass out, blood pressure needs to drop; when people panic, their blood
pressure rises. Police officers, ambulance drivers, and firefighters when
attending to an emergency feel anxious and don’t pass out. There are situations
where people can pass out, specifically if someone has a blood-injury-injection
phobia.
The role of adrenaline when anxious is to keep us safe rather than kill us. Panic
is about fear of our bodily sensations and where they might lead. Our reaction
to the fear is the same as if we came face to face with a lion.
Suffocation can occur only if your airways are blocked by something. Breathing
is an automatic process and not something we need to think about controlling;
our bodies self-regulate. Airways do not spontaneously close when anxious.
Blindness is primarily caused by age-related eye diseases that include macular
degeneration, cataracts, diabetic retinopathy, and glaucoma. These diseases are
progressive and develop over a long period of time. It is not something that is
caused or happens through experiencing emotion.
A stroke is when the circulation of blood to the brain declines. Decreased blood
flow and lack of oxygen results in loss of brain cells. Strokes can be caused by
either a blockage of blood flow to the brain or bleeding into the brain. Anxiety
does not clot blood.
Feeling frightened of symptoms of anxiety and the fear of what it means and
where it might lead, that is, to uncontrollable anxiety and losing one’s mind or
the fear that your brain is not working properly, serves to increase anxiety.
When in a heightened state of anxiety, it is normal to feel detached and
disconnected from ourselves, like walking on cotton wool or when hearing the
echo of our voice, which adds to our worry and fears and at the time feels very
real.
It is not possible to ‘go crazy’ through panic.
How often have you been convinced that (CM) will happen and it hasn’t
happened?
What reasons do you have to believe that (CM) is unlikely to happen in a panic
attack?
Alternative Explanations. At step 3 the aim is to explore and strengthen alternative explanations for the symptom other than the CM. This involves providing the
patient with some further psychoeducation and counter-evidence to the CM. It is
important that this be facilitated through guided discovery and Socratic questioning.
Some examples of alternative explanations are shown in Table 4.2.
Modifying Images (Sessions 3–13)
Although patients often verbalize their feared catastrophic misinterpretation, for
some this may represent itself more as an image or variety of images. In much
66
Robin D. Bailey
the same way as above, these images can be challenged using verbal-based
techniques. In some situations, working with imagery may be of additional
benefit to weaken belief and reduce the associated affect. One such technique
proposed in Clark and Salkovskis (2009) is image restructuring with the aim of
generating an alternative image to the feared image. This involves, first, helping
the patient make the distinction between imagery and reality. Second, ask the
patient to bring to mind a negative catastrophic image and hold it in mind until
they feel anxious. Third, once they feel an increase in anxiety, they transform all
aspects of the image into something more positive. This transformation enables
to patient to observe and experience affective change through manipulation of an
image. Fourth, the image may require changing in stages, with the most distressing components being chosen for transformation first. The patient is encouraged
to practice this over the course of a week for homework, to further reduce
frequency and believability.
Behavioral Experiments (Sessions 3–13)
A natural follow on from any form of verbal reattribution is to further test out the
patient’s specific predictions through behavioral experiments. Although cognitive
restructuring may bring around some belief change at a logical and intuitive level,
the strength of the belief can be further reduced through gathering experiential
evidence (Bennett-Levy, 2003). In essence, patients may understand theoretically
that they may not pass out; however, it becomes indisputable only when they see
that they do not. Additionally, behavioral experiments are a very powerful tool in
enabling the patient to find out that safety behaviors do not prevent feared catastrophes and in fact can maintain emotion and physiological effects. For a full review
on behavioral experiments, see the Oxford Guide to Behavioral Experiments
(Bennett-Levy et al., 2004).
One of the main forms of behavioral experiments used in panic disorder are
symptom induction experiments. The aim of these types of experiments is to enable
the patient to experience the emotional/physical/cognitive symptoms that closely
resemble those experienced when having a panic attack. From this they can find out
the non-catastrophic cause of the symptoms and that the sensations that accompany
panic attacks are not dangerous. The following specific steps are among those that
need to be considered before setting a behavioral experiment.
1. Ensure the patient understands and buys into the rationale for the experiment;
this is linked to the formulation and any verbal reattribution that has occurred.
2. Make sure the induction exercise is specific to the patient’s CM.
3. Rate the believability in the CM before and after the experiment, and through
all future experiments.
4. Make sure the patient is not engaging in any overt or covert safety behaviors
when doing the experiment.
5. This should be practiced for homework to accrue real in situ disconfirmation
CBT for Panic Disorder
Note that assessment of any diagnosed physical conditions is important and that it is
advisable to seek assurance from the patient’s doctor that it is safe to use interoceptive exposure. For further information, it is worth reading Bennett-Levy et al.’s
work on self-reflection and self-practice (Bennett-Levy et al., 2001)
Below are some examples of experiments that may be beneficial in testing out
and challenging specific catastrophic misinterpretations.
Catastrophic Misinterpretation: ‘I am going to faint/pass out/collapse’
The hyperventilation provocation task (HVPT) is the most useful behavioral experiment to use with this belief. Hyperventilation on its own is not necessary or
sufficient to induce panic; it is the patient’s misinterpretation of somatic symptoms
that are the issue.
1. The therapist models HVPT by breathing in and out as deeply and as quickly as
they can.
2. The patient and therapist stand together and breathe deeply and rapidly for a
few minutes (time duration may need to be graded depending on the patient’s
level of fear).
3. If safety seeking behaviors are being used, the patient is prompted nonverbally
to desist.
4. At the end of the HVPT, the patient is informed they should stay standing,
resume normal breathing for the next few minutes, and further desist from using
any safety-seeking behaviors.
5. The patient is then asked to re-rate their belief in the CM and comment on what
happened to the physical symptoms when left alone (these will have decreased).
6. Repeat until the belief in the CM is significantly reduced.
These experiments can be repeated with variations to help strengthen belief in
the non-occurrence of the catastrophe, and that the sensations that accompany
panic attacks are not dangerous. During the experiments it is worth asking the
patient to make predictions about what they think will happen and then assess
whether these predictions are correct.
If the patient believes they will pass out due to dizziness or derealization, the
above experiment can be carried out with the addition of asking the patient to spin
around three times. Spinning can mimic the symptoms of dizziness and derealization; however, the patient can learn that these symptoms do not lead to the feared
catastrophe of passing out.
Catastrophic Misinterpretation: ‘I am going to have a heart attack’
Inducing physical activity to enhance cardiac awareness can be used to challenge
this CM. This should include enough physical exercise to get the blood flowing and
the heart speeded up, which can bring on other feared physical sensations, such as
breathlessness, sweating, or dry mouth. Activities can be sitting to standing exercises, running on spot, walking up and down stairs, and so on.
67
68
Robin D. Bailey
Catastrophic Misinterpretation: ‘I am going to suffocate/choke/stop breathing’
In this behavioral experiment you can get the patient to engage in some paradoxical
experiments, through the ‘holding the breath test’. This involves asking the patient
to intentionally stop breathing (i.e., hold their breath) to see what happens. Patients
discover that it impossible to volitionally hold their breath for long periods of time.
After a while the brain will automatically start the breathing rhythm again, even if
they try very hard to hold their breath.
Catastrophic Misinterpretation: ‘I am going to lose my mind/go crazy’
This can be tested using the HVPT with the addition of instructing the patient to
intentionally worry about going crazy whilst doing the exercise. This can be further
modified if the patient has specific behaviors which they associate as a sign of losing
one’s mind, such as running around in circles or screaming. Again, the patient
completes the HVPT and during the exercise carries out the behaviors to see if
they actually go crazy/lose their mind.
Catastrophic Misinterpretation: ‘I am having a stroke’
In this experiment, first, we get the patient to compare the difference between the
specific symptoms experienced when someone is having a stroke (using the established acronym of FAST [see below]; Flynn et al., 2014) compared with when
someone is having a panic attack, to find out the symptoms are completely different.
Then, the patient can complete a HVPT and test out whether they have any of the
symptoms of a stroke.
F (Face): Because the face may have dropped to one side during a stroke, test
whether they are able to smile.
A (Arms): Because of weakness and numbness in the arm during a stroke, test
whether they are able to lift both arms and keep them there.
S (Speech): Because speech becomes slurred and garbled during a stroke, test
whether they can speak a number of sentences clearly.1
These are just some examples of behavioral experiments; therapists should be
creative when working with patients to devise and develop idiosyncratic
behavioral experiments.
If the patient has a pre-existing physical condition such as cardiac issues or
chronic obstructive pulmonary disease, use a specific treatment manual (Livermore
et al., 2010; Tully et al., 2017) that incorporates safe interoceptive exposure.
Behavioral Experiments and Safety Behaviors
It is important to reinforce with the patient that safety behaviors can often be part of
the problem in panic disorder rather than a solution. During the completion of all
1
The final ‘T’ in the acronym stands for time, which is of the essence in a real stroke situation.
CBT for Panic Disorder
behavioral experiments patients are encouraged to actively desist from engaging in
overt and covert safety behaviors. To highlight the problematic nature of safety
behaviors, patients can complete the above behavioral experiments and actively try
to control or stop symptoms by increasing their use of safety behaviors. For
example, patients who believe they are going to go insane when panicking can try
actively suppressing their thoughts, resulting in a paradoxical rebound effect. Or
patients who believe that they will stop breathing during panic attacks can try
deliberately focusing on/controlling their breathing, making breathing seem irregular/unnatural and increasing anxiety.
Behavioral Experiments as Homework
It is important and a more powerful learning experience for the patient to carry out
behavioral experiments away from the therapy room and on their own. This has a
number of therapeutic effects, such as the following.
1. Patients can believe that the security of a therapy room and the presence of a
professional are factors preventing the CM from happening. Practicing experiments on their own (at home and in feared situations) helps them find out that
feared catastrophes do not occur and that the sensations are not dangerous.
2. It strengthens beliefs in the ability to choose to respond differently to symptoms
of panic, reduces the perception of threat, and increases belief in their ability
to cope.
3. It facilitates long-term learning whereby the patient becomes more self-reliant
and less dependent upon the therapist and therapy.
All behavioral experiment homework should follow logically from the session
work and be collaboratively devised. Review of homework should always be
explored at the start of the next session. This can all be facilitated by using a
behavioral experiment record and the acronym SPEAR (Situation, Prediction,
Experiment, Actual Outcome, Review) (see Table 4.3), with the first three components of the acronym (SPE) completed in session, and the last two components (AR)
completed after the experiment and reviewed in the next session (see below).
Relapse Prevention and Blueprint for Therapy (Sessions 5–15, Depending on
How Well Patient Responds to Therapy)
Once the patient has reported a reduction in panic symptoms and severity and is
below the clinical cut-off on the outcome measures used, then therapy should move
into relapse prevention. To facilitate relapse prevention, some detailed questioning
regarding how the person will manage post-treatment is needed. This enables the
therapist to assess for any residual beliefs or behaviors that may still be problematic,
currently or in the future. It also addresses whether the patient has fully understood
what maintains their panic and whether they are clear on the strategies they need to
continue using.
69
70
Robin D. Bailey
Table 4.3. Behavioral experiment record modelled with SPEAR (situation, prediction, experiment,
actual outcome, review)
SITUATION
Where and
when.
Going into
lecture
theatre.
PREDICTION
What is your
feared
prediction?
(Belief rating
0–100%)
If I go and there
are lots of
people, I will
get so anxious
I will pass out.
EXPERIMENT
What will you do to
test the prediction?
What will you not do
to test the prediction
(safety behaviors)?
Go into lecture theatre
with the knowledge
that anxiety is not
dangerous and cannot
make me faint.
Avoid looking at
phone and do not do
breathing exercises
(these make me
worse).
Actually listen to the
lecture.
ACTUAL
OUTCOME
What really
happened?
Did the outcome
match the prediction?
I did not pass out.
I did feel a bit anxious
to begin with, but this
seemed to disappear.
I felt quite proud of
myself.
REVIEW
What have
you learned?
Re-rate belief.
How can apply this
for the future?
Anxiety really does
not make me pass
out.
I learned more in
the lecture than
I ever have.
Next time I will try
and not sit close to
an exit.
10%
This can be achieved by asking the patient some hypothetical questions such as
the following:
What happens in the future if you begin to feel some of the old symptoms? What
would you do?
If you begin to have thoughts that you may ‘pass out’, how can you respond to
that thought?
In the future, do you think it would be helpful to start using your old (safety)
behaviors again?
It is also worth normalizing with the patient that experience of panic and anxiety
may be something that will occur, as it does for many people. It is important to view
this not as a relapse but as a good chance to practice and develop skills learned.
It is also worth giving the patient copies of the formulation and treatment
techniques to review in the future if any issues arise. What may be even more
beneficial is giving the patient the option of recording each session, so that they have
an actual copy of everything that has occurred throughout the treatment. This
enables the patient to re-listen and strengthen their understanding post-treatment.
Key Learning Points
• Assessment of panic disorder requires detailed analysis of the key components
that maintain the disorder. Use the model to guide assessment.
• Formulation and socialization to the model are essential to help the patient
understand how panic disorder is maintained. It is also a powerful change
mechanism in itself.
CBT for Panic Disorder
(cont.)
• Both cognitive and behavioral change methods should be derived from the
idiosyncratic case formulation and be specific to the patient’s presentation.
• Homework is essential to help the patient discover that feared catastrophes do
not occur in their own environment.
• If residual beliefs exist towards the end of therapy, relapse prevention should
focus on modifying these before therapy ends.
RECOMMENDED READING
Bennett-Levy, J. E., Butler, G. E., Fennell, M. E., Hackman, A. E., Mueller, M. E., &
Westbrook, D. E. (2004). Oxford guide to behavioral experiments in cognitive therapy.
Oxford University Press.
Bennett-Levy, J., Turner, F., Beaty, T., Smith, M., Paterson, B., & Farmer, S. (2001). The
value of self-practice of cognitive therapy techniques and self-reflection in the training of
cognitive therapists. Behavioral and Cognitive Psychotherapy, 29(2), 203–220.
Clark, D. M. (1996). Panic disorder: From theory to therapy. In P. M. Salkovskis (Ed.),
Frontiers of cognitive therapy. Guilford Publications, 318–344.
Hackmann, A. (1998). Cognitive therapy with panic and agoraphobia: Working with
complex cases. In N. Tarrier, A. Wells, & G. Hoddock (Eds.), Treating complex cases.
Wiley, 27–43.
Livermore, N., Sharpe, L., & McKenzie, D. (2010). Panic attacks and panic disorder in
chronic obstructive pulmonary disease: A cognitive behavioral perspective. Respiratory
Medicine, 104(9), 1246–1253.
Tully, P. J., Sardinha, A., & Nardi, A. E. (2017). A new CBT model of panic attack treatment
in comorbid heart diseases (PATCHD): How to calm an anxious heart and
mind. Cognitive and Behavioral Practice, 24(3), 329–341.
Wells, A. (1997). Cognitive therapy of anxiety disorders: A practice manual and conceptual
guide. John Wiley and Sons.
REFERENCES
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.) (DSM-5). American Psychiatric Association.
Barlow, D. H. (1988). Anxiety and its disorders. Guilford Press.
Barlow, D. H., Gorman, J. M., Shear, M. K., & Woods, S. W. (2000). Cognitive-behavioral
therapy, imipramine, or their combination for panic disorder: A randomized controlled
trial. JAMA, 283(19), 2529–2536.
Beck, A. T., Emery, G., & Greenberg, R. L. (1985). Anxiety disorders and phoblas:
A cognitive perspective. Basic Books.
Bennett-Levy, J. (2003). Mechanisms of change in cognitive therapy: The case of automatic
thought records and behavioral experiments. Behavioral and Cognitive Psychotherapy,
31(3), 261–277.
Bennett-Levy, J. E., Butler, G. E., Fennell, M. E., Hackman, A. E., Mueller, M. E., &
Westbrook, D. E. (2004). Oxford guide to behavioral experiments in cognitive therapy.
Oxford University Press.
Bennett-Levy, J., Turner, F., Beaty, T., Smith, M., Paterson, B., & Farmer, S. (2001). The
value of self-practice of cognitive therapy techniques and self-reflection in the training of
cognitive therapists. Behavioral and Cognitive Psychotherapy, 29(2), 203–220.
71
72
Robin D. Bailey
Bernstein, A., Zvolensky, M. J., Sachs-Ericsson, N., Schmidt, N. B., & Bonn-Miller, M. O.
(2006). Associations between age of onset and lifetime history of panic attacks and
alcohol use, abuse, and dependence in a representative sample. Comprehensive
Psychiatry, 47(5), 342–349.
Casey, L. M., Oei, T. P., & Newcombe, P. A. (2004). An integrated cognitive model of panic
disorder: The role of positive and negative cognitions. Clinical Psychology Review, 24(5),
529–555.
Chambless, D. L., Caputo, G. C., Bright, P., & Gallagher, R. (1984). Assessment for fear of
fear in agoraphobics: The Body Sensations Questionnaire and the Agoraphobia
Cognitions Questionnaire. Journal of Consulting and Clinical Psychology, 52, 1090–1097.
Chambless, D. L., Caputo, G. C., Jasin, S. E., Gracely, E. J., & Williams, C. (1985). The
mobility inventory for agoraphobia. Behavior Research and Therapy, 23, 35–44.
Clark, D. M. (1986). A cognitive approach to panic. Behavior Research and Therapy, 24(4),
461–470.
Clark, D. M. (1988). A cognitive model of panic attacks. In S. Rachman & J. D. Maser
(Eds.), Panic: Psychological perspectives (pp. 71–89). Lawrence Erlbaum Associates.
(1996). Panic disorder: From theory to therapy. In: P. M. Salkovskis (Ed.), Frontiers of
cognitive therapy. Guilford Publications, 318–344.
Clark, D. M., & Salkovskis, P. (2009). Cognitive therapy for panic disorder: Manual for IAPT
high intensity therapists.
Clark, D. M., Salkovskis, P. M., Hackmann, A., Wells, A., Ludgate, J., & Gelder, M. (1999).
Brief cognitive therapy for panic disorder: A randomized controlled trial. Journal of
Consulting and Clinical Psychology, 67(4), 583.
De Cort, K., Hermans, D., Noortman, D., Arends, W., Griez, E. J., & Schruers, K. R. (2013).
The weight of cognitions in panic: The link between misinterpretations and panic
attacks. PloS One, 8(8).
De Jonge, P., Roest, A. M., Lim, C. C., Florescu, S. E., Bromet, E. J., Stein, D. J., & AlHamzawi, A. O. (2016). Cross-national epidemiology of panic disorder and panic attacks
in the world mental health surveys. Depression and Anxiety, 33(12), 1155–1177.
Deacon, B., & Abramowitz, J. S. (2008). Is hypochondriasis related to obsessive-compulsive
disorder, panic disorder, or both? An empirical evaluation. Journal of Cognitive
Psychotherapy, 22(2), 115–127.
Flynn, D., Ford, G. A., Rodgers, H., Price, C., Steen, N., & Thomson, R. G. (2014).
A time series evaluation of the FAST national stroke awareness campaign in England.
PLoS One, 9(8).
Furukawa, T. A., Watanabe, N., & Churchill, R. (2007). Combined psychotherapy plus
antidepressants for panic disorder with or without agoraphobia. Cochrane Database of
Systematic Reviews, (1).
Goodwin, R. D., Brook, J. S., & Cohen, P. (2005). Panic attacks and the risk of personality
disorder. Psychological Medicine, 35(2), 227–235.
Goodwin, R. D., Fergusson, D. M., & Horwood, L. J. (2004a). Panic attacks and
psychoticism. American Journal of Psychiatry, 161(1), 88–92.
(2004b). Panic attacks and the risk of depression among young adults in the
community. Psychotherapy and Psychosomatics, 73(3), 158–165.
Goodwin, R. D., & Gotlib, I. H. (2004). Panic attacks and psychopathology among youth.
Acta Psychiatrica Scandinavica, 109(3), 216–221.
Hiller, W., Leibbrand, R., Rief, W., & Fichter, M. M. (2005). Differentiating hypochondriasis
from panic disorder. Journal of Anxiety Disorders, 19(1), 29–49.
Hoffart, A. (2016). Cognitive models for panic disorder with agoraphobia: A study of
disaggregated within-person effects. Journal of Consulting and Clinical Psychology,
84(9), 839.
CBT for Panic Disorder
Hoffart, A., Sexton, H., Hedley, L. M., & Martinsen, E. W. (2008). Mechanisms of change in
cognitive therapy for panic disorder with agoraphobia. Journal of Behavior Therapy and
Experimental Psychiatry, 39(3), 262–275.
Houck, P. R., Spiegel, D. A., Shear, M. K., & Rucci, P. (2002). Reliability of the self-report
version of the panic disorder severity scale. Depression and Anxiety, 15(4), 183–185.
Huppert, J. D., Kivity, Y., Barlow, D. H., Gorman, J. M., Shear, M. K., & Woods, S. W.
(2014). Therapist effects and the outcome–alliance correlation in cognitive
behavioral therapy for panic disorder with agoraphobia. Behavior Research and
Therapy, 52, 26–34.
Kim, Y. W., Lee, S. H., Choi, T. K., Suh, S. Y., Kim, B., Kim, C. M., . . . & Song, S. K. (2009).
Effectiveness of mindfulness-based cognitive therapy as an adjuvant to pharmacotherapy
in patients with panic disorder or generalized anxiety disorder. Depression and
Anxiety, 26(7), 601–606.
King, M., Nazareth, I., Levy, G., Walker, C., Morris, R., Weich, S., . . . & Rifel, J. (2008).
Prevalence of common mental disorders in general practice attendees across
Europe. The British Journal of Psychiatry, 192(5), 362–367.
Lewis-Fernández, R., Hinton, D. E., Laria, A. J., Patterson, E. H., Hofmann, S. G., Craske,
M. G., . . . & Liao, B. (2011). Culture and the anxiety disorders: Recommendations for
DSM-V. Focus, 9(3), 351–368.
Livermore, N., Sharpe, L., & McKenzie, D. (2010). Panic attacks and panic disorder in
chronic obstructive pulmonary disease: A cognitive behavioral perspective. Respiratory
Medicine, 104(9), 1246–1253.
NICE. (2011). Common Mental Health Disorders: Identification and Pathways to Care.
Clinical Guideline 123. National Institute for Health and Care Excellence.
Noyes, R., Reich, J., Clancy, J., & O’Gorman, T. W. (1986). Reduction in hypochondriasis
with treatment of panic disorder. The British Journal of Psychiatry, 149(5), 631–635.
Olatunji, B. O., Deacon, B. J., Abramowitz, J. S., & Valentiner, D. P. (2007). Body vigilance
in nonclinical and anxiety disorder samples: Structure, correlates, and prediction of
health concerns. Behavior Therapy, 38(4), 392–401.
Pergamin-Hight, L., Naim, R., Bakermans-Kranenburg, M. J., van IJzendoorn, M. H., & BarHaim, Y. (2015). Content specificity of attention bias to threat in anxiety disorders:
A meta-analysis. Clinical Psychology Review, 35, 10–18.
Pompoli, A., Furukawa, T. A., Efthimiou, O., Imai, H., Tajika, A., & Salanti, G. (2018).
Dismantling cognitive-behavior therapy for panic disorder: A systematic review and
component network meta-analysis. Psychological Medicine, 48(12), 1945–1953.
Pompoli, A., Furukawa, T. A., Imai, H., Tajika, A., Efthimiou, O., & Salanti, G. (2016).
Psychological therapies for panic disorder with or without agoraphobia in adults:
A network meta-analysis. Cochrane Database of Systematic Reviews, (4).
Reinecke, A., Cooper, M., Favaron, E., Massey-Chase, R., & Harmer, C. (2011). Attentional
bias in untreated panic disorder. Psychiatry Research, 185(3), 387–393.
Reiss, S., & McNally, R. J. (1985). The expectancy model of fear. In S. Reiss & R. R. Bootzin
(Eds.), Theoretical issues in behavior therapy (pp. 107–122). Academic Press.
Salkovskis, P. M. (1991). The importance of behavior in the maintenance of anxiety and
panic: A cognitive account. Behavioral and Cognitive Psychotherapy, 19(1), 6–19.
Salkovskis, P. M., & Clark, D. M. (1993). Panic disorder and hypochondriasis. Advances in
Behavior Research and Therapy, 15(1), 23–48.
Salkovskis, P. M., Clark, D. M., & Gelder, M. G. (1996). Cognition-behavior links in the
persistence of panic. Behavior Research and Therapy, 34(5–6), 453–458.
Salkovskis, P. M., Clark, D. M., Hackmann, A., Wells, A., & Gelder, M. G. (1999). An
experimental investigation of the role of safety-seeking behaviors in the maintenance of
panic disorder with agoraphobia. Behavior Research and Therapy, 37(6), 559–574.
73
74
Robin D. Bailey
Salkovskis, P. M., Hackmann, A., Wells, A., Gelder, M. G., & Clark, D. M. (2007). Belief
disconfirmation versus habituation approaches to situational exposure in panic disorder
with agoraphobia: A pilot study. Behavior Research and Therapy, 45(5), 877–885.
Shear, M. K., Brown, T. A., Barlow, D. H., Money, R., Sholomskas, D. E., Woods, S. W., . . .
& Papp, L. A. (1997). Multicenter collaborative panic disorder severity scale. American
Journal of Psychiatry, 154(11), 1571–1575.
Teachman, B. A., Marker, C. D., & Clerkin, E. M. (2010). Catastrophic misinterpretations as
a predictor of symptom change during treatment for panic disorder. Journal of
Consulting and Clinical Psychology, 78(6), 964.
Tully, P. J., Sardinha, A., & Nardi, A. E. (2017). A new CBT model of panic attack treatment
in comorbid heart diseases (PATCHD): How to calm an anxious heart and
mind. Cognitive and Behavioral Practice, 24(3), 329–341.
Vroom, V. H. (1964). Work and motivation. Wiley & Sons.
Wells, A. (1997). Cognitive therapy of anxiety disorders: A practice manual and conceptual
guide. John Wiley and Sons.
Williams, P. G. (2004). The psychopathology of self-assessed health: A cognitive approach to
health anxiety and hypochondriasis. Cognitive Therapy and Research, 28(5), 629–644.
Wolpe, J., & Rowan, V. C. (1988). Panic disorder: A product of classical
conditioning. Behavior Research and Therapy, 26(6), 441–450.
Zickgraf, H. F., Chambless, D. L., McCarthy, K. S., Gallop, R., Sharpless, B. A., Milrod, B. L.,
& Barber, J. P. (2016). Interpersonal factors are associated with lower therapist adherence in cognitive-behavioral therapy for panic disorder. Clinical Psychology and
Psychotherapy, 23(3), 272–284.
5
The Cognitive Behavioral Treatment
of Agoraphobia
Vijaya Manicavasagar and Derrick Silove
Although many people may experience periods of anxiety and avoidance behaviors in response to situational stressors, only a minority develops agoraphobia, a
chronic condition involving widespread avoidance of places such as shopping
centres, bridges, tunnels, and crowds. Despite these lifestyle limitations, most
people with agoraphobia seek treatment only when their symptoms become highly
restrictive or they become overly dependent on others to pursue everyday activities (Greenberg et al., 1999; Lepine, 2003). The development of agoraphobia
often reflects a downward spiral characterized by a ‘fear of fear’, in which the
anticipation of anxiety leads to increasing avoidance behaviors and vice versa,
creating a vicious circle.
The key concern amongst people with agoraphobia is a fear that they will develop
severe physical symptoms in certain situations where escape or obtaining help from
others is impossible. In some instances, the presence of a trusted companion can
contain or lessen their fears (American Psychiatric Association, 2013 [DSM-5]). In
order to receive a full DSM-5 diagnosis of agoraphobia, a person must experience
significant psychosocial distress or disability related to their symptoms and the
problem must persist for at least six months. Once entrenched, agoraphobia is
associated with diminished quality of life and increased use of healthcare services.
These core features distinguish agoraphobia from the other anxiety disorders
that share superficial similarities. For example, in separation anxiety disorder, core
fears revolve around being separated from or harm befalling key attachment
figures, or being away from places of security such as the home (Bowlby, 1973); in
social phobia, feelings of embarrassment and negative appraisal by others leads to
avoidance of social interactions; and in obsessive-compulsive disorder, fears of
contamination or contagion may lead to avoidance of public places. In posttraumatic stress disorder, people avoid places which remind them of their original
trauma and/or which trigger traumatic memories (flashbacks). People experiencing
severe depression may avoid leaving home because they feel hopeless or lack
motivation and the energy to do so; and persons with psychotic illnesses may be
fear persecution or harm by others when venturing away from the house.
75
76
Vijaya Manicavasagar and Derrick Silove
Historical Context
The term ‘agoraphobia’, first coined by Westphal in the 1870s (Kuch & Swinson,
1992), was applied to people who experienced unexpected and situation-specific
panic attacks in public places, empty streets, bridges, and crowds. They typically
experienced anticipatory anxiety (‘fear of fear’) and sudden physical incapacitation
(such as gastrointestinal upset, dizziness, and difficulty breathing) in these situations
(Kuch & Swinson, 1992). In the 1970s, the focus turned to panic disorder which then
was regarded as the primary cause of agoraphobic symptoms, a model reflected in
the third edition of the Diagnostic and Statistical Manual of Mental Disorders
(American Psychiatric Association, 1980) (Barlow, 2002). Since then, debate has
persisted regarding the relationship of the two disorders. In DSM-5 (American
Psychiatric Association, 2013) and the eleventh edition of the International
Classification of Diseases (ICD-11; World Health Organization, 2018), agoraphobia
and panic disorder are classified as separate categories. Nevertheless, in clinical
practice, it is common to trace the onset of avoidant symptoms of agoraphobia to
pre-existing fear of experiencing panic attacks or at least panic-like symptoms when
approaching or entering the relevant locations.
Prevalence
The aforementioned serial changes in the classification of agoraphobia and its
association with panic disorder has contributed to the variability in prevalence rates
identified for the two disorders over time. Population studies prior to DSM-5
suggested that a third of people with panic disorder develop agoraphobia, whereas
agoraphobia without a history of panic attacks appeared to be relatively uncommon
(Sanchez-Meca et al., 2010). For example, a national epidemiological study in the
United States (Kessler et al., 2006) found a lifetime prevalence for panic disorder
with agoraphobia of 1.1%, whereas only 0.5% of participants reported agoraphobia
alone (Robins & Regier, 1991). The peak age of expression of agoraphobia is
between 20 and 30 years; woman make up three quarters of cases (Yonkers et al.,
1998) and also tend to develop more severe forms of the disorder (Turgeon et al.,
1998). Relatives of persons with agoraphobia are more likely to develop the disorder (Crowe et al., 1983), a pattern not seen in panic disorder alone (Rapee &
Murrell, 1988).
Causes and Pathways
Most people with agoraphobia who seek treatment can recall a clear point of
onset of anxiety (Aronson & Logue, 1987). At first, symptoms are commonly
misinterpreted as indicating serious physical illness, generating further anxiety
(Clark, 1986), a spiral that contributes to avoidance of situations in which
symptoms occur.
Cognitive Behavioral Treatment of Agoraphobia
Cognitive Distortions
Cognitive processes, or the way people think about and interpret events, can play a
key role in the maintenance of agoraphobia. In particular, the belief about the
meaning and implication of symptoms may be instrumental in generating anticipatory anxiety and avoidance. Extreme or catastrophic cognitions, such as a conviction
that the person will die, go mad, soil themselves, vomit, faint, or collapse if they
enter a certain place, can exacerbate symptoms. An important cognition that adds to
the spiral of avoidance is that the person will not be able to control themself,
resulting in chaotic or embarrassing behavior (Cox et al., 1995; Craske & Barlow,
1988; Craske et al., 1988; Telch et al., 1989). Evidence from experimental studies
suggests that cognitive distortions are more common amongst persons with agoraphobia; for example, persons with the disorder are more likely to interpret ambiguous physical sensations and novel situations as threatening and anxiety-provoking
than those who do not experience anxiety (McNally & Foa; 1987).
In summary, the types of cognitions that maintain agoraphobia include the
following:
Expecting negative outcomes if the person approaches or enters a particular
situation, including fear of becoming overwhelmingly distressed, experiencing a
catastrophic illness, or acting in an embarrassing way
Fear of uncontrollable anxiety symptoms, leading to collapse or madness
A conviction that the person will not be able to manage or cope with anxiety,
often associated with characteristics such as low self-confidence, poor coping
strategies, and an impaired sense of self-efficacy.
Conditioning to Situational Cues
Classical conditioning, in which repetitive exposure to environmental stimuli
becomes paired with adverse experiences such as anxiety, can lead to associative
learning. The consequence is that returning to the same or a similar situation can
provoke a fear reaction, even if there is no objective danger in that location. For
example, an individual who experienced severe anxiety symptoms at work in
response to a situational stress (e.g., an altercation with a manager) may develop
a conditioned learnt response in which neutral cues, such as the journey to work or
the physical workplace itself, trigger anxiety and avoidance.
Clinical Assessment and Tools
When assessing a person with suspected agoraphobia, it is important to obtain
precise information about the core symptoms including timing of onset, progression,
and the pattern of fear, anxiety, and avoidance as it evolved over time.
Differentiating agoraphobic from other forms of avoidance observed in other
anxiety subtypes is essential to making an accurate diagnosis. A detailed account
77
78
Vijaya Manicavasagar and Derrick Silove
should be elicited of factors that contribute to the onset and perpetuation of the
symptoms, including life stressors at work, home, and the wider environment. The
developmental history should also include inquiry into the style of parenting experienced in childhood, including evidence of overprotectiveness or neglect. Sensitive
inquiry should also be made into past experiences of trauma and abuse. A detailed,
chronological history of how avoidance behaviors developed, unfolded, and fluctuated, and how they relate to situational cues and life stressors, may provide useful
insights into what approaches and possible lifestyle modifications may be worth
pursuing to change the associated maladaptive responses and behaviors.
Assessment of the impact of avoidance is made in relation to all aspects of the
person’s life, that is, in the personal, social, work, study, and leisure domains.
Physical symptoms of anxiety need to be differentiated from those of organic
disorders which may be independent of or contribute to anxiety. Special attention
should be given to conditions known to cause or mimic anxiety, such as thyroid or
other endocrine diseases, cardiac arrhythmias or other heart conditions (such as
mitral valve disease), and drug withdrawal syndromes. A history of alcohol and drug
use (both prescribed and non-prescribed) is important to determine whether these
substances may be contributing to anxiety and/or providing relief to the person. Up
to a third of men and one in 10 women with agoraphobia misuse drugs or alcohol
(Markowitz et al., 1989). These substances and the complications from their use can
be a factor in the causal pathway to agoraphobia or represent an attempt at ‘selfmedication’, that is, to reduce anxiety. Invariably, the early transient anxietyreducing effect is lost, and, instead, escalating use of the drug or alcohol exacerbates
or complicates avoidant tendencies.
Comorbidity needs to be assessed, in relation to both past and concurrent
disorders. Agoraphobia commonly is comorbid with a range of other anxiety or
depressive disorders and, as indicated, with drug and alcohol misuse. If possible,
causal links should be identified amongst comorbid disorders, that is, which disorder
appears to have preceded and triggered the other, and identification of the disorder
that is dominant at the time of assessment. Psychiatric complications, such as the
onset of depressive symptoms or disorder and especially suicidal feelings/urges,
require close attention and may demand urgent intervention. Assessing personality
characteristics is important, including features of perfectionism, dependency, or
borderline behaviors, given that these traits are likely to play a role in determining
the prognosis and in the capacity of the person to engage in and adhere
to treatment.
When a high level of diagnostic reliability and validity is required, such as in
epidemiological studies and clinical trials, structured diagnostic interviews may be
used. The Structured Clinical Interview for DSM-5 (SCID-5; First et al., 2015) is one
of the most commonly used comprehensive assessment tools. Self-report measures
may also be used as an adjunct to the clinical interview or to gauge varying levels of
agoraphobic avoidance and the frequency and intensity of physical symptoms and
dysfunctional negative thoughts. Some of the more frequently used measures that
are specific to the assessment of agoraphobia are listed as follows:
Cognitive Behavioral Treatment of Agoraphobia
Agoraphobic Cognitions Questionnaire (ACQ) (Chambless et al., 1984) comprises 14 items (plus an open question) rated on a five-point Likert-type scale
measuring the frequency of catastrophic thoughts.
Body Sensations Questionnaire (BSQ) (Chambless et al., 1984) has 18 items
rated on a five-point Likert-type scale measuring fear of physical sensations.
Mobility Inventory for Agoraphobia (MIA) (Chambless et al., 1985) is a 27-item
scale which includes two agoraphobic avoidance scales measuring the severity
of phobic avoidance and the frequency of panic attacks depending on whether
the participant is alone (MIA-Alone) or accompanied (MIA-Accompanied).
Other measures that are frequently used tend to focus on panic attacks and include
the Panic Agoraphobia Scale (PAS; Bandelow, 1997) and the Anxiety Sensitivity
Index (ASI; Reiss, et al., 1986). The Panic and Agoraphobia Scale (P&A;
Bandelow, 1995) is a short clinician- or client-rated scale that covers panic frequency, severity, and duration; panic-related phobias; anticipatory anxiety; impairment and general quality of life; and global problem severity. A similar measure
is the clinician-scored diary the Panic-Associated Symptoms Scale (PASS; Scupi
et al., 1992).
Evidence-Based Treatments for Agoraphobia
Cognitive behavioral therapy (CBT) is the mainstay of therapy for agoraphobia.
This approach generally offers lower treatment costs and greater access than other
treatment modalities. An extensive body of research has shown that CBT, whether
individually delivered or group-based, is generally effective in treating agoraphobia
(Barlow et al., 2000; Hahlweg et al., 2001).
In summary, CBT interventions typically include a package of inter-related
components, including psychoeducation, normalizing symptoms and experiences
of anxiety, cognitive restructuring, slow breathing exercises, strategies to reduce
general physical symptoms of anxiety, exposure to interoceptive stimuli (e.g., rapid
heart rate or sweating), and gradual in vivo exposure to avoided situations.
Ultimately, CBT aims to encourage people with agoraphobia to utilize more adaptive thought patterns and behaviors (Craske et al., 2008) as the foundation
for recovery.
Psychoeducation
Understanding the nature of agoraphobia and the pathways leading to and maintaining the problem helps the person to gain a sense of mastery in coping with the
disorder. Psychoeducation about the physical manifestations of anxiety can help to
alleviate unrealistic and catastrophic fears while helping the person with anxiety to
understand the specific triggers and stressors that contribute to anxiety and avoidance behaviors. This assists in anticipating and using pre-emptive strategies to
79
80
Vijaya Manicavasagar and Derrick Silove
manage stressful situations (Craske & Barlow, 1988; Goldstein & Chambless, 1978;
Zarate et al., 1990).
Cognitive Therapy
Cognitive therapy helps people to address dysfunctional thoughts that lead to or
exacerbate symptoms, giving them confidence in their ability to cope with panic and
anxiety symptoms and to curb negative thoughts about themselves regarding their
capacity to overcome lifestyle restrictions and to reduce their dependence on others.
Thought challenging and cognitive restructuring can be used at various points in
therapy, for example, to re-appraise thoughts around anticipatory anxiety and
resistance to engage in exposure, and to address any negative thoughts regarding
their performance and progress in therapy.
Addressing Arousal
A key focus of treatment is to reduce high levels of autonomic arousal and acute
anxiety symptoms. Commonly used methods include hyperventilation control,
meditation or mindfulness, and relaxation exercises. These techniques relieve symptoms and facilitate the implementation of the exposure hierarchy by providing a
method to achieve de-arousal which can be used in vivo.
Exposure Therapy
Graded exposure to feared situations is a key component of CBT for agoraphobia
(Mattick et al., 1990; Teusch and Boehme, 1999). Exposure therapy is grounded on
the principles of habituation and extinction of learned fear responses. The assumption is that intrinsically non-harmful situations or places cannot continue to evoke
fear or anxiety if the person is repeatedly exposed to these conditions without any
adverse consequences. A hierarchy of situations is developed with the person in
which feared or avoided situations are ranked according to the severity of the
anxiety they provoke. It can be useful to apply a simple numerical rating scale
to identify the order of the hierarchy (e.g., on a visual analogue scale ranging
from 0 to 10). Additional questions that can guide anxiety ratings may include
‘What would make this worse?’ and ‘What would make this better?’
The person then is guided through a systematic program aimed at reducing
anxiety and avoidance in a progressive manner by graduated and repeated exposure
to the designated places or conditions listed in the hierarchy, starting with the least
anxiety-provoking items and moving serially up to the most severe. Remaining in
the feared situation long enough for the fear response to dissipate is important to
reverse conditioning. Ideally, the person should be there long enough for the
anxiety rating to reduce by at least 2 or 3 points during exposure exercises, a process
which may take 20–30 minutes to achieve. If in vivo exposure is not possible or can
be undertaken only infrequently, then exposure in imagination can be used
Cognitive Behavioral Treatment of Agoraphobia
(imaginal exposure) as a first step. Therapist-guided in vivo exposure, where the
professional initially accompanies the patient to coach and guide them through
specific situations (Gloster et al., 2011), remains a mainstay of therapy. In some
situations, a trusted companion can substitute for the therapist, but it is important to
gauge whether this strategy may increase dependency and how and when the
companion should withdraw from the process to ensure that the person becomes
autonomous in confronting the feared situation. Increasingly, virtual reality–based
treatments can be used, which are time-efficient and safe and allow direct monitoring of physiological arousal (Botella et al., 2004).
Throughout the process, the challenge for the person and the therapist is to
overcome in a sustained manner the initial discomfort incurred by confronting the
feared situation(s) on the hierarchy. The participant needs to be prepared for
possible setbacks and periods in which the task feels overwhelming to avoid feeling
discouraged and giving up by retreating to safe but maladaptive behaviors such as
finding reasons to stay at home or insisting on having a companion accompany them
on outings. These forms of resistance should be anticipated, discussed, and monitored from the outset to avoid treatment failure or dropout.
Other Strategies
Initially, the use of safety behaviors can facilitate the first stages of exposure, for
example, resorting to sipping water, carrying anxiolytic medication in a bag, or
talking to a friend on a mobile phone. Later in therapy, these behaviors should be
discussed and gradually withdrawn once the person’s confidence in managing anxiety has consolidated. Otherwise, improvements in mobility may be transitory.
Engaging in structured problem-solving to address specific impediments to pursuing
the exposure program and goal-setting to help maintain motivation to work through
the hierarchy can be used throughout the treatment process.
Homework tasks include practicing in vivo exposure and thought-challenging.
Again, engaging family members or friends in the process can facilitate motivation
and progress as long as clear instructions are given to these persons concerning the
extent and limitations of assistance that should be provided.
Evidence
In combination, cognitive therapy (Fava et al., 2001; Gloster et al., 2011) and
exposure (Barlow et al., 1984; Hahlweg et al., 2001; Michelson et al., 1985) appear
to achieve the best outcomes for agoraphobia (Mattick et al., 1990; Michelson et al.,
1985). The prognosis is less favourable if there is a pattern of comorbidity with other
common mental disorders related to depression, anxiety, and substance use; where
traits of personality disorder are evident and interfere with regular practicing of
skills; and/or where there are social or environmental barriers to focusing on
therapy, for example, in the presence of an unsupportive partner or family
(Gloster et al., 2015).
81
82
Vijaya Manicavasagar and Derrick Silove
In general, although most persons with agoraphobia respond positively to CBT,
a quarter gain only limited symptom relief (McPherson et al., 1980; Munby &
Johnston, 1980). For these people, other treatment approaches may be worth trying,
including acceptance and commitment therapy (Gloster et al., 2015) or psychodynamic approaches (Keefe et al., 2014) that address the factors that impede
motivation or generate resistance to engaging in CBT. At the same time, chronic,
treatment-resistant cases encountered in clinical practice usually involve persons
with long-term entrenched symptoms and a combination of the poor prognostic
factors listed above.
Medications Used to Manage Agoraphobia
Anxiety-reducing medications (tranquilizers) such as the benzodiazepines (diazepam, alprazolam) can help to reduce both state anxiety and anticipatory anxiety and
are frequently used in the treatment of agoraphobia. Some patients find that simply
having a tablet of diazepam in their bag helps them to have the confidence to go out.
Others take a benzodiazepine strategically, that is, just as they are anticipating
leaving the house. Problems of addiction, tolerance, and withdrawal symptoms
caution against using these medications for prolonged periods of time. Their use
should be restricted to the short term where the person’s anxiety or distress is so
overwhelming that they cannot engage in psychological interventions, or, for
patients with no risk of addiction, on an occasional basis (not more than twice a
week) for insomnia or to deal with specific anxiety-provoking situations.
The main classes of drugs used (loosely referred to as antidepressants) are the
selective serotonin reuptake inhibitors (SSRIs) such as sertraline, paroxetine, and
fluoxetine, or serotonin and noradrenalin (also known as norepinephrine) reuptake
inhibitors (SNRIs) such as mirtazapine, venlafaxine, and duloxetine, especially
where depression is a comorbid feature. In situations where there is a restriction
on availability of psychotropic medications, first-generation tricyclic medications
(doxepin, imipramine, nortriptyline) and, less commonly, the monoamine oxidase
inhibitors, such as phenelzine, can be used. Or these latter classes of drugs may be
trialled in treatment-resistant cases. Although CBT and medication offer similar
short-term outcomes, the former appears to result in longer-term maintenance of
gains, whereas discontinuation of medication is often associated with higher relapse
rates (Fava et al., 2001; Gould et al., 1995).
Although various chemical augmentation methods have been trialled to
improve the treatment response to CBT techniques, particularly exposure therapy,
there is little evidence as yet that these approaches are useful for agoraphobia.
Concerns have been raised that the use of psychotropic medications may impede
learning when CBT methods are used, but so far the evidence remains slender
(Basoglu et al., 1994). Clinical judgement therefore needs to be applied in deciding
whether or not to prescribe medication on an individual basis, taking into account
the severity of the problem, the capacity of the person to engage in CBT given their
level of anxiety, the preferences of the patient, and the risk of adverse effects.
Cognitive Behavioral Treatment of Agoraphobia
Case Vignette
ND was a 46-year-old married woman employed in a government service but
currently on leave following a workplace incident in the year prior to seeking
treatment. She had consulted a psychiatrist and a counsellor in the previous four
months. ND had lost confidence in herself and in her abilities, had experienced
repeated panic attacks, and, increasingly, agoraphobic avoidance. She was developing a pattern of avoiding driving, shopping, and even walking to the front gate to
collect her mail.
ND had been married for almost 20 years and had three adult sons who lived
elsewhere but whom she saw on a weekly basis. She was also involved in the care of
her elderly mother. She had been treated for cancer on two separate occasions
several years prior to the work incident and had made a full recovery.
Her agoraphobic avoidance had increased her reliance on her husband to take
her to appointments and to go shopping. ND’s husband worked long hours involving variable timing of shifts which made it difficult to plan ahead, leading to
arguments and considerable strain in their previously stable marriage.
Although there was no previous history of formal psychiatric disorder, it
became evident that ND had an obsessional personality style manifesting in controlling behaviours (such as keeping track of her husband’s whereabouts and closely
monitoring her adult sons’ activities). Associated features were intolerance of
uncertainty and unrealistic expectations of herself and others to behave in a what
she considered to be a ‘highly moral’ way at all times. Nevertheless, prior to the
development of her anxiety, ND was a highly independent woman holding a
responsible job.
Outline of ND’s Treatment
Phase 1: Setting the Stage for Change
After a clinical history was obtained, the next step was to examine and address the
interpersonal factors that could be contributing to her anxiety and avoidance
behaviors, such as uncertainty regarding her husband’s availability. Attention was
given to planning and regularizing activities requiring her husband’s help such as
shopping and keeping appointments with medical specialists. Other issues related to
her concerns about her diet, lack of exercise, and interpersonal difficulties. Some of
these could be addressed with simple problem-solving and encouraging ND to
discuss these issues with her husband. ND’s pride in maintaining a healthy organic
diet for herself and her family was also a source of tension given that shopping for
these specialized groceries required her husband to accompany her. ND was reasonably fit and healthy prior to the development of her agoraphobia but had
reduced her level of exercise because she could not drive to the gym or go for
walks. She had also developed high blood pressure, headaches, and severe muscle
tension in her neck. Her sleep was poor and she found herself unable to relax even
83
84
Vijaya Manicavasagar and Derrick Silove
while at home when her husband was at work. Her other stresses mainly involved
looking after her elderly mother and providing emotional support to her sons, who
were both in the process of changing jobs. Although she had some close friends with
whom she met on a regular basis prior to the development of her agoraphobia, ND
had stopped calling them and declined invitations to meet up with them. Problem
solving was used to assess and, where possible, resolve or lessen these obstacles and
problems with the aim of allowing ND to return to a more fulfilling and
satisfying life.
During this phase, ND was instructed in some basic anxiety management skills,
including progressive muscle relaxation, mindfulness meditation, and slow
breathing. Moreover, the process of challenging her extreme cognitions was commenced, particularly her critical appraisal of herself as a failure because she could
not leave the house.
ND was guided in recording a hierarchy of anxiety-provoking situations, using
the 0–10 rating system. She found this task helpful as it organized her experiences of
anxiety and assisted in compartmentalizing the steps involved in addressing them in
sequence. Her list of anxiety-provoking situations was further refined by discussing
actions that increased or decreased the fear ratings, that is, the strategies she could
use to minimize anxiety prior to or on entering these situations.
ND also was encouraged to document the negative thoughts she experienced
that were associated with those anxiety-provoking situations. In addition to her selfcritical thoughts, she became frustrated with the time it was taking to recover,
challenging herself as to why she could not simply exert control over her anxiety.
She was encouraged to examine how these cognitive-driven frustrations actually
hampered the process of therapy, thereby reinforcing a vicious cycle of maintenance
of symptoms. Suggestions were offered to substitute rigidly negative thoughts with
more helpful and realistic appraisals of her progress in therapy.
Once ND had learned some anxiety management skills such as slow breathing,
she was encouraged to increase her exercise again given that physical activity in
itself could contribute to improving her mental health. ND was at first hesitant to
engage in activities that would lead to a perceptible increase in her heart rate or
shortness of breath. She came to recognize the contextual differences in her bodily
responses to physical activity as compared with the physiological arousal related to
acute anxiety, allowing her to resume practicing yoga at home.
Phase 2: Implementing Cognitive and Behavioral Techniques
In phase 2, when ND had completed planning her program of exposure and had
gained skills in cognitive restructuring, she commenced the task of confronting the
problem of shopping (rated around 5 or 6). She practiced exposure in imagination
while controlling her level of arousal with a slow-breathing exercise. The scene was
repeated up to three times in each session and ND was instructed to remain in the
imagined situation for as long as it took for her anxiety ratings to decrease. After
each imaginal exposure session, ND was encouraged to discuss the effectiveness of
Cognitive Behavioral Treatment of Agoraphobia
the exercise in controlling her anxiety, any unhelpful thoughts that occurred during
the exposure session, and her anxiety rating during the process. Once she achieved a
reasonable level of confidence, she began to practice exposure in vivo, her husband
being briefed to provide the necessary support by being physically present in the
first stages. ND was encouraged to practice this scenario as many times as possible
between therapy sessions and to report back to her therapist on the processes and
outcomes of that activity.
Phase 3: Working through the Exposure Hierarchy
Subsequent sessions focused on each of the items in the hierarchy, in imagination
during the session if necessary, then in vivo. Subjective ratings of anxiety were
obtained pre- and post-activity in order to monitor progress, ensure motivation
was maintained, and offer enough time to troubleshoot problem areas.
In phase 3, with practice, ND was eventually able to drive short distances on her
own to the gym and to appointments for herself or her mother. She rated her
anxiety before and after these events, noting the anxiety management techniques
that worked best for her in each situation. Over a period of time, the frequency of
her therapy sessions was reduced as ND increasingly worked on overcoming her
anxieties on her own. Her therapy sessions began to focus on troubleshooting
situations that remained problematic for her. During this time the tensions in her
relationship with her husband lessened as her ability to drive short distances
resulted in fewer arguments about his availability.
Over a period of 18 months, ND began to regain a sense of control over her life
and engage in activities that she had been avoiding. She began exercising regularly
and joined several group programmes, both recreational and therapeutic. Her
husband’s involvement in her treatment programme was tapering off. Her therapy
sessions focussed on supporting her positive change in self-image as an increasingly
independent and confident person. However, ND’s perfectionistic character traits
continued to limit her progress and affect other areas of her life, such as her
frustration over her mother’s increasing frailty, intolerance over her friends’ perceived unreliability in attending social engagements, and her compulsive need to
source specific types of foodstuffs despite the difficulties in obtaining them far from
her home and thus having to rely on her husband to drive her.
Comments about This Model for Treatment
In vivo exposure during treatment should resemble the real situations avoided by
individuals as much as possible. Furthermore, patients need to be guided to confront
these situations as frequently and as long as possible until the anxiety subsides.
Partners and other family members may be initially included in the exposure-based
program, but their involvement should be gradually tapered off if the patient is to
become more independent.
85
86
Vijaya Manicavasagar and Derrick Silove
After working through several items on the exposure hierarchy, some patients
may become ‘stuck’ and reach a point where they believe that they cannot proceed
because the next item on the list seems too difficult. If this happens, exploration of
the reasons for the resistance together with reversion to imaginal exposure may be
required. In some cases, specific difficulties associated with that particular situation
may need to be re-analyzed and addressed before attempting in vivo exposure.
Exposure to feared situations can bring about positive cognitive changes, and these
changes should be discussed during therapy sessions as they can facilitate further
improvements in avoidance behaviors and serve as a reminder to patients when they
become ‘stuck’ again.
The presence of the therapist during the early stages of in vivo exposure may
assist in initiating change when there is hesitation to do so. The therapist can alert
patients to the use of positive rather than maladaptive ‘safety signals’ and ‘safety
behaviors’ in real time while the provision of immediate feedback through modelling and verbal guidance can assist patients through challenging situations. Whilst
some (see Bandura, 1977) have cautioned against the use of safety behaviors, it may
be prudent to allow this practice early in the treatment program rather than to risk
discontinuation. However, the excessive presence of the therapist as a safety signal
for the patient ultimately can inhibit progress, so that timely withdrawal by the
therapist allowing the patient to attempt the same situations on their own is vital.
Facilitators of Therapy
Progress during therapy can be affected by several factors, including the extent to
which a person is willing to confront their anxiety during treatment (Perreault et al.,
2014). Other issues include patient expectations (whether realistic or not) about the
procedures, technique, format, and duration of treatment, as well as a commitment
to make a sustained effort. These issues can be related to personality characteristics
and the strength of family and social supports (Constantino, 2012). Patients also
may have idiosyncratic expectations about their prognosis (either excessively optimistic or pessimistic) and the overall degree and speed of recovery (Constantino,
2012). These issues were discussed with ND prior to the commencement of treatment and were revisited throughout the program.
Flexibility in the choice and timing of each intervention depends on the overall
formulation of the patient’s clinical presentation, capacities, and supports (familial,
environmental). If applied rigidly, manualized treatments, although popular, may
overlook individual differences in patient characteristics, which may complicate
treatment. However, if they are used with discretion, manuals can provide general
guidance and practical information for patients and clinicians alike.
Apart from general therapist factors, such as empathy and warmth, therapists
treating agoraphobia need to be skilled and experienced in CBT and familiar with
the medications used to treat both anxiety and depressive disorders, given the high
levels of comorbidity between these two disorders. The therapist must have a
capacity to assess and, if necessary, refer for other mental health problems (such
Cognitive Behavioral Treatment of Agoraphobia
as drug and alcohol misuse), social assistance, and medical assessment and intervention where necessary. Where treatment outcomes are unsatisfactory, consideration should be given to referral to therapists using other approaches (Barlow et al.,
2000). For example, Gloster et al. (2015) have reported success in using acceptance
and commitment therapy (ACT) in the treatment of agoraphobia with treatmentresistant patients. Engaging other health professionals, such as physiotherapists,
dietitians, and general practitioners, as part of a multidisciplinary team may also
help to facilitate change especially where physical and social factors may be
impacting on the illness course.
Inhibitors to Recovery
Inhibitors to recovery should be addressed explicitly during assessment, in the collaborative development of a management plan, and in the initial stages of treatment.
Although partners and family members can play a vital role in facilitating
recovery, they may also constrain progress during the therapy process. For example,
families may be critical of the patient for not being able to overcome their avoidance
behaviors within a short time frame. In other situations, families may collude
inadvertently with the person in justifying or compensating for avoidant behaviors.
For example, the network of dependency that occurs when one member of the
household experiences agoraphobia may lead to changes in family dynamics. Where
these factors are present, it is essential to hold special sessions with partners and
other family members (if the patient agrees) in order to ensure a shared understanding of the disorder, the type of family support needed to facilitate the treatment
plan, and the expected pace of progress (Cerny et al., 1987). Gently but consistently
confronting a sick-role attitude is vital to ensure that the person is motivated to
recover. This involves acknowledging the gravity of agoraphobia and the genuine
difficulties in overcoming avoidance but, at the same time, reinforcing the principle
that recovery is possible as long as there is a sustained and concerted effort from all
contributors (the person with agoraphobia, their family, and the therapist) in
working in the same direction. It may also be important to discuss the gains and
‘losses’ relating to recovery with the person and their support network, in order to
ensure that the losses, such as the sick role, are worth giving up for the gains, such as
a greater sense of independence.
A further complexity arises from multiple treatment providers and/or insurers
involved in situations where patients experience chronic anxiety problems, especially when compensation for work-related stressors is involved. Various professionals may inadvertently contradict each other, and adversarial medico-legal issues
(e.g., compensation-seeking) can retard recovery.
Post-therapy Complications
Agoraphobia often needs prolonged treatment, commencing with more intensive
and frequent sessions at the outset but tapering off to infrequent visits over several
87
88
Vijaya Manicavasagar and Derrick Silove
years. An attachment may form between patients and their therapists fostering
dependency, which is clearly problematic given that the aim of therapy is to
encourage independence and self-confidence. This issue can be addressed by
estimating the ‘blocks’ of sessions that may be required at each phase of treatment, regularly reviewing progress, issuing a mutually agreed-upon treatment
completion date, and anticipating the changes that the person will experience
once therapy is concluded. As with people experiencing any long-term anxiety
disorder, those with agoraphobia often develop other forms of emotional problems in their lives. For example, a study has reported that people with agoraphobia frequently develop depression following improvements in their anxiety
symptoms and avoidance behaviors, possibly because they become more aware
of the psychosocial costs incurred as a consequence of their previous level of
disability (Gloster et al., 2011). Furthermore, their relationships with the people
around them may change and, if not adequately managed, may result in increased
interpersonal stress.
Dropout from Treatment
Treatment attrition rates for agoraphobia range from 10% to 55% (Issakidis &
Andrews, 2004; White et al., 2010). Contributing factors include symptom severity
at outset and the presence of comorbidities such as depressive illness (Issakidis &
Andrews, 2004; White et al., 2010), sociodemographic variables such as lower
household income and education (Swift & Greenberg, 2012), negative attitudes to
past or ongoing treatment (Perreault et al., 2014), and difficulties in intimate
relationships (El-Baalbaki et al., 2010; Marcaurelle et al., 2005).
Remedial factors that need to be assessed if there appears to be a risk of
dropout include the patient’s lack of understanding of the nature, procedure, and
intended duration of the treatment program, especially feelings of being overwhelmed by the pace of treatment; motivational factors which may be reflected in
the personality of the patient; and factors within the family and social environment
that inhibit recovery or maintain avoidant behaviors.
Maintenance and Relapse Prevention
Relapse (i.e., return of fear and avoidance) after successful situational exposure is
usually related to the recurrence of anxiety symptoms (Arnow et al., 1985). Booster
sessions can be especially helpful to prevent full relapse, especially if they are
scheduled as soon as early warning signs are detected (such as increasing feelings
of tension or hesitancy in approaching situations after regaining the capacity to do
so). If possible, booster sessions can be tentatively scheduled during the course of
therapy. The availability of these sessions can be very reassuring to patients and can
always be cancelled if they are not required.
Cognitive Behavioral Treatment of Agoraphobia
Future Developments in Agoraphobia Treatments
There are several online programs and apps, based on manualized CBT principles,
to assist the recovery of people with agoraphobia. These may be useful for mild to
moderate levels of avoidance. It is likely, however, that people who have more
severe or complex presentations will still require face-to-face therapist contact and
intervention. Virtual reality platforms may replicate real-life situations more effectively than imagination, and these technologies may become affordable in office
practice. Finally, neurobiological assessments may assist in predicting individual
responsiveness to therapies such as CBT, but, as yet, they depend on
specialist investigations such as advanced brain imaging which are costly and may
carry risk.
Significant numbers of patients continue to suffer from agoraphobia following
treatment. Further investigation needs to be given to the use of second and third
wave therapies for people who do not respond to CBT alone. Training of therapists
to diagnose and treat is also a critical feature, as the more accurate the initial
assessment, the greater the likelihood that treatment can be tailored to suit individual patient needs.
REFERENCES
American Psychiatric Association. (1980). Diagnostic and statistical manual of mental disorders (3rd ed.). American Psychiatric Publishing.
(2013). Diagnostic and statistical manual of mental disorders (5th ed.) (DSM-5). American
Psychiatric Publishing.
Arnow, D. A., Taylor, C. B., Agras, W. S., & Telch, M. I. (1985). Enhancing agoraphobia
treatment outcome by changing couple communication patterns. Behavior Therapy, 16,
452–467.
Aronson, T. A., & Logue, C. M. (1987). On the longitudinal course of panic disorder:
Development history and predictors of phobic complications. Comprehensive
Psychiatry, 28, 344–355.
Bandelow, B. (1995). Assessing the efficacy of treatments for panic disorder and agoraphobia:
II. The Panic and Agoraphobia Scale. International Clinical Psychopharmacology, 10,
73–81.
(1997). Panic and Agoraphobia Scale (PAS). Hogrefe & Huber.
Bandura, A. (1977). Self efficacy: Toward a unifying theory of behavioral change.
Psychological Review, 84, 191–205.
Barlow, D. H. (2002). Anxiety and its disorders: The nature and treatment of anxiety and panic.
Guilford Press.
Barlow, D. H., O’Brien, G. T., & Last C. G. (1984). Couples treatment of agoraphobia.
Behavior Therapy, 15, 41–58.
Barlow, D. H., Gorman, J. M., Shear, M. K., & Woods, S. W. (2000). Cognitive behavioral
therapy, imipramine, or their combination for panic disorder: A randomized controlled
trial. Journal of the American Medical Association, 283, 2529–2536.
Basoglu, M., Marks, I. M., Kilic, C., Brewin, C. R., & Swinson, R. P. (1994). Alprazolam and
exposure for panic disorder with agoraphobia attribution of improvement to medication
predicts subsequent relapse. British Journal of Psychiatry, 164, 652–659.
89
90
Vijaya Manicavasagar and Derrick Silove
Botella, C., Villa, H., Garcia Palacios, A., Quero, S., Banos, R. M., & Alcaniz, M. (2004). The
use of VR in the treatment of panic disorders and agoraphobia. Studies in Health
Technology and Informatics, 99, 73–90.
Bowlby, J. (1973). Attachment and loss, vol. 2: Separation: Anxiety and anger. Penguin Books.
Cerny, J. A., Barlow, D. H., Craske, M. G., & Himadi, W. G. (1987). Couples treatment of
agoraphobia: A two-year follow-up. Behavior Therapy, 18, 401–415.
Chambless, D. L., Caputo, G. C., Bright, P., & Gallagher, R. (1984). Assessment of fear in
agoraphobics: The Body Sensations Questionnaire and the Agoraphobic Cognitions
Questionnaire. Journal of Consulting and Clinical Psychology, 52, 1090–1097.
Chambless, D. L., Caputo, G. C., Jasin, S. E., Gracely, E. J., & Williams, C. (1985). The
Mobility Inventory for Agoraphobia. Behaviour Research and Therapy, 23, 35–44.
Clark, D. M. (1986). A cognitive approach to panic. Behaviour Research and Therapy, 24,
461–470.
Constantino, M. J. (2012). Believing is seeing: An evolving research program on patients’
psychotherapy expectations. Psychotherapy Research, 22, 127–138.
Cox, B. J., Endler, N. S., & Swinson, R. P. (1995). An examination of levels of agoraphobic
severity in panic disorder. Behaviour Research and Therapy, 33, 57–62.
Craske, M. G., & Barlow, D. H. (1988). A review of the relationship between panic and
avoidance. Clinical Psychology Review, 8, 667–685.
Craske, M. G., Kircanski, K., Zelikowsky, M., Mystkowski, J., Chowdhury, N., & Baker, A.
(2008). Optimizing inhibitory learning during exposure therapy. Behaviour Research and
Therapy, 46(1), 5–27.
Craske, M. G., Rapee, R. M., & Barlow, D. H. (1988). The significance of panic-expectancy
for individual patterns of avoidance. Behavior Therapy, 19, 577–592.
Crowe, R. R., Noyes, R., Pauls, D. L., & Sylmen, D. (1983). A family study of panic disorder.
Archives of General Psychiatry, 40, 1065–1069.
El-Baalbaki, G., Bélanger, C., Perreault, M., Fredman, S. J., & Baucom, D. (2010). Marital
interactions in predicting treatment outcome in panic disorder with agoraphobia.
International Journal of Humanities and Social Science, 5, 671–681.
Fava, G. A., Rafanelli, C., Grandi, S., Conti, S., Ruini, C., Mangelli, L., & Belluardo, P.
(2001). Long-term outcome of panic disorder with agoraphobia treated by exposure.
Psychological Medicine, 31, 891–898.
First, M. B., Williams, J. B. W., Karg, R. S., & Spitzer, R. L. (2015). Structured Clinical
Interview for DSM-5 Disorders, Clinician Version (SCID-5-CV). American Psychiatric
Association.
Gloster, A. T., Sonntag, R., Hoyer, J., Meyer, A. H., Heinze, S., Strohle, A., Eifert. G., &
Wittchen, H.-U. (2015). Treating treatment-resistant patients with panic disorder and
agoraphobia using psychotherapy: A randomized controlled switching trial.
Psychotherapy and Psychosomatics, 84, 100–109.
Gloster, A. T., Wittchen, H.-U., Einsle, F., Helbig-Lang, S., Hamm, A. O., Richter, J.,
Gerlach, A. L., Kircher, T., Zwanzger, P., Lang, T., Fydrich, T., Fehm, L., Alpers, G.
W., Ströhle, A., Deckert, J., Höfler, M., & Arolt, V. (2011). Psychological treatment for
panic disorder with agoraphobia: A randomized controlled trial to examine the role of
therapist-guided exposure in situ in CBT. Journal of Consulting and Clinical Psychology,
79(3), 406–420.
Goisman, R. M., Warshaw, M. G., Peterson, L. G., Rogers, M. P., Cuneo, P., Hunt, M. F.,
Tomlinalbanese, J. M., Kazim, A., Gollan, J. K., Epsteinkaye, T., Reich, J. H., & Keller,
M. B. (1994). Panic, agoraphobia, and panic disorder with agoraphobia: Data from a
multicenter anxiety disorders study. Journal of Nervous & Mental Disease, 182(2), 72–79.
Goldstein, A. J., & Chambless, D. L. (1978). A reanalysis of agoraphobia. Journal of
Behavior Therapy and Experimental Psychiatry, 1, 305–313.
Cognitive Behavioral Treatment of Agoraphobia
Gould, R. A., Otto, M. W., & Pollack, M. H. (1995). A meta-analysis of treatment outcome
for panic disorder. Clinical Psychology Review, 15, 819–844.
Greenberg, P., Sisitsky, T., Kessler, R., & Finkelstein, S. (1999). The economic burden of
anxiety disorders in the 1990s. Journal of Clinical Psychiatry, 60(7), 427–435.
Hahlweg, K., Fiegenbaum, W., Frank, M., Schroeder, B., & von Witzleben, I. (2001). Shortand long-term effectiveness of an empirically supported treatment for agoraphobia.
Journal of Consulting and Clinical Psychology, 69 (3), 375–382.
Issakidis, C., & Andrews, G. (2004). Pretreatment attrition and dropout in an outpatient clinic
for anxiety disorders. Acta Psychiatrica Scandinavica, 109, 426–433.
Keefe, J. R., McCarthy, K. S., Dinger, U., Zilcha-Mano, S., & Barber, J. P. (2014). A metaanalytic review of psychodynamic therapies for anxiety disorders. Clinical Psychology
Review, 34, 309–323.
Kessler, R., Chiu, W., & Jin, R. (2006). The epidemiology of panic attacks, panic disorder,
and agoraphobia in the National Comorbidity Survey Replication. Archives of General
Psychiatry, 63(4), 415–424.
Kuch, K., & Swinson, R. P. (1992). Agoraphobia: What Westphal really said. Canadian
Journal of Psychiatry, 37(2), 133–136.
Lepine, J.-P. (2003). The epidemiology of anxiety disorders: Prevalence and societal costs.
Journal of Clinical Psychiatry, 63(Suppl. 14), 4–8.
Marcaurelle, R., Bélanger, C., Marchand, A., Katerelos, T. E., & Mainguy, N. (2005). Marital
predictors of symptom severity in panic disorder with agoraphobia. Journal of Anxiety
Disorders, 19, 211–232.
Markowitz, J. S., Weissman, M. M., Quellette, R., Lish, J. D., & Klerman, G. L. (1989).
Quality of life in panic disorder. Archives of General Psychiatry, 46, 984–992.
Mattick, R. P., Andrews, G., Hadzi-Pavlovic, D., & Christensen, H. (1990). Treatment of
panic and agoraphobia: An integrative review. Journal of Nervous and Mental Disease,
178, 567–576.
McNally, R. J., & Foa, E. B. (1987). Cognition and agoraphobia: Bias in the interpretation of
threat. Cognitive Therapy and Research, 11, 567–582.
McPherson, F. M., Brougham, L., & McLaren, S. (1980). Maintenance of improvement in
agoraphobic patients treated by behavioral methods: Four year follow-up. Behaviour
Research and Therapy, 18, 150–152.
Michelson, L., Mavissakalian. M., & Marchione, K. (1985). Cognitive and behavioral treatments of agoraphobia: Clinical, behavioral and psychophysiological outcomes. Journal of
Consulting and Clinical Psychology, 53, 913–925.
Munby, J., & Johnston D.W. (1980). Agoraphobia: Long-term follow-up of behavioural
treatment. British Journal of Psychiatry, 135, 418–427.
Perreault, M., Julien, D., White, N. D., Bélanger, C., Marchand, A., Katerelos, T., & Milton,
D. (2014). Treatment modality preferences and adherence to group treatment for panic
disorder with agoraphobia. Psychiatric Quarterly, 85, 121–132.
Rapee, R. M., & Murrell, E. (1988). Predictors of agoraphobic avoidance. Journal of Anxiety
Disorders, 2, 203–218.
Reiss, S., Peterson, R. A., Gursky, D. M., & McNally, R. J. (1986). Anxiety sensitivity, anxiety
frequency and the prediction of fearfulness. Behaviour Research and Therapy, 24, 1–8.
Robins, L. N., & Regier, D. A. (1991). Psychiatric disorders in America. Macmillan.
Sanchez-Meca, J., Rosa-Alcazar, A. I., Marin-Martinez, F., & Gomez-Conesa, A. (2010).
Psychological treatment of panic disorder with or without agoraphobia: A meta-analysis.
Clinical Psychology Review, 30, 37–50.
Scupi, B. S., Maser, J. D., & Uhde, T. W. (1992). The National Institute of Mental Health
Panic Questionnaire: An instrument for assessing clinical characteristics of panic disorder. Journal of Nervous and Mental Disease, 180, 566–572.
91
92
Vijaya Manicavasagar and Derrick Silove
Swift, J. K., & Greenberg, R. P. (2012). Premature discontinuation in adult psychotherapy:
A meta-analysis. Journal of Consulting and Clinical Psychology, 80, 547–559.
Telch, M. J., Brouillard, M., Telch, C. F., Agras, W. S., & Taylor, C. B. (1989). Role of
cognitive appraisal in panic-related avoidance. Behaviour Research and Therapy, 27,
373–383.
Teusch, L., & Boehme, H. (1999). Is the exposure principle really crucial in agoraphobia?
The influence of client-centered ‘nonprescriptive’ treatment on exposure. Psychotherapy
Research, 9, 115–123.
Turgeon, L., March, A., & Dupuis, G. (1998). Clinical features in panic disorder with
agoraphobia: A comparison of men and women. Journal of Anxiety Disorders, 12,
539–553.
White, K. S., Allen, L. B., Barlow, D. H., Gorman, J. M., Shear, M. K., & Woods, S. W.
(2010). Attrition in a multicenter clinical trial for panic disorder. Journal of Nervous and
Mental Disease, 198, 665–671.
World Health Organisation. (2018). International Statistical Classification of Diseases and
Related Health Problems (11th Revision). https://icd.who.int/browse11/1-m/en
Yonkers, K. A., Zlotnick, C., Allsworth, J., Warshaw, M., Shea, T., & Keller, M. B. (1998). Is
the course of panic disorder the same in women and men? American Journal of
Psychiatry, 155, 596–602.
Zarate, R., Craske, M., & Barlow, D. H. (1990). Situation exposure treatment versus panic
control treatment for agoraphobia: A case study. Journal of Behaviour Therapy and
Experimental Psychiatry, 21(3), 211–224.
6
Cognitive Behavioral Therapy for Social
Anxiety Disorder
Michaela B. Swee, M. Taylor Wilmer, and
Richard G. Heimberg
Introduction
Social anxiety disorder (SAD), also known as social phobia, is one of the most
commonly occurring anxiety disorders, with an estimated lifetime prevalence of
12% (Kessler et al., 2005). SAD is characterized by significant and persistent fear
of social situations, such as initiating a conversation, giving a presentation, or
speaking up at a meeting (American Psychiatric Association [APA], 2013).
Individuals with SAD experience excessive fear of being negatively evaluated by
others, which often results in avoidance of social situations and causes significant
distress and impairment in social, occupational, and personal domains. SAD
commonly occurs with other anxiety disorders as well as substance use disorders,
mood disorders, and personality disorders (Aderka et al., 2012; Grant et al., 2005;
Ruscio et al., 2008).
SAD often develops in early adolescence and runs a chronic course if untreated
(Bruce et al., 2005; Kessler et al., 2005). Fortunately, there are several evidencebased psychotherapeutic interventions for SAD, with cognitive behavioral therapy
(CBT) garnering the most evidence in its support (Gordon et al., 2014). This chapter
presents an overview of an individual CBT protocol for SAD, developed by Hope
et al. (2010a, 2010b) and evaluated in randomized controlled trials (RCTs) (e.g.,
Goldin et al., 2012; Ledley et al., 2009).
We first discuss the theoretical model on which this treatment is based and
review relevant efficacy and effectiveness research. We follow with discussion of
methods and measures for diagnosing SAD and assessing symptom severity before,
during, and after treatment. We then discuss how treatment is introduced to clients,
as well as the socialization process and strategies for forming an initial case conceptualization. Finally, we outline the therapy protocol and describe the core aspects of
the sessions.
93
94
Swee, Wilmer, and Heimberg
Previous Research
A Model of Social Anxiety Disorder
Multiple models of SAD exist (e.g., Clark, 2001; Heimberg et al., 2014; Hofmann,
2007). The Hope et al. (2010b) protocol was developed in tandem with the model of
SAD proposed by Rapee and Heimberg (Heimberg et al., 2014; Rapee &
Heimberg, 1997), which proposes that a primary driver of SAD is the discrepancy
between a person’s perception of the demands of the social world and their perceived ability to meet those demands. Individuals with SAD hold a highly negative
view of themselves, and they spontaneously generate negative self-images in social
situations based on how they imagine other people see them (Hackmann et al.,
1998; Wells & Papageorgiou, 1999). This third-person viewpoint is particularly
problematic for individuals with SAD, because they believe that others (the ‘perceived audience’; Rapee & Heimberg, 1997) hold the same excessively high standards for social performance that they hold for themselves. Individuals with SAD
believe that they are unable to meet these high expectations and thus overestimate
the probability and consequences of negative evaluation by others. Cognitive,
physiological, and behavioral symptoms of SAD emerge as consequences of
this process.
Individuals with SAD also engage in negative and distorted thoughts before,
during, and after social situations (Kaplan et al., 2017). These negative thoughts
often reflect the individual’s concern that they will be unable to meet the high
expectations held by others in social situations (e.g., “I won’t have anything interesting to say”; “I’ll make a fool of myself”) or that others will judge them negatively
(e.g., “They will think I’m an idiot”; “They don’t want to spend time with me.”).
Individuals with SAD tend to accept these thoughts as fact, focusing on them as they
approach social situations and engaging in perseverative thinking afterward. They
also exhibit negative interpretation biases, making negative interpretations about
neutral or ambiguous social events and interpreting negative social outcomes as
catastrophic (Amir et al., 1998; Hertel et al., 2008; Stopa & Clark, 2000). This biased
cognitive processing may increase the tendency to ignore or discount positive social
outcomes (Alden et al., 2008; Hirsch & Mathews, 2000; Weeks, 2010), reinforcing
negative thought patterns and maintaining social anxiety.
In response to these negative thoughts and resulting anxiety, individuals with
SAD engage in avoidance and escape behaviors. Sometimes social situations are
completely avoided; other times, individuals with SAD exhibit more subtle forms of
avoidance, known as safety behaviors (Clark, 2001; Piccirillo et al., 2016; Wells
et al., 1995). Safety behaviors are typically employed to minimize negative evaluation or reduce anxiety when complete evasion of the situation is undesirable or
impossible. For instance, socially anxious individuals may go to a party but speak
only to familiar people, or they may attend class but avoid participating. Although
avoidance and safety behaviors serve to reduce anxiety in the moment, they prevent
socially anxious individuals from gathering disconfirmatory evidence related to their
CBT for Social Anxiety Disorder
negative thoughts, reinforce beliefs about social incompetence, and increase anxiety
about future social situations (Clark, 2001; Heimberg et al., 2014; McManus et al.,
2008). Thus, these behaviors perpetuate the cycle of anxiety and play an important
role in maintaining SAD symptoms.
Core Components of CBT for SAD
CBT protocols commonly incorporate three core components: cognitive restructuring, exposure to feared social situations, and between-session homework. Cognitive
restructuring helps individuals identify and challenge maladaptive thoughts and
reframe them in ways that are more realistic and helpful. Exposures are utilized
to confront anxiety-provoking situations, challenge the accuracy of anxious cognitions, and foster self-efficacy. Exposures can take many forms, including imaginal
exposures, in-session role-play, and exposure to real-life social situations during or
between therapy sessions (i.e., in vivo exposures). Cognitive restructuring and
exposure activities are often used in tandem because cognitive restructuring aids
in more realistic thinking, which facilitates effective coping and adaptive behavior.
Additionally, experiencing successful behavioral outcomes provides powerful evidence to challenge maladaptive thoughts. Finally, regular homework assignments
allow clients to independently practice skills learned during therapy and apply them
to a diverse range of everyday situations. As clients approach the termination of
formal sessions, therapists encourage them to continue applying their newly
acquired skills in everyday life to lower the chance of relapse and to maintain
treatment gains.
Evidence Base of CBT for SAD
Heimberg and colleagues first developed and evaluated a cognitive behavioral
group therapy (CBGT) for the treatment of SAD (Heimberg & Becker, 2002).
The initial efficacy study compared CBGT with an educational-supportive group
psychotherapy control condition (Heimberg et al., 1990). CBGT yielded greater
improvements in SAD from pre- to post-test and at six-month follow-up than did
the control condition, and gains were maintained approximately five years later
(Heimberg et al., 1993). CBGT has also performed well relative to other treatments
for SAD. When compared with mindfulness-based stress reduction, CBGT produced lower post-treatment scores for SAD, greater response and remission rates
(Koszycki et al., 2007), and greater decreases in safety behaviors (Goldin et al.,
2016). Thus, evidence points to CBGT as an efficacious treatment for SAD, even
when compared with credible placebo and active treatment conditions.
Heimberg and colleagues adapted the protocol for use in individual treatment,
resulting in a manualized and workbook-driven individual CBT for SAD, now in its
second edition (Hope et al., 2010b). RCTs have demonstrated lowered client- and
clinician-rated severity of social anxiety after 20 weeks of treatment, with gains
maintained at three-month (Ledley et al., 2009) and one-year follow-ups (Goldin
95
96
Swee, Wilmer, and Heimberg
et al., 2012). Additionally, individuals who received CBT experienced improvement
in comorbid mood and anxiety disorders. Individual CBT, based largely on this
protocol, is also efficacious compared with acceptance and commitment therapy
(ACT), producing equivalent symptom reduction across 12 weeks of treatment and
outperforming a wait-list control (Craske et al., 2014).
Meta-analyses provide further support for the efficacy of CBT for SAD,
yielding strong within-person treatment effects at post-treatment (d = 0.77) and at
follow-up (d = 0.95; Gil et al., 2001). Average post-treatment effect sizes were also
strong when compared with wait-list control groups (e.g., d = 0.71, Acarturk et al.,
2009; g = 0.84, Barkowski et al., 2016; d = 1.19, Mayo-Wilson et al., 2014). The
Heimberg model of CBGT (d = 0.80) and individual CBT (d = 1.02) showed effect
sizes comparable to other CBT models (Mayo-Wilson et al., 2014). Moreover,
multiple meta-analyses suggest that CBGT is as efficacious as individual CBT for
SAD (Acarturk et al., 2009; Barkowski et al., 2016; Gil et al., 2001; Powers et al.,
2008).
When compared with pharmacotherapy, CBT has been shown to be as efficacious at reducing SAD symptoms. Some studies suggest that CBT yields better longterm outcomes and lower relapse rates than pharmacotherapy (e.g., Liebowitz et al.,
1999). An RCT comparing CBGT with the monoamine oxidase inhibitor phenelzine
found that participants who received phenelzine improved more rapidly and
reported lower social anxiety after 12 weeks, but they also showed greater relapse
during the follow-up period, suggesting that CBGT conferred greater protection
against relapse (Heimberg et al., 1998; Liebowitz et al., 1999). More recent research
demonstrated a synergistic effect, with combined CBGT and phenelzine producing
a steeper rate of improvement, higher remission rates, and higher response rates
than CBGT alone, phenelzine alone, or placebo (Blanco et al., 2010). However,
Nordahl et al. (2016) recently demonstrated superior efficacy of CBT to paroxetine
pharmacotherapy at post-treatment and one-year follow-up. CBT was also superior
to combination treatment at follow-up. Meta-analyses suggest that individual and
group CBT are just as efficacious as pharmacotherapy for SAD in terms of symptom
reduction and response rates (Barkowski et al., 2016; Mayo-Wilson et al., 2014).
CBT for SAD has also produced positive outcomes in effectiveness trials,
demonstrating that CBT for SAD is both feasible and acceptable to individuals in
routine clinical care. Across four treatment clinics in Germany, clients receiving
individual CBT for SAD experienced a significant reduction in social anxiety, as
well as general anxiety and depression, after only six weeks (Lincoln et al., 2003).
CBGT for SAD in a community clinic produced similar results, yielding significant
decreases in social anxiety and depression after only seven sessions (McEvoy, 2007).
Longitudinal research has further indicated that social anxiety symptom reduction is
maintained up to 12 months following CBT in a routine clinical setting (McCarthy
et al., 2013). Direct comparisons have demonstrated that CBT for SAD administered in an outpatient clinic produces symptom reduction equivalent to administration in a research laboratory, even when the clinic sample had more severe social
anxiety, more comorbid disorders, and lower educational attainment than the
CBT for Social Anxiety Disorder
research sample (Gaston et al., 2006; McEvoy, Nathan et al., 2012). Taken together,
CBT (individual or group) appears to be both efficacious and effective in reducing
symptoms of SAD, with gains maintained up to five years later.
Methods of Assessment
Diagnostic Interviews
Diagnostic interviews are particularly useful tools for identifying SAD, differentiating SAD from other anxiety disorders, and capturing a broad diagnostic picture,
including comorbidity. Our clinicians administer the Anxiety and Related Disorders
Interview Schedule for DSM-5 (ADIS-5; Brown & Barlow, 2014), which focuses on
assessment of anxiety and related disorders. Although psychometric data for the
ADIS-5 have not yet been published, the ADIS-5 was closely modeled on the ADIS
for DSM-IV (Brown et al., 1994), which demonstrated substantial inter-rater reliability for SAD (κ = 0.77; Brown et al., 2001).
The ADIS-5 takes approximately three hours to administer and can provide a
wealth of information about the experience of SAD, including symptom severity,
perceived causes and triggers, age of onset, and current interference in functioning.
Other interviews that can be used to assess the presence of SAD include the
Structured Clinical Interview for DSM-5 (SCID-5; First et al., 2015) and the Mini
International Neuropsychiatric Interview for DSM-5 (M.I.N.I. 7.0; Sheehan, 2015).
Clinician-Administered Measures
A clinician-administered measure can be a helpful supplement to a full diagnostic
interview or useful when completing a diagnostic interview is difficult or unnecessary. The Liebowitz Social Anxiety Scale (LSAS; Liebowitz, 1987) was the first
clinician-rated scale to assess social anxiety and remains widely used today. It is a
24-item measure that assesses feared and/or avoided social situations. The measure
addresses social interaction situations (11 items; e.g., talking to people in authority)
and performance situations (13 items; e.g., working while being observed). For each
item, the evaluator asks for ratings of fear and avoidance on Likert-type scales, with
fear ranging from 0 (none) to 3 (severe) and avoidance ranging from 0 (never, 0%)
to 3 (usually, 68–100%). The evaluator may adjust ratings based on clinical judgment. Fear and avoidance ratings are summed to create a total score. The LSAS has
demonstrated excellent internal consistency (Cronbach’s α = 0.96), good convergent
validity with other measures of social anxiety, and sensitivity to treatment-related
changes (Heimberg et al., 1999). The LSAS performs well in identifying individuals
who meet criteria for SAD, with a clinical cutoff score of 30 providing the best
balance of sensitivity and specificity (Mennin et al., 2002). The LSAS has also been
validated as a self-report measure (Fresco et al., 2001; Table 6.1). The Brief Social
Phobia Scale (Davidson et al., 1991, 1997) is another psychometrically sound
clinician-administered measure of social anxiety.
97
98
Downloaded from https://www.cambridge.org/core, on subject to the Cambridge Core terms of use, available at https://www.cambridge.org/core/terms.
https://doi.org/10.1017/9781108355605.007
Table 6.1. Commonly used self-report measures of social anxiety and related constructs
Measure name
Domain
Items
Item score
range
Total score
range
Clinical cutoff
Reference
Brief Fear of Negative Evaluation
Scale (BFNE)
Liebowitz Social Anxiety Scale Self-Report Versionb (LSAS-SR)
Fear of negative evaluation and
judgment from other people
Fear and avoidance of social
interaction and performance situations
12a
1–5
12–60
n/a
24
0–144
30
Social Interaction Anxiety Scale
(SIAS)
Fear of interaction in social situations
involving dyads or groups
20c
0–3 for
fear
0–3 for
avoidance
0–4
Leary
(1983)
Fresco et al.
(2001)
0–80
34
Social Phobia and Anxiety
Inventory (SPAI)
Cognitive, somatic, and behavioral
dimensions of social fear
45
1–7
60 (Social
Phobia Subscale
only)
Social Phobia Inventory (SPIN)
Fear, avoidance, and physiological
symptoms characteristic of social
anxiety
Fear at the prospect of being observed
or watched by other people
17
0–4
0–192 (Social
Phobia
Subscale)
0–78
(Agoraphobia
Subscale)
0–68
19
Connor
et al. (2000)
20
0–4
0–80
24
Mattick &
Clarke
(1998)
Social Phobia Scale (SPS)
Mattick &
Clarke
(1998)
Turner et al.
(1989)
Note: n/a, not applicable.
a
Recent research suggests that scoring of only the eight straightforwardly worded Brief Fear of Negative Evaluation (BFNE) items provides a more valid score (see
Rodebaugh et al., 2011).
b
The LSAS-SR is psychometrically equivalent to the clinician-administered LSAS (Fresco et al., 2001).
c
Recent research suggests that scoring of only the 17 straightforwardly worded SIAS items provides a more valid score; a clinical cutoff score of 28 would be more
appropriate with that scoring (see Rodebaugh et al., 2011).
CBT for Social Anxiety Disorder
Self-Report Measures
Because a full diagnostic interview or clinician-administered measure is not always
feasible, much attention has been devoted to the assessment of social anxiety via
self-report. Self-report measures are quick to administer and a useful supplement to
clinician-gathered information. Although a full discussion of all self-report measures
is beyond the scope of this chapter, we provide a summary table that describes the
most commonly used self-report measures of social anxiety and related constructs
(Table 6.1).
Measuring Progress in Treatment
Continued monitoring of social anxiety is a useful process. Tracking progress on a
week-to-week basis can alert the clinician to client improvement or relapse and help
clients recognize and acknowledge incremental changes. Here we highlight the
Social Anxiety Session Change Index (SASCI; Hayes et al., 2008), a four-item
self-report measure that asks about change in four areas: (1) anxiety in anticipation
of and/or during social situations, (2) avoidance of social situations, (3) concern
about doing something embarrassing or humiliating in front of others, and (4)
functional interference. Each item is rated relative to the start of treatment on a
Likert-type scale ranging from 1 (much less than the start of treatment) to 7 (much
more than the start of treatment), and the SASCI is scored by summing the four
items. The SASCI has been shown to have good validity and internal consistency
(α = 0.84–0.94) and correlates positively with session-by-session changes in the Brief
Fear of Negative Evaluation Scale (Leary, 1983; Table 6.1). The SASCI offers a
brief assessment of subjective improvement in social anxiety symptoms that can be
useful in both treatment and research settings (Hayes et al., 2008).
Case Formulation and Conceptualization
For the remainder of this chapter, we discuss case conceptualization, socializing the
client to treatment, introducing the treatment model and rationale, and delivering
treatment content using the Hope et al. (2010a, 2010b) protocol. This is demonstrated through the case of Kayla, a 30-year-old Black heterosexual female who
grew up in Southern California with her biological parents and elder sister. At the
time of treatment initiation, she was enrolled as a full-time student in business
school and worked part-time as a server at a local restaurant.
Initial Diagnostic Assessment
During the ADIS-5, Kayla reported that she struggled with social anxiety since early
childhood; depression followed in middle school when she became keenly aware of
feeling as if she did not “fit in.” She stated that her depression would worsen when
she perceived that a social situation had gone poorly or worried that others viewed
99
100
Swee, Wilmer, and Heimberg
her as a failure, triggering feelings of hopelessness. Results from the interview
suggested that Kayla met DSM-5 criteria for SAD and major depressive disorder,
recurrent episode, in partial remission. SAD was determined to be her
primary diagnosis.
Kayla recognized fear and avoidance of numerous social situations, including
participating in her business school classes, speaking with people in authority (e.g.,
professors, her restaurant manager), refusing requests of dissatisfied customers at
work, and interacting with fellow students in class. She worried that she would
embarrass herself by saying something stupid, that her professors or manager
thought she was incompetent, and that her peers thought poorly of her. Kayla
reported that these fears interfered with her ability to perform effectively as a server
in the restaurant, making her highly nervous about interacting with customers and
hesitant to ask her manager for help.
Historically, she reported that social anxiety caused significant distress and
interference in school. She felt unable to make friends or network with peers, and
her grades suffered because she rarely participated in class and turned papers in late
due to fear of negative evaluation by her professors. Kayla identified goals for
treatment as connecting with her classmates, improving her confidence at work,
and being able to network in preparation for applying to jobs in line with her
career interests.
Psychoeducation and Client Socialization Process
The first therapy session was primarily aimed at socializing the client to treatment.
This process ensured that Kayla had an accurate understanding of SAD, how her
own experiences were related to SAD, and how CBT for SAD could be helpful.
During the session, Kayla’s therapist initiated a discussion about the universality
and dimensionality of social anxiety by presenting three case vignettes (Hope et al.,
2010b), which vary in the severity of social anxiety and associated impairment. The
therapist explained that mild symptoms of social anxiety are normal and experienced by most people (e.g., public speaking situations, job interviews). However,
when symptoms are sufficiently distressing and interfere with day-to-day life, treatment is warranted. The therapist then briefly summarized empirical evidence supporting the positive outcomes and long-term effects of CBT for SAD to emphasize
the potential value of treatment for Kayla.
Introduction to the Treatment Model and Rationale
The therapist introduced an abbreviated treatment model to Kayla through a
discussion of the three principal components of SAD: physical symptoms, thoughts,
and behaviors.1 Kayla then practiced identifying these components in her own life.
1
We suggest that therapists familiarize themselves with the full Rapee-Heimberg (1997) model of SAD,
but we find that this simplified model is more intuitive and relatable for clients.
CBT for Social Anxiety Disorder
Engaging in this exercise helped Kayla learn ways in which physical, cognitive, and
behavioral symptoms interact with each other in a process we call the “downward
spiral.” For example, Kayla became cognizant of how anxious thoughts increased
her physical symptoms and behavioral avoidance. She noted that these relationships
functioned bidirectionally. The therapist then introduced and described the three
key components of CBT for SAD: cognitive restructuring, exposures, and homework. Kayla was offered the opportunity to ask questions and clarify her understanding throughout these discussions.
Therapy Session Outline
Sessions 1–4: Psychoeducation, Tracking Components of SAD,
and Developing the Fear and Avoidance Hierarchy
Session 1. A priority for the first four sessions was rapport building. This created a
foundation of trust between Kayla and her therapist, allowing Kayla to feel safe and
supported when confronting anxiety-provoking material. Much of this was achieved
through psychoeducation, which helped Kayla to develop a framework and vocabulary for SAD. Through this process, the therapist also had an opportunity to better
understand Kayla’s past and current experiences and they together developed
collaborative goals for therapy.
The remainder of session 1 was devoted to a motivational exercise. Kayla
identified pros and cons of working versus not working on her SAD “right now.”
This exercise was intended to increase investment in treatment, proactively identify
potential barriers to engagement, and encourage adherence, particularly when
therapy would become more exposure-focused. The session concluded with a discussion of specific ways to maximize treatment gains and effectiveness, including
completing between-session homework and approaching rather than avoiding
anxiety-provoking situations.
Session 2. Kayla explored the three components of social anxiety in a recent
interaction with a complaining customer. Kayla described her physiological experience of heart palpitations, lightheadedness, and sweaty palms when confronted by
the customer. She identified the thoughts “He thinks this is my fault” and “He
thinks I am incompetent at my job.” Regarding behavioral symptoms, Kayla
remembered avoiding eye contact with the customer and leaving the table as quickly
as possible.
Kayla and her therapist discussed how these different symptoms interacted to
create a downward spiral of anxiety. Attending to her physical symptoms affirmed
her thoughts about appearing nervous or foolish, which intensified her physical
symptoms, leading to further thoughts of incompetence and the inclination to
escape. Kayla’s therapist emphasized that although avoidance/escape reduces anxiety in the short term, it increases feelings of guilt, shame, and depression; prevents
learning; and reduces feelings of self-efficacy in social situations in the long term.
101
102
Swee, Wilmer, and Heimberg
Next, the therapist introduced the three main components of treatment, which
together serve as valuable tools for interrupting the downward spiral of social
anxiety. The therapist explained that Kayla would first learn cognitive restructuring,
the process of identifying maladaptive negative thoughts and modifying them to
become more neutral, or less anxiety-provoking. The second component of treatment, which would begin after learning and practicing cognitive restructuring skills,
is exposure. The therapist explained that exposure gives Kayla evidence with which
to challenge her negative thoughts and gain confidence in her ability to persevere
through anxiety-provoking social situations. Finally, Kayla was introduced to the
third major component of treatment, between-session homework. Homework
would be utilized throughout treatment to continue momentum between sessions
and provide the opportunity to practice using cognitive restructuring and exposure
skills in her everyday life.2
Session 3. This session was devoted to creating the “fear and avoidance hierarchy,” an important step prior to beginning exposures. For homework, Kayla
generated eight to ten feared social situations that varied in level of anxiety evocation. Hierarchy items ranged from specific social scenarios (e.g., talking to her
supervisor at work) to more general categories of social situations (e.g., networking
with peers at business school events). Kayla and her therapist examined factors that
made each situation more or less distressing. For example, one item on Kayla’s
hierarchy was “sharing personal information.” The therapist asked if there were
people with whom Kayla was more or less comfortable speaking about herself.
Kayla reported that sharing personal information with her professors was more
difficult than sharing with fellow students, which was more difficult than sharing
personal information with coworkers at the restaurant. All of these situations were
included on the hierarchy. Next, Kayla rated each situation on two dimensions:
anxiety and avoidance (both on a scale from 0 to 100, where 0 is no anxiety or
avoidance, and 100 is the worst anxiety or avoidance imaginable). To evaluate
anxiety, the therapist described the Subjective Units of Distress Scale (SUDS;
Wolpe & Lazarus, 1967). Kayla used her SUDS levels to rank the situations on
her hierarchy from most to least difficult. Her therapist explained that they would
monitor SUDS ratings throughout exposures to track symptom change over time.
Session 4. This session focused on etiology and included a discussion of factors
involved in the development of Kayla’s SAD. The therapist described ways in which
genetics, family environment, and formative life experiences can influence SAD.
The therapist used Kayla’s homework, an exploration of where her SAD came
from, to guide the discussion. Kayla shared that there was a family history of social
anxiety on her father’s side. Specifically, she observed that her paternal uncle and
2
It should be noted that the first portion of every session is devoted to a check-in on homework
completion, as well as a review of highlights from the worksheets that were completed for homework.
This serves as a way to regularly monitor clients’ compliance and investment in therapy, as well as an
opportunity to ensure that material from the previous week’s session was appropriately and effectively
integrated and implemented before moving on to new material.
CBT for Social Anxiety Disorder
cousin always seemed shy and withdrawn when she spent time with them. When she
was a child, her father impressed upon her the importance of “staying under the
radar” and encouraged Kayla to stay home if she was feeling nervous about
attending school functions or birthday parties. Additionally, Kayla’s mother was
strict and demanding, keeping Kayla and her sister from spending time with other
children if their grades were not high enough or they had not finished their
household chores. Thus, Kayla identified genetics and family environment as having
the biggest impact on her development of SAD.
Sessions 5 and 6: Automatic Thoughts and Cognitive Restructuring
Session 5. The therapist opened this important phase of treatment with two
vignettes in which an objectively neutral social situation leads to two entirely
different outcomes for two individuals based on how they interpret the situation.
The pair of vignettes illustrated that people with SAD become anxious because of
their beliefs about a social situation, others involved, and themselves, not because of
the situation itself.
The therapist explained that negative thoughts about oneself, others, and the
world can be labeled as automatic thoughts (ATs). ATs are frequently erroneous
and/or unhelpful and have significant consequences for behaviors and emotions.
Hence, learning to evaluate and challenge them, rather than automatically accepting
them as fact, is fundamental to SAD treatment. Kayla and her therapist reviewed
her ATs (recorded in previous homework assignments) and noted several important
commonalities: they were emotionally based, largely negative, sometimes irrational,
and often future-oriented.
Kayla’s therapist next introduced the concept of thinking errors (Beck, 2011),
which are common dysfunctional thinking patterns that underlie ATs.3
Strengthening Kayla’s ability to identify thinking errors in her own ATs was the
first step in helping her question their validity and challenge them. Kayla practiced
identifying thinking errors in the ATs of a case vignette, then some of her own ATs.
Using the thought, “People will not like me when they meet me,” Kayla identified
thinking errors such as mind reading and fortune telling. With the thought, “My
writing is awful,” she identified all-or-nothing thinking, labeling, catastrophizing, and
emotional reasoning (Beck, 2011). Kayla’s therapist pointed out that an individual
AT may involve multiple thinking errors (as many of Kayla’s ATs did).
Next, the therapist guided Kayla toward an awareness of emotions triggered by
her ATs. For example, when Kayla experienced the thought that her writing was
awful, she noticed feeling angry, hopeless, and ashamed. Kayla’s therapist emphasized the interconnections between thoughts and feelings – thoughts affect emotions
and vice versa.
Session 6. Before Kayla began exposures, she learned the final steps of cognitive restructuring. The therapist reminded Kayla that the aim was to act like a
scientist by studying her thoughts and examining their validity and helpfulness.
Kayla learned to challenge her ATs through the use of an anxious-self/coping-self
103
104
Swee, Wilmer, and Heimberg
(AS/CS) dialogue. With this technique, Kayla would first identify an AS (anxious
self ) thought, then pose a disputing question (coping self ), next answer the question
expressing honest concern or disbelief in the response (anxious self ), then again
pose a question to address that response (coping self ), and so on. Finally, a CS
(coping self ) response is reached that Kayla could accept as tenable. Kayla’s AS/CS
dialogue concerning the AT “People will not like me when they meet me” follows:
Anxious self:
Coping self:
Anxious self:
Coping self:
Anxious self:
Coping self:
Anxious self:
Coping self:
Anxious self:
Coping self:
Anxious self:
People will not like me when they meet me.
How do you know that people will not like you when they
meet you?
Because there have been times when I have gotten the strong
feeling that someone does not like me.
When were these times?
Once in fifth grade when I met that girl, Brianna, and once in
high school with my teacher, Mr. Mattingly.
Any other times?
No, those are the two main ones I can think of.
How many people have you met in your life?
Over the course of my whole life? Oh, I don’t know if I can
even count. Hundreds.
Do you think Brianna and Mr. Mattingly’s possible dislike for
you is representative of most people’s first impressions of you?
No, I think most people feel positively, or at least neutral,
when they meet me.
By thoroughly examining these questions and answers, Kayla and her therapist
noticed an eventual shift in her original anxious self perspective. Kayla realized that
most people do not dislike her when they meet her. Next, the therapist asked Kayla
to take a step back from the dialogue and summarize the take-home message. Kayla
reported, “There have been a couple of people in my life who have not liked me, but
I have met many people in my life and most people have had no negative feelings
toward me at all; in fact, their reactions have been positive.” The therapist coached
Kayla to generate a rational response from this summary point. Effective rational
responses are typically short, concise, positive, or neutral statements that can be
used to counter negative ATs in the heat of an anxiety-provoking situation. When
the therapist asked Kayla for a rational response based on the conclusion of the AS/
CS dialogue, Kayla replied, “Most people I meet like me.” Kayla was encouraged to
use this and other helpful rational responses during exposures and future anxietyprovoking situations.
Sessions 7–11: First and Continued Exposures
These sessions focus on conducting exposures to feared social situations. Exposures
are perhaps the most important and impactful component of CBT for social anxiety,
CBT for Social Anxiety Disorder
particularly when paired with cognitive restructuring skills introduced in sessions
5 and 6. Kayla and her therapist discussed exposures several times prior to session 7,
including when the treatment model was introduced and during the development of
the fear and avoidance hierarchy.
Session 7. The therapist reviewed key aspects of exposures, explaining that they
would start with easier situations and steadily advance toward more anxietyprovoking situations. The rationale behind exposures was also reviewed, namely,
that exposures would allow Kayla to experience habituation to feared social situations. She would also be able to practice specific social skills, learn to cope with
anxiety while remaining in social situations, and gather additional evidence to
challenge ATs, ultimately building self-efficacy.
Kayla and her therapist collaboratively selected a situation from the lower
portion of Kayla’s hierarchy for the first in-session exposure (one that triggered a
SUDS level of 40–50 in real life). The chosen situation was relevant to Kayla’s
treatment goals and could feasibly be conducted within the therapy session. Kayla
selected “small talk at a business school networking event,” which she and her
therapist role-played for the first exposure. The therapist guided Kayla through
preexposure cognitive restructuring, in which Kayla identified her ATs and selected
a specific thought for restructuring, in this case: “I won’t have anything interesting to
say.” Kayla rated her belief in the AT at 80%. The therapist helped Kayla pinpoint
thinking errors in the AT (e.g., all or nothing thinking, fortune telling, and catastrophizing), challenge the AT using an AS/CS dialogue, and develop a rational
response: “All I have to do is talk about my favorite business school class.” Kayla
rated her belief in the rational response at 40%. Kayla and her therapist then
worked out the details of the exposure and, importantly, identified achievable
behavioral goals. The therapist explained that such goals are attainable, observable,
measurable, and focused on behaviors (i.e., how Kayla could act against SADrelated ATs) rather than oriented toward feelings (e.g., wishing to speak without
feeling any anxiety). Kayla identified a behavioral goal of making one statement
about her favorite class and asking at least two questions. The therapist reminded
Kayla that she would be prompted to share her SUDS level and repeat her rational
response throughout the exposure.
Kayla and her therapist then completed the exposure. The therapist prompted
Kayla to rate her SUDS level at one-minute intervals throughout the exposure and
recorded each rating. Kayla was also prompted to state her rational response aloud
whenever she gave a SUDS rating. After the exposure was completed, Kayla noted
that she was able to share more than one piece of information and she asked four
questions during the conversation. She stated that the conversation was easy
because she already felt comfortable with the therapist, and the therapist pointed
out the disqualifying the positive thinking error in that statement. The therapist and
Kayla discussed her pattern of SUDS ratings, which peaked at 65 and eventually
decreased to 20 by the end of the five-minute conversation. Kayla re-rated her belief
in the AT at 50% and the rational response at 60%. Kayla also noticed one
additional AT during the conversation, “This silent pause is so awkward.” The
105
106
Swee, Wilmer, and Heimberg
therapist reflected that she did not notice any long pauses during the conversation,
and she and Kayla used this information to challenge this AT. The therapist helped
Kayla celebrate the completion of her first exposure and praised her for her effort.
Finally, they collaborated on planning an out-of-session homework exposure. Kayla
decided that she would say “Hello” and ask one question (e.g., “How was your
weekend?”) to a classmate.
Sessions 8–11. The following four sessions continued with in-session exposures,
following a similar format and proceeding up Kayla’s hierarchy as appropriate.
Sessions 12–15: Advanced Cognitive Restructuring and Core Beliefs
Session 12. After conducting five in-session exposures, the skills of which were
reinforced through in vivo, out-of-session exposures, sessions 12–15 turn to
advanced cognitive restructuring techniques, with a particular focus on core beliefs.
Core beliefs are the central and enduring beliefs one holds about oneself, others, the
world, or the future (Beck, 2011). Individuals with SAD often hold negative core
beliefs with typical themes of being unlovable, defective, helpless, or worthless.
Because core beliefs are so fundamental and deeply held, they are often unarticulated prior to engaging in treatment. Thus, session 12 was aimed at helping Kayla
explore and verbalize her negative core belief(s).
The therapist guided Kayla to delve deeper into her ATs to examine her
underlying beliefs about herself, using a downward arrow technique called “peeling
the onion.” The therapist explained that the ATs Kayla had challenged in prior
sessions were like the outer layers of an onion, and the goal for the session would be
to “peel back” these layers and identify negative underlying beliefs that might be at
the onion’s “core.” Kayla gathered her worksheets from previous exercises and
homework assignments and reviewed her previously recorded ATs. She and the
therapist examined the list for emergent themes, and she selected the one that
seemed most important to how she thought about herself. She then addressed the
theme with the following questions: (1) Why is that important?, (2) What does it
mean if this is true?, and (3) What would be so bad about that? Similar to the AS/CS
dialogue, she answered the question, questioned the answer, answered the question,
and so on, until she reached a statement that she had believed for a long time, one
that seemed very personal and true, and that evoked strong emotion. A transcript of
this dialogue follows:
Therapist:
Kayla:
Therapist:
Thinking back to the automatic thoughts you challenged before
and after your exposures, did you notice any that came up over
and over again?
Well, I usually had thoughts like, “I won’t have anything
interesting to say” or “Everyone thinks I’m an idiot.”
Okay, let’s focus on those “outer layer” automatic thoughts and
see if we can peel back a few layers of the onion. First, do you
notice any common themes in those thoughts?
CBT for Social Anxiety Disorder
Kayla:
Therapist:
Kayla:
Therapist:
Kayla:
Therapist:
Kayla:
Therapist:
Kayla:
Therapist:
Kayla:
Therapist:
Kayla:
Well, I guess there seems to be a theme about other people not
liking me because I’m not interesting or smart enough.
And what would be so bad about that, if other people thought
you were not interesting or smart enough?
It would mean that I’m less than them in some way. It’s been like
this my whole life. I’ve been working as hard as I can to keep up
with everyone else, but I feel like I’ll never measure up.
So, one related thought you have is, “I’ll never measure up.” It’s
important to you to succeed in most, if not all, areas of your life.
What does it mean to “measure up” to other people?
I think of it as doing things really well or getting what you want,
like the grades you want, the friends you want, the promotion
you want.
So, when you set a goal for yourself, you feel like you should meet
or exceed that goal every time?
Well it sounds a little extreme when you put it like that, but yeah.
I don’t want to fall short, especially if I know I can do better.
How does it feel when you think you’ve fallen short of
your goals?
It feels terrible. I have this sinking feeling in my stomach, and
I feel really sad and ashamed. I get that way sometimes even if
I do well. I don’t want to tell anyone about successes I have
because I worry that they’re going to expect me to do even better
the next time.
So, you have the thought that even when you do something well
or reach your goals, you’ll still fall short or disappoint people?
Yeah, it’s like nothing I do will ever be good enough.
I think you may have reached the core of the onion. Does “I’m
not good enough” sound like it could be a core belief for you?
Yeah, I guess so. It’s like I’ll never be smart enough, fun enough,
or attractive enough, especially compared to my sister and my law
school classmates. I think “I’m not good enough,” captures all
of those.
Kayla identified the core belief, “I am not good enough.” The therapist then
explained that Kayla could approach the core belief like any other AT, by challenging and restructuring the belief using the same skills practiced since session 5. The
therapist explained how in vivo exposures could help Kayla gather additional realworld evidence against this core belief. During the sessions that followed, Kayla
used cognitive restructuring to identify and elaborate on past accomplishments. She
reflected on the strength of her friendships from college and engaged in “perspective-taking” about her mother’s criticism. She also completed in vivo exposures of
creating a resumé that highlighted her achievements, speaking to two recruiters at a
networking event, and inviting one of her classmates to get coffee with her. Over
107
108
Swee, Wilmer, and Heimberg
time, she chipped away at the belief that she was not good enough and gained strong
evidence in opposition to this belief.
Session 13 and Termination. A typical course of CBT for SAD runs 16–20
sessions, although some clients require more exposures or attention to core beliefs
to achieve their treatment goals. The final sessions are focused on evaluating
progress, establishing post-treatment goals, and discussing relapse prevention.
Kayla terminated therapy in 19 sessions. She had made good progress toward her
original goals and indicated that she felt comfortable applying her new skills independently. At the final session, Kayla and her therapist reviewed her SASCI scores,
which had dropped by 50% since the beginning of treatment. Kayla also re-rated
her fear and avoidance hierarchy and noted that her avoidance scores were considerably lower and her fear scores were moderately lower, especially for situations
directly targeted in therapy. Kayla wondered aloud whether she had worked hard
enough in her exposure sessions. The therapist again pointed out disqualifying the
positive and helped Kayla remember how she overcame the challenges of her
early exposures.
Kayla also identified ways in which her day-to-day life had changed since
beginning treatment. In addition to feeling less depressed, Kayla acknowledged
that she now felt comfortable speaking in class and talking to her professors and
was more confident in her ability to handle difficult customers at work. She further
noted that she recently made a friend in one of her classes and attended two
business school networking events. Kayla pointed out that the situations at the top
of her hierarchy, including dating and job interviews, still evoked high levels of
anxiety, but she felt more able to confront them without avoidance or safety
behaviors. She and her therapist used these situations to set behavioral goals for
Kayla’s first month following treatment. Kayla planned to create an online dating
profile and apply to at least three jobs by the end of the month. Her therapist
offered the option of booster sessions if Kayla were to get a job interview and
wanted to refresh her CBT skills beforehand. The therapist congratulated Kayla on
her hard work, her progress in treatment, and her openness to learning skills to
manage her social anxiety, and they concluded their treatment together.
Key Summary Points
Social anxiety disorder (SAD) is characterized by excessive fear of negative
evaluation and avoidance of social situations, which results in significant distress
and impairment in social, occupational, and personal domains.
Heimberg and colleagues developed a cognitive behavioral therapy (CBT)
protocol for SAD that has been shown to be efficacious when compared with
credible placebo and active treatment conditions, including pharmacotherapy,
and effective when administered in routine clinical settings.
Careful assessment of social anxiety and related symptomatology is essential to
treatment planning, case conceptualization, and symptom monitoring. A variety
CBT for Social Anxiety Disorder
of clinician-administered (e.g., ADIS-5, LSAS) and self-report measures (e.g.,
SIAS, SPS) are available for these purposes (Table 6.1).
The Heimberg CBT protocol is designed to be administered in 16–20 sessions.
Primary treatment components include cognitive restructuring, exposure to
feared social situations, and between-session homework.
Sessions 1–4 focus on psychoeducation for SAD, socializing clients to treatment,
and fostering treatment engagement. Clients are introduced to the treatment
model and rationale; practice identifying the physical, cognitive, and behavioral
symptoms of SAD; and create a hierarchy of feared social situations.
Sessions 5 and 6 target the cognitive restructuring component of treatment.
Clients learn to identify automatic thoughts, label thinking errors in the
thoughts, and challenge the thoughts using disputing questions and an anxious-self/coping-self dialogue.
Sessions 7–11 are devoted to conducting exposures to feared social situations,
based on the clients’ “fear and avoidance hierarchy.” Successful in-session
exposures are reinforced through cognitive restructuring beforehand, debriefing afterward, and in vivo exposures between sessions.
Sessions 12–16 focus on advanced cognitive restructuring skills and continued
exposures. Clients identify their core beliefs through a downward arrow exercise, and they work to challenge those beliefs through in-session restructuring
and out-of-session exposure homework.
Treatment termination may be considered when clients make progress toward
their original goals and feel comfortable applying CBT skills independently in
their own lives. The final session is devoted to evaluating progress, establishing
post-treatment goals, and discussing strategies for relapse prevention.
RECOMMENDED FURTHER READING
Clark, D. M., & Wells, A. (1995). A cognitive model of social phobia. In R. Heimberg, M.
Liebowitz, D. A. Hope, & F. R. Schneier (Eds.), Social phobia: Diagnosis, assessment,
and treatment (pp. 69–93). Guilford Press.
Gordon, D., Wong, J., & Heimberg, R. G. (2014). Cognitive-behavioral therapy for social
anxiety disorder: The state of the science. In J. W. Weeks (Ed.), The Wiley-Blackwell
handbook of social anxiety disorder (pp. 477–497). Wiley-Blackwell.
Heimberg, R. G., Brozovich, F. A., & Rapee, R. M. (2014). A cognitive-behavioral model of
social anxiety disorder. In S. G. Hofmann & P. M. DiBartolo (Eds.), Social anxiety:
Clinical, developmental, and social perspectives (3rd ed., pp. 705–728). Academic Press.
Heimberg, R. G., & Magee, L. (2014). Social anxiety disorder. In D. H. Barlow (Ed.), Clinical
handbook of psychological disorders: A step-by-step treatment manual (5th ed.,
pp. 114–154). Guilford Publications.
Hofmann, S. G. (2007). Cognitive factors that maintain social anxiety disorder:
A comprehensive model and its treatment implications. Cognitive Behaviour Therapy,
36, 193–209.
Hope, D. A., Heimberg, R. G., & Turk, C. L. (2010a). Managing social anxiety: A cognitivebehavioral therapy approach (Therapist Guide, 2nd ed.). Oxford University Press.
109
110
Swee, Wilmer, and Heimberg
(2010b). Managing social anxiety: A cognitive-behavioral therapy approach (Client
Workbook, 2nd ed.). New York: Oxford University Press.
Kaplan, S. C., Swee, M. B., & Heimberg, R. G. (2018). Psychological treatments for social
anxiety disorder. In O. Braddic (Ed.), Oxford research encyclopedia of psychology.
Oxford University Press.
Mayo-Wilson, E., Dias, S., Mavranezouli, I., Kew, K., Clark, D. M., Ades, A. E., & Pilling, S.
(2014). Psychological and pharmacological interventions for social anxiety disorder in
adults: A systematic review and network meta-analysis. The Lancet Psychiatry, 1,
368–376.
Powers, M. B., Sigmarsson, S. R., & Emmelkamp, P. M. G. (2008). A meta-analytic review of
psychological treatments for social anxiety disorder. International Journal of Cognitive
Therapy, 1, 94–113.
REFERENCES
Acarturk, C., Smit, F., de Graaf, R., van Straten, A., ten Have, M., & Cuijpers, P. (2009).
Economic costs of social phobia: A population-based study. Journal of Affective
Disorders, 115, 421–429. doi:10.1016/j.jad.2008.10.008
Aderka, I. M., Hofmann, S. G., Nickerson, A., Hermesh, H., Gilboa-Schechtman, E., &
Marom, S. (2012). Functional impairment in social anxiety disorder. Journal of Anxiety
Disorders, 26, 393–400. doi:10.1016/j.janxdis.2012.01.003
Alden, L. E., Taylor, C. T., Mellings, T. M. J. B., & Laposa, J. M. (2008). Social anxiety and
the interpretation of positive social events. Journal of Anxiety Disorders, 22, 577–590.
doi:10.1016/j.janxdis.2007.05.007
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Amir, N., Foa, E. B., & Coles, M. E. (1998). Automatic activation and strategic avoidance of
threat-relevant information in social phobia. Journal of Abnormal Psychology, 107,
285–290. doi:10.1037/0021-843x.107.2.285
Barkowski, S., Schwartze, D., Strauss, B., Burlingame, G. M., Barth, J., & Rosendahl, J.
(2016). Efficacy of group psychotherapy for social anxiety disorder: A meta-analysis of
randomized-controlled trials. Journal of Anxiety Disorders, 39, 44–64. doi:10.1016/j.
janxdis.2016.02.005
Beck, J. S. (2011). Cognitive behavior therapy: Basics and beyond (2nd ed.). Guilford Press.
Blanco, C., Heimberg, R. G., Schneier, F. R., Fresco, D. M., Chen, H., Turk, C. L., . . . &
Liebowitz, M. R. (2010). A placebo-controlled trial of phenelzine, cognitive behavioral
group therapy, and their combination for social anxiety disorder. Archives of General
Psychiatry, 67, 286–295. doi:10.1001/archgenpsychiatry.2010.11
Brown, T. A., & Barlow, D. H. (2014). Anxiety and Related Disorders Interview Schedule for
DSM-5 (ADIS-5L): Client interview schedule. Oxford University Press.
Brown, T. A., Di Nardo, P. A., & Barlow, D. H. (1994). Anxiety Disorders Interview Schedule
for DSM-IV (ADIS-IV): Client interview schedule. Oxford University Press.
Brown, T. A., Di Nardo, P. A., Lehman, C. L., & Campbell, L. A. (2001). Reliability of DSMIV anxiety and mood disorders: Implications for the classification of emotional disorders.
Journal of Abnormal Psychology, 110, 49–58. doi:10.1037/0021-843X.110.1.49
Bruce, S. E., Yonkers, K. A., Otto, M. W., Eisen, J. L., Weisberg, R. B., Pagano, M., . . . &
Keller, M. B. (2005). Influence of psychiatric comorbidity on recovery and recurrence
in generalized anxiety disorder, social phobia, and panic disorder: A 12-year
prospective study. American Journal of Psychiatry, 162, 1179–1187. doi:10.1176/appi.
ajp.162.6.1179
CBT for Social Anxiety Disorder
Clark, D. M. (2001). A cognitive perspective on social phobia. In W. R. Crozier & L. E. Alden
(Eds.), International handbook of social anxiety: Concepts, research and interventions
relating to the self and shyness (pp. 405–430). John Wiley & Sons.
Connor, K. M., Davidson, J. R. T., Churchill, L. E., Sherwood, A., Foa, E., & Weisler, R. H.
(2000). Psychometric properties of the Social Phobia Inventory. British Journal of
Psychiatry, 176, 379–386. doi:10.1192/bjp.176.4.379
Craske, M. G., Niles, A. N., Burklund, L. J., Wolitzky-Taylor, K. B., Vilardaga, J. P., Arch, J.
J., . . . & Lieberman, M. D. (2014). Randomized controlled trial of cognitive behavioral
therapy and acceptance and commitment therapy for social phobia: Outcomes and
moderators. Journal of Consulting and Clinical Psychology, 82, 1034–1048. doi:10.1037/
a0037212
Davidson, J. R. T., Miner, C. M., De Veaugh-Geiss, J., Tupler, L. A., Colket, J. T., & Potts,
N. L. S. (1997). The Brief Social Phobia Scale: A psychometric evaluation. Psychological
Medicine, 27, 161–166. doi:10.1017/s0033291796004217
Davidson, J. R. T., Potts, N. L. S., Richichi, E. A., Ford, S. M., Krishnan, K. R. R., Smith,
R. D., & Wilson, W. H. (1991). The Brief Social Phobia Scale. Journal of Clinical
Psychiatry, 52 Suppl., 48–51. doi:10.1037/t07672-000
First, M. B., Williams, J. B. W., Karg, R. S., & Spitzer, R. L. (2015). Structured Clinical
Interview for DSM-5 Disorders, clinician version (SCID-5-CV). American Psychiatric
Publishing.
Fresco, D. M., Coles, M. E., Heimberg, R. G., Liebowitz, M. R., Hami, S., Stein, M. B., &
Goetz, D. (2001). The Liebowitz Social Anxiety Scale: A comparison of the psychometric properties of self-report and clinician-administered formats. Psychological Medicine,
31, 1025–1035. doi:10.1017/S0033291701004056
Gaston, J. E., Abbott, M. J., Rapee, R. M., & Neary, S. A. (2006). Do empirically supported
treatments generalize to private practice? A benchmark study of a cognitive-behavioural
group treatment programme for social phobia. British Journal of Clinical Psychology, 45,
33–48. doi:10.1348/014466505x35146
Gil, P. M., Carrillo, F. M., & Meca, J. S. (2001). Effectiveness of cognitive-behavioural
treatment in social phobia: A meta-analytic review. Psychology in Spain, 5, 17–25.
Goldin, P. R., Morrison, A., Jazaieri, H., Brozovich, F., Heimberg, R., & Gross, J. J. (2016).
Group CBT versus MBSR for social anxiety disorder: A randomized controlled trial.
Journal of Consulting and Clinical Psychology, 84, 427–437. doi:10.1037/ccp0000092
Goldin, P. R., Ziv, M., Jazaieri, H., Werner, K., Kraemer, H., Heimberg, R. G., & Gross, J. J.
(2012). Cognitive reappraisal self-efficacy mediates the effects of individual cognitivebehavioral therapy for social anxiety disorder in a randomized controlled trial. Journal of
Consulting and Clinical Psychology, 80, 1034–1040. doi:10.1037/a0028555
Gordon, D., Wong, J., & Heimberg, R. G. (2014). Cognitive-behavioral therapy for social
anxiety disorder: The state of the science. In J. W. Weeks (Ed.), The Wiley-Blackwell
handbook of social anxiety disorder (pp. 477–497). Wiley-Blackwell.
Grant, B. F., Hasin, D. S., Blanco, C., Stinson, F. S., Chou, S. P., Goldstein, R. B., . . . &
Huang, B. (2005). The epidemiology of social anxiety disorder in the United States:
Results from the National Epidemiologic Survey on Alcohol and Related
Conditions. The Journal of Clinical Psychiatry, 66, 1351–1361. doi:10.4088/JCP.v66n1102
Hackmann, A., Surawy, C., & Clark, D. M. (1998). Seeing yourself through others’ eyes:
A study of spontaneously occurring images in social phobia. Behavioural and Cognitive
Psychotherapy, 26, 3–12.
Hayes, S. A., Miller, N. A., Hope, D. A., Heimberg, R. G., & Juster, H. R. (2008). Assessing
client progress session by session in the treatment of social anxiety disorder: The Social
Anxiety Session Change Index. Cognitive and Behavioral Practice, 15, 203–211.
doi:10.1016/j.cbpra.2007.02.010
111
112
Swee, Wilmer, and Heimberg
Heimberg, R. G., & Becker, R. E. (2002). Cognitive-behavioral group therapy for social
phobia: Basic mechanisms and clinical strategies. Guilford Press.
Heimberg, R. G., Brozovich, F. A., & Rapee, R. M. (2014). A cognitive-behavioral model of
social anxiety disorder. In S. G. Hofmann, & P. M. DiBartolo (Eds.), Social anxiety:
Clinical, developmental, and social perspectives (3rd ed., pp. 705–728). Academic Press.
doi:10.1016/B978-0-12-394427-6.00024-8
Heimberg, R. G., Dodge, C. S., Hope, D. A., Kennedy, C. R., Zollo, L. J., & Becker, R. E.
(1990). Cognitive behavioral group treatment for social phobia: Comparison with a
credible placebo control. Cognitive Therapy and Research, 14, 1–23. doi:10.1007/
bf01173521
Heimberg, R. G., Horner, K. J., Juster, H. R., Safren, S. A., Brown, E. J., Schneier, F. R., &
Liebowitz, M. R. (1999). Psychometric properties of the Liebowitz Social Anxiety Scale.
Psychological Medicine, 29, 199–212. doi:10.1017/S0033291798007879
Heimberg, R. G., Liebowitz, M. R., Hope, D. A., Schneier, F. R., Holt, C. S., Welkowitz,
L. A., . . . & Klein, D. F. (1998). Cognitive behavioral group therapy versus phenelzine
therapy for social phobia. Archives of General Psychiatry, 55, 1133–1141. doi:10.1001/
archpsyc.55.12.1133
Heimberg, R. G., Salzman, D., Holt, C. S., & Blendell, K. (1993). Cognitive behavioral group
treatment of social phobia: Effectiveness at 5-year follow-up. Cognitive Therapy and
Research, 17, 325–339. doi:10.1007/BF01177658
Hertel, P. T., Brozovich, F., Joormann, J., & Gotlib, I. H. (2008). Biases in interpretation and
memory in generalized social phobia. Journal of Abnormal Psychology, 117, 278–288.
doi:10.1037/0021-843x.117.2.278
Hirsch, C. R., & Mathews, A. (2000). Impaired positive inferential bias in social phobia.
Journal of Abnormal Psychology, 109, 705–712. doi:10.1037/0021-843x.109.4.705
Hofmann, S. G. (2007). Cognitive factors that maintain social anxiety disorder:
A comprehensive model and its treatment implications. Cognitive Behaviour Therapy,
36, 193–209. doi:10.1080/16506070701421313
Hope, D. A., Heimberg, R. G., & Turk, C. L. (2010a). Managing social anxiety: A cognitivebehavioral therapy approach (Therapist Guide, 2nd ed.). Oxford University Press.
doi:10.1093/med:psych/9780195336689.001.0001
(2010b). Managing social anxiety: A cognitive-behavioral therapy approach
(Client Workbook, 2nd ed.). Oxford University Press. doi:10.1093/med:psych/
9780195336696.001.0001
Kaplan, S. C., Morrison, A. S., Goldin, P. R., Olino, T. M., Heimberg, R. G., & Gross, J. J.
(2017). The Cognitive Distortions Questionnaire (CD-Quest): Validation in a sample of
adults with social anxiety disorder. Cognitive Therapy and Research, 41, 576–587.
doi:10.1007/s10608-017-9838-9
Kessler, R., Chiu, W., Demler, O., Merikangas, K., & Walters, E. (2005). Prevalence, severity,
and comorbidity of 12-month DSM-IV disorders in the National Comorbidity Survey
Replication. Archives of General Psychiatry, 62, 617–628. doi:10.1001/archpsyc.62.6.617
Koszycki, D., Benger, M., Shlik, J., & Bradwejn, J. (2007). Randomized trial of a meditationbased stress reduction program and cognitive behavior therapy in generalized social
anxiety disorder. Behaviour Research and Therapy, 45, 2518–2526. doi:10.1016/j.
brat.2007.04.011
Leary, M. R. (1983). A brief version of the Fear of Negative Evaluation Scale. Personality and
Social Psychology Bulletin, 9, 371–375. doi:10.1177/0146167283093007
Ledley, D. R., Heimberg, R. G., Hope, D. A., Hayes, S. A., Zaider, T. I., Dyke, M. V., . . . &
Fresco, D. M. (2009). Efficacy of a manualized and workbook-driven individual treatment for social anxiety disorder. Behavior Therapy, 40, 414–424. doi:10.1016/j.
beth.2008.12.001
CBT for Social Anxiety Disorder
Liebowitz, M. R. (1987). Social phobia. Modern Problems in Pharmacopsychiatry, 22,
141–173. doi:10.1159/000414022
Liebowitz, M. R., Heimberg, R. G., Schneier, F. R., Hope, D. A., Davies, S., Holt, C. S., . . . &
Klein, D. F. (1999). Cognitive-behavioral group therapy versus phenelzine in
social phobia: Long-term outcome. Depression and Anxiety, 10(3), 89–98. PMID:
10604081.
Lincoln, T. M., Rief, W., Hahlweg, K., Frank, M., von Witzleben, I., Schroeder, B., &
Fiegenbaum, W. (2003). Effectiveness of an empirically supported treatment for social
phobia in the field. Behaviour Research and Therapy, 41, 1251–1269. doi:10.1016/s00057967(03)00038-x
Mattick, R. P., & Clarke, J. C. (1998). Development and validation of measures of social
phobia scrutiny fear and social interaction anxiety. Behaviour Research and Therapy, 36,
455–470. doi:10.1016/s0005-7967(97)10031-6
Mayo-Wilson, E., Dias, S., Mavranezouli, I., Kew, K., Clark, D. M., Ades, A. E., & Pilling, S.
(2014). Psychological and pharmacological interventions for social anxiety disorder in
adults: A systematic review and network meta-analysis. Lancet Psychiatry, 1, 368–376.
doi:10.1016/s2215-0366(14)70329-3
McCarthy, O., Hevey, D., Brogan, A., & Kelly, B. D. (2013). Effectiveness of a cognitive
behavioural group therapy (CBGT) for social anxiety disorder: Immediate and longterm benefits. The Cognitive Behaviour Therapist, 6. doi:10.1017/S1754470X13000111
McEvoy, P. M. (2007). Effectiveness of cognitive behavioural group therapy for social phobia
in a community clinic: A benchmarking study. Behaviour Research and Therapy, 45,
3030–3040. doi:10.1016/j.brat.2007.08.002
McEvoy, P. M., Nathan, P., Rapee, R. M., & Campbell, B. N. C. (2012). Cognitive behavioural group therapy for social phobia: Evidence of transportability to community clinics.
Behaviour Research and Therapy, 50, 258–265. doi:10.1016/j.brat.2012.01.009
McManus, F., Sacadura, C., & Clark, D. M. (2008). Why social anxiety persists: An experimental investigation of the role of safety behaviours as a maintaining factor. Journal of
Behavior Therapy and Experimental Psychiatry, 39, 147–161. doi:10.1016/j.
btep.2006.12.002
Mennin, D. S., Fresco, D. M., Heimberg, R. G., Schneier, F. R., Davies, S. O., & Liebowitz,
M. R. (2002). Screening for social anxiety disorder in the clinical setting: Using the
Liebowitz Social Anxiety Scale. Journal of Anxiety Disorders, 16, 661–673. doi:10.1016/
S0887-6185(02)00134-2
Nordahl, H. M., Vogel, P. A., Morken, G., Stiles, T. C., Sandvik, P., & Wells. A. (2016).
Paroxetine, cognitive therapy or their combination in the treatment of social anxiety
disorder with and without avoidant personality disorder: A randomized clinical trial.
Psychotherapy and Psychosomatics, 85, 346–356. doi:10.1159/000447013
Piccirillo, M. L., Dryman, M. T., & Heimberg, R. G. (2016). Safety behaviors in adults with
social anxiety: Review and future directions. Behavior Therapy, 47, 675–687. doi:10.1016/
j.beth.2015.11.005
Powers, M. B., Sigmarsson, S. R., & Emmelkamp, P. G. (2008). A meta-analytic review of
psychological treatments for social anxiety disorder. International Journal of Cognitive
Therapy, 1, 94–113. doi:10.1521/ijct.2008.1.2.94
Rapee, R. M., & Heimberg, R. G. (1997). A cognitive-behavioral model of anxiety in
social phobia. Behaviour Research and Therapy, 35, 741–756. doi:10.1016/s0005-7967
(97)00022-3
Rodebaugh, T. L., Heimberg, R. G., Brown, P. J., Fernandez, K. C., Blanco, C., Schneier, F.
R., & Liebowitz, M. R. (2011). More reasons to be straightforward: Findings and norms
for two scales relevant to social anxiety. Journal of Anxiety Disorders, 25, 623–630.
doi:10.1016/j.janxdis.2011.02.002
113
114
Swee, Wilmer, and Heimberg
Ruscio, A. M., Brown, T. A., Chiu, W. T., Sareen, J., Stein, M. B., & Kessler, R. C. (2008).
Social fears and social phobia in the USA: Results from the National Comorbidity
Survey Replication. Psychological Medicine, 38, 15–28. doi:10.1017/s0033291707001699
Sheehan, D. V. (2015). Mini International Neuropsychiatric Interview 7.0. Medical Outcomes
Systems.
Stopa, L., & Clark, D. M. (2000). Social phobia and interpretation of social events. Behaviour
Research and Therapy, 38, 273–283. doi:10.1016/s0005-7967(99)00043-1
Turner, S. M., Beidel, D. C., Dancu, C. V., & Stanley, M. A. (1989). An empirically derived
inventory to measure social fears and anxiety: The Social Phobia and Anxiety Inventory.
Psychological Assessment: A Journal of Consulting and Clinical Psychology, 1, 35–40.
doi:10.1037/1040-3590.1.1.35
Weeks, J. W. (2010). The Disqualification of Positive Social Outcomes Scale: A novel
assessment of a long-recognized cognitive tendency in social anxiety disorder. Journal
of Anxiety Disorders, 24, 856–865. doi:10.1016/j.janxdis.2010.06.008
Wells, A., Clark, D. M., Salkovskis, P., Ludgate, J., Hackmann, A., & Gelder, M. (1995).
Social phobia: The role of in-situation safety behaviors in maintaining anxiety and
negative beliefs. Behavior Therapy, 26, 153–161. doi:10.1016/s0005-7894(05)80088-7
Wells, A., & Papageorgiou, C. (1999). The observer perspective: Biased imagery in social
phobia, agoraphobia, and blood/injury phobia. Behaviour Research and Therapy, 37,
653–658. doi:10.1016/s0005-7967(98)00150-8
Wolpe, J., & Lazarus, A. A. (1967). Behavior therapy techniques: A guide to the treatment of
neuroses. Pergamon Press.
7
CBT for Social Anxiety Disorder
Henrik Nordahl and Adrian Wells
Introduction
Social anxiety disorder (SAD) is characterized by a marked fear or anxiety concerning one or more social situations in which the individual may be scrutinized by
others (DSM-5; American Psychiatric Association [APA], 2013). Individuals with
SAD are concerned with their social performance and fear that others will perceive
them negatively, for example, as anxious, boring, stupid, weak, or unlikeable and
that this will lead to embarrassment, humiliation, rejection, or loss of self-worth.
These concerns can be so pronounced that the individual avoids most social situations or endures them with intense discomfort.
SAD is one of the most common mental health disorders, with a 12-month
prevalence estimate of 7% and a lifetime estimate of 12% in the United States
(Ruscio et al., 2008). The disorder often has an early onset with a median of 13 years
of age, and 75% of individuals with SAD have an onset between 8 and 15 years (APA,
2013). In addition, SAD is considered a relatively chronic disorder if left untreated and
is associated with lower quality of life. It is also considered to be a major risk factor for
developing major depressive disorder, other anxiety disorders, substance abuse/addiction, and work-related problems (APA, 2013; Wittchen & Fehm, 2003). Effective
clinical management and treatment of the disorder is therefore of great importance.
The Cognitive Model of SAD
Building on Beck’s schema theory (Beck, 1976) and the self-regulatory executive
function (S-REF) model of psychological disorder (Wells & Matthews, 1994), Clark
and Wells (1995) advanced a cognitive model of SAD that has received substantial
empirical support. Central to the model is the view that individuals with SAD
engage in an information processing style characterized by self-focused attention.
Self-attention is not capable of providing unambiguous disconfirmation of social
fears and concerns in feared social situations, as the primary source of information
comes from feelings and sensations rather than external information which has the
115
116
Henrik Nordahl and Adrian Wells
Social Situation
Activates assumptions
Perceived social danger
(negative automatic thoughts)
Processing of
Self as a Social
Object
Safety
Behaviors
Somatic and
cognitive
symptoms
Figure 7.1. A cognitive model of social phobia. From Wells (1997, p. 169). Copyright 1997 by
John Wiley & Sons Limited. Reprinted by permission
potential to correct distorted beliefs. Hence, the Clark and Wells model accounts for
the persistence of SAD with reference to a number of specific cognitive-behavioral
mechanisms involving vicious circles that are responsible for maintaining the problem. This model is depicted in Figure 7.1.
Assumptions and Beliefs
Resulting from past experience, individuals with SAD hold negative assumptions
and beliefs about social situations and the social self that are activated when
entering or anticipating a social situation. These beliefs contribute to negative
appraisals of social situations and the vicious cycles that maintain SAD. The
following three types of assumptions and beliefs are identified in the model:
1. Conditional assumptions (e.g., “If I show signs of anxiety, people will think I am
weak”; “If people like me they will begin the conversation first”)
2. Unconditional beliefs about the social self (e.g., “I am boring”; “I am unlikeable”)
3. High standards and rules for social performance (e.g., “I must always get
everyone’s approval”; “I have to appear intelligent and relaxed”)
CBT for Social Anxiety Disorder
Activation of such assumptions and beliefs leads to a perception of potential
social danger, which is evident as anticipatory worry or negative automatic
thoughts – for example, “What if I shake?”; “They will notice I’m anxious”; and
“I’ll babble and talk funny” – which increase the sense of threat and anxiety.
Furthermore, the somatic and cognitive symptoms that accompany anxiety are
themselves subject to negative appraisal and may be interpreted as evidence of
failure and impending social humiliation.
Processing of the Self as a Social Object
Negative automatic thoughts about the social self not only are associated with an
increase in anxiety symptoms but also are accompanied by a shift in attention in
which the individual becomes self-conscious and engages in self-processing with a
specific focus toward how they think they appear to others. The socially anxious
individual uses internal information in the form of feelings or an image of the self in
their mind’s eye to infer how they must appear. The image of the self typically
consists of an “observer perspective,” meaning that it entails seeing oneself as if
from someone else’s vantage point. In images of this type, anxiety symptoms and
personal inadequacies are seen as highly conspicuous, and the socially anxious
person assumes that the negative observer image accurately reflects what other
people can see in them. This process in turn reinforces negative self-appraisals of
performance and negative thoughts about evaluation by others.
Safety Behaviors
In order to prevent feared social outcomes, such as showing anxiety or failed
performance, safety behaviors are used. For example, individuals fearful of saying
something that others will judge as stupid will often say little or mentally rehearse
sentences before talking. Contrary to their intended effect, safety behaviors are
problematic and maintain social anxiety in several ways. They contribute to
heightened self-focused attention, prevent disconfirmation of negative beliefs and
assumptions, increase feared symptoms, can draw attention toward the individual,
and have the potential to contaminate the social situation by, for example, making
the person with SAD appear withdrawn or unfriendly.
In addition to the safety behaviors that are used during a social encounter,
avoidance of social situations is a problem because it prohibits experiences that
could disconfirm assumptions and beliefs. It is therefore crucial to deal with in order
to maximize treatment effects.
Post-Mortem and Anticipatory Processing
There are two other strategies that individuals with SAD use to deal with social
concerns that occur before or after social encounters. Before exposure to situations,
anticipatory worry and planning are used to try to deal with and avoid social threat,
117
118
Henrik Nordahl and Adrian Wells
for example, mentally rehearsing conversations. This is a negative process that
heightens the sense of danger and anxiety. On leaving the social situation the
individual may also engage in a “post-mortem” that consists of rumination, analyzing their performance, and recalling what might have gone wrong. This process can
transform a relatively positive experience into a sense of failure to perform well.
Each of these processes contributes to emotional distress and negative selfprocessing even in the absence of direct social threat.
Research Supporting CBT for SAD
There is substantial empirical support for the key aspects of the Clark and Wells
model such as the role of observer perspective imagery (e.g., Wells, Clark &
Ahmad, 1998) and reviews of self-focused attention (Ng et al., 2014), safety behaviors (Piccirillo et al., 2015), and dysfunctional self-beliefs (Gregory & Peters, 2017).
In terms of treatments for SAD, findings are summarized in a systematic review
and network meta-analysis by Mayo-Wilson and colleagues (2014) that aimed to
compare interventions for SAD and identify which were the most effective in adults.
They included 101 trials which explored 41 interventions or control conditions.
Compared with wait-list control, several classes of medications (monoamine oxidase
inhibitors, benzodiazepines, selective serotonin-reuptake inhibitors, serotoninnorepinephrine reuptake inhibitors, and anticonvulsants), individual CBT, group
CBT, exposure and social skills training, self-help with support, self-help without
support, and psychodynamic psychotherapy were efficacious. However, individual
CBT was found to be most effective, and the authors concluded that, in particular,
individual CBT that was based on the Clark and Wells (1995) model provided large
effects (standardized mean difference compared with wait-list reported to be 1.56,
with 95% confidence interval for recovery from 1.85 to 1.27 based on three
included studies). This finding is in line with the most updated clinical guidelines for
adults with SAD, for example, by the National Institute for Health and Care
Excellence (NICE, 2013); however, the meta-analysis has also been criticized for
being biased in favor of CBT in drawing its conclusions (Wampold et al., 2017).
The efficacy of CBT based specifically on the Clark and Wells (1995) model and
treatment manuals (e.g., Wells, 1997) has been evaluated in several randomized
controlled trials. Clark et al. (2003) reported that CBT was superior to fluoxetine
combined with self-exposure, and superior to placebo combined with self-exposure.
The effect size for CBT pre- to post-treatment on social anxiety symptoms was large
(Cohen’s d = 1.31). In a different study, Clark and colleagues (2006) demonstrated that
CBT was superior to exposure plus applied relaxation. The effect size for CBT was
reported to be large from pre- to post-treatment (Cohen’s d = 2.63) and 86% of patients
were classified as recovered at post-treatment. In this study, 78% of the patients who
met criteria for avoidant personality disorder at pretreatment no longer met the criteria
at one-year follow-up. Both these studies reported that treatment effects were maintained at follow-up one year after treatment. Moreover, CBT was also associated with
large effect sizes on secondary outcome measures such as depression.
CBT for Social Anxiety Disorder
Stangier and colleagues (2003) compared individual with group CBT and found
that the treatment was most efficacious in the individual format with a large mean
effect size for social anxiety measures at post-treatment (Cohen’s d = 1.17) and at
six-month follow-up (Cohen’s d = 1.57). In a similar study, Mörtberg and colleagues
(2007) reported individual CBT to be more efficacious than group and intensive
group CBT. The individual CBT was associated with a large mean effect size for
social anxiety measures post-treatment (Cohen’s d = 1.62) and at one-year follow-up
(Cohen’s d = 1.89).
CBT has been compared with interpersonal therapy (IPT) in two studies.
Stangier et al. (2011) reported that CBT was superior to IPT and that the response
rate for CBT was 65.8% and largely maintained at one-year follow-up. CBT was
associated with large controlled effect sizes on measures of social anxiety and
medium controlled effect sizes for depression symptoms. In another study, CBT
was evaluated against IPT in a residential setting: Borge and colleagues (2008)
reported a medium effect size for CBT from pre- to post-treatment (Cohen’s d =
0.76) and that only 31% of patients were classified as recovered post-treatment.
However, the effects of CBT were not statistically different from those of residential
interpersonal therapy.
Leichsenring et al. (2013) conducted a large RCT where they compared CBT to
psychodynamic therapy. They found CBT to be the superior intervention and
reported a remission rate for CBT of 36%. The effect size for CBT from pre- to
post-treatment was large (Cohen’s d = 1.32). However, it is important to note that
Leichsenring and colleagues delivered CBT over a mean of 25.7 sessions, suggesting
that the treatment manual was not adhered to.
To summarize, CBT based on the Clark and Wells model is associated with
moderate to large effects on primary and secondary outcome measures, which are
largely maintained at follow-up. The efficacy of the treatment seems to be reliant on
adherence and competence factors. Moreover, the treatment has been found to be
superior to other psychological treatments and medications in most direct
comparisons.
Clinical Management of SAD
In the remainder of this chapter, we will describe the clinical management of SAD
from a CBT orientation in terms of issues related to assessment, formulation, and
intervention. A more extensive and detailed description of this CBT for SAD can be
found in the treatment manual by Wells (1997, pp. 167–199).
Effective interventions for SAD such as CBT may not be accessed due to poor
recognition and inadequate assessment. Often, individuals with primary SAD present in the clinic with other problems such as depression or substance dependence,
and will reveal their problem with social anxiety only on direct questioning. In
addition, many patients with various disorders present with social evaluative concerns and anxiety in social situations that are not manifestations of SAD. For
example, patients with paranoid personality problems can present social anxiety
119
120
Henrik Nordahl and Adrian Wells
due to suspiciousness and mistrust in others, while patients with body dysmorphic
disorder can present social anxiety related to their obsessive belief about the
conspicuousness of a “flawed body part.” Thus, an initial and crucial step in the
successful formulation and treatment of these patients is a thorough clinical assessment and accurate diagnosis of the presenting problem. Two of the most commonly
used semi-structured interviews for diagnosing SAD are the Anxiety Disorders
Interview Schedule for DSM-IV (ADIS-IV; Brown et al., 1994) and the
Structured Clinical Interview for DSM-IV Axis I Disorders (SCID-IV; First et al.,
1996). Both instruments are based on the diagnostic criteria outlined in DSM-IV,
but recent revisions of these instruments have been published following the publication of DSM-5 (APA, 2013): ADIS-5 (Brown & Barlow, 2014) and SCID-5-RV
(First et al., 2015).
Assessment
Following a thorough general clinical assessment and accurate diagnosis of SAD,
more SAD-specific assessment and measurement will serve several aims. These
include assessing severity of the problem, identifying which situations are associated
with most fear and/or avoidance, elicitation of information about the maintaining
mechanisms for case conceptualization, and monitoring treatment outcome.
Measurement should be used to determine progress in meeting treatment goals
and as a guide to modifying components of treatment in a way that maximizes the
probability of cognitive behavioral change. For example, the continuation of counterproductive safety behaviors and avoidance should be monitored throughout
treatment as they prohibit necessary disconfirmation of social fears and the acquisition of new information.
There is a range of clinical assessment tools that can be used to assess social
anxiety and the components thought to maintain it. These have recently been
summarized in a review by Wong et al. (2016), which the interested reader can
consult for more detailed information. A brief overview of scales frequently used in
research and with good utility for clinical practice are outlined.
Clinician-Administered Outcome Measures
In addition to the ADIS-IV/ADIS-5, commonly used clinician-administered scales
include the Liebowitz Social Anxiety Scale (LSAS; Liebowitz, 1987) and the Brief
Social Phobia Scale (BSPS; Davidson et al., 1997). All these assessment tools
provide separate ratings of fear and avoidance for different social situations. Thus,
these instruments can be of great use in planning treatment as they provide information about the severity and breadth of social anxiety and which situations are
associated with the most fear and/or anxiety for the patient.
Self-Report Outcome Measures
There is a range of self-report measures that are established and widely used to
assess social anxiety. Although these measures overlap, there are slight differences
CBT for Social Anxiety Disorder
in terms of what they assess. The Liebowitz Social Anxiety Scale has also been
developed as a self-report version (LSAS-SR; Baker et al., 2002) and assesses fear
and avoidance across 24 different social situations. The Social Interaction Anxiety
Scale (SIAS; Mattick & Clarke, 1998) measures distress related to interaction with
others. The Social Phobia Scale (SPS; Mattick & Clarke, 1998) assesses anxiety
related to being observed or watched by others. The Social Phobia and Anxiety
Inventory (SPAI; Turner et al., 1989) assesses symptoms, cognitions, and behavior
in a range of social situations. The Social Phobia Inventory (SPIN; Connor et al.,
2000) assesses fear, avoidance, and physiological discomfort related to social situations. Moreover, shortened versions have been developed for several of the most
commonly used scales to reduce administration times, which can be an important
factor in clinical settings. For a comprehensive overview of these measures, the
interested reader should consult Wong et al. (2016).
Self-Report Measures of SAD-Maintenance Factors
Cognitive and behavioral factors that maintain social anxiety are usually assessed
with self-report measures. The Social Phobia Rating Scale (SPRS; Wells, 1997) was
published together with the original treatment manual and was designed as a clinical
assessment tool for assessment and monitoring of social anxiety and its cognitive
behavioral maintenance components when treating SAD patients with CBT. The
SPRS has 32 items and five subscales that assess distress, avoidance, selfconsciousness, use of safety behaviors, and negative beliefs characterizing SAD.
In addition, several measures more specifically assess cognitive and behavioral
components of the cognitive model that we briefly mention below.
Negative beliefs characterizing social anxiety can be assessed with the Social
Cognitions Questionnaire (SCQ; Wells et al., 1993). In addition, the Self-Beliefs
Related to Social Anxiety Scale (SBSA; Wong & Moulds, 2009; Wong et al., 2014)
was developed to assess high standards, unconditional beliefs, and conditional
beliefs based on the Clark and Wells (1995) model. Anticipatory processing can
be assessed with the Anticipatory Social Behaviours Questionnaire (ASBQ;
Hinrichsen & Clark, 2003), and post-event processing can be assessed with the
Post-Event Processing Questionnaire (PEPQ; Rachman et al., 2000). Self-attention
can be assessed with the Self-Consciousness Scale (SCS; Fenigstein et al., 1975),
and, in particular, one of its subscales (public self-consciousness) is relevant to the
Clark and Wells (1995) model. Safety behaviors can be assessed with the Social
Behaviours Questionnaire (SBQ; Clark et al., 1995). Avoidance of social situations
is best assessed with the previously described outcome measures.
Cognitive Behavioral Therapy for Social Anxiety Disorder
Structure of Treatment
A course of CBT treatment for SAD (Wells, 1997) is typically implemented over
12–14 sessions, in which sessions are held weekly and each session is normally
121
122
Henrik Nordahl and Adrian Wells
60 minutes in duration. Moreover, the treatment should follow a particular
sequence in maximizing the efficiency of cognitive behavioral change. It is a logical
derivation from the model to use strategies early in treatment that reconfigure the
patient’s behaviors and focus of attention in a way that maximizes subsequent
change in negative thoughts and beliefs. More specifically, if behavioral experiments
involving exposure proceed without modifying the use of maladaptive safety behaviors and reducing self-focused attention, the effectiveness of such strategies may
be impaired.
Sessions 1–3 of treatment usually consists of case formulation, socialization, and
cognitive preparation for restructuring, involving manipulations of safety behaviors
and of attention. Treatment sessions 4–6 typically focus on a continuation of
behavioral experiments, often involving exposure to test negative appraisals and
predictions, and video feedback methods to correct the distorted self-image.
Sessions 7–9 continue with cognitive and behavioral reattribution methods, and
introduce bandwidth maneuvers aimed at further interrogating the environment
and discovering that social catastrophes (e.g., everyone staring at you) are unlikely,
even in the event of failed performance. Finally, sessions 10–12 involve consolidation of material learned, relapse prevention, and a continuation of work on the
remaining issues (i.e., residual negative beliefs and avoidance). Clearly, there is
flexibility in the number of sessions devoted to these phases of treatment, but it
serves here to provide a basic conceptual structure for planning
specific interventions.
Formulation
In the following, we present a fictional but realistic case of Jenny to illustrate the
process from assessment to case formulation and socialization. First, we provide
some background information about Jenny.
Background Information
Jenny is a single, 32-year-old architect with no children. After graduating from
university three years ago, Jenny moved to a new city a long way from her family
and childhood friends to work for a medium-sized construction company. This was a
large step for Jenny as she had always been a cautious person and most comfortable
in safe and well-known surroundings. However, the construction company had a
great reputation, and Jenny was excited that she was offered a job there as a newly
qualified architect.
After three years in the city, Jenny did not know anyone outside work, and she
rarely spent time with colleagues outside the office. Most of her spare time was
spent going for walks, doing yoga, or just sitting in front of the TV. Jenny felt lonely
and depressed, and she also struggled at work. She very much enjoyed working by
herself on, for example, designing houses, but she found teamwork challenging as it
nearly always had her feel very anxious; she avoided eating lunch with her colleagues; and she hated attending meetings and other formal settings. Sometimes
CBT for Social Anxiety Disorder
Jenny found it difficult to ask her manager for help, even when she needed supervision to do her job effectively. On some occasions, typically before meetings where
she had to present some of her design drawings to several spectators, Jenny would
drink wine before presenting. During the past three years, Jenny had been on
several periods of short-term sick leave due to depression and fatigue, and her
general practitioner had prescribed antidepressant medications that had helped
somewhat. However, in her latest encounter with her general practitioner, Jenny
agreed to be referred to a mental health professional for assessment and treatment
as the problems with depression and fluctuating work performance endured.
Jenny’s therapist initially conducted a standardized diagnostic interview and
concluded that Jenny met the diagnostic criteria for social anxiety disorder and
recurrent depressive disorder. Jenny reported avoidance of performance situations
and concerns about being negatively evaluated since adolescence. She agreed with
the therapist that depression would be less of a problem if she could have a richer
social life and be more relaxed when interacting with others during the work day.
The therapist questioned Jenny about her worst fears in social situations at
work. The main problem as Jenny saw it was trembling in her whole body and in her
voice, and she was certain that others would think she was a stupid and weak person
if they noticed it. Therefore, she avoided as many performance situations as she
could. However, when she was left with no choice other than confronting her feared
situations, Jenny reported that she tried to control her body and voice with the aim
of concealing her anxiety.
Eliciting Information for Case Formulation
When working with SAD, there are two basic approaches that can be used to elicit
data for conceptualization. The first option is reviewing one or more recent episodes
in which the patient felt socially anxious by direct questioning. However, for some
individuals where avoidance of feared situations is a prominent problem, it might be
necessary to conduct a behavioral assessment test where the individual is exposed to
a feared situation constructed in session. Depending on the nature of the patient’s
fear, this will typically involve introducing a type of performance situation for the
patient such as making conversation with a stranger. For an example of a behavior
test to elicit data for conceptualization, the reader can consult Wells (1997, p. 180).
Four types of information are required to make an idiosyncratic case formulation. The therapist aims to identify (1) main negative automatic thoughts, (2)
anxiety symptoms, (3) contents of self-consciousness/inner image, and (4)
safety behaviors.
Negative Automatic Thoughts. Negative automatic thoughts occur in anticipation of and within social situations. Two types of content are relevant to problem
maintenance: (1) the meaning and significance that other people might attach to
symptoms and (2) an individual’s own appraisal of the significance of symptoms
such as overestimation of conspicuousness and implications for self-concept. Useful
questions to elicit negative automatic thoughts include (1) “What were you thinking
before you entered the situation/in the situation/when you noticed symptom X?”
123
124
Henrik Nordahl and Adrian Wells
(2) “What was the worst you thought could happen in the situation?” and (3) “What
did you think people would think or see?”
Anxiety Symptoms. Anxiety takes the form of cognitive and somatic symptoms.
They are maintained by negative appraisals, but are also a focus of negative
appraisals. Individuals with SAD are often most troubled by symptoms that can
be witnessed by others, such as shaking, sweating, blushing, crying, or their mind
going blank. In constructing the idiosyncratic formulation, it is useful to determine
the extent to which appraisals of symptoms contribute to negative automatic
thoughts and dysfunctional self-processing. The following questions can be used:
(1) “Which symptoms bother you most?” (2) “When you felt anxious in the
situation, what symptoms did you notice?” (3) “How conspicuous do you think
the symptoms are?” and (4) “If people did notice your symptoms what would
that mean?”
Contents of Self-Processing. There are three main ways to explore the content
of an individual’s self-processing: (1) exploring the content of heightened selfconsciousness, (2) questioning the appraised level of conspicuousness of symptoms,
and (3) determining if safety behaviors are linked to a particular self-perception.
The initial marker for self-processing in patients’ accounts of situations is a report of
increased self-consciousness. The therapist should specifically ask about the point in
time at which the patient became highly self-conscious, either through direct exploration of the content of processing in this state (e.g., “What aspect of yourself were
you most aware of?”) or by questioning the subjective impression of the self for
periods when symptoms were intense (e.g., “When you felt anxious, how do you
think you appeared?”). Moreover, some patients utilize safety behaviors to conceal
symptoms, and this provides an opportunity to determine the content of selfperception in two ways: (1) explore self-processing associated with implementing
safety behaviors (e.g., “When you try to conceal your symptoms, what is your
impression of how you look to others?”) and (2) explore self-processing in the
hypothetical absence of safety behaviors (e.g., “If you did not engage in your safety
behavior when you felt anxious, how would you look to others then?”). It is
important to try to determine the content of self-processing in as detailed and
precise a way as possible, and the therapist should ask about imagery, as the selfimpression often occurs as an image from the “observer perspective” (e.g., “Did you
have an image of the way you thought you looked when you were in the situation?
Can you describe the image”?).
Safety Behaviors. Safety behaviors can be overt or covert. It is important that
the therapist works to elicit all kinds of safety behaviors, which can be a challenge
when there is low awareness by the patient. In addition to questioning about safety
behaviors, exposure to feared situations in conjunction with questioning from the
therapist may be warranted. Examples of typical question to elicit safety behaviors
are: (1) “When you thought (feared event) was happening, did you do anything to
prevent it?” (2) “Do you do anything to control your symptoms/improve your
performance/hide your problem?” and (3) “Do you do anything to avoid drawing
attention to yourself?”
CBT for Social Anxiety Disorder
Collecting Data for Jenny’s Formulation
In the case of Jenny, reviewing a recent episode of social anxiety was chosen to elicit
data for her idiosyncratic case formulation. Below we have provided a description of
the conversation between Jenny and her therapist from the process of generating
her formulation. The formulation arising from the dialogue is presented in figure 2.
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
Jenny:
Therapist:
In today’s session, I would like to explore a recent social situation
in which you felt anxious and uncomfortable. In doing this we can
explore factors that are keeping your social anxiety going and
begin to examine ways that you can overcome your anxiety.
When was the last time you were anxious and distressed in a
social situation?
It was on Monday. I went to a meeting with two of my colleagues
to talk to some customers about a new project.
I am going to ask you some questions about your feelings and
thoughts in that situation. Is it clear in your memory?
Yes.
Just before you went to the meeting, were you feeling anxious?
Very much so, I did not really want to go, but it was an important
meeting and I did not want to let my colleagues down.
What were your anxious thoughts?
I was worried that I had to take the lead in the conversation, and
that my anxiety would lead to trembling in my voice and body. I do
not want others to see me shaking and being anxious for nothing.
So, your thoughts were something like “What if I shake and
everybody will notice?” Is that right?
Yes, that is right.
When you entered the situation, how did you feel?
I felt anxious, tense, hot, and shaky.
When you felt anxious in that situation, what thoughts went
through your mind?
I thought, “They will notice the shaking and think I am an
incompetent person.”
It sounds like you think others will perceive you as an
incompetent person if they saw you shaking. How conspicuous do
you think your symptoms are?
I think it is quite easy to notice that I am nervous
and uncomfortable.
If I could have seen you in that meeting, what would I see?
You would see a shivering wreck. It felt like my whole body
was shaking.
When you felt that your whole body was shaking, did you do
anything to prevent it?
125
126
Henrik Nordahl and Adrian Wells
Situation: Meeting at work
NAT: “They will notice the shaking and think I am an incompetent person”
Self-Conscious
Image of self as a
“shivering
wreck”
Safety Behaviors
Tense legs
Hide arms
Deep breaths
Control shaking
Say little
Anxiety
Anxious
Tense
Hot
Shaky
Figure 7.2. An idiosyncratic SAD case conceptualization for Jenny.
Jenny:
Therapist:
Yes, I tried to tense my legs to control the shaking. I hid my arms
behind my back, and I took deep breaths to calm down. I usually
say very little to avoid attracting attention.
Thank you for answering these questions, Jenny. Now, let us have
a look together at a diagram [Figure 7.2] I have mapped out to
see some important factors and cycles that help us understand the
causes of your anxiety problem.
Socialization
When the idiosyncratic formulation has been developed, the therapist presents the
model using guided discovery to begin to share an understanding of the factors
maintaining the problem. A goal in the socialization process is to engage the patient
in the treatment and to demonstrate that social anxiety is linked to negative selfevaluation based on internal information. In particular, the effect of self-focused
attention and use of safety behaviors on symptoms and performance is discussed
and illustrated by in-session experiments.
Behavioral Experiments in Socialization
Typically, a socialization experiment involves social performance under two conditions where the engagement in and disengagement from safety behaviors and/or
CBT for Social Anxiety Disorder
self-focused attention are contrasted. The first condition usually involves full
engagement in self-focus/safety behaviors, and this is followed by a second condition
where the patient is instructed to reduce these strategies as much as possible.
Several socialization experiments are usually required in order to demonstrate the
key elements in the model and the interactions between them. For examples and
further discussion, the reader should consult Wells (1997).
In the case of Jenny, one of the socialization experiments aimed to explore the
effects of her safety behaviors. She was asked to participate in two interactions with
a small group of the therapist’s colleagues. In the first interaction, Jenny was
instructed to engage all of her typical safety behaviors (e.g., tensing her legs, hiding
her arms, taking deep breaths, trying to control shaking, and saying little). In the
second interaction, Jenny was instructed to drop all safety behaviors, and focus
attention externally on other people in the situation. When the two conditions were
contrasted with appropriate questioning by the therapist, Jenny discovered that her
self-consciousness was greater and her trembling more noticeable in the first condition. She also felt more at ease in the second condition and experienced that
participating in the conversation felt more natural. The experiment had the effect
that Jenny was more curious about the role of safety behaviors and self-attention;
this increased her motivation for therapy and to reduce these responses.
Modifying Self-Processing with Video Feedback
Following socialization and successful work in reducing safety behaviors, the next
stage of treatment consists of modifying the content of self-processing, as it is the
main source of evidence used by the socially anxious individual to make negative
self-appraisals and inferences about the content of other people’s appraisals. Video
feedback provides an effective technique for correcting distorted self-images and
beliefs concerning the conspicuousness of anxiety symptoms, and can help patients
obtain a more realistic view of how they appear to other people. This procedure
should be managed in a particular way to prohibit adverse effects and maximize
change in self-image and negative beliefs. Clinical guidelines detailing a range of
procedures for successful implementation of the procedure have been published
elsewhere (Warnock-Parkes et al., 2017; Wells, 1997).
Video feedback can be used in a variety of different ways during a course of
CBT for SAD. Normally, a video of the behavioral experiment with two contrasting
conditions is used as part of the socialization process where the patient likely
experienced high levels of anxiety as described above. This experiment was originally set up to evaluate the effect of self-focused attention and safety behaviors, and
patients usually discover that they feel less anxious and think they came across more
favorably in the second condition. In the case of Jenny, watching the videos helped
her discover that her trembling was barely noticeable and that she came across more
favorably in both conditions than she thought. For Jenny, this insight meant that her
feelings are not a reliable judge of how she appears to others and that even when
127
128
Henrik Nordahl and Adrian Wells
feeling shaky it is barely noticeable to others. In addition, Jenny realized that the
safety behavior of tensing her legs likely contributed to the felt shakiness.
Verbal Reattribution
Throughout therapy, verbal reattribution techniques are used with a view to modifying negative self-appraisals and thoughts concerning other people’s reactions and
thoughts in social situations. In addition to video feedback, there are several strategies that can be used for challenging the patient’s belief in their negative automatic
thoughts, of which we present some of the most commonly used techniques here.
Questioning the Evidence
A typical strategy to use is to review the evidence that the socially anxious individual has for their belief. A central aim in doing so is to help the patient discover that
the evidence mostly stems from internal information such as feelings/symptoms and
self-appraisal rather than objective sources.
Thinking Errors
Educating patients to identify thinking errors is a good approach as a means of
helping the patient gaining distance from their negative appraisals and objectively
evaluating their validity. Common thinking errors in SAD include mind reading
(e.g., “She thinks I am stupid”), catastrophizing (e.g., “I won’t be able to say
anything”), and personalization (e.g., “No one is talking to me because I am
boring”). Some also engage in an error of projected self-appraisal (Wells, 1997) in
which they assume their own negative beliefs are held by others (e.g., “I sound
really boring; they must think I am boring”).
Dealing with Anticipatory Processing and the Post-mortem
These cognitive types of safety behaviors are typically dealt with by asking patients
to ban or reduce the time spent on these processes. To make this easier for the
patient, an analysis of the advantages and disadvantages of such processing is
usually undertaken with the aim to show how the disadvantages outweigh the
advantages. In the case of Jenny, her therapist made a list of all the advantages
and disadvantages of planning what to say (i.e., anticipatory processing) that Jenny
could think of. According to Jenny, the advantage with this coping strategy was that
it could help her come across as a competent person. The disadvantage was that she
became more self-focused and anxious when engaging in this strategy. In addition,
Jenny’s therapist helped her see other disadvantages with using the strategy, for
example, that her planning seldom fitted with how meetings and conversations
actually turn out and that the strategy had not helped her overcome her social
anxiety problem. In reviewing the advantages and disadvantages, Jenny concluded
that the disadvantages by far outweighed the advantages and that anticipating
potential negative social consequences contributed to her social anxiety problem.
CBT for Social Anxiety Disorder
She agreed to ban the activity as homework and to spend time on evaluating her
experiences from this in the next treatment session.
Using Rational Self-Statements
When more rational statements have been generated and/or validated by verbal
retribution and behavioral strategies during the course of treatment, these can be
used as self-statements in feared situations as a means of preventing full activation
of self-processing and of maintaining stability of the self-concept. For example,
Jenny was instructed to use a rational self-statement (“I look calm even when
I don’t feel it”) based on corrective video feedback in challenging social situations.
Behavioral Experiments
Behavioral experiments consisting of exposure to social situations with concurrent
dropping of safety behaviors and processing of external information are a crucial
component of CBT for SAD. They complement verbal reattribution procedures and
provide one of the most powerful means of modifying distorted appraisals and
beliefs by enabling testing of other people’s reactions and by evaluating the consequences of showing signs of anxiety or failed performance in feared social situations.
We recommend implementing behavioral experiments in accordance with the P-ET-S protocol described by Wells (1997), which consists of four phases: (1) preparation (P), (2) exposure to social situations (E), (3) testing the predictions based on
negative beliefs (T), and (4) a final summarizing (S) of the results.
Examples of test (T) phases in such experiments include deliberately focusing on
other people to determine their reactions, dropping safety behaviors, deliberately
showing signs of anxiety, producing “failures in performance,” or behaving in an
“unacceptable” manner and observing if predicted outcomes occur. Such testing
usually requires that the patient maintain an external focus of attention as a means to
gather objective information about other people’s reactions. After the testing phase,
the experiences are interpreted and summarized in light of the cognitive formulation,
and belief in negative thoughts and predictions is reassessed in light of these results.
Experiments conducted in session are normally repeated for homework in order to test
the predictions in more naturalistic settings for the patients with means to further
challenge negative thoughts and beliefs. In the case of Jenny, she did an experiment
in a meeting at work where she deliberately shook her arms while carefully observing
her colleagues’ reactions. To her surprise, Jenny discovered that only one of her
colleagues noticed, but this person did not seem to react to it in any negative may.
This discovery was used by the therapist to help Jenny discover that even in the event of
“failed performance,” strong negative reactions from others are unlikely.
Dealing with Underlying Assumptions and Beliefs
In the Clark and Wells model, individuals with SAD hold underlying negative
assumptions and beliefs (“schemas”) that are activated when entering or
129
130
Henrik Nordahl and Adrian Wells
anticipating a social situation and contribute to negative appraisals of social situations and the vicious cycle that maintains SAD. These schemas can be divided into
conditional assumptions, unconditional beliefs about the social self, and high standards and rules for social performance, and should be addressed and modified in the
later stages of the treatment. To accomplish this successfully, it is important to
generate a clear definition of concepts represented in the assumptions and to
operationalize them in a testable format. A good strategy in working with conditional assumptions is to review evidence and counterevidence for the assumptions,
as individuals with SAD often fail to process disconfirmatory information. This can
be followed up with a mini-survey to gather information about other people’s
thoughts and beliefs, and it may be useful to run behavioral experiments to test
the validity of the assumption.
When the therapist aims to modify high standards and rigid rules for social
performance, it is useful to explore the effects of behaving in ways that break these
personal rules. For example, the patient may be asked to deliberately “make
mistakes” or spill a drink in a social situation to test the reactions of others.
With unconditional negative self-beliefs, which often consist of dichotomous
concepts, continua work offers a means of introducing a more nuanced perspective
on the self. The positive data log and flashcards provide a means of accumulating
evidence in support of replacement positive beliefs with the aim of counteracting
negative cognitive bias in feared situations. In some cases, identifying early events
and images that might explain how a negative self-belief arose can also be helpful,
and either encouraging the patient to treat their negative self-beliefs as prejudice
against themselves or using elements from trauma interventions such as exposure or
memory rescripting to process the traumatic event and thus overcome the
negative belief.
Relapse Prevention
Toward the end of therapy, a central part of the treatment is relapse prevention.
Patients are typically asked to write a detailed summary of the information learned
during the course of treatment, a task that the therapist should supervise to make
sure most of the important learning points and experiences are included. In addition, several examples of the patient’s formulation that highlight the more specific
components responsible for maintaining social anxiety are included in the summary.
It is also typical to include a list of strategies that have been used in overcoming
social anxiety. Furthermore, booster sessions are normally scheduled at two and six
months after treatment and provide an opportunity to consolidate treatment gains
and reinforce the implementation of treatment strategies.
Recent Developments and Advances
Although it has been nearly 25 years since the Clark and Wells model and the
treatment derived from it was published, it is still considered the benchmark model
CBT for Social Anxiety Disorder
and treatment of SAD. In more recent years, treatment based on the model has
successfully been adapted to children and adolescents (Ingul et al., 2014; Leigh &
Clark, 2016, 2018) and to different formats such as delivery through the Internet
(see Carlbring et al., 2018, for a review of controlled trials) with promising results.
A Return to Theoretical Influences: The Metacognitive Approach.
A conceptual feature of the Clark and Wells model (1995) is that it draws on
different theoretical frameworks in an integrative way that may create upper limits
to what can be achieved. For example, it places the content of cognition in center
stage and argues that schemas or negative beliefs give rise to self-processing and
social anxiety. However, the S-REF model (Wells & Matthews, 1994) that partially
informed its development emphasizes the failure to adaptively regulate processing
such as worry and attention as the main features of disorder. This effect is thought to
emerge from metacognitive beliefs, and together these are the targets of metacognitive therapy (Wells, 2009). In line with this idea, recent studies have shown that
metacognitive (e.g., “I cannot control my worry”) rather than cognitive beliefs (e.g.,
“I am boring”) are more reliable predictors of social anxiety (Gkika et al., 2017;
Nordahl & Wells, 2017). Moreover, change in metacognitive rather than cognitive
beliefs is associated with symptom improvement in individuals undergoing treatment for SAD (Nordahl et al., 2017). Metacognitive but not cognitive beliefs are
statistical predictors of depression symptoms in patients with SAD (Nordahl et al.,
2018), and of work status among high socially anxious individuals (Nordahl & Wells,
2017). Two studies (Nordahl et al., 2016; Wells & Papageorgiou, 2001) have shown
that a briefer and more metacognitive-focused intervention can be highly effective
and time efficient for patients with SAD. Furthermore, Vogel and colleagues (2016)
isolated and tested two techniques: the attention training technique (ATT: Wells,
1990) and situational attentional refocusing (SAR: Wells & Papageorgiou, 1998)
often used in metacognitive therapy applications, and found that 46% of patients no
longer met diagnostic criteria for SAD after receiving them. Nordahl and Wells
(2018) have presented preliminary evidence suggesting that full metacognitive
therapy is associated with positive outcomes for patients with different presentations of SAD. These emerging data support the idea that moving toward a greater
metacognitive-focused implementation of treatment may contribute positively to
further treatment developments.
Key Points
Social anxiety disorder (SAD) is among the most common mental disorders,
with a lifetime prevalence of approximately 12%.
Building on Beck’s schema theory (Beck, 1976) and the self-regulatory
executive function model of psychological disorder (Wells & Matthews,
1994), Clark and Wells (1995) advanced a cognitive model of SAD.
The cognitive model emphasizes schemas/negative automatic thoughts, selfattention, and safety behaviors as the key maintenance factors of
social anxiety.
131
132
Henrik Nordahl and Adrian Wells
(cont.)
Wells (1997) developed the CBT manual for SAD based on the Clark and
Wells (1995) model.
Individual CBT is considered the most efficacious treatment of SAD and is
recommended as the treatment of choice in NICE guidelines.
Refinements of the model and treatment may be possible, as shown by recent
research on metacognitive factors.
RECOMMENDED READING
Clark, D. M., & Wells, A. (1995). A cognitive model of social phobia. In R. G. Heimberg, M.
R. Liebowitz, D. A. Hope, & F. R. Schneier (Eds.), Social phobia: Diagnosis, assessment,
and treatment (pp. 69–93). Guilford Press.
Mayo-Wilson, E., Dias, S., Mavranezouli, I., Kew, K., Clark, D. M., Ades, A. E., & Pilling, S.
(2014). Psychological and pharmacological interventions for social anxiety disorder in
adults: A systematic review and network meta-analysis. The Lancet Psychiatry, 1,
368–376.
Wells, A. (1997). Cognitive therapy of anxiety disorders: A practice manual and conceptual
guide. John Wiley & Sons.
(2007). Cognition about cognition: Metacognitive therapy and change in generalized anxiety disorder and social phobia. Cognitive and Behavioral Practice, 14, 18–25.
(2009). Metacognitive therapy for anxiety and depression. Guilford Press.
REFERENCES
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Baker, S. L., Heinrichs, N., Kim, H. J., & Hofmann, S. G. (2002). The Liebowitz social anxiety
scale as a self-report instrument: A preliminary psychometric analysis. Behaviour
Research and Therapy, 40, 701–715.
Beck, A. T. (1976). Cognitive therapy and the emotional disorders. International Universities
Press.
Borge, F. M., Hoffart, A., Sexton, H., Clark, D. M., Markowitz, J. C., & McManus, F. (2008).
Residential cognitive therapy versus residential interpersonal therapy for social phobia:
A randomized clinical trial. Journal of Anxiety Disorders, 22, 991–1010.
Brown, T. A., & Barlow, D. H. (2014). Anxiety and Related Disorders Interview Schedule for
DSM-5 (ADIS-5L): Lifetime Version. Client interview schedule. Oxford University Press.
Brown, T. A., Di Nardo, P. A., & Barlow, D. H. (1994). Anxiety Disorders Interview Schedule
for DSM-IV (ADIS-IV). Psychological Corporation.
Carlbring, P., Andersson, G., Cuijpers, P., Riper, H., & Hedman-Lagerlöf, E. (2018).
Internet-based vs. face-to-face cognitive behavior therapy for psychiatric and somatic
disorders: An updated systematic review and meta-analysis. Cognitive Behaviour
Therapy, 47, 1–18.
Connor, K. M., Davidson, J. R., Churchill, L. E., Sherwood, A., Weisler, R. H., & Foa, E.
(2000). Psychometric properties of the Social Phobia Inventory (SPIN): New self-rating
scale. The British Journal of Psychiatry, 176, 379–386.
Clark, D. M., Butler, G., Fennell, M., Hackmann, A., McManus, F., & Wells, A. (1995). Social
behaviour questionnaire [Unpublished manuscript].
CBT for Social Anxiety Disorder
Clark, D. M., Ehlers, A., Hackmann, A., McManus, F., Fennell, M., Grey, N., . . . & Wild, J.
(2006). Cognitive therapy versus exposure and applied relaxation in social phobia:
A randomized controlled trial. Journal of Consulting and Clinical Psychology, 74,
568–578.
Clark, D. M., Ehlers, A., McManus, F., Hackmann, A., Fennell, M., Campbell, H., . . . &
Louis, B. (2003). Cognitive therapy versus fluoxetine in generalized social phobia:
A randomized placebo-controlled trial. Journal of Consulting and Clinical Psychology,
71, 1058–1067.
Clark, D. M., & Wells, A. (1995). A cognitive model of social phobia. In R. G. Heimberg,
M. R. Liebowitz, D. A. Hope, & F. Schneier (Eds.), Social phobia: Diagnosis, assessment,
and treatment (pp. 69–93). Guilford Press.
Connor, K., Davidson, J., Churchill, L., Sherwood, A., Weisler, R., & Foa, E. (2000).
Psychometric properties of the Social Phobia Inventory (SPIN): New self-rating
scale. British Journal of Psychiatry, 176(4), 379–386. doi:10.1192/bjp.176.4.379
Davidson, J. R., Miner, C. M., De Veaugh-Geiss, J., Tupler, L. A., Colket, J. T., & Potts,
N. L. S. (1997). The brief social phobia scale: A psychometric evaluation. Psychological
Medicine, 27, 161–166.
Fenigstein, A., Scheier, M. F., & Buss, A. H. (1975). Public and private self-consciousness:
Assessment and theory, Journal of Consulting and Clinical Psychology, 43(4), 522–527.
First, M. B., Spitzer, R. L., Gibbon, M., & Williams, J. B. (1996). User’s guide for the
Structured Clinical Interview for DSM-IV axis I disorders. SCID-I: Clinician version.
American Psychiatric Publishing.
First, M. B., Williams, J. B. W., Karg, R. S., & Spitzer, R. L. (2015). Structured clinical
interview for DSM-5: Research version (SCID-5 for DSM-5, research version; SCID-5RV). American Psychiatric Association.
Gkika, S., Wittkowski, A., & Wells, A. (2017). Social cognition and metacognition in social
anxiety: A systematic review. Clinical Psychology & Psychotherapy, 25, 10–30.
Gregory, B., & Peters, L. (2017). Changes in the self during cognitive behavioural therapy for
social anxiety disorder: A systematic review. Clinical Psychology Review, 52, 1–18.
Hinrichsen, H., & Clark, D. M. (2003). Anticipatory processing in social anxiety: Two pilot
studies. Journal of Behavior Therapy and Experimental Psychiatry, 34, 205–218.
Ingul, J. M., Aune, T., & Nordahl, H. M. (2014). A randomized controlled trial of individual
cognitive therapy, group cognitive behaviour therapy and attentional placebo for adolescent social phobia. Psychotherapy and Psychosomatics, 83, 54–61.
Leichsenring, F., Salzer, S., Beutel, M. E., Herpertz, S., Hiller, W., Hoyer, J., . . . & Ritter, V.
(2013). Psychodynamic therapy and cognitive-behavioral therapy in social anxiety disorder: A multicenter randomized controlled trial. American Journal of Psychiatry, 170,
759–767.
Leigh, E., & Clark, D. M. (2016). Cognitive therapy for social anxiety disorder in adolescents:
A development case series. Behavioural and Cognitive Psychotherapy, 44, 1–17.
(2018). Understanding social anxiety disorder in adolescents and improving treatment
outcomes: Applying the cognitive model of Clark and Wells (1995). Clinical Child and
Family Psychology Review, 21, 388–414.
Liebowitz, M. R. (1987). Social phobia. In D. F. Klein (Ed.), Anxiety. Karger, 141–173.
Mattick, R. P., & Clarke, J. C. (1998). Development and validation of measures of social
phobia scrutiny fear and social interaction anxiety. Behaviour Research and Therapy, 36,
455–470.
Mayo-Wilson, E., Dias, S., Mavranezouli, I., Kew, K., Clark, D. M., Ades, A. E., & Pilling, S.
(2014). Psychological and pharmacological interventions for social anxiety disorder in
adults: A systematic review and network meta-analysis. The Lancet Psychiatry, 1,
368–376.
133
134
Henrik Nordahl and Adrian Wells
Mörtberg, E., Clark, D. M., Sundin, Ö., & Åberg Wistedt, A. (2007). Intensive group
cognitive treatment and individual cognitive therapy vs. treatment as usual in social
phobia: A randomized controlled trial. Acta Psychiatrica Scandinavica, 115, 142–154.
National Institute for Health and Care Excellence. (2013). Social anxiety disorder: recognition, assessment and treatment of social anxiety disorder (Clinical guideline 159). http://
guidance.nice.org.uk/CG159
Ng, A. S., Abbott, M. J., & Hunt, C. (2014). The effect of self-imagery on symptoms
and processes in social anxiety: A systematic review. Clinical Psychology Review, 34,
620–633.
Nordahl, H., Nordahl, H. M., Hjemdal, O., & Wells, A. (2017). Cognitive and metacognitive
predictors of symptom improvement following treatment of social anxiety disorder:
A secondary analysis from a randomized controlled trial. Clinical Psychology and
Psychotherapy, 24(6), 1221–1227.
Nordahl, H., Nordahl, H. M., Vogel, P. A., & Wells, A. (2018). Explaining depression
symptoms in patients with social anxiety disorder: Do maladaptive metacognitive beliefs
play a role? Clinical Psychology and Psychotherapy, 25, 457–464.
Nordahl, H. M., Vogel, P. A., Morken, G., Stiles, T. C., Sandvik, P., & Wells, A. (2016).
Paroxetine, cognitive therapy or their combination in the treatment of social anxiety
disorder with and without avoidant personality disorder: A randomized clinical trial.
Psychotherapy and Psychosomatics, 85, 346–356.
Nordahl, H. M., & Wells, A. (2017). Social anxiety and work status: The role of negative
metacognitive beliefs, symptom severity and cognitive-behavioural factors. Journal of
Mental Health, 29(6), 665–669.
(2018). Metacognitive therapy for social anxiety disorder: An A-B replication series across
social anxiety subtypes. Frontiers in Psychology, 9, 540.
Piccirillo, M. L., Dryman, M. T., & Heimberg, R. G. (2015). Safety behaviors in adults with
social anxiety: Review and future directions. Behavior Therapy, 47, 675–687.
Rachman, S., Grüter-Andrew, J., & Shafran, R. (2000). Post-event processing in social
anxiety. Behaviour Research and Therapy, 38, 611–617.
Ruscio, A. M., Brown, T. A., Chiu, W. T., Sareen, J., Stein, M. B., & Kessler, R. C. (2008).
Social fears and social phobia in the USA: Results from the National Comorbidity
Survey Replication. Psychological Medicine, 38, 15–28.
Scheier, M. F., & Carver, C. S. (1985). The Self-Consciousness Scale: A revised version for
use with general populations. Journal of Applied Social Psychology, 15, 687–699.
Stangier, U., Heidenreich, T., Peitz, M., Lauterbach, W., & Clark, D. M. (2003). Cognitive
therapy for social phobia: Individual versus group treatment. Behaviour Research and
Therapy, 41, 991–1007.
Stangier, U., Schramm, E., Heidenreich, T., Berger, M., & Clark, D. M. (2011). Cognitive
therapy vs interpersonal psychotherapy in social anxiety disorder: A randomized controlled trial. Archives of General Psychiatry, 68, 692–700.
Turner, S. M., Beidel, D. C., Dancu, C. V., & Stanley, M. A. (1989). An empirically derived
inventory to measure social fears and anxiety: The Social Phobia and Anxiety Inventory.
Psychological Assessment: A Journal of Consulting and Clinical Psychology, 1, 35–40.
Vogel, P. A., Hagen, R., Hjemdal, O., Solem, S., Smeby, M. C. B., Strand, E. R., Fisher, P.,
Nordahl, H. M., & Wells, A. (2016). Metacognitive therapy applications in social phobia:
An exploratory study of the individual and combined effects of the attention training
technique and situational attentional refocusing. Journal of Experimental
Psychopathology, 7, 608–618.
Wampold, B. E., Flückiger, C., Del Re, A. C., Yulish, N. E., Frost, N. D., Pace, B. T., . . . &
Hilsenroth, M. J. (2017). In pursuit of truth: A critical examination of meta-analyses of
cognitive behavior therapy. Psychotherapy Research, 27, 14–32.
CBT for Social Anxiety Disorder
Warnock-Parkes, E., Wild, J., Stott, R., Grey, N., Ehlers, A., & Clark, D. M. (2017). Seeing is
believing: Using video feedback in cognitive therapy for social anxiety disorder.
Cognitive and Behavioral Practice, 24, 245–255.
Watson, D., & Friend, R. (1969). Measurement of social-evaluative anxiety. Journal of
Consulting and Clinical Psychology, 33, 448–457.
Wells, A. (1990). Panic disorder in association with relaxation induced anxiety: An attentional
training approach to treatment. Behavior Therapy, 21, 273–280.
(1997). Cognitive therapy of anxiety disorders: A practice manual and conceptual guide.
John Wiley & Sons.
(2009). Metacognitive therapy for anxiety and depression. Guilford Press.
Wells, A., Clark, D. M., & Ahmad, S. (1998). How do I look with my mind’s eye?: Perspective
taking in social phobic imagery. Behaviour Research and Therapy, 36, 631–634.
Wells, A., & Matthews, G. (1994). Attention and emotion: A clinical perspective. Erlbaum.
Wells, A., & Papageorgiou, C. (1998). Social phobia: Effect of external attention on anxiety,
negative beliefs and perspective taking. Behavior Therapy, 29, 357–370.
(2001). Brief cognitive therapy for social phobia: A case series. Behaviour Research and
Therapy, 39, 713–720.
Wells, A., Stopa, L., & Clark, D. M. (1993). Social cognition questionnaire [Unpublished
manuscript]. Department of Psychiatry, University of Oxford, Warnerford Hospital.
Wittchen, H. U., & Fehm, L. (2003). Epidemiology and natural course of social fears and
social phobia. Acta Psychiatrica Scandinavica, 108, 4–18.
Wong, Q. J., Gregory, B., & McLellan, L. F. (2016). A review of scales to measure social
anxiety disorder in clinical and epidemiological studies. Current Psychiatry Reports, 18,
38–53.
Wong, Q. J., & Moulds, M. L. (2009). Impact of rumination versus distraction on anxiety and
maladaptive self-beliefs in socially anxious individuals. Behaviour Research and Therapy,
47, 861–867.
Wong, Q. J., Moulds, M. L., & Rapee, R. M. (2014). Validation of the self-beliefs related to
social anxiety scale: A replication and extension. Assessment, 21, 300–311.
135
8
CBT for Somatic Symptom Disorder and Illness
Anxiety Disorder
Lillian Reuman and Jonathan S. Abramowitz
Introduction
According to the Diagnostic and Statistical Manual of Mental Disorders (5th ed.;
American Psychiatric Association, 2013), the essential feature of somatic symptom
disorder (SSD) and illness anxiety disorder (IAD) is a preoccupation with one or
more physical symptoms that are attributed to a non-psychiatric disease. This
preoccupation is often distressing and results in significant disruptions of daily life.
For example, SSD or IAD may be characterized by worry about stomach pains that
are not consistent with a medical diagnosis or by the persistent fear that frequent
“chest tightening” is indicative of a heart attack despite no evidence of cardiac
disease. This preoccupation is accompanied by inaccurate and persistent thoughts
about the seriousness of the perceived symptoms, a high level of anxiety related to
one’s health, and excessive time and energy devoted to thinking about or responding to the perceived symptoms and health concerns. Accordingly, an individual
preoccupied with concerns about stomach pains or chest tightening may devote
hours to researching medical treatments online, spend excessive amounts of money
pursuing unnecessary medical tests and procedures, and enlist family members and
doctors to provide reassurance regarding worrisome symptoms.
In addition to maladaptive cognitions and unhelpful accompanying behaviors,
bodily sensations play a large role in the development and maintenance of SSD. For
example, physical discomfort or shifts in body sensations (internal triggers) are
often interpreted as signs of serious disease (e.g., the sensations that accompany a
headache indicate a brain tumor). Individuals may also experience anxiety as a
result of external triggers (e.g., reading about an illness) that cue thoughts about
one’s own risk of illness and selective attention to benign (yet perceptible) bodily
perturbations. The emotional response of anxiety further evokes (or exacerbates)
bodily sensations (e.g., racing heart) that may be misinterpreted as signs of serious
illness. This perception of intensifying bodily sensations can then lead to a vicious
cycle of selective attention toward the perceived threat, misinterpretation of the
nature and severity of bodily sensations, and maladaptive coping.
136
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
Somatic sensations may result from normal “bodily noise,” the experience of
general stress (including stress related to worrying about one’s health), and a
heightened awareness of the body. Given the conceptualization of SSD and IAD
as conditions having to do with maladaptive thinking patterns and accompanying
repetitive behaviors, the literature on the treatment of SSD and IAD supports the
use of cognitive behavioral therapy (CBT) for reducing symptomatology
and improving physical functioning and quality of life. In this chapter we
summarize the research base for CBT for somatic concerns and provide a clinical
guide to formulating, assessing, and implementing CBT for clients with
such concerns.
Clinical Case Formulation, Assessment, and Treatment
Case Formulation and Treatment Implications
The cognitive behavioral formulation of SSD/IAD explains why health-related
anxiety persists despite no good evidence of ill health. This conceptualization
includes environmental and learning factors, as well as a range of cognitive and
behavioral mechanisms (Abramowitz & Braddock, 2008). From this perspective,
dysfunctional beliefs about bodily sensations and illnesses (e.g., “My body is weak,
and I am vulnerable to illness”) and the inability to tolerate uncertainty and anxiety
(e.g., “Not having a guarantee about my health is unbearable”) increase the likelihood of catastrophic misinterpretations of benign bodily sensations and perturbations (i.e., normal “body noise”), as well as ambiguous health-related information,
as indicators of illness. Such misinterpretations trigger anxiety, an increased sense of
uncertainty, and increased vigilance for signs of illness, as well as the urge to gain
assurances about health to reduce the subjective discomfort.
The catastrophic misinterpretations are then maintained (despite contradictory
information and repeated reassurance of good health from medical professionals)
by maladaptive strategies used to cope with illness-related anxiety. These strategies
include exaggerated attempts to prevent the feared illness or its consequences (e.g.,
staying near a hospital), avoidance (e.g., of triggers or illness cues), and attempts to
attain certainty about health status (e.g., checking medical resources, body
checking). Such safety behaviors prevent individuals with SSD/IAD from disconfirming their mistaken beliefs about being ill and learning that they can in fact
manage uncertainty and anxiety.
This approach to understanding SSD/IAD, which is partially derived from
conceptualizations of other anxiety-related problems, provides the foundation for
cognitive behavioral interventions discussed later in this chapter, which generally
have the aim of helping the individual develop healthier responses to the ubiquitous
internal and environmental cues, as well as uncertainty, related to one’s health. One
such intervention is psychoeducation, which involves providing the individual with
information about the nature of their presenting concerns and potential strategies
for addressing them. Exposure and response prevention (ERP) is a set of procedures
137
138
Lillian Reuman and Jonathan S. Abramowitz
used to help health anxious individuals confront fear triggers (external and internal)
to learn that they can manage such cues, as well as the anxiety and uncertainty they
provoke, without the use of safety behaviors or other strategies for trying to gain
reassurance. In some instances, cognitive restructuring might be used to identify and
challenge inaccurate beliefs about the meaning of bodily sensations.
Assessment
The cognitive behavioral model also informs what is necessary to assess when
working with clients affected by SSD/IAD. We recommend using the following
instruments and approaches to gain an understanding of SSD/IAD severity and
related behavioral and cognitive features that will help inform a treatment plan and
assess treatment response.
The Yale-Brown Obsessive Compulsive Scale
The Yale–Brown Obsessive Compulsive Scale (Y-BOCS; Goodman et al., 1989) is a
semi-structured interview developed to assess obsessive-compulsive symptoms and
also serves as a measure of illness anxiety severity. It includes 10 items (healthrelated thinking, items 1–5; compulsions, items 6–10) that assess the following five
parameters: (a) time, (b) interference, (c) distress, (d) efforts to resist, and (e)
perceived control. Each item is rated on a 0–4 scale, and the item scores are summed
to produce a total score ranging from 0 (no symptoms) to 40 (extreme). Behaviors
such as checking, reassurance seeking, or consulting with doctors are scored on the
compulsions (safety behaviors) subscale. Scores of 0–7 represent subclinical symptoms, 8–15 represent mild symptoms, 16–23 relate to moderate symptoms, 24–31
suggest severe symptoms, and scores of 32 or greater imply extreme symptoms.
The Short Health Anxiety Inventory
The Short Health Anxiety Inventory (SHAI; Salkovskis et al., 2002) is an 18-item
instrument that assesses health anxiety independent of physical health status. For
each item the respondent chooses from a series of four statements (ranging in
severity from 0 to 3) that best reflects their feelings over the past several months.
Items address worry about health, awareness of bodily sensations and changes, and
feared consequences of having an illness.
The Cognitions about Body and Health Questionnaire
The Cognitions about Body and Health Questionnaire (CBHQ; Rief et al., 1998)
is a 31-item self-report scale that assesses various domains of exaggerated and
dysfunctional cognitions and assumptions about illness, bodily sensations, and
health habits. The respondent rates each item (e.g., “My body can tolerate a lot of
strain”) on a four-point Likert scale ranging from 0 (completely wrong) to 3 (completely right).
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
Functional Assessment of Illness-Related Anxiety
We also suggest collecting detailed, patient-specific information about internal and
external health anxiety cues and behavioral responses to these cues. As indicated by
the conceptualization described previously, the key domains to assess include
the following.
Bodily Signs, Sensations, and Perturbations. Signs and sensations might be
internal but can also be associated with the skin, hair, or products that are expelled
from the body (e.g., “Which bodily signs and sensations are you concerned with?”).
External Situations and Stimuli. Such triggers often include hospitals, doctors,
seeing or hearing about sick people, reading about illnesses, and other stimuli that
serve as reminders of health concerns. Being alone or “too far” from a hospital or
medical center can also be situational triggers, as can seeing oneself naked or giving
oneself an examination (e.g., breast or testicular) (e.g., “What situations trigger your
health-related thoughts and concerns?” “What situations do you avoid because they
might trigger your health concerns?”).
Misinterpretations of Bodily Signs and Sensations. Beliefs concerning the presence and meaning of unexpected, unwanted, or disconcerting bodily sensations are
common. For example, patients may believe “When my head hurts, I think I’m
getting a brain tumor” or “When I feel a lump in my throat, it reminds me that
I have a serious neck problem that no doctors have been able to figure out.”
Therapists should query, “What do you think this sensation means?” and “What
do you worry is causing you to have this feeling?”
Feared Consequences. Beliefs about the probability and severity of becoming ill
or dying must be assessed. Some individuals have fears of explicit disastrous consequences, whereas for others, the internal and external cues evoke only a vague sense
that “something is wrong.” Others fear they cannot tolerate uncertainty associated
with their health (i.e., not having a 100% guarantee that they are not ill).
Avoidance. Avoidance patterns (usually related to external triggers) might
include staying away from hospitals, people with certain illnesses or diseases,
funerals, or cemeteries, or avoiding disease-related movies, TV shows, news articles,
or other information in the media. Such avoidance can often be predicted from
external triggers and feared consequences. Therapists can ask, “What kinds of
things do you not do because of your health concerns?” and “What situations do
you routinely avoid?”
Checking, Reassurance Seeking, and Other Safety Behaviors and Safety Signals
It is important to assess body checking, which might include examination of the
body’s vital signs (using medical equipment) or the structure and function of a
particular body part (e.g., probing bodily orifices, smelling certain body parts or
bodily excretions). Individuals might also perform other forms of checking and
reassurance seeking, such as scheduling extraneous doctor visits, repetitively
reviewing medical test results and reports, and searching the internet for information about diseases of concern. Furthermore, therapists ought to inquire about
139
140
Lillian Reuman and Jonathan S. Abramowitz
other strategies the person engages in to reduce health-related anxiety or fear, for
example, the use of a cane to avoid putting “too much stress” on one’s “rotting” legs
or being sure to get exactly eight hours of sleep. Finally, safety signals are situations
or objects that are associated with safety, such as cell phones, medications, and
medical alert bracelets. Many individuals do not recognize these behaviors, situations, or objects as part of their problems with SSD or IAD. Questions to help
elucidate safety behaviors and signals are: “Are there things you do to protect
yourself from illnesses?” and “What precautions do you take so that you are
prepared if you should have a medical emergency?”
Illustration of Assessment and Case Formulation
Theo, a 37-year-old accountant, noticed a small discoloration of the skin on his left
shoulder. At his routine biyearly physical exam, he asked his physician to evaluate
the skin mark for possible skin cancer. The doctor measured its diameter and told
Theo she believed the mark was a solar lentigo, a benign discoloration of the skin
caused by sun exposure. The doctor suggested Theo wear sunscreen with zinc oxide
to help prevent additional sun damage and felt there was no need at that time to
biopsy or remove the lentigo. Theo was initially relieved by this diagnosis, yet a few
months later, he began thinking about it again when a family friend was diagnosed
with skin cancer. Theo examined the skin mark in the mirror, and for a few weeks,
took pictures of it and compared them to see whether the lentigo was growing.
Although there did not seem to be any worsening, Theo became preoccupied with
checking the lentigo every day. He rubbed and picked at it to see if it was raised and
noticed that it seemed to be looking redder and “angrier.” He had the sensation that
it was itchy and sore, and he found himself searching other parts of his body for
more signs of skin cancer, from which Theo now believed he was suffering.
Theo’s next step was to be evaluated by a dermatologist, who provided the
same diagnosis of benign lentigo and suggested that no follow-up was necessary.
The irritation, she suggested, was due to Theo’s persistent rubbing and picking of
the area. But Theo continued to press the dermatologist for a biopsy “just to be
sure.” After his pleas were denied several times, Theo took to searching the internet
for information about skin cancer. Every day, he examined images of benign and
malignant discolorations and growths. He spoke with his family and friends about
their knowledge of skin cancer and frequently asked for reassurance that the
problem was benign, never believing the feedback he received. He was losing sleep,
his appetite was reduced, and he was spending more time reading about skin cancer
than with his wife and two children. At that point, Theo’s wife encouraged Theo
to talk with a mental health professional, which is how he ended up in our
treatment clinic.
Theo’s therapist used the cognitive-behavioral framework (1) to generate a
formulation of Theo’s illness anxiety complaints and (2) to assess his concerns to
further refine the formulation and develop a treatment plan. Numerous triggers and
cues to Theo’s health concerns were present in his environment, including looking
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
at himself in the mirror, seeing spots on his skin, talking about cancer, and relating
to his family friend with cancer. Theo’s belief that he had undiagnosed skin cancer
was thought to lead to anxiety, which maintained his excessive health concerns by
causing him to feel stressed and fatigued. Theo’s safety behaviors were conceptualized as natural responses to anxiety aimed at reducing distress and leading to some
degree of certainty about the nature of his “symptoms.” These behaviors often
resulted in temporary or short-lived reductions in anxiety. However, they ultimately
maintained Theo’s health anxiety. For example, excessive body-checking led Theo
to become sensitive to any changes on his skin; picking and rubbing led to irritation
of the lentigo, which seemed to confirm (to Theo) that it was cancerous; and
reassurance seeking always left some degree of ambiguity about the actual nature
of the skin mark, thus sustaining the belief that he had been misdiagnosed.
Theo’s score on the Y-BOCS was 28, indicating somewhat severe illness anxiety. His responses on the CBHQ indicated all-or-nothing beliefs about health and
illness, such as the belief that good health means having no symptoms whatsoever.
Finally, functional assessment led to identifying a list of illness anxiety triggers and
additional safety behaviors that would need to be addressed in treatment.
Cognitive Behavioral Therapy
CBT for somatic-related anxiety is an active intervention that includes components
of psychoeducation, cognitive therapy, self-monitoring, exposure and response
prevention, and relapse prevention (e.g., Abramowitz & Braddock, 2008; Barlow,
2014). Numerous studies suggest that multicomponent CBT treatment packages
involving between 12 and 20 treatment sessions are efficacious for somatic
symptoms; however, not all patients respond equally, and some do not achieve
improvement at all. The primary treatment components are described in the
following sections.
Psychoeducation
Psychoeducation involves providing information about bodily symptoms, the development and maintenance of SSD/IAD, and a rationale for using CBT. Therapists
can offer statistics about the prevalence of health anxiety (up to 25% of patients in
some medical clinics; Tyrer et al., 2011), as this can help clients to recognize that
they are not alone in their suffering. Clinicians should also share that symptoms of
SSD exist along a continuum of severity ranging from mild to severe distress and
impairment. This can help clients recognize that a mild degree of illness-related
anxiety is normal and adaptive but that increased levels of concern are maladaptive
and impairing. Within this conversation, the client and therapist can discuss the
ways in which mild to moderate anxiety may be beneficial (i.e., anxiety may
motivate an individual to act and alert us to danger), whereas severe levels of
anxiety can be costly and debilitating.
141
142
Lillian Reuman and Jonathan S. Abramowitz
Importantly, clinicians should educate clients about the aforementioned model
of the maintenance of illness worry. Important points to convey include that dysfunctional beliefs about bodily symptoms and illness increase anxiety and worry.
Once anxious about the possibility of acquiring an illness, the individual exhibits
heightened awareness for any signs of illness and seeks to reduce their worry by
gaining certainty about their health. Dysfunctional beliefs are maintained by maladaptive strategies used to cope with health-related anxiety, as we have discussed.
Therapists, for example, can explain how avoidance of anxiety-provoking situations
prevents the disconfirmation of feared consequences and how seeking reassurance
from health-related websites strengthens fears about the situation. This information
is best delivered in a collaborative, interactive approach, and the clinician should
also check with the client to gauge their understanding of the model by asking
questions such as, “Can you describe to me how this model applies to your
situation?”
Once clients have a grasp of the processes implicated in the development and
maintenance of SSD, they should receive the rationale for CBT, which aims to
modify unhelpful behavioral and thinking patterns that maintain SSD. Cognitive
restructuring techniques are used to address catastrophic thinking and beliefs about
the need for certainty. Behavioral methods include refraining from engaging in
actions that provide temporary reassurance (e.g., seeking medical advice, browsing
the internet) while engaging in activities that facilitate engagement with daily living
(e.g., attending school/work, engaging in pleasurable activities).
Psychoeducation is different from providing reassurance in that the individual is
presented with new information, rather than the repeated presentation of old information (e.g., repeating assurances of good health, unnecessary medical tests to
placate concerns). Therapists should also ask clients to identify the ways in which
moderate levels of health-related anxiety can be normal, safe, and adaptive. By
understanding that anxious responses are common, not dangerous, and adaptive,
individuals may be more likely to proceed with treatment.
Self-Monitoring
Self-monitoring involves recording instances of body-related sensations, any external triggers, and accompanying emotions, cognitions, and behaviors as they occur in
real time between therapy sessions. Self-monitoring not only helps increase awareness of such episodes and their antecedents and sequalae, but also aids in identifying
treatment targets and detecting patterns between sensations, cognitions,
and emotions.
Cognitive Restructuring
Cognitive restructuring involves the systematic use of Socratic questioning to help
patients identify and challenge mistaken thoughts, beliefs, and assumptions about
health, illnesses, and their external and internal cues (e.g., “Anxiety will harm me”;
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
“Doctors can’t be trusted”), to arrive at more useful, healthy, and empirically or
logically consistent beliefs and assumptions (e.g., “I have been anxious many times
in the past and it has never harmed me”; “Doctors are very knowledgeable, and
I can generally trust them”).
Exposure Therapy
Aims of exposure therapy for SSD/IAD are to help the individual (1) modify
exaggerated estimates of probability and severity of feared outcomes and (2) learn
that anxiety and uncertainty are tolerable and do not have to interfere with functioning. Exposure is conducted both during the treatment session and for homework. A repetition of exposure exercises in a variety of contexts (e.g., location, time
of day) is essential to promote generalization of learning (e.g., Craske et al., 2008).
Three types of exposure are discussed in turn.
Interoceptive Exposure
As bodily sensations are often cues for illness anxiety, interoceptive exposure (IE)
represents an important treatment strategy. IE involves deliberately and repeatedly
producing feared bodily sensations, focusing on the sensations without trying to
bring them to an end, and trying to maintain the sensations for a period of time
(Walker & Furer, 2008). Two main goals of IE for SSD/IAD are (1) to increase
acceptance of such sensations and (2) to decrease associated distress. Exercises such
as spinning, hyperventilation, and breathing through a straw are common fearinducing strategies; for a complete list of suggestions, see Huppert and BakerMorissette (2003).
Before beginning interoceptive exposure exercises, the clinician should conduct
a thorough assessment to determine which bodily sensations are most anxietyprovoking and which interoceptive activities bring about feared sensations. For
exposure to such bodily sensations, the clinician and client jointly and repeatedly
practice the activity in session. Interoceptive exposure might be produced by having
the patient hyperventilate, move vigorously (e.g., jumping jacks), or spin in an office
chair. The therapist should demonstrate the activity, intensify the activity as needed,
encourage the client to note their reactions, and debrief following the exposure. The
therapist can coach the client to “lean in” to the feared body sensations, and practice
observing them without trying to change them (Walker & Furer, 2008). The clinician should encourage repeated IE practice for extended periods of time between
sessions in order to promote generalization of learning that such sensations are safe
and tolerable.
Imaginal Exposure
Imaginal exposure for SSD/IAD usually entails writing scripts or creating images
(e.g., of one’s death) based on the client’s somatic concerns. Exposure to such
stories via writing, reading, and listening allows the client to experience feared
situations (e.g., getting a cancer diagnosis) that cannot be accessed through in vivo
143
144
Lillian Reuman and Jonathan S. Abramowitz
or interoceptive exposure. Imaginal exposure can also be used to amplify feelings of
uncertainty about feared illnesses to help the client learn that uncertainty
is manageable.
In practice, the therapist guides the client in creating stories or images that
are detailed and individually tailored to the client’s concerns. Next, the client
focuses on the written or visual material (i.e., reread the story for an extended
period of time) as a cue to vividly imagine the scenario and adds details as necessary
to intensify the experience. The client may wish to record the reading to listen to
the scenario repeatedly for homework practice. The therapist encourages the
client to note physical and emotional discomfort that arises as a result of the
imaginal exposure. To intensify the experience, imaginal exposure may be combined with IE.
In Vivo Exposure
In vivo exposure involves repeatedly and systematically facing a fear-provoking
situation until the individual learns that (1) the situation or cue is safe, (2) they can
tolerate the discomfort associated with the trigger, and (3) anxiety does not persist
indefinitely or spiral out of control. For example, individuals may read stories (or
obituaries) of individuals with chronic illness, watch medical documentaries that
depict medical suffering and disability, or visit sites of painful physical memories
(e.g., a chemotherapy center). The procedures for conducting exposure are
described in detail elsewhere (e.g., Abramowitz et al., 2012).
Response Prevention
Response prevention entails voluntarily refraining from reassurance seeking and
other anxiety-reducing (safety) behaviors both during and after exposure exercises.
This technique is used so the individual with SSD/IAD can learn that safety behaviors are not necessary for safety and for anxiety tolerance and that, moreover,
health anxiety subsides naturally, even when safety behaviors are not performed.
Specific response prevention guidelines for the patient to follow can be determined
collaboratively, and whereas some patients are able to immediately stop all
checking, reassurance seeking, and other safety behaviors, others require a gradual
approach. It is important to discuss the negative effects of checking, reassurance
seeking, and other safety behaviors as well as the rationale for stopping them when
implementing response prevention. Patients are often surprised at how much less
attention they pay to their bodies when they do not engage in these maladaptive
behaviors.
Therapists can emphasize that refraining from safety behaviors is a difficult
choice and can work with the client to define guidelines for response prevention. If
relatives or friends are involved in the patient’s symptoms, such individuals are also
encouraged to refrain from “helping” with the performance of safety behaviors. The
patient is to record violations of response prevention on a symptom record and
bring these to the therapist so that areas in need of additional attention can be
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
addressed. Violations of the response prevention plan can be self-monitored and
addressed. The following are some examples of response prevention plans and
strategies for various types of safety behaviors often observed in individuals with
SSD/IAD.
Body Checking
Patients are not to check bodily signs (e.g., pulse) or levels (blood sugar), or the
skin. Inspecting or scrutinizing body fluids and excreta (aside from a simple glance)
is also prohibited.
Reassurance Seeking
Medical appointments for previously evaluated symptoms are off-limits. Any “new
symptoms” can be discussed with the therapist, but no doctor visits are to occur.
Telephoning medical personnel or “help lines” is off-limits. Internet searches for
medical information are also prohibited. If the patient must be online for work or
school, they should avoid medical websites. It may be helpful to ask a partner or
relative to monitor internet use. Reviewing one’s own medical records is also
prohibited. Patients are also not to ask for reassurance about health matters from
friends and family members and are not to discuss symptoms with others (e.g., “I
have had this headache for two days now”).
We suggest helping the patient draft a letter to friends and relatives explaining
the detrimental effects of reassurance seeking and providing suggestions for how to
respond if asked for reassurance. For example, “I’m sorry but I can’t answer your
question because I agreed to help you with treatment.”
Mental Analyzing
Patients are not to mentally devote time to going over what doctors have told them
or trying to analyze situations related to their health. Instead, they should purposely
imagine being uncertain (as in exposure).
Dietary Restrictions
Patients are discouraged from taking dietary supplements, herbs, and other similar
agents if their use reduces health anxiety or unexplained body sensations. Similarly,
adhering to special diets because of health anxiety is prohibited. The therapist should
be alert for additional safety behaviors and signals that will require response
prevention rules.
Homework
Each stage of CBT for SSD includes homework components to practice skills and
reinforce session content. To emphasize the central role of homework, the therapist
should assign homework beginning with the first session. For example, homework
during the psychoeducation stage might include reading handouts about the
145
146
Lillian Reuman and Jonathan S. Abramowitz
patient’s particular problem and how the CBT approach addresses conceptualization and treatment. Homework during the self-monitoring phase is described above.
For exposure homework assignments, it is important to explain that such tasks
provide opportunities for the client to challenge their fear-provoking assumptions
and form new beliefs about situations that are realistically safe and tolerable (e.g.,
“Even if I have a stomach ache before flying, I can still travel”). In explaining outof-session exposure practice, the therapist can emphasize the importance of
repeated practice in various contexts to maximize treatment effects and enhance
generalizability. Additionally, conducting exposure outside the medical setting and
without the therapist present allows the patient to develop self-efficacy and learn
that they can approach these situations even if help is not immediately available or if
the bodily discomfort persists. More broadly, homework presents an opportunity to
practice coping with anxiety and physical discomfort.
It is important to consider that homework non-completion may occur for a
variety of reasons. Huppert and colleagues (2006) posit that patients may misunderstand or refuse to complete a homework assignment due to beliefs about the utility
of the assignment or low motivation to change. For example, patients with SSD may
believe that a particular response prevention assignment (e.g., not consulting a
doctor for a perceived uncomfortable body sensation) is harmful or irresponsible.
Patients with SSD may also experience pain and exhaustion, effectively impairing
their ability to complete their homework. In each situation, it is important to
understand the patients’ reasons for non- or partial homework completion and
integrate this information into the case formulation.
The therapist and patient should work jointly during homework review to
address and problem-solve any obstacles to homework compliance that arise. In
the case of a misunderstanding, the therapist might explain the assignment in
different terms, provide clear illustrations or examples, and/or check that the patient
understands homework expectations by asking the patient to teach the therapist
about the importance of homework. Cognitive therapy and motivational interviewing (e.g., identifying pros and cons) can be useful for addressing homework compliance barriers such as beliefs about safety (e.g., “conducting IE on my own is
dangerous”) or low motivation for change. In the context of emotional barriers
(e.g., fear, shame) to homework (non)compliance, psychoeducation about emotions
(e.g., normalizing fear) can be useful. Given logistical barriers (e.g., little time to
complete homework), the therapist and patient could work jointly to schedule a
specific time of day to complete homework practice or identify an individual that
can help the patient remain accountable.
Treatment Outcome
Abundant research supports the efficacy of CBT for SSD/IAD and their precursors
in earlier editions of the DSM (i.e., hypochondriasis), and findings are well summarized in a meta-analysis of this literature by Olatunji and colleagues (2014).
Considering 13 randomized controlled trials (RCTs) with a total sample size of
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
1,081 participants, Olatunji and colleagues (2014) found that CBT for hypochondriasis outperformed control conditions (e.g., treatment as usual, wait-list) on primary symptom outcome measures at post-treatment and showed a large effect size.
A follow-up analysis of seven studies revealed that CBT for patients with health
anxiety also outperformed control conditions on secondary outcomes (i.e., exhibited
significant reductions in symptoms of depression). This suggests that the acute
therapeutic effects of CBT for patients with hypochondriasis/health anxiety may
generalize to other symptoms.
The researchers examined the extent to which effect size varied as a function of
treatment factors (e.g., number of sessions) and participant characteristics (e.g.,
percentage of females, mean age). More sessions of CBT for hypochondriasis were
associated with larger effect sizes at post-treatment. Higher pre-treatment hypochondriasis/health anxiety was found to be associated with an increase in CBT effect
size at follow-up visits; however, participant demographics were not differentially
associated with effect sizes. Although these findings are promising, additional
research with longer follow-up intervals is necessary to offer more definitive conclusions regarding the strength of CBT for SSD/health anxiety.
Recent Advances
Although CBT for SSD/IAD is most commonly offered in an individual, in-person
format, a number of recent advances lend empirical support to varied styles of
treatment delivery such as group therapy, internet-based formats, and
mindfulness-based approaches.
Group Therapy
Group therapy has been shown to be effective for a variety of psychiatric conditions,
such as anxiety disorders (e.g., panic disorder) and hypochondriasis (Hedman et al.,
2010). In comparison to individual therapies, a group approach for SSD/IAD offers
support and advantages for participants in various ways. First, learning about fellow
group members’ experiences can help to normalize the individual’s concerns.
Second, working together in a group setting can provide a supportive, encouraging
environment that motivates individuals to persist with out-of-session practice. Third,
group members can collectively brainstorm and problem-solve treatment obstacles
by providing a range of perspectives and first-hand accounts. Further, given that
patients can be treated jointly, a group approach can facilitate scheduling by
maximizing therapist availability and decreasing costs for the group members
(Espejo et al., 2016). This is particularly important given that somaticizing patients
exhibit increased utilization of medical services and incur higher costs by virtue of
more specialist and emergency department visits as a function of perseverative
worry (Barsky et al., 2005).
In a recent open trial, Hedman and colleagues (2010) investigated the effectiveness and cost impact of a group CBT protocol in a psychiatric setting for individuals
with somatic concerns. Using a CBT model, the treatment emphasized challenging
147
148
Lillian Reuman and Jonathan S. Abramowitz
interpretations of bodily sensations as signs of serious illness and reducing avoidance and safety behaviors (Furer et al., 2007; Taylor & Asmundson, 2004). The
groups included four to seven patients, and treatment consisted of 10 weekly group
sessions. Each session lasted two hours, covered a particular theme (e.g., cognitive
restructuring, interoceptive exposure, response prevention), and followed a structured format (e.g., agenda-setting, homework review, and psychoeducation).
Results revealed large within-group effect sizes on self-report measures of health
anxiety. At post-treatment, there was a significant reduction in the number of
participants meeting diagnostic criteria for hypochondriasis following treatment.
Treatment gains were maintained at follow-up on all measures, and participants
continued to improve on measures of depression and anxiety sensitivity (i.e., the
“fear of fear”). Taken together, findings suggest that group CBT can be delivered in
a regular psychiatric healthcare context for individuals with SSD. The researchers
also concluded that CBT for health-related worry can be highly cost-effective, as the
intervention generated a total cost offset within one year, primarily because of a
decrease in direct medical costs.
Internet-Based Treatment
Hedman and colleagues (2011) also investigated an internet-based treatment platform for somatic concerns (i.e., DSM-IV hypochondriasis). The 12-week CBT-based
protocol, developed by their research team, included thematic modules (e.g., psychoeducation) and accompanying homework. Further, participants received online
access to fellow study participants for group discussion/support and to a therapist who
was available for online feedback and encouragement. Study findings lent support for
the feasibility of providing online educational modules and therapeutic support for
somatic concerns. The researchers noted that minimal therapist resources (approximately 10 minutes per patient per week) and the flexibility of internet-based CBT
with respect to delivery make internet therapy a promising means for improving the
availability of CBT for hypochondriasis. In particular, an online CBT protocol may
be well suited to individuals with severe somatization symptoms who are unable to
leave their home due to somatic complaints (e.g., pain) or time-consuming, debilitating worry and ritualistic behavior.
Mindfulness-Based Approaches
Mindfulness-based cognitive therapy (MBCT; Lovas & Barsky, 2010) represents
another recent advance in treatment for somatic concerns. An emphasis on nonjudgment and relaxation may be particularly useful for reducing physiological
arousal and interrupting cycles of unproductive worry that accompany somatic
complaints among individuals with illness anxiety. Lovas and Barsky (2010) conducted a small pilot study of an eight-week group MBCT intervention for severe
health anxiety. Educational in focus, the intervention was based on preexisting
models of mindfulness-based stress reduction (MBSR) and MBCT, by Kabat-Zinn
(1990) and Segal et al. (2002), respectively. Participants were taught mindfulness
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
meditation, including body scan, mindful breathing, and mindful movement practices. For homework, participants practiced the meditations for at least 30 minutes
daily. Following treatment, participants exhibited significant improvements in measures of health anxiety, disease-related thoughts, somatic symptoms, and mindfulness, which were sustained at three-month follow-up. These preliminary findings
simultaneously highlight the various cognitive behavioral options for treating
SSD, as well as the need for exploring novel treatment options for this ill
patient population.
Key Points
Assessment of SSD
Rule out organic medical conditions.
Assess external and internal triggers of SSD.
Assess beliefs about internal body sensations.
Assess safety behaviors associated with SSD.
Use the functional assessment and self-monitoring to develop an idiosyncratic
case conceptualization.
Treatment of SSD
Begin with psychoeducation about cognitive behavioral model and rationale
for cognitive behavioral therapy.
Incorporate cognitive restructuring to challenge beliefs about bodily sensations.
Use a mix of interoceptive, in vivo, and imaginal exposure.
Eliminate all safety behaviors.
FURTHER RECOMMENDED READING
Abramowitz, J. S., & Braddock, A. E. (2008). Psychological treatment of health anxiety and
hypochondriasis. Hogrefe.
Deacon, B., & Abramowitz, J. S. (2008). Is hypochondriasis related to obsessive-compulsive
disorder, panic disorder, or both? An empirical evaluation. Journal of Cognitive
Psychotherapy, 22(2), 115–127.
Furer, P., Walker, J. R., & Stein, M. B. (2007). Treating health anxiety and fear of death:
A practitioner’s guide. Springer.
Olatunji, B. O., Kauffman, B. Y., Meltzer, S., Davis, M. L., Smits, J. A., & Powers, M. B.
(2014). Cognitive-behavioral therapy for hypochondriasis/health anxiety: A metaanalysis of treatment outcome and moderators. Behaviour Research and Therapy, 58,
65–74.
Reuman, L., & Abramowitz, J. S. (2015). Illness anxiety disorder. In K. A. Phillips & D. J.
Stein (Eds.), Handbook on obsessive-compulsive and related disorders (pp. 225–246).
American Psychiatric Publishing.
Rief, W., Hiller, W., & Margraf, J. (1998). Cognitive aspects of hypochondriasis and the
somatization syndrome. Journal of Abnormal Psychology, 107(4), 587.
149
150
Lillian Reuman and Jonathan S. Abramowitz
Salkovskis, P. M., Rimes, K. A., Warwick, H. M. C., & Clark, D. M. (2002). The Health
Anxiety Inventory: Development and validation of scales for the measurement of health
anxiety and hypochondriasis. Psychological Medicine, 32(5), 843–853.
Taylor, S., & Asmundson, G. J. (2004). Treating health anxiety: A cognitive-behavioral
approach. Guilford Press.
REFERENCES
Abramowitz, J. S., & Braddock, A. E. (2008). Psychological treatment of health anxiety and
hypochondriasis. Hogrefe.
Abramowitz, J. S., Deacon, B. J., & Valentiner, D. P. (2007). The Short Health Anxiety
Inventory: Psychometric properties and construct validity in a non-clinical sample.
Cognitive Therapy and Research, 31(6), 871–883.
Abramowitz, J. S., Deacon, B. J., & Whiteside, S. P. (2012). Exposure therapy for anxiety:
Principles and practice. Guilford Press.
American Psychiatric Association. (2013). Diagnostic and statistical manual of Mental
Disorders (5th ed.). American Psychiatric Association.
Barlow, D. H. (Ed.) (2014). Clinical handbook of psychological disorders: A step-by-step
treatment manual. Guilford Press.
Barsky, A. J., Orav, E. J., & Bates, D. W. (2005). Somatization increases medical utilization
and costs independent of psychiatric and medical comorbidity. Archives of General
Psychiatry, 62(8), 903–910.
Craske, M. G., Kircanski, K., Zelikowsky, M., Mystkowski, J., Chowdhury, N., & Baker, A.
(2008). Optimizing inhibitory learning during exposure therapy. Behaviour Research and
Therapy, 46(1), 5–27.
Espejo, E. P., Castriotta, N., Bessonov, D., Kawamura, M., Werdowatz, E. A., & Ayers, C. R.
(2016). A pilot study of transdiagnostic group cognitive-behavioral therapy for anxiety in a
veteran sample. Psychological Services, 13(2), 162.
Furer, P., Walker, J. R., & Stein, M. B. (2007). Treating health anxiety and fear of death:
A practitioner’s guide. Springer Science & Business Media.
Goodman, W. K., Price, L. H., Rasmussen, S. A., Mazure, C., Fleischmann, R. L., Hill, C.
L., . . . & Charney, D. S. (1989). Yale–Brown Obsessive Compulsive Scale (YBOCS). Archives of General Psychiatry, 46, 1006–1011.
Hedman, E., Andersson, G., Andersson, E., Ljótsson, B., Rück, C., Asmundson, G. J., &
Lindefors, N. (2011). Internet-based cognitive-behavioural therapy for severe health anxiety: Randomised controlled trial. The British Journal of Psychiatry, 198(3), 230–236.
Hedman, E., Ljótsson, B., Andersson, E., Rück, C., Andersson, G., & Lindefors, N. (2010).
Effectiveness and cost offset analysis of group CBT for hypochondriasis delivered in a
psychiatric setting: An open trial. Cognitive Behaviour Therapy, 39(4), 239–250.
Huppert, J. D., & Baker-Morissette, S. L. (2003). Beyond the manual: The insider’s guide to
panic control treatment. Cognitive and Behavioral Practice, 10(1), 2–13.
Huppert, J. D., Roth Ledley, D., & Foa, E. B. (2006). The use of homework in behavior
therapy for anxiety disorders. Journal of Psychotherapy Integration, 16(2), 128–139.
Kabat-Zinn, J. (1990). Full catastrophe living: Using the wisdom of your body and mind to face
stress, pain, and illness. Dell Publishing.
Lovas, D. A., & Barsky, A. J. (2010). Mindfulness-based cognitive therapy for hypochondriasis, or severe health anxiety: A pilot study. Journal of Anxiety Disorders, 24(8),
931–935.
Salkovskis, P. M., Rimes, K. A., Warwick, H. M. C., & Clark, D. M. (2002). The Health
Anxiety Inventory: Development and validation of scales for the measurement of health
anxiety and hypochondriasis. Psychological Medicine, 32(5), 843–853.
CBT for Somatic Symptom Disorder and Illness Anxiety Disorder
Segal, Z. V., Teasdale, J. D., Williams, J. M., & Gemar, M. C. (2002). The mindfulness-based
cognitive therapy adherence scale: Inter-rater reliability, adherence to protocol and treatment distinctiveness. Clinical Psychology and Psychotherapy, 9(2), 131–138.
Taylor, S., & Asmundson, G. J. (2004). Treating health anxiety: A cognitive-behavioral
approach. Guilford Press.
Tyrer, P., Cooper, S., Crawford, M., Dupont, S., Green, J., Murphy, D., et al. (2011).
Prevalence of health anxiety problems in medical clinics. Journal of Psychosomatic
Research, 71, 392–394.
Walker, J. R., & Furer, P. (2008). Interoceptive exposure in the treatment of health anxiety
and hypochondriasis. Journal of Cognitive Psychotherapy, 22(4), 366–378.
151
9
Cognitive Behavioral Therapy for Generalized
Anxiety Disorder: Targeting Intolerance
of Uncertainty
Cara R. Dunkley and Melisa Robichaud
Despite being a relatively common disorder, generalized anxiety disorder (GAD) is
known as one of the more challenging mental health conditions to accurately
diagnose and treat. The diagnostic criteria for GAD in the current edition of the
Diagnostic and Statistical Manual of Mental Disorders (DSM-5) include excessive
and uncontrollable worry about a number of daily life events, accompanied by
anxiety and somatic symptoms, occurring more days than not for a period of at
least six months (American Psychiatric Association [APA], 2013). Among the
general population, 12-month prevalence rates for GAD are between 2 and 4%,
and lifetime prevalence rates are between 4 and 7% (Blazer et al., 1991; Hunt et al.,
2002; Wittchen et al., 1994). The incidence of GAD is markedly greater among
clinical populations, with GAD being the most common anxiety disorder and the
second most common mental health concern within primary care facilities (Barrett
et al., 1988; Wittchen et al., 2002). As GAD is associated with significant social,
occupational, and functional impairment (e.g., Stein & Heimberg, 2004), there is a
great need for a highly effective psychological treatment.
In this chapter, we provide an overview of the research and evidence-based
treatment protocol for a GAD-specific treatment, cognitive behavioral therapy
targeting intolerance of uncertainty (CBT-IU). Within the theoretical model
for CBT-IU, intolerance of uncertainty (IU) represents the underlying mechanism
that drives the development and maintenance of excessive worry and contributes to
additional model components that serve as maintenance factors in GAD.
Relevant Research
CBT-IU is a theoretically derived treatment protocol for GAD that has been
refined and validated over 25 years of clinical research (see Robichaud et al.,
2019, for review). Its central focus is to address GAD symptoms by targeting what
underlies the development and maintenance of excessive worry, namely, intolerance of uncertainty. IU can be defined as a dispositional characteristic that results
152
CBT for Generalized Anxiety Disorder
from a set of negative beliefs about uncertainty and its consequences (e.g., uncertainty is unfair, leads to negative and catastrophic outcomes, and is beyond one’s
ability to cope) (Hebert & Dugas, 2018; Robichaud et al., 2019). Research related to
CBT-IU for GAD has focused not only on the efficacy of the treatment itself but
also on the processes targeted within the protocol and its corresponding
theoretical conceptualization.
CBT-IU Processes
The four cognitive processes targeted in treatment include intolerance of uncertainty, positive beliefs about the function of worry, negative problem orientation,
and cognitive avoidance. Each of these factors has been extensively researched with
respect to its association to GAD symptoms, and is conceptualized as contributing
to the development and maintenance of excessive and uncontrollable worry. IU,
however, is viewed as a higher-order process that drives the worry cycle, and at least
partially accounts for the associations between the remaining factors and GAD.
That is, positive beliefs about worry, negative problem orientation, and cognitive
avoidance can be explained, at least in part, by the level of intolerance of uncertainty in individuals with GAD. IU is thus the key feature of this theoretical model,
and should be addressed at all phases of treatment.
Intolerance of Uncertainty
Given the primacy of IU in the CBT-IU protocol, a great deal of research has been
conducted over the years to identify evidence for a potential causal relationship
between IU and worry in GAD (e.g., Bomyea et al., 2015; Dugas et al., 2004b;
Koerner & Dugas, 2006; Meeten et al., 2012). Evidence for this link comes from
various sources, including longitudinal research, wherein variations in IU were
found to predict increases in excessive worry over time (Dugas et al., 2012).
A bidirectional relationship between IU and worry also emerged, whereby habitual
worry predicted greater IU. In addition, research using an experimental design has
found that heightening IU momentarily increases worry. Specifically, when the level
of IU is experimentally manipulated, participants in the group who underwent IU
manipulation reported higher levels of worry compared with those in the control
condition who did not have an IU manipulation (Ladouceur et al., 2000; Meeten
et al., 2012). Evidence in support of IU as a causal risk factor for GAD can also be
found in the temporal sequence of change, with changes in IU preceding changes in
worry during treatment (Dugas et al., 1998).
It is worth noting that research has also linked IU to symptoms of many other
disorders, including obsessive-compulsive disorder, social anxiety disorder, posttraumatic stress disorder, panic disorder, depression, and eating disorders (see
Carleton, 2016, for review), making IU a transdiagnostic process. However, several
studies have identified the association of IU to worry as stronger than its relationship to other anxiety and mood disorders (Dugas et al., 1997, 2004a; Ladouceur
et al., 1999), as well as to conceptually related processes, such as perfectionism and
153
154
Cara R. Dunkley and Melissa Robichaud
need for control over all facets of life (Buhr & Dugas, 2006). Moreover, a study
examining whether the level of IU predicted GAD symptom severity found that
clients with severe GAD had higher levels of IU than those with more moderate
symptoms of GAD (Dugas et al., 2007). These findings suggest that despite its
significant relationship to other mental health conditions, IU maintains a sensitive
and specific association to the excessive, uncontrollable worry characteristic
of GAD.
Positive Beliefs about Worry
Although most people view the process of worry as helpful to some degree on
occasion, individuals with GAD appear to hold positive beliefs about the function
of worry to a greater degree than nonclinical worriers (Ladouceur et al., 1998,
1999). Common positive beliefs include the notion that worry (1) aids in
problem-solving, (2) represents a motivational force, (3) is a positive personality
trait, (4) can prevent negative outcomes in and of itself, and (5) protects against
negative emotions. The extent to which nonclinical individuals hold a combination of these beliefs is associated with problematic worry (Robichaud et al.,
2003). Overall, the belief that worry is useful perpetuates the ongoing use of
worry as a means of reducing uncertainty, and can lead to ambivalence about
change with treatment.
Negative Problem Orientation
Problem orientation describes one’s attitude and confidence about problems and
problem-solving ability. Individuals with a positive problem orientation view problems as normal challenges that can be effectively overcome, while individuals with a
negative problem orientation view problems as insurmountable obstacles. Research
into the relationship between problem-solving and worry suggests that individuals
with GAD endorse a strong negative problem orientation via self-report, which is
marked by the tendency to view daily life problems as threatening, to doubt their
problem-solving ability, and to be pessimistic about the outcome of problem solving
(Robichaud & Dugas, 2005a, 2005b). As daily life problems are inherently marked
by uncertainty, it is not surprising that individuals with GAD view problems as
threatening. Indeed, research has found negative problem orientation to predict
GAD symptom severity (Dugas et al., 2007) and to be associated with worry above
and beyond worry’s relationship to anxiety, depression, and IU (Dugas et al., 1997;
Robichaud & Dugas, 2005b). Yet negative problem orientation in individuals with
GAD does not reflect deficits in actual problem-solving ability; rather, having a
negative problem orientation appears to lead to increased anxiety and avoidance
when confronted with problems (Davey, 1994), which in turn interferes with the
proper application of problem-solving skills (D’Zurilla & Nezu, 2006). As such,
holding negative perceptions about problems and problem-solving, as well as mentally reviewing problems through worry, impedes individuals with GAD from
engaging in active problem-solving.
CBT for Generalized Anxiety Disorder
Cognitive Avoidance
Individuals with GAD tend to engage in a number of cognitive avoidance strategies
as an attempt to cope with worry and avoid threatening cognitive and emotional
content. Worry represents an implicit form of cognitive avoidance, in that it is a
verbal-linguistic process that reduces physiological arousal and inhibits emotional
processing (Borkovec et al., 2004). Cognitive avoidance of mental imagery involving
disturbing events is therefore a particularly common process among individuals with
GAD. A number of ineffectual cognitive avoidance strategies are associated with
GAD, including thought suppression, substituting worries with positive thoughts,
mental distraction, and situational avoidance (Sexton & Dugas, 2008). Research has
shown that writing about feared hypothetical situations elicits vivid mental imagery,
which promotes emotional processing of core fears and results in subsequent
reductions in worry among pathological worriers (Fracalanza et al., 2014;
Ovanessian et al., 2018). Repeated writing about core fears is thus theorized to
reduce negative beliefs about emotion, as well as shift perceptions about feared
hypothetical situations and coping ability.
Treatment Efficacy Research
The efficacy of the CBT-IU protocol in treating GAD has been demonstrated
through several clinical trials. First, significant reductions in GAD symptoms from
pre- to post-treatment have been observed in both individual and group formats
when compared with wait-list controls (Dugas et al., 2003; Ladouceur et al., 2000).
Specifically, treatment gains and diagnostic remission of GAD were maintained for
77% participants at one-year follow-up. CBT-IU has also been compared to wait-list
controls and applied relaxation (AR) therapy in a randomized controlled trial
(Dugas et al., 2010). CBT-IU emerged as being markedly superior to wait-list
controls and marginally superior to AR. Treatment gains were largely maintained
among those in the CBT-IU group, and diagnostic remission remained high in the
majority of participants at two-year follow-up. Although AR was beneficial to those
with GAD, only CBT-IU produced ongoing improvement over time in both symptom measures and diagnostic remission. Among individuals in the CBT-IU group,
parallel decreases in IU and GAD symptoms were observed during treatment and
follow-up. Further, reductions in IU predicted reductions in GAD symptoms above
and beyond several other important therapy factors. A new single-component
treatment that focuses entirely on behavioral experiments targeting IU has been
developed in recent years. Preliminary testing of this treatment showed statistically
and clinically significant improvements in GAD severity and associated symptoms,
pathological worry, and IU, comparable to that observed in studies testing the
efficacy of the entire CBT-IU protocol (Hebert & Dugas, 2018). Collectively, these
findings provide additional evidence for the importance of targeting IU in treating
GAD and suggest that CBT-IU leads to substantial short- and long-term improvements in GAD symptoms.
155
156
Cara R. Dunkley and Melissa Robichaud
Assessment
GAD is one of the more challenging anxiety disorders to accurately diagnose. There
are several likely reasons for this. For example, clients with GAD are often reluctant to seek help as worry may not be viewed as a legitimate mental health
complaint, and the main diagnostic criteria for GAD is ambiguous, given that the
central feature of GAD is “excessive and uncontrollable worry” about daily life
events. Furthermore, individuals with GAD largely report worrying about the same
topics as nonclinical worriers. The content of worry is therefore less relevant in
discriminating between clinical and nonclinical worry. Rather, it is the extent to
which reported worries are experienced as frequent, excessive, and uncontrollable
that distinguishes worry characteristic of GAD. As such, the frequency and severity
of worry is the primary focus of the assessment. In order to accurately diagnose
GAD, excessive worries relating to several life domains should be examined. Clients
who meet diagnostic criteria for GAD will endorse at least two worry domains as
frequent, excessive, and uncontrollable.
In addition to excessive worry, at least three of the seven GAD-associated
somatic and cognitive symptoms (i.e., restlessness, fatigue, irritability, muscle tension, sleep difficulties, concentration, and memory impairment) are required for
diagnosis. Only muscle tension, however, is specific to a diagnosis of GAD, as the
other associated symptoms can also be seen in various anxiety and mood disorders.
GAD-associated somatic and cognitive symptoms should therefore not be a primary
consideration when diagnosing GAD.
Given the diagnostic challenges, GAD is best assessed using a structured
diagnostic interview. The Anxiety and Related Disorders Interview Schedule for
DSM-5 (ADIS-5; Brown & Barlow, 2014) and Structured Clinical Interview for
DSM-5 Disorders, Clinician Version (SCID-5-CV; First et al., 2016) are the most
common measures for the structured assessment of GAD. Therapists assessing for
GAD should consider whether the presence of excessive worry is best accounted for
by GAD or another diagnosis, and whether particular worry themes, such as those
pertaining to social settings or one’s health, are best captured by GAD or indicative
of an additional separate diagnosis.
Structured diagnostic interviews can be augmented with a battery of self-report
measures. The Intolerance of Uncertainty Scale (IUS; Buhr & Dugas, 2002) assesses
negative beliefs about uncertainty, and the Penn State Worry Questionnaire
(PSWQ; Meyer et al., 1990) assesses the tendency for excessive and uncontrollable
worry; these can be administered across all sessions. Additional measures that may
be employed include the Negative Problem Orientation Questionnaire (NPOQ;
Robichaud & Dugas, 2005a), which assesses negative problem orientation); the
Why Worry-II (WW-II; Hebert et al., 2014), which assesses positive beliefs about
worry; and the Cognitive Avoidance Questionnaire (CAQ; Sexton & Dugas, 2008),
which assesses the tendency to engage in cognitive avoidance. Administering these
questionnaires before, during, and after treatment provides a means by which
treatment efficacy and client gains can be measured.
CBT for Generalized Anxiety Disorder
Key Points
Accurate identification and differential diagnosis of GAD can be challenging,
and a comprehensive assessment is therefore recommended.
The distinction between GAD and nonclinical worry is quantitative (frequent,
excessive, and uncontrolled), rather than qualitative (content of worry).
GAD is best assessed using a structured assessment tool alongside selfreport questionnaires.
Formulation and Case Conceptualization
An appropriate theoretical model for any clinical disorder should be able to account
for the particular symptom presentation within that disorder. The CBT-IU model
emphasizes the primary role of intolerance of uncertainty (IU) in developing and
maintaining excessive and uncontrollable worry. As such, IU needs to account for
the symptoms and phenomenology in GAD, specifically, its cardinal feature of
frequent, excessive, and uncontrollable worry, as well as its dynamic and varied
content over time.
Why would IU account for the worry that is characteristic of GAD? In order to
address this question, it is first important to clarify how worry in GAD is conceptualized. Worry can be viewed as an attempt to mentally plan and prepare for all
potential negative outcomes prior to their occurrence (e.g., “What if X happened?
Then I might do . . . But what if Y happened?”). However, this elaborate scenario
building tends to generate further worry among individuals with GAD, as worries
centers upon what could happen, which allows for infinite iterations of hypothetical
outcomes that could occur. Moreover, worry is highly dynamic in GAD, as worry
content tends to shift from day to day, typically involving daily life events such as
finances, work or school, family, and health of self and loved ones. In fact, people
with GAD often report worrying about “everything.”
Within the CBT-IU model, it is posited that individuals with GAD worry
excessively as an attempt to reduce, avoid, or otherwise circumnavigate uncertainty
by thinking about and planning for all potential outcomes in an uncertain situation.
However, given that daily life is inherently fraught with uncertainty, there is always
something to worry about. In addition, given that the particular uncertainties that an
individual might face can change daily, worry topics can be expected to shift
accordingly; hence, worry can be about multiple daily life events.
Although everybody occasionally worries, most people can tolerate some
uncertainty in daily life without the urge to mentally plan and prepare as a result.
This is because IU can be conceptualized as a dimensional construct, with individuals with GAD displaying higher levels of intolerance than low or moderate worriers, such that even a minute amount of uncertainty in a given situation has the
potential to incite a worry episode. In other words, the threshold for uncertainty in
daily life situations is much lower among individuals with GAD; as such, their
157
158
Cara R. Dunkley and Melissa Robichaud
worries are more easily triggered than is the case with individuals with a higher
tolerance or threshold for uncertainty. Although the fear of uncertainty is present in
many anxiety disorders, it is likely that it is the “general state of uncertainty” that
characterizes GAD, hence the presence of varied and changing worry themes in
GAD. Much like the fear of negative evaluation typically represents the source of
threat in social anxiety, the fear of uncertainty is posited as the theme of threat
in GAD.
For example, consider an individual who spends much of their day occupied by
various worries, including concerns about the health and well-being of their children, finances, career, and social interactions. To cope with these worries, they
might engage in a number of safety behaviors, such as repeatedly checking on their
children via text, asking multiple people for their opinions when making decisions,
overpreparing for work presentations, or rereading emails several times before
sending them. The worries that they experience, as well as all of these disparate
safety behaviors, represent cognitive and behavioral attempts to reduce or avoid
uncertainty. As such, despite the varied content of their worries and associated
behaviors, their thoughts and actions can be parsimoniously understood as strategies to manage the fear of uncertainty. With this in mind, CBT-IU centers on
targeting IU by changing negative beliefs about uncertainty and its consequences,
rather than focusing on the specific content of worry. Given the frequent change in
worry topics from day to day that is seen in GAD, CBT interventions that target
worries themselves, rather than the underlying process that drives worry, would be
akin to chasing a moving target. CBT-IU therefore aims to indirectly reduce GAD
symptoms by directly targeting the fear of uncertainty.
Overview of CBT-IU
The CBT-IU treatment protocol can vary in length from 12 to 16 sessions and
encompasses multiple modules that can be flexibly applied according to clients’
particular presentation. Though all aspects of CBT for IU are helpful for individuals
with GAD, the primary emphasis across therapy is increasing tolerance to uncertainty by developing more balanced beliefs about uncertainty and its consequences.
The goal of CBT-IU is not to free clients entirely of worry but to help them worry
less as a result of challenging negative beliefs about uncertainty.
Module 1: Psychoeducation about GAD and the Cycle of Worry
Psychoeducation
The first module centers on providing psychoeducation about CBT and the
meaning of a GAD diagnosis. The diagnostic characteristics of GAD are outlined, with emphasis being placed on the primary symptom of frequent, excessive, and uncontrollable worry about daily life events. The somatic symptoms
associated with GAD are reviewed, and anxiety is described as the physiological
CBT for Generalized Anxiety Disorder
Figure 9.1. Initial clinical model of GAD for CBT-IU.
result of excessive worry. GAD can be presented to clients as existing on a
continuum, with everyone falling somewhere from low to high anxiety and worry.
In this way, the diagnosis of GAD is conceptualized as a matter of degree, with
individuals meeting diagnostic criteria if their worry is excessive and creates
significant distress or impairment. The distinction between nonclinical and clinical worries is thus not qualitative in nature, as GAD is seen as an excessive
manifestation of a normal phenomenon.
Clients are presented with an initial pictorial model of GAD that describes the
basic worry cycle (see Figure 9.1). Negative beliefs about uncertainty and its impact
on model components are introduced in later sessions when IU is presented. The
initial CBT-IU model begins with a triggering situation marked by novelty (e.g.,
going to a new restaurant), ambiguity (e.g., noticing an unclear ache in one’s body),
or unpredictability (e.g., taking a test at school). Triggers lead to initial “what if”
questions (e.g., “What if I fail my exam?”), which in turn lead to worry. Worry as a
strategy is described as elaborate scenario building of potential negative outcomes
and attempts at mental problem-solving (e.g., “What if I fail my exam? I could
perhaps retake it, but what if the teacher refuses? Maybe I could do an extra credit
assignment?”). Given that the predicted feared negative outcomes have not
happened yet (and may never happen), there are endless permutations of what
“could” occur. This process engenders further “what if” thoughts and further
worries, resulting in a cycle of worry (e.g., “What if I do poorly on the extra credit
159
160
Cara R. Dunkley and Melissa Robichaud
assignment? I could fail the class. What if I am unable to graduate?”). It is this
elaborate mental process that ultimately leads to the physiological experience
of anxiety.
To cope with worry and anxiety, people typically engage in safety behaviors
(e.g., excessively checking of the course syllabus to repeatedly reassure oneself
that they’ve studied all of the test material). Safety behaviors are deliberate
actions designed to reduce anxiety and feared outcomes in the short term;
however, such actions prevent the acquisition of corrective information, as the
nonoccurrence of feared negative outcomes is taken as evidence that they otherwise would have occurred if not for the use of safety behaviors. As a consequence, fears are maintained in the long term, thereby perpetuating the cycle
of worry.
Worry Awareness Training. Following the exposition of the initial GAD model,
clients are presented with their first between-session exercise, which involves selfmonitoring. Clients are invited to record a worry episode three times day, noting (1)
the internal (thought or physical sensation) or external (actual situation) triggering
event, (2) the original “what if” thought, (3) the level of anxiety (on a 0–10 scale),
and (4) any associated safety behaviors. Through worry awareness training, clients
are encouraged to become more aware of the content of their thoughts, as well as
the association between their worries, physical sensations, and behaviors. Worry
monitoring forms can then be debriefed in subsequent sessions, with the therapist
inquiring about specific worry themes, as well as potentially common safety
behaviors.
Key Points
Psychoeducation involves providing clients with information about the principles of CBT, as well as normalizing information about GAD diagnostic
criteria.
Clients are introduced to an initial model of GAD, with an emphasis on the
worry cycle, and the maintaining role of safety behaviors.
Worry awareness training helps clients become more cognizant of the interrelationship between their worries, anxiety, and associated safety behaviors.
Module 2: Reevaluation of the Usefulness of Worry
Identifying Positive Beliefs about Worry
As individuals with GAD tend to hold positive beliefs about the usefulness of worry,
the primary goal of this module is to help clients identify and challenge their beliefs
regarding the actual utility of worry. So long as worries are perceived as beneficial,
there remains a potential barrier to working toward worry reduction. Yet because
clients present for treatment in order to reduce their excessive worry, they may be
CBT for Generalized Anxiety Disorder
unaware of, or hesitant to discuss, any ambivalence they hold toward actually letting
go of their worries and their perceived benefits. However, as clients have typically
spent a debilitating amount of time occupied by worry, there is often a need to see
worry as serving some benefit, even while acknowledging its problems. The therapist can normalize conflicting beliefs about worry through an analogy:
Therapist:
Let’s say that a student consistently studies all night before an
exam, and they usually get good grades. Although they are
exhausted because of their overnight cram sessions, and know
that it probably isn’t a very good way to study, it nevertheless
feels like a helpful strategy because they are getting good grades.
So too with worry, people with GAD might find it exhausting and
stressful to worry all the time, yet they still might think that it is
useful to worry about their finances or their daily schedules, for
example, as it keeps them organized and on track in daily life.
Research has identified five main categories of positive beliefs about worry: (1)
worry helps to find solutions to problems; (2) worry can serve as a motivating
function; (3) worry can guard against negative emotions by helping to prepare for
terrible outcomes, should they occur; (4) worry, in and of itself, can prevent negative
outcomes; and (5) worrying is a positive personality trait. When introducing these
beliefs to clients, it is best accomplished through guided discovery, by asking clients
about the perceived benefits they derive from their specific worries (e.g., “What are
the positives about worrying about the health and well-being of your children?”),
rather than discussing worry in general (e.g., “Do you think worrying helps to
motivate you?”). This addresses the conflicting beliefs clients have about worry in
general (e.g., “It’s bad that I worry”), as opposed to their own particular worries
(e.g., “It’s good that I worry about my children, as it shows that I’m a loving and
caring parent”).
Challenging Positive Beliefs about Worry
Positive beliefs about worry are investigated through cognitive challenging to determine whether worry is indeed useful. One strategy to help reevaluate the function of
worry is the lawyer/prosecutor role-play, wherein clients first take on the role of a
lawyer tasked with convincing a jury about the usefulness of a specific worry (rather
than a belief in general), and then as the prosecutor, convincing the jury that worry
is not useful. Socratic questioning can be used to aid clients with this (see Table 9.1
for possible challenges according to worry belief ). Therapists can also ask clients to
consider the overall cost of worry in their lives, such as time spent worrying and
difficulties enjoying the present moment.
Focusing on specific worries, as opposed to the positive beliefs about worries
themselves, allows the case made for and against the usefulness of worry to be more
concrete, and prevents the discussion from becoming overly intellectualized.
161
162
Cara R. Dunkley and Melissa Robichaud
Table 9.1. Examples of challenges to positive beliefs about the usefulness of worry
Positive belief
Challenge
Worry helps to find solutions
to problems.
Do you actually solve your problems when you worry, or are you just
thinking a lot about the problem? Does worrying ever interfere with
problem-solving?
Does your worry really improve your performance? Are there times when
you got so worried about something that you were less likely to follow
through on it?
Does worrying about things that might never occur actually make you feel
better in the here and now? Has anything bad ever happened that you had
worried about before? If so, did worrying beforehand make you less
upset?
Is your belief about worry preventing negative outcomes based on real
evidence, or is it an assumption? Is it possible that you remember only
instances that support this belief?
Do you know any caring/conscientious/organized individuals who do not
worry excessively? Have you suffered any negative consequences from
friends and family (e.g., being seen negatively by others) because of your
excessive worry?
Worry can serve a motivating
function.
Worry can guard against
negative emotions.
Worry can prevent negative
outcomes.
Worry is a positive personality
trait.
Gathering evidence about the actual usefulness of worries also promotes insight into
the potentially paradoxical nature of beliefs about worry. For example, an individual might believe worrying about their health is positive, thinking that it motivates
them to exercise; however, worrying about health may also leave that individual so
stressed that they end up avoiding exercise altogether.
It is important to note that significant cognitive shifts are not expected at this
phase. Rather, clients are encouraged to recognize the positive beliefs they hold
about the utility of worry, and identify whether worry is providing any actual
benefits that cannot be obtained through other means.
Shifting Beliefs about Worry: Implications
A secondary goal of this module is to allow clients to consider what it would truly
mean to worry less. What would they do with all their time that was previously spent
worrying? What would it say about them as a person if they were no longer “a
worrier”? As many clients cannot remember a time without excessive worry, this
can be an emotional experience. Exploring what it really means to worry less allows
clients to address any fears or feelings of loss they might experience with respect to
this change, as well as begin to conceive of a future free of excessive worry.
It should be noted that not all positive beliefs about worry are mistaken or
incorrect; rather, discussion on this topic is intended to assist clients in ascertaining
whether or not their excessive worries are as helpful as they think, and whether it is
possible to obtain any perceived uses of worry through alternative means if there
are in fact benefits achieved through worry.
CBT for Generalized Anxiety Disorder
Key Points
People with GAD tend to hold positive beliefs about the usefulness of worry
that might lead to ambivalence toward the goals of treatment.
Socratic questioning is encouraged to help clients identify the specific positive
beliefs they hold about their own worries.
Cognitive challenging using a lawyer/prosecutor role-play can help clients to
challenge their positive beliefs about worry by identifying evidence for and
against their beliefs and recognizing paradoxes within their evidence.
Exploring what it would mean to worry less is a valuable exercise that can help
clients prepare for a life after treatment without excessive worry as
a backdrop.
Module 3: Reevaluation of Negative Beliefs about Uncertainty
IU and Negative Beliefs about Uncertainty
Module 3 focuses on IU and negative beliefs about uncertainty, which are the basis
of the GAD model and the primary component of treatment. IU is posited as the
underlying source of the excessive worry characteristic of GAD, and is therefore
directly targeted in this module. Clients are introduced to negative beliefs about
uncertainty and their consequences within the GAD model, as well as their impact
on the development and maintenance of GAD. Graduated behavioral experiments
are the primary therapeutic strategy introduced in this section, and they are
designed to directly test the accuracy of negative beliefs about uncertainty.
Therapists are encouraged to devote time to explaining the relationship
between negative beliefs about uncertainty and excessive worry. Specifically, people
with GAD are susceptible to worry even in situations with small amounts of
uncertainty, as their tolerance threshold for uncertainty is low. As a result, they
engage in worry as a means of decreasing uncertainty and increasing their sense of
control by mentally preparing for hypothetical eventualities. In fact, situations that
initially trigger the worry cycle, that is, novel, unpredictable, or ambiguous situations, all share uncertainty as a common factor. However, uncertainty is not
inherently threatening. For example, many people likely would not perceive going
to dinner with friends at a new restaurant as particularly threatening. Rather, it is an
individual’s beliefs about the consequences of uncertainty in triggering situations
that are perceived as threatening, and ignite a worry cycle as a consequence (e.g.,
“What if I don’t like the food at the restaurant? What if I order something I don’t
like, and waste time and money as a result?”).
The basic model of GAD is expanded through the inclusion of negative beliefs
about uncertainty, and its impact on worry and safety behaviors (Figure 9.2). There
are four primary negative beliefs about uncertainty that individuals with GAD tend
163
164
Cara R. Dunkley and Melissa Robichaud
Figure 9.2. Expanded clinical model of GAD for CBT-IU model featuring negative beliefs
about uncertainty.
to hold, each of which can contribute to perceptions of threat in uncertaintyinducing situations:
1. Events with ambiguous or unknown outcomes will turn out negatively. This
belief involves the overestimation of the likelihood of threat, wherein individuals inflate the expectation that uncertain situations are highly likely to have
negative outcomes.
2. Negative outcomes will be catastrophic. This belief involves the overestimation
of the severity of threat.
3. Inability to cope with negative outcomes. This involves the tendency to greatly
underestimate one’s ability to cope or act effectively in the face of a
negative outcome.
4. Being uncertain is unacceptable, unpleasant, or unfair. This involves the belief
that the general state of uncertainty is upsetting and unjust.
CBT for Generalized Anxiety Disorder
When introducing the expanded model, it is important to normalize the experience of excessive worry considering the client’s negative beliefs about uncertainty.
Regardless of the accuracy of one’s beliefs about uncertainty, it makes sense that
someone with these beliefs would view uncertain events as threatening. The goal of
this module is therefore to develop more balanced beliefs about uncertainty and its
outcomes, with the expectation that this would reduce threat perceptions toward
uncertain situations, and reduce worry and anxiety as a result. Balanced beliefs
about uncertainty and its consequences include (1) belief that uncertain events will
probably turn out all right, (2) confidence in one’s ability to cope with negative
outcomes should they arise, and (3) understanding that some uncertainty is a
normal part of life. It is worth noting that individuals with balanced beliefs
about uncertainty are not expected to never worry; rather, their tolerance
threshold is increased, such that some uncertainty in daily life is viewed as less
threatening overall.
Safety Behaviors
In order to challenge negative beliefs about uncertainty, it is necessary to help
clients identify the various ways in which IU manifests through behavior. As noted
earlier, safety behaviors maintain fears through the nonoccurrence of feared outcomes, and thereby prevent the acquisition of corrective information. In the case of
GAD, individuals are prevented from learning about the actual outcome of uncertain situations and their correspondent coping ability due to the use of
safety behaviors.
GAD-related safety behaviors involve a host of actions that are designed to
either avoid uncertainty altogether or reduce the uncertainty of the situation as
much as possible. Common safety behaviors seen among clients with GAD include
excessive information seeking, reassurance seeking, double-checking, avoidance of
uncertainty-inducing situations, procrastination, refusal to delegate tasks to others,
and the maintenance of a predictable routine.
Behavioral Experiments
In order to allow clients to directly test the accuracy of their beliefs about uncertainty and its consequences, behavioral experiments are introduced. Behavioral
experiments involve deliberately refraining from engaging in a safety behavior,
making a prediction about the feared outcome, and then observing the actual
outcome. In this manner, clients have the opportunity to discover whether
uncertainty-inducing situations do in fact consistently lead to negative outcomes,
whether those outcomes are catastrophic, and whether they are able to cope with
any negative outcomes, if any. The development of balanced beliefs therefore
occurs through direct testing and the accrual of discomfirmatory disconfirmatory
evidence through experience. However, given the anxiety-provoking nature of
165
166
Cara R. Dunkley and Melissa Robichaud
behavioral experiments, it is helpful to provide clients with a compelling and logical
rationale for their use, as in the following dialogue:
Therapist:
Client:
Therapist:
In general, we tend to change our beliefs when there is
compelling evidence that contradicts our current beliefs and
supports alternative beliefs. For example, if I supported a
particular political candidate, and you wanted to convert me to
support another candidate, how would you sway me?
I would probably list the various platforms, credentials, and
virtues of my candidate, and discuss your candidate’s weaknesses.
Precisely. You would provide evidence that supports your belief
and challenges mine. We approach negative beliefs about
uncertainty in the same way. You are unlikely to change your
mind about the consequences of uncertainty without direct
evidence. Unfortunately, your safety behaviors prevent you from
finding out what would actually happen if you allowed some
uncertainty into your life. For example, if you always look up
your exact route before driving somewhere new in order to be
sure that you know the way, then you never have the opportunity
to find out whether you would have gotten lost without advance
directions, or what you would have done if you had lost your way.
Behavioral experiments should be planned and intentional, with the client
recording (1) the experiment they are conducting, (2) their feared outcome(s), (3)
the actual outcome, and (4) how they coped with the situation if the outcome was
negative. Table 9.2 provides an example of a behavioral experiment record form.
Clients are encouraged to have an air of curiosity about the outcome of experiments, and debriefing within session should adopt the same exploratory tone.
Clients can be queried about whether the actual outcome matched the feared
Table 9.2. Example of behavioral experiment record form
(What did you
do?)
Deliberately
not
overpreparing
for a work
presentation
Ordering a meal
that you have
never tried
before
Feared outcome
(What were you worried
would happen?)
Actual outcome
(What actually happened?)
Coping
(If outcome was negative,
how did you cope?)
The presentation will be
horrible, and I won’t be
able to answer all of the
questions.
The presentation went
well, and I was able to
answer most questions
without difficulty.
No coping was necessary.
I won’t like it, I will not
be able to eat it, and my
friend will judge me.
I actually didn’t enjoy what
I ordered. I told my friend
and he suggested I order
something else.
I only ate a little bit of the
meal I ordered. I asked the
waiter for something else,
and they gave it to me
without any problem.
CBT for Generalized Anxiety Disorder
outcome; whether the outcome was negative, neutral, or positive; whether it was
viewed as catastrophic if the outcome was negative, whether it was viewed as
catastrophic; and how they handled a negative outcome if it occurred. Given that
belief change is expected to occur only with a preponderance of evidence, it is
recommended that multiple sessions be devoted to developing, and then debriefing,
behavioral experiments. Every few sessions, the therapist can help the client examine all of the experiments completed to date with the goal of identifying the
following overarching conclusions:
How often were the outcomes of experiments negative? Neutral? Positive?
When the outcomes were negative, how often were they catastrophic?
How did you manage negative outcomes when they arose?
How do you view your ability to cope when negative outcomes occurred?
What conclusions can you draw so far about the accuracy of your negative
beliefs about uncertainty?
By engaging in this process over the course of several weeks, clients are
encouraged to discover that negative outcomes are relatively infrequent, seldom
catastrophic, and typically manageable. In addition, it is hoped that clients gain
improved self-efficacy and confidence concerning their ability to cope with difficulty
through direct experience.
Key Points
Individuals with GAD tend to hold several negative beliefs about uncertainty
and its consequences, including an overestimation of the likelihood and
severity of threat in uncertain situations, underestimating of coping with
negative outcomes, and a tendency to view uncertainty as unacceptable
and unfair.
GAD-related safety behaviors designed to reduce or avoid uncertainty maintain negative uncertainty beliefs through the nonoccurrence of feared outcomes and the prevention of corrective information.
A primary goal of treatment is to encourage clients to obtain direct evidence
about the actual threat of uncertain situations in order to ultimately develop
balanced beliefs about uncertainty. This can be accomplished through
behavioral experiments.
Module 4: Problem-Solving Training
Modules 4 and 5 differ from earlier modules due to their treatment focus.
Specifically, while previous modules have largely targeted what underlies and
maintains excessive worry, the next sections involve interventions that address
specific worry content. Each module offers strategies for worries that persist
following an extensive focus on shifting negative beliefs about uncertainty.
167
168
Cara R. Dunkley and Melissa Robichaud
Module 4 addresses worries about current problems over which clients have some
degree of control. Clients are taught that problem-solving is a more practical
alternative to worry and are encouraged to target excessive worries about current
problems through the active strategy of problem-solving. Problem-solving training
involves addressing an individual’s negative problem orientation, followed by education on the effective use of problem-solving skills.
Negative Problem Orientation
Problem orientation concerns how we view problems and view ourselves as problem
solvers. Individuals with a negative problem orientation typically view problems as
negative and threatening, anticipate negative outcomes irrespective of effort, and
doubt their capacity to solve problems effectively. As a negative problem orientation can impede or block individuals from actually solving their problems, the goal is
to improve their problem orientation. There are three main strategies recommended for improving problem orientation: (1) viewing problems as a normal part
of life, (2) looking for benefits and opportunities within problems instead of focusing
solely on the threatening aspects, and (3) recognizing problems early by using one’s
emotions as a cue to be alerted to the presence of a problem in life, and developing a
recurrent problems list as a result (see D’Zurilla & Nezu, 2006).
Active Problem-Solving
Active problem-solving describes the deliberate and rational process of effectively
solving a problem through a series of steps, including (1) problem definition, (2)
goal formulation, (3) generation of possible solutions, (4) decision making, and (5)
solution implementation and verification (see D’Zurilla and Nezu, 2006, for a
thorough description of the application of these steps). Individuals with GAD not
only tend to have difficulty with the effective use of these skills when dealing with
their own worries, but they also struggle with moving forward in the problemsolving process. An emphasis is thus placed on helping them complete each stage
and move on to the next by seeking out the best solution given the information at
hand, not the “perfect solution.”
Key Points
After targeting negative beliefs about uncertainty, some clients with GAD
continue to experience residual excessive worries about current problems.
These worries can be addressed by targeting negative problem orientation
and engaging in the effective use of problem-solving skills.
Negative problem orientation involves the tendency to view problems as
threatening, to be pessimistic about the outcome of problem-solving, and to
underestimate one’s problem-solving ability. Clients with GAD can learn to
improve their problem orientation by identifying their problems early,
CBT for Generalized Anxiety Disorder
(cont.)
recognizing them as a normal part of life, and shifting their perceptions of
problems from threats to potential challenges or opportunities.
Problem-solving is an active strategy for managing worries about current
problems. Treatment at this stage focuses on teaching clients about the
effective use of problem-solving skills.
Module 5: Written Exposure
This module provides a means of addressing residual excessive worries about
hypothetical situations through written exposure. Research has shown that individuals with GAD tend to avoid feared thoughts and corresponding negative emotions
in order to reduce distress. Yet attempts to avoid or neutralize distressing thoughts
about hypothetical problems prevents clients from learning what would actually
happen if they allowed themselves to think about and visualize the situations they
fear. The futility of cognitive avoidance should be discussed prior to starting written
exposure; specifically, clients are told that attempts to suppress or avoid distressing
worries not only tend to be ineffective, but lead to a paradoxical increase in worries.
By engaging in written exposure, clients can tackle cognitive avoidance of core
fears. This method of exposure enables clients to test the accuracy of two negative
beliefs: (1) thinking about worst-case scenarios is dangerous and can heighten the
probability of the feared outcome occurring and (2) negative emotions brought on
by feared outcomes are dangerous, in that they are overwhelming and will result in a
loss of control. Through experiencing and emotionally processing hypothetical
worries, clients are able to shift perspective about how realistic the threat of their
feared worst-case scenarios actually is. It is worth noting that the goal of this module
is not to eliminate worries about distressing feared outcomes (e.g., a loved one
dying). Rather, by focusing on worst-case scenarios and reevaluating the actual
threat of both the situation and the attendant emotions it engenders, clients are
expected to shift their worries from the “front of the mind” (i.e., from a constant
worry) to the “back of the mind” (i.e., an upsetting thought that occurs on occasion
but is not ever-present).
As written exposure targets core fears, it is typically unnecessary for clients to
engage in this exercise more than once, since most hypothetical worries tend to from
a single core fear. Therapists can help clients identify their worst-case scenario
through the use of the following “downward arrow” technique (Provencher et al.,
2000; Vasey & Borkovec, 1992):
1. Clients are asked to describe their worry in simple terms (e.g., “What if my
partner is involved in a car crash?”).
2. They are then queried, “If your worry came true, what would that mean? What
would happen next?”
169
170
Cara R. Dunkley and Melissa Robichaud
3. After the client has responded with the subsequent link in the worry chain (e.g.,
“He could be seriously injured”), therapists continue to repeat the same question with the intent of getting progressively closer to the worst-case scenario.
Once the client is unable to provide an answer to the prompt, the core fear has
likely been identified (e.g., “He could die, and I would be alone, overwhelmed
with grief and unable to function”).
After identifying the core fear, clients are asked to write about their worst-case
feared scenario as concretely as possible in the first-person present tense, and to
include as much sense imagery as possible (smell, touch, sight, sound, taste) in order
to generate a vivid visual image. Clients are asked to complete this exercise consistently for at least a week (e.g., five to six times per week), for approximately
20 minutes on each occasion. After each written exposure session, clients are asked
to record the following: (1) level of distress, (2) estimated probability of the feared
outcome occurring, (3) estimate of how catastrophic the feared outcome would be,
and (4) estimated ability to cope with the feared outcome. As this exercise is often
anxiety-provoking for clients, it is helpful to normalize any anticipated distress,
provide a cogent rationale, and emphasize the goal of bringing excessive worries
about feared hypothetical situations to the back of the mind.
Key Points
Excessive worries about hypothetical situations can be addressed through
written exposure.
Clients with GAD tend to engage in cognitive avoidance strategies that not
only are futile, but also allow for the maintenance of negative beliefs about
their ability to cope with negative emotions and the overestimation of the
dangerousness of feared outcomes.
Through written exposure, clients can face core fears by repeatedly writing
about their feared worst-case scenarios in vivid and concrete visual images.
In addition to allowing for a reevaluation of the actual danger of feared
scenarios, written exposure can help clients shift their worries about hypothetical situations to the “back of the mind” rather than being a frequent and
recurrent worry theme.
Module 6: Relapse Prevention
The final sessions should focus on reviewing the skills gained throughout treatment
and creating a relapse prevention plan. First, therapists can clarify the difference
between a lapse and relapse, with a lapse being understood as the result of normal
fluctuations in worry and anxiety, and a relapse as a return to pre-treatment
symptom levels. A clients’ reaction to, or catastrophic interpretation of, a lapse
can influence whether the lapse becomes a relapse; it is thus important to stress that
CBT for Generalized Anxiety Disorder
occasional increases in worry and anxiety are unavoidable and are not necessarily a
sign of relapse. Additional skills to review in this module can include the following:
1. Check-ups for good mental health hygiene. It can be helpful for clients to get in
the habit of routinely setting aside 20–30 minutes (weekly or monthly) to
evaluate their overall mental health. Useful points to assess can include current
level of worry and anxiety, progress with behavioral experiments targeting
uncertainty beliefs, review of current exercises and generation of new ones,
and evaluation of short- and long-term goals.
2. Identification of at-risk situations. To ensure long-term maintenance of treatment gains, therapists can assist clients in identifying “red flags” that might
signal a return of problematic symptoms. This can include specific situations
(e.g., sleep difficulties), stressors (e.g., increased work demands), or behaviors
(e.g., increased use of safety behaviors). Recognizing warning signs early
encourages clients to take action quickly to remediate the situation.
3. Preparation for at-risk situations. Once at-risk situations have been identified, it
is helpful for clients to have a plan of action to address the situation. This plan
may entail increasing the frequency of mental health check-ins, reengaging with
various treatment modules, or getting in touch with their therapist.
The overall goal of this module is to ensure not only that clients maintain the gains
they have acquired in treatment, but that they also are able to build upon and
consolidate those gains over the long term. In essence, by becoming “their own
therapist,” clients are encouraged to normalize lapses and manage them independently over the long term.
Key Points
Relapse prevention involves both the maintenance of treatment gains and
preparation for potential lapses in the future.
The difference between a lapse and a relapse can be reviewed, with an
emphasis on normalizing the occurrence of fluctuations in worry and anxiety
over time.
It is helpful for clients to have self-led sessions on a consistent basis to review
their overall mental health, as well as identify and plan for potential atrisk situations.
FURTHER RECOMMENDED READING
Hebert, E. A., & Dugas, M. J. (2018). Behavioral experiments for intolerance of uncertainty:
Challenging the unknown in the treatment of generalized anxiety disorder. Cognitive and
Behavioral Practice, https://doi.org/10.1016/j.cbpra.2018.07.007
Koerner, N., Mejia, T., & Kusec, A. (2017). What’s in a name? Intolerance of uncertainty,
other uncertainty-relevant constructs, and their differential relations to worry and generalized anxiety disorder. Cognitive Behavior Therapy, 46, 141–161.
171
172
Cara R. Dunkley and Melissa Robichaud
Robichaud, M., & Dugas, M. J. (2015). The generalized anxiety disorder workbook:
A comprehensive CBT guide for coping with uncertainty, worry, and fear. New
Harbinger.
Robichaud, M., Koerner, N., & Dugas, M. J. (2019). Cognitive-behavioral treatment for
generalized anxiety disorder: From science to practice. Routledge.
REFERENCES
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Barrett, J., Oxman, T. E., & Gerber, P. D. (1988). The prevalence of psychiatric disorders in
primary care practice. Archives of General Psychiatry, 45, 1100–1106.
Blazer, D. G., Hughes, D., George, L. K., Schwartz, M., & Boyer, R. (1991). Generalized
anxiety disorder. In L. N. Robins & D. A. Regier (Eds.), Psychiatric disorders in
America. The Epidemiologic Catchment Area Study (pp. 180–203). Free Press.
Bomyea, J., Ramsawh, H., Ball, T. M., Taylor, C. T., Paulus, M. P., Lang, A. J., & Stein, M. B.
(2015). Intolerance of uncertainty as a mediator of reductions in worry in a cognitive
behavioral treatment program for generalized anxiety disorder. Journal of Anxiety
Disorders, 33, 90–94.
Borkovec, T. D., Alcaine, O., & Behar, E. (2004). Avoidance theory of worry and generalized
anxiety disorder. In R. G. Heimberg, C. L. Turk, & D. S. Mennin (Eds.), Generalized
anxiety disorder: Advances in research and practice (pp. 78–108). Guilford Press.
Brown, T. A., & Barlow, D. H. (2014). Anxiety and related disorders interview schedule
for DSM-5 (ADIS-5) – Adult and lifetime version: Clinical manual. Oxford University
Press.
Buhr, K., & Dugas, M. J. (2002). The Intolerance of Uncertainty Scale: Psychometric
properties of the English version. Behavior Research and Therapy, 40, 931–945.
(2006). Investigating the construct validity of intolerance of uncertainty and its unique
relationship with worry. Journal of Anxiety Disorders, 20, 222–236.
Carleton, R. N. (2016). Into the unknown: A review and synthesis of contemporary models
involving uncertainty. Journal of Anxiety Disorders, 39, 30–43.
Davey, G. C. (1994). Worrying, social problem-solving abilities, and social problem-solving
confidence. Behaviour Research and Therapy, 32, 327–330.
Dugas, M. J., Brillon, P., Savard, P., Turcotte, J., Gaudet, A., Ladouceur, R., Leblanc, R., &
Gervais, N. J. (2010). A randomized clinical trial of cognitive-behavioral therapy and
applied relaxation for adults with generalized anxiety disorder. Behavior Therapy, 41,
46–58.
Dugas, M. J., Buhr, K., & Ladouceur, R. (2004b). The role of intolerance of uncertainty in
etiology and maintenance. In R. G. Heimberg, C. L. Turk, & D. S. Mennin (Eds.),
Generalized anxiety disorder: Advances in research and practice. Guilford Press, 143–163.
Dugas, M. J., Freeston, M. H., & Ladouceur, R. (1997). Intolerance of uncertainty and
problem orientation in worry. Cognitive Therapy and Research, 21, 593–606.
Dugas, M. J., Gagnon, F., Ladouceur, R., & Freeston, M. H. (1998). Generalized anxiety
disorder: A preliminary test of a conceptual model. Behaviour Research and Therapy,
36(2), 215–226.
Dugas, M. J., Laugesen, N., & Bukowski, W. M. (2012). Intolerance of uncertainty, fear of
anxiety, and adolescent worry. Journal of Abnormal Child Psychology, 40, 863–870.
Dugas, M. J., Ladouceur, R., Léger, E., Freeston, M. H., Langlois, F., Provencher, M. D.,
et al. (2003). Group cognitive-behavioral therapy for generalized anxiety disorder:
Treatment outcome and long-term follow-up. Journal of Consulting and Clinical
Psychology, 71, 821–825.
CBT for Generalized Anxiety Disorder
Dugas, M. J., Savard, P., Gaudet, A., Turcotte, J., Laugesen, N., Robichaud, M., Francis, K.,
& Koerner, N. (2007). Can the components of a cognitive model predict the severity of
generalized anxiety disorder? Behavior Therapy, 38, 169–178.
Dugas, M. J., Schwartz, A., & Francis, K. (2004a). Intolerance of uncertainty, worry, and
depression. Cognitive Therapy and Research, 28, 835–842.
D’Zurilla, T. J., & Nezu, A. M. (2006). Problem-solving therapy: A positive approach to
clinical intervention (3rd ed.). Springer Publishing.
First, M. B., Williams, J. B. W., Karg, R. S., & Spitzer, R. L. (2016). Structured Clinical
Interview for DSM-5 Disorders – Clinician version (SCID-5-CV). American Psychiatric
Association Publishing.
Fracalanza, K., Koerner, N., & Antony, M. M. (2014). Testing a procedural variant of written
imaginal exposure for generalized anxiety disorder. Journal of Anxiety Disorders, 28, 559–569.
Hebert, E. A., & Dugas, M. J. (2018). Behavioral experiments for intolerance of uncertainty:
Challenging the unknown in the treatment of generalized anxiety disorder. Cognitive and
Behavioral Practice, https://doi.org/10.1016/j.cbpra.2018.07.007
Hebert, E. A., Dugas, M. J., Tulloch, T. G., & Holowka, D. W. (2014). Positive beliefs about
worry: A psychometric evaluation of the Why Worry-II. Personality and Individual
Differences, 56, 3–8.
Hunt, C., Issakidis, C., & Andrews, G. (2002). DSM-IV generalized anxiety disorder in the
Australian National Survey of Mental Health and Well-Being. Psychological Medicine,
32, 649–659.
Koerner, N., & Dugas, M. J. (2006). A cognitive model of generalized anxiety disorder: The
role of intolerance of uncertainty. In G. C. L. Davey & A. Wells (Eds.), Worry and its
psychological disorders: Theory, assessment and treatment. Wiley, 201–216.
Ladouceur, R., Blais, F., Freeston, M. H., & Dugas, M. J. (1998). Problem solving and
problem orientation in generalized anxiety disorder. Journal of Anxiety Disorders, 12,
139–152.
Ladouceur, R., Dugas, M. J., Freeston, M. H., Rhéaume, J., Blais, F., Boisvert, J.-M., et al.
(1999). Specificity of generalized anxiety disorder symptoms and processes. Behavior
Therapy, 30, 191–207.
Ladouceur, R., Gosselin, P., & Dugas, M. J. (2000). Experimental manipulation of intolerance of uncertainty: A study of a theoretical model of worry. Behavior Research and
Therapy, 38, 933–941.
Meeten, F., Dash, S. R., Scarlet, A. L. S., & Davey, G. C. L. (2012). Investigating the effect of
intolerance of uncertainty on catastrophic worrying and mood. Behavior Research and
Therapy, 50, 690–698.
Meyer, T. J., Miller, M. L., Metzger, R. L., & Borkovec, T. D. (1990). Development and
validation of the Penn State Worry Questionnaire. Behavior Research and Therapy, 28,
487–495.
Ovanessian, M. M., Koerner, N., Antony, M. M., & Dugas, M. J. (2018). A preliminary test of
the therapeutic potential of written exposure with rescripting for generalized anxiety
disorder [Manuscript submitted for publication].
Provencher, M. D., Freeston, M. H., Dugas, M. J., & Ladouceur, R. (2000). Catastrophizing
assessment of worry and threat schemata among worriers. Behavioral and Cognitive
Psychotherapy, 28, 211–224.
Robichaud, M., & Dugas, M. J. (2005a). Negative problem orientation (part I): Psychometric
properties of a new measure. Behavior Research and Therapy, 43, 391–401.
(2005b). Negative problem orientation (part II): Construct validity and specificity to worry.
Behavior Research and Therapy, 43, 403–412.
Robichaud, M., Dugas, M. J., & Conway, M. (2003). Gender differences in worry and
associated cognitive-behavioral variables. Journal of Anxiety Disorders, 17, 501–516.
173
174
Cara R. Dunkley and Melissa Robichaud
Robichaud, M., Koerner, N., & Dugas, M. J. (2019). Cognitive-behavioral treatment for
generalized anxiety disorder: From science to practice. Routledge.
Sexton, K. A., & Dugas, M. J. (2008). The Cognitive Avoidance Questionnaire: Validation of
the English translation. Journal of Anxiety Disorders, 22, 355–370.
Stein, M. B., & Heimberg, R. G. (2004). Well-being and life satisfaction in generalized anxiety
disorder: Comparison to major depressive disorder in a community sample. Journal of
Affective Disorders, 79, 161–166.
Vasey, M. W., & Borkovec, T. D. (1992). A catastrophizing assessment of worrisome
thoughts. Cognitive Therapy and Research, 16, 505–520.
Wittchen, H.-U., Kessler, R. C., Beesdo, K., Krause, P., Höfler, M., & Hoyer, J. (2002).
Generalized anxiety and depression in primary care: Prevalence, recognition, and management. Journal of Clinical Psychiatry, 63, 24–34.
Wittchen, H.-U., Zhao, Z., Kessler, R. C., & Eaton, W. W. (1994). DSM-III-R generalized
anxiety disorder in the National Comorbidity Survey. Archives of General Psychiatry, 51,
355–364.
PART TWO
COGNITIVE BEHAVIORAL
THERAPY FOR POSTTRAUMATIC
STRESS DISORDER
10
CBT for PTSD
Lily A. Brown and Edna B. Foa
Research Supporting CBT for PTSD
A handful of cognitive behavioral therapies (CBTs) for PTSD have achieved
significant empirical support. The most studied CBTs are prolonged exposure
(PE) therapy (Foa et al., 2007), cognitive processing therapy (CPT; Resick &
Schnicke, 1992), and cognitive therapy (CT; Ehlers & Clark, 2000). While other
evidence-based therapies for PTSD exist, including eye-movement desensitization
and reprocessing (EMDR; Shapiro, 2001) and present-centered therapy (PCT;
Schnurr et al., 2007), these therapies do not fall under the domain of CBT either
because of therapeutic content (in the case of PCT) or because of preference from
the treatment developer (in the case of EMDR).
There have been several meta-analyses that have attempted to determine the
efficacy of CBT on PTSD. One recent meta-analysis found that across all CBTs for
PTSD, there was a medium effect on PTSD symptoms (Hedges’ g = 0.48, 95% CI:
0.26, 0.71) and a small effect on depression (Hedges’ g = 0.32, 95% CI: 0.18, 0.46),
general anxiety (Hedges’ g = 0.38, 95% CI: 0.23, 0.54), and quality of life (Hedges’ g =
0.24, 95% CI: 0.02, 0.47) relative to placebo control (Carpenter et al., 2018).
Individual CBT outperformed group CBT for PTSD; however, CBT was associated
with significantly higher dropout (29%) compared with placebo (17%; Carpenter
et al., 2018). Another meta-analysis separated effects by type of CBT for PTSD. This
study found that exposure therapy, including PE (Cohen’s d = 1.27, 95% CI: 1.00,
1.54), CPT (Cohen’s d = 1.40, 95% CI: 0.85, 1.95), and CT (Cohen’s d = 1.33, 95% CI:
0.67, 1.99) each had strong reductions on PTSD symptoms (Cusack et al., 2016).
Prolonged exposure outperformed relaxation for PTSD, but there were no other
outcome differences among the CBTs (Cusack et al., 2016). These findings replicated
an early study in which PE significantly outperformed control conditions in terms of
both PTSD outcomes (Hedge’s g = 1.08; 95% CI: 0.69, 1.46) and secondary clinical
characteristics (Hedges’ g = 0.77, 95% CI: 0.53, 1.01; Powers et al., 2010). A fourth
meta-analysis found that internet-delivered CBT for PTSD outperformed treatment
as usual (Hedges’ g = 0.71, 95% CI: 0.49, 0.93; Sijbrandij et al., 2016). Thus, there is
sufficient evidence to conclude that CBT is associated with significantly greater
177
178
Lily A. Brown and Edna B. Foa
reductions in PTSD symptoms and secondary targets compared with control conditions, and CBT can be effectively delivered in diverse contexts.
The efficacy of CBT for PTSD has been established across a wide range of
populations. In adolescents, PE is associated with significantly greater reductions in
PTSD symptoms compared with supportive counseling (Foa et al., 2013b). Both PE
and CPT have demonstrated efficacy in veteran (Acierno et al., 2017; Sloan et al.,
2018) and military samples (Foa et al., 2018; P. A. Resick et al., 2017). Survivors of
sexual assault respond well to both PE and CPT compared with minimal attention
or supportive counseling (Foa et al., 1991; P. A. Resick et al., 2002). Similar results
have emerged across various cultures and international locations (Asukai et al.,
2010; Schiff et al., 2015; Valentine et al., 2017)
There is also a growing evidence base to suggest that CBT for PTSD is effective
for complex comorbidities. For example, PE and CPT are both associated with
significant reductions in suicidal ideation over time (Bryan et al., 2016; Cox et al.,
2016), though most randomized controlled trials for PTSD exclude participants with
more than mild suicidal ideation. PE is also effective in patients with a concurrent
alcohol use disorder (Foa et al., 2013a), substance use disorders (Ruglass et al.,
2017), or a tobacco use disorder (Foa et al., 2017). Similarly, PE is effective in
individuals with psychosis (Grubaugh et al., 2017). Both CPT and PE have preliminary efficacy for those with traumatic brain injury (Ragsdale & Voss Horrell, 2016;
Wolf et al., 2015). The literature on CBT for PTSD and comorbid conditions is
likely to grow in coming years, as several studies to this effect are already underway.
Summary: There are several evidence-based treatments for PTSD, including
prolonged exposure therapy (PE), cognitive processing therapy (CPT), and
cognitive therapy (CT). These treatments consistently outperform control conditions, but there are few differences between active CBTs.
KEY READING
Cusack, K., Jonas, D. E., Forneris, C. A., Wines, C., Sonis, J., Middleton, J. C., . . . & Gaynes,
B. N. (2016). Psychological treatments for adults with posttraumatic stress disorder:
A systematic review and meta-analysis. Clinical Psychology Review, 43, 128–141.
Carpenter, J. K., Andrews, L. A., Witcraft, S. M., Powers, M. B., Smits, J. A. J., &
Hofmann, S. G. (2018). Cognitive behavioral therapy for anxiety and related disorders: A meta-analysis of randomized placebo-controlled trials. Depression and
Anxiety. doi:10.1002/da.22728
Powers, M. B., Halpern, J. M., Ferenschak, M. P., Gillihan, S. J., & Foa, E. B. (2010).
A meta-analytic review of prolonged exposure for posttraumatic stress disorder.
Clinical Psychology Review, 30(6), 635–641. doi:10.1016/j.cpr.2010.04.007
Clinical Management of PTSD
In the remainder of this chapter, we will discuss the clinical management of PTSD
from a CBT orientation. We will discuss issues related to assessment and
CBT for PTSD
formulation, and we will provide an overview of the treatment. A vignette of a
fictitious but realistic case is provided throughout to facilitate comprehension. While
many similar chapters could be written from a CPT or CT approach, we base our
discussion in the PE approach. However, many of the core concepts will be shared
across variants of CBT for PTSD.
Assessment
Evidence-based assessment of PTSD is a required component of CBT for PTSD. In
this section, we provide suggestions for administering state-of-the-science assessments of PTSD. We review considerations for conducting a standardized assessment
to derive an initial PTSD diagnosis and provide recommendations for assisting in
this diagnosis. In addition, we include a discussion of strategies for determining
patient readiness for CBT. Finally, we discuss approaches to monitor progress
throughout CBT.
Preliminary Diagnosis
The Clinician-Administered PTSD Scale for DSM-5 (CAPS-5; Weathers et al.,
2013a) and the PTSD Severity Scale Interview for DSM-5 (PSSI-5; Foa et al.,
2016a) are two empirically supported and widely used clinical interviews for diagnosing PTSD in adults. Both the CAPS-5 and the PSSI-5 have strong psychometric
properties (Foa et al., 2016a; Weathers et al., 2013a). Assessors using either measure consider intensity and frequency in evaluating each PTSD symptom.
There are several important differences between the measures. First, the
CAPS-5 provides a separate section to assess the dissociative subtype of PTSD, a
new subcategory of PTSD that was introduced in the Diagnostic and Statistical
Manual of Mental Disorders, 5th edition (DSM-5; American Psychiatric
Association, 2013), whereas the PSSI-5 includes only a single item to assess for
dissociation. Second, the CAPS-5 is always administered in conjunction with the
Life Events Checklist (Weathers et al., 2013b), a self-report questionnaire, in order
to facilitate identification of the index trauma (i.e., the most distressing trauma at
the time of assessment). In contrast, the PSSI-5 identifies the index trauma through
several items at the beginning of the administration. Finally, whereas both measures
have been developed for diagnosing current (i.e., past month) PTSD, the CAPS-5
also has a version to test for past week and worst point (lifetime) symptoms. Both
measures are used across research and clinical settings alike and are available on
request (CAPS-5: www.ptsd.va.gov/professional/assessment/ncptsd-instrumentrequest-form.asp; PSSI-5: email foa@pennmedicine.upenn.edu).
While a formal standardized clinical interview is the ideal method for determining PTSD, many clinicians may be unable to administer these measures due to
constraints on time or resources. In these cases, the PTSD Diagnostic Scale for
DSM-5 (PDS-5; Foa et al., 2016b) and the PTSD Checklist for DSM-5 (PCL-5;
Blevins et al., 2015) are two commonly used self-report screening measures to
identify which patients that should receive a formal PTSD assessment.
179
180
Lily A. Brown and Edna B. Foa
As mentioned above, the index trauma is the most distressing trauma at the
time of the assessment. However, many patients with PTSD have been exposed to
repeated trauma or to more than one type of trauma. In these cases, it may be
helpful to validate that the patient is not being asked to make a judgment as to
which trauma was the “worst.” Instead, the patient is asked to indicate which
trauma is distressing them most at the time of the assessment. This can be facilitated
by querying about whether the patient has been most bothered by particular
intrusive thoughts or nightmares in the past month, or if there are places that
they are avoiding due to reminders of a specific trauma memory. For patients
who feel equally bothered by multiple trauma memories, choosing one to start
with in therapy will likely result in a generalization of learning to their other
memories. Therefore, choosing one trauma to assess is a suitable approach in
these cases.
Beyond assessing general symptoms of PTSD, it is often useful to formally
assess negative post-trauma cognitions because they are critical mediators of change
in PTSD symptoms in treatment (McLean et al., 2015a, 2015b). In other words,
reductions in negative cognitions often precede and lead to reductions in PTSD
symptoms. The Posttraumatic Cognitions Inventory (PTCI; Foa et al., 1999) is a
measure of three types of negative post-trauma cognitions, namely, negative cognitions about the self, negative cognitions about the world, and self-blame. This
measure has strong psychometric properties (Foa et al., 1999) and is available on
request (email foa@pennmedicine.upenn.edu).
Assessing Readiness for CBT for PTSD
During CBT for PTSD trainings, clinicians commonly request guidelines for assessing whether a given treatment is appropriate for the patient. Despite the abundance
of discussion in the literature on this topic, there are few studies well suited to
address this empirical question; however, the literature base on the topic is growing
(for relevant studies on suicide, see Cox et al., 2016; for relevant studies on alcohol
use, see Foa et al., 2013a). The challenge with identifying robust moderators of
treatment (the variables that dictate whether someone is likely to respond to
treatment or not) is that randomized controlled trials often have exclusion criteria
that limit variance in the sample. While we await the literature to expand in this
area, we present a few key readiness indicators that are likely to be relevant for the
average patient with PTSD.
The first indicator of readiness for CBT for PTSD is a willingness to focus on
PTSD, largely to the exclusion of other topic areas, for the duration of the treatment. Most CBT protocols are flexible to adapt to unexpected emergencies and
atypical circumstances, such as the death of a loved one or the breakup of a
romantic relationship. Deviation from the protocol is recommended as needed
during these rare circumstances to provide support. However, patients should be
willing to dedicate most of their treatment time to addressing PTSD. The second
indicator of readiness for CBT is willingness to engage in the treatment components,
which may become apparent only after discussing treatment expectations.
CBT for PTSD
All psychotherapy focused on treating PTSD, including CBT, can be emotionally
challenging; however, most patients can decide whether they are ready for the
treatment after they are provided with information about the treatment format,
content, and about PTSD in general. The third indicator of readiness for CBT is
whether the patient is reasonably unlikely to be retraumatized. This concern most
frequently, though not exclusively, arises in cases of domestic violence or familial
abuse. If there is a significant risk that the patient will be retraumatized regularly
(e.g., the patient is still living with an abusive partner or family member), the
patient’s immediate safety needs to be prioritized prior to engaging in CBT.
However, the therapist need not wait until conditions of guaranteed safety are
met, as this may be unachievable. For example, if a patient lives in an unsafe
neighborhood but is at no greater risk of exposure to trauma than their neighbors,
they could still be eligible for CBT. As untreated PTSD can increase risk-taking
behaviors (American Psychiatric Association, 2013), it may be very important to
proceed with treatment in such circumstances.
Additional considerations are required for some comorbid behaviors and diagnoses in determining readiness for CBT for PTSD. In therapist workshops, clinicians frequently ask about two common comorbidities or secondary conditions:
substance use and suicide risk. These are common concerns because individuals
with PTSD are significantly more likely both to abuse substances (Yalch et al., 2018)
and to report suicidal thoughts and behaviors (Brown et al., 2016, 2018) compared
with individuals without PTSD. Therefore, therapists providing CBT for PTSD
often need to consider the management of substance use and suicide risk. In terms
of substance use, a medical evaluation may be useful for determining whether the
patient requires medical detoxification prior to engaging in CBT. Otherwise, CBT
may be appropriate for patients who are willing to abstain from substance use for at
least two hours before and after engaging in therapy and homework practice. The
period of required abstinence may be longer depending on the substance of use. In
terms of suicide risk, patients who have engaged in nonsuicidal self-injurious behavior or have made a suicide attempt in the prior two months may be referred for
treatment focused on abstaining from these behaviors, including dialectical behavior
therapy (DBT; Linehan, 1993). Otherwise, therapists may consider generating a
crisis response plan (Bryan et al., 2017) with patients at the beginning of treatment
to identify sources of support, problem-solving, and distress tolerance skills to be
used as needed.
Monitoring Progress throughout CBT for PTSD
As with any CBT treatment, the therapist should monitor progress throughout
treatment. This typically involves assessment of PTSD symptoms, depression, and
key secondary targets (e.g., suicide risk, substance use, interpersonal conflict)
approximately every other session. Common measures that are used to this end
are the PDS-5 or PCL-5 and the Beck Depression Inventory-II (Beck et al., 1996).
Often, clinicians also assess negative post-trauma cognitions using the PTCI on a
regular basis in treatment. The therapist asks patients to arrive 10–15 minutes early
181
182
Lily A. Brown and Edna B. Foa
for session to complete the self-report measures, which the patient then scores and
discusses at the outset of the session.
Assessment Summary: Frequent, evidence-based assessment of PTSD and
related symptoms is a critical component of CBT for PTSD. Commonly used
measures include (1) for clinician interviews, the Clinician-Administered PTSD
Scale for DSM-5 (CAPS) and the PTSD Severity Scale Interview for DSM-5
(PSSI-5); (2) for self-report measures, the PTSD Diagnostic Scale for DSM-5
(PDS-5) and the PTSD Checklist for DSM-5 (PCL-5); and (3) for related targets:
the Beck Depression Inventory (BDI) and the Posttraumatic Cognitions
Inventory (PTCI).
Formulation
To illustrate a CBT formulation of PTSD, we provide the realistic but fictional case
vignette of Joyce. We present some background information, describe the process of
assessment and conceptualization of a treatment plan, and provide an example of
the socialization process to the therapeutic model. We opted to provide a formulation for a case that is appropriate for PE, though many features of this formulation
would extend to other variants of CBT for PTSD.
Background Information
Joyce is a married, 35-year-old pharmacist with two children under the age of 10.
She presented for treatment approximately three years following an armed robbery at her place of work. Joyce reported that one evening, a man and woman
entered the pharmacy; about five minutes later, the customers brandished loaded
guns and demanded opiates and money from the cash register. The perpetrators
threatened to kill Joyce and her assistant, Bill, if their demands were not met. At
some point during the robbery, Joyce attempted to press the emergency button
under the counter to alert the police. The female perpetrator witnessed Joyce’s
attempt to alert the authorities, and hit her over the head with a pistol. Joyce’s
head bled profusely as she waited for the perpetrators to collect the money and
pills. The robbers threatened Joyce and Bill that if they did not wait at least five
minutes to call the police, the robbers would return on a later date and kill them.
In addition to the pain of the assault, Joyce felt horrified and helpless. After the
robbers left, she broke down sobbing on the floor of the pharmacy from terror and
shock. Bill, who also appeared shocked and fearful, said nothing throughout the
robbery; he ran to the emergency button to contact the police as soon as the
robbers exited. Joyce remembered screaming, “What have you done?” to Bill as
he pressed the button and the alarm sounded. She reported feeling even more
panicked at this point for fear that the robbers would return to murder her and Bill
in retaliation.
CBT for PTSD
183
Table 10.1. Joyce’s baseline assessment scores
Date: December 1, 2017
Score
PTSD Severity Scale Interview for DSM-5 (PSSI-5)
PTSD Severity Scale for DSM-5 (PDS-5)
Beck Depression Inventory (BDI)
Posttraumatic Cognitions Inventory (PTCI)
60
55
32
140
Joyce telephoned the PTSD clinic requesting to discuss the aftermath of this
trauma. She reported that she was on medical leave from her job intermittently
due to fears of being retraumatized. When she was at work, she visually scanned
the store for signs of threat and positioned her workstation to be closer to the
emergency response button. Joyce identified that the feeling of not being safe at
work or other places was novel to her; this was a dramatic shift from her pretrauma expectations about danger and safety. Since the robbery, she struggled
with feeling constantly anxious and had extreme sleeping difficulties, which motivated her to seek help. The therapist agreed to meet with Joyce for a
formal evaluation.
At the time of the evaluation, Joyce’s therapist described that they would
complete a formal assessment to understand the ways in which Joyce’s life was
affected by the trauma. The therapist completed the PSSI-5 with Joyce. On this
measure, Joyce endorsed nearly every symptom. Beyond those already reported,
Joyce avoided being in crowded places, doing the shopping for the family, and
intimacy with her husband. She reported that there were times when she could not
control her emotions at work, resulting her “breaking down crying” in front of
customers and coworkers. She reported trying to distract herself constantly to avoid
remembering the trauma and “how close she had come to dying.” When she slept,
she was plagued by nightmares of the trauma and generally bad dreams. She also
endorsed feelings of self-blame and negative post-trauma cognitions. These
included beliefs that she should have acted more quickly to press the emergency
button, that she should have known that the two customers were dangerous as soon
as they entered the store. She also felt guilty for feeling angry with Bill for pressing
the emergency response button as soon as the robbers left the store. Finally, she
reported thinking that her perception of danger and safety had been warped since
the trauma, and that nowhere felt safe. Considering these negative trauma-related
cognitions, the therapist administered the PTCI to assess whether Joyce endorsed
other key areas of cognition alterations. Joyce’s baseline assessment scores are
provided in Table 10.1.
Formulation
From the perspective of emotional processing theory (EPT; Foa & Kozak, 1986),
the theoretical model from which PE therapy was derived, the robbery and
assault resulted in the formation of an emotional cognitive structure. This structure included negative associations between previously neutral stimuli (e.g., her
184
Lily A. Brown and Edna B. Foa
place of work, being in stores in general) and a sense of danger. Her trauma
structure also included negative self-perceptions stemming from her responses
during and after the trauma. Joyce expected that if she was at work or at another
store, she was very likely to be hurt again. This expectation contributed to her
avoidance of scenarios where she felt unsafe. Unfortunately, continuing to avoid
these situations also resulted in the maintenance of her fear and shame.
According to EPT and PE, PTSD symptoms are maintained because avoidance
(1) becomes habitual, (2) prevents learning that situations Joyce feels are dangerous are indeed safe, (3) prevents learning that anxiety will diminish over time,
and (4) promotes negative trauma-related cognitions. Relatedly, Joyce described
engaging in frequent avoidance of the memory of the trauma. Despite feeling
that she wanted to think of anything else, she described being constantly preoccupied by thoughts of the robbery. As a result, Joyce attempted to distract
herself with music, television, or browsing the internet – anything to keep her
from remembering the trauma. As with avoidance of real-world situations,
avoidance of the trauma memory prevents Joyce from emotionally processing
the trauma. According to EPT and PE, avoidance of the trauma memory results
in (1) a disorganized trauma memory, (2) confusion about the trauma recurring
versus being remembered, (3) missed opportunities for anxiety to diminish over
time, (4) the promotion of negative associations about the trauma and similar
events that result in altered perceptions of danger and safety, and (5) the
promotion of negative post-trauma cognitions.
Socialization to PE
When coming to a clear formulation of the factors that are maintaining Joyce’s
symptoms of PTSD, it is important to share this formulation with the patient. In so
doing, it is critical to provide a rationale justifying why the selected treatment, in this
case PE, is ideal for her. Therapists should ensure that the patient understands the
treatment model prior to formally beginning therapy. This can often be accomplished with a 15–20-minute conversation following the assessment, especially
because psychoeducation and rationale are provided on an ongoing basis on beginning the treatment. The following is an example of such a socialization conversation
between Joyce and her therapist.
Therapist:
Thank you for sharing the details of the robbery and the ways in
which it affects you today. It is very brave for you to come in and
talk about this. I know that you have told me how tempting it is to
pretend as if the trauma never happened; today, you took an
important first step in starting to process what happened to you.
In fact, talking about what you have been through is an important
component of prolonged exposure therapy, or PE, for PTSD.
I am going to tell you a bit about PE so that you can decide if you
are willing to give the therapy a try. Does that sound okay to you?
CBT for PTSD
Joyce:
Therapist:
Joyce:
Therapist:
185
Yes, I would like that. I have already tried so many things to
manage my PTSD, and none of it has worked. At this point, I am
ready to try anything.
It is very common for individuals to try to manage their PTSD on
their own for some time before coming to therapy. I know that in
your case, you have been suffering with this problem for over
three years, and I am very pleased to hear that you are open to
trying a new way of dealing with this.
For you to decide whether you want to start this therapy, you
first need to know what the therapy entails. I am going to give you
an overview of PE today and I will give you plenty of time to ask
any questions about it. You do not need to decide today whether
this is a therapy that you want to pursue; we can even meet again
to discuss it in more detail before you decide. If you do decide to
start PE, we will revisit some of the information that I am about
to share with you as we go through the treatment together.
Today, I will just briefly introduce you to the therapy.
PE is a time-limited therapy that is typically completed in eight
to fifteen ninety-minute treatment sessions. Occasionally, some
patients complete the treatment in fewer than eight sessions, and
occasionally some patients complete the treatment in more than
twelve sessions. To date, there are over twenty clinical trials that
demonstrate PE reduces PTSD and related symptoms, including
depression and relationship problems. Many of the people in PE
studies had similar symptoms to yours and were survivors of
assaults and robberies. For this reason, I am very hopeful that this
treatment will be very helpful for you.
I had assumed that most of the people that you have worked with
would be combat veterans – that is really all that I knew about
PTSD before the robbery. It is comforting to hear that people like
me have been through this before too.
Yes, I often hear that. In fact, PE was developed broadly for
individuals with PTSD stemming from a variety of traumatic
events. The treatment directly targets the core symptoms of
PTSD, regardless of the type of trauma that initiated the PTSD.
Throughout the treatment, you and I will work together to
identify the ways in which your life has changed since the trauma,
and we will plan for how to get your life headed in the direction that
you want. For instance, you told me that since the trauma, there are
many things that you no longer do, like going to work or shopping,
because you fear being hurt again. This is very common for
individuals with PTSD. In PE, you and I would use what we call “in
vivo exposure exercises.” These exercises will assist you in
186
Lily A. Brown and Edna B. Foa
approaching people, places, and situations that, despite being
objectively safe, have made you feel anxious since the trauma. We
will go at a pace that feels reasonable to you and I will guide you
through the process. We would also use imaginal exposure exercises
to help you to emotionally process the robbery. This is important
because while you try very hard not to think of the robbery, you also
told me that you find yourself having intrusive thoughts of it
frequently throughout the day. Imaginal exposure will help digest
the experience of the robbery, which is now unfinished business.
Both in vivo and imaginal exposures will help to reduce anxiety,
change negative beliefs that you have about yourself and the world,
and help to regain a sense of empowerment.
As we continue throughout the treatment, you will complete
practice exercises between sessions, which will include listening to
audio recordings of your therapy sessions and practicing in vivo
exposure exercises on your own. You will also complete
questionnaires every couple of sessions so that we can see how
your symptoms are changing over time. Do you have any
questions about this?
In some circumstances, scheduling one to three pre-treatment sessions can
provide an opportunity for rapport building, answering questions, or ensuring that
appropriate clinical management strategies are in place. For instance, the therapist
may consider using this time to construct a crisis response plan with a patient or to
orient them to monitoring of substance use in treatment, as relevant. These preparatory sessions can facilitate a smooth introduction into the treatment and help patients
decide whether they are interested and willing to participate in the treatment.
Formulation and Socialization Summary. It is critical to link your formulation of
the patient’s symptoms to the rationale for treatment. Provide a transparent but
brief description of what the patient can expect should they decide to complete the
treatment. Occasionally, it may be necessary to schedule one to three preparatory
sessions to build rapport and ensure that the patient is ready for treatment.
Overview of Therapy
Joyce’s therapy overview is presented in Table 10.2; this table includes information
about the session content, Joyce’s reaction to the content, and her progress. In
total, Joyce attended nine 90-minute sessions of PE for PTSD. Throughout the treatment, which included a combination of psychoeducation, breathing retraining, imaginal
exposure and processing, and in vivo exposure, Joyce reported significant habituation in
PTSD symptom severity, depression, and negative post-trauma cognitions.
CBT for PTSD
187
Table 10.2. Session content
Session
Content
Assessments
1: December
27, 2017
Psychoeducation
The therapist described that most individuals exposed to a trauma
have symptoms of PTSD in the immediate aftermath of the trauma,
but for many of them, those symptoms resolve on their own. Those
who go on to develop PTSD are often experts at avoiding reminders of
the trauma. Joyce’s examples include working, shopping, and thinking
about the trauma in general, among others.
Trauma history interview
Joyce provided enough details about her traumatic event to determine
the starting and stopping point of the trauma. She described the
physical, emotional, cognitive, and legal consequences of the trauma.
Breathing retraining
The therapist taught Joyce how to slow down her breathing.
Assigned homework
PDS-5 = 56
BDI = 31
PTCI = 140
1. Listen to audio recording of the full session.
2. Read treatment rationale.
3. Practice breathing retraining.
2: January 3,
2018
Homework review
Joyce listened to nearly the entire session audio recording and
practiced the breathing retraining twice. She also read the assigned
homework and asked a few questions about the material.
Common reactions to trauma
The therapist described the common reactions to trauma and discussed
the ones of high relevance to Joyce’s experience.
In vivo exposure rationale
The therapist described an in-depth rationale for using in vivo
exposure, drawing on examples from Joyce’s life experiences.
Introduction to subjective units of distress (SUDs)
The therapist explained the concept of SUDs to Joyce and identified
key anchor points. These anchors reflected times when Joyce’s distress
was at 100, 50, and 0. These anchor points are used to gauge future
distress ratings.
Construction of in vivo hierarchy
Joyce and her therapist generated a list of 12 items for her hierarchy
(see Table 10.3). The therapist queried about examples of strategies to
make each of those exercises more or less difficult. After generating
this list, Joyce and the therapist made SUDs ratings for each item on
the list. They selected an item that was rated as a 40 – going to a small
gift store in the early afternoon – to practice three times. They also
selected a behavioral activation item of taking a walk three times.
Assigned homework
1.
2.
3.
4.
5.
Listen to audio recording of the full session.
Read common reactions to trauma worksheet.
Practice breathing retraining.
Complete in vivo exposure homework exercise.
Review hierarchy and make alterations as needed.
188
Lily A. Brown and Edna B. Foa
Table 10.2. (cont.)
Session
Content
Assessments
3: January 10,
2018
Homework review
Joyce listened to the entire session audio recording and practiced the
breathing retraining one. She completed her first in vivo exercise three
times, and the second exercise twice. She reviewed her hierarchy and
added two items to the list. She read through the common reactions to
trauma worksheet. The therapist provided her with a lot of praise for
completing the exercises as prescribed.
Imaginal exposure rationale
The therapist provided an in-depth rationale for imaginal exposure.
Joyce asked questions about this and reported feeling very nervous but
willing to try it.
Imaginal exposure
Joyce completed an imaginal exposure exercise for 41 minutes. She
repeated the exposure three times. Her initial rendition was somewhat
disconnected and devoid of emotional expression, but she became
tearful throughout the second rendition. Her tearfulness subsided by
the third rendition.
Processing
The therapist praised Joyce for her bravery in conducting the imaginal
exposure. She asked open-ended questions to explore Joyce’s
emotional reaction to the imaginal exposure. Joyce described feeling
terrified during the robbery and angry at her coworker for not
following the assailants’ orders. Core cognitions that emerged from the
processing centered on beliefs such as “I am helpless” and “This could
happen to me at any time.”
Homework Assigned
PDS-5 = 52
BDI = 31
PTCI = 137
Pre-imaginal
SUDs = 88
Peak imaginal
SUDs = 95
Post-imaginal
SUDs = 80
1.
2.
3.
4.
4: January 17,
2018
Listen to audio recording of the full session.
Practice breathing retraining.
Complete in vivo exposure homework exercise.
Listen to imaginal exposure daily.
Homework review
Joyce listened to the entire session audio-recording but did not practice her
breathing retraining. She completed her assigned in vivo exercise four
times: peak SUDs scores were 75, 70, 70, and 69. She listened to the
imaginal exposure on two additional occasions beyond the full tape review,
for a total of three times. Her peak SUDs ratings were 89, 88, 86, and 86.
Imaginal exposure
Joyce completed an imaginal exposure exercise for 40 minutes. As with
session 3, she repeated the exposure three times. She was tearful
throughout the first rendition, but less tearful during the second and third
rendition.
Processing
The therapist praised Joyce again. As usual, she asked open-ended
questions to explore Joyce’s emotional reaction to the imaginal exposure.
Joyce reported some surprise that the exercise was not as difficult as on the
prior session.
Homework assigned
See session 3.
Pre-imaginal
SUDs = 80
Peak imaginal
SUDs = 85
Post-imaginal
SUDs = 69
CBT for PTSD
189
Table 10.2. (cont.)
Session
Content
Assessments
5: January 24,
2018
Homework review
Joyce completed all her homework. For in vivo exposure, her peak
SUDs scores were 65, 65, 60, and 59. For imaginal exposure, her peak
SUDs scores were 80, 65, and 60.
Imaginal exposure
Joyce completed an imaginal exposure exercise for 40 minutes. As in
prior sessions, she repeated the exposure three times.
Processing
Key themes discussed in processing included habituation of distress
over time and Joyce’s shifting sense of threat versus safety.
Homework assigned
See sessions 3 and 4.
PDS-5 = 35
BDI = 19
PTCI = 115
Pre-imaginal
SUDs = 65
Peak imaginal
SUDs = 75
Post-imaginal
SUDs = 50
6: January 31,
2018
Homework review
Joyce completed most of her homework. For in vivo exposure, her
peak SUDs scores were 55 and 40. For imaginal exposure, her peak
SUDs scores were 65, 40, and 35.
Imaginal exposure: Hot spots
Joyce completed her first hot spot imaginal exposure, wherein a select
portion of the most difficult part of the trauma memory was repeated
instead of the full trauma memory. She selected one portion of the memory
that was approximately four minutes long and repeated it 10 times.
Processing
Key themes discussed in processing included beliefs about why she had not
acted more quickly to press the emergency response button.
Homework assigned
See sessions 3–5.
Pre-imaginal
SUDs = 45
Peak imaginal
SUDs = 65
Post-imaginal
SUDS = 50
7: February 7,
2018
Homework review
Joyce completed all her homework. For in vivo exposure, her peak
SUDs scores were 40, 40, and 35. For imaginal exposure, her peak
SUDs scores were 50, 40, and 30.
Imaginal exposure: Hot spots
Joyce completed her second round of hot spot imaginal exposure.
Processing
The key theme discussed in processing was the observation that she
has moved on in her life in a positive direction.
Homework assigned
See sessions 3–6.
PDS-5 = 15
BDI = 15
PTCI = 101
Pre-imaginal
SUDs = 30
Peak imaginal
SUDs = 35
Post-imaginal
SUDs = 20
8: February
14, 2018
Homework review
Joyce completed most of her homework. For in vivo exposure, her
peak SUDs scores were 30, 20, and 15. For imaginal exposure, her
peak SUDs scores were 30, 30, and 20.
Imaginal exposure: Hot spots
Joyce completed her third round of hot spot imaginal exposure.
Processing
The key theme for discussion was Joyce’s readiness to terminate
therapy, and the evidence that she has made significant progress in her
symptoms.
Homework assigned
See sessions 3–7.
Pre-imaginal
SUDs = 25
Peak imaginal
SUDs = 30
Post-imaginal
SUDs = 15
190
Lily A. Brown and Edna B. Foa
Table 10.2. (cont.)
Session
Content
Assessments
9: February
21, 2018
Homework review
Joyce completed all her homework. For in vivo exposure, her peak
SUDs scores were 20, 10, and 5. For imaginal exposure, her peak
SUDs scores were 20, 15, and 15.
Imaginal exposure
Joyce completed one final rendition of the full trauma memory from
start to finish.
Processing
Joyce reflected on the difference in emotional upset and negative
cognitions from the beginning of treatment to this point in therapy.
Progress review and thinking ahead
Joyce reported that she feels ready to return to work and can live her
life as though she had not been through the robbery/ assault. She
reflected on strategies that she needs to continue practicing. Joyce and
her therapist terminated therapy.
PDS-5 = 9
BDI = 10
PTCI = 75
Pre-imaginal
SUDs = 20
Peak imaginal
SUDs = 25
Post-imaginal
SUDs = 10
Table 10.3. Joyce’s in vivo hierarchy
Item
Exposure
SUDs
Session
1
2
3
4
5
5
6
7
8
9
10
Going to a small gift store, 2 p.m.
Talking a walk around her neighborhood, noon
Watching a TV show about a crime
Going to a busy store in a safe neighborhood, 3 p.m.
Sitting in a restaurant, in the middle of the room, during lunch
Going to a pharmacy in a safe neighborhood, 2 p.m.
Going to her work pharmacy at lunch
Watching a video of a pharmacy robbery
Taking a bus around town
Going to her work pharmacy in the evening
Going to a new pharmacy in the evening
40
45
50
55
60
60
70
70
80
90
95
2
2
3
3
4
4
5
5
6
6
7, 8
Summary: In following the PE manual, the therapist offered psychoeducation,
breathing retraining, in vivo exposure, and imaginal exposure in the treatment of
Joyce’s PTSD. Joyce reported significant symptom resolution throughout PE.
FOR ADDITIONAL READING
Foa, E. B., Hembree, E. A., & Rothbaum, B. O. (2007). Prolonged exposure therapy for
PTSD: Emotional processing of traumatic experiences therapist guide (Treatments that
work). Oxford University Press.
CBT for PTSD
REFERENCES
Acierno, R., Knapp, R., Tuerk, P., Gilmore, A. K., Lejuez, C., Ruggiero, K., . . . & Foa, E. B.
(2017). A non-inferiority trial of prolonged exposure for posttraumatic stress disorder: In
person versus home-based telehealth. Behaviour Research and Therapy, 89, 57–65.
doi:10.1016/j.brat.2016.11.009
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Asukai, N., Saito, A., Tsuruta, N., Kishimoto, J., & Nishikawa, T. (2010). Efficacy of exposure
therapy for Japanese patients with posttraumatic stress disorder due to mixed traumatic
events: A randomized controlled study. Journal of Traumatic Stress, 23(6), 744–750.
doi:10.1002/jts.20589
Beck, A. T., Steer, R. A., & Brown, G. K. (1996). Manual for the Beck Depression InventoryII. Psychological Corporation.
Blevins, C. A., Weathers, F. W., Davis, M. T., Witte, T. K., & Domino, J. L. (2015). The
Posttraumatic Stress Disorder Checklist for DSM-5 (PCL-5): Development and initial
psychometric evaluation. Journal of Traumatic Stress, 28(6), 489–498. doi:10.1002/jts.22059
Brown, L. A. Armey, M. A., Sejourne, C., Miller, I. W., & Weinstock, L. (2016). Suicide risk
in psychiatric inpatients with PTSD. Psychiatry Research, 243, 191–197.
Brown, L. A., Fernandez, C., Kohn, R., Saldivia, S., & Vincente, B. (2018). Pre-disaster PTSD
as a moderator of the relationship between natural disaster and suicidal ideation over
time. Journal of Affective Disorders, 230, 7–14.
Bryan, C. J., Clemans, T. A., Hernandez, A. M., Mintz, J., Peterson, A. L., Yarvis, J. S., &
Resick, P. A. (2016). Evaluating potential iatrogenic suicide risk in trauma-focused group
cognitive behavioral therapy for the treatment of PTSD in active duty military personnel.
Depression and Anxiety, 33(6), 549–557. doi:10.1002/da.22456
Bryan, C. J., Mintz, J., Clemans, T. A., Leeson, B., Burch, T. S., Williams, S. R., Maney, E., &
Rudd, M. D. (2017). Effect of crisis response planning vs. contracts for safety on suicide
risk in U.S. Army Soldiers: A randomized clinical trial. Journal of Affective Disorders, 212,
64–72.
Carpenter, J. K., Andrews, L. A., Witcraft, S. M., Powers, M. B., Smits, J. A. J., & Hofmann,
S. G. (2018). Cognitive behavioral therapy for anxiety and related disorders: A metaanalysis of randomized placebo-controlled trials. Depression and Anxiety. doi:10.1002/
da.22728
Cox, K. S., Mouilso, E. R., Venners, M. R., Defever, M. E., Duvivier, L., Rauch, S. A., . . . &
Tuerk, P. W. (2016). Reducing suicidal ideation through evidence-based treatment for
posttraumatic stress disorder. Journal of Psychiatric Research, 80, 59–63.
Cusack, K., Jonas, D. E., Forneris, C. A., Wines, C., Sonis, J., Middleton, J. C., . . . &
Gaynes, B. N. (2016). Psychological treatments for adults with posttraumatic stress
disorder: A systematic review and meta-analysis. Clinical Psychology Review, 43,
128–141.
Ehlers, A., & Clark, D. M. (2000). A cognitive model of posttraumatic stress disorder.
Behaviour Research and Therapy, 38(4), 319–345. doi:10.1016/S0005-7967(99)00123-0
Foa, E. B., Asnaani, A., Rosenfield, D., Zandberg, L. J., Gariti, P., & Imms, P. (2017).
Concurrent varenicline and prolonged exposure for patients with nicotine dependence
and PTSD: A randomized controlled trial. Journal of Consulting and Clinical
Psychology, 85, 862–872. doi:10.1037/ccp0000213
Foa, E. B., Hembree, E. A., & Rothbaum, B. O. (2007). Prolonged exposure therapy for
PTSD: Emotional processing of traumatic experiences – Therapist guide. Oxford
University Press.
Foa, E. B., & Kozak, M. J. (1986). Emotional processing of fear: Exposure to corrective
information. Psychological Bulletin, 99(1), 20–35.
191
192
Lily A. Brown and Edna B. Foa
Foa, E. B., McLean, C. P., Capaldi, S., & Rosenfield, D. (2013b). Prolonged exposure vs
supportive counseling for sexual abuse–related PTSD in adolescent girls: A randomized
clinical trial. JAMA, 310(24), 2650–2657. doi:10.1001/jama.2013.282829
Foa, E. B., McLean, C. P., Zang, Y., Rosenfield, D., Yadin, E., Yarvis, J. S., . . . & Peterson,
A. L. (2018). Effect of prolonged exposure therapy delivered over 2 weeks vs 8 weeks vs
present-centered therapy on PTSD symptom severity in military personnel:
A randomized clinical trial. JAMA, 319(4), 354–364. doi:10.1001/jama.2017.21242
Foa, E. B., McLean, C. P., Zang, Y., Zhong, J., Rauch, S., Porter, K., . . . & Kauffman, B. Y.
(2016a). Psychometric properties of the Posttraumatic Stress Disorder Symptom Scale
Interview for DSM-5 (PSSI-5). Psychological Assessment, 28(10), 1159–1165.
doi:10.1037/pas0000259
Foa, E. B., McLean, C. P., Zang, Y., Zhong, J., Powers, M. B., Kauffman, B. Y., . . . &
Knowles, K. (2016b). Psychometric properties of the Posttraumatic Diagnostic Scale for
DSM-5 (PDS-5). Psychological Assessment, 28(10), 1166–1171. doi:10.1037/pas0000258
Foa, E. B., Rothbaum, B. O., Riggs, D. S., & Murdock, T. B. (1991). Treatment of
posttraumatic stress disorder in rape victims: A comparison between cognitivebehavioral procedures and counseling. Journal of Consulting and Clinical Psychology,
59(5), 715–723.
Foa, E. B., Tolin, D. F., Ehlers, A., Clark, D. M., & Orsillo, S. M. (1999). The posttraumatic
cognitions inventory (PTCI): Development and validation. Psychological Assessment, 11,
303–314.
Foa, E. B., Yusko, D. A., McLean, C. P., et al. (2013a). Concurrent naltrexone and prolonged
exposure therapy for patients with comorbid alcohol dependence and PTSD:
A randomized clinical trial. JAMA, 310(5), 488–495. doi:10.1001/jama.2013.8268
Grubaugh, A. L., Veronee, K., Ellis, C., Brown, W., & Knapp, R. G. (2017). Feasibility and
efficacy of prolonged exposure for PTSD among individuals with a psychotic spectrum
disorder. Frontiers in Psychology, 8, 977. doi:10.3389/fpsyg.2017.00977
Linehan, M. (1993). Cognitive behavior treatment for borderline personality disorder. Guilford
Press.
McLean, C. P., Su, Y. J., & Foa, E. B. (2015b). Mechanisms of symptom reduction in a
combined treatment for comorbid posttraumatic stress disorder and alcohol dependence.
Journal of Consulting and Clinical Psychology, 83(3), 655–661. doi:10.1037/ccp0000024
McLean, C. P., Yeh, R., Rosenfield, D., & Foa, E. B. (2015a). Changes in negative cognitions
mediate PTSD symptom reductions during client-centered therapy and prolonged exposure for adolescents. Behaviour Research and Therapy, 68, 64–69. doi:10.1016/j.
brat.2015.03.008
Powers, M. B., Halpern, J. M., Ferenschak, M. P., Gillihan, S. J., & Foa, E. B. (2010). A metaanalytic review of prolonged exposure for posttraumatic stress disorder. Clinical
Psychology Review, 30(6), 635–641. doi:10.1016/j.cpr.2010.04.007
Ragsdale, K. A., & Voss Horrell, S. C. (2016). Effectiveness of prolonged exposure and
cognitive processing therapy for U.S. veterans with a history of traumatic brain injury.
Journal of Traumatic Stress, 29(5), 474–477. doi:10.1002/jts.22130
Resick, P. A. (1992). Cognitive treatment of crime-related post-traumatic stress disorder. In
R. D. Peters, R. J. McMahon, & V. L. Quinsey (Eds.), Aggression and violence throughout the life span (pp. 171–191). Sage Publications.
Resick, P. A., Nishith, P., Weaver, T. L., Astin, M. C., & Feuer, C. A. (2002). A comparison of
cognitive-processing therapy with prolonged exposure and a waiting condition for the
treatment of chronic posttraumatic stress disorder in female rape victims. Journal of
Consulting and Clinical Psychology, 70(4), 867–879.
Resick, P. A., & Schnicke, M. K. (1992). Cognitive processing therapy for sexual assault
victims. Journal of Consulting and Clinical Psychology, 60(5), 748–756.
CBT for PTSD
Resick, P. A., Wachen, J. S., Dondanville, K. A., Pruiksma, K. E., Yarvis, J. S., Peterson, A. L., . . .
& Young-McCaughan, S. (2017). Effect of group vs individual cognitive processing therapy
in active-duty military seeking treatment for posttraumatic stress disorder: A randomized
clinical trial. JAMA Psychiatry, 74(1), 28–36. doi:10.1001/jamapsychiatry.2016.2729
Ruglass, L. M., Lopez-Castro, T., Papini, S., Killeen, T., Back, S. E., & Hien, D. A. (2017).
Concurrent treatment with prolonged exposure for co-occurring full or subthreshold
posttraumatic stress disorder and substance use disorders: A randomized clinical trial.
Psychotherapy and Psychosomatics, 86(3), 150–161. doi:10.1159/000462977
Schiff, M., Nacasch, N., Levit, S., Katz, N., & Foa, E. B. (2015). Prolonged exposure for
treating PTSD among female methadone patients who were survivors of sexual abuse in
Israel. Social Work Health Care, 54(8), 687–707. doi:10.1080/00981389.2015.1058311
Schnurr, P. P., Shea, M. T., Friedman, M. J., & Engel, C. C. (2007). Posttraumatic stress
disorder and cognitive behavioral therapy – In reply. JAMA, 297(24), 2965.
Shapiro, F. (2001). Eye movement desensitization and reprocessing (EMDR): Basic principles,
protocols, and procedures (2nd ed.). Guilford Press.
Sijbrandij, M., Kunovski, I., & Cuijpers, P. (2016). Effectiveness of internet-delivered cognitive behavioral therapy for posttraumatic stress disorder: A systematic review and metaanalysis. Depression and Anxiety, 33(9), 783–791. doi:10.1002/da.22533
Sloan, D. M., Marx, B. P., Lee, D. J., & Resick, P. A. (2018). A Brief exposure-based
treatment vs cognitive processing therapy for posttraumatic stress disorder:
A randomized noninferiority clinical trial. JAMA Psychiatry. doi:10.1001/
jamapsychiatry.2017.4249
Valentine, S. E., Borba, C. P., Dixon, L., Vaewsorn, A. S., Guajardo, J. G., Resick, P. A., . . .
& Marques, L. (2017). Cognitive processing therapy for Spanish-speaking Latinos:
A formative study of a model-driven cultural adaptation of the manual to enhance
implementation in a usual care setting. Journal of Clinical Psychology, 73(3), 239–256.
doi:10.1002/jclp.22337
Weathers, F. W., Blake, D. D., Schnurr, P. P., Kaloupek, D. G., Marx, B. P., & Keane, T. M.
(2013a). The Clinician-Administered PTSD Scale for DSM-5 (CAPS-5). Available from
the National Center for PTSD at www.ptsd.va.gov.
(2013b). The Life Events Checklist for DSM-5 (LEC-5). Available from the National
Center for PTSD at www.ptsd.va.gov.
Wolf, G. K., Kretzmer, T., Crawford, E., Thors, C., Wagner, H. R., Strom, T. Q., . . . &
Vanderploeg, R. D. (2015). Prolonged exposure therapy with veterans and active duty
personnel diagnosed with PTSD and traumatic brain injury. Journal of Traumatic Stress,
28(4), 339–347. doi:10.1002/jts.22029
Yalch, M. M., Hebenstreit, C. L., & Maguen, S. (2018). Influence of military sexual assault
and other military stressors on substance use disorder and PTS symptomology in female
military veterans. Addictive Behaviors, 80, 28–33. doi:10.1016/j.addbeh.2017.12.026
193
PART THREE
COGNITIVE BEHAVIORAL
THERAPY FOR OBSESSIVECOMPULSIVE DISORDER AND
ASSOCIATED DISORDERS
11
Obsessive-Compulsive Disorder: An Updated
Cognitive Behavioral Approach
Oliver Sündermann and David Veale
Introduction
Obsessive-compulsive disorder (OCD) is the fourth most common mental disorder
with a lifetime prevalence of 2% (Kessler et al., 2005). The WHO ranks OCD among
the 10 most debilitating disorders (2008); OCD affects all cultural groups and is
associated with poor quality of life, unemployment, social exclusion, stigma, and suicide
risk (Eisen et al., 2006; Hollander & Benzaquen, 1996). Onset tends to be in adolescence or early adulthood, and without adequate treatment, OCD usually takes a
chronic course (Fineberg et al., 2013). OCD is poorly recognized, and on average
people struggle for 10 years until they receive appropriate help (Hollander et al., 1997).
Diagnostic Criteria and Changes in ICD11 and DSM-5
OCD is characterized by the presence of obsessions and/or compulsions, which are
time-consuming or cause distress or impairment in various areas of functioning.
Obsessions are defined as ‘recurrent and intrusive, urges, or images’, and compulsions are repetitive behaviors or mental acts aimed at preventing or reducing
distress caused by the obsessions (DSM-5; American Psychiatric Association,
2013). Obsessions are usually ego-dystonic and recognized as the product of one’s
own mind. Common obsessions refer to preventing harm to self and others from
contamination (e.g., dirt, germs, bodily fluids, chemicals) or from making a mistake
(e.g., not turning off the gas taps); intrusive sexual, religious, or violent thoughts;
and obsessions of incompleteness, order, and symmetry. DSM-5 and ICD11 have
‘shifted’ OCD from the anxiety disorders to a new chapter, ‘Obsessive-Compulsive
and Related Disorders’, grouped alongside other disorders with seemingly similar
phenomenology of repetitive behaviors such as body dysmorphic disorder, trichotillomania, and hypochondriasis. We regard this regrouping as problematic because
the groups consist of repetitive behaviors that are only superficially similar (e.g., tics,
hair pulling, compulsive checking) but have very different functions. Other anxietyreduction strategies are not captured adequately (e.g., mental neutralizing, avoidance) (Abramowitz & Jacoby, 2014).
197
198
Oliver Sündermann and David Veale
CBT Model of OCD
A cognitive-behavioral approach to OCD is based on the observation that intrusive
thoughts, urges, and images are normal experiences (Rachman & de Silva, 1978 that
cause distress through appraisals of personal significance (‘Having bad thoughts
means, I am bad’), control (‘I must control my thoughts’), and responsibility (‘I am
responsible for preventing harm’) (Salkovskis, 1999). Hence, the difference between
a normal intrusion and a clinical obsession lies in the way the person with OCD
interprets these and responds to them. Compulsive rituals (covert and overt),
avoidance, safety seeking, and neutralizing behaviors (e.g., removing/replacing a
thought) are conceptualized as attempts to control or remove intrusions and to avert
feared consequences, all of which maintain obsessional distress through negative
reinforcement, preventing disconfirmation of threat beliefs, and increase frequency
of obsessions because rituals and avoidance act as constant reminders of their
occurrence. While environmental and biological factors may increase vulnerability
to acquire obsessional concerns (Taylor & Jang, 2011), compulsive responding to
them and cognitive biases maintain OCD and are therefore the target in CBT
(Abramowitz, 2006).
Outcome Research and New Avenues
Selective serotonin reuptake inhibitors (SSRIs) are the recommended pharmacological treatment for OCD, but gains typically subside when discontinued (Soomro
et al., 2008). The psychological treatment of choice for OCD remains CBT, which
refers to exposure and response prevention (E/RP) either with or without cognitive
interventions. A recent meta-analysis by Öst et al. (2015) (n = 37 RCTs) found CBT
the only effective psychological treatment for OCD, with an overall moderate effect
(0.57) on the Yale–Brown Obsessive Compulsive Scale (Y-BOCS) changes from
pre- to post-treatment. There were very large effect sizes in favour of CBT compared with wait-list (1.31) and placebo conditions (1.33), with minimal or no differences between individual and group treatments. Findings are generally in line with
previous meta-analyses (Abramowitz, 1996; Olatunji et al., 2013; Rosa-Alcázar
et al., 2008). Long-term outcome data are limited but promising, with some studies
showing that gains from E/RP can be maintained at one year (Olatunji et al., 2013)
and at five years (van Oppen et al., 2005).
While CBT clearly is an effective intervention for OCD, up to 50% of individuals with OCD do not benefit from CBT, drop out of treatment, or find treatment,
especially E/RP, too challenging. Around 75% live with residual symptoms after
completing treatment (Abramowitz et al., 2003; Foa et al., 2005; Rufer et al., 2006;
Tolin et al., 2004). It also remains unclear how much comorbidity (Öst et al., 2015),
heterogeneity of symptoms (Sookman & Fineberg, 2015), and complexity require
treatment adaptations (Sündermann & Veale, 2017). CBT-related research has
advanced significantly over the last 10 years, and we will briefly describe four areas
that we consider promising in informing OCD treatment, namely, (1) an inhibitory
Obsessive-Compulsive Disorder
learning approach to exposure therapy, (2) disgust in OCD, (3) imagery in OCD,
and (4) complex OCD and specialized care.
Exposure Therapy and Inhibitory Learning
For many years E/RP was theoretically underpinned by emotional processing
theory (Foa & Kozak, 1986) according to which conditionally learned obsessional
fears are negatively reinforced and perpetuated through avoidance and compulsions. Consequently, the focus in E/RP had been on fear habituation. However, the
concept of fear habituation is scientifically problematic because successful habituation of non-OCD fears fails to predict long-term outcomes, and, conversely,
positive long-term outcomes can occur in the absence of habituation during E/RP
(Craske et al., 2012, 2014). Applying updated knowledge on basic learning and
memory processes, inhibitory learning has been suggested as the core mechanism in
extinction (Bouton, 1993; Bouton et al., 2006; Vervliet et al., 2013), according to
which the originally learned associations between conditioned and unconditioned
stimulus remains intact. Therefore, the goal of E/RP should be to enhance inhibitory learning, through maximizing the likelihood that non-danger associations (e.g.,
‘Obsessions are meaningless’; ‘Uncertainty is tolerable’) inhibit access and retrieval
of threat associations (e.g., ‘Obsessions are unacceptable’; ‘Uncertainty is intolerable’). Principles of inhibitory learning that facilitate extinction involve maximal
violation of expectancies of feared outcomes, eliminating safety-seeking behaviors,
including retrieval cues, varying the context and time of E/RP (e.g., exposures offsite and at different times), and reconsolidating memory of adverse events that
relate to the onset of OCD (Craske et al., 2014). Optimizing E/RP for OCD using
inhibitory learning principles is promising (Arch & Abramowitz, 2015) but requires
further evaluation.
Disgust in OCD
Disgust is a central but understudied emotion in OCD, and generally E/RP outcome
in disgust-OCD is poorer than in fear-OCD, with smaller, slower, and more temporary effects (Mason & Richardson, 2010; Olatunji et al., 2009). Disgust differs
from fear with regard to phenomenology, acquisition, and maintenance. For
example, a case series of 11 residential patients with contamination-related OCD
found that exposure with disgust-inducing stimuli was associated with an increased
vagal tonus (Duncko & Veale, 2016), contrary to the concept of adrenergic activation of heightened fear. Disgust-avoidance has also been conceptualized as a
distinct motivational ‘core dimension’ to harm-avoidance (Melli et al., 2015) in
contamination-related OCD. Disgust may be acquired through evaluative conditioning where the reference between conditioned and unconditioned stimulus is
learned rather than the expectancy of the occurrence of the unconditioned stimulus
(Schienle et al., 2001; Woody & Teachman, 2000). These functional differences
between evaluative and fear conditioning may require alternative intervention
199
200
Oliver Sündermann and David Veale
strategies, and it was recently proposed that counter-conditioning (pairing the
conditioned stimulus with an unconditioned stimulus of opposite/pleasant valence)
and unconditioned stimulus revaluation (pairing the unconditioned stimulus with
another unconditioned stimulus of opposite/pleasant valence) may be more effective in reducing disgust (Ludvik et al., 2015). Disgust is also related to the experience
of mental contamination, the feeling of pollution or dirtiness in the absence of a
physical contaminant (Rachman, 1994, 2006). Cognitive interventions appear more
effective than E/RP in treating mental contamination (Coughtrey et al., 2013).
Mental contamination is often linked with past trauma, particularly those involving
moral violation (humiliation, betrayal, sexual, physical, or emotional harm)
(Rachman, 2010; Zysk et al., 2018); thus, trauma-focused approaches (e.g., imagery
rescripting) may enhance outcome of CBT for OCD, particularly when past aversive experiences continue to affect the person.
Imagery in OCD
People with OCD experience intrusive images (Speckens et al., 2007), and these can
be linked emotionally with past fear-, disgust-, or shame-based experiences that
continue to haunt the person. For example, a person who developed mental contamination in response to childhood sexual abuse may continue to experience
disgust-based memories and sensations that drive attentional processes to threat,
avoidance, and decontamination rituals. When intrusive images or sensations are
linked with earlier trauma, these need to be addressed in treatment, in addition to
cognitive and behavioral interventions. A recent single-case experimental case
series (n = 12; Veale et al., 2015) found that a single session of imagery rescripting
in patients with images linked to aversive memories was associated with a drop on
the Y-BOCS scale from 24.1 pre-treatment to 10.7 at three-month follow-up.
Complex OCD and Specialized Services
Clients with OCD often present with a multitude of difficulties, and are sometimes
described as ‘complex’. Although the term ‘complex OCD’ is widely used, the
literature lacks a clear definition and guidelines for treatment. We therefore
recently described, in a case series, how complexity in OCD arises, the challenges
various dimensions of complexity pose to therapy, how they are inter-linked, and
how best to work with them (Sündermann & Veale, 2017). Complexity can be
incurred through non-specific factors (language and culture barriers, unemployment, cognitive disabilities, etc.), and more specifically through comorbid problems,
often in the context of personality and interpersonal difficulties, dissociation, and
trauma. ‘Complexity’ is not the same as ‘severity’ or ‘chronicity’, but is more likely
to occur in those with long-standing OCD with multiple treatment failures. Complex
OCD does not require ‘complex’ treatment, but should be formulated by understanding and processing relevant past experiences, the context and the function of
Obsessive-Compulsive Disorder
OCD behaviors, and drawing on compassion-focused techniques, and will likely
require a multidisciplinary approach.
Clients with severe and/or complex OCD or those for whom adequate treatments have failed (CBT and/or SSRI) may require ‘stepping up’ to secondary or
tertiary care services (see NICE, 2006). Residential or inpatient settings with
specialized staff and the resources to provide intensive CBT may be indicated when
there is risk of suicide, severe neglect, or low body weight or when rituals are found
to be too debilitating (e.g., 24-hour shower rituals) or risky (e.g., drinking bleach)
for outpatient services. Specialized residential and inpatient clinics should provide a
caring and safe therapeutic community modelled on compassion-focused and social
learning principles (Veale et al., 2014), encouraging clients to provide
mutual support.
Assessment and Treatment of OCD
Assessment and treatment of OCD can take place in many different settings and
contexts, and depends on various factors such as the nature, severity, complexity,
and subtype of the OCD; the availability of resources; accessibility of treatment; and
the client’s motivation for change (McKay et al., 2015). Therapy needs to be
culturally sensitive, and although OCD themes tend to be similar across cultures
and societies (Clark & Radomsky, 2014; Radomsky et al., 2014), religious and
sexual intrusions in particular vary with regard to themes of homosexuality, selfpurification, premarital sex, and prescribed prayers (Sookman, 2015). Table 11.1
gives an overview of a CBT protocol for assessment and treatment of OCD,
including core measures for each therapy stage.
Assessment
The main purpose of the assessment is to gain a shared understanding of how the
client’s OCD developed and how it is maintained. Assessment should be developmentally adapted, multidimensional, and include self-reports of subjective experiences of obsessions and compulsion, corroborated by clinical interviews and
standardized self-report measures, as applicable to the client’s presenting OCD
(Sookman, 2015); for example, someone suffering from mental contamination could
complete
the
Vancouver
Obsessional
Compulsive
Inventory–Mental
Contamination Scale (VOCI-MC) (Rachman, 2005).
Screening for OCD
The National Institute for Health and Clinical Excellence (NICE) provides the
following guidance on questions for OCD screening:
Do you wash or clean a lot?
Do you check things a lot?
201
202
Oliver Sündermann and David Veale
Table 11.1. A cognitive behavioral assessment and treatment protocol for OCDa
Session
Therapy stage
Key elements and aims
1 and 2
Assessment and
formulation
Screening for OCD
Clarify hopes, expectations, motivation, readiness for change
Define the problem; set specific, measurable, attainable, realistic, and
time-based (SMART) goals (short, medium, long)
Obtain history and relevant early experiences
Assess nature of obsessions and compulsions (content, context,
distress, appraisals, emotions)
Psychoeducation and normalization of unwanted thoughts, urges,
images
Assessment of OCD-related imagery
Theory A/Theory B: build alternative understanding of the problem
Formulation: Identify main appraisals of obsessions, emotions,
imagery, and coping responses
Home-based assessment (if indicated and feasible)
Self-monitoring to enhance awareness of obsessions and compulsions
(e.g., using diaries, tally counter)
Involve key significant others (e.g., partner, parents)
3–6
Work phase 1
Therapy-assisted and modelled behavioral experiments and exposures
in session, off-site, and home-based (if feasible)
Rehearse E/RP principles, including ‘going beyond’ anti-OCD
behaviors (i.e., not simply resisting rituals, but actively
triggering OCD)
Cognitive restructuring and Socratic dialogues to target obsessional
appraisals and reflect on exposure outcomes
Relate all therapy work to Theory A/Theory B and obtain regular
belief ratings in both theories
Use of metaphors is helpful (‘OCD as bully’; ‘Digging yourself out of a
hole’; ‘Ghosts from the past’; etc.)
Strong emphasis on homework tasks
Imagery rescripting (if indicated)
Home visits and in vivo off-site experiments (if indicated, do as quickly
as possible)
7–10
Work phaseb
Space out sessions to agreed frequency, e.g. biweekly, monthly
More emphasis on independent planned exposures: ‘Client becoming
their own therapist’
11 and 12
Termination
13–15
Follow-up
Review of progress
a
Generalization of experiments/exposures
Review of treatment
Dealing with setbacks
Blueprint (‘Staying well plan’)
Protocol can/should be adapted according to severity, complexity, heterogeneity of OCD, and availability
of resources.
b
Consider specific-subtype measures if indicated, e.g., for VOCI-MC mental contamination; monitor mood/anxiety/
quality of life and other difficulties as required.
Obsessive-Compulsive Disorder
Is there any thought that keeps bothering you that you would like to get rid of
but cannot?
Do your daily activities take a long time to finish?
Are you concerned about putting things in a special order or are you very upset
by mess?
Do these problems trouble you?
If the client answers any of the above affirmatively, a more thorough clinical
assessment should be conducted.
Hopes, Expectations, and Problem Definition
A good clinical assessment should start by clarifying the client’s hopes and expectations for treatment. The therapist should also inquire about any ambivalence about
seeking help. Does the client feel pressured to seek treatment, for example, by their
partner or family, or are they sent or referred by someone else, for example, their
general practitioner? The therapist may consider asking why they are seeking help
now, what they hope to gain, and how they feel about seeing a therapist. Clients may
feel ashamed about their OCD, and find it difficult to discuss their obsessions or
compulsions. Normalization and psychoeducation about the universality of intrusions is therefore key (Rachman & de Silva, 1978).
The therapist should also discuss expectations of what is required in therapy of
the client, for example, engaging in homework tasks, carrying out planned exposures independently, or keeping daily diaries of intrusions, interpretations, and
responses. This will help to gain an understanding of the client’s motivation for
treatment, which can depend on different factors such as insight into their problem,
overvalued ideas (e.g., preferring things to be neat and clean), having different ideas
about how treatment should work (e.g., by discussion of childhood instead of doing
exposures), frequency and duration of treatment (e.g., wanting a quick fix), or
comorbid problems that require addressing first (e.g., alcohol problems may not
be seen as a problem). The concept of ‘becoming your own therapist’ should be
conveyed, that is, the idea of the client taking more responsibility for their recovery
as treatment progresses. Client and therapist should then agree on what the problem is, and this should be translated into specific, measurable, attainable, realistic,
and time-based (SMART) goals to help monitor treatment progress. A client with
contamination fears may want to work on reducing washing rituals; someone with
concerns about incompleteness and symmetry may aim to reduce ordering rituals.
History and Early Experiences
The developmental history is key to understanding the client’s problem, and often
helps the client to ‘feel understood’. The goal should be to understand the onset of
the problem and make clear links between possible past aversive memories and
their OCD. We find drawing out a timeline helpful as an aid to go through the
client’s early childhood, adolescence, and adulthood while focusing on key events in
the person’s life, including the family history of mental health problems, attachment
203
204
Oliver Sündermann and David Veale
history and any traumatic experiences, bereavements, or experiences of discrimination. Parenting characterized by high levels of criticism, perfectionism, riskaversion, or overprotection is related to OCD and should be inquired about
(Frost et al., 1994). The therapist should provide a rationale as to why it is important
to have a good grasp of the past. We find the metaphor of ‘ghosts from the past’
helpful in explaining how unprocessed difficult events can continue to affect
(‘haunt’) the person in the here-and-now by shaping their experiences and interpretations of events; for example, a client who soiled herself as a child in school and
was teased for smelling may have developed disgust-based memories of the event
that now drive disgust-avoidance and cleaning rituals. A childhood memory of being
racially teased (e.g., ‘Your skin colour is dirty’) can fuel mental contamination in
the present.
Assessment of Obsessions and Compulsions
The assessment of obsessions and compulsions should be guided and informed by
the CBT model of OCD; that is, the therapist should gain a good understanding of
the context in which the OCD arises, how obsessions are appraised, and what coping
strategies the person employs.
A good starting point is to ask about a recent episode or trigger when the client
felt distressed by obsessions/compulsions, and elicit a typical sequence of triggerintrusion-interpretation-response. We recommend drawing out a maintenance
model (‘vicious flower’) jointly with the client on a whiteboard to clarify content
of intrusion(s), appraisals, and the underlying principles and responses to the
intrusion, including compulsive ritualizing, neutralizing, reassurance seeking, and
avoidance. It is usually helpful to provide examples of typical OCD appraisals to
further elicit discussion about these, alongside normalizing them. For example, if the
client suffers from contamination-related OCD with harm-avoidance, inquire about
whether they feel responsible for preventing harm. The (unintended) consequences
of each coping behavior should be examined, and how these possibly strengthen
OCD appraisals and maintain their OCD.
Rather than focusing on understanding the content of an intrusion, we recommend identifying the processes by which the client concludes that their thoughts are
dangerous or confirm danger. Typical principles involve ‘acting on thoughts’ (e.g.,
fear of losing control can lead to loss of control), ‘thoughts cause harm’ (e.g., belief
that presence of thought has power to make it happen), ‘moral equivalence’ (e.g.,
having an intrusion is morally the same as acting on it), and emotional reasoning
(e.g., I feel dirty, hence I am contaminated). This will enable clients to be able to
identify and label these processes when they occur.
The therapist should identify a recent episode in which the intrusion occurred
but the person was not or less bothered by it, and discuss the differences. For
example, someone with harm-obsessions about killing his wife may be less distressed when not around his spouse; the therapist should discuss how distress results
from appraisals (e.g., ‘I must avoid being near her to prevent accidentally killing
her’), avoidance, and failed disconfirmation. In case of incompleteness or ‘just-right’
Obsessive-Compulsive Disorder
obsessions characterized by sensory perceptions preceding rituals, appraisals may
focus more on the ability to tolerate the discomfort (e.g., ‘If I don’t feel right
I cannot walk through the door’) rather than the meaning of the urge to ritualize.
OCD-Related Imagery
Intrusive images are common in OCD and are often linked with past adverse
experiences. A thorough assessment includes checking if the client experiences
any visual images when triggered. Similar to imagery in social phobia or body
dysmorphic disorder, images may be experienced as a ‘felt impression’, that is, the
perception of their trigger or feared catastrophe. For example, someone with
contamination OCD may have images of oversized bugs crawling over their skin
after contact with contaminants. The therapist should clarify the perspective of how
images are perceived, that is, whether they are experienced from an observer
perspective (as if looking at oneself from another person’s perspective), which often
relates to more distorted imagery and greater distress or field perspective (looking
out of one’s own body). Images often provide an emotional (‘hot’) bridge to the
onset of the difficulties, hence it is important to ask when they first experienced their
images. The therapist should also ask how old they feel when they are triggered in
the here-and-now. The meaning of any images and their current relevance should be
carefully explored.
Engagement into the CBT Approach to OCD: Theory A/Theory B
A common technique to help clients build an alternative view of their difficulties
and to engage them into a cognitive approach to obsessional problems is ‘Theory A/
Theory B’ (see Table 11.2) (Salkovskis, 1999). This can be introduced by first asking
the client what they believe their problem is from the OCD perspective (e.g., ‘My
problem is that I am at risk of contracting and spreading HIV, and it is my responsibility to prevent this’). If this were true, then the therapist should ask what the client
would need to do (e.g., wear only ‘safe’ footwear, scan floor for needles, check body
for pricks), followed by inquiring what the future would be like if Theory A was
true. Subsequently, the therapist should propose a different view on their problem,
namely, Theory B, which states that their problem is one of worry instead of being
factual (e.g., ‘The problem is that I worry that I am at risk of contracting HIV and
infecting my partner’). Discuss implications of what they would need to do if Theory
B were true, which usually elicits answers of ‘doing the opposite’ to their compulsions and living in line with their values. Understandably, the outlook for a Theory
B–led life is much brighter.
The therapist should highlight that only one of the two theories can logically be
correct and that therapy is about finding out which theory is a better, more accurate
account of their experiences. Of course, the only way to test both theories is by
acting in reverse to Theory A, and this can lead to a discussion of E/RP and
behavioral experiments. Inquire whether the client would be prepared to act ‘as if
their problem were Theory B’. Commonly, clients express doubts about Theory B,
205
206
Oliver Sündermann and David Veale
Table 11.2. Worksheet for Theory A/Theory B with example of client suffering from sexual obsessions
about children
Theory A (OCD)
My problem is that I am a pedophile and I am
going to harm my children.
Theory B (Worry problem)
My problem is that I worry about being a pedophile. I am
a caring person, who simply worries about causing harm
to those I love the most.
Evidence
I have thoughts about hurting children.
Evidence
I have never caused any harm.
Everyone tells me that I worry too much.
I have OCD.
What do I need to do if Theory A is true?
Stay away from all children, including my own.
Monitor sensations in my genitals.
Seek reassurance from my wife and doctors.
Suppress any thoughts about children through
distraction, counting rituals, and alcohol.
Avoid reading newspaper and watching TV to
minimize risk of seeing children.
Mentally review if I was aroused by the thought
of children.
Report myself to the police.
What do I need to do if Theory B is true?
I need to live my life in accordance with my values. This
involves spending time with my wife and children, going
to work, meeting friends, and engaging in my hobbies.
I need to do all these activities without engaging in any
rituals, compulsions, and avoidance behaviors.
The future
Dark, depressing; would not have a future
The future
Positive, optimistic
or reluctance to ‘act in line with Theory B’. This should be normalized as an
understandable response whilst stressing the importance of challenging and testing
out OCD fears. It is also helpful is to ask the client if they have noticed how their
solutions (Theory A behaviors) have in fact become their problem (time-consuming
and distressing rituals) and keep them stuck. The therapist can also suggest ‘fake it,
to make it’ in order to challenge Theory A, and that the client can always go back to
Theory A, if they wish – after fully committing to treatment for a set period of time.
Once both theories are drawn out, belief ratings should be obtained, and it should
be explained that both theories provide the framework of therapy; that is, all
therapy work will be related to Theory A/Theory B, and change in belief rating
should be monitored.
Cognitive Behavioral Treatment for OCD
Overview
Once therapist and client have reached a shared formulation of the OCD, the actual
work phase begins. The main purpose of treatment is to build up evidence for
Theory B through repeatedly testing out fears and expectations and building up
tolerance of the client’s specific OCD distress (fear, disgust, just-so sensations)
through planned exposures and behavioral experiments – while not engaging in
Obsessive-Compulsive Disorder
any compulsive or safety-seeking behaviors. We usually highlight that OCD treatment is very much a ‘doing’ treatment as opposed to a ‘talking’ treatment, requiring
the client to take responsibility for homework assignments and becoming gradually
independent in conducting and generalizing therapy tasks.
Delivery of CBT
The delivery of therapy – that is, the number, length, and frequency of sessions;
their format (group vs. individual); and context (home-based, outpatient, residential, or inpatient) – depends on severity, complexity, and available resources.
Usually, outpatient sessions occur weekly for up to 20 sessions (mild to moderate
severity), and last between 45 and 90 minutes. Longer sessions for prolonged
exposures (e.g., 2–3 hours) should be considered. In residential or inpatient settings,
sessions can also be shorter and more frequent. Sessions should be audio recorded
or videotaped for clients to listen to, and treatment progress should be monitored.
The OCD protocol described here (Table 11.2) can and should be adapted
according to presentation and setting.
Formulation and Treatment
To illustrate formulation and treatment for OCD, we present the case of Eric (who
attended intensive inpatient treatment in a UK national specialist service for OCD)
with reference to challenges, pitfalls, and learning points from his therapy. We chose
Eric because we think his case is informative in terms of enhancing CBT with
insights from recent OCD research (see Sündermann & Veale, 2017, for a more
detailed description of Eric’s OCD).
CASE STUDY: ERIC
Eric is a 27-year-old single man with Bosnian Muslim origin who suffered from
debilitating OCD characterized by disgust avoidance of his and other people’s
semen. He was admitted to a UK national specialist clinic for OCD and his YBOCS score on admission was 40/40 (extremely severe).
His main worry was that he might leak and spread semen. He was easily triggered
by sexual thoughts, arousal, or anything vaguely related to, or perceived as, semen.
When triggered, he experienced vivid images of spermatozoa, which were associated with high levels of disgust and shame, emotionally linked with childhood wet
dreams, and related to appraisals of being dirty and responsible for preventing
semen from spreading. He engaged in extreme decontamination rituals (showering
for 30 hours) using hard cleaning products (bleach, scrubs) and stayed indoors (he
had not left the house in seven years at admission). Eric mentally replayed events,
checking for contact with contaminants, suppressing thoughts with mental counting
rituals, seeking reassurance from his mother (‘Is this semen?’; ‘Have you been in
contact with semen?’), and ridding himself of everything perceived contaminated.
207
208
Oliver Sündermann and David Veale
His relationship with his mother was highly enmeshed to the point that she had
entirely bought into his OCD belief system fearing contamination and experiencing
disgust by the thought of semen. She started wearing gloves and kept the house
microscopically clean even though Eric was in hospital. They had not been in direct
contact with each other for seven years, and kept a ‘safety distance’ of one meter,
with Eric worrying that semen would magically transfer between them. Eric’s OCD
developed during his early teenage years when he started masturbating, secretly
watching pornographic material, and experienced wet dreams, all of which caused
feelings of guilt, shame, and dirtiness. He coped by showering in the middle of the
night to feel clean.
Past treatment courses of CBT with E/RP for his OCD were largely unsuccessful,
and our shared understanding for treatment failure was that previous therapies had
overly focused on E/RP without formulating the emotional origins of his OCD. This
continued to affect Eric through vivid imagery of semen fused with past shame and
disgust-based memories.
Eric’s OCD was also tightly linked with anger and aggression. Irritation interfered
with ritual completion, which fuelled his OCD, and often escalated his anger. On the
other hand, aggression also provided relief from OCD distress, all of which made
sense in the context of his violent upbringing. His uncle was physically abusive
towards him and his mother; in school he was bullied for being foreign; and he
observed that the most aggressive male peers appeared to be most popular among
the female students – all of which contributed to warped ideas about masculinity and
needing to be aggressive to survive and be attractive.
Case Formulation in OCD
Case formulation is an integral part of CBT for OCD, and it is crucial to first reach a
shared understanding of the problem before embarking on interventions. Theory A/
Theory B socializes the client into the CBT model and provides the groundwork for
the initial formulation with the elicited Theory A behaviors mapping onto the OCD
vicious cycle. The therapist might start by pulling together the information they
have gathered so far, including interview and questionnaire data, by jointly drawing
out a diagram of the model on a whiteboard. Figure 11.1 shows an example of Eric’s
formulation. We have adapted this OCD model from previous established CBT
models of OCD (Salkovskis, 1999; Veale, 2007). Note that the model includes clear
past-present links between early relevant incidences and current OCD characterized by a triangulated experience of OCD-related imagery, emotions, and appraisals, which drive idiosyncratic OCD behaviors and processes. Each OCD coping
behavior is briefly described, alongside the specific unintended consequences. For
example, Eric discovered that seeking reassurance from his mother increased his
uncertainty and anxiety. Decontamination rituals, besides feeding his preoccupation
with contamination of semen, incurred significant financial debt and feelings of guilt
for ‘wasting money’ on cleaning products. Staying indoors and avoiding social life
caused feelings of disconnection and loneliness, increasing depression.
Obsessive-Compulsive Disorder
209
Relevant past experiences (2,3,6)
• 3 years: Fled civil war in Bosnia with mother; father died in genocide
• 3–15 years: Mother suffered from PTSD and was emotionally unavailable to him
• 10–16 years: Wet dreams and other shame/disgust-based relevant experiences
(e.g., being told that ‘masturbation is the work of the devil’
Trigger (1,2,3)
Any sexual thought, being aroused or worrying about arousal, seeing white
stains perceived as semen, actual semen, semen-like smell; suspecting
other people of having had contact with semen
(1, 4)
My attention narrows to:
Other ways of coping: (1, 2)
Mum 'protects' me from contamination;
buys me OCD products and smuggles them
into hospital; entirely dependent on her.
Engaging in mental counting rituals to block
out any thoughts about semen.
Anything that could be
semen. Constantly
scanning and monitoring
environment for threat
Unintended consequences:
Unintended cosequences:
Mum now suffers from OCD, feeling
guilty; increases my anxiety, more
preoccupied with semen
Meaning or past
associations: (3, 6)
I am dirty. l must
prevent semen from
spreading
I seek reassurance by: (2)
Constantly asking mum to clarify if
something is semen, if she came in
contact with semen, and if I am safe
Unintended consequences:
I became more uncertain, and
get more anxious
Spotting 'semen'
everywhere, more
anxious and agitated
Image or physical
sensations: (3, 6)
Images of
semen, linked
with early
experiences
I 'undo' the event by:
(i.e., washing) (1, 2)
Extreme showering rituals using hard
cleaning products (bleach, sponges) to
scrub myself for up to 30 hours, assisted
by my mother
Throwing away clothes, shoes, and
everything perceived contaminated
Unintended consequences:
I get more agitated and hardly
feel clean; getting stuck and
losing sense of time
Severe debt and distress, also
on mum's part
I avoid: (1, 2)
Emotions: (3, 5, 6)
Overwhelming
disgust and
anxiety
Stay indoors in my
room at all times;
move as little as
possible
Unintended consequences:
Missing out on life;
becoming depressed
and stuck in OCD
(1, 4)
I replay past events in my
mind or physically check by:
Meticulously rehearsing and trying
hard to recall whether I came in
contact with own or other people's
semen
Unintended consequences:
Becoming more
anxious and never getting
clarity and certainty
Figure 11.1. Eric’s case formulation, demonstrating how his contamination-related OCD characterized
by disgust of semen developed and was maintained by compulsive rituals and avoidance, which all
served the function of disgust-avoidance and brought about multiple unintended consequences.
Adapted with permission from Sündermann & Veale (2017)
210
Oliver Sündermann and David Veale
Confirmatory biases of narrowing his attention and ‘scanning’ for semen (often
labelled as ‘looking for trouble’) substantially increased the frequency of obsessions,
thus triggering anxiety, frustration, and agitation. Eric found two aspects of his
formulation particularly helpful. First, after discussion of the function of each
behavior and its unintended consequences, Eric felt he ‘finally got how my OCD
works, and backfires’. We translated each OCD behavior from his formulation into
a treatment goal and avoidance hierarchy. Second, a developmental understanding
of his OCD, that is, contextualizing his early shame and disgust-based experiences
and linking these with his current experiences of semen-related imagery and disgustavoidance, was a greatly normalizing experience and motivated him to work on
processing the relevant memories.
Exposure-Based Interventions
Exposure and behavioral experiments are deliberate and planned activities that help
clients experience the main emotion avoided (e.g., fear, disgust, sensations of just-so/
right, and incompleteness), test out their predictions and build the evidence as to
whether their experience best fits with a worry problem (Theory B), and learn to
tolerate their distress without any compulsions or safety-seeking behaviors. A common
misconception is that exposures are ‘simple’, ‘uncomplicated’, and ‘easy to deliver’.
However, successful exposure based-interventions depend on a multitude of factors
(Bouton, 2002; Craske et al., 2014; McKay et al., 2015) concerning decisions about
context, content, timing, and process of delivery, that is, what to do, when, how, and at
what pace. The following principles briefly outline how to set up and deliver exposure
tasks, which are informed by an inhibitory approach to exposure-based interventions
and testing out expectations (Arch & Abramowitz, 2015; Craske et al., 2014):
1. Set up a hierarchy of exposure tasks with feared/avoided triggers/situations
2. Exposure should be graded, but we recommend ‘jumping’ around the hierarchy
instead of working sequentially from the bottom of the list to the top.
3. The focus is on maximal expectancy violation of what the client predicts, rather
than habituation. Similar to behavioral experiments, test beliefs about coping
(‘I can’t tolerate the disgust, will lose control’), and always reflect on the
outcome of specific predictions with regard to formulation and Theory A/
Theory B. Ask what was learned, and how the outcome best fits the theories.
4. Habituation during sessions is less important than extinction between sessions.
5. All tasks should be generalized across different times and contexts (e.g., on
their own, in unfamiliar places, at home).
6. Labelling of emotions during exposure can be helpful.
7. Overcome all avoidance and compulsive/safety-seeking behaviors during and
after exposures; if client ritualizes subsequently, retrigger (e.g., touch
contaminant again).
8. Aim for daily exposures, and do not leave long gaps between exposures to
avoid spontaneous reinstatement of OCD distress and responses.
Obsessive-Compulsive Disorder
Various pitfalls should be kept in mind when conducting exposure-based interventions. First, response/ritual delay is highly common, which refers to clients
carrying out an exposure without a ritual but ‘storing it’ for later, for example,
tolerating contaminant triggers with the therapist, but completing a decontamination ritual after the session. This is common, and clients should be encouraged to
redo the exposure (e.g., re-contaminate). Self-monitoring (e.g., using tally counters)
can help clients to keep track of rituals (and resist them) post-exposures.
Second, termination criteria for rituals should be specific and objective, and
not feeling-based; for example, specify the frequency of and duration of
showering. Note that emotional termination criteria (e.g., having to feel comfortable, ‘just right’) are not abnormal but elevated criteria to normal routine; that is,
they are relevant for important decisions (e.g., who to get married to) but take
excessive amount of time when applied to rituals. Clients should be encouraged
to stop rituals feeling not right or uncomfortable, and if completed feeling right
or comfortable, they should do re-exposure until they can stop the ritual feeling
not right.
Third, often therapy overly focuses on response prevention, rather than
planned exposures, for example, not carrying out a counting ritual when the impulse
arises instead of actively triggering the impulse (e.g., by wishing their parents to die
in an accident). Exposure interventions should always be carried out together with
ritual prevention to maximize effectiveness.
Fourth, while the aim of E/RP should be to eliminate all ritualized responses,
judicious use of safety-seeking behaviors (or better termed as approachenhancing behaviours) can be helpful (Rachman et al., 2011). This could involve
confronting contaminants with reduced rituals instead of eliminating them immediately. For example, Eric was initially unable to masturbate because of overwhelming feelings of disgust and fear of decontamination rituals. To help Eric
approach exposures of masturbation, we agreed to several achievable exposure
levels, including the use of judicious safety behaviors such as masturbating only
in the shower (as opposed to his room), a five-minute shower afterwards (as
opposed to hour-long showers), wiping the shower drainage hole, and flushing his
semen (as opposed to keeping it for further exposures and to build up disgust
tolerance).
In line with inhibitory E/RP principles, we focused on violation of expectancies
of his coping beliefs. For example, Eric predicted he would not be able tolerate the
smell of semen, and was surprised that he could with a graded approach using many
repetitions. He also scored his emotions during each exposure (0–100), and during
most E/RP his disgust dropped below 30/100 after 10–15 minutes.
Uncertainty about contact with semen was a particularly strong trigger; therefore, another line of experiments involved shaking hands with the therapist who had
put his hand into a plastic cup out of his sight in the inside of his jacket (without him
seeing the cup), and telling him that it ‘may or may not contain’ semen. Since
physiology of disgust responses during exposures is associated with a decrease in
heart rate (Duncko & Veale, 2016), we speculated that accelerating Eric’s heartbeat
211
212
Oliver Sündermann and David Veale
immediately before E/RP may enhance effectiveness. We found some anecdotal
evidence that his disgust habituation was several minutes faster when Eric sprinted
for one minute right before exposure (compared with not sprinting or doing a
relaxation exercise). Counter-conditioning has been suggested as potentially enhancing E/RP in disgust-OCD; and we recommend exploring (and researching) if
pairing disgust-triggers with idiosyncratically identified pleasant stimuli during
exposures is effective.
Imagery Rescripting and Stimulus Discrimination
Imagery rescripting aims to change the meaning of past childhood events that
continue to shape the person’s experience (Arntz, 2011). This may be relevant to
clients with OCD, particularly where current imagery is fused with shame or disgustbased memories. The protocol is based on Arntz’s manual (Arntz & Weertman,
1999) and is described in Chapter 15. In short, the stages include reliving the event
as a child, re-entering as an adult, then reviewing and rescripting the event as a child
once more to a less catastrophic outcome that focuses on the child feeling safe. Eric
was able to revisit and rescript several key events that were associated with the
emotional origins of his OCD.
For example, he rescripted the memory of his first wet dream that continued to
cause him great distress, as it was not normalized due to the absence of his father.
He was able to imagine soothing his younger self (‘Little Eric’) as an adult by
normalizing the experience. Another key incident he rescripted involved his uncle
telling him that ‘masturbation is the work of the devil’ and that ‘a good Muslim does
not masturbate’. Imagery rescripting seemed to help Eric in several ways: he experienced a sharp decrease in shame and disgust, which also boosted exposure work as
he was better able to tolerate disgust and uncertainty of contamination; frequency of
intrusive semen-related imagery was reduced; and masturbation became more
enjoyable. Imagery rescripting also brought about feelings of sadness for the loss
of his father and aided a delayed bereavement process, and he experienced anger
and guilt towards his mother for not being emotionally available when his
OCD developed.
Stimulus discrimination can further aid ‘putting aversive memories to rest’. Eric
learned to identify associative past-present links and was able to tell himself, in a
compassionate voice, that he did not feel safe in his abusive home environment and
that he had no one to turn to when he felt lonely, ashamed, and dirty. Through
becoming aware of these emotional links, he was able to distance himself from these
memories and contextualize them, which in turn facilitated courage in exposures.
Cognitive Interventions and Reassurance-Seeking for OCD
Cognitive interventions for OCD involve challenging misappraisals of obsessional
thoughts and discussing evidence for and against key appraisals. Caution is warranted when discussing feared consequences that relate to unspecific or far distant
Obsessive-Compulsive Disorder
time points in the future (e.g., causing accidents at any time or going to hell in the
afterlife). Rather than attempting to disprove appraisals, discussions should focus on
the helpfulness and costs of these beliefs and drawing on Theory A/Theory B to
examine how rituals are the problem and keep the person stuck. Salkovskis (1999)
uses the metaphor of OCD rituals being an extremely costly insurance premium
with the cost being greater than the risk justifies.
Sometimes ‘experts’ such as religious leaders with more credibility than the
therapist with regard to the clients’ belief system can be involved or consulted in
therapy to normalize and refute unhelpful spiritual beliefs. Disconfirmation strategies work best when clients exhibit clear thought–action fusion (e.g., imagining
harm causes harm), and these can be subjected to experiments where the client
deliberately attempts to bring on the feared outcome; for example, encourage the
client to repeatedly wish/imagine their parents dying in car accident the same
afternoon (of course, the therapist should also wish a loved one dead to normalize
and model the experiment).
Clients may also learn meta-cognitive skills to distance themselves from obsessions and reappraise the process. For example, a client who believes that ‘thinking of
stabbing a baby is as bad as killing it’ can learn to label these as moral–action fusion,
thereby distancing themself from it. However, we advise against ‘labelling’ thoughts
as obsessions (e.g., ‘It’s my OCD’) as this in itself can become a ritualized response
and may encourage engagement with OCD thoughts. It is usually best to practice
acknowledging and ignoring (without ‘fighting’ or attempting to control) thoughts.
Instead of engaging with the content of the intrusion, it can be helpful to identify and
label the emotion that accompanies the thought (e.g., anxious) (Craske et al., 2014).
And it is helpful to seek or give oneself emotional support (e.g., ‘I’m feeling anxious;
can I have a hug, or can we go for a walk?’). Many people with OCD seek reassurance
with regard to their OCD fears, for example, asking their partner if they killed
someone or whether something is safe to touch. Generally, we encourage replacing
reassurance seeking with providing emotional support instead. For example, if a
client asks their partner for reassurance (‘Can I touch this?’), the partner could say:
‘I can see that you are anxious, and I am here with you. Do you remember what you
should be doing?’ or ‘What would Theory B behavior consist of?’, rather than giving a
point-blank refusal to give reassurance. Of course, the tone of one’s own voice and of
the other people supporting the client should always be compassionate, warm, and
accepting. The courage of challenging OCD is best met with compassion, including
self-compassion (Veale et al., 2014).
A Note on the Therapeutic Relationship
As with any treatment, the therapeutic alliance is pivotal, and CBT takes place
within a context of a safe and compassionate alliance where the client feels understood and trusts the therapist and their abilities. Two therapist qualities are particularly important for effective delivery of CBT for OCD. First, the therapist needs to
be able to tolerate and contain distress that is likely to arise during exposures.
213
214
Oliver Sündermann and David Veale
Second, the therapist needs the ability to model helpful coping responses in
moments of peak distress. The therapist must be careful not to collude with avoidance of necessary behavioral tasks that they may find unacceptable or ‘abnormal’
such as touching the inside of a toilet, smelling bodily fluids, wishing their partner to
die, or voicing blasphemous insults. Some of Eric’s exposures involved him masturbating to pornographic material, which his therapist had to organize and normalize,
and exposure to the smell of semen; his previous therapists were not comfortable
with these exposures, thus possibly reinforcing disgust-avoidance.
Family Involvement
Family accommodation is common in OCD, with family members or loved ones
often becoming a part of the client’s OCD system. Family accommodation can take
many different forms, such as clients asking and receiving reassurance for obsessional fears, ritual participation, or assisting clients in avoidance. There are various
assessment tools to assess family accommodation (see Table 11.3). Family accommodation predicts poorer outcomes, and clients whose family members received a
short intervention to reduce family accommodation seemed to recover more quickly
(Stewart et al., 2008; Thompson-Hollands et al., 2015). Hence, whenever indicated,
relevant others should be included in therapy (with consent) as co-therapists. In
Eric’s case, his mother’s involvement, most notably through providing constant
reassurance, worrying herself about semen, and ‘assisting’ constant showers,
required her own CBT for OCD. In less severe cases, it may be sufficient to build
a shared understanding, joint exposures, and practice resisting provision of reassurance (instead providing emotional support).
Recovery from OCD and Staying Well (‘Blueprint’)
Mindfulness meditation should not be offered instead of CBT and E/RP but can aid
the recovery process. Mindfulness can help to reduce responding to OCD thinking
styles, such as emotional reasoning (‘I feel anxious, therefore I must be in danger’),
thought–action fusion (‘If I think I am a pedophile, then I must be one’), and
distancing from OCD urges; becoming more aware of them; and choosing not to
carry out compulsions. Both therapist and client should be cautious that mindfulness
exercises are not carried out compulsively, as happened with one client who was a
diligent mindfulness practitioner and who responded to worries of being a pedophile with meditation. Although he reported that he found mindfulness useful, it
was discovered that he had not been aware of his underlying belief that he needed
to meditate to prevent him from harming children. His meditation had become a
safety-seeking behavior.
As with recovery from any mental disorder that has occupied the client pervasively for many years, recovery can be a daunting process. When Eric’s OCD
improved, he became aware of how much OCD had consumed his life and
expressed sadness about how little else there was in his life. Recovery should focus
Downloaded from https://www.cambridge.org/core, on subject to the Cambridge Core terms of use, available at https://www.cambridge.org/core/terms.
https://doi.org/10.1017/9781108355605.014
Table 11.3. Assessment measures for OCD
Outcome /
Severity
Name of measure
Type of
measure
Function of
measure
Yale–Brown Obsessive
Compulsive Scale (Y-BOCS)
Semi-structured
Clinician-rated
Severity of OCD
Brown Assessment of Beliefs Scale
(BABS)
Semi-structured
Clinician-rated
Insight into OCD
Obsessive Compulsive Inventory–
Revised (OCI-R)
Self-report
Screening
Symptom profiling
Severity of OCD
Dimensional ObsessiveCompulsive Scale (DOCS)
Self-report
Symptom profiling
Severity of OCD
Florida Obsessive-Compulsive
Inventory (FOCI) – Severity Scale
Self-report
Severity of OCD
Responsibility Interpretations
Questionnaire (RIQ)
Self-report
Interpretation of
intrusive thoughts
Description
Reference
Ten items measuring OCD symptoms,
severity, and response to treatment. Each
item is scored from 0 to 4, with a maximum
score of 40; higher scores indicating
greater severity and symptomatology.
Most widely used OCD outcome measure.
Seven items assessing conviction of a
belief, fixity of ideas, attempts to disprove,
and insight on a scale from 0 (nondelusional) to 4 (delusional). First six
items are summed with scores 12,
indicating poor insight.
Eighteen-item scale rated on four-point
Likert scale, measuring six subscales:
obsessions, checking, neutralizing,
hoarding, ordering, and washing.
Twenty items assessing four dimensions of
OCD: concerns about (1) germs and
contamination; (2) responsibility for harm,
injury, or bad luck; (3) unacceptable
thoughts; and (4) symmetry,
completeness, just-right.
Five items assessing presence and severity
of obsessions and compulsions (obtained
from the FOCI, a 20-item symptom
checklist). Recommended for tracking
treatment progress.
Twenty-two items assessing the
interpretation of intrusive thoughts about
potential harm by rating their frequency of
occurrence
Goodman
et al. (1989)
Eisen et al.
(1998)
Foa et al.
(2002)
Abramowitz
et al. (2010)
Storch et al.
(2007)
Salkovskis
et al. (2000)
215
216
Downloaded from https://www.cambridge.org/core, on subject to the Cambridge Core terms of use, available at https://www.cambridge.org/core/terms.
https://doi.org/10.1017/9781108355605.014
Table 11.3. (cont.)
Type of
measure
Function of
measure
Self-report
Mental
contamination
Self-report
Disgust
University of São-Paulo Sensory
Phenomena Scale (UP-SPS)
Self-report
Sensory
phenomena
Family Accommodation Scale
(FAS)
Clinician-rated
Family
accommodation
and distress
Family Accommodation Scale SelfRated (FAS-SR)
Self-report
Family
accommodation
and distress
Name of measure
Specific
OCD
measures
Family
participation
Vancouver Obsessional
Compulsive Inventory–Mental
Contamination Scale (VOCI-MC)
Disgust Emotion Scale (DES)
Description
Reference
Twenty-item self-report assessing mental
contamination and internal dirtiness; with
a potential maximum score of 80
Thirty items measuring disgust sensitivity
across various domains: (1) animals, (2)
injections and blood draws, (3) mutilation
and death, (4) rotting foods, and (5)
smells. Five-point Likert scale from 0 (no
disgust or repugnance at all) to 4 (extreme
disgust or repugnance).
Investigates presence and severity of
different sensory phenomena occurring
before or during repetitive behaviors,
including ‘just-so’, ‘just-right’, and ‘urge
only’ perceptions, feeling of
incompleteness sensations, and ‘energy
release’ sensations
Thirteen items assessing extent of family
members’ accommodating rituals, and
family distress associated with
participation. Higher scores indicate
greater involvement. A parent-report
version (FAS-PR) is available for children
and adolescents with OCD (Flessner et al.,
2011).
A self-report version of the clinician-rated
FAS; a valid, time-saving, and costeffective alternative
Rachman,
(2005)
Olatunji
et al. (2007)
Rosario et al.
(2009)
Calvocoressi
et al. (1995)
Pinto et al.
(2013)
Obsessive-Compulsive Disorder
on helping clients to move towards their valued goals – which often involves
building meaningful relationships, occupational work, hobbies, and so on – while
also acknowledging and helping clients process sadness about the losses OCD
has incurred.
Measurement of Outcome
Outcome measures are an important part of every evidence-based intervention,
including CBT for OCD. Treatment progress should be measured continually, using
self-report measures, assessing adherence to homework tasks, progress during E/RP
and behavioral experiments, and (with consent) reports from significant others
(Sookman, 2015). Table 11.3 gives an overview of specific OCD self-report measures and interviews that can be administered at various points during the course of
treatment. Ideally, OCD-self-reports (e.g., Florida Obsessive-Compulsive Inventory
[FOCI]), alongside general mood and anxiety measures, are obtained at every point
of contact, preferably filled out before the session, so that progress can be reviewed
jointly with the client. Clinical judgement should complement quantitative measures; for example, the therapist should regularly review engagement with therapy
tasks, and progress during exposures in and between sessions, including asking
relevant others how they perceive the client’s progress. More comprehensive outcome measures should be obtained at the beginning and end of therapy. The most
common clinician-led OCD measure is the Yale–Brown Obsessive Compulsive
Scale (Y-BOCS), which is more likely to be conducted in specialist services, and
may be required by funding panels.
Summary and Conclusion
CBT for OCD remains the treatment of choice, and E/RP with testing of expectations lies at the heart of every therapy course. Inhibitory learning principles should
optimize exposure-based interventions, for example, by focusing on maximizing
expectancy violation of predicted concerns (e.g., ‘I cannot tolerate disgust’), with
frequent repetitions of varying exposures in different contexts, always dropping all
coping behaviors. A thorough developmental formulation is key, making clear links
between past relevant experiences and current OCD. Cross-sectional formulations
should describe the context and the function of each coping behavior and its
unintended consequences.
Imagery rescripting should be considered when past shame- or disgust-based
memories continue to affect the client (as described here in Eric’s case). Selfcompassion skills can de-shame the clients’ experiences and further help to disengage from threat monitoring and ‘self-attacking’ thinking. We emphasize to clients
that while their OCD is not their fault, it is their responsibility to do something
about it, and the concept of ‘becoming your own therapist’ is particularly useful in
OCD recovery that requires the client to continue with planned exposures and align
their lives with their values.
217
218
Oliver Sündermann and David Veale
Key Learning Points
Behavioral interventions to test out expectations remain the key ingredient in
the treatment of OCD.
Inhibitory learning principles should inform all behavioral tasks.
Theory A/Theory B is a helpful and engaging framework to conceptualize the
problem and helps to build an alternative, more benign view of the problem.
The formulation should describe the context and function of each safetyseeking behavior and the related unintended consequences.
Imagery rescripting and stimulus discrimination can be considered when past
aversive events are meaningfully linked to the person’s OCD.
Compassion-focused interventions may be a helpful adjunct to therapy.
Relevant others should be involved in therapy where possible and indicated.
REFERENCES
Abramowitz, J. S. (1996). Variants of exposure and response prevention in the treatment of
obsessive-compulsive disorder: A meta-analysis. Behavior Therapy, 27(4), 583–600.
(2006). The psychological treatment of obsessive-compulsive disorder. The Canadian
Journal of Psychiatry, 51(7), 407–416.
Abramowitz, J. S., Deacon, B. J., Olatunji, B. O., Wheaton, M. G., Berman, N. C., Losardo,
D., . . . & Adams, T. (2010). Assessment of obsessive-compulsive symptom dimensions:
Development and evaluation of the Dimensional Obsessive-Compulsive Scale.
Psychological Assessment, 22(1), 180.
Abramowitz, J. S., Franklin, M. E., Schwartz, S. A., & Furr, J. M. (2003). Symptom presentation and outcome of cognitive-behavioral therapy for obsessive-compulsive disorder.
Journal of Consulting and Clinical Psychology, 71(6), 1049.
Abramowitz, J. S., & Jacoby, R. J. (2014). Obsessive-compulsive disorder in the DSM-5.
Clinical Psychology: Science and Practice, 21(3), 221–235.
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Arch, J. J., & Abramowitz, J. S. (2015). Exposure therapy for obsessive-compulsive disorder:
An optimizing inhibitory learning approach. Journal of Obsessive-Compulsive and
Related Disorders, 6, 174–182.
Arntz, A. (2011). Imagery rescripting for personality disorders. Cognitive and Behavioral
Practice, 18(4), 466–481.
Arntz, A., & Weertman, A. (1999). Treatment of childhood memories: Theory and practice.
Behaviour Research and Therapy, 37(8), 715–740.
Bouton, M. E. (1993). Context, time, and memory retrieval in the interference paradigms of
Pavlovian learning. Psychological Bulletin, 114(1), 80.
(2002). Context, ambiguity, and unlearning: Sources of relapse after behavioral extinction.
Biological Psychiatry, 52(10), 976–986.
Bouton, M. E., Westbrook, R. F., Corcoran, K. A., & Maren, S. (2006). Contextual and
temporal modulation of extinction: Behavioral and biological mechanisms. Biological
Psychiatry, 60(4), 352–360.
Calvocoressi, L., Lewis, B., Harris, M., & Trufan, S. J. (1995). Family accommodation in
obsessive-compulsive disorder. The American Journal of Psychiatry, 152(3), 441.
Obsessive-Compulsive Disorder
Clark, D. A., & Radomsky, A. S. (2014). Introduction: A global perspective on
unwanted intrusive thoughts. Journal of Obsessive-Compulsive and Related
Disorders, 3(3), 265–268.
Coughtrey, A. E., Shafran, R., Lee, M., & Rachman, S. (2013). The treatment of mental
contamination: A case series. Cognitive and Behavioral Practice, 20(2), 221–231.
Craske, M. G., Liao, B., Brown, L., & Vervliet, B. (2012). Role of inhibition in exposure
therapy. Journal of Experimental Psychopathology, 3(3), 322–345.
Craske, M. G., Treanor, M., Conway, C. C., Zbozinek, T., & Vervliet, B. (2014). Maximizing
exposure therapy: An inhibitory learning approach. Behaviour Research and Therapy,
58, 10–23.
Duncko, R., & Veale, D. (2016). Changes in disgust and heart rate during exposure for
obsessive compulsive disorder: A case series. Journal of Behavior Therapy and
Experimental Psychiatry, 51, 92–99.
Eisen, J. L., Mancebo, M. A., Pinto, A., Coles, M. E., Pagano, M. E., Stout, R., & Rasmussen,
S. A. (2006). Impact of obsessive-compulsive disorder on quality of life. Comprehensive
Psychiatry, 47(4), 270–275.
Eisen, J. L., Phillips, K. A., Baer, L., Beer, D. A., Atala, K. D., & Rasmussen, S. A. (1998).
The brown assessment of beliefs scale: Reliability and validity. American Journal of
Psychiatry, 155(1), 102–108.
Fineberg, N. A., Hengartner, M. P., Bergbaum, C., Gale, T., Rössler, W., & Angst, J. (2013).
Remission of obsessive-compulsive disorders and syndromes: Evidence from a prospective community cohort study over 30 years. International Journal of Psychiatry in Clinical
Practice, 17(3), 179–187.
Flessner, C. A., Freeman, J. B., Sapyta, J., Garcia, A., Franklin, M. E., March, J. S., & Foa, E.
(2011). Predictors of parental accommodation in pediatric obsessive-compulsive disorder: Findings from the Pediatric Obsessive-Compulsive Disorder Treatment Study
(POTS) trial. Journal of the American Academy of Child and Adolescent Psychiatry,
50(7), 716–725. https://doi.org/10.1016/j.jaac.2011.03.019
Foa, E. B., Huppert, J. D., Leiberg, S., Langner, R., Kichic, R., Hajcak, G., & Salkovskis,
P. M. (2002). The Obsessive-Compulsive Inventory: Development and validation of a
short version. Psychological Assessment, 14(4), 485.
Foa, E. B., & Kozak, M. J. (1986). Emotional processing of fear: Exposure to corrective
information. Psychological Bulletin, 99(1), 20.
Foa, E. B., Liebowitz, M. R., Kozak, M. J., Davies, S., Campeas, R., Franklin, M. E., . . . &
Schmidt, A. B. (2005). Randomized, placebo-controlled trial of exposure and ritual
prevention, clomipramine, and their combination in the treatment of obsessivecompulsive disorder. American Journal of Psychiatry, 162(1), 151–161.
Frost, R. O., Steketee, G., Cohn, L., & Griess, K. (1994). Personality traits in subclinical and
non-obsessive-compulsive volunteers and their parents. Behaviour Research and
Therapy, 32(1), 47–56.
Goodman, W. K., Price, L. H., Rasmussen, S. A., Mazure, C., Fleischmann, R. L., Hill,
C. L., . . . & Charney, D. S. (1989). Yale–Brown Obsessive Compulsive Scale
(Y-BOCS). Archives of General Psychiatry, 46, 1006–1011.
Hollander, E., & Benzaquen, S. D. (1996). Is there a distinct OCD spectrum? CNS Spectrums,
1(1), 17–26.
Hollander, E., Stein, D. J., Kwon, J. H., Rowland, C., Wong, C. M., Broatch, J., & Himelein,
C. (1997). Psychosocial function and economic costs of obsessive-compulsive disorder.
CNS Spectrums, 2(10), 16–25.
Kessler, R. C., Berglund, P., Demler, O., Jin, R., Merikangas, K. R., & Walters, E. E. (2005).
Lifetime prevalence and age-of-onset distributions of DSM-IV disorders in the National
Comorbidity Survey Replication. Archives of General Psychiatry, 62(6), 593–602.
219
220
Oliver Sündermann and David Veale
Ludvik, D., Boschen, M. J., & Neumann, D. L. (2015). Effective behavioural strategies for reducing
disgust in contamination-related OCD: A review. Clinical Psychology Review, 42, 116–129.
Mason, E. C., & Richardson, R. (2010). Looking beyond fear: The extinction of other
emotions implicated in anxiety disorders. Journal of Anxiety Disorders, 24(1), 63–70.
McKay, D., Sookman, D., Neziroglu, F., Wilhelm, S., Stein, D. J., Kyrios, M., . . . & Veale, D.
(2015). Efficacy of cognitive-behavioral therapy for obsessive-compulsive disorder.
Psychiatry Research, 225(3), 236–246.
Melli, G., Chiorri, C., Carraresi, C., Stopani, E., & Bulli, F. (2015). The two dimensions of
contamination fear in obsessive-compulsive disorder: Harm avoidance and disgust avoidance. Journal of Obsessive-Compulsive and Related Disorders, 6, 124–131.
Olatunji, B. O., Davis, M. L., Powers, M. B., & Smits, J. A. (2013). Cognitive-behavioral
therapy for obsessive-compulsive disorder: A meta-analysis of treatment outcome and
moderators. Journal of Psychiatric Research, 47(1), 33–41.
Olatunji, B. O., Sawchuk, C. N., de Jong, P. J., & Lohr, J. M. (2007). Disgust sensitivity and
anxiety disorder symptoms: Psychometric properties of the disgust emotion scale.
Journal of Psychopathology and Behavioral Assessment, 29(2), 115–124.
Olatunji, B. O., Wolitzky-Taylor, K. B., Willems, J., Lohr, J. M., & Armstrong, T. (2009).
Differential habituation of fear and disgust during repeated exposure to threat-relevant
stimuli in contamination-based OCD: An analogue study. Journal of Anxiety Disorders,
23(1), 118–123.
Öst, L.-G., Havnen, A., Hansen, B., & Kvale, G. (2015). Cognitive behavioral treatments of
obsessive-compulsive disorder. A systematic review and meta-analysis of studies published 1993–2014. Clinical Psychology Review, 40, 156–169.
Pinto, A., Van Noppen, B., & Calvocoressi, L. (2013). Development and preliminary psychometric evaluation of a self-rated version of the Family Accommodation Scale for
Obsessive-Compulsive Disorder. Journal of Obsessive-Compulsive and Related
Disorders, 2(4), 457–465.
Rachman, S. (1994). Pollution of the mind. Behaviour Research and Therapy, 2(3), 311–314.
(2005). Vancouver Obsessional Compulsive Inventory Mental Contamination Scale (VOCIMC). Rachman Lab.
(2006). The fear of contamination: Assessment & treatment. Oxford University Press.
(2010). Betrayal: A psychological analysis. Behaviour Research and Therapy, 48(4),
304–311.
Rachman, S., & de Silva, P. (1978). Abnormal and normal obsessions. Behaviour Research
and Therapy, 16(4), 233–248.
Rachman, S., Shafran, R., Radomsky, A. S., & Zysk, E. (2011). Reducing contamination by
exposure plus safety behaviour. Journal of Behavior Therapy and Experimental
Psychiatry, 42(3), 397–404.
Radomsky, A. S., Alcolado, G. M., Abramowitz, J. S., Alonso, P., Belloch, A., Bouvard,
M., . . . & Wong, W. (2014). Part 1: You can run but you can’t hide: Intrusive thoughts on
six continents. Journal of Obsessive-Compulsive and Related Disorders, 3(3), 269–279.
Rosa-Alcázar, A. I., Sánchez-Meca, J., Gómez-Conesa, A., & Marín-Martínez, F. (2008).
Psychological treatment of obsessive-compulsive disorder: A meta-analysis. Clinical
Psychology Review, 28(8), 1310–1325.
Rosario, M. C., Prado, H. S., Borcato, S., Diniz, J. B., Shavitt, R. G., Hounie, A. G., . . . &
Perin, E. A. (2009). Validation of the University of São Paulo sensory phenomena scale:
Initial psychometric properties. CNS Spectrums, 14(6), 315–323.
Rufer, M., Fricke, S., Moritz, S., Kloss, M., & Hand, I. (2006). Symptom dimensions in
obsessive-compulsive disorder: Prediction of cognitive-behavior therapy outcome. Acta
Psychiatrica Scandinavica, 113(5), 440–446.
Salkovskis, P. M. (1999). Understanding and treating obsessive-compulsive disorder.
Behaviour Research and Therapy, 37, S29–S52.
Obsessive-Compulsive Disorder
Salkovskis, P. M., Wroe, A. L., Gledhill, A., Morrison, N., Forrester, E., Richards, C., . . . &
Thorpe, S. (2000). Responsibility attitudes and interpretations are characteristic of
obsessive compulsive disorder. Behaviour Research and Therapy, 38(4), 347–372.
Schienle, A., Stark, R., & Vaitl, D. (2001). Evaluative conditioning: A possible explanation
for the acquisition of disgust responses? Learning and Motivation, 32(1), 65–83.
Sookman, D. (2015). Specialized cognitive behavior therapy for obsessive compulsive disorder:
An expert clinician guidebook. Routledge.
Sookman, D., & Fineberg, N. A. (2015). Specialized psychological and pharmacological
treatments for obsessive-compulsive disorder throughout the lifespan: A special series
by the Accreditation Task Force (ATF) of the Canadian Institute for Obsessive
Compulsive Disorders (CIOCD). Psychiatry Research, 227(1), 74–77.
Soomro, G. M., Altman, D., Rajagopal, S., & Oakley-Browne, M. (2008). Selective serotonin
re-uptake inhibitors (SSRIs) versus placebo for obsessive compulsive disorder (OCD).
The Cochrane Database of Systematic Reviews, (1), CD001765.
Speckens, A., Ehlers, A., Hackmann, A., Ruths, F., & Clark, D. (2007). Intrusive memories
and rumination in patients with post-traumatic stress disorder: A phenomenological
comparison. Memory, 15(3), 249–257.
Stewart, S. E., Beresin, C., Haddad, S., Egan Stack, D., Fama, J., & Jenike, M. (2008).
Predictors of family accommodation in obsessive-compulsive disorder. Annals of
Clinical Psychiatry, 20(2), 65–70.
Storch, E. A., Bagner, D., Merlo, L. J., Shapira, N. A., Geffken, G. R., Murphy, T. K., &
Goodman, W. K. (2007). Florida Obsessive-Compulsive Inventory: Development, reliability, and validity. Journal of Clinical Psychology, 63(9), 851–859.
Sündermann, O., & Veale, D. (2017). Complexity in obsessive-compulsive and body dysmorphic disorder: A functional approach to complex difficulties. The Cognitive
Behaviour Therapist, 10.
Taylor, S., & Jang, K. L. (2011). Biopsychosocial etiology of obsessions and compulsions: An
integrated behavioral-genetic and cognitive-behavioral analysis. Journal of Abnormal
Psychology, 120(1), 174.
Thompson-Hollands, J., Abramovitch, A., Tompson, M. C., & Barlow, D. H. (2015).
A randomized clinical trial of a brief family intervention to reduce accommodation in
obsessive-compulsive disorder: A preliminary study. Behavior Therapy, 46(2), 218–229.
Tolin, D. F., Maltby, N., Diefenbach, G. J., Hannan, S. E., Worhunsky, P., et al. (2004).
Cognitive-behavioral therapy for medication nonresponders with obsessive-compulsive
disorder: A wait-list-controlled open trial. Journal of Clinical Psychiatry, 65(7), 922–931.
van Oppen, P., van Balkom, A. J., de Haan, E., & van Dyck, R. (2005). Cognitive therapy and
exposure in vivo alone and in combination with fluvoxamine in obsessive-compulsive
disorder: A 5-year follow-up. The Journal of Clinical Psychiatry, 66(11), 1415–1422.
Veale, D. (2007). Cognitive-behavioural therapy for obsessive-compulsive disorder.
Advances in Psychiatric Treatment, 13(6), 438–446.
Veale, D., Gilbert, P., Wheatley, J., & Naismith, I. (2014). A new therapeutic community:
Development of a compassion-focussed and contextual behavioural environment.
Clinical Psychology & Psychotherapy, 22(4), 285–303.
Veale, D., Page, N., Woodward, E., & Salkovskis, P. (2015). Imagery rescripting for obsessive
compulsive disorder: A single case experimental design in 12 cases. Journal of Behavior
Therapy and Experimental Psychiatry, 49, Part B, 230–236.
Vervliet, B., Craske, M. G., & Hermans, D. (2013). Fear extinction and relapse: State of the
art. Annual Review of Clinical Psychology, 9, 215–248.
Woody, S. R., & Teachman, B. A. (2000). Intersection of disgust and fear: Normative and
pathological views. Clinical Psychology: Science and Practice, 7(3), 291–311.
Zysk, E., Shafran, R., & Williams, T. (2018). The origins of mental contamination. Journal of
Obsessive-Compulsive and Related Disorders, 17, 3–8.
221
12
CBT for OCD: Evidence-Based Treatment for
Contamination and Checking
Elizabeth A. Forrester
Introduction and Research
Did I clean the toilet with my husband’s toothbrush?
There are warnings on cleaning products to keep them away from children and pets
for a reason.
Am I really in love with my girlfriend?
The cat could die in a fire if I’ve left the gas on.
How can I be sure the marks on my car aren’t from clipping a cyclist without realizing?
I might have contracted HIV by stepping on a discarded syringe.
Despite the fact that any idea can become an obsession and any action a compulsion, contamination concerns and checking remain two of the most prevalent forms
of presentation in OCD. Contamination fears are reported by 25–50% of OCD
sufferers, with checking the most common compulsion (Ruscio et al., 2010). While
washing, cleaning, and checking are the most prevalent behaviors in OCD, they
cover a broad spectrum of concerns and complexity.
How Widespread Is Checking as a Compulsion?
It is difficult to envisage any forms of OCD devoid of checking in some shape or
form. Checking is a consistent feature across the range of OCD presentations and
can take a variety of forms, including the following:
Overt checking, for example, physically checking doors and windows, feeling
pockets for keys, retracing the route driven
Visual checking, for example, looking at an object, rereading a letter
Reassurance seeking, for example, asking others directly, surreptitiously watching what others do (or gauging their reactions), reviewing psychoeducational
materials, doing internet research
Covert checking, for example, mentally reviewing an action, checking an
internal state (Does this feel right? Do I feel certain?)
222
CBT for OCD
223
Usually a combination of checking behaviors is used and would be carried out
more than once. Frequently, repeating actions becomes part of the ritual used in an
attempt to alleviate worry and reduce anxiety.
If a person has concerns about faeces (i.e., contamination), checking will be a
regular behavior, whilst someone whose key concerns are connected with home
safety will not automatically have concerns about contamination. Yet often obsessional concerns can range across various domains. This is particularly the case when
someone has struggled with OCD over a long period of time: the initial onset of the
problem may begin with a specific intrusive thought or worry and generalize to
other areas. OCD has a tendency to morph to reflect the concerns of the day.
Further examples of the range of obsessions and compulsions involving worries
about contamination and checking behaviors can be seen in Table 12.1.
Table 12.1. Examples illustrating the range of contamination and checking concerns
Contamination
Checking
Obsessional concerns
Blood, bodily waste, or secretions
Dirt or germs
Illness or disease
Chemicals, e.g., insecticides, medicines
Environmental contaminants, e.g., asbestos, lead
paint, bisphenol A (BPA)
Household cleaners, e.g., bleach
Animals or insects
Mental contamination or tainted by association, e.g.,
not buying from cancer or learning disability charity
shop
Accidental or deliberate contamination, e.g., broken
glass in food, mishandling medication
Obsessional concerns
Have I done something dangerous?
Fear of being responsible for something bad
happening due to not being careful enough, e.g., fire,
burglary, illness
Welfare of self or others (including pets)
Responding to doubts
Making a mistake
Is my hair falling out?
Throwing something away accidentally
Have I committed a crime?
Do I love my partner?
Perfectionism, e.g., cleaning, making the bed, writing
a report at work, decorating, dressing and grooming
Compulsions
Excessive or ritualized hand washing
Excessive or ritualized showering or bathing
routines
Toileting rules, e.g., never sit on seat, wash clothes
after opening bowels, avoid public toilets
Excessive use (or avoidance) of cleaning products
Frequent laundering (including items of apparel not
usually washed regularly, e.g., coats, bags, footwear)
Actions to prevent contact with contamination, e.g.,
keeping groceries in bags, separating ‘clean’ and
‘unclean’ clothing, keeping doors and windows
closed at all times, wearing gloves for certain
activities
Careful cleaning of certain items, e.g., mobile
phone, door handles
Asking for reassurance
Compulsions
Checking doors, windows, gas, electric, appliances
Taking photos that can be checked later
Checking the car: Is it locked? Signs of damage?
Wallet, keys, phone, valuable items, e.g., jewellery
Driving route or retracing steps to check for signs of
an accident
Checking for signs of injury, e.g., puncture wounds
from syringes
Checking for possible dangers, e.g., contamination,
trip hazards, sharp items, causing offence, making
mistakes
Bank statements, till receipts
Repeated checking
Rereading, e.g., books, letters, emails
Counting whilst checking
224
Elizabeth A. Forrester
The overlap is not necessarily immediately obvious: for example, checking for
hair loss may not be due to a diagnosis of body dysmorphic disorder but the result of
worrying whether parabens in a shampoo previously used regularly may be harmful.
Similarly, someone who is driven to check repeatedly before throwing something
away may be concerned that it is contaminated (e.g., with bodily waste from
nappies), harmful (e.g., sharp items), or contain something of importance (e.g.,
jewellery or passport).
What Do Checking and Contamination Have in Common?
Key features of both checking and contamination are fears about harm, danger, and
safety. This may be about harm to themselves or to others, either accidental or
intentional. Furthermore, the consequences may be immediate or (more often) in
the future or even historically:
The bin man will cut himself if I’ve put broken glass in the rubbish (immediate).
I will become ill if I use a supermarket trolley that had contained raw chicken
(future).
What if the building rubble removed from my first flat contained asbestos?
(historical)
Responsibility, Blame, and Harm
A number of belief domains have been postulated, such as beliefs about being a
dangerous or harmful person (Riskind et al., 2007) and ideas about one’s own
vulnerability and fear of self (Melli et al., 2016). Initially proposed by Salkovskis
et al. (2000), responsibility continues to play an important role (Romero-Sanchiz
et al., 2015).
Overestimation of Threat
Overestimation of threat is a frequent clinical feature, although the more recent
development of inference-based therapy (IBT) (O’Connor et al., 2009) proposes
that the person with OCD develops a doubting narrative which increases the sense
of danger: ‘Although it looks clean, the last person to use this toilet may have a
sexually transmitted disease.’
Contamination and Disgust
A growing body of literature suggests that disgust should be taken into consideration where contamination fears are present (Thorpe et al., 2011). Many studies
have found that exposure-based treatments are less effective where disgust is a
predominant feature (Ludwik et al., 2015). It is suggested that disgust is less
amenable to habituation and there is a greater likelihood of behavioral avoidance,
which makes it less likely that therapeutic gains are sustained.
CBT for OCD
Checking and Memory
Beliefs about memory appear to be more important in OCD than actual poor
memory. Tackling maladaptive beliefs has been demonstrated to reduce checking
symptoms (Alcolado & Radomsky, 2016). Furthermore, repeated checking
increases doubt and uncertainty. The detrimental effect it has on clarity and detail
of memory has implications for memory confidence (Toffolo et al., 2016).
Not Just Right Feelings
While Fergus (2014) proposes that not just right feelings are not exclusive to OCD,
there is substantive evidence that obsessive checkers use more objective and subjective criteria compared with anxious and non-clinical controls when making decisions to terminate checking (Coles & Ravid, 2016; Salkovskis et al., 2017).
Mental Contamination
Previously overlooked, mental contamination is now recognized as a distinct concept separate from direct contamination, although there are similarities
A preliminary study found that 46% of participants felt dirty without direct physical
contact with a contaminant (Coughtrey et al., 2012). According to Mathes et al.
(2019), with mental contamination there is a greater propensity for feeling dirty
inside, which is less likely to be ameliorated by washing.
So, What Does This Mean for Treatment?
It can seem that every variant of OCD needs a different approach. So far, cognitive
theories fail to provide an adequate explanation why some intrusions are appraised
as significant and distressing whilst others are not (Melli et al., 2016). At a more
fundamental level, what needs to be addressed in therapy is the overvalued importance of intrusive thoughts, doubts, images, urges, and beliefs regarding their control
as these are associated with obsessive-compulsive symptoms at all levels of severity
(Kim et al. 2016).
McCubbin and Sampson (2006) argue there is a parallel between the intolerance of unwanted thoughts and the intolerance of unwanted emotions in OCD.
Low distress tolerance has been found to correlate with OCD symptoms (Robinson
& Freeston, 2014). It has also been suggested that compulsions prevent confrontation of distressing emotion and inhibit access to alternative information (Calkins
et al., 2013).
Third Wave CBT and OCD
Third wave CBT has led to interesting changes of emphasis on cognitive aspects of
treatment and uses various methods which place this important distinction at
their core.
225
226
Elizabeth A. Forrester
Mindfulness
Mindfulness proposes that obsessive thoughts should be observed simply as mental
events rather than an accurate reflection of reality. Outcomes from studies into the
use of mindfulness in OCD have so far been inconsistent: in experimental settings
they appear to show promise (e.g., Wahl et al., 2013), but there is insufficient
evidence of clinically important improvements compared with ERP (Strauss et al.,
2018).
Metacognitive Therapy (MCT)
MCT for OCD focuses on beliefs about inner experiences such as thoughts and
urges to carry out compulsions (Wells, 2009) rather than beliefs such as probability,
blame, or responsibility. An important feature is the concept of exposure and
response commission. This involves engaging in rituals but deliberately invoking
unwanted thoughts, for example, showering in their usual meticulous fashion whilst
repeatedly thinking, ‘I’m smeared in faeces.’
Acceptance and Commitment Therapy (ACT)
ACT utilizes the concept of holding a thought lightly, being an observer of thoughts
in the manner of mindfulness so they do not interfere with acting in accordance with
the individual’s values rather than in the pay of OCD (Twohig et al., 2015).
How Big Is the ‘B’ in CBT?
Exposure is an essential component in treatment of all anxiety disorders. Getting
the right balance between ‘cognitive’ and ‘behavioral’ in CBT can be tricky for even
the most experienced therapists. Sometimes it is the result of therapist ‘drift’ or a
preference for discussing cognitive matters (Waller, 2009). For those who have
become therapists via the Improving Access to Psychological Therapies (IAPT)
route, it is likely that their training has been based on Beck’s CBT approach.
Without an adequate grounding in behavior therapy, behavioral interventions rely
on behavioral experiments (a cognitive intervention intended to test the validity of
beliefs and assumptions) rather than exposure (Binnie, 2015).
Through a combination of observations and case examples from clinical practice
this chapter will do the following:
Take a fresh look at exposure and considers its focus and function
Examine the role of thought processes in OCD
Consider how changing relationships with thoughts and feelings can facilitate
exposure
Change our perspective on goals, processes, and defining success
Move away from ‘experiments’ and ‘homework’ to living in the real world.
CBT for OCD
Assessment
Although a structured interview protocol (such as the Structured Clinical Interview
for DSM-5 [SCID-5] or the Anxiety and Related Disorders Interview Schedule for
DSM-5 [ADIS-5]) can be useful, diagnostic tools are rather blunt objects and will
not in themselves yield sufficient detail about the person’s lived experience of OCD.
The publication of the American Psychiatric Association’s Diagnostic and
Statistical Manual (DSM-5) (2013) has been controversial since it removed OCD
from the anxiety disorders category and created a new listing of ‘ObsessiveCompulsive and Related Disorders’ which includes other loosely related problems
such as hoarding and skin picking. This has spurred various debates. Without
discussing the appropriateness of this decision, for the purpose of this chapter the
assertion is that psychiatric diagnoses do not indicate which treatment options
are beneficial.
Psychiatric diagnosis systems have frequently been criticized, not least because
they have little utility in the clinical setting since they do not indicate which
treatment options are beneficial (Kinderman et al., 2015). They often fail to take
into account psychological or sociological aspects.
Finding the meaning in the individual’s experiences and responses through the
use of formulation enables an understanding of what keeps the problem going
(Johnstone, 2018). This is far more useful in clinical practice, particularly since the
experience of OCD is unique to each person. It also helps to normalize rather than
pathologize the problem. The formulation should be drawn up collaboratively, using
the person’s own description and quite possibly their own words. For the OCD
sufferer, these invariably include ‘I worry that . . .,’ I feel anxious when . . .,’ and so
on. The treatment protocol described in this chapter is formulation driven.
Use of Assessment Measures: 1
An objective measure of the severity of symptoms is helpful to enable progress to be
monitored. As well as demonstrating validity and reliability as an outcome measure,
the following are of therapeutic use to identify key areas of difficulty.
Obsessive Compulsive Inventory – Revised (OCI-R)
The OCI-R (Foa et al., 2002) continues to demonstrate good reliability as a selfreport screening measure (Wootton et al., 2015).
Yale–Brown Obsessive Compulsive Scale (Y-BOCS)
The Y-BOCS (Goodman et al., 1989) continues to be considered the ‘gold standard’
for OCD assessment, although a newer version has been developed (Rapp et al.,
2016). Administered by the therapist, its checklist of obsessions and compulsions is
particularly helpful as a prompt for less troublesome concerns that may
go unreported.
227
228
Elizabeth A. Forrester
Ratings of mood and anxiety are also helpful additions. While the Beck Anxiety
Inventory (BAI) and Beck Depression Inventory (BDI) are excellent measures,
they create an unnecessary burden as part of a wider battery of questionnaires.
Therefore, brief measures are sufficient, unless clinically indicated (Kroenke et al.,
2001; Spitzer et al., 2006).
How Much? How Often?
Measures should be kept to a minimum and do not need to be repeated each
session. From clinical experience, a significant number of OCD patients have difficulties completing questionnaire measures; thus, there is a paucity of research on
this topic. Many checkers, for example, may take a long time to fill in self-report
measures due to urges to check or indecisiveness about how to score an item. Whilst
beginning and end of therapy would be the minimum data set, consider using
assessment measures at pivotal points during treatment where change has clearly
occurred. If follow-up appointments are offered once active therapy has ended,
measures can also be used to track maintenance.
Use of Assessment Measures: 2
There may be occasions during treatment when other measures may be used to rate
specific features of clinical presentation such as responsibility appraisals, the importance of thoughts, or thought-fusion. The following are useful measures:
Responsibility Attitudes Scale (RAS) (Salkovskis et al., 2000)
Metacognitions Questionnaire (MCQ-30) (Wells & Cartwright-Hatton, 2004)
(in Wells, 2009)
Thought Fusion Instrument (TFI) (Wells et al., 2001) (in Wells, 2009).
Obtaining an Overview
Without a feel for both the content and extent of the problem, treatment might miss
some important aspects that could hamper progress. OCD is the most idiosyncratic
of the anxiety disorders, with many subtleties and differences. Not all contamination
fears are about dirt and germs, in the same way that not all checkers worry about
leaving the gas on. A familiarity with the many different ways in which OCD
presents and can affect a person’s life is essential in order to understand the most
appropriate questions to ask to assist in making a rudimentary formulation.
Case Vignette
Ask any of his colleagues and they’d say that Matthew was a brilliant paramedic.
Professional and knowledgeable, yet thorough and calm in a crisis. He never balked
when called to an emergency, no matter how gory. Inside his head, however, and
CBT for OCD
especially away from work it was another story. His mind was constantly on alert for
potential dangers from bodily fluids or unknown diseases of the people he ministered to. He would keep a mental track of everything he’d handled or stepped on,
keep a close eye on what his colleagues touched, and was careful never to touch his
face after he’d removed the surgical gloves until he’d been able to thoroughly wash
them. At the end of a shift, he’d change into ‘civvies’ before going home to his wife
and children, stowing all his work clothes (right down to socks and underwear) in a
bag he kept specially for this purpose at the bottom of his locker. Once a week, he’d
make a trip to a launderette a few miles away from the ambulance station. Although
he had to make a special detour to get there, he figured it was worth it despite the
cost. He admitted he spent a small fortune on cleaning products and replacing many
items of clothing he’d simply got rid of because they seemed too dirty. Once he got
home, only after a thorough shower with chlorhexidine gluconate in the downstairs
shower room used exclusively by him, followed by a swift wipe-round with disinfectant wipes, was he ready to put his feet up and relax. After all, he couldn’t risk the
well-being of his family by bringing any source of contamination into the house.
The following provide the basis for useful lines of questioning:
Are there particular ‘rules’ about the way you do things or have to follow
certain rituals?
How does it impact on your daily living?
What other consequences does OCD cause?
Is there anything you don’t do because of this problem?
Rules and Rituals. An inability to vary routines or procedures is frequently a
feature. For example, hand washing is carried out equally meticulously regardless of
the activity that it follows (such as throwing a scrap of paper into an open waste bin,
putting away groceries, changing the baby’s nappy). Similarly, checking routines are
carried out thoroughly, whether it’s scribbling a note to remind themselves to buy
some milk on the way home or preparing their tax return. In other words, actions
taken do not reflect the individual needs of specific situations.
Impact on Daily Living. The severity of impact on daily living may vary greatly.
Subtle changes to routine or small avoidances have often crept in over time; the
initial reason for these changes has been lost as these alterations become absorbed
as the usual way of doing things, for example, using paper kitchen roll instead of a
tea towel, keeping windows and doors locked when at home, storing cleaning
products in a locked cupboard even though the ‘children’ are adults. Without
over-pathologizing habits and practices, the therapist must develop an awareness
of subtle behaviors that may need addressing as therapy progresses.
Unwanted or Unintended Consequences. Contamination fears can unexpectedly lead to quite the opposite. Routine domestic chores are so terrifying or timeconsuming that they are avoided; for example, clutter accumulates when rubbish
can’t be put out for fear of contamination or losing something.
There may be financial costs. With contamination concerns, a lot of money may
be spent on cleaning products and toiletries, or replacing items discarded as unclean
229
230
Elizabeth A. Forrester
or simply worn out from over-cleaning. Vigorous checking can damage locks,
handles, or taps, while worries about potential dangers from items (like domestic
appliances) may mean they are regularly replaced for a newer version that promises
enhanced safety features.
Impact on Activities. OCD may restrict leisure and social life, leaving little
scope for pleasure and enjoyment. With checking and contamination worries,
activities such as leaving the house for any length of time, travel, or eating out are
affected. Avoidance may not be deliberate: there may not be time to do anything
beyond rituals and compulsions.
Identifying Covert Compulsions. Because these happen internally, the individual may not recognize them as behaviors. These actions may not even seem to be
conscious and often are not differentiated from the experience of thoughts.
However, overlooking covert compulsions is often a major reason for treatment
failure (Pence et al., 2010). Common covert strategies include the following:
Counting whilst carrying out a compulsion
‘Should’ statements, for example, ‘It should be okay’
Repeating special words, images, or numbers
Reviewing actions
Mentally ‘erasing’ an unwanted thought or image (e.g., trying to picture
something clean)
Completing a mental check list (e.g., ‘I hung my bag on the back of the
washroom door, tore off the first six sheets of toilet paper, covered the seat
with toilet paper, didn’t touch the walls’)
Create mental ‘paper trail’ tracking actions for future reference (to make them
more memorable – different to reviewing actions, as this is either whilst activity
taking place or very soon after)
More detailed accounts of the clinical assessment can be found in Forrester
(2015) and Van Niekerk (2018).
From Assessment to Formulation
A comprehensive clinical picture in cognitive behavioral therapy requires more than
a list of symptoms. The effects of OCD are far-ranging and affect many aspects of
the person’s life. As an initial between-sessions task, the therapist should ask for a
(written) list of the following:
Things you do too much
Things you do too little of
Anything you avoid.
While there will inevitably be some replication of compulsions discussed during
assessment, it also gives an idea of how the client’s world might have shrunk or be
compromised by the problem. An effective intervention needs to take this into
account. Whilst many behaviors are self-explanatory, others are less so. Lavinia
CBT for OCD
avoids listening to the radio so she would be able to hear if the dog was in difficulty.
She prefers not to go out as she worries so much about leaving the dog at home
(even though she puts him in a cage). In addition, she read that many things are
toxic to dogs – such as certain flowers and food stuffs. This means she doesn’t like to
cook at home and takes precautions after preparing meals. An awareness of the
wider picture is useful when considering goals for therapy.
Overview of Treatment
CBT for OCD should be considered as a brief, time-limited intervention even when
the problem is severe and/or chronic. This approach is generally delivered over
12–15 sessions of an hour’s duration. For optimal impact, the first few sessions
should occur weekly while the patient is socialized into the model and developing
an understanding of both what maintains the problem and what they need
to change.
Frequency of subsequent therapy sessions is at the discretion of the therapist;
more time may be required for therapeutic tasks to be practiced between sessions
and (towards the end of therapy especially) to foster autonomy and independence
prior to discharge.
Additional time may be necessary when in vivo tasks are carried out during a
session. The approach can be condensed hours into a briefer time frame without
compromising outcomes (Challacombe & Salkovskis, 2011; Oldfield et al., 2011).
This may be particularly appropriate where timing of an intervention is an important factor, such as impending parenthood or physical health considerations.
Structure of Treatment
Following initial assessment, structure of treatment can be broadly considered as
having the following five phases:
1. Formulation
2. Developing a less threatening view of the problem
3. Focusing on change using a combination of behavioural and cognitive
interventions
4. Building on progress
5. Maintaining change and preventing relapse.
Case Formulation
OCD can seem so random and senseless to the patient that they’re left puzzling
about what it all means. Case formulation (see Figure 12.1) is the key to developing
a shared understanding of the problem. The following steps create a cohesive thread
through what’s often nicknamed the ‘vicious flower’:
1. Can you recall a recent situation that triggered one of your worries (or intrusive
thought)?
2. What thought did that trigger?
231
232
Elizabeth A. Forrester
2. Intrusive thought, urge, image
or doubt
•
Image of houses on fire
1. Situation
Leaving the house to take
my daughter to school
Did my routine check
5. Neutralizing
• Checking
• Use random
word to make it
memorable
• Take photo of
hob and door
4. Feelings
• Anxious
• Tearful
• Feeling sick
3. What this thought meant to
me
e.g., Why I believed I shouldn’t ignore it
•
6. Counterproductive
safety-seeking
behaviors
• Try to feel
completely
certain
• Try not to have
any doubts ( or
horrible
images)
•
I can’t trust my
judgment
If something went
wrong I couldn’t live with
the guilt
7. Attention and
Reasoning biases
• Looking for
trouble: I notice
all kinds of
potential
dangers
• Emotional
reasoning: If it
feels dangerous
it must be
dangerous
Figure 12.1. Case formulation.
3. Why did the thought seem so important to you at that moment? What made it
hard to ignore or dismiss it?
4. How did it make you feel? Did you notice any physical sensations?
5. What did you do to make yourself feel better? (Prompt for overt and covert
rituals)
6. Is there anything that you did to help keep you safe?
7. Are there particular attention or reasoning biases?
Situation. Although invariably the person may try to describe a number of
situations (especially if they have a variety of obsessional concerns), emphasize
the importance of just discussing a recent one. The rationale for this is that they
will have a clearer recall for details, which will be helpful for the formulation.
CBT for OCD
Intrusive Thought. Thoughts may also occur in the form of doubts, urges,
or images.
Meaning. The meaning isn’t always readily accessible, often because the person
immediately engages in safety behaviors to prevent the occurrence of beliefs such as
being responsible for harm or being a dangerous person. Stopping or delaying a
compulsion will usually elicit the meaning.
Feelings. As well as emotion responses such as anxiety, anger, or even depression, include physical feelings (e.g., tense, hot) and any mental references such as
stressed, doubtful, or a sense of something terrible about to happen. Although there
is no research evidence to date, clinical observation suggests that, in a similar way in
which panic results from catastrophic misinterpretations of normal bodily sensations, it is possible that a person with contamination OCD may misinterpret the
sensations of their sweating palms (an anxiety response) as feeling dirty. Similarly,
frequent washers often have extremely dry skin on their hands and become sensitized to changes to the feel of their skin.
Neutralizing. Any action (i.e., compulsion) to counteract the thought or reduce
anxiety is neutralizing. The link between the neutralizing action and the harm it is
intended to prevent may not be a direct one, for example, superstitious behaviors such
as touching wood, or other idiosyncratic actions. Overt compulsions such as cleaning,
checking, or repeating are outwardly observable. Covert actions (such as mental
debate or thinking a good thought after a bad one) may be overlooked as behaviors.
Counterproductive Safety-Seeking Behaviors. Many safety-seeking behaviors
are in fact counterproductive. Attempts to suppress thoughts lead to further intrusions. Striving to be perfectly clean, safe, or certain is attempting to achieve the
impossible. Avoiding, say, cleaning the bathroom due to worries about toilet germs
means the bathroom gets dirtier from neglect.
Attention and Reasoning Biases. Attention is drawn towards things that have
personal relevance or importance. Thus, the patient is more likely to notice threat
cues, and reason that this is further evidence for how dirty or dangerous the world is.
Reasoning bias plays a role in another way: feeling anxious can be misinterpreted as
a sign of danger.
More detailed guidance to developing the vicious flower formulation with the
patient can be found in Forrester (2015) or Bream et al. (2017).
Socialization to the Model
So it isn’t the thought (doubt, image or urge) that’s the main problem, but what you
believe it means – and what you do in response to it. While various illustrations of
how thought suppression is ineffective (try not to think of a polar bear), the
following helpfully demonstrates the interplay between thoughts, emotions, and
behavior and where it is possible (as well as impossible) to intervene:
Imagine a masked gunman bursts into the room and puts a pistol to your head and
shouts, ‘Don’t have any scary thoughts!’ Would you be able to stop such thoughts?
233
234
Elizabeth A. Forrester
I reckon not. And if they said, ‘And don’t feel frightened!’ could you prevent yourself
from feeling afraid? Again, highly unlikely. But imagine they then demanded you sang a
nursery rhyme and danced to the tune, could you do that?
While it isn’t possible to control what comes into our mind or prevent our emotional
response, we are able to make choices over our actions.
Instead of trying to persuade yourself that things are safe or clean before
changing what you do – change behavior and belief change follows.
Features of Treatment
Shared understanding of maintenance factors
Changing relationship with thoughts and feelings
Exposure third wave style
Tackling rumination and overthinking
Use of anti-OCD.
Will There Be Homework?
As all CBT therapists know, therapy doesn’t begin and end in the therapist’s office.
What the patient does between sessions is even more important:
Therapy for OCD is like going to a slimming club. It’s helpful to go along each week, get
some helpful tips, recipes, and encouragement to help you lose weight. If you keep eating
cakes, chips, and chocolate in between times, nothing will change – you might even gain
more weight. It won’t always be easy, but we can work together to help you stick to
the diet.
The idea of ‘homework’ suggests a specific, discrete activity separate to activities of daily living (including those aspects defined by OCD). It’s often put off until
the last minute. To continue the slimming club analogy, the process of therapy is
akin to healthy eating and lifestyle rather than a crash diet. This is a more holistic
approach which facilitates individuals to thrive, as opposed to focusing on the
absence of symptoms or psychopathology (Hayes & Hofmann, 2017). In other
words, there won’t be homework, but there will be something far more dynamic:
a plan of action.
Plan of Action: From Formulation to Treatment
Since the formulation is based mainly on a specific occurrence of OCD, it is not
certain whether it has explanatory power for other situations. Hence the patient is
asked to draw up some further formulations before the next session when OCD
occurs. This is particularly useful when there are other kinds of presentation.
A helpful way to phrase it is: ‘Let’s see if it still makes sense when OCD affects
you in other ways, with other kinds of concerns or if we need to revise it in any way.’
In addition to ascertaining that the person understands the maintenance cycle,
the process of change can begin by suggesting they either resist a compulsion to gain
CBT for OCD
235
further insight (it is possible they haven’t done this for a long while) or simply do
something differently.
A Picture Paints a Thousand Words
Although particulars of routines, rituals, and compulsions will have been obtained
at the initial meeting (and added to as further features emerge during treatment),
direct observation provides a wealth of detail in a glance. It may be possible to carry
out an exposure task in the consulting room, for example, touching something
‘dirty’, or at the very least to demonstrate how a particular ritual is carried out,
for example, hand washing. A home visit can be invaluable (see Table 12.2). Of
course, this is not always practicable, in which case a step-by-step breakdown (even
‘miming’ a routine) can be done.
Developing a Less Threatening View of the Problem
The key to understanding OCD and what keeps it going is the meaning attached to
the occurrence of particular intrusive thoughts, and the implications this has in terms
of what is required to prevent this dreaded outcome. By continuing to take
Table 12.2. A picture paints a thousand words
Presenting problem: Justyna describes significant worries about contamination, that dirt is brought into her
home from outside; in particular, she fears insect infestation. ‘What really gets me down is that vacuuming
takes hours. It’s really exhausting.’
Therapist’s mental picture: So, we have a spotlessly clean house with every room carefully vacuumed, right
up to the edges. No wonder it’s exhausting if the furniture – including beds, sofas, and wardrobes – is moved
every time in order to clean beneath and not a bit of fluff or dirt around the skirting boards.
The reality: On a visit to the home, the therapist is ushered to the only chair that does not have things piled
on it. Justyna explains that the plastic boxes on the other chairs and sofas contain items she fears may be
contaminated, which (in time) she plans to deal with but she’s too stressed at the moment to take on the task.
She moves one of the boxes so she can sit down. Embarrassed, she admits that another reason for putting
stuff everywhere is so she can control where the therapist would be able to sit. She has also placed throws on
every chair because they can be washed more easily than the upholstery. Prior to the visit, Justyna agreed
that she would demonstrate how she did the vacuuming. The therapist observed that the carpet did not look
clean, and there was a distinct border about 20–30 cm in breadth around the perimeter of the room and items
on the floor which was thick with dust and fluff.
Before commencing vacuuming, Justyna put on slippers she kept exclusively for the task – first wiping the
soles with a disinfectant wipe which she then discarded. A fresh wipe was used to wipe down the vacuum
cleaner, a precaution she always took just in case part of it had been in contact with something dirty (perhaps
the cable had touched a ‘dirty’ patch of floor). Often she had to repeat this if a doubt came into her mind, or if
she was interrupted while attending to disinfecting the appliance. She’d begin by vacuuming a small area of
carpet, stretching only as far as she could reach so her feet would not encroach on the newly cleaned area. By
wiping the soles of her slippers again, she could then proceed to the cleaned area and repeat the process on
the next patch. Meticulous care was taken not to get too close to furniture, walls, or other potential sources of
contamination, hence the unvacuumed margins.
236
Elizabeth A. Forrester
precautions in an attempt to feel safe or reduce risk, we never discover how
necessary these behaviors really are, which in turn reinforces the belief that they
are necessary actions.
Helga believed that her judgment was poor and she lacked ability to assess
safety or cleanliness in everyday situations.
Helga:
The thing is, I’ve had this problem for so long I just don’t know
whether or not things are clean enough, or if I’ve done something
that could put others at risk in some way. I’ve no idea how to do
things properly.
Therapist: Is it that you consider that your judgment is poor?
Helga:
Yes. I’m constantly wondering if I’ve done something wrong or
dangerous. I just couldn’t live with myself if I thought I had.
Therapist: So, it sounds like you have a theory about what your problem
is . . . that it would be dangerous or irresponsible to ignore the
thoughts?
Helga:
That’s right.
Theory A/Theory B
Theory A/Theory B is a common and effective tool for introducing alternative
explanations for OCD-driven thoughts and behavior (see Table 12.3). For more
details about developing Theory A/Theory B, refer to Forrester (2015), Bream et al.
(2017), or to Chapter 11.
The Right Solution for the Problem? If the problem was indeed harm or danger,
then all the actions the person has been taken would of course be sensible solutions. If
something toxic had been spilled, it’s entirely reasonable to wipe it up and clean the
area thoroughly. It’s appropriate to check whether any shards remained if some glass
smashed on the floor. However, it’s the wrong solution if the problem is worry about
danger. Ideas generated by asking, ‘What would we do if we were treating this as a
worry problem?’ help form the basis of the intervention and aims of treatment.
Already, change becomes a possibility. Previous precautions are no longer necessary.
Debunking the Myth of Graded Hierarchies. To grade or not to grade? That is
indeed the question. While exposure is the essential ingredient in any anxiety
treatment, consideration is required to optimize its efficacy (Jordan et al., 2017).
Reasons in support of not taking a stepped approach include the following:
The stepped approach is time consuming, especially if habituation is required
before moving to the next step.
Habituation does not fit the traditional neat reduction shown in
textbook graphs.
Habituation may not occur (some things will always induce a degree of fear or
discomfort).
The stepped approach reinforces the idea that some situations are more dangerous than others.
The stepped approach does not reflect real life.
CBT for OCD
237
Table 12.3. Theory A/Theory B
Theory A: My problem is that my judgement is poor
and I wouldn’t be able to live with myself if I’d
harmed someone (i.e., danger or harm problem)
Theory B: My problem is that I worry that my
judgement is poor and I wouldn’t be able to live with
myself if I’d harmed someone (i.e., a worry problem)
What I need to do
Act on every thought about danger or harm.
Identify all possible hazards or dangers.
Clean everything thoroughly.
Wash my hands a lot.
Keep clean and dirty things apart.
Use antibacterial products on everything to avoid
spreading my germs.
Give ‘dirty’ things a wide berth.
If I have a doubt, treat it as dirty.
Reduce/stop any potentially dangerous behaviours,
e.g., driving, buying ‘hazardous’ gifts, cooking for
others.
Allow others to make decisions to reduce my
responsibility.
Lock myself in the house so I can be sure I haven’t
done something.
Do things slowly and methodically so I can
remember exactly what I did.
What I need to do
Not do the things on Theory A list!
Allow thoughts and worries to come and go without
acting on them.
Don’t think too hard about hazards and dangers.
Clean and wash like most people do, e.g., just use a
damp cloth to wipe kitchen counters after cooking,
wash hands only when greasy or visibly dirty.
Sit down in public places, walk past bins, etc.
Mix things up at home, e.g., put shoes in the kitchen,
put coats and bags on the bed.
Drive, cook for the family, buy gifts such as glass,
candles, or other ‘hazardous’ items.
Make my own decisions even if they’re scary.
Deliberately go out several times each day (and not
lock myself in).
Do things in a breezy and relaxed way.
If I have a doubt, treat it as OCD!
Evidence for Theory A
Things are dirty and people get ill through contact
with dirt.
It feels dangerous.
Checking and cleaning makes me feel better.
I don’t know what good enough cleaning is like.
There have been times when I can’t remember
whether I’ve done something that could
be harmful.
Evidence for Theory B
Others don’t take the precautions I do yet don’t
seem to get ill more than me.
I get more of these worries when I’m stressed for
other reasons.
The focus of my worry changes – it’s often specific
and not logical.
People move on after bad things happen, even if it’s
something that they’ve done.
Most importantly, if viewing OCD as a worry problem (Theory B), there is no
need for it: a worry is a worry. Grading compulsions give them extra meaning
and power.
There is substantial evidence for the effectiveness of using random schedules of
exposure (e.g., Craske et al., 2008). Similarly, by increasing the range of contexts in
which new learning takes place, more retrieval cues are generated, which increase
the likelihood of the person remembering to make a different (more helpful)
response (e.g., Bjork & Bjork, 2006).
Although grading is unnecessary, it is still important to have targets to help
orientate the intervention. They provide a guide towards the patient’s desired
outcome: put simply, being able to do things the way most people do. See
Table 12.4 for an example.
238
Elizabeth A. Forrester
Table 12.4. OCD/non-OCD way
OCD way
Non-OCD way
Wash hands thoroughly before touching or feeding
my dog.
Read label on dog food carefully each time.
Rinse her dishes (and ours) several times to ensure
no detergent traces.
Don’t allow her in the garden unless I’m with her.
Keep cleaning products in cloakroom with the door
shut or locked in under-sink cupboard.
Check lights and appliances several times before
leaving the house.
Take a photo of the front door so I know it’s closed.
Keep all receipts to check against bank statement
each week.
Wash hands only when necessary.
Just pick up the can or packet and feed her.
Wash and dry dishes without rinsing.
Let her play in the garden while I’m at home,
either indoors or outdoors.
Leave door open to air the cloakroom, remove
child-lock from kitchen cupboards.
Switch things off as I go, don’t check (or only
have a quick glance).
No photos! Just lock and go.
Keep receipts only for high value items.
Check statement once a month only.
Exposure starts by doing things differently, changing habits, and learning from
those experiences. The advantages of approaching it in this way are the following:
1.
2.
3.
4.
5.
It’s more naturalistic and fits in with daily life.
It increases opportunities for exposure to other things.
It moves away from the idea of ERP as a discrete ‘homework’ activity.
There is always scope for doing something differently.
It increases cognitive flexibility.
The Role of Habit in OCD. ‘If you always do what you’ve done, you’ll always
have what you’ve always had’ is a useful adage for tackling OCD. The role of habit
in OCD has been widely investigated (e.g., Gillan et al., 2014). Such findings are
consistent with frequent clinical observations that people with OCD often engage in
compulsions ‘by accident’ rather than triggered by a threat appraisal or physiological arousal, when their intention had been to adhere to the treatment protocol of
ERP. The implications for treatment are that alternative instructions are needed
about how to respond in given situations.
It is not always easy to differentiate between an OCD-driven behavior and
habits, preferences, and conventions of individuals. It can be helpful to view everything we do on a continuum (e.g., Mataix-Cols et al., 2005). After all, an isolated
thought or action is not by itself an indicator of OCD, but when thoughts and
actions are observed in a cluster with other features, they can then be considered
symptoms. The fixedness of certain characteristics can be an indicator. For example,
repeated checking leads to it becoming an automatic response in OCD (Dek et al.,
2015). Anything that is done regularly and repeatedly becomes habit and the
default way of doing things. This applies not only to what we do but also to what
we don’t do:
I always
Take a photo of the door when I go to work.
Use a tissue to open the door in public places.
CBT for OCD
Check my car for signs of damage before and after a journey.
Closely examine the pavement for signs of blood, vomit, faeces.
I never
Use the same towel twice or share my partner’s.
Leave the kettle or microwave plugged in when not in use.
Wear clothes that can’t be washed.
Drive on motorways or dual carriage ways.
Safety Behaviors and Effective Exposure. While all responses to reduce anxiety, suppress unwanted thoughts, or otherwise ‘feel better’ should be considered
safety behaviors and therefore need to be targeted in therapy (e.g., Abramowitz,
2006), it can also be argued that any change in the right direction is good. Safety
behaviors can play an important role in encouraging engagement in exposure as
long as they do not interfere with learning that a feared situation is in fact innocuous
(Levy & Radomsky, 2016; Milosovic & Radomsky, 2008): ‘Charlene would have
been unable to take her child to a public toilet if she hadn’t been allowed to wash
her own hands thoroughly afterwards, and would not have discovered that her child
did not become ill as a consequence.’
Fears relate not only to external outcomes but also to the emotions the feared
stimulus elicits (Van den Hout et al., 2012). In Charlene’s case, it also imbued a
sense of control over her feelings of disgust but perhaps more importantly allowed
her to discover that these feelings did not persist (or escalate) but receded over time.
She discovered that she was able to cope with them.
Rationality, Risk, and Cognitive Restructuring. The clinical efficacy of exposure
therapy is enhanced by the addition of cognitive interventions (Rector et al., 2019).
Cognitive restructuring within the context of CBT for contamination and checking is
focused on appraisals of the likelihood of being able to cope with feelings of
discomfort (whether that be intrusive thoughts, physical sensations of anxiety, or
feelings of distress, doubt, or uncertainty).
The primary aim of therapy is not to prove or disprove the likelihood of feared
outcomes, although that may occur as a by-product of therapy. A major obstacle in
attempting to do so is that such concerns are not always amenable to testing: ‘Just
because I haven’t run someone over on this occasion doesn’t mean it won’t happen
in the future.’ However, the person may coincidentally discover that they do not lie
awake all night after resisting urges to check.
Box: Risk versus Uncertainty
Julia was uneasy about letting her son go on the school trip to London. Anything
could happen: the coach could crash, maybe he’d be abducted, there could be a
terrorist attempt. It couldn’t be guaranteed that they definitely wouldn’t – so there’s a
risk. Equally, there’s no guarantee that something similar wouldn’t happen if he were
kept at home for the day (or taken to an amusement park by his parents, as she’d
suggested). She was asked to weigh that risk against the certainty of him feeling left
out when his classmates chattered excitedly about their shared experience.
239
240
Elizabeth A. Forrester
Dichotomous Thinking and Overestimation of Threat. A tendency to dichotomous
thinking exacerbated obsessional worries. If it isn’t clean or hygienic, it must be dirty or
even contaminated. If we’re not convinced a situation is safe, it must be dangerous. The
notion of risk is exchanged for absolute certainty, where in reality a feared outcome is
only one of many possibilities (the majority of which may well be benign). ‘Any human
activity involves some risk to life or health. Although it is possible to reduce the existing
risk of a particular activity, it is not possible to reach the “zero risk” or “absolute safety”
that is often demanded. Once this general fact is recognized, it then becomes necessary
to define an acceptable level of risk’ (Black & Niehaus, 1980).
Constant striving for certainty is an attempt to achieve the impossible. Certainty
research often fails to differentiate between pathological and normative contexts
(Cougle & Lee, 2014). In an age of instant access to ‘facts’, intolerance of uncertainty is rife.
Risk Isn’t Only about Bad Things Happening. Just because something can
happen doesn’t mean it will happen. It’s also often overlooked that risk isn’t only
about bad things happening:
Shirley:
Therapist:
Shirley:
Therapist:
But what if I put the dirty laundry on the bed and I can’t get the
worry out of my head and my anxiety gets worse and worse?
Don’t forget that risk isn’t only about bad things happening. How
about taking the risk that by doing this you find it quite a
challenge and discover it’s not as hard as you think it’s going to
be, that you can cope with it. How would that feel?
Ha, you’re right. I know it’s just my glass-half-empty way of
looking at things. When I stopped washing my clothes with
disinfectant I thought I’d never be able to wear them and feel
comfortable, but actually it wasn’t that bad.
So, maybe it’s worth taking the risk that it could go well.
Thoughts and Thinking
Understanding the difference between thoughts (the event of having a thought) and
thinking (the process or action one takes in response to that thought) is a key factor in
overcoming obsessional concerns. Normalizing the occurrence of intrusive thoughts
is essential, but it is equally important for the patient to develop an understanding of
how thoughts come and go, ebb and flow without any apparent act of will or effort
from them. It is interesting to note that higher reports of spontaneous mindwandering are associated with increased OCD symptomatology (Seli et al., 2017),
yet participants in the study were less likely to make negative or threatening appraisals when they deliberately chose to allow their minds to wander. This is further
evidence, in fact, that attempts to control thoughts are counterproductive.
Thought Bingo
Encouraging patients to take a step back to observe what they’re actually doing is
crucial. It should not be overlooked that thinking is also a behavior, and one that is
CBT for OCD
often used in an attempt to reduce uncertainty or anxiety. An exercise
called Thought Bingo (see Appendix 12.1) can help the person discover the different ways in which they typically get caught up in rumination – the different ways
in which they think. Amongst many other possibilities, these are typical
manifestations:
Reasoning
Analyzing
Debating
Problem-solving
Imagining.
Helping people to learn about and understand their own thinking enables them
to gain a little critical distance from it, as well as an awareness of its role as a
maintaining factor.
Allowing Thoughts without Thinking
While thought suppression is impossible, developing the skill of allowing thoughts to
occur without thinking about them is essential. Overthinking leads to increased
worry as illustrated in the following.
My husband let the cat in last night. It just ran straight through the kitchen, into the hall
and up the stairs. I shouted at him to go and catch Misty while I checked the kitchen
floor. I couldn’t see anything obvious so I knelt down to have a closer look. There were a
few dark spots – I wasn’t sure what they were, they could have been anything. You’ve no
idea where they go when they’re outside. She goes to the toilet out there, other cats do
too . . . and foxes.. . . There could be anything on her paws. It’s disgusting. I can’t believe
he didn’t wipe her feet before letting her through. I cleaned the tiles with bleach and then
some disinfectant, but it still bothers me. I wanted to clean the carpet with it too but my
husband stopped me so I just scrubbed at it with a soapy cloth when he wasn’t looking.
Maybe we should get rid of the carpet. It’s just not hygienic.
When I eventually went to bed, I just couldn’t settle. What if Misty had been on the bed?
She might have run across the pillows. There are mice in the garden, and bird poo.. . . What
if some of the lawn fertilizer hadn’t dissolved? I put my pillows in the bath so they could be
washed in the morning. At least the sheet underneath them would still be okay.
The Role of Rumination
In the context of OCD, rumination refers to (ego-syntonic) repeated mental acts
(e.g., mental debate or argument) carried out in response to (ego-dystonic) intrusive
thoughts, doubts, or images in an attempt to reduce anxiety. Depressive rumination
dwells on causes, meanings, and implications of negative symptoms, feelings, and
problems with attempts to make sense of why something happened Whilst for both
it may be conceptualized as a mental habit, there is a strongly purposive element
in OCD rather than a passive focus on negative content (Watkins & NolenHoeksema, 2014).
241
242
Elizabeth A. Forrester
There is a growing body of evidence for rumination as a transdiagnostic feature
of both anxiety and depression (e.g., McEvoy et al., 2013). Indeed, the processes
have many similarities. However, in OCD the use of ‘thinking solutions’ is very
much a part of the problem and is more prevalent and distressing (Wahl et al.,
2011).
Rumination Is Never about Now
Rumination is more prevalent where concerns are connected with something that
has not yet happened (i.e., an imagined event in the future) or that happened in the
past that is not amenable to physically checking:
If I’m concerned there might be a hazardous substance in my house, it’s possible to
check – but if I have a doubt whether or not there was a hazardous substance in the
house I lived in several years ago which has now been demolished, I can’t.
That’s particularly true for doubts about abstract concepts, for example, ‘Do
I really love my partner?’ These kinds of moral questions – or questions about the
nature of ‘being’ – elicit strong feelings but, even more importantly, they have no
absolute answer. These things are not ‘knowable’ in the ‘fact’ sense (Forrester,
2017).
Rumination Increases Likelihood of Further Intrusions
Rumination prevents the natural ‘decay’ of memories of actual events. Repetition
creates memories of thoughts, whether or not the thought was factual. By regularly
bringing to mind a specific event associated with their obsessional concerns (e.g.,
losing a packet of tablets on the bus), it won’t just fade away to become a distant
memory (or even be forgotten about, as would happen with many autobiographical
memories that we don’t attribute a special significance to). Someone who doubts
whether they may have run someone over in their car pictures something that hasn’t
happened while trying to make themselves certain that it didn’t, and will recall
that image.
Rumination Prevents the Mind Wandering Freely
Selectively attending to and considering threat-related intrusions may prevent other
kinds of thoughts – nice or interesting or creative thoughts –that might distract them
from their thinking (e.g., Welz et al., 2018).
I could have cleaned the toilet with my husband’s toothbrush.. . . When did I last clean
it?. . . It was when Naomi popped in.. . . She’s so funny . . . what was that joke? Must ask
her again soon. I wonder where she got her dress from . . . such a lovely color.
Although it may seem as if they have no choice, they may be actively choosing
to attend to thoughts they consider important. This internal focus reduces cognitive
resources left for other tasks and has a detrimental effect on performance (Koch &
Exner, 2015).
CBT for OCD
Thinking Is not Doing!
Although rumination is universal, in OCD the tendency is to do only this and very
little else. In addition to addressing cognitive processes, the emphasis needs to
remain on behavioral change. Encouraging the question, ‘What could I do differently to challenge OCD?’ means every activity of daily living becomes
an opportunity.
It’s Not What You Do but How You Do It
We all wash, we all check, we all take care not to step in dog mess or to run
pedestrians down in the street. The difference between someone who struggles with
OCD and someone who doesn’t is a matter of degree. Rules are often used to create
certainty – although they are impossible criteria (what’s the perfect hand wash?).
What is needed is a lightness of touch. Focusing too hard, too closely on what you’re
doing is closely related to overthinking.
Initially, however, some new rules are required. Using the list of non-OCD
actions for guidance, these instructions might include:
Dry the laundry outside instead of indoors.
Cook a meal with chicken twice a week.
Decline receipts for purchases under £5.
Leave kitchen appliances plugged in and switched on at the wall.
Guidelines, not Rules!
Guidelines offer flexibility. Development of flexibility is encouraged: it’s nicer to be
able to use a well-kept toilet, but if you need the toilet and the only one available
has a floor swimming in goodness-knows-what it would be most people’s preference
to hitch up their trouser hems and hover over the seat rather than the discomfort of
trying to hold on (or the imminent likelihood of wetting themselves). On a less
graphic level, at home we might choose to wash an apple before eating it, but when
having fun at a picnic would just give it a quick ‘polish’ on our sleeve. Dichotomous
thinking is a feature of rigid rules: if I don’t do it the ‘right’ way it must be ‘wrong’; if
it’s not ‘safe’ it must be ‘dangerous’.
Building on Progress
Spending less time on rituals or compulsions can dramatically improve quality of
life, but it leaves scope for OCD to creep back in. Doing things the way that most
people do (i.e., the non-OCD way) is not enough.
How Far Is Far Enough?
In order to change unhelpful habits, it’s necessary to act as if the less threatening
alternative is true, rather than wait until we believe it or feel confident. After all,
confidence results from doing the kinds of things that lead to anxiety/distress. Not
only does doing more extreme things expand the person’s comfort zone; it means
they are taking a proactive role in tackling OCD. Rather than simply viewing the
243
244
Elizabeth A. Forrester
absence of obsessive thoughts as desirable, it should be also viewed as a potential
indicator that the patient needs to push themself to do more. A timely reminder may
be needed that exposure to thoughts is what is required.
Anti-OCD
More intensive exposure leads to greater reductions in anxiety levels and fear
beliefs (Norberg et al., 2018). While this does not mean doing more dangerous
things (although, as previously stated, nothing is without risk), it requires exposure
to more challenging thoughts. By introducing a modicum of playfulness in therapy,
creative anti-OCD ideas can be generated (see Table 12.5). Initial resistance is often
encountered with the patient concerned that they will have to do any of the things
Table 12.5. OCD/non-/anti-OCD
OCD way
Non-OCD way
Anti-OCD way
Avoid walking near dog poo or
even looking at it
Walk in areas where dogwalkers go.
Go for a picnic and sit on the
grass.
Unplug all appliances off before
leaving the house.
Leave the kettle, TV, alarm
clock, and table lights plugged
in all the time.
Ask mum to check my mail before
shredding it.
Decide for myself what to
throw out. Shred only bank
statements.
Read label on dog food carefully
each time.
Rinse her dishes (and ours)
several times to ensure no
detergent traces are left.
Just pick up the can or packet
and feed her.
Wash and dry dishes without
rinsing.
Check if I get aroused by images
of men or women.
Avoid any films or TV that might
feature homosexuality.
Try not to notice anything that
looks like male genitalia.
Be friendly and chatty with
both sexes without wondering if
I fancy them.
Watch whatever I want to.
Deliberately step on dark marks on
the pavement.
Make some chocolate cakes that
look like dog poo for my son.
Print pictures of dog poo and put
our shoes on them.
Switch lights, TV, radio, oven,
kettle, and toaster on and go out
for the afternoon.
Use an image of a burning house as
a screensaver on my phone.
Put some bank or credit card
statements in recycling without
tearing them.
Write my bank account number on
a piece of paper and leave it on the
bus.
Stick ‘Poison/toxic’ symbols on the
packets.
Deliberately leave a dollop of
detergent froth on dishes.
Leave a bleach bottle on the floor
where she eats.
Play ‘Glad to Be Gay’ on my
MP3 . . . on repeat.
Agree with my thoughts.
Deliberately watch Broke Back
Mountain and ‘Queer Eye’.
Buy a phallic shaped cactus for my
desk and call it Dick.
CBT for OCD
245
they have suggested. While it is partly a brain-storming exercise, it serves an
important function:
Considering possibilities is in itself exposure
Writing them down facilitates repeated exposure.
Outcomes from agreeing to complete anti-OCD tasks can be surprising, for
example, inability to eat a sandwich that had been placed on a picture of raw
chicken which they had purposely printed moments earlier. Such difficulties indicate
further need for discussion of the nature of thoughts; that is, thoughts about danger
are not the same as actual danger.
Are We Nearly There Yet?
Throughout therapy, the concept of exposure is maintained as an ongoing process,
that is, a series of steps towards a goal. The size of the steps taken can vary. Of
course, if only smaller steps are taken, change will take longer, but it also means
there is always something the person can do to take them in the right direction.
A dialogue between therapist and patient is presented below.
What Would You Do?
Patient:
Therapist:
Patient:
Therapist:
Patient:
I couldn’t imagine actually sitting on the seat in a café toilet!
If that seems a bit too challenging at the moment, what could you
do that would move you in the right direction?
Well, I’m not sure . . . it’s just really hard. Maybe I could hover, or
put some tissue on the seat?
That sounds like a good compromise. Of course, if you’re able to
just sit on it, that’s great. But if not, at least you’d still be using a
public loo.
It would be brilliant if I could do that. At the moment I can’t go
out for more than a short time because I’m petrified I’ll need
a wee
Yet the goal of living life free from OCD does not have a clearly defined end.
Earlier in this chapter, therapy was likened to going to a slimming club. To continue
with this analogy would be to describe the process as a diet for life – hardly
appealing! The following is a more palatable parallel.
You join the gym because you would like to lose weight and get fitter. It isn’t always
enjoyable – it’s often really hard work, it makes you hot, sweaty, and aching. Some days
you’re slower on the treadmill than others. But you keep going because you accept that is
what you need to do to keep trim and stay fit. When you’re feeling fitter and looking
trimmer, would you stop going and stay at home watching TV whilst eating crisps and
chocolate?
246
Elizabeth A. Forrester
Maintaining Change and Preventing Relapse
A therapy blueprint is generated over the last couple of sessions to summarize key
learning points. It includes an example of the formulation, initial beliefs along with
the less threatening alternative view of the problem, and a brief account of behavioral changes along with what the client learned. It includes what they need to do to
maintain progress, how to detect signs of relapse, and how to deal with it. A copy of
the blueprint is presented in Appendix 12.2. Further guidance can be found in
Forrester (2015). OCD ‘Rules’ are shown in Appendix 12.3.
Looking at the Problem from the Other Side of Therapy
We’ll always have worries.
To be human is to feel anxious sometimes.
Some thoughts will always be horrible, scary, unpleasant.
These thoughts are more likely to keep coming back if I try not to have them.
It’s fine to feel anxious.
We don’t need to ‘fix’ anxiety for it to go away.
Coping doesn’t necessarily feel great.
Confidence comes from doing things differently despite how I feel.
The world may be a dirty and dangerous place, but it doesn’t matter as much as
I used to think
If I’m willing to accept doubts, worries, and discomfort, I can live my life how
I please.
Summary and Conclusion
There is not just one truth but many. Theories abound over how contamination
concerns develop, the role of disgust, factors that lead to prolonged checking, and so
on. Research seeks to simplify, but in clinical practice we encounter multifaceted
presentations; thus, the breadth of research findings can seem overwhelming. While
theory should inform practice and provide a general direction, it should not constrain it. Therapists (like their patients) should avoid becoming absorbed in finding
The Way.
Inclusion of third wave therapy techniques allows the effective therapist to hold
many thoughts (and theories) lightly. It allows us to hover above such details to
achieve an overarching view of OCD, that is, that too much importance and
attention are given to thoughts and feelings, and that behavior change is possible
in their presence. The emphasis on change as a process is powerful: keep on living
until you feel alive is a pithy adage, in the absence of continual checking of course.
This chapter explored what is required for exposure to be really effective, why
we should redefine goals and success, and how to utilize creativity and playfulness to
help clients free themselves from the grip of OCD as part of evidencebased practice.
CBT for OCD
FURTHER READING FOR THERAPISTS
Bream, V., Challacombe, F., Palmer, A., & Salkovskis, P. (2017). Cognitive behaviour therapy
for OCD. Oxford University Press.
van Niekerk, J. (2018). A clinician’s guide to OCD. New Harbinger Publications.
FURTHER READING FOR PATIENTS
Challacombe, F., Salkovskis, P. M., & Oldfield, V. B. (2011). Break free from OCD:
Overcoming obsessive compulsive disorder with CBT. Vermilion.
Forrester, E (2015). How to deal with OCD. John Murray Learning, Hodder & Stoughton.
REFERENCES
Abramowitz, J. (2006). Understanding and treating obsessive-compulsive disorder:
A cognitive-behavioral approach. Lawrence Erlbaum.
Abramowitz, J. S., & Arch, J. J (2014). Strategies for improving long-term outcome in
obsessive-compulsive disorder: Insights from learning theory. Cognitive and Behavioral
Practice, 21, 20–31.
Alcolado, G. M., & Radomsky, A. S. (2016). A novel cognitive intervention for compulsive
checking: Targeting maladaptive beliefs about memory. Journal of Behavior Therapy &
Experimental Psychiatry, 53, 75–83. https://doi.org/10.1016/j.jbtep.2015.02.009
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
Binnie, J. (2015). Do you want therapy with that? A critical account of working within
IAPT. Mental Health Review Journal, 20(2), 79–83. https://doi.org/10.1108/MHRJ-112014-0044
Bjork, R. A., & Bjork, E. L. (2006). Optimizing treatment and instruction: Implications of a
new theory of disuse. In L.-G. Nilsson & N. Ohta (Eds.), Memory and society:
Psychological perspectives (pp. 116–140). Psychology Press.
Black, S. C., & Niehaus, F. (1980). How safe is ‘too’ safe? IAEA Bulletin, 22(1).
Bream, V., Challacombe, F., Palmer, A., & Salkovskis, P. (2017). Cognitive behaviour therapy
for OCD. Oxford University Press.
Calkins, A. W., Berman, N. C., & Wilhelm, S. (2013). Recent advances in research on
cognition and emotion in OCD: A review. Current Psychiatry Reports, 15, 357. https://
doi.org/10.1007/s11920–013-0357-4
Challacombe, F. L., & Salkovskis, P. M. (2011). Intensive cognitive-behavioural treatment for
women with post-natal obsessive-compulsive disorder: A consecutive case series.
Behaviour Research & Therapy, 49(6–7), 422–426.
Coles, M. E., & Ravid, A. (2016). Clinical presentation of not-just right experiences (NJREs)
in individuals with OCD: Characteristics and response to treatment. Behaviour Research
and Therapy, 87, 182–187. https://doi.org/10.1016/j.brat.2016.09.013
Coughtrey, A. E., Shafran, R., Knibbs, D., & Rachmann, S. (2012). Mental contamination in
obsessive-compulsive disorder. Journal of Obsessive-Compulsive and Related Disorders,
1(4), 244–250.
Cougle, J. R., & Lee, H.-J. (2014). Pathological and non-pathological features of obsessivecompulsive disorder: Revisiting basic assumptions of cognitive models. Journal of
Obsessive-Compulsive and Related Disorders, 3(1), 12–20. https://doi.org/10.1016/j.jocrd
.2013.11.002
Craske, M. G., Kircanski, K., Zelikowsky, M., Mystkowski, J., Chowdhury, N., & Baker, A.
(2008). Optimizing inhibitory learning during exposure therapy. Behaviour Research and
Therapy, 46(1), 5–27. https://doi.org/10.1016/j.brat.2007.10.003
247
248
Elizabeth A. Forrester
Dek, E. C. P., Van den Hout, M. A., Engelhard, I. M., Giele, C. L., & Cath, D. C. (2015).
Perseveration causes automisation of checking behaviour in obsessive-compulsive disorder. Behaviour Research & Therapy, 71, 1–9. https://doi.org/10.1016/j.brat.2015.05.005
Fergus, T. A. (2014). Are ‘not just right experiences’ (NJREs) specific to obsessivecompulsive symptoms? Evidence that NJREs span across symptoms of emotional disorders. Journal of Clinical Psychology, 70, 353–363. doi:10.1002/jclp.22034
Foa, E. B., Huppert, J. D., Leiberg, S., Langner, R., Kichic, R., Hajcak, G., & Salkovskis,
P. M. (2002). The obsessive-compulsive inventory: Development and validation of a
short version. Psychological Assessment, 14(4), 485–496. https://doi.org/10.1037//10403590.14.4.485
Forrester, E (2015). How to deal with OCD. Hodder & Stoughton.
(2017). Working with obsessional ruminations in clinical practice. Invited paper. Getting
stuck in repetitive negative thoughts: A transdiagnostic phenomenon illuminated from
different perspectives: Symposium on Repetitive Negative Thinking (RNT). 30 March.
University of Basel.
Gillan, C. M., Morein-Zamir, S., Urcelay, G. P., Sule, A., Voon, V., Apergis-Schoute, A. M.,
Fineberg, N. A., Sahakian, B. J., & Robbins, T. W. (2014). Enhanced avoidance habits in
obsessive-compulsive disorder. Biological Psychiatry, 75(8), 631–638. https://doi.org/10
.1016/j.biopsych.2013.02.002
Goodman, W. K., Price, L. H., Rasmussen, S. A., Mazure, C., Fleischmann, R. L., Hill, C. L.,
Henninger, G. R., Charney, D. S. (1989). The Yale–Brown Obsessive Compulsive Scale:
Part I. Development, use, and reliability. Archives of General Psychiatry, 46, 1006–1012.
Hayes, S. C., & Hofmann, S. G. (2017). The third wave of cognitive behavioral therapy and
the rise of process-based care. World Psychiatry: Official Journal of the World Psychiatric
Association (WPA), 16(3), 245–246. doi:10.1002/wps.20442
Johnstone, L. (2018). Psychological formulation as an alternative to psychiatric
diagnosis. Journal of Humanistic Psychology, 58(1), 30–46. https://doi.org/10.1177/
0022167817722230
Jordan, C., Reid, A. M., Guzick, A. G., Simmons, S., & Sulkowski, M. L. (2017). When
exposures go right: Effective exposure-based treatment for OCD. Journal of
Contemporary Psychotherapy, 47, 31–39. https://doi.org/10.1007/s10879–016-9339-2
Kim, S.-K., McKay, D., Taylor, S., Tolin, D., Olatunji, B., Timpano, K., & Abramowitz, J.
(2016). The structure of obsessive-compulsive symptoms and beliefs: A correspondence
and bi-plot analysis. Journal of Anxiety Disorders, 38, 79–87. https://doi.org/10.1016/j
.janxdis.2016.01.003
Kinderman, P., Read, J., Moncrieff, J., & Bentall, R. P. (2013). Drop the language of disorder.
Evidence-Based Mental Health, 16, 2–3. http://dx.doi.org/10.1136/eb-2012-100987
Koch, J., & Exner, C. (2015). Selective attention deficits in obsessive-compulsive disorder:
The role of metacognitive processes. Psychiatry Research, 225(3), 550–555. https://doi
.org/10.1016/j.psychres.2014.11.049
Kroenke, K., Spitzer, R. L., & Williams, J. B. (2001). The PHQ-9: Validity of a brief
depression severity measure. Journal of General Internal Medicine, 16, 606–613.
Levy, H. C., & Radomsky, A. (2016). Are all safety behaviours created equal? A comparison of
novel and routinely used safety behaviours in obsessive-compulsive disorder. Cognitive
Behaviour Therapy, 45(5), 367–379. https://doi.org/10.1080/16506073.2016.1184712
Ludvik, D., et al. (2015). Effective behavioural strategies for reducing disgust in
contamination-related OCD: A review. Clinical Psychology Review, http://dx.doi.org/10
.1016/j.cpr.2015.07.001
Mataix-Cols, D., Rosario-Campos, M. C., & Leckman, J. F. (2005). A multidimensional
model of obsessive-compulsive disorder. American Journal of Psychiatry, 162(2),
228–238.
CBT for OCD
Mathes, B. M., McDermott, K. A., Okey, S. A., Vazquez, A., Harvey, M. J., & Cougle, J. R.
(2019). Mental contamination in obsessive-compulsive disorder: Associations with contamination symptoms and treatment response. Behavior Therapy, 50(1), 15–24.
McCubbin, R. A., & Sampson, M. J. (2006). The relationship between obsessive-compulsive
symptoms and appraisals of emotional states. Journal of Anxiety Disorders, 20(1), 42–57.
https://doi.org/10.1016/j.janxdis.2004.11.008
McEvoy, P. M., Watson, H., Watkins, E. R., & Nathan, P. (2013). The relationship between
worry, rumination, and comorbidity: Evidence for repetitive negative thinking as a
transdiagnostic construct. Journal of Affective Disorders, 151(1), 313–320. https://doi
.org/10.1016/j.jad.2013.06.014
Melli, G., Aardema, F., & Moulding, R. (2016). Fear of self and unacceptable thoughts in
obsessive-compulsive disorder. Clinical Psychology & Psychotherapy. 23(3), 226–235.
https://doi.org/10.1002/cpp.1950
Milosovic, I., & Radomsky, A. S. (2008). Safety behaviour does not necessarily interfere with
exposure therapy. Behaviour Research & Therapy, 46(10), 1111–1118. https://doi.org/10
.1016/j.brat.2008.05.011
Norberg, M., Newins, A., Jiang, Y., Xu, J., Forcadell, E., Alberich, C., & Deacon, B. (2018).
The scarier the better: Maximizing exposure therapy outcomes for spider
fear. Behavioural and Cognitive Psychotherapy, 46(6), 754–760. doi:10.1017/
S1352465818000437
O’Connor, K., Koszegi, N., Aardema, F., van Niekerk, J., & Tallon, A. (2009). An inferencebased approach to treating obsessive-compulsive disorder. Cognitive and Behavioural
Practice, 16, 420–429.
Oldfield, V., Salkovskis, P. M., & Taylor, T. (2011). Time-intensive cognitive behaviour
therapy for obsessive-compulsive disorder: A case series and matched comparison group.
British Journal of Clinical Psychology, 50(1), 7–18.
Pence, S. L., Sulkowski, M. L., Jordan, C., & Storch, E. A. (2010). When exposures go wrong:
Trouble-shooting guidelines for managing difficult scenarios that arise in exposure-based
treatment for obsessive-compulsive disorder. American Journal of Psychotherapy, 64(1),
39–53.
Rapp, A. M., Bergman, R. L., Piacentini, J., & McGuire, J. F. (2016). Evidence-based
assessment of obsessive-compulsive disorder. Journal of Central Nervous System
Disease, 8, 13–29. doi:10.4137/JCNSD.S38359
Rector, N. A., Richter, M. A., Katz, D., & Leybman, M. (2019). Does the addition of
cognitive therapy to exposure and response prevention for obsessive compulsive disorder enhance clinical efficacy? A randomized controlled trial in a community
setting. British Journal of Clinical Psychology, 58(1), 1–18.
Riskind, Ayers & Wright (2007). Simulated interpersonal provocation and fears of a loss of
impulse control as determinants of aggressive mental intrusions. Journal of Cognitive
Psychotherapy, 21, 285–294. doi: 10.1891/088983907782638752
Robinson, L. J., & Freeston, M. H. (2014). Emotion and internal experience in obsessive
compulsive disorder: Reviewing the role of alexithymia, anxiety sensitivity and distress
tolerance. Clinical Psychology Review, 34(3), 256–271. https://doi.org/10.1016/j.cpr.2014.03
.003
Romero-Sanchiz, P., Nogueira-Arjona, R., Godoy-Ávila, A. Gavino-Lázaro, A., & Freeston,
M. H. (2015). Narrow specificity of responsibility and intolerance of uncertainty in
obsessive-compulsive behavior and generalized anxiety symptoms. International
Journal of Cognitive Therapy, 8(3), 239–257. https://doi.org/10.1521/ijct_2015_8_04
Ruscio, A., Stein, D., Chiu, W., & Kessler, R. (2010). The epidemiology of obsessivecompulsive disorder in the National Comorbidity Survey Replication. Molecular
Psychiatry, 15(1), 53–63. https://doi:10.1038/mp.2008.94
249
250
Elizabeth A. Forrester
Salkovskis, P. M., Forrester, E., Richards, H. C., & Morrison, N. (1998). The devil is in the
detail: Conceptualising and treating obsessional problems. In N. Tarrier, A. Wells, &
G. Haddock (Eds.), Treating complex cases: The cognitive behavioural therapy approach
(pp. 46–80). Wiley.
Salkovskis, P. M., Millar, J., Gregory, J., & Wahl, K. (2017). The termination of checking and
the role of just right feelings: A study of obsessional checkers compared with anxious and
non-clinical controls. Behavioural and Cognitive Psychotherapy, 45(2), 139–155.
doi:10.1017/S135246581600031X
Salkovskis, P. M., Wroe, A., Gledhill, A., Morrison, N., Forrester, E., Richards, H. C.,
Reynolds, M., & Thorpe, S. (2000). Responsibility attitudes and interpretations are
characteristic of obsessive-compulsive disorder. Behaviour Research & Therapy, 38(4),
347–372. https://doi.org/10.1016/S0005–7967(99)00071-6
Seli, P., Risko, E. F., Purdon, C., & Smilek, D. (2017). Intrusive thoughts: Linking spontaneous mind-wandering and OCD symptomatology. Psychological Research, 81, 392.
https://doi.org/10.1007/s00426–016-0756-3
Spitzer, R. L., Kroenke, K., Williams, J. B., & Löwe, B. (2006). A brief measure for assessing
generalized anxiety disorder: The GAD-7. Archives of Internal Medicine, 166, 1092–1097.
Strauss, C., Lea, L., Hayward, M., Forrester, E., Leeuwerik, T., Jones, A., & Rosten, C.
(2018). Mindfulness-based exposure and response prevention for obsessive compulsive
disorder: Findings from a pilot randomised controlled trial. Journal of Anxiety Disorders,
57, 39–47. https://doi.org/10.1016/j.janxdis.2018.04.007
Thorpe, S. J., Barnett, J., Friend, K., & Nottingham, K. (2011). The mediating roles of disgust
sensitivity and danger expectancy in relation to hand washing behaviour. Behavioural
and Cognitive Psychotherapy, 39(2), 175–190.
Toffolo, M. B. J, Van den Hout, M. A., Radomsky, A. S., & Engelhard, I. M. (2016). Check,
check, double check: Investigating memory deterioration within multiple sessions of
repeated checking. Journal of Behavior Therapy & Experimental Psychiatry, 53, 59–67.
https://doi.org/10.1016/j.jbtep.2015.09.001
Twohig, M. P., Abramowitz, J. S., Bluett, E. J., Fabricant, L. E., Jacoby, R. J., Morrison, K. L.,
Reuman, L., & Smith, B. M. (2015). Exposure therapy for OCD from an acceptance and
commitment therapy (ACT) framework. Journal of Obsessive-Compulsive and Related
Disorders, 6, 167–173. https://doi.org/10.1016/j.jocrd.2014.12.007.
van den Hout, M. A., Reininghaus, J. K., van der Stap, D., & Engelhard, I. M. (2012). Why
safety behaviour may not be that bad in the treatment of anxiety disorders: The
commitment to future exposures. Psicoterapia Cognitiva e Comportamentale, monograph
supplement, 111–126.
van Niekerk, J. (2018). A clinician’s guide to OCD. New Harbinger Publications.
Wahl, K., Huelle, J. O., Zurowski, B., & Kordon, A. (2013). Managing obsessive thoughts
during brief exposure: An experimental study comparing mindfulness-based strategies
and distraction in obsessive-compulsive disorder. Cognitive Therapy and Research, 37,
752. https://doi.org/10.1007/s10608–012-9503-2
Wahl, K., Schönfeld, S., Hissbach, J., Küsel, S., Zurowski, B., Moritz, S., Hohagen, F., &
Kordon, A. (2011). Differences and similarities between obsessive and ruminative
thoughts in obsessive-compulsive and depressed patients: A comparative study.
Journal of Behaviour Therapy & Experimental Psychiatry, 42(4), 454–461. https://doi
.org/10.1016/j.jbtep.2011.03.002
Waller, G. (2009). Evidence-based treatment and therapist drift. Behaviour Research and
Therapy, 47, 119–127.
Watkins, E. R., & Nolen-Hoeksema, S. (2014). A habit–goal framework of depressive
rumination. Journal of Abnormal Psychology, 123(1), 24–34. http://dx.doi.org/10.1037/
a0035540
CBT for OCD
Wells, A. (2009). Metacognitive therapy for anxiety and depression. Guilford.
Wells, A., & Cartwright-Hatton, S. (2004). A short form of the metacognitions questionnaire:
Properties of the MCQ-30. Behaviour Research and Therapy, 42(4), 385–396. https://doi
.org/10.1016/S0005-7967(03)00147-5
Wells, A., Gwilliam, P., & Cartwright-Hatton, S. (2001). The Thought Fusion Instrument.
Unpublished self-report scale, University of Manchester, UK.
Welz, A., Reinhard, I., Alpers, G. W. & Kuehner, C. (2018). Happy thoughts: Mind
wandering affects mood in daily life. Mindfulness, 9, 332. https://doi.org/10.1007/
s12671–017-0778-y
Wootton, B. M., Diefenbach, G. J., Bragdon, L. B., Steketee, G., Frost, R. O., & Tolin, D. F.
(2015). A contemporary psychometric evaluation of the Obsessive Compulsive
Inventory-Revised (OCI-R). Psychological Assessment, 27(3), 874–882. doi:10.1037/
pas0000075
251
13
Obsessive Compulsive Disorder: Treating
Obsessions and Associated Behavioral Responses
Gillian M. Alcolado
Background Theory and Research
Researchers and clinicians once considered “pure” obsessions difficult to treat because
they did not necessarily have associated overt compulsions (see Clark, 2000, for a
review). These obsessions are repugnant in nature and are highly distressing to the
sufferer as they run contrary to personal values (e.g., aggressive, blasphemous, sexual,
or amoral thoughts) (Rachman, 1997). Thankfully, experts now recognize that obsessions can be treated (for a review, see Shafran et al., 2013). This is particularly due to
the development of cognitive therapy (CT) for beliefs regarding obsessions (e.g.,
Rachman, 2003) and because of the recognition that there are behavioral responses
to these intrusive thoughts (e.g., avoidance behavior, neutralization attempts;
Rachman, 1997). The latter can be targeted effectively through cognitive or traditional
exposure and response prevention (ERP) techniques (e.g., Clark, 2004). This chapter
will cover both CT and ERP for treating obsessions and their behavioral sequelae and
will therefore take a cognitive behavioral therapy (CBT) approach.
Definitions
The terms “intrusive thoughts” and “obsessions” will be used throughout. These
words can be used interchangeably with clients once they have been socialized to
the treatment language and model. When clinicians explain how obsessions
develop, however (which will form an integral part of the psychoeducation; see
below), they must make the following important difference clear: most people
experience intrusive thoughts from time to time (as first demonstrated by
Rachman & de Silva, 1978). It is only once the thoughts become frequent, distressing, and impairing that they become clinically significant, and are therefore called
obsessions (American Psychiatric Association, 2013).
Universality of Intrusive Thoughts
Intrusive thoughts are very common in the general population. In a sample of
nonclinical individuals, the overwhelming majority of participants reported having
252
Obsessive-Compulsive Disorder
experienced an intrusive thought (80%; Rachman & de Silva, 1978). Researchers
replicated this finding in a worldwide representative sample, where 94% of nonclinical participants endorsed having had at least one unwanted intrusive thought in
the previous three months (Radomsky et al., 2014). Research findings from several
studies (e.g., Moulding et al., 2014; Rowa & Purdon, 2003) support the theory
(Rachman, 1997) that it is not the experience, but the interpretation of the intrusion
that causes it to become an obsession.
From Intrusion to Obsession
According to Rachman’s (1997, 1998) cognitive theory of obsessions, intrusive
thoughts lead to obsession via the “catastrophic misinterpretation” of what it
means to have these thoughts. This misinterpretation is the target for treatment,
rather than the obsessional content itself (see Clark, 2004; Rachman, 2003).
Individuals who make this type of misinterpretation are understandably highly
vigilant for the potential presence of such thoughts. They also often develop rituals
aimed at reducing and/or neutralizing the thought and associated distress. In
clients with obsessions, these thoughts are therefore occurring much more frequently, repeatedly, and are accompanied by a great deal of anxiety. By contrast, a
person who is not suffering from OCD makes no negative interpretation. These
intrusive thoughts are therefore more likely be fleeting, largely unnoticed, and
tend not to cause suffering (Rachman, 1997). As such, no ritual to neutralize the
thought is needed (Rachman, 1997). Research aligns with these tenets. In a recent
worldwide study of nonclinical individuals, the intrusive thoughts that were rated
as most highly distressing were the ones that occurred most frequently, were
interpreted most negatively, and were most strongly attempted to be controlled
(Moulding et al., 2014). Hence, the person who is vigilant for the recurrence of
these thoughts because a catastrophic misinterpretation has been made, and
engages in a behavior to obtain relief from any associated anxiety or distress, will
be more likely to turn to the same strategies the next time the thought recurs
(Rachman, 1997). These behaviors may include avoidance of objects/environments
that trigger the thoughts or attempts to stop or cancel the thoughts (“neutralizing”
their effect) through behavioral or mental tactics (Rachman, 1997). This type of
individual is therefore then more likely to develop a problem with obsessions over
time (Rachman, 1997).
Types of Misinterpretations
There are various ways in which individuals with OCD may catastrophically interpret their intrusive thoughts, and all are potential targets for treatment (see below).
Rachman described these beliefs as typically fitting into one of these categories,
mainly, the fears that the thought means one is “immoral, evil, dangerous, insane,
or . . . a combination of these qualities” (Rachman, 2003, p. 4). In the subsequent
theoretical and research literature, these types of beliefs have been elaborated on
and expanded. The belief that having a thought means something about one’s true
253
254
Gillian M. Alcolado
values or that the thought is more likely to “come true” is a type of thought–action
fusion (Rachman & Shafran, 1999). Another common (and erroneous) belief is that
thoughts are controllable and that inability to control thoughts is a sign one might
lose control or go crazy (Clark & Purdon, 1993). Indeed, the Obsessive Compulsive
Cognitions Working Group (OCCWG) has developed three factors of belief
domains thought to be central to OCD, one of which is “importance and control
over thoughts.” Another cognitive theory of obsessive thoughts is the inferencebased theory (O’Connor & Robillard, 1999). It postulates that individuals with
obsessions are bothered by them because they are making incorrect logic inferences
regarding the meaning of the presence of these thoughts (O’Connor & Robillard,
1999). An entire treatment has been developed that focuses solely on this type of
misinterpretation and has been shown to be effective (O’Connor et al., 2005).
Interested readers are directed to its treatment manual (O’Connor & Aardema,
2011).
Evidence-Based Interventions for Obsessions: ERP and CBT
Exposure and response prevention (ERP) was the first successful psychological
intervention for OCD (Meyer, 1966). The focus is behavioral: the client gradually
engages with situations that would normally prompt compulsive rituals, but practices refraining from doing so (see Foa et al., 2012, for the definitive ERP practitioner treatment manual). ERP has long been considered the gold standard of
psychological interventions for OCD (Chambless et al., 1998). Recent treatment
research examining outcome by symptom dimension, however, has shown that
primary obsessions are less responsive to ERP than are the more behaviorally
based symptom dimensions (Williams et al., 2014). In interventions with a heavy
ERP focus but that contain some mixed CBT techniques, researchers have also
found that in individuals with both obsessions and compulsions, their obsessive
symptoms responded less well to intervention (Chase et al., 2015; Farrell et al.,
2016). Encouragingly, however, combined approaches that incorporate cognitive
rationale and challenges to cognitive beliefs in conjunction with ERP techniques
have exhibited good outcomes for individuals with primary obsessions (Freeston
et al., 1997; O’Connor et al., 2005).
Evidence-Based Interventions for Obsessions: CT
The first successful case study of cognitive therapy (CT) for primary obsessions was
published in 1994 (Van Oppen, 1994). A woman with harming obsessions was
treated with cognitive interventions targeting her overestimation of negative consequences as well as the personal significance of what her thoughts meant. She made
clinically significant change, which was maintained at follow-up. A case series study
targeting the obsessive belief domains outlined by OCCWG (2005) found similarly
promising results (Freeston et al., 2001). In 2010, Whittal et al. conducted a randomized controlled trial (RCT), using Rachman’s (2003) cognitive therapy protocol
Obsessive-Compulsive Disorder
for obsessions. They found robust decreases in symptoms with large effect sizes that
were stable at 12-month follow-up. Cognitive therapy seems to have a superior
effect on obsessions regarding unwanted intrusive thoughts (i.e., the repugnant
obsessions that are the focus of this chapter) than for obsessions relating to traditional compulsions (e.g., contamination obsessions related to washing behavior;
Belloch et al., 2010), suggesting it is particularly appropriate for use in
this population.
Why Evidence-Based Treatments Work
All psychological interventions for OCD generally exhibit the same trajectory of
treatment change, whereby the behavioral symptoms change before cognitions
(Anholt et al., 2008). A rigorous outcome trial on exposure and response prevention
(ERP) for OCD suggested that cognitive change is not the mechanism of change (Su
et al., 2016). In CBT for OCD, by contrast, it appears that change in cognition is an
important factor, although the direction of that relationship might not be as straightforward as hypothesized (e.g., Andersson et al., 2015). The same has been found for
CBT specifically for obsessions (Woody et al., 2011). A review by Polman et al.
(2010) concluded that there is evidence that both cognitive change and behavioral
change can lead to improvement and that there is bidirectionality between them.
The authors recommended that as process and mechanism research in OCD outcomes is still in its infancy, it should be conducted separately by specific symptom
dimension in order to best understand the mechanisms of change (Polman et al.,
2010). Despite the lack of such research, ERP and cognitive therapy (CT) are both
effective (e.g., Chambless et al., 1998; Woody et al., 2011). Therefore, these
approaches continue to be used to target obsessions (e.g., Clark, 2004; Rachman,
2003), even though the mechanisms remain unknown.
Assessment of Obsessions and Associated Behaviors
The clinician will first determine the nature, frequency, and triggers for the obsessions. It is also important to ascertain the appraisals that the sufferer is making
about the intrusive thoughts and any underlying beliefs that are affecting these
interpretations. Additionally, any neutralizing, avoidance, and/or reassuranceseeking behaviors need to be identified, as they maintain the obsessions.
Together, this information helps guide the case formulation and provides points
of intervention.
Assessment of Symptoms
Structured Interviews
The Yale–Brown Obsessive Compulsive Scale (Y-BOCS; Goodman et al., 1989b) is
the gold standard structured interview for assessing OCD (Kim et al., 1989). It
exhibits good internal consistency and inter-rater reliability (Goodman et al., 1989b)
255
256
Gillian M. Alcolado
as well as validity (Goodman et al., 1989a). It contains a checklist with multiple
examples of aggressive, sexual, and religious obsessions that can be used as probes
for the clinician. This is particularly helpful as clients may not be as comfortable
spontaneously reporting repugnant obsessions as compared with more benign compulsions. A psychometrically sound self-report version of the Y-BOCS has also been
developed (Steketee et al., 1996). Alternatively, Rachman’s (2003) treatment
manual provides a semi-structured interview for assessing obsessions. Although
not validated, it is comprehensive and highly clinically relevant. If OCD symptoms
are to be assessed in the context of other disorders, the Anxiety and Related
Disorders Interview Schedule for DSM-5 (ADIS-5; Brown & Barlow, 2014), a
semi-structured interview, includes in its section on OCD a useful checklist of
possible obsessions and compulsions. It does not yet have published reliability and
validity statistics, but its predecessor, the ADIS for DSM-IV, on which it is modeled,
has strong psychometric properties (Brown et al., 2001).
Self-Report Measures
A number of questionnaires exist that assess OCD symptomatology, with subscales
pertaining to obsessions. These tools include the Vancouver Obsessional
Compulsive Inventory (VOCI; Thordarson et al., 2004), which has an obsessions
subscale (12 items), some of which are specific to repugnant obsessional domains
(e.g., harm, sex, religion). A shorter measure, the Obsessive-Compulsive
Inventory – Revised (OCI-R; Foa et al., 2002) contains three items pertaining to
obsessive thoughts, although without specifying type. The Dimensional Obsessive
Compulsive Scale (DOCS; Abramowitz et al., 2010, measure appended to the
article) contains two subscales that address obsessions, one relating to harm
(although this subscale also contains items relating to checking compulsions) and
the other to unacceptable thoughts. Another group has since amended the DOCS to
include a supplemental five-item scale that assesses sexually intrusive thoughts
(Wetterneck et al., 2015; subscale appended in the article) that would be highly
relevant for the clinician working with this type of obsession. For a recent and
comprehensive review of all OCD symptom interviews, measures, and their psychometric properties, see Rapp et al. (2016).
Unstructured Interviews
Obsessions can also be explored using open-ended questions as part of a nonstructured diagnostic intake interview. First, information on presence and type of intrusion can be collected. These would be intrusive thoughts of aggression, sex,
morality, and religion. This can be asked in a manner such as, “Have you ever
had unwanted thoughts of harming someone even though you don’t want to?” and
“Have you ever had unwanted intrusive thoughts that go against your religious
beliefs, sexual preferences, or moral values?” If these statements are endorsed, it is
important to always follow up by asking the client for examples, as it will be the
clinician who ultimately makes the determination whether a thought is an obsession
or not. For example, having the desire to “throttle” someone during an argument is
Obsessive-Compulsive Disorder
not typically an obsession, as this type of thought tends to be mood-congruent and
nonintrusive. A more likely example of an obsession would be an intrusive thought
or image of harming a loved one while holding a sharp implement. Similarly, feeling
guilty about having sexual fantasies with someone outside a partnership would not
typically fit the criteria for an unwanted intrusive thought. A more likely example of
an obsession would be an intrusive thought or image of engaging in a sexual act with
an inappropriate or undesirable figure (e.g., a stranger, family member). In either
case, however, determining the intrusiveness, interpretation, and distress associated
with the thought will clarify whether it is an obsession or some other type of
negative thought process.
It is important to include unstructured questions regarding neutralizing behaviors, as they are not necessarily well captured otherwise. The clinician can ask in an
open-ended manner: “How do you respond to those thoughts?” and/or “Is there
anything you do to try to make the thoughts go away?” In order to determine
whether the action they are describing is indeed a neutralizing strategy, it is helpful
to ascertain the goal of said behavior. For example, “What are you trying to
accomplish when you engage in that behavior?” If the client is not sure what is
meant, clarify by asking whether they are trying to neutralize/cancel the thought, get
rid of the thought, or stop the thought. It is also very clinically useful to directly ask
regarding the presence of behaviors associated with the obsessions, such as avoidance and reassurance seeking: “Do you ever avoid anything out of fear of having
these thoughts?” and “Do you ever seek reassurance from others about what it
means that you have these thoughts?”
Assessment of Beliefs
Structured Interview
Once symptoms are understood, the underlying beliefs must be probed in order to
complete the clinical formulation and understand the client. There are currently no
validated structured clinical interviews that assess these beliefs. However, for the
clinician wanting some practical guidelines for this type of questioning, Rachman’s
(2003) interview is useful.
Self-Report Measures
The Interpretation of Intrusions Inventory (III; OCCWG, 2005) focuses solely on
beliefs about intrusive thoughts. It contains 31 items that assess beliefs about control
of thoughts, importance of thoughts, and responsibility. These items load onto a
single factor, and the measure has strong psychometric properties (OCCWG, 2005).
The Obsessive Beliefs Questionnaire (OBQ-44) is a validated 44-item questionnaire for which the “importance and control over thoughts” subscale would be
relevant to this population (OCCWG, 2005; the questionnaire is appended within
the research article). This subscale includes items such as “Having nasty thoughts
means I am a terrible person” and “If I have aggressive thoughts or impulses about
my loved ones, this means I may secretly want to hurt them” (OCCWG, 2005).
257
258
Gillian M. Alcolado
The Responsibility Interpretations Questionnaire (RIQ; Salkovskis et al., 2000)
assesses beliefs associated with whatever is the most commonly occurring obsession
they experienced within the last two weeks. To make it relevant to the assessment of
“pure” obsessions, the clinician directs the client to fill out the questionnaire
thinking about those unacceptable blasphemous thoughts. Items are highly pertinent to obsessional treatment, as they include statements such as “I must regain
control of my thoughts” and “Because these thoughts come from my own mind
I must want them to happen.” This questionnaire also asks about the degree to
which an individual endorses a more adaptive interpretation, such as “There’s
nothing wrong with letting such thoughts come and go naturally” and “Thoughts
cannot make things happen.” This measure has good psychometric properties and
can be found in the appendix of the research article (Salkovskis et al., 2000).
Shafran et al. (1996) developed a measure of one domain of catastrophic
misinterpretation, thought–action fusion (TAF). The TAF scale exhibits sound
psychometric properties and is comprised of three subscales: (1) moral TAF (e.g.,
“Having a blasphemous thought is almost as sinful to me as having a blasphemous
action”), (2) likelihood TAF for self (e.g., “If I think of myself in a car accident, this
increases the risk that I will have a car accident”), and (3) likelihood of TAF for
others (e.g., “If I think of a relative/friend falling ill, this increases the risk that they
will fall ill”) (Shafran et al., 1996). The items are appended in the article, and the
authors recommend the TAF as a useful starting point for discussion rather than
one strictly to be used to monitor strict score changes (Shafran et al., 1996).
Finally, the Personal Significance Scale (PSS; Rachman, 2003) can be completed
by clients in order to assist them in understanding their beliefs about their intrusions. In order to tailor this measure to the assessment of relevant obsessions, ask
the clients to think of their blasphemous thoughts when answering the items. A copy
of this measure, which has yet to be validated, can be found in Rachman’s
(2003) manual.
Unstructured Interview
To ascertain the beliefs that underlie the distress associated with the obsessions, one
can ask, “What is most upsetting to you about the fact that you are experiencing this
thought?” Follow-up questions can be more direct as needed. These may include
queries such as “What do you think the thought means about you?” and “What do
you fear would happen if you could not stop the thought?”
Case Formulation
An individualized case formulation guides the intervention. The clinician develops
this using the information from the assessment and solidifies it through discussion
with the client. As with any CBT model, predisposing factors that make an individual vulnerable to having obsessions are considered. These include genetics, brain
chemistry, environment, learning, and/or life stressors. Core beliefs predispose an
Obsessive-Compulsive Disorder
individual toward making catastrophic misinterpretations of intrusions in
the moment.
Framework for Understanding Obsessions
The important distinguishing factor in the case formulation of the obsession is that
the obsession is considered not to be the automatic thought but instead a trigger for
such thoughts. Thus, the clinician does not target the obsession itself in treatment,
but rather the client’s interpretation of the obsession. The maintenance factors of
the model (i.e., thoughts, emotions, behaviors) are the responses to the presence of
the obsession or a stimulus that elicits the obsession (the trigger). The catastrophic
misinterpretation of the intrusive thought as dangerous and personally significant in
some way and/or a belief about what such thoughts mean (e.g., that they should be
controllable and/or should not occur) is the automatic thought. This interpretation
of the obsession leads to an increase in the emotion of anxiety, physiological
arousal, and a behavioral response aimed at neutralizing the thought and decreasing
the anxiety. If the behavior is successful in decreasing the anxiety, it becomes
reinforced and more likely to occur the next time the client has the obsessive
thought. Specific to a model of obsessions, included in the behavioral component
of the model are any physical or mental neutralizing rituals and/or reassurance
seeking. In the long term, the behavioral response will likely also include any
avoidance of triggers as a strategy to prevent the cycle from occurring.
Case Vignette. Elena is a 30-year-old married woman who works in administration. She had a Catholic upbringing and an anxious mother. After the birth of her
daughter, she began experiencing unwanted intrusive thoughts of harming her. She
presented reporting difficulty caring for her independently out of fear of “going
crazy” and acting on these harm thoughts. An intake assessment was conducted
using a semi-structured interview to assess symptom domains, as well as self-report
questionnaires and unstructured clinical interviewing for further clarification of
her obsessions.
Developing the Model: Vulnerability Factors
Clients often want to understand “where the anxiety came from.” It is helpful to
incorporate this information into the broader case formulation to satisfy this desire
and to demonstrate that, regardless of the origin of the anxiety (or whether one can
even ascertain such a root cause, which is not always possible), the maintenance
factors can still be targeted to successfully treat the problem (see below).
Elena may have a genetic predisposition to OCD as her mother also struggled
with anxiety. Elena confirmed the clinician’s guess that modeling of OCD behaviors
by her mother was observed from a young age. Further, Elena’s religious background influenced her belief about the immorality of “impure thoughts,” which
contributed to the development of some of her core beliefs. These beliefs were
assessed via the unstructured clinical interview and self-report measures. The TAF
was administered, as it was suspected Elena held some TAF beliefs. It revealed that
259
260
Gillian M. Alcolado
she felt that having a “bad” thought is as bad as committing an action, and also that
having a thought means it is more likely to occur “in real life.” Her scores on the
OBQ were elevated, in particular, on the importance and control of thoughts
subscale, demonstrating that Elena also felt that she should have control over her
thoughts. The life stressor that catalyzed the onset of the current episode of OCD
was identifiable in her case as the birth of her child.
Developing the Model: Maintenance Factors
Details of the OCD symptoms, including obsessions and related behaviors that
Elena experiences, are gathered through the tools described above, including the
self-report measures and the semi-structured and unstructured interviews. She
exhibited elevated scores on the OCI-R obsession items. Further open-ended
follow-up questions were asked to determine her understanding of the meaning of
the intrusive thoughts, what she did in response to these thoughts, and how effective
these strategies were. In Elena’s case, her obsessive thoughts include harming her
daughter by dropping her or stabbing her with sharp objects. Elena had no desire to
harm her child, but did not know how to reconcile this with her experience of having
these thoughts. She responded to the thought by putting distance between herself
and her child as well as between herself and any potentially dangerous objects (e.g.,
scissors, knives). She tried to avoid handling such objects in front of her daughter,
and preferred to have help with her when she could so that a second person could
observe their interactions and “keep her daughter safe.” She also engaged in several
rituals, including praying over and over again to protect her child as well as trying to
mentally replace the thought with a “loving and safe” thought. She recognized these
rituals as unnecessary, but she could not resist engaging in them, as completing the
rituals resulted in a temporary decrease in anxiety.
Socialization to the Model: Specific Techniques
Once the CBT model above is explained, the therapist provides an explanation of
OCD. The clinician can begin by asking “Who experiences intrusive thoughts?” The
client may not be sure, but through discussion with the clinician will come to
understand that all individuals experience intrusive thoughts. Some clients may
appreciate learning that there is a wealth of research to support this assertion.
Having the client review a handout listing typical intrusive thoughts endorsed by
the general population can also be very powerful (see example in Rachman, 2003,
extracted from Rachman & de Silva, 1978). The clinician can then ask the client to
guess what kind of people listed these thoughts. The client is typically surprised and
relieved to know that individuals without OCD experience these thoughts.
The clinician may then ask the client in Socratic fashion, “So, if everyone has
these thoughts but not everyone has OCD, what is the difference?” This can lead to
a productive discussion about the difficulty in OCD being the interpretation of the
obsessive thought, not the obsessive thought itself. Individuals without OCD also
Obsessive-Compulsive Disorder
have intrusive thoughts, but they interpret them as meaningless noise. Minimal
anxiety is provoked, and therefore no ritual is necessary. The goal of examining
the client’s thoughts and behavioral processes is to help them understand this type
of reaction to the intrusion and subsequent lack of necessary behavior that is seen in
the general population. A clear explanation of the CBT model of obsessions is a
crucial step in engaging the client in treatment.
The other important salient point to make at treatment outset is that the goal is
not to stop the thoughts. This can be initially disappointing for the client. The
therapist can also provide the more hopeful caveat that as clients become less
distressed by the thoughts over time (as a result of the treatment) they will
likely find that the intensity and frequency of the obsessional thoughts also
gradually declines.
Socialization to the Model: Addressing Resistance
If the client is reluctant to adopt this model, a couple of techniques can be used. The
clinician can start with Socratic questioning and discussion regarding how well
efforts to suppress and eliminate these thoughts have gone so far. Many clients
readily acknowledge that their existing strategies have not been effective. Second,
evidence from studies on the ubiquity of intrusive thoughts in the general population can be summarized, and, as suggested above, a handout listing common
intrusive thoughts that people without OCD endorse may also be helpful. Finally,
the clinician can conduct the “white bear” behavioral experiment (see Wilhelm &
Steketee, 2006, for more details) to further demonstrate that trying not to have
certain thoughts has a paradoxical effect, and that is what we are trying to avoid in
this treatment. The white bear experiment is typically done without explaining the
purpose ahead of time. The client is asked to spend 30–60 seconds trying to think of
the white bear as much as they can. The clinician times them and gets the client to
count how many times they think of the white bear. Then this experience is
discussed. The client often will find it was hard to stay concentrated on the white
bear. Then the opposite task is assigned for another 30–60 seconds. The client is
instructed that no matter what, they cannot think of a white bear, and again
instructed to count how many times they think of the white bear. A debrief follows
again. This exercise demonstrates to the client how trying not to have obsessive
thoughts is likely achieving exactly the opposite result. Thus, the goal of treatment
will be not to suppress the thoughts but to learn to respond to them differently.
Therapy
Overview
Treatment consists of three phases: (1) psychoeducation, (2) therapeutic techniques
(including cognitive restructuring, ERP, and behavioral experiments), and (3)
relapse prevention. Most of the sessions are concentrated on phase 2. The number
of sessions will vary based on the number of obsessive belief domains to work on.
261
262
Gillian M. Alcolado
A minimum of 12–16 sessions is typical, with 1–2 sessions for phase 1, 8–12 sessions
for phase 2, and 1–2 sessions for phase 3.
During phase 2, clients may prefer to begin with cognitive interventions and/or
behavioral experiments, as they may be less anxiety-producing than ERP.
Furthermore, alternative interpretations generated from these exercises can be used
as coping statements during exposures to increase engagement with them.
Alternatively, if clients prefer to start with ERP to gain control over some of their
behaviors and avoidance, treatment can begin here and cognitive elements can be
woven into the intervention if and when the client struggles or plateaus.
Phase 1: Psychoeducation
Definitions of CBT and OCD are provided. Socialization to the model (described
above) also occurs here. When discussing the role of behavioral responses to
obsessions, it is important to probe for ambivalence that clients might have.
Emphasize that this is understandable, given that in the short term the anxiety
and the relief they produce is so high. A good between-session exercise for these
introductory sessions may include a pro/con list of engaging in therapy. The client
can refer to this list in the future if ambivalence resurfaces.
Have the client monitor their symptoms for one week. These would include
obsessions, interpretations, and associated rituals. This provides a more exact
picture of what the pertinent obsessions are and which behaviors to work on. It
often also reveals some symptoms that have been overlooked. Rachman (2003) has
a sample daily record form for the frequency and distress associated with an
obsession. Foa et al. (2012) have a form for monitoring rituals. Either may be used
or a hybrid that captures both elements (see Wilhelm & Steketee, 2006), depending
on the focus for that particular week.
Measuring Outcome
A weekly monitoring form (see above) tracks symptom change in the most personalized fashion. Given that the goal is not for the obsessions to decrease, it is
important to include the intensity and distress associated with the occurrence of
the intrusion. Avoidance and mental ritualizing should be explicitly tracked. The
obsessions subscale from the OCI-R (Foa et al., 2002) is a general and brief
alternative, although the information gleaned from this will not be as nuanced.
The Personal Significance Scale (Rachman, 2003) tracks a range of the beliefs
being targeted.
Phase 2: Cognitive Interventions and Behavioral Experiments
Choose a relevant automatic thought to begin with based on the formulation and/or
monitoring form. If desired, a thought record to modify beliefs about thoughts (not
the obsessive content itself ) can be used (see Wilhelm & Steketee, 2006). The
behavioral experiment approach was originated by Beck et al., (1979). For more
Obsessive-Compulsive Disorder
examples of behavioral experiments in the context of OCD treatment, refer to Clark
(2004) or Rachman (2003). Common interventions relevant to obsessions are
discussed below.
To target the likelihood of TAF (i.e., that the occurrence of a thought means it
is more likely to happen in reality), a behavioral experiment can be designed to test
this belief. The client can begin by purposefully thinking about a good thing
happening to the therapist (e.g., winning the lottery) and check in to see if it
happens. They can then progress to more negative thoughts (e.g., thinking of the
therapist tripping while walking, getting parking tickets). The therapist may model
the safety of this behavior by also engaging in the task. A behavioral experiment can
be conducted in-session where both write out “I wish (loved one) would get in a car
accident.” The take-home message is that although it might feel unpleasant to have
such thoughts, they do not actually influence reality. If the person is still struggling
with such thoughts, then one can move on to ERP where the target is to not respond
to thoughts that trigger likelihood TAF beliefs (see below).
Morality TAF (i.e., that having a bad thought is as bad as doing a bad thing) can
be targeted through a cognitive exercise. The goal is to shift how evil the client
concludes they are for having such thoughts. The client is asked to consider who is
the worst person and the best person they can think of (typical anchors are Hitler
and Mother Teresa). The client is then encouraged to add other people along the
continuum, for example, murderers, rapists, but also more ambiguous examples
such as individuals who cheat, lie, evade taxes, don’t pay library fines, and so on. As
the last step, the client is asked to consider where they might place someone who has
repugnant thoughts that go against their values. This helps the client begin to see
how far they are from being a bad person. Relevant ERP exercises to cues that elicit
morality TAF could then be designed.
Discussion of control over thoughts will likely have been covered through
socialization to the model (see above), but if not, the white bear behavioral experiment would be appropriate here. Additionally, clients can be asked to manipulate
how much they attempt to control their own intrusions using alternate days of
attempting control versus days of not attempting control and tracking how frequent
and intense the obsessions are on these days. They will likely find the obsessions are
actually much better on days in which they do not attempt any such control.
To target beliefs about the importance of thoughts, the discussion from the
socialization to the model can be reviewed here as well. A behavioral experiment
can be conducted similar to the one for control of thoughts. The client can alternate
days on which they attend very closely to the thought, and give it importance, versus
attempting to let it go and rate how important the thought actually feels. The client
will again likely find that paying less attention to the thoughts will make them feel
less important. If the client believes the thought must be true because of their
behavioral reaction to it, the following demonstration and discussion can be useful.
The clinician shares a story of how they made homemade pasta sauce on the
weekend. The clinician and client can then discuss the thoughts (e.g., “I’m hungry”)
and physical reactions (e.g., “salivation”) this story caused. The clinician then
263
264
Gillian M. Alcolado
reveals the story is false. The point to be discussed is that thoughts (even when not
true) can elicit reactions and that this should not be taken as a marker of
their importance.
Phase 2: Behavioral Interventions (Sessions 3–10)
ERP can also be very helpful in addressing obsessions. See Foa et al. (2012) for the
definitive guide to this type of work and Grayson (2014) for sample hierarchies. The
client generates a list of cues and situations that typically elicit obsessions. They
engage with these in planned, controlled circumstances and are prompted to apply
response prevention. Response prevention may at first be a brief delay in neutralizing behavior, avoidance, or reassurance seeking. They then may continue to reduce
the number of behavioral strategies employed, and continue to refrain from any
overt or covert behavioral or mental rituals that would typically neutralize the
thought. Clients are encouraged to sit with the intrusion and the subsequent anxiety
for as long as planned. This can be accomplished by setting a goal of staying in the
exposure either until the anxiety reduces by a certain amount (e.g., subjective
anxiety decreasing from 95/100 to 65/100), or for a predetermined length of time
(e.g., 20 minutes).
When targeting reassurance seeking it might be helpful to have loved ones
attend a session (with the client’s permission). They can then be provided with
psychoeducation and the best strategies for supporting the client. The clinician will
remind the loved one that the client is in the control of how and when they are
reducing the behavior.
If there are certain catastrophic feared outcomes of the intrusive thought that
are not alleviated through the aforementioned techniques, an imaginal exposure
exercise may be appropriate. For example, fear of having an intrusion, losing
control, and harming loved ones could be targeted in this way. The goal of the
imaginal exposure is for the client to have a reduced anxious reaction to that
scenario through explicitly confronting it. What often happens is that when clients
write out the scenario, they begin to see how unlikely it would be to occur.
Phase 3: Relapse Prevention (Sessions 11 and 12)
Typical CBT relapse principles apply with this treatment approach to obsessions.
Strategies such as coping statements, maintenance ERP exercises, and education on
lapses versus relapses are all important. The clinician should ensure that the client
recalls that it is not the intrusive thoughts but their interpretation that leads to
distress. This same principle (and associated cognitive and behavioral strategies the
client has learned) would apply to any new intrusions not targeted in the therapy
that become upsetting in the future. ERP exercises would include the continued
vigilance for and elimination of mental rituals in response to intrusive thoughts.
A lapse would not be the occurrence of an obsession, but a reaction of distress and
impairment to its presence.
Obsessive-Compulsive Disorder
Summary of Key Learning Points
Intrusive thoughts are universal; it is misinterpretation of intrusive thoughts that
converts them to obsessions.
The goal of therapy is not to get rid of the obsessive thoughts but to interpret
them more adaptively.
Some rituals may be covert (i.e., mental) and must be assessed prior
to treatment.
Obsessions and related behaviors can be effectively targeted in CBT using
cognitive techniques, behavioral experiments, and exposure and response
prevention.
FURTHER RECOMMENDED READING
Clark, D. A. (2003). Cognitive-behavioural therapy for OCD. Guilford.
Foa, E. B., Yadin, E., & Lichner, T. K. (2012). Exposure and response (ritual) prevention for
obsessive-compulsive disorder: Therapist guide (Treatments that work) (2nd ed.). Oxford
University Press.
Grayson, J. (2014). Freedom from obsessive-compulsive disorder. Berkley.
Rachman, S. (2003). The treatment of obsessions. Oxford University Press.
Wilhelm, S., & Steketee, G. S. (2006). Cognitive therapy for obsessive-compulsive disorder:
A guide for professionals. New Harbinger.
REFERENCES
Abramowitz, J. S., Deacon, B. J., Olatunji, B. O., Wheaton, M. G., Berman, N. C., Losardo,
D., . . . & Hale, L. R. (2010). Assessment of obsessive-compulsive symptom dimensions:
Development and evaluation of the Dimensional Obsessive-Compulsive Scale.
Psychological Assessment, 22, 18019–8.
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.) (DSM-5). American Psychiatric Association.
Andersson, E., Ljotsson, B., Hedman, E., Hesser, H., Enander, J., Kalod, V., . . . & Ruck, C.
(2015). Testing the mediating effects of obsessive beliefs in internet-based cognitive
behaviour therapy for obsessive-compulsive disorder: Results from a randomized controlled trial. Clinical Psychology and Psychotherapy, 22, 722–732.
Anholt, G. E., Kempe, P., de Haan, E., van Oppen, P., Cath, D. C., Smit, J. H., & van Balkom,
A. J. L. M. (2008). Cognitive versus behavior therapy: Processes of change in the
treatment of obsessive-compulsive disorder. Psychotherapy and Psychosomatics, 77,
38–42.
Beck, A. T., Rush, A. J., Shaw, B. F., & Emery, G. (1979). Cognitive therapy of depression.
Guilford Press.
Belloch, A., Cabedo, E., Carrio, C., & Larsson, C. (2010). Cognitive therapy for autogenous
and reactive obsessions: Clinical and cognitive outcomes at post-treatment and 1-year
follow-up. Journal of Anxiety Disorders, 24, 573–580.
Brown, T. A., & Barlow, D. H. (2014). Anxiety and Related Disorders Interview Schedule for
DSM-5 (ADIS-5) – Lifetime Version. Oxford University Press.
Brown, T. A., Di Nardo, P. A., Lehman, C. L., & Campbell, L. A. (2001). Reliability of DSMIV anxiety and mood disorders: Implications for the classification of emotional disorders.
Journal of Abnormal Psychology, 110, 49–58.
265
266
Gillian M. Alcolado
Chambless, D. L., Baker, M. J., Baucom, D. H., Beutler, L. E., Calhoun, K. S., CristsChristoph, P., . . . & Woody, S. R. (1998). Update on empirically validated therapies.
II. The Clinical Psychologist, 51, 3–16.
Chase, T., Wetterneck, C. T., Bartsch, R. A., Leonard, R. C., & Riemann, B. C. (2015).
Investigating treatment outcomes across OCD symptom dimensions in a clinical sample
of OCD patients. Cognitive Behaviour Therapy, 44, 365–376.
Clark, D. A. (2000). Cognitive behavior therapy for obsessions and compulsions: New
applications and emerging trends. Journal of Contemporary Psychotherapy, 30, 129–147.
(2004). Cognitive-behavioural therapy for OCD. Guilford.
Clark, D. A., Abramowitz, J., Alcolado, G. M., Alonso, P., Belloch, A., Bouvard, M., . . . &
Wong, W. (2014). Part 3. A question of perspective: The association between intrusive
thoughts and obsessionality in 11 countries. Journal of Obsessive-Compulsive and
Related Disorders, 3, 292–299.
Clark, D. A., & Purdon, C. (1993). New perspectives for a cognitive theory of obsessions.
Australian Psychologist, 28, 161–167.
Farrell, N. R., Ouimet, A. J., Rowa, K., Soreni, N., Swinson, R. P., & McCabe, R. E. (2016).
Who gets better when? An investigation of change patterns in group cognitive behavioral therapy for obsessive-compulsive disorder. Journal of Obsessive Compulsive and
Related Disorders, 10, 35–41.
Foa, E. B., Huppert, J. D., Leiberg, S., Lagner, R., Kichic, R., Hajcak, G., & Salkovskis, P. M.
(2002). The obsessive-compulsive inventory: Development and validation of a short
version. Psychological Assessment, 14, 485–495.
Foa, E. B., Yadin, E., & Lichner, T. K. (2012). Exposure and response (ritual) prevention for
obsessive-compulsive disorder: Therapist guide (Treatments that work) (2nd ed.). Oxford
University Press.
Freeston, M. H., Ladouceur, R., Gagnon, F., Thibodeau, N., Rhéaume, J., Letarte, H., &
Bujold, A. (1997). Cognitive-behavioral treatment of obsessive thoughts: A controlled
study. Journal of Consulting and Clinical Psychology, 1997, 405–413.
Freeston, M. H., Léger, E., & Ladouceur, R. (2001). Cognitive therapy of obsessive thoughts.
Cognitive and Behavioral Practice, 8, 61–78.
Goodman, W. K., Price, L. H., Rasmussen, S. A., Mazure, C., Delgado, P., Heniger, R., et al.
(1989a). The Yale–Brown Obsessive Compulsive Scale. II. Validity. Archives of General
Psychiatry, 46, 1012–1016.
Goodman, W. K., Price, L. H., Rasmussen, S. A., Mazure, C., Fleishman, R. L., Hill, C. L.,
et al. (1989b). The Yale–Brown Obsessive Compulsive Scale. I. Development, use, and
reliability. Archives of General Psychiatry, 46, 1006–1011.
Grayson, J. (2014). Freedom from obsessive-compulsive disorder. Berkley.
Kim, S. W., Dysken, M. W., & Katz, R. (1989). Rating scales for obsessive compulsive
disorder. Psychiatric Annals, 9, 74–79.
Meyer, V. (1966). Modification of expectations in cases with obsessional rituals. Behaviour
Research and Therapy, 4, 273–280.
Moulding, R. M., Coles, M. E., Abramowitz, J., Alcolado, G. M., Alonso, P., Belloch, A., . . .
& Wong, W. (2014). Part 2. Do we all dislike intrusive thoughts for the same reasons?
Links between appraisals, control strategies and intrusions across countries. Journal of
Obsessive-Compulsive and Related Disorders, 3, 280–291.
Obsessive Compulsive Cognitions Working Group [OCCWG]. (2005). Psychometric validation
of the obsessive belief questionnaire and interpretations of intrusions inventory – Part 2:
Factor analyses and testing of a brief version. Behaviour Research and Therapy, 43,
1527–1542.
O’Connor, K., & Aardema, F. (2011). Clinician’s handbook for obsessive compulsive disorder:
Inference based therapy. Wiley-Blackwell.
Obsessive-Compulsive Disorder
O’Connor, K. P., Aardema, F., Bouthillier, D., Fournier, S., Guay, S., Robillard, S., Pelissier,
M. C., . . . & Pitre, D. (2005). Evaluation of an inference-based approach to treating
obsessive-compulsive disorder. Cognitive Behaviour Therapy, 34, 148–163.
O’Connor, K. P., Freeston, M. H., Gareau, D., Careau, Y., Dufour, M. J., Aardema, F., &
Todorov, C. (2005). Group versus individual treatment in obsessions without compulsions. Clinical Psychology and Psychotherapy, 12, 87–96.
O’Connor, K. P., Koszegi, N., Aardema, F., van Niekerk, J., & Taillon, A. (2009). An
inference-based approach to treatment obsessive-compulsive disorders. Cognitive and
Behavioral Practice, 16, 420–429.
O’Connor, K. P. & Robillard, S. (1999). A cognitive approach to the treatment of primary
inferences in obsessive-compulsive disorder. Journal of Cognitive Psychotherapy: An
International Quarterly, 13, 359–375.
Polman, A., Bouman, T. K., van Hout, W. J. P. J., de Jong, P. J., & den Boer, J. A. (2010).
Progress of change in cognitive-behavioural treatment of obsessive-compulsive disorder:
Current status and some future directions. Clinical Psychology and Psychotherapy, 17,
1–12.
Rachman, S. (1997). A cognitive theory of obsessions. Behaviour Research and Therapy, 35,
793–802.
(1998). A cognitive theory of obsessions: Elaborations. Behaviour Research and Therapy,
36, 385–401.
(2003). The treatment of obsessions. Oxford University Press.
Rachman, S., & de Silva, P. (1978). Abnormal and normal obsessions. Behaviour Research
and Therapy, 16, 233–238.
Rachman, S., & Shafran, R. (1999). Cognitive distortions: Thought–action fusion. Clinical
Psychology and Psychotherapy, 6, 80–85.
Radomsky, A. S., Alcolado, G. M., Abramowitz, J., Belloch, A., Bouvard, M., Clark, D. A.,
et al. (2014). Part 1. You can run but you can’t hide: Intrusive thoughts across 13 cities on
6 continents. Journal of Obsessive-Compulsive and Related Disorders, 3, 269–279.
Rapp, A. M., Bergman, L., Piacentini, J., & McGuire, J. F. (2016). Evidence-based
assessment of obsessive-compulsive disorder. Journal of Central Nervous System
Disease, 8, 13–29.
Rowa, K., & Purdon, C. (2003). Why are certain intrusive thoughts more upsetting than
others? Behavioural and Cognitive Psychotherapy, 31, 1–11.
Salkovskis, P. M., Wroe, A. L., Gledhill, A., Morrison, N., Forrester, E., Richards, C., . . . &
Thorpe, S. (2000). Responsibility attitudes and interpretations are characteristic of
obsessive compulsive disorder. Behaviour Research and Therapy, 38, 347–372.
Shafran, R., Radomsky, A. S., Coughtrey, A. E., & Rachman, S. (2013). Advances in the
cognitive behavioural treatment of obsessive-compulsive disorder. Cognitive Behaviour
Therapy, 42, 265–274.
Shafran, R., Thordarson, D. S., & Rachman, S. (1996). Thought action fusion in obsessive
compulsive disorder. Journal of Anxiety Disorders, 10, 379–391.
Steketee, G., Frost, R., & Bogart, K. (1996). The Yale–Brown Obsessive Compulsive Scale:
Interview versus self-report. Behavior Research and Therapy, 34, 675–684.
Su, Y.-J., Carpenter, J. K., Zandberg, L. J., Simpson, H. B., & Foa, E. B. (2016). Cognitive
mediation of symptom change in exposure and response prevention for obsessivecompulsive disorder. Behavior Therapy,47, 474–486.
Thordarson, D. S., Radomsky, A. S., Rachman, S., Shafran, R., Sawchuk, C. N., & Hakstian,
A. R. (2004). The Vancouver Obsessional Compulsive Inventory (VOCI). Behaviour
Research and Therapy, 42, 1289–1314.
Van Oppen, P. (1994). Cognitive therapy for obsessive-compulsive disorder. Clinical Case
Studies, 3, 333–349.
267
268
Gillian M. Alcolado
Wetterneck, C. T., Siev, J., Adams, T. G., Slimcowitz, J., & Smith, A. H. (2015). Assessing
sexually intrusive thoughts: Parsing unacceptable thoughts on the Dimensional
Obsessive-Compulsive Scale. Behaviour Therapy, 46, 544–556.
Whittal, M. L., Woody, S. R., McLean, P. D., Rachman, S. J., & Robichaud, M. (2010).
Treatment of obsessions: A randomized controlled trial. Behaviour Research and
Therapy, 48, 295–303.
Whittal, M. L., Thordardson, D. S., & McLean, P. D. (2005). Treatment of obsessivecompulsive disorder: Cognitive behavior therapy vs. exposure and response prevention.
Behaviour Research and Therapy, 43, 1559–1576.
Wilhelm, S., Berman, N. C., Keshaviah, A., Schwartz, R. A., & Steketee, G. (2015).
Mechanisms of change in cognitive therapy for obsessive compulsive disorder: Role of
maladaptive beliefs and schemas. Behavior Research and Therapy, 65, 5–10.
Wilhelm, S., & Steketee, G. S. (2006). Cognitive therapy for obsessive-compulsive disorder:
A guide for professionals. New Harbinger.
Williams, M. T., Farris, S. G., Turkheimer, E. N., Franklin, M. E., Simpson, H. B., Liebowitz,
M., & Foa, E. B. (2014). The impact of symptom dimensions on outcome for exposure
and ritual prevention therapy in obsessive-compulsive disorder. Journal of Anxiety
Disorders, 28, 553–558.
Woody, S. R., Whittal, M. L., & McLean, P. D. (2011). Mechanisms of symptom reduction in
treatment for obsessions. Journal of Consulting and Clinical Psychology, 79, 653–664.
14
Other Specified and Unspecified ObsessiveCompulsive and Related Disorders
Jan van Niekerk and Kieron O’Connor*
Introduction
The diagnosis of “other specified” obsessive-compulsive and related disorder
(OCRD) is made when the clinician describes the features of the presenting condition, which has similarities to an OCRD, but does not meet the full diagnostic
criteria. “Other unspecified OCRD” is diagnosed when the clinician does not
describe the presenting symptoms. DSM-5 (American Psychiatric Association
[APA], 2013) provides seven examples of conditions in this category:
Body dysmorphic-like disorder with actual flaws
Body dysmorphic-like disorder without repetitive behaviors
Body-focused repetitive behavior disorder (including behaviors such as repetitive nail biting or lip chewing, but excluding trichotillomania, excoriation disorder, stereotypic movement disorder and nonsuicidal self-injury)
Obsessional jealousy
Shubo-kyofu (from an indigenous Japanese diagnostic system: excessive fear of
having a bodily deformity)
Jikoshu-kyofu (related to shubo-kyofo: fear of having an offensive body odor;
the alternative term, olfactory reference syndrome, will be used hereafter)
Koro (a diagnostic label with origins in South East Asia: an episode of sudden
and intense anxiety that the penis, or the vulva and nipples in females, will
recede into the body, which may lead to death). The Malay word koro means
“to shrink”; alternatively, kura means “tortoise” – the “head of the tortoise” is
often used as an expression for the penis by the Malays and Chinese (Cheung,
1998).
* Sadly, Kieron O’Connor passed away on August 27, 2019, just after the final submission of this
manuscript. We would like to acknowledge his huge contribution to the field of obsessive-compulsive
and related disorders. He will be greatly missed by his colleagues and collaborators.
269
270
Jan van Niekerk and Kieron O’Connor
However, the above-listed conditions are not meant to be exhaustive. We will
discuss CBT interventions in the sections below for the following presentations
more likely to be encountered by the clinician:
Obsessional jealousy
Body-focused repetitive behavior disorders – not otherwise specified
Body dysmorphic disorder with a visual defect
Olfactory reference syndrome.
Where the presentation falls outside this range, the clinician will have to
carefully consider the available OCRD psychological models and how these may
need to be modified to address the underlying mechanisms of the presenting
problem. Further, transdiagnostic protocols (e.g., Barlow et al., 2011) could provide
a helpful background framework for guiding formulation and intervention.
Obsessional Romantic Jealousy
Jealousy is a normal, complex social emotion experienced from infancy into old age.
It has been defined as “the negative emotional state generated in response to a
threatened or actual loss of a valued relationship due to the presence of a real or
imagined rival” (DeSteno et al., 2006, p. 627). It serves an adaptive purpose, channeling cognition and behavior toward attainment of social goals, with a benefit to health.
When predicated on realistic data (e.g., one’s partner flirting with someone
else), jealousy is a reasonable response. However, it evolves into a clinical disorder
when the concern strays from having a bearing in objective reality, such as when
there is a “preoccupation with a partner’s sexual unfaithfulness based on unfounded
evidence” (Kingham & Gordon, 2004, p. 207) and where the concern is unresolved
by rational discussion. We will call this pathological jealousy, but other terms have
been used (e.g., morbid jealousy).
Pathological jealousy can manifest in varying degrees of conviction that the
jealous concerns are justified: as a delusion (high, stable conviction); within the
nondelusional category, as overvalued ideation (moderate conviction); or as an
obsession (lower conviction).
The current discussion will mainly be restricted to nondelusional, obsessional
romantic jealousy (OJ), in line with the chapter remit. Ecker (2012) lists the
following criteria for distinguishing non-delusional OJ from ordinary jealousy: (1)
more time-consuming and distressing, (2) greater functional impairment, (3) greater
negative impact on the partner, and (4) irrationality of the fears (e.g., they persist
despite contradictory evidence).
Prevalence and Impact
Pathological jealousy is considered a relatively common clinical problem; however,
epidemiological data are lacking (Ecker, 2012). From Shakespeare (Othello) to Jimi
Hendrix (“Hey Joe”), culture is replete with examples of its toxic impact – it impairs
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
psychological health and can culminate in suicide, physical aggression, and even
homicide, directed against the partner or the rival.
Clinical Model
Cognitive-behavioral formulation requires assessment of the following:
1. Learning history. Past experience and the meanings derived from them (i.e.,
beliefs about the self or others, and about ways of coping) constitute the
template for the information-processing biases that fire up OJ. Examples of
relevant experience/beliefs include insecure childhood attachment, prior traumatic loss (belief: “The people I love can leave me at any moment”), previous
exposure to infidelity (“People can’t be trusted”), low self-esteem, a perception
of sexual inferiority, a catastrophic expectation of relationship breakdown, and
rules for staying safe (“I won’t be caught napping”).
2. Immediate precipitants (or triggers). A wide variety of stimuli can trigger OJ,
including internal (e.g., imagined events, memories, thoughts about diminished
physical attractiveness) or external (self-comparison with rivals, a partner being
friendly with a rival).
3. Cognition. Jealous interpretations ensue: that the partner favors someone else
(a present rival or a previous partner), is engaged in infidelity, or is considering
cheating. The form of thinking may entail repetitive automatic intrusive
thoughts (similar to OCD obsessions), which may be resisted, and/or lengthy
ruminations (“Why can’t I be ever be good enough?”) or worries. The anticipated catastrophe is interpersonal – a defining feature.
4. Emotion. This can be complex: anxiety, fear (of rejection and loss), or anger (at
the partner or rival for moral transgression or betrayal, or about causing
jealousy), and shame, embarrassment, and guilt (when reflecting on the experience and consequences of OJ).
5. Behavior. Varied verification or repetitive safety behaviors (again, similar to
OCD compulsions) may be deployed to “find out what is really going on” or to
disconfirm the jealous worries, entailing a narrowed focus on, and response to,
the particular concern. Examples include aggressively interrogating the partner
about their movements or about past sexual experiences, examining bed linen
for sexual activity, checking the partner’s phone, or repetitive reassuranceseeking from the partner. Avoidant strategies may be used, for example,
requiring the partner to break off contact with opposite-gender friends. These
behaviors are negatively reinforced by a temporary reduction in jealousyrelated distress; however, given the typical omnipresence of jealousy-relevant
cues, coping or problem-solving strategies have to be ritualistically repeatedly
enacted (similar to OCD rituals).
6. Secondary feedback loops. Jealous interpretation may be fed by the consequent
negative affect, via an emotional reasoning bias, or as a result of conflict with the
partner (e.g., after the partner refuses to provide reassurance: “You’re hiding
something!”). Or jealous attention may make the partner feel special or powerful,
271
272
Jan van Niekerk and Kieron O’Connor
and thus, be encouraged. The OJ sufferer may appraise their symptoms as being
out of control, increasing their distress and inviting the use of maladaptive coping
strategies (e.g., thought suppression, which paradoxically may achieve the opposite, or substance abuse, spiraling into aggression); however, simultaneously, jealousy may be considered helpful for staying safe and preventing surprises. Worry
and rumination, ostensibly toward valued ends (such as distress reduction), result
in protracted jealous torment, achieving the opposite.
Assessment
The diagnosis will need to be carefully considered (e.g., delusional vs. nondelusional
OJ will require a different strategy), as well as assessment and management of risk
to self and others. The bulk of the data enabling formulation will derive from the
clinical interview; however, helpful supplementary information can be gained from
psychometric assessment scales (listed in Ecker, 2012) and situational diary keeping
of symptom triggers, emotional distress, and mediating factors (interpretations and
behaviors). Interviewing the partner can be particularly helpful, setting the stage for
their possible involvement in treatment.
Formulation and Therapy Socialization
The building blocks for cognitive-behavioral formulation are described above. The aim
is to present the client with an alternative account of their problems residing in their
thinking patterns and behavioral response, rather than in their partner. Ecker (2012)
stresses the helpful contribution of a historical formulation. This allows the alternative
account to be emboldened by having a plausible developmental underpinning and
dovetails with a goal of normalizing jealous feelings, but seeking to modify the response
to them. An example of the formulation of a jealous client is provided in Figure 14.1.
The following elements may be helpful for socializing the client to the model:
Jealousy is a normal emotion – it helps humans to bond and maintain their alliances;
however, it becomes a problem when it leads you incessantly to doubt your relationship,
and causes you distress. This can happen when you have developed powerful, unhelpful
misgivings and apprehensions about yourself and others on the basis of your past experience (list the relevant experiences). They lead you to look out for what you fear. You
then connect the dots and worry that something could be horribly wrong. You try to
investigate your suspicions and allay your fears, but your strategies backfire (list any
identified cons of the coping strategies). Suspicions linger and fester and the cycle repeats
itself. Your relationship suffers – you squeeze your partner to keep them close, but you
end up squeezing all the life out of the relationship, pushing them away.
Treatment
Therapist Stance and Psychoeducation
Because people who suffer from OJ frequently feel ashamed of or defensive about
their problems, the standard guidelines for establishing rapport in clinical
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Learning history
Mother has affair in early childhood; parents divorced;
father loses interest in parenting.
Discovered teenage steady cheated after
relationship ended.
Core beliefs and assumptions
You can’t trust your partner.
I’m not physically attractive.
I’m at risk if my partner finds anyone else
attractive.
Losing someone you love is a terrible thing.
Hypervigilant for infidelityrelevant cues
Situational trigger
Partner unexpectedly back late
from work.
Emotion
Jealous
Anxious
Angry
Guilty
Cognitive
They have been distracted lately/they didn’t
want to have sex last weekend - they’re
seeing someone.
I will lose them.
They have been lying.
I haven’t been trying hard enough.
Outcome
Disbelieve them.
Argument with partner ® ruminate.
I have to be certain ® worry.
Anxious, angry, depressed, ashamed
Hypervigilant
Retreat/distancing
Figure 14.1. Cognitive-behavioral formulation of obsessional jealousy.
Behavior
Interrogate them.
Confront them with
the evidence.
Blame and
threaten them.
273
274
Jan van Niekerk and Kieron O’Connor
interviewing apply ten times over. The therapist reassures the client that jealousy is
a normal, commonly experienced feeling. Whether it is damaging depends on how it
is managed and responded to.
Increasing Motivation
Carefully explore the client’s likely ambivalence about change: list the perceived
pros of jealousy (e.g., preventing the partner from straying, preventing surprises,
getting the partner back in line) and the cons (protracted personal/partner distress,
relationship conflict and stagnation). Then consider the pros and cons of change in
light of the likely future relationship trajectory if the status quo prevails (“Will your
relationship continue to grow?”). Seek to question and reframe any progresssabotaging pros of jealousy or cons of change (e.g., “Even if your partner has
broken off contact with his friends of the opposite gender, does that leave him
happier with you?”). Consider whether benefits can be pursued using a healthier
strategy (“Could there be ways of keeping your partner close without restricting
them?”). Use of strategies derived from motivational interviewing may also be
helpful; for example, address ambivalence using autonomy-affirming open questions
and reflective listening, rather than persuasion (see Simpson & Zuckoff, 2011).
Cognitive Strategies
A common area of focus is the OJ sufferer’s interpretation that there is a risk of
infidelity which has to be acted on. Standard CBT strategies may be used for
restructuring of situation-specific automatic thoughts and underlying beliefs:
thought elicitation, downward arrow (e.g., “What would it mean if they were seeing
someone and you did nothing?”), and evaluation of validity (evidence for and
against) and utility (costs and benefits). The aim is to introduce dissonance with
OJ interpretations and encourage consideration of alternatives, while not arguing
that the (sometimes unfalsifiable) jealous thought could not be true. Educate the
client about the pitfalls of thought distortions (e.g., mind-reading: “They have a
connection with each other”; soothsaying: “They will make a pass at them”) and
work on articulating simpler, more helpful perspectives from within the parameters
set by the existing context-relevant data. Cognitive restructuring work can also find
a useful application in reconsideration of catastrophic beliefs regarding relationship
breakdown (e.g. “I won’t be able to survive without them”).
Insights from Inference-based Therapy for OCD (IBT; O’Connor et al., 2009)
may have an application; here the jealous cognition is conceptualized as a doubt
(“doubting what you know”) which derives from reasoning with its origin in the
faculty of the imagination (which deals with “what could be” versus perception
which deals with “what is”). The aim is to guide the client back to the simpler,
commonsensical reasoning approach that they use in other domains of functioning.
It can be helpful to examine the selectivity of their reasoning to OJ-relevant themes:
“You’re not doubting that your partner is stealing your money, or earning extra
income by working as a sex worker – how do you approach things
differently there?”
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
The rationale for any jealousy-focused or jealousy-guided dysfunctional coping
strategies (cognitive, such as worry or rumination, or behavioral, such as challenging
or restricting the partner) needs to be carefully considered. What is the evidence
against the pros? Overall, does the strategy deliver? Is the outcome attainable, that
is, is it realistic to demand complete certainty about your partner’s inner experience,
or about their movements? How does the strategy deviate from a healthy approach
to relationship building? What is a sounder alternative to worrying or ruminating?
(This line of questioning may expose unhelpful metacognitive beliefs about lacking
control over worry/rumination, which need to be addressed – see Wells, 2009.)
Overlapping with OCD approaches (see Frost & Steketee, 2002; Van Niekerk,
2018), the interpretation of OJ intrusive thoughts may benefit from consideration.
Rather than designating actual threat, or a responsibility to act, have the client
cultivate a stance toward their thoughts and feelings which recognizes that these are
frequently irrelevant, transient, subjective mental events. In the absence of “hard
evidence” to support their validity, it is better to accept and mindfully observe them,
rather than try to control or act on them. The person is encouraged “to make room”
for contradictory thoughts and feelings (e.g., resentment, love, and ire), and recognize that they have a right to their experience, but then refrain from unhelpful
responses. A view of trust as a desirable, relationship-building attitude is promoted,
and this entails not demanding 100% certainty about a partner’s behavior. A model
of trust and acceptance of uncertainty can be explored in other interpersonal
domains (such as letting your best friend stay in your house while you are away).
If this works for the client, it can be helpful to name an alternative, healthier
identity, guiding and subsuming therapy change, for example, “wiser Pat.”
Leahy and Tirch (2008) suggest that insights and techniques from acceptance
and commitment therapy (ACT; Hayes et al., 2012) and metacognitive therapy
(MCT; Wells, 2009) can be helpful in promoting decentering from jealous feelings.
Such insights and techniques might include metaphors (e.g., “Passengers on a bus” –
the mind is noisy; jealous feelings are best accepted and disregarded, rather than
avoided or acted on; Hayes et al., 2012) and detached mindfulness technique, to
practice a more adaptive way of relating to thoughts (Wells, 2005).
These approaches are not incompatible with the occasional adjunctive use of
emotional regulation strategies, such as distraction or self-soothing behaviors, when
jealous distress is high and there is a high risk of lapsing into destructive behavior.
Yet, ideally, the longer-term focus should be on increasing the ability to practice
acceptance and mindful observation of distressed experience (or the more workaday alternative: to tolerate distress better and catastrophize about it less), while
refraining from unhelpful action.
Where there is evidence of dysfunctional reasoning in this domain, decatastrophizing about the meaning of actual infidelity and relationship breakdown can be
productive: “Are you really worthless if your partner cheats on you?” “You’ve
coped with failed relationships before, why should it be different this time?”
Core belief/self-narrative interventions (e.g., with themes of being an unlovable
or worthless person) require good therapy rapport and are important for increasing
275
276
Jan van Niekerk and Kieron O’Connor
the likelihood of enduring therapeutic change. Follow guidelines outlined in Young
et al. (2003) and Beck (2011).
As suggested above, use of strategies from different CBT approaches, for
example, cognitive therapy (CT), IBT, ACT, or MCT, holds much promise in the
treatment of OCRDs, but care should be taken that their combined use is coherent
in terms of the case formulation and does not confuse the client. “More” can
sometimes be less – a simpler strategy could be more powerful and efficient.
Possible unhelpful consequences of the sequencing of techniques need to be considered (e.g., Gkika & Wells, 2015). When considering whether to combine traditional CBT and third wave methods, it may be helpful to reflect on both
commonalities (Mennin et al., 2013) and differences (e.g. Hayes et al., 2013;
Herbert & Forman, 2013) in the treatment models, to ensure clinical alignment.
Farmer and Chapman (2016) provide further guidance.
Behavioral Strategies
A core focus of behavioral experimentation is on testing predictions about actual
and/or mental consequences of reduction of dysfunctional coping or problemsolving responses. Ecker (2012) suggests that a useful strategy for identifying
suitable options entails collaborating with the partner, by asking in which ways they
would be acting differently if the client did not have OJ. Using standard guidelines, a
list of mutually agreed experiments can then be designed in which the partner
modifies their behavior so as not to be encroached on by jealousy. The client then
responds by refraining from unhelpful responses and instead introducing valueguided behavior (e.g., jointly watch an arthouse film with erotic content and afterward discuss the artistic merits of the film instead of jealous worries). Relevant
predictions are tested (e.g., that distress will continue interminably or that the client
will not be able to function).
Ultimately, the range of experiments is limited by what the client is willing to
engage in. Where the client’s agreement cannot initially be obtained, further preparatory cognitive work may be helpful; however, be careful not to risk treatment
disengagement by being perceived by the client to be invalidating, resulting in
polarization. Experimentation can follow a graded approach, similar to exposure
and response prevention for OCD. Note the caveats regarding the use of some
imaginal exposure techniques in OJ described in Cobb and Marks (1979).
Couple Interventions
Involving the client’s partner can be a helpful adjunct to individual therapy.
Furthermore, couple-focused intervention may be useful where skill deficits become
apparent, such as in communication or sexual functioning, or where existing skills
are not brought to bear in the problematic domain. Focus on how couples can
communicate constructively about the jealousy problem, for example, negotiating
an agreed definition of their commitment, giving clear feedback about helpful
responses, and embracing a compassionate attitude.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Body-Focused Repetitive Behavior Disorders
The term “body-focused repetitive behaviors” (BFRBs) refers to a group of maladaptive, injurious, and nonfunctional behaviors directed toward the body, including hair-pulling, skin-picking, and nail-biting (O’Connor et al., 2005; Teng et al.,
2004). Individuals with BFRBs report diminished control over their behavior
(Schreiber et al., 2011) and a range of physical and psychological sequelae
(Diefenbach et al., 2005).
Pathological hair-pulling (HP) and skin-picking (SP) are included in DSM-5 as
trichotillomania and excoriation disorder, respectively, and these disorders are
discussed in Chapter 16. However, several other BFRPs are mentioned in the
diagnostic category of “Other Specified” OCRD, such as nail-biting, lip-biting,
and cheek-chewing.
Nail-biting or chewing (also called onychophagia) involves recurrent biting past
the nail bed and cuticles, often drawing blood, creating scarring, or resulting in red,
sore, and infected fingers (Penzel, 1995). Other lesser known BFRBs include
trichotemnomania (hair cutting), dermatophagia (compulsive skin biting), rhinotillexomania (nose picking), and scab eating disorder. Finally, there is neck and
knuckle cracking, or crepitation, often also termed “over-manipulation syndrome,”
where a person forces sudden movements up and down or left or right, to the neck
or spine; this causes rapid torsion, thereby compromising cervical or spinal stability
and potentially resulting in ligament damage.
The negative physical and psychological consequences of BFRBs are considerable (Diefenbach et al., 2002). Chronic nail-biting can damage fingers (Salmon-Ehr
et al., 1999), gums (Krejci, 2000), and teeth (Johansson et al., 1991). Individuals may
have infected and scarred fingers (Penzel, 1995) and be self-conscious and reluctant
to show their hands (Williams et al., 2007). Although occasional knuckle cracking
can be harmless, frequent cracking may lead to arthritis and dislocation of joints.
These torsions often break bubbles in the joints, making a snap, crackle, pop sound
and giving some satisfaction. Neck cracking is more serious and can lead to torn
cervical ligaments and even a stroke. Individuals with BFRBs may avoid seeing
doctors or dentists out of shame or fear of discovery, exacerbating their problems.
Clinical Model and Formulation
BFRBs are painful and produce physical consequences that result in shame and
distress (Bohne et al., 2002; Diefenbach et al., 2002, 2005; Woods et al., 2001), and
seem to be a distraction and a waste of time. However, they also seem to satisfy an
urge and deliver some form of gratification or reward (APA, 2000; Penzel, 1995).
One model, receiving recent research attention, is the emotional regulation
(ER) model. ER refers to the way in which individuals identify and respond to
emotional experience (Diefenbach et al., 2008). ER can be further understood as
the process through which individuals influence the presence, experience, and
expression of emotions (Gross, 1998). The ER model for BFRBs proposes that
277
278
Jan van Niekerk and Kieron O’Connor
BFRBs function as a maladaptive mechanism for regulating emotions. That is,
individuals with problematic body-focused behavior have difficulty regulating emotions and engage in BFRBs in an effort to avoid, decrease, or attenuate negative
affect. BFRBs persist despite aversive consequences because they are negatively
reinforced by relief from unpleasant emotions (Roberts et al., 2013).
The ER model has been supported by studies that demonstrate change in
affective states over the course of BFRB episodes. In clinical and nonclinical samples,
individuals with HP and SP consistently report that emotions such as boredom,
anxiety, tension, and frustration are present prior to BFRB and decrease during or
after episodes of HP or SP (Roberts et al., 2013). Guilt, shame, sadness, and anger
may develop subsequent to an episode of BFRB, but feelings of satisfaction, indifference, and relief are often present as well (e.g., Bohne et al., 2002; Duke et al., 2009;
Mansueto et al., 2007; Neal-Barnett & Stadulis, 2006; Wilhelm et al., 1999).
O’Connor and colleagues (O’Connor et al., 2001; Pélissier & O’Connor, 2004)
observed that many individuals with BFRBs demonstrate a style of planning characterized by unwillingness to relax and difficulty with appropriate pacing of tasks.
Individuals with this maladaptive planning style often try to do too much at once
and feel the need to always be productive. They are consequently susceptible to
frustration, impatience, and dissatisfaction when standards are not met, and to
boredom when productivity is impossible. The authors suggest that one of the
functions of BFRBs may be to release the tension generated by frustrated action
(O’Connor, 2002, 2005). That is, impatience, boredom, frustration, and dissatisfaction build, creating tension. Tension and negative emotions are subsequently
released or diminished through BFRBs.
This frustrated action model also ties in negative emotions with aspects of
perfectionism. In particular, there is a strong link between aspects of perfectionism
and emotional regulation (Roberts et al., 2015). Although there are various types of
perfectionism, the perfectionist components linked with pathology tend to be high
standards and self-criticism (Dunkley et al., 2006). Self-criticism seems to be the key
dysfunctional element.
According to Thompson and Zuroff (2004), there are two forms of selfcriticism: (1) comparative, where the person compares the self unfavorably to others
and (2) internalized, where the person falls short of their own standards, resulting in
feelings of shame, for example, “I’m ashamed to be me” or “I’m ashamed I cannot
live up to my own high standards.” Gilbert and Procter (2006) postulated two types
of shame: (1) internal shame, characterized by a devaluation of the self as inadequate and (2) external shame, when the person projects the thoughts and feeling of
others on to the self. Noble (2012) found shame to be an important mediator
between perfectionism and symptoms of HP. Shame is also frequently reported in
BFRBs (Weingarden & Renshaw 2015), to the extent that these authors propose
that shame is the principal trigger for BFRBs, particularly shame about the body.
According to this model, BFRBs serve to neutralize the shame. But of course, the
consequences of BFRBs (e.g., damaged nails) elicit further shame and so produce a
negative reinforcement cycle.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Maladaptive
cognitive
emotion
regulation
strategies
Perfectionism
Self-criticism
Shame
BFRBs
Figure 14.2. BFRB model.
Hence the model shown in Figure 14.2 might further refine the mechanism of
BFRB onset. Here perfectionism creates the core emotion of shame, mediated by
self-criticism, and frustrated action is a side consequence.
Treatment
Current treatment approaches generally involve identification of multimodal triggers and implementation of stimulus and response control. But current treatments
rarely directly address emotional regulation, although adjuncts of mindfulness and
ACT may indirectly influence mood (Flessner et al., 2008). Habit reversal training
(HRT) has been used to treat a wide variety of BFRBs and allows short-term
improvement, although this was not sustained over the long term (Morris et al.,
2013). Mansueto and colleagues (2007) developed the comprehensive behavioral
(ComB) model, which adds to HRT by encompassing a wide range of cognitive,
affective, motor, sensory, and environmental modalities.
When doing HRT, the therapist and client conduct a functional analysis and
identify triggers. They then implement the following:
1. Awareness exercises, involving monitoring the habit
2. Stimulus control (either internal or external stimuli, that is, state or signal
control), for example, becoming aware that a certain activity provokes the habit
and so avoiding it
3. A competing response, which does not necessarily need to be anatomically
antagonist but should prevent the BFRB. Since most BFRBs start with the
hand, or a manipulation of the hand, this initial move needs preventing by
introduction of an opposing action, like keeping the hand on the knee or in
contact with a hard surface.
An alternative is the cognitive psychophysiological (CoPs) approach, which has
proved effective on a range of specified and unspecified BFRBs (see O’Connor
et al., 2015). This involves addressing the underlying emotions triggering BFRBs, as
well as more multimodal stimulus control. Stages include increasing awareness of
but also understanding the way trigger situations are evaluated. For this purpose, an
adaptation of George Kelly’s construct theory (Blowers & O’Connor, 1996) is used,
where evaluations are elicited specific to each situation. The way the person
reacts in the situation is then addressed cognitively and behaviorally with the
aim of introducing greater flexibility, in particular, transplanting approaches in
low-risk situations to high-risk situations – so adding habit control and modifying
279
280
Jan van Niekerk and Kieron O’Connor
tension-producing thoughts and the style of planning of actions. The latter refers to
how people plan and approach action and whether they prepare in an adaptive or
maladaptive manner. Meta-cognitions of judgment and self-blame are also
addressed. (A complete manual can be found in O’Connor et al., 2017.)
BFRB Case Study. The following case is of an atypical BFRB treated with the
CoPs approach (described above). The client presented with repetitive biting of the
inner lip and the skin around the fingernails, and then rubbing her arms up and
down symmetrically, all in the same movement, accompanied by tension in her jaw
and shoulders. She would repeat the habit in front of the mirror, observing herself
for several minutes, several times per day.
The habit developed at the age of 13 years when she claimed she became more
self-conscious about comments at school about her exposed body parts (arms and
face). Auto-observation revealed that she performed the habit about 20 times a day
for several minutes. On evaluation, she had mild depression and she reported that
she was very emotional and had problems controlling negative emotions, frequently
ending up crying. She said she reacted emotionally very quickly and had problems
regulating stress.
She scored high on the personal standards and organization subscales of the
Frost Multidimensional Perfectionism Inventory and was quite rigid in her planning
and ordering of events. This was, she claimed, so as not to waste time. She found it
difficult when routines were changed or things went wrong.
She also judged her self-worth entirely on her appearance and presentation to
others and was particularly concerned that her arms and hands should appear
correctly groomed. She also walked with a posture where her hands were kept close
to her face as though ready to correct some blemish.
She showed a classic overactive style of planning (as measured on the Style of
Planning Action Questionnaire; O’Connor et al., 2015), where she found it difficult
to sit still, and she was constantly attempting to do too much. She was afraid of
wasting time and would rapidly become impatient when, for example, her computer
malfunctioned at work, or when engaged in what she considered unnecessary
conversations and social events. Sometimes she dealt with stress by suddenly feeling
very tired and then “tuning out” of the situation.
In the initial stages of the 10-week treatment, she had problems completing the
relaxation exercises since she felt that she was not tense and therefore she also felt
that she was wasting her time. This response is not atypical since people with BFRBs
often do not appreciate that their tension may be high. Eventually, she realized that
she was tense and the relaxation and discrimination exercises helped her acknowledge tension and therefore enhance her ability to respond more flexibly.
She recognized that her thoughts and attitude contributed to her high levels of
impatience. Hence, she profited from cognitive restructuring to change her thoughts
about conversations with others, her idea of how others perceived her, and the need
to be and appear perfect. Rather than constantly dividing her attention between the
task and the conversation at hand, and self-observation, she dropped the selfobservation in favor of immersion in the task.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
At the eighth session, her habit intensity and duration were reduced, and at
post-treatment, habit frequency was significantly reduced. At post-treatment, there
was still some tension and she reported 70% control over the habit. At the sixmonth follow up, she said she had maintained the changes in her thinking and
action, and claimed that she did not even think about the habit and controlled it
unconsciously. She reported that the goals of therapy had been achieved, and this
was supported by clinical and psychometric pre-post evaluations.
Body Dysmorphic Disorder with Visible Defect
Although most body dysmorphic disorder (BDD) is invisible and only the person
sees the defect, sometimes there is a minimal defect. Typically, this may involve a
mild facial blemish or, in men, a mild body asymmetry or muscle underdevelopment. In these cases, more than in regular BDD, there is frequently other underlying pathology – in particular, impaired social judgment, social phobia,
overinvestment in appearance, and doubts about being generally unattractive, or
having a general insecurity, such as about gender identity (Philips, 2009).
The behaviors found in more typical BDD, such as mirror-checking, applying
makeup, camouflaging, frequent clothes changing, avoidance, and reassuranceseeking, may have greater apparent justification due to the objective defect.
Recourse to surgery may be more frequent, and greater family accommodation of
dysfunctional responding is problematic and may sanction avoidance. Exposure is
difficult to achieve without prior belief change.
Standard CBT Approaches
Veale and Neziroglu (2010) recommend several CBT strategies for BDD, some of
which are listed below. In the first instance, offer the client psychoeducation and
deal with ambivalence about change. Identify and present the self-sabotaging loops
of coping behaviors such as staying at home and avoidance, and identify the best
alternative options for moving toward goals. Although support groups can help, it is
important to avoid Google chat groups and internet searches, which may feed BDD.
Address unhelpful judgments and metacognitions (e.g., “Why can’t I be like
everyone else? I shouldn’t be suffering like this”). Educate the client in addressing
self-critical appraisals using rational responding. Alternatively, mindfulness training
can help in promoting decentering from self-flagellating thinking.
At the same time, it is important to learn to direct attentional focus outward/
externally, since it is so often fixated internally. Mirror retraining can be helpful and
involves looking in a mirror for a specified period of time and not selectively
overfocusing on specific features. Other exercises in attentional retraining involve
listening and talking exercises where the person learns to switch attention and
distract themselves.
Gradually giving up off safety behaviors is a standard behavioral technique
where the person eliminates self-attacking behaviors, hypervigilance, and
281
282
Jan van Niekerk and Kieron O’Connor
disguising, and examines the results. For example, the person ceases comparing
their present appearance with that in old photos. Instead, new photos can be taken
and the person asked to judge them as a third person (Wilhelm et al., 2013).
Other cognitive strategies involve examining the questionable utility of rumination and worry, and how they get in the way of solving the real problems. Imagery
rescripting can be helpful – here the person replays a scene where they felt their
body part was demeaned in the past and rescript it from a position of power. This
involves reframing the demeaner, and replying to the criticism from the perspective
of the adult self, who comes to the rescue. Further, behavioral activation is important since so many clients become inactive and restrict themselves to their homes as a
mechanism of defense.
Finally, if the BDD entails skin- or scab-picking, then habit reversal (see above)
may be combined with other CBT techniques. An alternative is to introduce CoPs
as a combined or standalone strategy, which is described above in the BFRBs
section on treatment.
Inference-Based Therapy
Traditional cognitive-behavioral models propose that unwanted intrusive thoughts
are a universal experience, but that if they are appraised as holding negative
implications for the person (e.g., “People will reject me because I look hideous”),
the person will become preoccupied and try to “neutralize” or otherwise suppress
the thought, consequently maintaining the preoccupation. On the other hand, IBT
conceptualizes obsessions as doubts that are supported by an inductively generated
and purely idiosyncratic narrative. In fact, IBT considers that there is a subjective
narrative that precedes and supports the obsessional doubt before the intrusion is
even appraised. The reasoning process, hypothesized to be common to OCD and
BDD, and to lead to obsessions, is termed inferential confusion (O’Connor et al.,
2005).
Those whose reasoning processes are characterized by inferential confusion
tend to distrust their senses and common sense, and to invest in remote and often
imaginary possibilities at the expense of reality. See Table 14.1 for a list of inferential reasoning errors with examples applied to BDD. The aim of IBT is to modify the
reasoning narrative producing the doubt, and to return the person to the world of
commonsense perception.
In IBT, obsessions are conceptualized as a two-step process. The person first
arrives at one or more primary doubts about imperfection through a subjective
narrative (inferential confusion) that leads them to confuse reality in the here and
now with subjective possibilities.
In BDD, overvalued doubting beliefs are common, that is, doubting with high
conviction that certain aspects of their appearance are normal, for example, “My
body is weirdly shaped” or “My skin is too oily.” The primary inference (e.g., “My
nose is ugly”) is generally followed by one or more secondary inferences, that is, the
negative consequences that are expected to derive from the primary doubt, such as:
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
283
Table 14.1. Overview of reasoning errors in IBT (from Taillon et al., 2013)
Reasoning error
Definition
Example
Category errors
Confusing two logically distinct
properties or objects
Apparently
comparable
events
Selective use of
out-of-context
facts
Purely imaginary
sequences
Distrust of
normal
perception
Inverse inference
Confusing two distinct events
separated by time or place
My sister had a nose job and got a lot of
compliments. This procedure is therefore also
justified for me.
Teenagers with acne get mocked, which means
that I can also be laughed at if my skin is imperfect.
Inappropriately applying abstract
facts to specific personal contexts
Actors with physical flaws don’t get as many
contracts.
Making up convincing stories and
living them
Disregarding the senses in favor
of going deeper into reality
When people look at me, I feel my nose grows
even bigger and I become even uglier.
People say I look okay but it doesn’t mean that
I actually do.
Inferences about reality precede
rather than follow from
observation
People in the room have laughed, which might
mean I look ugly.
“Nobody will ever love me because of my hideous nose.” Contrary to traditional
cognitive behavioral approaches, IBT therefore addresses the initial content of the
BDD doubt by intervening in the process of inferential confusion that led to it,
rather than focusing on the perceived negative consequences arising from its
appraisal. The rationale behind IBT is that addressing the initial BDD doubt will
ultimately remove the trigger for the secondary distressing appraisals (Aardema &
O’Connor, 2003).
All the other BDD maintenance factors ultimately have to be eliminated, for
example, attentional hypervigilance, ritualistic behaviors, thought suppression, and
avoidant strategies, but the primary focus is on eliminating inferential confusion. In
the most elegant solution, when the primary doubt is invalidated, the need for the
use of dysfunctional coping strategies is eliminated. The IBT formulation applies
equally to BDD with and without visible defect.
BDD Case Study 1. Joe was a 31-year-old single man who worked as a fireman.
He presented complaints of disabling preoccupations concerning his muscle mass
and bone structure that have been present for the past 18 years. Joe stated that he
was scarred by experiences of being mocked as a child, when he remembered being
called ugly and scrawny. In fact, his body shape was objectively asymmetrical and he
was weaker on one side.
Joe’s main obsession was that if he were not “bigger,” the consequence would
be that he would be unable to find a girlfriend. Joe engaged in a series of compulsive
and safety-seeking behaviors such as working out excessively (two hours per day,
five days per week), eating large amounts of food, checking his appearance in the
mirror (for approximately 20 minutes, four to five times per day), and comparing his
284
Jan van Niekerk and Kieron O’Connor
appearance to people around him and people on the internet (approximately two to
three hours per day). His social network and daily functioning were only mildly
impaired – he had a few friends and was functional in most domains of his life. Joe
avoided bars, discos, and gyms (he worked out at home) and had stopped weighing
himself for eight years.
Apart from a primary diagnosis of BDD (in his case, “muscle dysmorphia”), Joe
also met criteria for social anxiety disorder and presented borderline and avoidant
personality traits. He was not taking any medication.
At baseline, before therapy, Joe’s total BDD–Yale–Brown Obsessive
Compulsive Scale (BDD-Y-BOCS) score was 28, in the severe symptomatic range.
Severity of obsessions and compulsions was equivalent, and insight was fair, with
mild associated depressive and anxious symptoms.
Joe’s therapist assessed him over four sessions. Obsessions and compulsions
were listed and clarified (see above). Obsessions were then explored further and
elaborated in terms of IBT’s primary and secondary inferences. The question:
“What do you fear would happen if you were prevented from engaging in your
compulsive and reassuring behaviors?” will often allow specification of the person’s
secondary inference. In Joe’s case, he feared that if he did not perform his rituals
(e.g., working out), then he “would not find a girlfriend” and “would be less
attractive and popular.” Joe was 90% convinced that these consequences would
be realistic if he did not perform his compulsions.
Starting from the identified feared consequences, the therapist can then work
back to the initial obsessional doubt by posing the question: “Which doubt would
have to be eliminated for you to be able to resist the compulsions?” In Joe’s case,
the primary initial BDD doubt was: “Perhaps I’m not big enough to get noticed/
respected/loved.” Joe rated his doubt as 100% probable.
The first part of the treatment aims at clarifying the distinction between subjective and real doubt. Real doubt is justified by new sense-based information (e.g.,
wondering if it is cold outside after seeing people wearing jackets and scarves),
whereas subjective/imaginary doubt is not based on any new information that signals
problems in the here and now (e.g., when there is no realistic precipitant for Joe
ruminating, when waking up in the morning, on whether he is attractive enough to be
loved). There may be an external trigger (e.g., seeing one’s reflection in the mirror),
but the content of the doubt does not rely on normal attention to new sense-based
information in the present (e.g., someone telling Joe that he is ugly). Thus, Joe’s
reaction to the doubt (e.g., mirror gazing) does not lead to further information which
allows him to resolve the doubt, but rather pushes him to go beyond his senses and
commonsensical interpretation, unproductively venturing into the imagination.
The next stage of therapy reveals to the person the reasoning supporting their
doubts. The justification can seem quite logical since the arguments supporting the
doubt are not necessarily incorrect on their own (e.g., “People do sometimes get
rejected because of their looks”).
It is often not the content of the justification which renders the doubt “illogical”
but rather the context in which it occurs. Joe came up with the following arguments
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
to justify his doubt: “In movies, it’s always the attractive guy that ends up with the
pretty girl” and “There are always women lining up at male strip clubs, therefore
this is the kind of man (attractive and muscular) that women are attracted to.”
These arguments did not derive from realistic information in the present, but rather
from previous experiences and his own logical calculations that were irrelevant to
Joe’s own situation in the here and now. He was able to recognize this, but still
found it difficult not to give credence to these justifications.
The objective of IBT’s third stage is for the person to recognize that the BDD
doubt originates from them rather than from their senses. IBT therefore does not
rely on convincing the person of the unlikelihood of the doubt, but proposes its
irrelevancy to the context in which it arises. This was illustrated in Joe’s case by
using an OCD example which was neutral to him – someone doubting that their
hands might be dirty without any sense-based information justifying such a doubt
(e.g., seeing dirt on their hands or smelling a bad odor from their hands).
The next stage of IBT highlights the power of the imagination and explains how
convincing, subjective reasoning leads the person into the BDD doubt. The “story”
behind the doubt is countered by developing an alternative story exclusively based
on realistic information in the here and now. The aim is to come up with an
alternative story, rather than to “disprove” the BDD story. Although Joe was able
to recognize both stories as being equally possible, he still continued to invest more
in the BDD story, as is normal at the start:
Joe’s BDD story:
Joe’s alternative
story:
Maybe I am not big enough to get noticed/respected/
loved. In the past, I have elicited positive reactions from
women because I was in good shape. This proves that
I must continue working out. The bigger I will be, the
more I will get noticed, just like the dancers in the clubs.
These men look good and there are always women lining
up at clubs to see them. This is therefore the kind of man
that attracts women. Also, in movies, it is always the guy
with the good looks that ends up with the pretty girl. It is
not surprising that men who are in shape feel better,
have more confidence, and are more popular.
I don’t need to be big to get noticed/respected/loved.
I can very well be, noticed, respected, and loved
regardless of my appearance or physical condition. I am
a good listener, I have good communication skills, and
I am respectful of others. I enjoy helping people; in fact,
that’s what I chose to do as a job. I have a good sense of
humor and I don’t take myself too seriously. I am easy to
approach and I enjoy being with people, talking to them
and reassuring them. I am more than a weight or a body.
All these qualities contribute much more to the love and
respect that people can have for me.
285
286
Jan van Niekerk and Kieron O’Connor
IBT’s step 5 is to help the person create distance from their BDD doubt by
learning to identify a cross-over point when they leave reality behind and get caught
in a “BDD bubble,” where the imagination takes over. A metaphor of a bridge is
useful here: the therapist asked Joe to visualize himself crossing a bridge. On one
side was reality, his senses, and his normal everyday responses. On the other side
was the BDD doubt. The therapist asked Joe to move across the bridge toward the
doubt and identify the specific moment when the imagination started to jump in.
Although Joe understood the principle, he had to reflect to identify a precise
moment when his BDD took over. This exercise was repeated at home and in
sessions before Joe mastered identifying the cross-over point.
The next two steps of IBT cover the reasoning devices (see Table 14.1) in the
person’s narrative, which invalidly convinces them to be invested in the doubt. In
Joe’s case, the principal reasonings were category errors and selective use of out-ofcontext facts. Joe achieved a good understanding of this stage and participated well.
In step 8, the therapist covers the selectivity of the BDD doubt by exploring
situations where Joe did not experience such doubts. If the person can become
aware of other situations where they adopt a “normal” style of reasoning, it will
highlight the irrelevance of obsessional doubts in BDD situations. Joe’s therapist
used the example of crossing the street to illustrate this point. Joe agreed that in this
situation, he did trust reality to decide whether it was safe to cross the street (e.g.,
looking left and right, waiting for the green light). He did not invoke far-fetched
possibilities such as, “A car may cross the corner suddenly,” which would stop him
from ever crossing the street. Joe was able to appreciate that his reasoning differed
in BDD and “neutral” situations and that he chose not to trust his senses when it
came to BDD situations.
The next stage of IBT probably was most productive for him. The aim of step
9 is to discover the person’s vulnerable self-theme by exploring why the person
experiences doubts in one area, but not in others. Joe realized that his muscularity
was directly linked to his desire to be strong and a “real man” and that the BDD
doubt of not being big enough made him feel weak and lacking character. He was
gradually able to develop a more realistic view of himself by exploring and challenging the meaning of being “strong” and by considering and listing his other personal
attributes that illustrated strength of character.
The objective of IBT’s last step is to train the person in the proper use of their
senses in BDD situations. Joe spent a lot of time each day analyzing his reflection in
the mirror. Rather than representing appropriate reasoning about the meaning of
sensory feedback, these behaviors in fact reflect a distrust of sensory feedback. In
order to help Joe understand a “proper use of the senses,” his therapist used the
neutral example of combing his hair in the morning. However, as Joe gradually
learned to see his body and trust his senses using the same method as he did when
grooming his hair, he reported feeling that he was “not doing enough” or that he
was “missing something.” People with BDD often report this feeling, but as Joe’s
therapist told him, it was temporary and should disappear over time as he again
learned to trust his senses and reacquaint himself with a commonsensical reasoning
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
approach. Trusting the senses involves just accepting what the senses say using a
simple reasoning approach – therefore, not applying extra effort and invalidating
the initial impression by trying to go “deeper” – in other words, using the senses
exactly as the person does in other non-BDD walks of life.
Overall, Joe collaborated well in therapy and was compliant with homework
and session attendance. Although his symptoms improved consistently over the
weeks, working on his vulnerable self-theme (IBT’s step 9) appeared to have had
the biggest impact in reducing the strength of conviction in his obsession. In fact,
Joe’s score on the Overvalued Ideas Scale went from 6 at pre-treatment to 2 at
post-treatment. At the end of the therapy, a 79% reduction was observed in Joe’s
total BDD-Y-BOCS score (which fell in the subclinical range), with obsessions
slightly more severe than compulsions. Although already low at the beginning of
treatment, depressive and anxiety symptoms also decreased considerably (from
14 to 3 on the Beck Depression Inventory-II, and from 12 to 3 on the Beck
Anxiety Inventory).
Treatment of cases other than Joe shows that there tend to be differences in the
benefits derived from and the obstacles encountered at each treatment step (see
further examples described in Taillon et al., 2013). The focus of IBT on faulty
inferences and overinvestment in obsessional doubts fits with the recommendation
of Labuschagne et al. (2010) to include techniques from models of delusional
thinking in the treatment of BDD. This focus on reasoning processes occurring
before – and leading to – overinvested primary inferences fundamentally differentiates IBT from traditional CBT interventions, which typically target the feared
consequences derived from the initial doubts. Given the chronic nature of BDD,
it is noteworthy that with IBT, therapeutic gains appear to be maintained – or in
some cases even improved – six months following treatment, suggesting that
changes in reasoning processes and inferences endure (Taillon et al., 2013).
The protocol may benefit from adding a module addressing depression. This is
highlighted by the case study described below, which considers some of the difficulties encountered when attempting to treat a somatic delusion of having a terrible
appearance defect, in the context of a small, but objectively visible defect.
BDD Case Study 2. Dor was a 25-year-old female who had suffered from severe
BDD for seven years, during which she was confined to home. Her central concern
was that she was disfigured because of her facial acne. She claimed that she quit
school after numerous negative comments from peers, and with the acquiescence of
her parents. There were no stated reasons other than the acne for discontinuing
school.
Hence, the first treatment obstacle: significant others are more likely to agree with,
sympathize and accommodate clients with BDD with visible defect. Due to the
accommodation of her parents, Dor had missed several years of schooling, which
further complicated her recovery. The family dynamic was complex: Dor’s older
sister had broken off contact with her, considering her a “fake.” Her mother and
stepfather were very accommodating of her difficulties, agreeing to her requests to
stay in her room and be left alone, not go on outings, disengage from prescribed
287
288
Jan van Niekerk and Kieron O’Connor
home-based education, and not perform domestic tasks. If her stepfather insisted on
her doing the tasks, Dor would express anger and throw tantrums until she got her
way. Even when she became aggressive with her parents and had spent 28 days in
self-isolation, no behavioral limits were imposed using strategies of either positive
or negative reinforcement.
When questioned, in addition to what was wrong with her facial complexion,
Dor would identify another “60,000” things wrong with her, including hip size,
breast size, height, and body shape. The second obstacle: The initial (objective) label
of defect generalizes to multiple additional nondesired physical features.
Dor scored high on perfectionism, in particular, on the Personal Standards
Scale. She had poor to moderate insight on the Brown Assessment of Beliefs
Scale, low resistance to the obsessions, and low control over the compulsions. The
third obstacle: Despite some insight that her concerns might be unjustified, this did not
translate into an ability to resist or control the responses to her obsessions. Dor
admitted there was a psychological element in her BDD, but still wanted to change
her features surgically.
The fourth obstacle: Anticipation of social scrutiny was a large source of anxiety
for Dor, and before going out she would heavily camouflage herself with makeup in
order to hide her complexion – to a greater degree than is typical in BDD. Fifth, Dor
would exacerbate the barely visible facial marks by rubbing and tweaking at her skin
(also described in Veale & Neziroglu, 2010), hence exacerbating the signs and making
them more visible – so confirming the defect.
Finally, as well as clinical depression, and social anxiety in the moderate range,
Dor had antisocial personality traits – this raises the possibility that a manipulative
tactic may have been a factor in some of her BDD avoidance behavior. Thus,
additional comorbidity presents an amplified treatment challenge.
Treatment. Dor sporadically attended six CBT therapy sessions over four
months. She admitted that she would not judge others by their complexion. She
acknowledged that her views were selective and that her BDD did not present an
objective impediment to pursuing her life goals. A large component of therapy time
was focused on improving her mood by using behavioral activation strategies,
aiming to introduce greater planning and structuring of her activities. Some exercise
routines were established and Dor undertook more regular short trips outside her
home; however, more intensive exposure work was refused despite Dor having
benefited from brief exposure.
The therapist also allocated considerable effort to addressing maintenance
factors within the family system, such as improved parenting skills and negotiating
a decrease in voluntary accommodation of problem behavior and using strategies of
reinforcement in a consistent way to diminish symptom accommodation.
Outcome. Dor’s parents decided, against cautionary advice, to fund her pursuit
of surgery. She embraced this option and disengaged from therapy with a promise to
return after having had laser surgery. She was convinced that surgery would be her
salvation and would enable her to feel comfortable in society. With her parents’
agreement, she terminated therapy.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Olfactory Reference Syndrome
Olfactory reference syndrome (ORS) is a form of dysmorphophobia where the
person is afraid that they emit a body odor, either from a specific part of the body
or generally. This can include bad breath, genital scent, anal odor, armpit smell,
chemical or tainted aroma. In particular there is a belief that others can detect the
smell even if they do not communicate this.
The disorder verges on delusional and there may be a paranoiac element, where
the person is extra-vigilant to any possible social signal of smell detection, without
further proof (e.g., nose wrinkling, coughing, or sighing). Other compulsive behaviors
include typical BDD safety behaviors and strategies of over-disguising smells, avoiding
close gatherings or social occasions, and frequently checking for smells. The checking
behavior, as with OCD, frequently increases the doubt that there is a smell.
Phillips and Menard (2011) report the following ORS preoccupations: bad
breath, bad-smelling armpit/genitals/anus/feet/skin, flatulence, odd non-body emissions (old tires, rubbish), with symptoms present for one hour or more. Compulsive
responses included smelling oneself (80%), showering (68%), dietary modification
(45%), brushing teeth (40%), scrubbing nails (40%), laundering clothes (30%),
sniffing others for comparison (30%), and reassurance seeking: “Do I have bad
breath?” Other problem behaviors include camouflage, professing embarrassment,
social avoidance, drug use, quitting jobs or leaving situations abruptly, and even
suicide attempts. Mood disturbance, social anxiety, shame, and low self-esteem are
common. ORS prevalence is unknown.
The person often mis-smells and is inaccurate partly due to repetition but also due
to misinterpretation of normal body odors (www.katharinephillipsmd.com/). Phillips
and Menard (2011) recruited 20 patients, 60% female. The majority developed ORS
in their mid-twenties, with no remission at two-year follow-up. Their ORS-Y-BOCS
score was in the severe range, with moderate to severe interference in daily living.
They were smelling a mean of 2.9 (SD: 1.4) body locations. The Brown Assessment of
Beliefs Scale score overall was in the delusional range, with no to poor insight; but not
all were classified as delusional. Eighty-five percent reported they smelled their
unwanted body odor; 95% that others did; 88% had ideas of reference; 55–70%
masked the smell with powder, deodorant, perfume, or mints. Forty-four percent
sought alternative treatments (e.g., prescription mouthwash, tonsillectomy), which
may have displaced some ORS symptoms, but the disorder prevailed.
People with ORS may actually hallucinate smells, but as stated above, not all
are delusional; 15% seem to have poor to fair insight. Some medical problems can
lead to bodily excretions such as fish malodor syndrome (trimethylaminuria), but
there are no reports of overlap with ORS cases.
ORS can culminate in suicidal and violent behavior associated with strong
reactions to what patients see as deceptive responses by others. There is a strong
comorbidity with both social phobia and major depressive disorder, which generally
develop subsequent to ORS. For a screening test, see https://ocdla.com/olfactoryreference-syndrome-test.
289
290
Jan van Niekerk and Kieron O’Connor
Treatment for ORS is the same CBT as applied in BDD, but addressing ORS.
But there is a danger that unless the general theme of dysfunctional thinking
regarding smelling is addressed, and the smells are just dealt with one by one, the
person will replace the resolved body smell with another. Inference-based therapy
(IBT), discussed above, which unravels the person’s narrative about their smell,
could be of assistance here, as the following case study illustrates.
ORS Case Study. Mel was a 39-year-old male who had suffered from ORS since
early puberty. He traced the onset to concern about his grandfather, who lived with
the family and often through negligent hygiene would smell of urine or stale clothes,
details that were commented on by members of the family. Mel thought he himself
might emit such smells and hence felt constantly embarrassed or shameful.
Mel had comorbidities of depression and social anxiety. He had poor insight
and also paranoid tendencies, which classified him as between BDD and a somatic
delusion. He self-referred to our study on BDD.
Among other compensatory behaviors, Mel would try to camouflage his smell
with cologne, and frequently changed his clothes and left social situations and
spaces. He frequently quit jobs. He would not board a crowded bus and would
change carriages in the metro, convinced everyone could smell him. Interference
with daily living was severe, and apart from occasional visits by his family, he
was isolated.
An important feature of his ORS, which mimicked a paranoid presentation, was
his constant vigilance for the reactions of others. Any nose movement, sniffing, even
change in breathing pattern or head movements was confirmation that the other
person had smelled him. Reassurance from family members that he did not smell
was met with distrust and even a suspicion of deception: “Oh, they would say that to
make me feel better” or “They don’t want to admit the truth.’’
Mel was treated with IBT (protocol described in BDD section above) where the
justifications for his doubt about smelling and the reasoning errors in the narrative
were identified and replaced with trust in normal sense data as in other walks of life.
Reasoning errors included jumping prematurely to conclusions, imaginings, and
interpreting categories of events as similar when they were not associated. And of
course, he distrusted his senses and deviated from commonsense interpretations
of social signals. His ORS-Y-BOCS reduced to the mild range after 16 weeks of
treatment, as did his Beck Depression Inventory and ratings of social anxiety. Some
of his safety behaviors and vigilance were more difficult to eliminate, and here more
conventional techniques of attentional refocusing, distraction, and strategies aimed
at enhancing cognitive flexibility proved helpful additions to reinforce reality
sensing and eliminating confirmation bias.
Final Remarks
The chapter aimed to provide an overview of the CBT treatment strategies on offer
for addressing obsessional jealousy, body-focused repetitive behavior disorders,
body dysmorphic disorder with a visible defect, and olfactory reference syndrome.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Strategies covered include earlier CBT and behavior therapy approaches and newer
methods such as inference-based therapy, metacognitive therapy, and ACT. The
heterogeneity of presentation and therefore the treatment approach required is
considerable; in fact, the OCRD diagnostic category has been criticized as lacking
coherence (see Abramowitz & Jacoby, 2015). We therefore provide a very cursory
overview, but have made recommendations for further reading in the text. We hope
that the chapter can make a useful contribution to helping clients presenting with
these challenging problems.
FURTHER READING
Obsessional Jealousy
Ecker, W. (2012). Non-delusional pathological jealousy as an obsessive-compulsive spectrum
disorder: Cognitive-behavioural conceptualization and some treatment suggestions.
Journal of Obsessive-Compulsive and Related Disorders, 1, 203–210.
Leahy, R. L. (2018). The jealousy cure: Learn to trust, overcome possessiveness, and save your
relationship. New Harbinger.
Body-Focused Repetitive Behavior Disorders
O’Connor, K. P., Lavoie, M. E., & Schoendorff, B. (2017). Managing tic and habit disorders:
A cognitive psychophysiological approach with acceptance strategies. Wiley-Blackwell.
Body Dysmorphic Disorder
Wilhelm, S., Phillips, K. A., & Steketee, G. (2013). Cognitive-behavioral therapy for body
dysmorphic disorder: A treatment manual. Guilford Press.
Olfactory Reference Syndrome
Phillips, K. A., & Menard, W. (2011). Olfactory reference syndrome: Demographic and
clinical features of imagined body odor. General Hospital Psychiatry, 33, 398–406.
REFERENCES
Aardema, F., & O’Connor, K. (2003). Seeing white bears that are not there: Inference
processes in obsessions. Journal of Cognitive Psychotherapy, 17, 23–37.
Abramowitz, J. S., & Jacoby, R. J. (2015). Obsessive-compulsive and related disorders:
A critical review of the new diagnostic class. Annual Review of Clinical Psychology, 11,
165–186.
American Psychiatric Association [APA]. (2000). Diagnostic and statistical manual of mental
disorders (4th ed., text revision). American Psychiatric Association.
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Association.
Barlow, D. H., Farchione, T. J., Fairholme, C. P., Ellard, K. K., Boisseau, C. L., Allen, L. B.,
& Ehrenreich-May, J. (2011). Unified protocol for transdiagnostic treatment of emotional
disorders. Oxford University Press.
Beck, J. S. (2011). Cognitive behavior therapy: Basics and beyond (2nd ed.). Guilford Press.
291
292
Jan van Niekerk and Kieron O’Connor
Blowers, G., & O’Connor, K. P. (1996). Personal construct psychology in the clinical context.
University of Ottawa Press.
Bohne, A., Wilhelm, S., Keuthen, N. J., Baer, L., & Jenike, M. A. (2002). Skin picking in
German students: Prevalence, phenomenology, and associated characteristics.
Behavioural Modifications, 26, 320–339.
Cheung, F. M. (1998). Cross-cultural psychopathology. In A. S. Bellack & M. Hersen
(Eds.), Comprehensive clinical psychology, vol. 10: Sociocultural and individual differences (pp. 35–51). Pergamon.
Cobb, J. P., & Marks, I. M. (1979). Morbid jealousy featuring as obsessive-compulsive
neurosis: Treatment by behavioural psychotherapy. British Journal of Psychiatry, 134,
301–305.
DeSteno, D., Valdesolo, P., & Bartlett, M. Y. (2006). Jealousy and the threatened self:
Getting to the heart of the green-eyed monster. Journal of Personality and Social
Psychology, 91, 626–641.
Diefenbach, G. J., Mouton-Odum, S., & Stanley, M. A. (2002). Affective correlates of
trichotillomania. Behaviour Research and Therapy, 40, 1305–1315.
Diefenbach, G. J., Tolin, D. F., Hannan, S., Crocetto, J., & Worhunsky, P. (2005).
Trichotillomania: Impact on psychosocial functioning and quality of life. Behaviour
Research and Therapy, 43, 869–884.
Diefenbach, G. J., Tolin, D. F., Meunier, S., & Worhunsky, P. (2008). Emotion regulation and
trichotillomania: A comparison of clinical and nonclinical hair pulling. Journal of
Behavior Therapy and Experimental Psychiatry, 39, 32–41.
Duke, D. C., Bodzin, D. K., Tavares, P., Geffken, G. R., & Storch, E. A. (2009). The
phenomenology of hairpulling in a community sample. Journal of Anxiety Disorders,
23, 1118–1125.
Dunkley, D. M., Zuroff, D. C., & Blankstein, K. R. (2006). Specific perfectionism components
versus self-criticism in predicting maladjustment. Personality and Individual Differences,
40, 665–676.
Ecker, W. (2012). Non-delusional pathological jealousy as an obsessive-compulsive spectrum
disorder: Cognitive-behavioural conceptualization and some treatment suggestions.
Journal of Obsessive-Compulsive and Related Disorders, 1, 203–210.
Farmer, R. F., & Chapman, A. L. (2016). Behavioral interventions in cognitive behavior
therapy: Practical guidance for putting theory into action. (2nd ed.). American
Psychological Association.
Flessner, C. A., Busch, A. M., Heideman, P. W., & Woods, D. W. (2008). Acceptanceenhanced behavior therapy (AEBT) for trichotillomania and chronic skin picking:
Exploring the effects of component sequencing. Behavior Modification, 32, 579–594.
Frost, R., & Steketee, G. (2002). Cognitive approaches to obsessions and compulsions:
Theory, assessment and treatment. Elsevier.
Gilbert, P., & Procter, S. (2006). Compassionate mind training for people with high shame
and self-criticism: Overview and pilot study of a group therapy approach. Clinical
Psychology and Psychotherapy, 13, 353–379.
Gkika, S., & Wells, A. (2015). How to deal with negative thoughts? A preliminary comparison of detached mindfulness and thought evaluation in socially anxious individuals.
Cognitive Therapy and Research, 39, 23–30.
Gross, J. (1998). The emerging field of emotion regulation: An integrative review. Review of
General Psychology, 2, 271–299.
Hayes, S. C., Levin, M. E., Plumb-Vilardaga, J., Villatte, J. L., & Pistorello, J. (2013).
Acceptance and commitment therapy and contextual behavioral science: Examining
the process of a distinctive model of behavioral and cognitive therapy. Behavior
Therapy, 44, 180–198.
Other Specified and Unspecified Obsessive-Compulsive and Related Disorders
Hayes, S. C., Strosahl, K. D., & Wilson, K. G. (2012). Acceptance and commitment therapy:
The process and practice of mindful change (2nd ed.). Guilford Press.
Herbert, J. D., & Forman, E. M. (2013). Caution: The differences between CT and ACT may
be larger (and smaller) than they appear. Behavior Therapy, 44, 218–223.
Johansson, A., Fareed, K., & Omar, R. (1991). Analysis of possible factors influencing the
occurrence of occlusal tooth wear in a young Saudi population. Acta Odontologica
Scandinavica, 49, 139–145.
Kingham, M., & Gordon, H. (2004). Aspects of morbid jealousy. Advances in Psychiatric
Treatment, 10, 207–215.
Krejci, C. B. (2000). Self-inflicted gingival injury due to habitual fingernail biting. Journal of
Periodontology, 71, 1029–1031.
Labuschagne, I., Castle, D. J., Dunai, J., Kyrios, M., & Rossell, S. L. (2010). An examination
of delusional thinking and cognitive styles in body dysmorphic disorder. Australian and
New Zealand Journal of Psychiatry, 44, 706–712.
Leahy, R. L. (2018). The jealousy cure: Learn to trust, overcome possessiveness, and save your
relationship. New Harbinger.
Leahy, R. L., & Tirch, D. D. (2008). Cognitive behavioral therapy for jealousy. International
Journal of Cognitive Therapy, 1, 18–32.
Mansueto, C. S., Thomas, A. M., & Brice, A. L. (2007). Hair pulling and its affective correlates
in an African-American university sample. Journal of Anxiety Disorders, 21, 590–599.
Mennin, D. S., Ellard, K. K., Fresco, D. M., & Gross, J. J. (2013). United we stand: Emphasizing
commonalities across cognitive-behavioral therapies. Behavior Therapy, 44, 234–248.
Morris, S. H., Zickgraf, H. F., Dingfelder, H. E., & Franklin, M. E. (2013). Habit reversal
training in trichotillomania: Guide for the clinician. Expert Review of Neurotherapeutics,
13, 1069–1077.
Neal-Barnett, A., & Stadulis, R. (2006). Affective states and racial identity among AfricanAmerican women with trichotillomania. Journal of the National Medical Association, 98,
753–757.
Noble, C. L. (2012). The relationships among multidimensional perfectionism, shame and
trichotillomania symptom severity. Dissertation Abstracts International: Section B: The
Sciences and Engineer, 74(2-B(E)).
O’Connor, K. (2002). A cognitive-behavioral/psychophysiological model of tic disorders.
Behaviour Research and Therapy, 40, 1113–1142.
O’Connor, K., Audet, J.-S., Julien, D., Aardema, F., Laverdure, A., & Lavoie, M. E. (2015).
The style of planning action (STOP) questionnaire in OCD spectrum disorders.
Personality and Individual Differences, 86(Supplement C), 25–32.
O’Connor, K., Koszegi, N., Aardema, F., Van Niekerk, J., & Taillon, A. (2009). An inferencebased approach to treating obsessive-compulsive disorder. Cognitive and Behavioral
Practice, 16, 420–429.
O’Connor, K. P. (2005). Cognitive behavioral management of tic disorders. John Wiley & Sons.
O’Connor, K. P., Aardema, F., & Pélissier, M.-C. (2005). Beyond reasonable doubt:
Reasoning processes in obsessive compulsive disorder and related disorders. John Wiley
& Sons.
O’Connor, K. P., Brault, M., Robillard, S., Loiselle, J., Borgeat, F., & Stip, E. (2001).
Evaluation of a cognitive-behavioural program for the management of chronic tic and
habit disorders. Behaviour Research and Therapy, 39, 667–681.
O’Connor, K. P., Lavoie, M. E., Robert, M., Stip, E., & Borgeat, F. (2005). Brain–behaviour
relations during motor processing in chronic tic and habit disorder. Cognitive and
Behavioral Neurology, 18, 79–88.
O’Connor, K. P., Lavoie, M. E., & Schoendorff, B. (2017). Managing tic and habit disorders:
A cognitive psychophysiological approach with acceptance strategies. Wiley-Blackwell.
293
294
Jan van Niekerk and Kieron O’Connor
Pélissier, M.-C., & O’Connor, K. (2004). Cognitive-behavioural therapy of trichotillomania,
targeting perfectionism. Clinical Case Studies, 3, 57–69.
Penzel, F. (1995). Skin-picking and nail biting: Related habits. In Touch, 2, 1–10.
Philips, C. B. (2009). Student portfolios and the hidden curricullum on gender: Mapping
exclusion. Blackwell Publishing.
Phillips, K. A., & Menard, W. (2011). Olfactory reference syndrome: Demographic and
clinical features of imagined body odor. General Hospital Psychiatry, 33, 398–406.
Roberts, S., O’Connor, K., Aardema, F., & Bélanger, C. (2015). The impact of emotions on
body-focused repetitive behaviors: Evidence from a non-treatment-seeking sample.
Journal of Behavior Therapy and Experimental Psychiatry, 46, 189–197.
Roberts, S., O’Connor, K., & Bélanger, C. (2013). Emotion regulation and other psychological
models for body-focused repetitive behaviors. Clinical Psychology Review, 33, 745–762.
Salmon-Ehr, V., Mohn, C., & Bernard, P. (1999). Longitudinal melanonychia consecutive to
nail biting. Annales de Dermatologie et de Vénéréologie, 126, 44–45.
Schreiber, L., Odlaug, B. L., & Grant, J. E. (2011). Impulse control disorders: Updated review
of clinical characteristics and pharmacological management. Frontiers in Psychiatry, 2,
1–11.
Simpson, H. B., & Zuckoff, A. (2011). Using motivational interviewing to enhance treatment
outcome in people with obsessive-compulsive disorder. Cognitive and Behavioral
Practice, 1, 28–37.
Taillon, A., O’Connor, K., Dupuis, G., & Lavoie, M. (2013). Inference-based therapy for
body dysmorphic disorder. Clinical Psychology and Psychotherapy 20, 67–76.
Teng, E. J., Woods, D. W., Marcks, B. A., & Twohig, M. P. (2004). Body-focused repetitive
behaviors: The proximal and distal effects of affective variables on behavioral expression. Journal of Psychopathology and Behavioral Assessment, 26, 55–64.
Thompson, R., & Zuroff, D. C. (2004). The Levels of Self-Criticism Scale: Comparative selfcriticism and internalized self-criticism. Personality and Individual Differences, 36,
419–430.
Van Niekerk, J. (2018). A Clinician’s guide to treating OCD: The most effective CBT
approaches for obsessive-compulsive disorder. New Harbinger.
Veale, D., & Neziroglu, F. (2010). Body dysmorphic disorder: A treatment manual. Wiley.
Weingarden, H., & Renshaw, K. D. (2015). Shame in the obsessive compulsive related
disorders: A conceptual review. Journal of Affective Disorders, 15(171), 74–84.
Wells, A. (2005). Detached mindfulness in cognitive therapy: A metacognitive analysis and
ten techniques. Journal of Rational-Emotive & Cognitive-Behavior Therapy, 23, 337–355.
(2009). Metacognitive therapy for anxiety and depression. Guilford Press:.
Wilhelm, S., Keuthen, N. J., Deckersbach, T., Engelhard, I. M., Forker, A. E., Baer, L.,
O’Sullivan, R. L., & Jenike, M. A. (1999). Self-injurious skin picking: Clinical characteristics and comorbidity. Journal of Clinical Psychiatry, 60, 454–459.
Wilhelm, S., Phillips, K. A., & Steketee, G. (2013). Cognitive-behavioral therapy for body
dysmorphic disorder: A treatment manual. Guilford Press.
Williams, T. I., Rose, R., & Chisholm, S. (2007). What is the function of nail biting? An analog
assessment study. Behavior Research and Therapy, 45, 989–995.
Woods, D. W., Friman, P. C., & Teng, E. J. (2001). Physical and social impairment in persons
with repetitive behavior disorders. In D. W. Woods & R. G. Miltenberger (Eds.), Tic
disorders trichotillomania, and other repetitive behavior disorders: Behavioral approaches
to analysis and treatment (pp. 33–52). Springer.
Young, J. E., Klosko, J. S., & Weishaar, M. E. (2003). Schema therapy: A practitioner’s guide.
Guilford Press.
15
A Cognitive Behavioral Approach to Body
Dysmorphic Disorder: Assessment, Treatment,
and New Developments
Oliver Sündermann and David Veale
Introduction
Body dysmorphic disorder (BDD) is a condition that is characterized by a disabling
preoccupation with a perceived defect in one’s normal physical appearance. BDD
affects about 2% of the general population (Veale et al., 2016) with similar rates in
men and women, but prevalence rates are much higher in clinical settings, particularly in dermatological clinics (11.3%), general cosmetic settings (13.2%), and
rhinoplasty clinics (20%). The onset of BDD tends to be during adolescence,
remains chronic if untreated, and is associated with poor quality of life, high risk,
and significant impairment in school, interpersonal, and work domains (Angelakis
et al., 2016; Bjornsson et al., 2013; Didie et al., 2012). Hospitalization is common,
and clients often accumulate significant financial debt from cosmetic surgeries.
Earlier onsets are associated with higher severity, comorbidity, and risk
(Bjornsson et al., 2013). Suicidality is a grave concern (Angelakis et al., 2016) with
rates of completed suicide around 45 times higher in BDD than in the general
population (Phillips & Menard, 2006). BDD is both under-recognized and underreported. Shame and stigma are main treatment barriers (Marques et al., 2011), and
those who seek help are often misdiagnosed (e.g., with social anxiety), have their
difficulties minimized, or receive inappropriate and ineffective treatments.
Diagnostic Criteria and Phenomenology
The Diagnostic and Statistical Manual of Mental Disorders (DSM) lists in its fifth
edition the following criteria for BDD: a preoccupation with one or more perceived
flaws in physical appearance that are not or only slightly noticeable by others, for at
least an hour a day; repetitive overt or covert compulsive behaviors (e.g., mirror
checking, excessive grooming, reassurance seeking) or mental acts (e.g., comparing,
rumination) in response to appearance concerns, causing significant distress or
impairment, and appearance concerns are not better explained by concerns with
body fat or weight. DSM-5 requires clinicians to specify whether the preoccupation
295
296
Oliver Sündermann and David Veale
is better described as muscle dysmorphia, that is, a concern of too small or insufficient muscle mass, and to make a judgement about the person’s insight regarding
the conviction of their BDD beliefs (e.g., ‘I am ugly’; ‘I am deformed’). Historically,
BDD has been categorized as a somatoform disorder, because of its focus on the
body. However, DSM-5 classifies BDD in a new chapter, ‘Obsessive-Compulsive
and Related Disorders’ (OCRD), because of its similar comorbidity, family history,
and phenomenology of obsessive-compulsive disorder and other difficulties that
involve compulsive behaviors such as hoarding disorder or excoriation disorder.
Other diagnostic changes include referring to the person’s ‘defects or flaws in
appearance’ as ‘perceived’ and no longer as ‘imagined’ to better capture the person’s experience, and the inclusion of ‘repetitive behaviors’ as a characteristic BDD
feature that describes the person’s response to the perceived defect. The focus here
is on the form and content of the behavior rather than the function, and other
typical BDD behaviors that are less compulsive and repetitive may not be
adequately captured (e.g., seeking cosmetic surgery). Note that BDD is now also
recognized as a separate disorder by the International Classification of Diseases
(ICD) in its latest version ICD-11, linkcode 6B21 (https://icd.who.int/browse11/l-m/
en#/http%3a%2f%2fid.who.int%2ficd%2fentity%2f1321276661; see Veale &
Matsunaga, 2014, for a description of the ICD-11 Working Group’s proposal on
OCRD to revise the classification of BDD in ICD-11).
All areas of the body can be the focus of concern, but usually people with BDD
are most concerned about visible parts, especially their skin (e.g., perceived
wrinkles, scarring, acne, or ‘too dark’), hair (e.g., perceived baldness, thinning, too
straight, or ‘too curly’), and nose (e.g., shape and size) (Phillips et al., 2006), and the
preoccupation is usually focused on several body parts. Other common concerns
focus on body shape (e.g., hips, waist, stomach, or buttocks), size and structure of
face (e.g., jaw or chin), or concerns of asymmetry (e.g., asymmetric eyes, jaw, or
hairline); and sometimes complaints are non-specific (e.g., feeling ugly or ‘not
right’). Although clinical features of BDD appear similar across different cultures
and societies (Bohne et al., 2002; Cansever et al., 2003; Fontenelle et al., 2006), there
is some indication that Asian people are more concerned with straight hair and dark
skin, and have fewer body shape concerns than Caucasians (Marques et al., 2011),
possibly reflecting ethnic differences in beauty ideals. However, there is a general
lack of cross-cultural research of differences in phenomenology of appearance
concerns, related coping, and prevalence rates in non-Western countries (Veale
et al., 2016).
Cognitive, Perceptual, and Attentional Processes in BDD
Several processes and biases in attention and perception characterize BDD. For
example, people with BDD tend to have a detailed rather than holistic processing of
visual information (Deckersbach et al., 2000; Greenberg et al. 2014) and selectively
attend to the perceived flaws and other people’s responses towards them. A key
attentional process, albeit understudied, appears to be self-focused attention
(Windheim et al., 2011) onto internal imagery of how the person feels they look
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
to others. They are also more aesthetically sensitive to facial information than
controls, similar to people in the art and design profession (Lambrou et al., 2011);
have higher and more perfectionistic beauty standards (Buhlmann et al., 2002); and
overvalue the importance of appearance and self-objectify more than those without
BDD (Lambrou et al., 2012). People with BDD are more likely to misinterpret faces
as contemptuous or angry in self-referent (‘He’s looking at me’) but not in otherreferent scenarios (‘He’s looking at someone else’) than controls (Buhlmann et al.,
2006, 2004). Other distinct features involve the experience of frequent and more
vivid intrusive body image–related imagery that is also more negatively judged (‘I’m
ugly, disgusting’) compared with controls (Osman et al., 2004).
Cognitive behavioral models of BDD propose that these biases contribute to
appearance-related threat appraisals that drive safety-seeking behaviors (e.g.,
grooming and camouflaging), avoidance, and self-focused attention, which aim to
reduce perceived appearance-related threats but maintain the disorder and are
therefore the target in CBT.
Evidence Base for Cognitive Behavioral Therapy
Outcome research is limited, but an emerging evidence base supports CBT as the
treatment of choice for BDD. A recent meta-analysis by Harrison et al. (2016; N = 7
RCTs) found CBT to be superior to wait-list or credible psychological placebo
control groups in reducing BDD (d = 1.22), depressive symptoms, and insight/
delusionality (d = 0.56), with improvement of BDD likely to be maintained at least
in the short term at 2–4 months follow-up, in line with previous meta-analyses (Ipser
et al., 2009; Williams et al., 2006). No significant predictors of outcome were
identified, and there is no clear empirical support for a particular modality of
CBT. Only one RCT compared CBT with a credible psychological control condition, that is, anxiety management (Veale et al., 2014). Participants were largely
white and Western; hence it is unclear if CBT is acceptable to other ethnic groups.
Long-term outcome data are rare, but gains can be maintained for several years as
shown in a recent naturalistic case series (Veale et al., 2015).
Despite the growing evidence supporting CBT for BDD, there is an urgent
need to improve and validate current treatment protocols. Many clients drop out of
treatment or do not or only minimally benefit from CBT, and many continue to live
with chronic symptoms (Veale et al., 2015). On average, only 40–54% of trial clients
were classed as responders, with many still in the moderate to severe range posttreatment. Thus response rates to CBT for BDD are significantly lower than those
found in CBT trials for OCD (Öst et al., 2015). Many clients find engaging with
treatment too difficult, possibly due to poor insight into their BDD, low mood, or
high risk (Harrison et al., 2016). Furthermore, it remains unclear for whom CBT
works, how best to address comorbidity (Buhlmann et al., 2008), and how to adapt
treatment for severe and complex cases (Sündermann & Veale, 2017). Finally,
current CBT protocols for BDD predominantly focus on anxiety and therefore
may be less efficacious in addressing other emotions such as shame, anger, or disgust
(Cassiello-Robbins & Barlow, 2016; Sündermann et al., 2016).
297
298
Oliver Sündermann and David Veale
Functional and Contextual Approach to BDD
While the phenomenology of BDD is well described in terms of appearance-related
belief content, attentional biases, and the nature of repetitive behaviors, less is
understood about the function of BDD behaviors in relation to a perceived threat
from distorted body imagery. Veale and Gilbert (2014) recently described the
functional and contextual aspects of the phenomenology of BDD as part of
threat-based strategies. Taking an evolutionary angle, the authors argue that all
BDD processes and behaviors serve the function of averting social threat and
keeping the person safe. For example, attentional biases and checking function as
threat detection and monitoring; comparing with others and camouflaging appearance aim to monitor and avoid rejection, shame, and humiliation. Those who suffer
from BDD have often experienced trauma and social threats in the form of shaming,
bullying, humiliation, and rejection (Buhlmann et al., 2012; Weingarden et al.,
2017), and therefore it is understandable that their thinking and coping styles are
rooted and driven by these emotional aversive experiences. Veale and Gilbert
(2014) suggest that CBT can be improved by developing a shared functional analysis
of the functions and contexts of the person’s BDD and linking their body-related
fears to more underlying fears of rejection, exclusion, and loneliness, as well as
aversive relevant memories where appropriate. The authors consequently propose
to complement CBT with compassion-focused interventions, but more research is
needed to evaluate these.
A Cognitive-Behavioral Model: The Self as an Aesthetic Object
Veale and colleagues have developed a cognitive behavioral model of BDD that is
centered on the concept of the self as an aesthetic object, which refers to the
experience of extreme self-consciousness and self-focused attention on distorted
images of their disliked features (Baldock & Veale, 2017; Veale, 2004). The model
proposes that mental imagery in BDD is at least in part constructed from intrusive
imagery based on past aversive experiences that is meaningfully linked with the
person’s BDD, especially shame-based memories (e.g., of being bullied or being
different). The ‘felt impression’ of how the person fears they look is predominantly
visual, experienced from an observer perspective (Osman et al., 2004) and associated with negative appearance-related beliefs (e.g., ‘I am only acceptable if I look
good’) and emotions, most notably, shame. It is proposed that the person becomes
intensely self-focused on all aspects of this triangulated experience of appearancefocused imagery, beliefs, and emotions, which in turn leads the person to engage in
safety-seeking and avoidance behaviors that serve the function of keeping the
person safe through monitoring and averting appearance-related social threat (judgment, rejection) but that paradoxically maintain and often worsen their concerns
through preventing disconfirmation of feared outcomes, increasing dissatisfaction,
preoccupation, and isolation. The model has clinical implications and aims to help
the person reframe their difficulties as a problem of preoccupation with appearance
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
rather than actual appearance, stemming from mental imagery fused with past
aversive memories conditioned to the person’s body image and maintained through
their attempts to cope. Below we outline a CBT protocol for BDD, which is
informed by the model of the self as an aesthetic object.
Assessment and Treatment of Body Dysmorphic Disorder
Overview
The main aim, as with any CBT approach, is to gain a sound and shared understanding of how the person’s BDD developed and what maintains it. In most cases, CBT
will be delivered on an outpatient basis for at least 16–20 weekly sessions, with
sessions typically lasting between 45 and 90 minutes. More severe or complex cases
may require ‘stepping up’ to specialist services. See Table 15.1 for a session-bysession breakdown.
Screening for BDD
People with BDD may not reveal the degree of their preoccupation unless asked
directly because of shame about their appearance, which may also lead them to seek
inappropriate help. Consider the following screening questions to identify appearance concerns:
Some people are very bothered by the way they look. Is that a problem for you?
If the answer is positive, explore further.
What concerns do you have about your appearance? How noticeable do you
think it is? (Is there a difference between their perceived defect and actual or
between actual and their ideal?)
On a typical day, how many hours per day is your appearance at the forefront of
your mind?
Do you think about your appearance a lot and wish you could think less
about it?
Do you have to check your appearance a lot?
How many hours per day do you spend thinking about your appearance?
Is it very distressing for you?
Is there anything you avoid because of the way you feel about your
appearance?
Does it interfere with your ability to study or work?
Does in interfere in dating or your relationship?
Does it interfere in your social life?
Brief validated self-report measures can also be helpful to screen for BDD (see
Table 15.2), for example, the Body Image Disturbance Questionnaire (BIDQ)
(Cash et al., 2004), the Dysmorphic Concern Questionnaire (DCQ) (Mancuso
299
300
Oliver Sündermann and David Veale
Table 15.1. A cognitive behavioral therapy protocol for BDDa
Session
Therapy stage
Key elements and aims
1–3
Assessment and
formulation
4–10
Work phase 1
11–15
Work phase 2
Focus on engagement, be flexible about location (e.g., phone session,
home visit) and allow camouflaging if required.
Clarify hopes, expectations, motivation for treatment, readiness
for change.
Assess disliked feature, related beliefs, and coping behaviors (content,
context, distress, appraisals, emotions).
Provide psychoeducation and normalization of BDD experience
(evolutionary functions, ‘better safe than sorry’).
Assess BDD-related imagery.
Theory A/Theory B: build alternative understanding of BDD as a
problem with preoccupation (Theory B) rather than physical appearance
(Theory A)
Formulation: draw idiosyncratic version of client’s model: identify main
trigger, appraisals, emotions, imagery, and coping responses and
cognitive processes; discuss the function and unintended consequences of
these; and, where possible, link to early experiences (e.g., bullying,
overvaluation of appearance in family).
Involve key significant others (e.g., partner, parents) where appropriate.
Imagery rescripting (if indicated) of relevant early experiences. This is
usually done at the beginning of treatment.
Behavioral experiments and exposures in session, off-site (if feasible)
with the aim to resist/reduce safety-seeking behaviors (SSBs). Conduct
behavioral experiments with or without SSBs to determine the effect on
self-consciousness and frequency of BDD thoughts.
Relate all therapy work to Theory A/Theory B and obtain regular belief
ratings for both theories.
Functional analysis of cognitive processes (e.g., ruminating, comparing,
self-focused attention) through building awareness and labeling of these;
discuss function of these and their unintended consequences and develop
alternatives to them (e.g., focusing outwards, self-compassion).
Mirror retraining (identify motivation for mirror checking, criteria for
termination, practice viewing holistically focusing on whole body from a
distance).
Practice refocusing of attention away from self (e.g., by attending/
listening to environmental cues).
Target beliefs about appearance using CBT techniques (e.g., Big ‘I’,
Little ‘i’, overidentification of self with appearance, etc.).
Address perfectionism if person has unrelenting high standards; identify
criteria for ‘perfect looks’ (often subjective feeling–based, ‘just right’).
Compassion-focused interventions, e.g., safe imagery exercises, mindful
breathing, compassionate letter writing.
Space out sessions to agreed frequency, e.g., biweekly, monthly.
More emphasis on independent planned exposures: ‘Client becoming
their own therapist’.
Generalization of experiments/exposures
Review of treatment
Dealing with setbacks
Blueprint (‘Staying well plan’)
Review of progress
16 and 17 Termination
18–20
a
Follow-up
Protocol should be adapted according to severity, complexity, heterogeneity of BDD, and availability of resources.
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
et al., 2010), or the Cosmetic Procedure Screening Questionnaire (COPS) (Veale
et al., 2012).
Challenges in Engaging Clients in Assessment and Therapy
Clinicians face a number of challenges when working with clients suffering from
BDD, especially in the initial assessment phase. These commonly involve clients
having poor insight into their BDD, and thus they may not be motivated to engage
with a psychological way of working. Often clients are brought or sent for an
assessment by someone else, such as a family member, friend, or cosmetic surgeon.
Those with strong delusional and fixed ideas about their problem being physical in
nature (rather than psychological) may not believe that the interviewer can understand their problem or help in a meaningful way. Compared with mental health
assessments of other disorders, it is pivotal to spend extra time on rapport building,
motivational interviewing to address ambivalence (e.g., about the pros and cons of
treatment or their quality of life with no treatment), as otherwise the client may
disengage. Clients should be allowed to camouflage during the assessment (e.g., to
hide their face), and clinicians should be flexible about where and how to see the
client (e.g., Skype or Zoom sessions if the client feels unable to leave their house).
Be transparent about the diagnosis and provide this with a good psychological
explanation of what the problem is, including the concept of ‘body image’, which
we explain to clients as ‘not just being a photo on the back of your eye, but is
constructed by our brain based on thoughts, feelings, and past aversive experiences’.
The way the person uses mirrors to check their disliked feature also contributes to
distorted body imagery, for example, by using a magnifying glass to check for
pimples, or close-up examinations. Some professionals, especially when inexperienced with BDD or those with negative beliefs about the condition, can be dismissive of the client’s concerns (e.g., ‘You look fine’) and of course this is likely to
aggravate the client’s distress. We strongly recommend not to argue or debate with
clients about their disliked feature, overvalued ideas of appearance, or their diagnosis but instead maintain an ‘inquiring’ stance and focus on validating feelings of
shame and distress. In the case of slight visible differences (e.g., mild acne), it can be
helpful to acknowledge that their concern is minimally noticeable and the interviewer may emphasize that they can see what the client is describing only when
‘taking a very close up look and raising my aesthetic standards’.
Assessment of Disliked Feature(s), Appearance-Related Beliefs, and Coping
A good starting point is to ask the client to describe the features of their body they
dislike or would like to improve. Ask specifically whether the client is motivated to
seek a medical procedure such as dermatological treatment or cosmetic surgery. For
example, if the concern is regarding the size or shape of the nose, ask whether the
client is motivated to have rhinoplasty or has undergone surgery in the past. In case
the client is preoccupied with several areas of their body, it is helpful to ask the
301
302
Oliver Sündermann and David Veale
Box 1 Common safety-seeking behaviors in people with BDD
Mirror gazing or avoiding mirrors
Checking perceived defect, inspecting, touching or measuring it
Comparing self with others or to old photographs
Grooming behaviors, e.g., combing, smoothening, straightening, plucking, or
cutting hair
Camouflaging rituals (e.g., make-up, scarves, hats, oversized clothes)
Skin cleaning, picking, face peeling, scrubbing, or bleaching
Trying to convince others that defect exists
Facial exercises, e.g., practicing showing the ‘best face’
Monitoring other people’s reactions and eye movement
Controlling body posture to avoid showing ‘defect’
client to draw out a pie chart adding up the percentages of concern for each feature.
Ask to estimate how much time they spend thinking on average about each feature
during the day, how much distress their preoccupation causes them, and how much
they are impaired in the various domains of functioning such as relationships,
friendships, and work. If the client is not in a romantic relationship, assess the
extent their condition has on potential future relationships, for example, would they
avoid meeting someone new because of their appearance-related distress, or could
not enjoy sexual activity because of their BDD. Elicit a detailed list of activities and
situations the client avoids because of their preoccupation, and discuss all
appearance-related coping behaviors, their function, and unintended consequences.
Box 1 contains a list of typical safety-seeking behaviors. Self-report measures and
observer-rated scales such as the BDD-YBOCS are recommended to complement
the assessment (see Table 15.3 below).
The therapist should also probe for typical coping behaviors if these are not
volunteered (e.g., mirror checking) and normalize accordingly, that is, as an understandable response to appearance distress. A helpful way of capturing a sense of the
client’s felt impression is by asking the client to draw a self-portrait of how they think
their perceived defect appears to others. The emphasis is explicitly not on artistic
skills, but instead the client is instructed to draw a portrait from the picture in their
mind or impression that they have of their disliked feature. Pre- and post-treatment
self-portraits of successful CBT for BDD can impressively visualize the change in the
person’s experience of their body image (see Sündermann et al., 2016).
Engagement: Theory A/Theory B
The main purpose of the initial treatment stage is to socialize and engage the client
into a psychological way of thinking about their problem. ‘Theory A/Theory B’ (see
Table 15.2) is a helpful technique to introduce a psychological approach to BDD
and proposes to the client two fundamentally different accounts of their difficulties.
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
303
Table 15.2. Worksheet for Theory A/Theory B with example of client suffering from a preoccupation
with balding hair
Theory A (appearance problem)
‘My problem is that I am balding and that my
hairline is receding, which makes me look ugly and
inferior to other men, and means that I’ll never be
able to have a romantic relationship.’
Theory B (preoccupation problem)
‘My problem is that I am preoccupied with hair loss,
which makes me excessively self-conscious and
constantly worry that I am inferior.’
What do I need to do if Theory A is true?
Camouflage, particularly through using my hat.
Avoid all situations where I need to take off my hat.
Avoid bright lights, and spend more time in low
lights.
Compare myself with others who have ‘better’ hair.
Worry ‘what if’’ a hair transplant does not work.
Focus attention on my hair and monitor people’s
response to it.
Drink alcohol to avoid thinking about my baldness.
What do I need to do if Theory B is true?
Live my life in accordance with my values, in
particular:
If this is true, what does it say about the future?
The future would be dark and grim, and I will be
lonely forever.
If this is true, what does it say about the future?
I would feel optimistic and hopeful. I would be
able to have relationships, work, and live a
meaningful life.
Get a job.
Pursue a relationship.
Socialize with friends.
Spend time outside in the sun.
Do all of these activities without engaging in safetyseeking behaviors.
Theory A encompasses their beliefs about BDD and states that their problem is an
actual appearance problem (e.g., ‘My problem is that my nose is deformed, which
makes me look disgusting’). As such, Theory A is the theory according to which
they had been living their life, and which suggests to ‘solve’ the problem accordingly, for example, camouflage, avoid, seek cosmetic surgery. The therapist gently
suggests that these ‘solutions’ have become their actual problem, because they keep
them stuck and maintain a poor quality of life. Hence, if the client continued to live
their life according to Theory A, the future is very grim. Subsequently, the therapist
explains Theory B, an alternative account of their difficulties stating that the
problem is one of preoccupation with appearance rather than an actual appearance
flaw (e.g., ‘My problem is that I am preoccupied with my nose not being “right”, and
that I worry others will not like me’), suggesting that the preoccupation with
appearance may stem from an emotional issue (i.e., BDD). The therapist then
discusses what the client needs to do if their problem were indeed one of preoccupation. This usually elicits responses of doing the ‘opposite’ to Theory A behaviors,
and acting and living by their values. Of course, Theory B has more benign and
hopeful implications for the client’s life and future. The clinician might also ask
whether they noticed that solving their difficulties as an appearance problem actually increases their distress and preoccupation. Clients may be quick to express
doubts about Theory B or may not be willing to engage with the idea of an
alternative theory of their problem, particularly when appearance-related beliefs
are fixed. The clinician should normalize any doubts as understandable responses
arising from their BDD, while inviting the client to test out whether Theory B better
304
Oliver Sündermann and David Veale
fits their experiences. The therapist may also propose to the client that they can
always go back to Theory A if therapy does not work for them, while stressing that
only by acting in reverse to Theory A can they truly test out and experience whether
their difficulties fit better with the idea of a preoccupation problem. Once both
theories are drawn out, belief ratings should be obtained. Theory A/Theory
B should serve as a framework for discussing all therapy-related tasks and the
resulting client’s experience in subsequent sessions.
Cognitive Behavioral Treatment of BDD
Overview
CBT for BDD helps the client to build up evidence for Theory B through testing out
fears and expectations without avoidance and safety-seeking behaviors. BDD
behaviors that are performed in private (e.g., mirror checking, comparing) are
usually targeted first. Early goals in therapy usually involve reducing self-focused
attention and linking BDD behaviors with heightened preoccupation. If indicated,
relevant early adverse experiences (e.g., appearance-related bullying) should be
processed as soon as possible. We also recommend building up self-compassion
and self-soothing skills. Similar to other CBT approaches, we emphasize therapy as
a ‘doing’ treatment, with the client taking responsibility for their recovery through
increasing independence in conducting experiments as treatment progresses.
Treatment outcome should be monitored using BDD-specific self-report measures
(e.g., Appearance Anxiety Inventory [AAI]; Veale et al., 2014).
Formulation and Treatment
To illustrate our treatment protocol, we present the case of Tom, who attended
outpatient sessions with one of the authors.
Case vignette: Tom. Tom was a 20-year-old student who suffered from severe
BDD characterized by a preoccupation with height (‘being too short’), and his nose
(‘too large’). He had received 24 therapy sessions, some of them remotely via Skype.
His main preoccupation was about his height (50 700 ), which he felt made him
unattractive, inferior, and less of a man. He was also preoccupied with his nose
being too large in proportion to his face, and he worried that it would project too
much especially on the side profile. His appearance distress severely affected his
quality of life, interfered with romantic relationships, and impaired his
academic performance.
He was constantly triggered by his own intrusive thoughts of not being tall
enough, catching a glimpse of himself in reflective surfaces (e.g., cycling past cars),
and seeing taller men. When triggered he experienced a felt impression of a
shrunken, dwarf-like man worthy of ridicule and pity from women. Tom engaged
in numerous avoidance and safety-seeking behaviors, such as wearing insoles to
increase his height, avoiding bright lights, monitoring his body posture and
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
straightening his back to appear taller, binge drinking on social occasions, researching risky leg-lengthening surgical procedures, and performing mental surgery
imagining a positive outcome of the procedure. He also relentlessly compared
himself unfavourably to taller men and sought constant reassurance from his mother
while simultaneously trying to convince her of the severity of his physical flaws. Tom
constantly checked the shape of his nose with his finger, and recorded daily videos
of himself, which he stored to ‘keep track of his progress’.
From a young age Tom had unrelenting high and perfectionistic academic
standards and always saw good grades as his main compensatory ‘asset’ (‘I’m short,
but at least I’m smart’). His preoccupation ignited in early adolescence following a
number of comments from his aunt, who compared Tom unfavourably with his
‘attractive’ brother. Various comments made by women about not wanting to be
with ‘short’ men as well as some dating profiles stating ‘I would not date a man
shorter than me’ seemed solid proof of his overvalued ideas about the importance of
appearance, especially height. His BDD deteriorated when he felt rejected by a
woman, after which he sought professional help.
CBT Formulation for BDD
Case formulation for BDD should comprise a developmental part, linking the
client’s BDD to past adverse experiences, and a cross-sectional part, showing how
the felt impression, related appraisals, and emotions drive coping behaviors that
maintain the current preoccupation. The case formulation presented here is derived
from Veale’s model of the self as an aesthetic object (Veale, 2004), and is shown in
Figure 15.1. We have adapted the diagram from Baldock and Veale (2017) by
including past-present links between relevant shame-based experiences and the
client’s current felt impression. Each coping behavior is noted in one of the petals
of the vicious flower, and the therapist should discuss the intended function of each
behavior and the related unintended consequences. For example, Tom was constantly comparing himself with taller men, which served the function of monitoring
threat and establishing his place in the social hierarchy, but it also increased his
preoccupation, self-consciousness, and distress. Researching leg lengthening surgeries gave him momentary glimmers of hope but was quickly overridden with despair
when realizing that he could not afford it, and also triggered guilt for considering
‘wasting money’. Self-recorded videos were driven by the hope of looking better
than he thought, but usually made him feel worse about his appearance. Excessive
alcohol consumption allowed him to socialize but included the cost of memory loss,
triggering prolonged mental reviewing and self-doubt. Although Eric initially held
firm beliefs about Theory A, he found several aspects of his formulation allowed
him to think more flexibly about his BDD. First, he agreed that his coping strategies
increased his preoccupation and as such were ‘the problem, instead of the solution’.
Understanding his BDD developmentally as a plausible response to feeling body
shame when he was bullied and received comments critical of appearance (e.g., a
teacher called him ‘tiny’) made him doubt that he would have developed BDD
without such experiences (suggesting his difficulties were emotional rather than
305
306
Oliver Sündermann and David Veale
Relevant early experiences
• 6 years: Overvaluation of appearance in family, compared unfavorably with brother
• 13–15 years: Appearance-related bullying in school (‘you are tiny’, ‘short’)
• 17–20 years: Overheard derogatory comments by women about male height
Appearance-related beliefs
‘Only tall men are sexually attractive and will find a partner’
‘I am short and laughable, and destined to be alone and ridiculed’
‘Appearance is everything’
Avoidance
Staying indoors, avoiding social
situations: binge drinking when out
to numb/avoid feelings of shame
Trigger
Social situations
Taller men,
Own reflections
Self-focused attention
Constant and intense self-focus
on felt impression
Unintended consequences:
Relying on internal information
on how I feel I look; feeling selfconscious and can’t engage with
others
Unintended cosequences:
Feeling alienated and lonely
Can’t disconfirm that people don’t
joke about me
Attempts to correct
Researching and fantasizing about
leg surgery and rhinoplasty
Unintended consequences
Heightened preoccupation,
increased despair and
hopelessness
Meaning and past
associations
I am less of a man;
laughable, powerless,
and disgusting
Image/felt
impression
Impression of self
as a dwarf, with a
crooked, oversized
nose
Cover/hide features
Wearing insoles to gain
height; carefully adjust
posture to avoid showing
profile
Unintended consequences
More self-conscious about
my height; feel link a
‘fraud’, worry about ‘being
found out’
Figure 15.1. Case formulation.
Emotions
Shame, sadness,
anger (selfdirected for not
being taller)
Comparing/rumination
Relentlessly comparing
unfavorably will other men;
constant rumination why I was
born short
Unintended consequences
Fuels my preoccupation, makes
me feel hopeless, inferior, and
depressed
Checking
Check by touch (feeling
nose); video-recording of
self, uploading and storing
it, and mirror checks driven
by hope to look better than
felt impression
Unintended consequences
Feeling more hopeless and
preoccupied
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
physical). The formulation should be reviewed regularly and adapted as new information emerges in therapy. For example, in Tom’s case we did not learn until
session 10 that high academic achievements served as a powerful way of dealing
with feeling ugly and inferior (‘at least I have good grades’). When his BDD
interfered with university performance, this aggravated his vicious cycle of selfdepreciating hostile thoughts, preoccupation, and hopelessness.
Cognitive Behavioral Work: Addressing Beliefs about Appearance
Based on the formulation, therapist and client agree on a hierarchy of avoided
situations, coping behaviors, and processes to target (e.g., comparing, rumination,
self-focused attention), always with a view of testing out Theory B and building a
more helpful perspective of the client’s distress. Similar to CBT for OCD, behavioral tasks should be designed according to inhibitory learning principles (Craske
et al., 2014), that is, maximizing violation of expectancies, repetition, generalizing to
different contexts, and affect labelling. Beliefs about being judged for one’s appearance are often not testable in a credible way (‘Others are too nice to tell me how
ugly I am’) and, in fact, can be counterproductive; for example, the client asking
others directly what they think about their looks is likely to backfire because it may
evoke anger for feeling dismissed or not understood. Therefore, predictions of
experiments tend to focus more on tolerance of distress and building up confidence
in social situations. For example, Tom predicted that by going out without wearing
his insoles, his friends (and also strangers) would pity or tease him for being short;
however, he believed that while they would surely judge him, they would not reveal
their true judgements. However, he agreed to test out whether he would be able to
tolerate and ‘survive’ without them. To his surprise, he was able to attend a party
without his insoles and – importantly – without drinking alcohol. He was convinced
that he was negatively evaluated, but as the evening progressed he became less selfconscious and learned that he could be more confident than he thought. Clients can
be judicious with their use of safety-seeking behaviors in the early stages of treatment. For example, while Tom was able to remove his insoles, he initially could do
so only with his ‘safe’ shoes, namely, his boots with thicker soles.
Clients should be encouraged to always reflect on their learning and stimulate
alternative explanations (that usually fit better with Theory B). Even though Tom’s
strong beliefs about the importance of height were initially unshakable, he was able
to reflect that his fluctuating levels of preoccupation fitted better with Theory B,
suggesting his problems were emotional in nature.
Mirror Retraining
Mirror retraining is a standard component of CBT for BDD, and has the purpose of
helping the client to develop a healthier and more functional relationship with
mirrors and reflective surfaces. First, therapist and client should discuss and understand the motivation for mirror checks. Mirror gazing is often driven by the hope of
not looking as bad as the client fears, coupled with a belief that they would feel
307
308
Oliver Sündermann and David Veale
worse if they resisted (Veale & Riley, 2001). Common BDD behaviors often
performed in front of the mirror involve skin picking, cleaning or removing perceived blemishes, facial exercises, or make-up and camouflaging rituals. Many
patients carry pocket mirrors with them or use magnifying mirrors, and should be
encouraged to desist. The actual steps of mirror retraining involve cutting down on
frequency and duration of mirror usage and addressing the way mirrors are used.
Instead of zooming in on perceived flaws, which magnifies the felt impression,
clients are encouraged to step back, using larger mirrors to view themselves briefly
and holistically. They are encouraged to scan their whole body from a slight distance
while purposefully focusing outwards on external reflections, rather than internal
feelings. A helpful experiment can be for the client to compare their level of
preoccupation when using the mirror in a dysfunctional way and in a healthy way.
Results can be used to strengthen conviction in Theory B. Patients often dissociate
or ‘get stuck’ in front of the mirror and lose sense of time. Sometimes skin-picking or
other mirror-based rituals serve additional functions, such as purposefully bringing
on dissociative states to tolerate difficult memories or to avoid difficult feelings.
Imagery Rescripting
Imagery rescripting (ImRs) is indicated when the person’s sense of threat is associated with aversive memories. ImRs aims to change the meaning of past childhood
events that continue to shape the person’s experience (Arntz, 2011) through processing and re-evaluating them. The felt impression of a person with BDD is often
fused with imagery arising from shame-based memories of being teased, bullied, or
humiliated. This is also reflected in clients’ reporting that, when triggered, they feel
as young as when they were shamed as a child about their appearance. It is not
uncommon for clients to become less avoidant of social situations yet continue to
feel ugly. Old feelings of shame become easily triggered despite knowing that others
may not judge them. Clients who continue to be affected by ‘ghosts from the past’
should receive ImRs as early as possible in treatment. The protocol we use is briefly
outlined in Box 2, and follows the steps originally described by Arntz and colleagues
(Arntz & Weertman, 1999) and adapted by Willson et al. (2016) for BDD.
Unlike trauma reliving, it is not necessary to go over moments of peak distress.
Also, the therapist can ask the client to pause before the scene changes to its worst
point. The rationale of ImRs should be discussed beforehand, using metaphors such
Box 2 Imagery Rescripting for BDD
Stage 1: The client relives the image from the child’s perspective, and
describes their needs that were not met at the time.
Stage 2: The client enters the scene as an adult to provide the child’s needs and
provide a different perspective
Stage 3: The client returns to the image as a child with the adult self present at
the scene to determine if all needs have been met.
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
as ‘mental time travel’ to gain a different view on their experience (rather than
changing what happened). Metaphors from trauma-focused CBT can be used to
explain the aim of processing memories to contextualize them (Stott et al., 2010).
ImRs proved useful in Tom’s treatment. He rescripted several key events linked
with the emotional origins of his BDD. For example, Tom’s aunt used to compare
his appearance as a six-year-old child unfavourably to his older brother, which was
deeply shaming. The body shame experienced back then conditioned his presentday body image. Through rescripting, Tom was able to ‘stand up to my aunt, tell her
off and make my younger self feel safe’, thus successfully processing old feelings of
shame. The benefits were clearly noticeable. Tom was less frequently triggered, and
when he was, he felt older, was less focused on his internal impressions, and was
more able to tolerate distress. ImRs should be conducted alongside stimulus discrimination to further contextualize past experiences. This involves focusing on as
many details as possible that discriminate the past from the here and now. Tom
learned to be more mindful when triggered and was able to compassionately remind
himself that the present is very different from the past and that he was safe.
Self-Soothing and Compassionate Therapy Interventions
Clients with BDD commonly suffer from self-deprecating thoughts and rumination,
with the emotional tone of their inner voice being hostile and threat-based (‘It’s
your own fault that you are ugly’). Compassion-focused interventions are therefore
recommended to complement CBT for BDD to help de-shame the client’s experience (Veale & Gilbert, 2014). A key compassion intervention is psychoeducation
about how our brains have evolved and how they are designed to keep us safe
(‘better safe than sorry’). The therapist can explain how our old brain and our new
brain can get caught in tricky loops, with the old brain alerting to social threat, and
the new brain ruminating in an effort to solve the ‘appearance problem’. The
therapist highlights that the way brains process threat is a design problem, and
therefore not the client’s fault (albeit the client is responsible for change). The
therapist should not label the client’s thinking style as ‘distorted’ or ‘biased’ because
clients may perceive it as blaming (‘It’s my fault that I am thinking wrongly’).
Instead, the therapist may say, ‘It is very understandable that you became preoccupied with height when you were teased in school, and the impression has stayed with
you.’ Hence, the emphasis is on responsibility for change, which includes empathic
understanding of how their problems developed and are maintained, as well as the
importance of testing out an alternative view. Many clients find this evolutionary
explanation de-shaming and motivating. Furthermore, practical exercises are recommended to build skills in self-compassion and self-soothing. This includes safe
imagery exercises, compassionate breathing, and compassionate letter writing.
BDD Measures for Screening, Diagnosing, and Outcome
A range of BDD assessment measures is available for screening purposes, aiding
diagnosis, and monitoring treatment outcome. Table 15.3 gives an overview of a
309
310
Downloaded from https://www.cambridge.org/core, on subject to the Cambridge Core terms of use, available at https://www.cambridge.org/core/terms.
https://doi.org/10.1017/9781108355605.018
Table 15.3. Assessment measures for BDD
Type of
measure
Function of
measure
Body Dysmorphic Disorder
Questionnaire (BDDQ)
Self-report
Screens for BDD
Body Image Disturbance
Questionnaire (BIDQ)
Self-report
Screens for BDD
dimensional
Cosmetic Procedure Screening
(COPS)
Self-report
Screens for BDD
severity
Dysmorphic Concern
Questionnaire (DCQ)
Self-report
Screens for BDD
Structured Clinical Interview for
DSM-5 (SCID-5), BDD module
Body Dysmorphic Disorder
Diagnostic Module
Brown Assessment of Beliefs
Scale (BABS)
Semi-structured
Clinician-rated
Semi-structured
Clinician-rated
Semi-structured
Clinician-rated
Diagnoses BDD
Yale–Brown Obsessive
Compulsive Scale modified for
BDD (BDD-YBOCS)
Semi-structured
Clinician-rated
Severity of BDD
Appearance Anxiety Inventory
(AAI)
Self-report
Body Image Quality of Life
Inventory (BIQL)
Self-report
Assesses BDDprocesses and
behaviors
Monitoring
progress
Quality of life
Name of measure
Screening
Diagnostic
Outcome /
severity
Diagnoses BDD
Insight into BDD
Description
Reference
One to four items of yes/no questions mapping
DSM criteria for BDD; maximum score of
4 suggests likely diagnosis of BDD
Seven items derived from BDDQ; five-point Likert
scale, with higher scores indicating greater body
image disturbance
Nine items scored from 0 to 8, with high scores
indicating severity of BDD. Scores above 40 reflect
a likely diagnosis of BDD
Seven items assessing cognitive and behavioral
symptoms of BDD, sum scores ranging from 0 to
21, with scores above 9 indicating possible BDD
Maps onto the DSM-5 criteria for BDD; also allows
diagnosis of muscle dysmorphia
Five-item measure, which also allows diagnosis of
muscle dysmorphia
Seven-item scale measuring the strength of conviction
in a belief. Items are rated from 0 to 4, with scores of
18 or above indicating delusional BDD
Twelve-item scale, ranging from 0 to 48, with higher
scores indicating increased severity of BDD; widely
seen as the ‘gold standard’ outcome measure for
BDD
Ten-item questionnaire ranging from 0 to 4,
measures frequency of avoidance behavior and
threat-monitoring that are characteristic of a
response to a distorted body image; ideal for
tracking progress
Nineteen items measuring impact of body image
concerns on various life domains, using a sevenpoint scale (3 to +3) ranging from negative to
positive, with lower scores reflecting a more
negative impact.
K. A. Phillips
(1996)
Cash et al.
(2004)
Veale et al.
(2012)
Mancuso
et al. (2010)
First et al.
2002
K. A. Phillips
(2005)
Eisen et al.
(1998)
K. A. Phillips
et al., 2014
Veale et al.,
2014
Cash &
Fleming, 2002
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
selection of measures we use in our services. Screening measures can help in
identifying BDD and prompting further BDD-specific assessments. The diagnostic
process should include the use of validated clinician-rated semi-structured interviews such as the Structured Clinical Interview for DSM-5 (SCID-5) for BDD. The
Brown Assessment of Beliefs Scale (BABS) is commonly used to gauge the level of
insight a person has into their BDD. The Yale–Brown Obsessive Compulsive Scale
(Y-BOCS) modified for BDD remains the ‘gold standard’ outcome measure and is
typically used pre- and post-therapy to ascertain severity of BDD. Specialist clinics
are likely to use the Y-BOCS modified for BDD, as it is also required by many
funding panels to determine eligibility for accessing higher-level care services. We
strongly advocate using BDD-specific measures to monitor treatment progress. For
example, the Appearance Anxiety Inventory (AAI) provides a brief 10-item measure of the most common BDD avoidance and threat monitoring processes and
behaviors that are characteristic of a response to a distorted body image. Thus,
the AAI provides a helpful process measure that enables effective monitoring of
treatment progress, and should be used in conjunction with more generic mood,
anxiety, quality of life, and function measures. We strongly recommend incorporating these actively into treatment and jointly reviewing them with the client.
Conclusion
CBT is the treatment of choice for BDD. This chapter has outlined the key
principles of effective delivery of CBT for BDD. Key principles include formulating
BDD as a body image problem that has developed in response to shaming experiences, is maintained through avoidance and threat, is further maintained through
monitoring behaviors that aim to protect the person from rejection and judgement,
but have the unintended consequences of increasing preoccupation.
Conceptualizing BDD within a contextual and evolutionary framework can be
normalizing, engaging, and de-shaming. Compassion-focused interventions can
complement CBT and give the client courage to test out an alternative understanding of their problem (Theory B). Additionally, they can reduce the inner critique
and aid development of competency in relating more compassionately to self and
others. ImRs is indicated when past aversive memories continue to shape the
person’s experiences.
Key Learning Points
BDD is a common but under-recognized and under-reported problem.
The treatment of choice for BDD is CBT, but only 50% make a
satisfactory recovery.
Engagement into treatment can be challenging, particularly when insight
is low.
Theory A/Theory B provides a helpful framework to socialize clients
into therapy.
311
312
Oliver Sündermann and David Veale
(cont.)
Treatment aims to build a more helpful perspective of appearance distress
(Theory B) through exposure tasks and behavioral experiments.
Understanding functional and contextual aspects of BDD may enhance
engagement and treatment outcome.
Imagery rescripting and compassion-focused techniques can help to process
related past aversive experiences.
RECOMMENDED READING
Phillips, K. A. (2005). The broken mirror: Understanding and treating body dysmorphic
disorder. Oxford University Press.
(2017). Body dysmorphic disorder: Advances in research and clinical practice. Oxford
University Press.
Sündermann, O., & Veale, D. (2017). Complexity in obsessive-compulsive and body dysmorphic disorder: A functional approach to complex difficulties. The Cognitive
Behaviour Therapist, 10.
Veale, D., & Gilbert, P. (2014). Body dysmorphic disorder: The functional and evolutionary
context in phenomenology and a compassionate mind. Journal of Obsessive-Compulsive
and Related Disorders, 3(2), 150–160.
Veale, D., Gilbert, P., Wheatley, J., & Naismith, I. (2014). A new therapeutic community:
Development of a compassion-focussed and contextual behavioural environment.
Clinical Psychology and Psychotherapy, 22(4), 285–303.
Veale, D., & Neziroglu, F. (2010). Body dysmorphic disorder: A treatment manual. John
Wiley and Sons.
Veale, D., Willson, R., & Clark, A. (2009). Overcoming body image problems including body
dysmorphic disorder. Constable Robinson.
Wilhelm, S., Phillips, K. A., & Steketee, G. (2012). Cognitive-behavioral therapy for body
dysmorphic disorder: A treatment manual. Guilford Press.
REFERENCES
Angelakis, I., Gooding, P. A., & Panagioti, M. (2016). Suicidality in body dysmorphic
disorder (BDD): A systematic review with meta-analysis. Clinical Psychology Review,
49, 55–66.
Arntz, A. (2011). Imagery rescripting for personality disorders. Cognitive and Behavioral
Practice, 18(4), 466–481.
Arntz, A., & Weertman, A. (1999). Treatment of childhood memories: Theory and practice.
Behaviour Research and Therapy, 37(8), 715–740. doi:10.1016/S0005-7967(98)00173-9
Baldock, E., & Veale, D. (2017). The self as an aesthetic object: Body image, beliefs about
the self, and shame in a cognitive-behavioural model of body dysmorphic disorder. In
Body dysmorphic disorder: Advances in research and clinical practice. Oxford University
Press.
Bjornsson, A. S., Didie, E. R., Grant, J. E., Menard, W., Stalker, E., & Phillips, K. A. (2013).
Age at onset and clinical correlates in body dysmorphic disorder. Comprehensive
Psychiatry, 54(7), 893–903.
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
Bohne, A., Keuthen, N. J., Wilhelm, S., Deckersbach, T., & Jenike, M. A. (2002). Prevalence
of symptoms of body dysmorphic disorder and its correlates: A cross-cultural comparison. Psychosomatics, 43(6), 486–490.
Buhlmann, U., Etcoff, N. L., & Wilhelm, S. (2006). Emotion recognition bias for contempt
and anger in body dysmorphic disorder. Journal of Psychiatric Research, 40(2), 105–111.
Buhlmann, U., Marques, L. M., & Wilhelm, S. (2012). Traumatic experiences in individuals with
body dysmorphic disorder. The Journal of Nervous and Mental Disease, 200(1), 95–98.
Buhlmann, U., McNally, R. J., Etcoff, N. L., Tuschen-Caffier, B., & Wilhelm, S. (2004).
Emotion recognition deficits in body dysmorphic disorder. Journal of Psychiatric
Research, 38(2), 201–206.
Buhlmann, U., McNally, R. J., Wilhelm, S., & Florin, I. (2002). Selective processing of emotional
information in body dysmorphic disorder. Journal of Anxiety Disorders, 16(3), 289–298.
Buhlmann, U., Reese, H. E., Renaud, S., & Wilhelm, S. (2008). Clinical considerations for the
treatment of body dysmorphic disorder with cognitive-behavioral therapy. Body Image, 5(1),
39–49.
Cansever, A., Uzun, Ö., Dönmez, E., & Özşahin, A. (2003). The prevalence and clinical
features of body dysmorphic disorder in college students: A study in a Turkish sample.
Comprehensive Psychiatry, 44(1), 60–64.
Cash, T. F., & Fleming, E. C. (2002). The impact of body image experiences: Development of
the Body Image Quality of Life Inventory. International Journal of Eating Disorders,
31(4), 455–460.
Cash, T. F., Phillips, K. A., Santos, M. T., & Hrabosky, J. I. (2004). Measuring ‘negative body
image’: Validation of the Body Image Disturbance Questionnaire in a nonclinical population. Body Image, 1(4), 363–372.
Cassiello-Robbins, C., & Barlow, D. H. (2016). Anger: The unrecognized emotion in emotional disorders. Clinical Psychology: Science and Practice, 23(1), 66–85. doi:10.1111/
cpsp.12139
Craske, M. G., Treanor, M., Conway, C. C., Zbozinek, T., & Vervliet, B. (2014). Maximizing
exposure therapy: An inhibitory learning approach. Behaviour Research and Therapy,
58, 10–23.
Deckersbach, T., Savage, C. R., Phillips, K. A., Wilhelm, S., Buhlmann, U., Rauch, S. L., . . . &
Jenike, M. A. (2000). Characteristics of memory dysfunction in body dysmorphic disorder. Journal of the International Neuropsychological Society, 6(6), 673–681.
Didie, E. R., Loerke, E. H., Howes, S. E., & Phillips, K. A. (2012). Severity of interpersonal
problems in individuals with body dysmorphic disorder. Journal of Personality Disorders,
26(3), 345.
Eisen, J. L., Phillips, K. A., Baer, L., Beer, D. A., Atala, K. D., & Rasmussen, S. A. (1998).
The Brown Assessment of Beliefs Scale: Reliability and validity. American Journal of
Psychiatry, 155(1), 102–108.
First, M. B., Spitzer, R. L., Gibbon, M., & Williams, J. B. (2002). Structured clinical interview
for DSM-IV-TR axis I disorders, research version, patient edition. SCID-I/P.
Fontenelle, L. F., Telles, L. L., Nazar, B. P., De Menezes, G. B., Do Nascimento, A. L.,
Mendlowicz, M. V., & Versiani, M. (2006). A sociodemographic, phenomenological, and
long-term follow-up study of patients with body dysmorphic disorder in Brazil. The
International Journal of Psychiatry in Medicine, 36(2), 243–259.
Greenberg, J. L., Reuman, L., Hartmann, A. S., Kasarskis, I., & Wilhelm, S. (2014). Visual
hot spots: An eye tracking study of attention bias in body dysmorphic disorder. Journal
of Psychiatric Research, 57, 125–132.
Harrison, A., de la Cruz, L. F., Enander, J., Radua, J., & Mataix-Cols, D. (2016). Cognitivebehavioral therapy for body dysmorphic disorder: A systematic review and meta-analysis
of randomized controlled trials. Clinical Psychology Review, 48, 43–51.
313
314
Oliver Sündermann and David Veale
Ipser, J. C., Sander, C., & Stein, D. J. (2009). Pharmacotherapy and psychotherapy for body
dysmorphic disorder. Cochrane Database of Systematic Reviews, (1).
Lambrou, C., Veale, D., & Wilson, G. (2011). The role of aesthetic sensitivity in body
dysmorphic disorder. Journal of Abnormal Psychology, 120(2), 443.
(2012). Appearance concerns comparisons among persons with body dysmorphic disorder
and nonclinical controls with and without aesthetic training. Body Image, 9(1), 86–92.
Mancuso, S. G., Knoesen, N. P., & Castle, D. J. (2010). The Dysmorphic Concern
Questionnaire: A screening measure for body dysmorphic disorder. Australian and
New Zealand Journal of Psychiatry, 44(6), 535–542.
Marques, L., LeBlanc, N., Weingarden, H., Greenberg, J. L., Traeger, L. N., Keshaviah, A., &
Wilhelm, S. (2011). Body dysmorphic symptoms: Phenomenology and ethnicity. Body
Image, 8(2), 163–167. doi:10.1016/j.bodyim.2010.12.006
Osman, S., Cooper, M., Hackmann, A., & Veale, D. (2004). Spontaneously occurring images
and early memories in people with body dysmorphic disorder. Memory, 12(4), 428–436.
Öst, L.-G., Havnen, A., Hansen, B., & Kvale, G. (2015). Cognitive behavioral treatments of
obsessive-compulsive disorder. A systematic review and meta-analysis of studies published 1993–2014. Clinical Psychology Review, 40, 156–169.
Phillips, K. (2017). Body dysmorphic disorder: Advances in research and clinical practice.
Oxford University Press.
Phillips, K., & Menard, W. (2006). Suicidality in body dysmorphic disorder: A prospective
study. American Journal of Psychiatry, 163(7), 1280–1282.
Phillips, K. A. (2005). The broken mirror: Understanding and treating body dysmorphic
disorder. Oxford University Press.
(1996). Instruments for assessing BDD: The BDDQ: A self-report screening instrument for
BDD. The Broken Mirror, 321–333.
Phillips, K. A., Didie, E. R., Menard, W., Pagano, M. E., Fay, C., & Weisberg, R. B. (2006).
Clinical features of body dysmorphic disorder in adolescents and adults. Psychiatry
Research, 141(3), 305–314.
Phillips, K. A., Hart, A. S., & Menard, W. (2014). Psychometric evaluation of the Yale–
Brown Obsessive-Compulsive Scale Modified for Body Dysmorphic Disorder (BDDYBOCS). Journal of Obsessive-Compulsive and Related Disorders, 3(3), 205–208.
Stott, R., Mansell, W., Salkovskis, P., Lavender, A., & Cartwright-Hatton, S. (2010). Oxford
guide to metaphors in CBT: Building cognitive bridges. Oxford University Press.
Sündermann, O., & Veale, D. (2017). Complexity in obsessive-compulsive and body dysmorphic disorder: A functional approach to complex difficulties. The Cognitive
Behaviour Therapist, 10.
Sündermann, O., Wheatley, J., & Veale, D. (2016). ‘If you have good skin, you are god. If you
have bad skin, you are a piece of rubbish.’ Mastery of shame and anger in treatmentresistant body dysmorphic disorder: A single case study. The Cognitive Behaviour
Therapist, 9.
Veale, D. (2004). Advances in a cognitive behavioural model of body dysmorphic disorder.
Body Image, 1(1), 113–125. doi:10.1016/S1740-1445(03)00009-3
Veale, D., Ellison, N., Werner, T. G., Dodhia, R., Serfaty, M. A., & Clarke, A. (2012).
Development of a cosmetic procedure screening questionnaire (COPS) for body dysmorphic disorder. Journal of Plastic, Reconstructive and Aesthetic Surgery, 65(4), 530–532.
Veale, D., Eshkevari, E., Kanakam, N., Ellison, N., Costa, A., & Werner, T. (2014). The
Appearance Anxiety Inventory: Validation of a process measure in the treatment of
body dysmorphic disorder. Behavioural and Cognitive Psychotherapy, 42(5), 605–616.
Veale, D., & Gilbert, P. (2014). Body dysmorphic disorder: The functional and evolutionary
context in phenomenology and a compassionate mind. Journal of Obsessive-Compulsive
and Related Disorders, 3(2), 150–160.
A Cognitive Behavioral Approach to Body Dysmorphic Disorder
Veale, D., Gilbert, P., Wheatley, J., & Naismith, I. (2014). A new therapeutic community:
Development of a compassion-focussed and contextual behavioural environment.
Clinical Psychology and Psychotherapy, 22(4), 285–303.
Veale, D., Gledhill, L. J., Christodoulou, P., & Hodsoll, J. (2016). Body dysmorphic disorder
in different settings: A systematic review and estimated weighted prevalence. Body
Image, 18, 168–186.
Veale, D., & Matsunaga, H. (2014). Body dysmorphic disorder and olfactory reference
disorder: Proposals for ICD-11. Revista Brasileira de Psiquiatria, 36, 14–20.
Veale, D., Miles, S., & Anson, M. (2015). Long-term outcome of cognitive behavior therapy
for body dysmorphic disorder: A naturalistic case series of 1 to 4 years after a controlled
trial. Behavior Therapy, 46(6), 775–785.
Veale, D., & Neziroglu, F. (2010). Body dysmorphic disorder: A treatment manual. John
Wiley and Sons.
Veale, D., & Riley, S. (2001). Mirror, mirror on the wall, who is the ugliest of them all? The
psychopathology of mirror gazing in body dysmorphic disorder. Behaviour Research and
Therapy, 39(12), 1381–1393.
Veale, D., Willson, R., & Clarke, A. (2009). Overcoming body image problems including body
dysmorphic disorder. Constable and Robinson.
Weingarden, H., Renshaw, K. D., Davidson, E., & Wilhelm, S. (2017). Relative relationships
of general shame and body shame with body dysmorphic phenomenology and psychosocial outcomes. Journal of Obsessive-Compulsive and Related Disorders, 14, 1–6.
Wilhelm, S., Phillips, K. A., & Steketee, G. (2012). Cognitive-behavioral therapy for body
dysmorphic disorder: A treatment manual. Guilford Press.
Williams, J., Hadjistavropoulos, T., & Sharpe, D. (2006). A meta-analysis of psychological
and pharmacological treatments for body dysmorphic disorder. Behaviour Research and
Therapy, 44(1), 99–111.
Willson, R., Veale, D., & Freeston, M. (2016). Imagery rescripting for body dysmorphic
disorder: A multiple-baseline single-case experimental design. Behavior Therapy,
47(2), 248–261.
Windheim, K., Veale, D., & Anson, M. (2011). Mirror gazing in body dysmorphic disorder
and healthy controls: Effects of duration of gazing. Behaviour Research and Therapy,
49(9), 555–564.
315
16
Comprehensive Behavioral Treatment for
Trichotillomania (Hair Pulling Disorder)
and Excoriation (Skin Picking) Disorder
Charles S. Mansueto
Scene in a brightly lit bathroom. A young woman lies in her bathtub, her knees
pulled up near her face. She has lost track of time, but has been there for hours. With
one hand her fingertips glide over legs that are riddled with scars and scabs. Beneath
her tortured skin lie the objects of her consuming efforts – the tiniest of hairs – the
new growth that barely breaks above the skin’s surface. Once detected her other
hand, holding small silver tweezers, moves to the spot in a desperate effort to grip and
extract that hair and every other hair that she can find. The sharp tweezers’ tips dig
deep beneath the skin’s surface, but she hardly notices any pain. Digging and
scratching at her once delicate skin, now raw and unsightly, is the seemingly endless
cycle of picking and pulling that consumes untold hours of her young life. She stops to
rest only when her fingers detect no hairs remaining to be pulled or scabs to be
scratched off with her fingernails. Finally, when her fingers are too tired and aching
she stops and the episode ends. She lies back, filled with shame and self-loathing, and
cries, no longer noticing that the bathwater is spotted pink with her blood.
Body-Focused Repetitive Behaviors (BFRBs)
Repetitive behavior patterns involving hair pulling, skin picking, nail biting, cheek
and lip biting, and other similar behavior patterns, when taken together, are
referred to as body-focused repetitive behaviors (BFRBs).* Among these, hairpulling disorder (HPD), also called trichotillomania, and skin-picking disorder
(SPD), also called excoriation disorder, have received the most attention from the
psychological and psychiatric research and clinical communities. Both have been
accepted within official psychiatric nomenclature of the Diagnostic and Statistical
Manual of Mental Disorders of the American Psychiatric Association (HPD in
1987 and SPD in 2013, while other BFRBs are not specifically included). Over the
* The author wishes to thank David Keuler, Michael Lent, and Sherrie Vavrichek for their assistance in
preparation of this chapter.
316
Trichotillomania and Excoriation Disorder
past two decades professional attention paid to BFRBs has substantially increased
as research has established that these conditions are neither rare nor benign. While
milder forms of these behavior patterns are common in the general population, in
many adults and children these rise to the level of legitimate clinical disorders. Still,
HPD and SPD remain largely outside the awareness of the general public, the
scientific community, and the majority of clinical practitioners. For that reason, this
chapter will provide some detailed information about these disorders, since effective
treatment requires an in-depth understanding of their nature.
Diagnosis
Both HPD and as SPD are categorized within “Obsessive-Compulsive and Related
Disorders” in DSM-5 (APA, 2013). The diagnostic criteria for HPD include recurrent
pulling out of hair resulting in hair loss; SPD criteria include recurrent skin picking
resulting in skin lesions. Criteria for both disorders specify: repeated attempts to
decrease or stop the behaviors, significant distress or impairment as a result of these
behaviors, that they are not attributable to the other medical disorders or conditions,
nor better explained as symptoms of another mental disorder.
Epidemiology
HPD and SPD are both considered more common than previously understood.
Individuals with these disorders usually attempt to conceal the physical damage
from others with makeup, clothing, hairpieces, and hairstyles and can live lives of
secrecy and isolation. This probably contributes to the widespread view that these
disorders are rare. While large-scale studies documenting their prevalence within
the general population have not yet been conducted, reported rates for HPD have
been as high as 4.4% (Grant et al., 2005). However, wide discrepancies between
reported prevalence rates of HPD are notable across reports. Such discrepancies are
probably due to methodological limitations, such as small sample sizes, and inconsistencies in operational definitions and diagnostic criteria applied by different
researchers (Mansueto & Rogers, 2012). Reasonable estimates suggest that 1–2%
of adolescents and young adults suffer from the disorder (APA, 2013).
Regarding estimates of SPD prevalence rates, similar limitations apply.
According to DSM-5 (APA, 2013) between 1.4 and 5.4% of the population are
SPD sufferers. Among childhood hair pullers and skin pickers of all ages, incidence
seems to be evenly distributed between sexes. However, in the case of adult BFRB
sufferers, most clinical samples find females outnumbering males by substantial
margins (Mansueto & Rogers, 2012; Odlaug & Grant, 2012).
Course, Comorbidity, and Associated Problems
HPD and SPD can appear at any age. Onset at adolescence appears to be most
common with childhood onset being second most common. Variations in age of
317
318
Charles S. Mansueto
onset have led researchers to question whether there are phenomenological differences such as increased symptom severity and persistence, increased treatment
resistance, and increased comorbidity between early and later onset cases
(Keuthen et al., 2001a, b; Walsh & McDougle, 2001). In most cases these disorders
are chronic, fluctuate over time, and wax and wane in intensity during their course.
For both disorders, diagnostic criteria require significant distress or impairment
associated with the behaviors. Patients with these conditions often attempt to
conceal physical damage from others using makeup, clothing, hairstyles, and hairpieces. Significant numbers of sufferers report decreased self-esteem, a diminished
sense of attractiveness, shame and embarrassment, anxiety, and depression associated with their conditions. Many avoid common activities such as haircuts, sports,
sexual intimacy, and social situations (Flessner & Woods, 2006; Mansueto, 1991;
Stemberger et al., 2000; Woods et al., 2006). Clinical comorbidity is prevalent
among HPD and SPD sufferers, with anxiety and mood disorders among the most
common. Other less common but not infrequent disorders comorbid with BFRBs
include obsessive-compulsive disorder, body dysmorphic disorder, and an additional
BFRB such as the coexistence of skin picking and hair pulling (Flessner, 2012).
Besides exacting psychological and social tolls, there are also economic and
medical consequences associated with HPD and SPD. Time spent performing the
BFRB can interfere with academic and occupational functioning. Sufferers spend
hours each day (and money), in attempts to conceal resulting cosmetic damage
(Flessner & Woods, 2006).
In a subset of patients, HPD and SPD can result in several kinds of medical
problems. Most obvious is the damage to the hair or skin at the site (or sites)
targeted. Hair loss and skin lesions are predictable outcomes, but scarring, infections, and other medical complications are not unusual. Repeated trauma from
repetitive hair pulling and digging at the targeted sites with the fingernails or
implements such as tweezers and pins can produce degrees of tissue damage that
result in changes to the appearance and ultimate health of the hair and skin. In more
extreme cases of HPD, hair may not regrow, or regrowth may show color or textural
changes. The ingestion of the products of hair pulling, that is, the eating of all or
parts of the hair, can result in the development of trichobezoars, (hairballs).
Trichobezoars can produce gastrointestinal complications and may require surgery.
In rare cases, they can be fatal (Christenson & Mansueto, 1999). Skin picking often
produces tissue damage such as swelling, bleeding, weeping, infections, and scarring.
In more extreme cases of SPD, corrective medical procedures such as dermabrasion
or plastic surgery may be required.
Clinical Presentation of HPD and SPD
While earlier reports of HPD and SPD emphasized their mechanistic simplicity,
later descriptions have highlighted their heterogeneous nature and phenomenological complexity (O’Sullivan et al, 2000). Mansueto (1990) described an array of
variables that comprised the complexity and heterogeneity which distinguishes one
Trichotillomania and Excoriation Disorder
HPD sufferer from another on the basis of sensory experiences, cognitive elements,
affective states, motor habits, and situational variables. This view of the disorder
was affirmed in later reports (Christenson at al., 1991, 1993). HPD today is widely
regarded as diverse, idiosyncratic, and heterogeneous. With research interest
increasingly focused on SPD, it is clear that similar conclusions can be drawn
regarding that disorder as well (Grant & Odlaug, 2009).
HPD
Regarding HPD, Mansueto and colleagues (1997) acknowledged the wide variety of
behaviors associated with HPD that go beyond those specified within the diagnostic
criteria, and integrated them in a conceptual framework. Their conceptual model of
HPD has become known as the comprehensive behavioral (ComB) model. A wide
array of associative behaviors is seen most clearly when hair is pulled in what has
been termed a “focused style.” Focused hair pulling occurs when an individual is
highly conscious of pulling activities occurring. These activities tend to be accompanied by urges, thoughts about pulling, and feelings of discomfort. This can be
contrasted with “automatic pulling” in which pulling is performed with little or no
awareness, typically while the individual is engaged in some sedentary activity like
watching TV or reading. While some individuals have one or another predominant
style, most experience combinations of focused and automatic pulling (Christenson
& Mackenzie, 1994).
With the ComB model, the many potential activities associated with hair pulling
are organized into three phases: preparatory behaviors, pulling behaviors, and
disposal behaviors. Preparatory behaviors include such preliminary activities as
seeking privacy, accessing implements (such as mirrors and tweezers), and selecting
hairs to be pulled (such as thick, dark, rough, sharp, gray, or out-of-line hairs), either
visually or by touch. During the pulling phase, hair is most commonly extracted
from the scalp. However, eyebrows, eyelashes, and pubic hair are frequent sites as
well. Less commonly, hair is pulled from arms, legs, face, armpits, or other body
areas. While there may be only one exclusively targeted site, hair is often removed
from more than one site. Individuals may remove only a few or hundreds of hairs at
any given episode. An episode may last from seconds to hours at a time.
During the disposal phase, what is done with the pulled hairs can be elaborate
and idiosyncratic. Although quick disposal of the hair is a possibility, many individuals engage in activities with the hairs or hair root. It is not unusual for hair to be
visually examined or physically manipulated. Typical activities include rubbing of
the hair or its root between the fingers, on the face, or lips, and a variety of oral
activities such as biting, nibbling, chewing, or swallowing the hair root, bits of the
shaft, or, in some cases, entire hairs. Less commonly, hairs are saved for later
inspection, manipulation, or collection (Mansueto, 1991).
SPD
The clinical presentation of SPD is remarkably similar to that of HPD. While the
ComB conceptual model was developed for hair pulling, it is easily applied to skin
319
320
Charles S. Mansueto
picking, with only minor adjustments. As with HPD, the focused, automatic, and
combined styles also apply. While picking is mostly done with fingernails, in the
cases of focused picking, implements such as scissors, tweezers, pins, and mirrors are
also employed. Seeking of privacy and accessing implements are common preparatory activities for skin picking,
As with HPD, suffers can spend minutes to hours per day engaged in the
activity. The face is the most common site for picking. However, fingers, arms, legs,
chest, back, shoulders, feet, and pubic areas may also be targeted. Most individuals
pick from more than one site. Picking is usually directed at imperfections or
anomalies of the skin such as acne pimples, bug bites, scabs, bumps, or dry, flaking,
and rough patches of skin. Many individuals pick at healthy skin as well. Regarding
disposal, it is not unusual for products of the picking, such as bits of skin or scabs, to
be bitten and swallowed (Odlaug & Grant, 2012).
BFRB Treatment
A wide variety of therapeutic approaches drawing from various models of pathology
has been employed in efforts to help BFRB sufferers. These include traditional
psychotherapy, hypnosis, biofeedback, meditation, acupuncture, pharmacotherapy,
and behavior therapy. Among these, the two considered most promising, based on
results from limited clinical trials, are pharmacotherapy and behavioral treatments.
In a recent review of pharmacotherapy for BFRBs (Grant et al., 2012), the
authors deplored the shortage of adequate studies examining efficacy for these
disorders. However, they concluded that there is probably sufficient evidence from
randomized controlled trials to support the use of the opioid antagonist naltrexone,
the atypical antipsychotic olanzapine, and the glutamatergic agent N-acetylcysteine
for HPD, and the selective serotonin reuptake inhibitors (SSRIs) fluoxetine and
citalopram for SPD (Chamberlain et al., 2012). While pharmacological agents may
be helpful in treatment of BFRBs, particularly when significant comorbid disorders
are present, there are no psychotropic medications approved by the US Federal
Drug Administration for BFRBs.
Currently, the best supported of contemporary therapy approaches is behavior
therapy. In a meta-analysis of randomized controlled trials for HPD, McGuire and
colleagues (2014) reported that behavior therapy had a large effect in reducing hair
pulling severity, while SSRIs had a smaller effect. Similarly, Schumer et al. (2016)
found behavioral treatments superior to pharmacotherapy for skin picking in their
meta-analysis. Because behavioral approaches are currently considered treatments
of choice for BFRBs (Golomb et al., 2011), the remainder of this chapter will focus
on behavioral treatment.
Behavioral Treatment
Two behavioral treatment approaches stand out from others because of their
association with BFRB treatment. These are habit reversal training (HRT),
Trichotillomania and Excoriation Disorder
developed by Azrin and Nunn (1973), and comprehensive behavioral (ComB)
treatment developed by Mansueto and colleagues (1999). HRT was an early treatment and is still influential. Because it has the most empirical support of any single
treatment for BFRBs (Bloch et al., 2007), a brief overview will be provided of HRT
and its variants.
Habit Reversal Training (HRT)
Azrin and Nunn’s HRT viewed a mixed group of problematic repetitive behaviors,
such as tics and nail biting, as “nervous habits”: tension-reducing motor behaviors
that became largely automatic because, through frequent repetition, they became
strengthened. Later, HRT was applied to hair pulling (Azrin et al.,1980). From their
perspective, hair pulling and skin picking were viewed as well-practiced habits,
embedded in chains of internal and situational cues and connected responses,
through conditioning mechanisms, that could occur without conscious awareness.
In accordance with this conceptual model, HRT consists of a varying number of
components designed to compete with and eliminate the problem behaviors. The
essential component of HRT is competing response intervention, in which a motor
response that is incompatible with the undesired behavior, such as fist clenching, is
practiced in place of the problem behavior. Various therapeutic components were
added and deleted in later reports of HRT. These include awareness training,
motivation-enhancing procedures, imaginal rehearsal, self-monitoring, relaxation
training, and social support (e.g., Azrin & Nunn, 1977; Azrin et al.,1980). More
recently, Bloch and colleagues (2007) described HRT as encompassing four components: (1) self-monitoring, (2) awareness training, (3) stimulus control, and (4)
stimulus–response intervention (competing response training).
Early reports of successful treatment of hair pulling using variations of HRT
treatment (e.g., Rosenbaum & Ayllon, 1981; Miltenberger & Fuqua, 1985;
Tarnowski et al., 1987) were promising. However, they were criticized for substantial methodological shortcomings (Keuthen et al., 1999). Later indications of treatment effectiveness were less favorable with problems of relapse still significant
(Lerner et al., 1998; Mouton & Stanley, 1996; Van Minnen et al., 2003).
Augmented HRT and Mixed CBT Approaches
Recognition of the phenomenological complexity of BFRBs highlighted the possibility that Azrin’s behavioral model may have oversimplified the fundamental
nature of BFRBs by failing to identify some of the critical factors that govern them.
In efforts to increase treatment effectiveness, some researchers grafted other treatment components onto HRT that were designed to address factors not formally
addressed by HRT. One such later addition was stimulus control. Environmental
cues, such as the presence of a magnifying mirror in a brightly lit bathroom, that can
trigger an individual’s BFRB are altered or removed (e.g., Rothbaum, 1992).
Interest in possible cognitive contributions to BFRBs for individuals with strongly
held ideas, such as “I’ve got to get rid of these gray hairs,” led to the addition of
321
322
Charles S. Mansueto
cognitive therapy techniques, such as cognitive restructuring, to augment HRT
treatment (Gluhoski, 1995, Lerner et al., 1998).
In recognition of research evidence that BFRBs help modulate aversive emotions and other kinds of unwanted internal experiences (e.g., Arnold et al., 1998),
researchers have included emotional regulation skills as adjunctive components to
traditional HRT in BFRB treatment outcome studies. More recent efforts to bolster
HRT with newer CBT formulations designed to target these presumed variables,
and thereby to enhance treatment effectiveness, have included the addition of
dialectical behavior therapy skills (Keuthen et al., 2010) and acceptance and commitment therapy elements (Flessner et al., 2008; Woods et al., 2006). In clinical
practice, adult and pediatric BFRBs are likely to incorporate multiple intervention
strategies that include any of the abovementioned treatment components, such as
psychoeducation, reward systems, and relapse prevention techniques. The various
components of such “package” treatments can be applied in a modular fashion;
therefore, modifications to suit individual patient characteristics are possible
(Gianoli & Tolin, 2012).
It is generally accepted that the myriad forms of CBT, typically with HRT
elements at their core, are reasonably effective in the short term (Bloch et al.,
2007). However, the effectiveness of these approaches varied, and relapse was a
significant problem (Keijsers et al., 2006; Lerner et al., 1998). In particular, the
problem of failure to maintain gains indicates that there is room for improvement in
treatments for BFRBs.
The Comprehensive Behavioral (ComB) Model
Possible shortcomings in HRT and similar approaches to BFRBs can be identified.
First, there is an absence of a guiding, comprehensive, theoretical framework that
encompasses and integrates the behavioral, affective, cognitive, and sensory variables. Also, the general use of “package” treatments may not be individualized
enough to address the heterogeneity of BFRB presentations. Finally, treatment
models may not provide adequate guidance through the therapeutic steps of assessment, case conceptualization, and clinical decision-making to provide for more
effective treatment. The ComB conceptual model (Mansueto et al., 1997) was
designed to describe and capture the complex and heterogeneous nature of HPD.
While it is clear that CBT practitioners have utilized and tested a wide variety of
treatment techniques for BFRBs, this range of options can present a dilemma for
clinicians. From what can seem like a hodgepodge of available techniques derived
from standard CBT approaches, how can a clinician provide the most efficient and
effective treatment plan for any individual patient? This is the question that the
ComB approach is designed to answer.
The ComB treatment model was developed from the outset to address the
complexity and heterogeneity of hair pulling and to guide clinical decision-making
throughout the therapeutic process. Therefore, it is useful to be familiar with the
ComB conceptual model on which it is based (Mansueto et al., 1997) in order to
Trichotillomania and Excoriation Disorder
comprehend and most effectively apply ComB treatment to HPD and other BFRBs.
Extensive clinical experience suggests that ComB treatment, with minor modifications, can effectively address SPD.
ComB Conceptual Model
The ComB model of HPD was originally developed as a framework for organizing
and analyzing hair pulling data for research and clinical purposes, that is, as a
conceptual model. ComB described the broad array of variables associated with
hair pulling and, by extension, skin picking and other BFRBs in a functional analytic
(i.e., A-B-C) formulation, in which the antecedents (A) and consequences (C) by
which behaviors (B) associated with hair pulling are instigated and maintained.
These incorporate antecedents – cues that generate or otherwise facilitate impulses
and urges to engage in the BFRB, the array of behaviors involved in performance of
the BFRB, and the consequences that influence the repetition of those behaviors.
These variables are described as linked in accordance with familiar cognitive
behavioral principles. Thus, from the ComB perspective, BFRBs are a set of wellpracticed behaviors that are triggered by various cues, occurring internally or externally to the individual. These are maintained by positive and negative reinforcing
experiences. In other words, the performance of the BFRB behaviors is a function
of factors that immediately precede and follow them.
The ComB model specifies five categories (domains) in which variables functionally related to BFRBs can be identified. In this regard ComB has features in
common with the multimodal therapy of Arnold Lazarus (1981). There are significant differences, however. While multimodal therapy employs a seven-modality
framework to guide assessment and treatment of a wide range of disorders, ComB
specifies five categories of variables that are derived from BFRB research
(Mansueto, 1990) and are functionally relevant to these disorders. The five domains
comprise sensory, cognitive, affective, motor, and place (environmental) variables.
Note that sensory and cognitive variables are uniquely highlighted in combination
with other, familiar behavioral components. The anagram SCAMP provides a
convenient memory aid for recalling the five critical domains for patients and
clinicians throughout treatment. Some examples of variables within the SCAMP
domains are provided in Table 16.1.
Overview of ComB Treatment
At the core of ComB treatment (Mansueto et al., 1997) is the clinician’s task of
working collaboratively with the patient, first to identify and then to change relationships between the antecedent and consequential variables presumed to instigate
and maintain the BFRB. Treatment begins with a functional analysis employing an
in-depth structured assessment to identify an individual’s unique pattern of SCAMP
antecedents and consequences that foster performance of the BFRB. Change is
accomplished through an individualized treatment plan in which established behavior patterns are interrupted and nonharmful alternative behaviors are substituted to
323
324
Charles S. Mansueto
Table 16.1. Five domains of the ComB model
Sensory domain
Antecedents
Consequences
Cognitive domain
Antecedents
Consequences
Affective domain
Antecedents
Consequences
Motor domain
Antecedents
Consequences
Place domain
Antecedents
Consequences
Urges to pull hair or pick skin; the feel or sight of coarse hairs or blemished skin; itching
or irritation at the site; etc.
Satisfaction at accomplishing goals like removing unwanted hairs or smoothing out rough
skin; achieving desired sensations through pulling or picking, or through employment of
secured hair skin products (e.g., manipulating, nibbling hairs or skin), etc.
Facilitating thoughts or beliefs like “I’ve got to pop this pimple”; “These crooked
eyelashes have to go”; “I’ll just pull a few”; “This scab looks disgusting”; etc.
Thoughts like “That was a good pop”; “I’ve got that annoying stub”; “That feels nice
and smooth now”; “Got a nice plump root”; etc.
Unwanted feeling like being bored, stressed, anxious, depressed, tired, lethargic,
overstimulated, etc.
Satisfying feeling states achieved by hair pulling or skin picking like pleasurable
feelings, alleviation of boredom, stress and anxiety reduction, feeling energized, etc.
Habits involving facilitative postural cues and unconscious motor habits like twirling or
stroking the hair, running fingertips across skin, resting fingers against the face, rubbing
the eyes, etc.
Note: In the case of the consequences in the motor modality, the presumption is that the
expression of the habit serves as a reinforcing consequence.
External, environmental cues that trigger BFRB behaviors such as the presence of
physical implements like mirrors or tweezers; settings like the bathroom, car, or
bedroom; opportunities like being alone; etc.
Note: reinforcers in the place modality are not ordinarily functional in maintaining with
a few exceptions, such as when performance of the BFRB elicits desirable reactions
from others.
meet the idiosyncratic functions served by hair pulling or skin picking. Alternative
behaviors are carefully chosen within the domains that are identified, as relevant to
each individual’s needs. Treatment is modified, as necessary, in response to feedback regarding the utility of specific treatment recommendations. With practice, it is
expected that predispositions to pull hairs or to pick skin (and their accompanying
urges) will weaken as healthy alternative habits are established. This process utilizes
the SCAMP formulation as a guide for the assessment of BFRB-relevant features,
for the functional analysis of those features and for decision-making in
treatment implementation.
The ComB Treatment Process
As the ComB conceptual model is based on familiar behavioral principles, ComB
treatment encompasses a wide range of techniques drawn from standard behavioral,
cognitive, and CBT practice. These are familiar to CBT practitioners as they are
Trichotillomania and Excoriation Disorder
widely used to treat a broad range of disorders. Therapists are encouraged to draw
on their training, knowledge, and skills as client needs become apparent. For
example, a therapist skilled in motivational interviewing (Rollnick & Miller, 1995)
may incorporate those techniques to increase an individual patient’s motivation and
willingness to employ interventions taught in ComB treatment. If a BFRB patient is
burdened with stress or anxiety and has poor coping mechanisms, a therapist with
expertise in the use of mindfulness-based stress reduction (e.g., Kabat-Zinn &
Chapman-Waldrop, 1988), acceptance and commitment therapy (Hayes, 1999;
Woods et al., 2006) or dialectical behavior therapy skills (Keuthen et al., 2010;
Linehan, 1993) might provide any of these as therapeutic interventions. Because
such interventions require a significant amount of specialized training and experience, other established and widely used cognitive behavioral techniques such as
more traditional stress management techniques (e.g., diaphragmatic breathing,
progressive muscle relaxation, cognitive coping techniques, problem solving) will
suffice in most cases.
Because ComB treatment is uniquely tailored to the individual needs of each
patient, the treatment process is expected to be both creative and organic rather
than rigidly applied. The flexible and patient-oriented nature of the treatment
process results in therapy that may appear quite different from one patient to
another, due to factors such as the therapist’s skill set, judgment, and decisionmaking as well as the patient’s preferences.
There are two important preliminary considerations for the therapist to address
prior to beginning formal ComB treatment: First, the therapist should determine
that the BFRB is the appropriate target for treatment and that there are no more
prominent conditions overshadowing it. This is significant, because BFRBs frequently co-occur with other disorders such as depression, anxiety, and OCD. The
question of whether to address other disorders first or concurrently with BFRB
treatment should be determined at the outset. Some comorbid conditions, such as
milder forms of depression or anxiety, may be secondary to hair pulling or skin
picking. They may either abate with successful treatment of the BFRB or may not
significantly undermine treatment if left unaddressed. When significant comorbidity
renders an individual incapable of benefiting from ComB treatment, other conditions may be more appropriately targeted prior to efforts to treat the BFRB.
Second, more so than those with many other disorders, people who suffer from
BFRB are highly sensitive to the self-induced aspects of the problem. It is typical
therefore, for secondary features such as shame, poor self-esteem, and impaired
social functioning to be significant issues among these patients. The therapist needs
to be sensitive to this from the first encounter. Care should be taken throughout the
course of treatment to help the patient move toward healthy self-acceptance and
normalized social functioning. Subtle, nonverbal cues – a brief look of disgust fleeting
across the therapist’s face, for example – can damage efforts of rapport-building and
thus may severely compromise prospects for successful treatment. A casual,
accepting, and nonjudgmental tone adopted by the therapist during detailed and
frank exchanges about the BFRB can facilitate resolution of these issues.
325
326
Charles S. Mansueto
As mentioned above, ComB treatment is administered in a flexible, creative,
and individually tailored manner. Treatment does proceed methodically through a
series of four phases: (1) psychoeducation and assessment, (2) functional analysis
(identification and organization of maintaining factors within relevant domains), (3)
implementation of specific interventions, and (4) evaluation, termination, and
relapse prevention. However, these phases are not rigidly prescribed. Elements
associated with any one of the phases can be provided in any of the other phases
as determined by the therapist’s judgment and the patient’s needs.
A case description of a patient who both pulls out her hair and picks at her skin
will be provided to illustrate all phases of treatment.
HPD and SPD Combined: A Case Description
Kaitlyn, 22 and single, is one year out of college and is living alone in Florida where
she works at her first “real” job. Since she was a teenager she had a problem with
hair pulling. At first it was her eyebrows, which she would pull sporadically when
she had the necessary privacy; she said she does not remember why. When her
mother noticed that she had patches of missing eyebrows, she made quite a fuss and
never let up. Kaitlyn made a sincere effort to stop pulling and allow her eyebrows to
grow back. At some point, in desperation, she began to pull out hairs from her legs
instead of her brows, as it was easier to keep that pulling a secret. It worked pretty
well. Her eyebrows mostly grew back, and missing leg hairs were easier to hide.
Other problems began to develop, though. Sometimes it was hard to grab hold
of the hairs on her legs with just her finger, so she began using tweezers to pull the
short hairs and to dig out deeply buried hairs. Her legs bled when she dug too
deeply and scabs would form on the damaged skin. It was impossible for Kaitlyn to
resist picking off scabs as she had to shave her legs and their roughness bothered her
anyway. During high school this was more of an inconvenience, though at times
there was enough visible damage to prevent her from wearing clothes that bared her
legs. Sometimes she resorted to using makeup to camouflage the damage she had
done. Still, the problems were relatively minimal and she was reasonably happy and
well adjusted through the remaining years of high school. Things were even better
most of the time after she went away to college. She found that dorm life, as well as
a busy academic and social life, helped her keep her problem manageable. It was
still occurring but much less frequently and with less severity.
Now, living alone, with more privacy and more free time, things were getting
out of hand. Fortunately, she could wear slacks at work, but in the Florida climate
she longed to wear shorts and skirts, and swimsuits at the beach. Another big
problem was that she had met a guy whom she liked very much. Their relationship
had all the indications of becoming more serious, but she had so far avoided his
invitations to accompany him to pool parties and some other outdoor activities.
Moreover, she wanted more intimacy with him but had disallowed that for fear of
his reaction when he saw her bare legs. The more upset she became about her
Trichotillomania and Excoriation Disorder
circumstances, the worse her BFRBs became. Her new life was being spoiled by
problems over which she had no control.
A new problem added to her woes. Living in her own apartment, she had been
enjoying her growing independence and freedom from the constraints that she had
felt when she lived at home. Unfortunately, she remained dependent on her mother
for financial assistance as she struggled to pay back college loans and to cover other
living expenses. Her relationship with her mother was always difficult, but now she
was an adult and wished she were treated like one. Hundreds of miles away from
home, her mother, always an anxious and controlling parent, was phoning her
almost every afternoon just as Kaitlyn arrived back from work. She had a way of
asking intrusive questions and providing unwanted advice during those calls.
Though she tried to maintain her composure, Kaitlyn inevitably felt trapped, unable
to get a word in edgewise, and grew more frustrated and angry as the conversations
became prolonged and more one-sided. Kaitlyn’s usual pattern was to fall down on
her couch holding her cell phone and pull out leg hairs. She realized she couldn’t
blame everything on her mother because a lot of damage was done in situations
where her mother had nothing to do with it.
On top of that, a new development was pushing her over-the-edge. Seemingly
out of nowhere, she began pulling hair from her scalp. She had no idea where that
came from and could not recall even thinking about doing it. It wasn’t clear to her
when it started. All she knew was that, for at least two months, she had a growing,
egg-shaped bald spot above her left ear, which was increasingly difficult to keep
hidden from others.
One night after a particularly frustrating episode in which a lot of hairs were
pulled, Kaitlyn went on line, discovered the TLC Foundation for BFRBs’ website,
and found a trained therapist near her. What follows is a description of her therapy.
Phase 1: Assessment and Psychoeducation
Kaitlyn made the appointment with Dr. Benton, but she was very nervous about the
first visit. No one knew all the details about her pulling and picking practices. For
example, sometimes her skin picking didn’t stop at her upper legs but continued to
the pubic area. She had damaged her skin there also. She had other embarrassing
secrets as well. She would scratch off little scabs and pieces of dry skin with her
fingernails, put them in her mouth, nibble, and swallow them. She could never tell
anybody that.
Dr. Benton, however, put her at ease during the first session. Kaitlyn listened
dumbfounded as he spent some time describing how common these problems were,
how difficult it could be to live with them, and how their impact on his patients’ lives
could be profound. She felt relieved when he mentioned that pulling pubic hair was
occurring in at least half of the people who pulled out hair. Later, he told her that
more than half of the people who pull hair or pick skin did something oral with the
products of the pulling and picking. He seemed to understand that there were many
ways that pulling hair or picking skin could result in some desirable outcomes for the
person who did it. Dr. Benton said for some, it just felt really good; for others, it
327
328
Charles S. Mansueto
helped them handle emotions that they just didn’t want to feel. It gave them
something to explore with the fingers, eyes, or mouth as a distraction when they
were bored. He knew about and seemed to understand the “weird” things that
people did with hairs, scabs, and skin – even people who didn’t have a BFRB
problem. He seemed to understand that, while part of a person with BFRBs really
wants to stop because of the damage done to their bodies and the impact on their
lives, another part of them doesn’t want to give up the shorter-term, positive returns
of engaging in those behaviors. That was the therapist’s job, he said, to mobilize the
part of her that wanted to cease damaging her body, and to find healthy ways to
meet the needs currently being met by BFRBs. Kaitlyn had feared the worst of this
initial session and now was feeling safe, understood, and in good hands. Dr. Benton
gave her some informative pamphlets from the TLC Foundation about BFRBs to
read at home that included information about online support groups.
Dr. Benton made it clear that BFRBs are not an indication of any deep-seated
problems and that her therapy would directly address the hair pulling and skin
picking. He would use a therapy approach called comprehensive behavioral treatment, or ComB, that she would learn more about during the following sessions.
However, he did give her a brief overview of SCAMP domains. She immediately
understood that there was more complexity to her hair pulling and skin picking than
she had recognized. Dr. Benton’s obvious familiarity with BFRBs helped Kaitlyn
trust the process that would follow.
Assessment
Having gained sufficient information, Dr. Benton confirmed that Kaitlyn met criteria for two disorders: HPD and SPD. He believed that her sadness, anxiety, and
anger issues were largely secondary to BFRB problems.
Dr. Benton had Kaitlyn fill out two brief measures: the Massachusetts General
Hospital (MGH) Hair Pulling Scale (Keuthen et al., 1995), and the Skin Picking
Severity Scale (SPSS) (Keuthen et al., 2001a). (See Appendices 16.1 and 16.2 for
these measures.) These would be administered weekly throughout her treatment.
At this early stage of treatment, Kaitlin was introduced to self-monitoring and
recording of information about her BFRBs. She was given self-monitoring forms to
be used weekly, which would help her keep track of incidences of hair pulling and
skin picking and other information that would be used in the course of treatment (a
sample Weekly Self-Monitoring Form is provided in Table 16.2). There is no ideal
or definitive method of monitoring BFRBs. The sample form provided may be
modified in any number of ways to better suit individual patients. For instance, hair
pulling severity might be monitored as the number of hairs pulled daily.
Note that because there is no perfect metric for all forms of BFRBs, a subjective
rating by the patient (severity Score, 1–10) was employed to help increase awareness and to uncover other useful information.
These self-monitoring forms were to be completed between sessions. Kaitlyn
was helped to understand that this would allow her and Dr. Benton to identify
specific cues that preceded hair pulling and skin picking episodes, and to document
Trichotillomania and Excoriation Disorder
329
Table 16.2. Weekly self-monitoring form
Date
Place
Activity
Internal cues
thoughts,
sensations,
emotions
External cues
time of day,
implements, etc.
Interventions
used
(if any)
# of Hairs pulled or
severity score
(1–10)
the desirable effects derived from her BFRBs. With help from Dr. Benton they
organized information she brought back into SCAMP domains. With a little practice, Kaitlyn incorporated SCAMP concepts into her conversations with Dr. Benton
as well as into the ways she came to think about hair pulling and skin picking. What
follows is a summary of the information she provided, organized into SCAMP
domains, that Dr. Benton used to complete the functional analyses of her HPD
and SPD.
Phase 2: Functional Analysis: Identification and Organization of Maintaining
Factors within Relevant Domains
Most of Kaitlyn’s BFRB problems could be organized into three main clusters:
focused pulling and picking while bathing, focused pulling and picking while on the
phone with her mother, and largely automatic pulling and picking in a wide variety
of situations – chiefly sedentary ones or while deeply absorbed in an activity. Each
cluster would be subjected to separate analyses.
Bathing: Uses tweezers to pull out unwanted hairs on her legs and to dig deeply
through her skin to remove ingrown hairs from her legs, but also from the pubic
area.
Sensory Antecedents: Visual and tactile detection of “stubs” – short, dark, new
hair growth on her legs, and detection of ingrown hairs.
Sensory Consequences: Desirable feeling of smoothness when fingertips explore
skin after hair removal, sight of skin devoid of stubs.
Cognitive Antecedents: “Hate these stubs”; “Got to get the ingrown hairs out or
they’ll get sore and infected.”
Cognitive Consequences: “Got that one!”; “Mission accomplished!”
Affective Antecedents: Neutral feelings turn quickly to intense distress after
detection of the sharp hairs and the ingrown hairs.
Affective Consequences: Immediate reduction of distress on removal of hairs.
Motor Antecedents: None – all BFRB behaviors are done with
complete awareness.
Motor Consequences: None.
330
Charles S. Mansueto
Place Antecedents: Bright bathroom lights, tweezers within reach (a wellpracticed behavioral pattern when bathing).
Place Consequences: None.
On Phone with Mother: Pulls leg hairs with tweezers and digs for ingrown hairs
while talking on the phone.
Sensory Antecedents: None initially to trigger the BFRB behaviors, but once
picking and pulling has begun, she targets the new, prickly hairs and the
ingrown hairs that she detects by sight and touch.
Sensory Consequences: Minimal; in these situations, she gets little actual satisfaction from the removal of those hairs, and doesn’t bother to “finish the job”
or to enjoy the smoothness of her skin on her fingertips.
Cognitive Antecedents: No hair or skin-related thoughts – just angry thoughts in
response to her mother’s comments.
Cognitive Consequences: Not as notable as when bathing, but still an element of
“Got that one!” occurring sporadically.
Affective Antecedents: Anger, frustration, and feelings of being trapped.
Affective Consequences: Distraction from negative feelings, reduced level
of distress.
Motor Antecedents: Minimal; phone in left hand, tweezers in right is typical
position on the couch, which facilitates performance of the BFRB; however,
all BFRB behaviors are done with complete awareness.
Motor Consequences: None.
Place Antecedents: A second pair of tweezers kept between the couch cushions
(leading to a well-practiced behavioral pattern in this situation).
Place Consequences: None.
Automatic Picking and Pulling in Various Situations: With self-monitoring and
Kaitlyn’s growing self-awareness, it became clear that in many situations it
seemed that her left hand had “a mind of its own.” The situations in which
this type of pulling occurred were expanding – even to her desk at work. All
pulling from the scalp was done with her fingers, never with tweezers. In
those situations, her fingers would go to her scalp as she concentrated on
something else. She had a “favorite spot,” over her left ear, from which she
removed hair and produced a bald spot, roughly the size and shape of an egg.
At those times her fingertips would explore the edges of the spot and pull out
individual hairs, but more often a few at a time. Otherwise, her fingertips
explored the denuded area, pulling out any new growth she could grasp.
When she caught her fingers at her scalp, usually she would pull her hand
away, but before long, the pattern repeated itself. Sometimes she would just
give up and let it happen.
Kaitlyn eventually told Dr. Benton about one aspect of her BFRB that she
had been holding back from him: she enjoyed picking scabs off her legs,
usually while lounging around her apartment. She acknowledged that she
would bite and swallow bits of scabs, particularly enjoying the “crunchy
ones.” This behavior pattern also had an element of unconsciousness to it
Trichotillomania and Excoriation Disorder
some of the time, so Dr. Benton combined it with hair pulling from the scalp
for the functional analysis.
Sensory Antecedents: None that she was fully aware of prior to pulling hair or
picking scabs; after pulling from the scalp she sometimes rolled hairs between
her fingertips; when scratching scabs from her legs, she had more awareness
of the biting and swallowing than of the picking.
Sensory Consequences: Sometimes gets satisfaction out of clearing her bald spot
of new growth; her fingertips detect smoothness in that denuded area, eliminating scabs on her legs again produces a detectable smoothness – that
smoothness and the nibbling on scabs are experienced as pleasurable.
Cognitive Antecedents: Few hair- or skin-related thoughts in either case.
Cognitive Consequences: None notable, but occasional positive thoughts like
“Got that stub”; ”Nice and smooth”; “Good crunchy one.”
Affective Antecedents: Sometimes boredom, sometimes mild restlessness in
both cases.
Affective Consequences: Mildly stimulating when bored, mildly soothing
when restless.
Motor Antecedents: Without awareness, hands move to the pulling or
picking site.
Motor Consequences: Habit is expressed by activity at the pulling or picking site.
Place Antecedents: No fixed place requirement; however, privacy, a sitting
position, at least one hand free, and access to the pulling or picking site
are precursors.
Place Consequences: None.
Phase 3: Implementation of Specific Interventions
Now Kaitlyn and her therapist were both aware of the many antecedents and the
consequences associated with hair pulling and skin picking organized within the
SCAMP domains. It was time to implement some of the techniques that interfered
with “business as usual.” The place domain variables, once identified, pinpointed
the environments in which other contributing SCAMP variables would have to be
addressed. Information from Dr. Benton’s semi-structured interview and Kaitlyn’s
self-monitoring forms helped determine that therapeutic interventions would target
elements associated with three discretely different circumstances: the two high-risk
situations (the bath and the phone calls with her mother) as well as the various
random places in which the less mindful, more automatic pulling and picking
typically happened.
Dr. Benton told Kaitlyn during the next session that they would put treatment
elements in place that were designed to meet some of the needs currently met by her
BFRBs. These elements would also interfere with BFRB performance, allowing
healthier behavior patterns to be established in their place. He provided Kaitlyn
with an “intervention ideas list” (see Table 16.3) and asked her to review it during
the week. He explained that these were specific interventions organized according
to SCAMP domains.
331
332
Charles S. Mansueto
Table 16.3. Intervention ideas list
Sensory (decrease cues through healthy grooming; stimulate the senses in other ways)
Interventions that can soothe or stimulate the whole body: Warm bath, cool shower, exercise, dance
Cognitive (dispute unhelpful beliefs and attitudes; replace with more helpful ones)
How to think about yourself, the growth and healing of hair and skin, and the nature of BFRBs in ways that
will help the recovery process:
Cognitive restructuring: Restate BFRB-facilitating thoughts using accurate undistorted,
nonjudgmental language.
Learn more about BFRBs and treatment. Go to responsible websites and other sources for more
information about your condition and ways to recover from it.
Adaptive self-talk: Talk to yourself in positive ways that are encouraging – as you would with your
best friend.
Problem-solving: Explore alternative solutions to problems, such as assertiveness.
Mindfulness: Learn mindfulness techniques to recognize the sensations, emotions, and thoughts that
trigger picking or pulling and then sit with the experiences without acting on them.
Affective: Find better ways to respond to and balance uncomfortable emotions/other internal states
Calm the affective system
Activate, energize the affective system
Schedule pleasurable activities (e.g., socialize, sports,
gardening, hiking, crafts)
Cool shower
Effective communication/assertiveness
Gentle, moderate, or vigorous exercise
Volunteer work
Make a realistic to-do list (daily, longer term)
Deep breathing
Relaxation exercise (scheduled or as needed)
Meditation, yoga
Positive visualization
Mindfulness exercises
Acceptance
Warm bath
Interventions That Can Both Calm or Simulate the
Talk things through with friends or a confidant
Medications
Journal writing
Use affirmations
Affective System, Depending on the Need
Create daily gratitude lists
Do housework
Listen to music
Motor: Increase awareness, interfere with access, engage in alternative activities
Keep fingers from target area
Wear finger bandages
Cover fingertips with medical tape
Wear driving gloves
Keep hands moist with lotion
Cut fingernails short
Wear light cotton gloves
Wear elbow brace
Make an effort to not cross the boundary to
BFRB site (e.g., hands stop at necklace)
Keep hands busy in other ways
Use “fiddle toys”
Groom nails: manicure/pedicure
Engage in knitting or other craft
Engage in physical exercise
Clench fists
Make it harder to pick or pull
Tape/bandage on picking site
Wear clothes that cover the picking site
Wear glasses
Put ophthalmological ointment on eyelids
Wear false eyelashes or makeup
Wear a hat/bandana/headband
Put hair in pony tail, braids, or other hair style that
decreases access to hair
Get short haircut
Wear a wig or hairpiece
Wet hair and/or leave conditioner in hair
Get professional help (e.g., haircut, laser treatment,
electrolysis, prescription medicine)
Trichotillomania and Excoriation Disorder
333
Table 16.3. (cont.)
Place/environment: Decrease triggers (physical location, activity, time of day, privacy)
Decrease environmental cues
Adjust schedule or activities
Try to avoid using computer or TV during high-risk
times, or use motor interventions to block access
Contact someone for support
Avoid being alone in high-risk situations
Keep hands “busy” during high-risk times
Plan “wind down” activities at night
Put “caution” sign on bathroom door or mirror
Keep “fiddle” toys in trigger locations
Put a timer in the bathroom
Remove/cover mirrors
Eliminate tweezers/magnifying mirror
Put sticky note reminders on mirrors
Cover mirrors
Dim lights while using toilet
Stay out of certain rooms when possible or
leave the door open when possible
The items on the list were intended to stimulate Kaitlyn’s creativity. She might
select some of the interventions directly from the list or generate some of her own.
They would be introduced methodically but flexibly, deciding which targets to
choose, which interventions to implement, and when to add or delete interventions.
These choices would be determined by success achieved in gaining control over
problem behaviors, and by Kaitlyn’s preferences in consultation with Dr. Benton.
Interventions in the Bath: Kaitlyn and Dr. Benton decided to target the bath in
the first effort to gain the upper hand over her BFRBs. What follows are specific
interventions that were chosen by Kaitlyn. Also indicated are a few that were
rejected for various reasons. For example, shifting from baths to showers was
rejected because Kaitlyn thoroughly enjoyed her baths and would miss having them.
Also, she worried that showers offered similar opportunities for BFRB behaviors
to occur.
Note that the interventions were not necessarily implemented in accordance
with the order of the SCAMP domains. Often interventions in the place domain are
implemented early in the process because of the relative ease of making environmental changes. Also, intervention tactics and their effects are not likely to be
limited to any one SCAMP domain. Finally, all of the interventions indicated below
were not implemented at the same time. Different combinations of them were
chosen, implemented, and evaluated based on the judgment of Kaitlyn and Dr.
Benton.
Sensory Interventions: Limit visual cues, use candles instead of overhead lights,
use bubble bath in the water. Limit tactile cues, wear light cotton gloves in
bath when possible.
Cognitive Interventions: A problem-solving approach was used. A decision was
made to engage her dermatologist for laser removal of ingrown hairs.
Regarding removal of “stubs,” Kaitlyn was encouraged to reframe them as
“baby hairs” and get used to allowing them to grow out (later they would be
removed with a depilation device recommended by her dermatologist).
334
Charles S. Mansueto
Affective Interventions: Problem-solving in the cognitive and sensory domains
greatly reduced Kaitlyn’s stress during bathing, thereby reducing the need to
provide specific affective interventions for the bath.
Motor Interventions: None deemed necessary, as all BFRB behaviors
were focused.
Place Interventions: Removed tweezers from the bathroom (see other tactics
above, under Sensory Interventions).
Interventions on the Phone with Mother: These recurrent BFRB episodes
required a very different SCAMP-based plan than the bath.
Sensory Interventions: None.
Cognitive Interventions: Learned the importance of straightforward and assertive communication and, with some reading and practice, used her assertiveness skills with her mother; her mother agreed to prevent their phone
conversations from falling into the patterns that Kaitlyn found so distressing;
agreement was reached to limit phone calls to three times per week and to
fifteen minutes’ duration.
Affective Interventions: Practiced diaphragmatic breathing to reduce stress
during conversations with her mother (also prepared for calls with five
minutes of diaphragmatic breathing); Kaitlyn also practiced diaphragmatic
breathing to alleviate work-related stress and other life stressors.
Motor Interventions: Free hand was kept busy with manipulation of objects that
provided pleasing sensations (after sampling a number of possibilities, she
found that she preferred sensory substitutes with smooth, cool surfaces like
polished stones, metal balls, etc.). She intentionally wore pants that were tight
around the ankles and had drawstrings, which she would intentionally knot
when she felt particularly vulnerable to picking.
Place Interventions: Removed tweezers from the couch cushions and placed
basket containing polished stones, bracelets, and other sensory substitutes
within reach of the couch.
Interventions for Mainly Automatic Pulling and Picking: The third SCAMPbased plan would address BFRB activity that was occurring in many different
situations where hair was pulled from the scalp and scabs were picked from
the legs, nibbled, and ingested.
Sensory Interventions: Preferred sensory substitutes were kept available in
places where automatic performance of BFRB behaviors were very likely
(i.e., desk at work, armchair where she watched TV, etc.). Wearing bracelets
consisting of large, smooth stones, keeping small hard candies available to
provide an alternative to ingestion of BFRB products; hard candies were
eventually replaced with taco chips that could be nibbled and swallowed in
small pieces. Kaitlyn applied medicated ointment to her legs, which softened
the scabs and made them taste unpleasant.
Cognitive Interventions: Planning ahead to ensure that sensory interventions
were available at necessary times and places; Kaitlyn reminded herself the
Trichotillomania and Excoriation Disorder
335
baby hairs growing in her bald spot would help her regain healthy and
attractive hair.
Affective Interventions: Diaphragmatic breathing for emotional-regulation in
“tense” situations where she might be vulnerable to BFRB activity; anticipated situations where she might be bored and kept stimulating activities
available (e.g., apps on smartphone, crossword puzzles, news sites, email,
etc.).
Motor Interventions: Ensured free hands were occupied with sensory substitutes; bandages were worn on fingers employed in automatic pulling and
picking (when it was not awkward to do so) to promote awareness. She
engaged in activities that occupied her hands (e.g., grooming nails, needlepoint). She kept her fingernails short.
Place Interventions: Sensory substitutes and stimulating activities were kept
readily available; the occasional use of head coverings was discontinued
because they made her scalp itch and that “invited pulling.”
Phase 4: Evaluation, Termination, and Relapse Prevention
Evaluation: Progress in therapy was monitored by weekly scores on the MGH Hair
Pulling Scale (MGH/HPS) and the MGH Skin Picking Scale (MGH/SPS). These
scales can be found in Appendices 16.1 and 16.2. Weekly scores through the course
of 15 therapy sessions are shown in Figure 16.1.
As Figure 16.1 shows, from the first therapy session through to the fifteenth
therapy session, Kaitlyn’s scores on the MGH/HPS declined from 18 to 6. On the
MGH/SPS her scores declined from 24 to 10. Self-recorded data, with approximate
Progress in ComB Treatment
30
Score
23
15
8
0
1
2
3
4
5
6
7
8
9
10
11
12
13
Session
MGH Hair Pulling Scale
MGH Skin Picking Scale
Figure 16.1. Kaitlyn’s progress in ComB treatment for two BFRBs.
14
15
336
Charles S. Mansueto
numbers of hairs pulled, proved unreliable as a measure of BFRB severity.
However, record keeping was used for other therapeutic purposes, such as raising
awareness about critical variables associated with BFRBs. By all indications,
Kaitlyn achieved a substantial reduction in her BFRB symptoms.
Termination: Kaitlyn and Dr. Benton agreed to terminate treatment at session
fifteen. Kaitlyn did not achieve total symptom cessation (which, while less than
ideal, is not unusual), but she was pleased with her improvement. New hair growth
was filling in her bald spot, she had begun laser treatments, and her skin was looking
much better. She was hopeful that the techniques she learned in therapy would
continue to serve her well. She was very optimistic about her prospects for a life
unencumbered by BFRBs.
Relapse Prevention: Dr. Benton had one final issue to discuss with Kaitlyn
before she left his care. He cautioned her about the dangers of complacency in
managing BFRBs. Research and clinical experience tell us that “slips” (sudden
symptom recurrence) are almost inevitable when recovering from BFRBs. It is illadvised to think in terms of “cure” and prudent to think in terms of “management.”
In precaution against a full-blown relapse, Kaitlin was provided with a relapse
prevention plan to implement should a slip occur. This included the following steps:
1.
2.
3.
4.
Phase out interventions gradually, and reinstate them as needed.
Remain vigilant about returning urges.
Maintain involvement with an online BFRB support group.
Anticipate challenging life circumstances, and remain prepared
BFRB resurgence.
5. Return to a multifaceted self-help plan in the event of a major slip.
6. Schedule an additional “booster” therapy session on an as-needed basis.
for
Comments and Conclusions
Evidence in Support of ComB Treatment
ComB treatment is a conceptual model based on established behavioral principles
and sound theoretical formulations derived from decades of laboratory and clinical
research. Many specific interventions, tailored for ComB treatment, are familiar
transdiagnostic CBT techniques. These treatment components comprise a wide
range of behavioral and cognitive techniques frequently described in the scientific
literature and used extensively by thousands of CBT practitioners to treat a broad
range of disorders.
While ComB has yet to be tested in a randomized controlled paradigm,
research efforts are underway to bolster empirical support. A treatment manual
for ComB was developed (Mouton-Odum et al., 2013) and was utilized in a recent
pilot test of ComB efficacy (Falkenstein et al., 2016). Uncontrolled preliminary data
showed that ComB treatment reduced HPD symptom severity and impairment with
substantial effects, and with a modest decline in improvement at three-month
Trichotillomania and Excoriation Disorder
follow-up. A current study is underway to study ComB treatment in a randomized
trial with a minimal attention-placebo control. A future study is planned in which
the efficacy of ComB is compared head-to-head with HRT.
In the clinical realm, hundreds of therapists have been trained in the ComB
treatment approach through the Professional Training Institute of the TLC
Foundation for BFRBs. Many clinicians have adopted the ComB approach, having
acquired informal training through various mechanisms, such as exposure to written
accounts in the professional literature (Mansueto et al., 1999; Stemberger et al.,
2000, 2003) and by viewing a ComB training series, TLC’s Virtual Professional
Training Institute, produced and distributed by the TLC Foundation. While this
chapter has focused on treatment for adults with BFRBs, with minor modifications
the ComB treatment approach has been adapted for use with children and adolescents by many clinicians for individual therapy applications and for self-help
(Golomb & Vavrichek, 2000).
ComB Treatment in Perspective
While the need exists for further outcome research to determine whether ComB
treatment is most effective and efficient for the individual who suffers from BFRB,
decades of clinical observations and testimonials by numerous expert clinicians
support the following conclusions:
ComB provides a likely effective alternative to existing CBT approaches.
ComB addresses the diverse and idiosyncratic elements that foster BFRBs in
user-friendly form.
ComB guides assessment of information relevant to an individual’s BFRB and
organizes it into categories recognized as valid and useful.
ComB helps generate a wide variety of possible therapeutic interventions.
ComB guides the therapist and patient through a collaborative process of
clinical decision making, ensuring that therapy fits well with the unique characteristics of each patient.
At this time, no other single treatment for BFRBs has been recognized as providing
these advantages within an integrated conceptual framework and comprehensive
treatment approach.
REFERENCES
American Psychiatric Association. (1987). Diagnostic and statistical manual of mental disorders (3rd ed., revised) (DSM-III-R). American Psychiatric Association.
(2013). Diagnostic and statistical manual of mental disorders (5th ed.) (DSM-5). American
Psychiatric Association.
Arnold, L. M., McElroy, S. L., Mutasim, D. F., Dwight, M. M., Lamerson, C. L., & Morris,
E. M. (1998). Characteristics of 34 adults with psychogenic excoriation. Journal of
Clinical Psychiatry, 59(10), 509–514.
337
338
Charles S. Mansueto
Azrin, N. H., & Nunn, R. G. (1973). Habit-reversal: A method of eliminating nervous habits
and tics. Behaviour Research and Therapy, 11(4), 619–628. doi:10.1016/0005-7967(73)
90119-8.
(1977). Habit control: Stuttering, nail biting, and other nervous habits. Simon and Schuster.
Azrin, N. H., Nunn, R. G., & Frantz, S. E. (1980). Treatment of hairpulling (trichotillomania):
A comparative study of habit reversal and negative practice training. Journal of Behavior
Therapy and Experimental Psychiatry, 11(1), 13–20. doi:10.1016/0005-7916(80)90045-2
Bloch, M. H., Landeros-Weisenberger, A., Dombrowski, P., Kelmendi, B., Wegner, R.,
Nudel, J., Pittenger, C., Leckman, J. F., & Coric, V. (2007). Systematic review:
Pharmacological and behavioral treatment for trichotillomania. Biological Psychiatry,
62(8), 839–846. doi:10.1016/j.biopsych.2007.05.019.
Chamberlain, G. A., Fineberg, N. A., & Odlaug, B. L. (2012). Pharmacotherapy. In J. E.
Grant, D. J. Stein, D. Woods, & N. J. Keuthen (Eds.), Trichotillomania, skin picking, and
other body-focused repetitive behaviors. American Psychiatric Publishing.
Christenson, G. A., & Mackenzie, T. B. (1994). Trichotillomania. In M. Henson & R. T.
Ammerman (Eds.), Handbook of prescriptive treatments for adults. Plenum.
Christenson, G. A., Mackenzie, T. B., & Mitchell, J. E. (1991). Characteristics of 60 adult chronic
hair pullers. American Journal of Psychiatry, 148(3), 365–370. doi:10.1176/ajp.148.3.365
Christenson, G. A., & Mansueto, C. S. (1999). Trichotillomania: Descriptive characteristics
and phenomenology. In D. J. Stein, G. A. Christenson, & E. Hollander (Eds.),
Trichotillomania. American Psychiatric Press.
Christenson, G. A., Ristvedt, S. L., & Mackenzie, T. B. (1993). Identification of trichotillomania cue profiles. Behaviour Research and Therapy, 31(3), 315–320. doi:10.1016/00057967(93)90030-x
Falkenstein, M. J., Mouton-Odum, S., Mansueto, C. S., Golomb, R. G., & Haaga, D. A. F.
(2016). Comprehensive behavioral treatment of trichotillomania: A treatment development study. Behavior Modification, 40(3), 414–438. doi:10.1177/0145445515616369
Flessner, C. A. (2012). Diagnosis and comorbidity. In J. E. Grant, D. J. Stein, D. Woods, &
N. J. Keuthen (Eds.), Trichotillomania, skin picking, and other body-focused repetitive
behaviors. American Psychiatric Publishing.
Flessner, C. A., Busch, A. M., Heideman, P. W., & Woods, D. W. (2008). Acceptanceenhanced behavior therapy (AEBT) for trichotillomania and chronic skin
picking. Behavior Modification, 32(5), 579–594. doi:10.1177/0145445507313800
Flessner, C. A., & Woods, D. W. (2006). Phenomenological characteristics, social problems,
and the economic impact associated with chronic skin picking. Behavior Modification, 30
(6), 944–963. doi:10.1177/0145445506294083
Gianoli, M. O., & Tolin, D. F. (2012). Cognitive-behavioral therapy for pediatric trichotillomania. In J. E. Grant, D. J. Stein, D. Woods, & N. J. Keuthen (Eds.), Trichotillomania,
skin picking, and other body-focused repetitive behaviors. American Psychiatric
Publishing.
Gluhoski, V. L. (1995). A cognitive approach for treating trichotillomania. Journal of
Psychotherapy Practice and Research 4, 277–285.
Golomb, R., Franklin, M., Grant, J. E., Keuthen, N. J., Mansueto, C. S., Mouton-Odum, S.,
Novak, C., & Woods, D. (2011). Expert consensus treatment guidelines for trichotillomania, skin picking, and other body-focused repetitive behaviors. Scientific Advisory Board
of the Trichotillomania Learning Center.
Golomb, R. G., & Vavrichek, S. M. (2000). The hair pulling “habit” and you: How to solve the
trichotillomania puzzle. Writers’ Cooperative of Greater Washington.
Grant, J. E., Chamberlain, S. R., & Odlaug, B. L. (2012). Alternative treatments. In J. E.
Grant, D. J. Stein, D. Woods, & N. J. Keuthen (Eds.), Trichotillomania, skin picking, and
other body-focused repetitive behaviors. American Psychiatric Publishing.
Trichotillomania and Excoriation Disorder
Grant, J. E., Levine, L., Kim, D., & Potenza, M. N. (2005). Impulse control disorders in adult
psychiatric inpatients. American Journal of Psychiatry, 162(11), 2184–2188. doi:10.1176/
appi.ajp.162.11.2184
Grant, J. E., & Odlaug, B. L. (2009). The obsessive-compulsive spectrum and disorders of the
skin: A review. Expert Review of Dermatology, 4, 523–532.
Hayes, S. C., Strosahl, K., & Wilson. K. G. (1999). Acceptance and commitment therapy: An
experiential approach to behavior change. Guilford Press.
Kabat-Zinn, J., & Chapman-Waldrop, A. (1988). Compliance with an outpatient stress
reduction program: Rates and predictors of completion. Journal of Behavioral
Medicine, 11, 333–352.
Keijsers, G. P. J., Van Minnen, A., Hoogduin, C. A., Klaassen, B. N. W., Hendriks, M. J., &
Tanis-Jacobs, J. (2006). Behavioural treatment of trichotillomania: Two-year follow-up
results. Behaviour Research and Therapy, 44(3), 359–370. doi:10.1016/j.brat.2005.03.004
Keuthen, N. J., Aronowitz, B., Badenoch, J., & Wilhelm, S. 1999. Behavioural treatment for
trichotillomania. In D. J. Stein, G. A. Christenson, & E. Hollander (Eds.),
Trichotillomania. American Psychiatric Press.
Keuthen, N. J., Fraim, C., Deckersbach, T., Dougherty, D. D., Baer L., & Jenike, M. A. (2001a).
Longitudinal follow-up of naturalistic treatment outcome in patients with trichotillomania.
The Journal of Clinical Psychiatry, 62(2), 101–107. doi:10.4088/jcp.v62n0205
Keuthen, N. J., O’Sullivan, R. L., Ricciardi, J. N., Shera, D., Savage, C. R., Borgmann, A. S.,
Jenike, M. A., & Baer, L. (1995). The Massachusetts General Hospital (MGH)
Hairpulling Scale: 1. Development and factor analyses. Psychotherapy
and Psychosomatics, 64(3–4), 141–145.
Keuthen, N. J., Rothbaum, B. O., Welch, S. S., Taylor, C., Falkenstein, Martha, Heekin, M.,
Jordan, C. A., Timpano, K., Meunier, S., Fama, J., & Jenike, M. A. (2010). Pilot trial of
dialectical behavior therapy-enhanced habit reversal for trichotillomania. Depression
and Anxiety, 27(10), 953–959. doi:10.1002/da.20732
Keuthen, N. J., Wilhelm, S., Deckersbach, T., Engelhard, I. M., Forker, A. E., Baer L., &
Jenike, M. A. (2001b). The Skin Picking Scale: Scale construction and psychometric
analyses. Journal of Psychosomatic Research, 50(6), 337–341.
Lazarus, A. A. (1981). The practice of multimodal therapy. McGraw-Hill.
Lerner, J., Franklin, M. E., Meadows, E. A., Hembree, E., & Foa, E. B. (1998). Effectiveness
of a cognitive behavioral treatment program for trichotillomania: An uncontrolled
evaluation. Behavior Therapy, 29(1), 157–171. doi:10.1016/s0005-7894(98)80036-1
Linehan, M. M. (1993). Cognitive behavioral treatment of borderline personality disorder.
Guilford Press.
Mansueto, C. S. (1990). Typography and phenomenology of trichotillomania. Paper presented at meeting of the Association for the Advancement of Behavior Therapy, San
Francisco, CA, November.
(1991). Trichotillomania in focus. OCD Newsletter of the Obsessive Compulsive
Foundation, 5(3), 10–11.
Mansueto, C. S., Goldfinger-Golomb, R., McCombs-Thomas, A., & Townsley-Stemberger,
R. M. (1999). A comprehensive model for behavioral treatment of trichotillomania.
Cognitive and Behavioral Practice, 6(1), 23–43. doi:10.1016/s1077-7229(99)80038-8
Mansueto, C. S., & Rogers, K. E. (2012). Trichotillomania: Epidemiology and clinical characteristics. In J. E. Grant, D. J. Stein, D. Woods, & N. J. Keuthen (Eds.),
Trichotillomania, skin picking, and other body-focused repetitive behaviors. American
Psychiatric Publishing.
Mansueto, C. S., Townsley-Stemberger, R. M., McCombs-Thomas, A., & GoldfingerGolomb, R. (1997). Trichotillomania: A comprehensive behavioral model. Clinical
Psychology Review, 17(5), 567–577. doi:10.1016/s0272-7358(97)00028-7
339
340
Charles S. Mansueto
Mansueto, C. S., Vavricheck, S. M., & Golomb, R. G. (n.d.). Overcoming hair pulling and skin
picking for good: A self-help guide to comprehensive behavioral (ComB) treatment
[Unpublished manuscript].
McGuire, J. F., Ung, D., Selles, R. R., Rahman, O., Lewin, A. B., Murphy, T. K., & Storch, E.
A. (2014). Treating trichotillomania: A meta-analysis of treatment effects and moderators for behavior therapy and serotonin reuptake inhibitors. Journal of Psychiatric
Research 58, 76–83. doi:10.1016/j.jpsychires.2014.07.015
Miltenberger, R. G., & Fuqua, R. W. (1985). A comparison of contingent vs non-contingent
competing response practice in the treatment of nervous habits. Journal of Behavior
Therapy and Experimental Psychiatry, 16(3 ),195–200. doi:10.1016/0005-7916(85)90063-1
Mouton, S. G., & Stanley, M. A. (1996). Habit reversal training for trichotillomania: A group
approach. Cognitive and Behavioral Practice, 3(1), 159–182. doi:10.1016/s1077-7229(96)
80036-8
Mouton-Odum, S., Mansueto, C. S., & Golomb, R. G. (2013). Manual for comprehensive
behavioral treatment of trichotillomania [Unpublished manual].
Odlaug, B. L., & Grant, J. E. (2012). Pathological skin picking. In J. E. Grant, D. J. Stein, D.
Woods, & N. J. Keuthen (Eds.), Trichotillomania, skin picking, and other body-focused
repetitive behaviors. American Psychiatric Publishing.
O’Sullivan, R. L., Mansueto, C. S., Lerner, E. A., & Miguel, E. C. (2000). Characterization of
trichotillomania: A phenomenological model with clinical relevance to obsessivecompulsive spectrum disorders. Psychiatric Clinics of North America, 23(3), 587–604.
Rollnick, S., & Miller, W. (1995). What is motivational interviewing? Behavioural and
Cognitive Psychotherapy, 23, 325–334. doi:10.1017/S135246580001643X
Rosenbaum, M. S., & Ayllon, T. (1981). The habit-reversal technique in treating
trichotillomania. Behavior Therapy, 12(4), 473–481. doi:10.1016/s0005-7894(81)80085-8
Rothbaum, B. O. (1992). The behavioral treatment of trichotillomania. Behavioral
Psychotherapy, 20, 85–90.
Schumer, M. C., Bartley, C. A., & Bloch, M. H. (2016). Systematic review of pharmacological
and behavioral treatments for skin picking disorder. Journal of Clinical
Psychopharmacology, 36(2), 147–152. doi:10.1097/JCP.0000000000000462
Stemberger, R. M., Stein, D. J., & Mansueto, C. S. (2003). Behavioral and pharmacological
treatment of trichotillomania. Brief Treatment and Crisis Intervention, 3, 339–352.
Stemberger, R. M. T., Thomas, A. M., Mansueto, C. S., & Carter, J. G. (2000). Personal toll of
trichotillomania: Behavioral and interpersonal sequelae. Journal of Anxiety Disorders,
14(1), 97–104. doi:10.1016/S0887-6185(99)00028-6
Tarnowski, K. J., Rosén, L. A., McGrath, M. L., & Drabman, R. S. (1987). A modified habit
reversal procedure in a recalcitrant case of trichotillomania. Journal of Behavior Therapy
and Experimental Psychiatry, 18(2), 157–163. doi:10.1016/0005-7916(87)90030-9
van Minnen, A., Hoogduin, K. A. L., Keijsers, G. P. J., Hellenbrand, I., & Hendriks, G.-J.
(2003). Treatment of trichotillomania with behavioral therapy or fluoxetine. Archives of
General Psychiatry, 60(5), 517–522. doi:10.1001/archpsyc.60.5.517
Walsh, K. H., & McDougle, C. J. (2001). Trichotillomania: Presentation, etiology, diagnosis,
and therapy. American Journal of Clinical Dermatology 2, 327–333.
Woods, D. W., Wetterneck, C. T., & Flessner, C. A. (2006). A controlled evaluation of
acceptance and commitment therapy plus habit reversal for trichotillomania. Behaviour
Research and Therapy, 44(5), 639–656. doi:10.1016/j.brat.2005.05.006
17
Cognitive Behavioral Therapy for
Hoarding Disorder
Caitlin A. Stamatis, McKenzie K. Roddy,
and Kiara R. Timpano
Overview of Hoarding Disorder
The three hallmark characteristics of hoarding disorder (HD) include difficulties
discarding, excessive acquiring, and clutter (Frost & Hartl, 1996; Pertusa et al.,
2010). HD symptoms are dimensionally distributed (Timpano et al., 2013), ranging
from normative saving and collecting behaviors to extreme hoarding tendencies,
with approximately 1.5–3% of the population meeting criteria for HD (Nordsletten
et al., 2013; Timpano et al., 2011). In extreme cases, excess clutter hinders basic
activities of daily living and contributes to dangerous living conditions (Frost et al.,
2000), while acquiring tendencies can cause substantial monetary difficulties (Frost
et al., 2009; Mueller et al., 2007). Beyond the core features, HD is also associated
with high levels of comorbidity across both psychiatric (e.g., depression) and healthrelated (e.g., chronic pain) syndromes. Given these considerations, along with the
fact that hoarding-related impairments extend past the individual to include economic and societal burdens, HD has been identified as a significant public health
crisis (Tolin et al., 2008). Consequently, developing and refining therapeutic interventions for individuals with hoarding disorder constitutes a critical step in reducing
hoarding-related burden.
Case Vignette
Ron is a 71-year-old, single, Caucasian male who self-referred to at an outpatient
clinic for individual therapy to address symptoms of hoarding, which he stated had
worsened since losing his wife 10 years prior. He presented primarily with distress
around the amount of clutter in his two-bedroom house, as well as in his two storage
units. Ron also endorsed low mood, which he attributed to a combination of his
hoarding difficulties and perceived uncertainty about the future. Ron had recently
retired from working as an accountant, lost his house to foreclosure, and moved into
a smaller home owned by his son. Ron hoped to spend his retirement traveling and
spending time with his grandchildren. However, he felt “trapped” and “dirty” due to
the clutter, which also prevented him from having visitors in his home.
341
342
Stamatis, Roddy, and Timpano
Etiology of Hoarding
Understanding the etiological model for HD, including vulnerability and maintaining factors, is essential for accurately conceptualizing cases and executing treatment
plans. According to cognitive behavioral models of hoarding, risk for HD stems
from a combination of genetic and familial factors, information-processing deficits,
cognitive beliefs, emotional responses, and reinforcement learning. Research on
risk factors points to a strong heritable component, with approximately 50% of the
variance explained by genetic factors (Hirschtritt & Mathews, 2014; Iervolino et al.,
2009). Various candidate genes, particularly those regulating serotonergic function,
have been posited as relevant to hoarding (Hirschtritt & Mathews, 2014), though
conclusive findings are lacking. Family values and parenting behaviors have also
been found to influence hoarding risk (Landau et al., 2011), with studies indicating
that family beliefs about saving and wastefulness are commonly reported in adults
with HD (Gilliam & Tolin, 2010). Additionally, early life stress and trauma have
been linked with risk of hoarding behaviors (Cromer et al., 2007; Tolin et al., 2010).
Individual differences in personality traits (e.g., perfectionism, dependency, and
paranoia), along with transdiagnostic cognitive and emotional risk factors (e.g.,
distress tolerance, anxiety sensitivity), represent additional facets that may contribute to HD (Timpano et al., 2011).
Patients with HD often exhibit patterns of cognitive impairment, which are
theorized to reflect a more general information processing risk that may be present
prior to the onset of clinically significant hoarding symptoms (Woody et al., 2014).
Deficits in executive function, attention, and memory constitute the primary
information-processing abnormalities observed in and reported by patients with
HD (Morein-Zamir et al., 2014). Patients with HD often display symptoms of
attention-deficit/hyperactivity disorder (ADHD), including tangentiality and difficulty sustaining attention, which may interfere with sorting and discarding processes
(Tolin & Villavicencio, 2011; Weintraub et al., 2018). Difficulties with categorizing
objects and, in particular, difficulties with making decisions have also been associated with the core features of HD. Finally, while overall memory deficits are not
observed in HD, patients with hoarding report poorer confidence in their memory,
along with greater reliance on visual memory cues (Hartl et al., 2004). Jointly, these
findings point toward the notion that HD is associated with a general deficit in selfregulatory abilities (Timpano & Schmidt, 2013), a hypothesis supported by
emerging neuroimaging research on HD (Slyne & Tolin, 2014). The handful of
extant neuroimaging studies on HD indicate that saving tendencies are linked with
abnormalities in cognitive and affective brain circuits, including the orbitofrontal
cortex, dorsal anterior cingulate cortex, and temporal regions (Mataix-Cols et al.,
2004; Saxena et al., 2004; Tolin et al., 2009, 2012).
Along with the aforementioned cognitive and biological risk factors, core
beliefs contributing to hoarding symptoms include cognitions about the self, possessions, responsibility, memory, and control (Frost & Hartl, 1996). Individuals at risk
for HD may report greater valuation of possessions in terms of instrumental value,
CBT for Hoarding Disorder
intrinsic beauty, sentimental value, and comfort (Frost et al., 1995; Grisham et al.,
2009). In the same way as individuals with depressive and anxiety spectrum disorders, patients with hoarding often report negative beliefs about the self, including
a sense of vulnerability and helplessness, along with negative thoughts about likability and social relationships. More unique to hoarding pathology are thoughts
relating to saving objects, including beliefs about memory (e.g., “This object tells a
story”), responsibility (e.g., “I am responsible for this possession”), and control
(e.g., “I must ensure that things are sorted the right way”; Frost et al., 1998;
Steketee et al., 2003). Similarly, core beliefs about control, safety, and opportunity
have been linked with excessive acquiring (Kyrios et al., 2004). Negative beliefs in
hoarding are strengthened through a combination of positive and negative
reinforcement loops, which contribute to the core symptoms of excessive acquiring,
difficulty discarding, and clutter. For example, discarding will invariably elicit strong
negative emotions for individuals with hoarding (e.g., distress, anger, grief ), and
patients will subsequently be less likely to actually discard in order to avoid these
emotions. In contrast, the positive emotions experienced when acquiring new
objects can reinforce collecting behaviors.
Assessment
Assessment for HD covers five major symptom categories: difficulties discarding
and acquiring, clutter, problems of living, saving cognitions, and comorbid conditions (Frost & Hristova, 2011). In addition to helping define the severity of hoarding
for a particular patient, the assessment process represents a key step in conceptualizing the case. Throughout the assessment and case conceptualization process, it is
helpful to keep the general etiological model of hoarding in mind (see Figure 17.1)
and to begin mapping the patient’s own experiences and relevant components onto
the model. The different assessment categories described below, along with relevant
measures for each category, are intended to help clinicians map out an individual
patient’s idiographic model of hoarding (see Figure 17.2 for a blank copy of
the model).
The assessment process with patients with HD is complicated by several factors
that deserve separate consideration. First and foremost is the issues present by
variable levels of insight and motivation. For example, patients may minimize
reports of clutter for a number of distinct reasons: one patient could underreport
impairment due to clutter because she views her possessions as positive and egosyntonic, whereas a second may minimize reports of clutter due to shame or
embarrassment. By using mutual language to describe hoarding behaviors and
possessions, the clinician may increase transparency of reporting. It is particularly
important to avoid the use of judgmental language (e.g., “junk”) and to help reduce
the impact of stigma the patient may be experiencing, including self-stigma. For
example, if a client refers to the clutter in their home as their “collection of
treasures,” the therapist might use the phrase “collection” during the assessment
phase to reinforce the no-judgmental stance. During the treatment phase, the
343
344
Stamatis, Roddy, and Timpano
Environment
Indecision
Depression
and Anxiety
Stressful
Life Events
Core
Vulnerabilities
and Comorbidity
Genetic
Factors
Selfreferential
beliefs
Neurobiology
Coping
InformationProcessing
Deficits
Object
Intrinsic
valuation
Emo-tion-al-ly
Cognitive
Styles and
Beliefs
Individual
Differences
Object
Attachment
Poor
planning
Memory
Object
Instrumental
valuation
Problemsolving
deficits
Organization
difficulties
Attention
deficits
Interpersonal
Attachment
Emotional Responses
and Processes
Positive
Reinforcement
Negative
Reinforcement
· Positive Emotions · Negative Emotions
· Approach behaviors · Avoidance Behaviors
Acquiring
Difficulties
Discarding
Clutter
Figure 17.1. Etiological model of hoarding disorder.
therapist would then help the client understand that their “collection” is the result of
HD. Another example might be the term used to describe HD. If a patient is more
comfortable with being called a “pack rat” or a “collector,” then these terms should
be used by the therapist.
A second issue to consider is that the average treatment-seeking patient with
HD is likely to be an older adult. Consequently, clinicians should be prepared for
the assessment process to take more time and potentially require special accommodations. The American Psychological Association’s “Guidelines for Psychological
Practices with Older Adults” represents a helpful framework for managing additional issues that may arise during assessment with this population (American
Psychological Association, 2014).
A third consideration is that family members may be directly or indirectly
impacted by the HD symptoms. It is not uncommon for family members to be a
driving force behind treatment-seeking behavior, which may reflect the patient’s
ambivalence about changing. Sometimes family members can be helpful in providing a more objective assessment of the hoarding symptoms and associated
CBT for Hoarding Disorder
345
Memory
Core
Vulnerabilities
and Comorbidity
InformationProcessing
Deficits
Cognitive
Styles and
Beliefs
Approach Behaviors
·
·
·
·
Emotional Responses
and Processes
Positive
Reinforcement
· Positive Emotions • Negative Emotions
·
·
·
·
Acquiring
SIR A =
CAS =
Negative
Reinforcement
Difficulties
Discarding
SIR D =
HRS =
Avoidance Behaviors
·
·
·
·
Clutter
SIR C =
CIR =
Figure 17.2. Blank copy of hoarding case conceptualization model.
difficulties. At the same time, the family may also be experiencing extremely high
levels of discord (e.g., arguments, criticism, and rejection) relating to the patient’s
hoarding behaviors (Tolin et al., 2008). Final consideration for completing an
assessment with hoarding patients is that the level of clutter may present true safety
risks for the patient and anyone living in the hoarded home. Inasmuch as the
impacted individuals include children or elderly individuals, relevant reporting laws
may have to be taken into account.
Symptom Severity
Symptom severity of HD should be assessed at intake and throughout treatment to
monitor progress. The Saving Inventory-Revised (SI-R; Frost et al., 2004) is a 23item self-report measure that divides into three subscales: difficulty discarding,
acquiring, and clutter. The SI-R is both reliable and valid within a hoarding population and demonstrates discriminative validity between hoarding and other mood
symptoms. A cutoff total score of 41 is associated with probable diagnosis, with
62 representing the average score for patients with HD and 24 representing the
346
Stamatis, Roddy, and Timpano
average score for individuals without HD (Frost et al., 2004; Muroff et al., 2014;
Tolin et al., 2011).
The Hoarding Rating Scale (HRS; Tolin et al., 2013) is an additional severity
instrument that is ideal for ongoing monitoring. The HRS can be used either as a
clinician-administered interview or as a self-report measure. This scale is commonly
used in community settings due to its high validity and brief format, with five items
rated on a Likert-type scale from no problem (0) to extreme difficulties (8). The five
items assess the main features of HD, including difficulty discarding, acquisition,
clutter, functional impairment, and distress. Of note, a client must score at or above
a 4 on the items measuring clutter, difficulty discarding, and either distress or
functional impairment in order to meet criteria for HD. A total score of 14 or
greater is indicative of clinical levels of HD (Tolin et al., 2010).
Clutter
As previously discussed, clients often struggle to accurately estimate the amount of
clutter in their homes. A helpful tool for more accurately assessing clutter severity is
the Clutter Image Rating Scale (CIR; Frost et al., 2008). The CIR provides nine
photographs of a bedroom, kitchen, and living room, each with increasing amounts
of clutter (rated 1 to 9, with 4 considered the clinical cutoff ). Clients are instructed
to view the pictures and rate from 1 to 9 the amount of clutter for each room in their
house is that is considered their domain. Using the general living room photo from
the CIR, clinicians should also assess storage areas such as garages, basements,
storage units, attics, and any additional rooms in the home. It is helpful to emphasize
that clients should rate the volume of clutter rather than the content, which may
look very different from the items pictured. In general, ratings should be based only
on areas that are fully under the client’s control, as spouses or family members will
often limit or control the amount of clutter in shared spaces. The CIR is especially
informative when used in conjunction with photos of the client’s home or an actual
visit to the home, which allows clinicians to consider both client perceptions and
their own clinical ratings of available data. To facilitate this process, clinicians may
ask patients to print photos of their living spaces prior to the assessment session, or
to bring in photos on their phone.
Acquiring
Although the SIR provides a nice snapshot of the severity of any acquiring behavior, it may be necessary to include a more detailed assessment of the types of
acquiring that a patient may be doing (Frost et al., 2009). While the most common
method of acquisition is purchasing items (active acquiring), other objects may be
acquired by collecting free items (e.g., giveaways or items discarded by others). The
Compulsive Acquisition Scale (CAS; Frost et al., 2002) includes two subscales that
capture buying behaviors and the excessive acquisition of free objects. A cutoff of
48 has been found to distinguish those with clinically significant collecting behaviors
from normative acquiring tendencies (Frost et al., 2009). A final consideration is
that some individuals with HD may also endorse stealing objects (e.g., taking
CBT for Hoarding Disorder
magazines from waiting rooms or a roll of toilet paper from work) as a means of
acquiring (Timpano et al., 2011). Although this form of acquiring appears to be rare,
clinicians should be aware of the possibility (Frost et al., 2013).
Problems of Living
Clinical levels of clutter can interfere with necessary tasks (e.g., bills get lost and go
unpaid) and restrict functionality of the home (e.g., cluttered furniture prevents
cooking in the kitchen or bathing in the shower). In more extreme cases, clutter can
obstruct pathways or entire rooms, creating fire and safety hazards; consequently,
problems of living represent a crucial area of consideration during assessment of HD
symptoms. Along with patient and family member reports of problems of living,
professionals in the community who interact with patients with HD (e.g., social
workers and firefighters) may provide valuable insight into the severity of HD-related
interference in daily life. The HOMES Multidisciplinary Risk Assessment (HOMES;
Bratiotis, 2011) was designed as in inclusive measure of assessing HD-associated
problems of living. The checklist is a straightforward means of assessing health, mental
health, obstacles, endangerment, and structure and safety. For a more in-depth view,
the Activities of Daily Living for Hoarding (ADL-H; reported in Grisham et al., 2006)
is a self-report scale that assesses the extent to which HD symptoms interfere with
everyday activities such as bathing, preparing meals, and accomplishing daily tasks.
The ADL-H contains 16 items assessing activities of living, as well as seven items
assessing quality of living conditions and six items assessing health and safety.
Saving Cognitions
A measure of saving cognitions is particularly useful for identifying problematic
thinking patterns that can be addressed during cognitive behavioral therapy (CBT)
for HD. The Savings Cognitions Inventory (SCI; Steketee et al., 2003) is a 23-item
scale that consists of four subscales regarding beliefs about saving. Specifically, the
subscales measure desire for control over items, responsibility for possessions,
emotional attachment, and concerns about memory. Clinicians should additionally
assess for interpersonal or attachment difficulties, along with patient coping styles
and emotionality, all of which can shape an individual’s manifestation of HD.
Comorbid Conditions
In addition to hoarding-specific symptomology, it is important to complete a thorough intake assessment and identify any comorbid conditions. Depressive symptoms, social anxiety, generalized anxiety, and ADHD are all commonly comorbid
with HD and can interfere with treatment if not properly addressed (Frost et al.,
2011; Hall et al., 2013). Additionally, although HD is classified by DSM-5 as an
obsessive-compulsive spectrum disorder and a diagnosis separate from OCD, individuals with comorbid OCD with contamination concerns may struggle with components of HD treatment such as allowing assessors or therapists into their home.
Understanding how additional diagnoses contribute to individual vulnerabilities is
critical to case conceptualization, discussed below.
347
348
Stamatis, Roddy, and Timpano
Case Vignette: Assessment
During Ron’s intake session, the therapist conducted the Structured Clinical
Interview for DSM-5 (SCID-5) (First & Williams, 2016), with the goal of gaining
insight into Ron’s hoarding and depressive symptoms, as well as ruling out OCD
given his comments about the “dirty” feeling surrounding clutter. Based on the
SCID-5, Ron met criteria for hoarding disorder, as well as major depressive disorder, mild, single episode. His concerns about dirt relating to the clutter were
attributable to the dust and mold growing in his storage units, and he denied
ritualistic behaviors relating to these concerns, indicating that they were unlikely
to represent a symptom of OCD but rather represented realistic appraisals of
hoarding-related consequences. Ron did not endorse any excessive acquiring
behaviors.
Various hoarding-specific measures were utilized to obtain a more comprehensive picture of Ron’s condition. From the HRS, Ron’s symptoms appeared to be on
the more severe end (total score = 34/40), which was supported by his ratings of
6 and higher on the CIR of photos of his bedroom, which he considered his personal
domain in the home. Ron’s primary reasons for saving, gleaned from the SCI, were
generally related to memory and responsibility. This information was used to
conceptualize Ron’s case (Figure 17.3), which was subsequently used for treatment
planning. Ron had very few acquiring behaviors, thus treatment was focused on his
difficulty discarding. Additionally, we did not assess for problems with living (e.g.,
HOMES) because Ron functioned successfully in the professional sphere for many
years, and was currently living with his son.
Overview of Available Treatments for Hoarding Disorder
Given the complexity of both the clinical characteristics and associated features of
HD, it is perhaps not surprising that hoarding has historically been acknowledged
as a difficult treatment target (Abramowitz et al., 2003; Bloch et al., 2014; Pertusa
et al., 2010). In addition to heterogeneous presentations and complex comorbidity
patterns, clients with HD often exhibit low insight and motivation. Furthermore,
very few clinicians are trained to accurately assess and diagnose HD or provide
effective treatments. In spite of these challenges, a handful of empirically supported treatments have been developed and tested to treat hoarding disorder. In
particular, since the addition of hoarding disorder as an independent diagnostic
entity to the fifth edition of the Diagnostic and Statistical Manual of Mental
Disorders (DSM-5; American Psychiatric Association, 2013), the literature on
treating hoarding independent of OCD has grown substantially (Timpano et al.,
2016; Tolin et al., 2015). The best-supported form of HD treatment is CBT for
hoarding, which is discussed in detail in a later section. Immediately below, we
briefly summarize alternative treatment modalities applied to HD and the current
evidence for their efficacy.
CBT for Hoarding Disorder
349
Recent
widower
Decisionmaking
difficulties
Depression
Core
Vulnerabilities
and Comorbidity
Pressure
from
chilldren
Lost home
to
foreclosure
Memory
Responsibility
Cognitive
Styles and
Beliefs
Recent
retirement
Need to
control
objects
InformationProcessing
Deficits
Fear of
regret
Poor
planning
Instrumental
value
Avoidance Behaviors
Emotional Responses
and Processes
Approach Behaviors
anything tied with
fond memories
· Souvenirs from travels
· Unproductive churning
· Saving
Positive
Negative
Reinforcement Reinforcement
· Positive Emotions · Negative Emotions
Pleasure; joy;
Sadness; anxiety
nostalgia
Acquiring
Difficulties
Discarding
Clutter
SIR A = 9
CAS = 24
SIR D = 26
HRS = 34/40
SIR C = 24
CIR = 6
· Decisions about objects
· No planning for future
· Multiple (unnecessary)
storage units
· Lack of follow-through
· Avoidance of certain stores
Figure 17.3. Ron’s case conceptualization.
Pharmacological Treatments
A limited number of pharmacological interventions have been tested as a treatment
for hoarding with and without comorbid OCD (Timpano et al., 2016). Results from a
recent meta-analysis indicate that selective serotonin reuptake inhibitors (SSRIs)
(specifically, paroxetine and sertraline), along with the serotonin and norepinephrine
reuptake inhibitor (SSNRI) venlafaxine, are the most well-supported pharmacologic
treatments for HD (Brakoulias et al., 2015). More limited evidence exists regarding
the efficacy of medications indicated for the treatment of ADHD, such as methylphenidate and atomoxetine (Grassi et al., 2016; Rodriguez et al., 2013), to target
deficits in attention and executive function frequently observed in HD. A larger issue
relevant for using pharmacotherapy to treat HD, which is documented in several case
studies, is a general reluctance of patients with HD to pursue medications.
Family Treatments
Given the influence of the patient’s clutter, saving, and acquiring on the household,
hoarding tendencies can have a secondary, albeit significant, negative impact on
Attentional
focus
350
Stamatis, Roddy, and Timpano
family members, who in turn may be involved in managing or accommodating
patient behaviors. Family-focused interventions for hoarding may constitute a particularly helpful alternative treatment when patients with HD exhibit poor motivation or insight (Tompkins & Hartl, 2013). Components of family therapy for HD
include psychoeducation about HD, harm reduction techniques, and motivational
interviewing skills. In addition, relatives are taught about the role of family accommodation in maintaining HD symptoms, as well as ways of reducing accommodation
(Chasson et al., 2014).
Community-Focused Interventions
HD may confer a number of problems impacting the community, including potential
evictions, fire and health hazards, and suspected cases of neglect. Accordingly,
mental health professionals must often collaborate with other service providers
and professionals enforcing health and safety codes to best serve a client with
HD. Various models have been developed to characterize coordinated service
intervention for HD (Bratiotis & Woody, 2014). Community-based models range
from informal partnerships to formalized hoarding task forces, which function in the
areas of case consultation and treatment, while also pursuing policy change and
advocacy work (Bratiotis, 2013). Poised to tackle a unique challenge among psychological treatments, community interventions for HD must balance the needs of
the patient (e.g., manage housing issues, retaining control over decision-making
regarding possessions) and of the broader community (e.g., reducing safety risks
and sanitation issues, promoting compliance with local laws). For the clinician, this
may involve implementing shorter-term harm-reduction strategies, which aim to
reduce clutter to a level that is no longer an active safety concern, prior to beginning
longer-term work on core hoarding symptoms such as difficulty discarding
(Tompkins, 2015).
Cognitive-Behavioral Therapy for Hoarding
Evidence Basis for CBT for HD
As previously indicated, CBT for hoarding disorder boasts the greatest empirical
support. CBT for hoarding was created to provide therapists with a manualized,
skills-based approach to treating individual clients with HD (Steketee et al., 2000).
The specialized protocol for hoarding has since been adapted and tested in group
format, with a growing number of clinical trials supporting the efficacy of group
CBT for HD (Gilliam et al., 2011; Muroff et al., 2009, 2012; Steketee et al., 2010).
Additionally, CBT for HD has been adapted for special populations, including
group CBT for older adult clients (Ayers et al., 2011). There have also been versions
of the treatment created in self-help and web-based formats (Frost et al., 2012;
Muroff et al., 2010; Tolin et al., 2013), which provide a promising means of targeting
HD symptoms in areas with limited access to specialized service providers.
CBT for Hoarding Disorder
To date, the data from empirical studies on CBT for HD are promising, with
certain aspects of HD showing greater response to treatment. In two important
translational studies, CBT for hoarding was tested in more naturalistic, real-world
settings (Mathews et al., 2016; Moulding et al., 2016), and both of these naturalistic
studies yielded significant HD symptom reductions. A recent meta-analysis revealed
that CBT produced the greatest reductions in difficulty discarding (Tolin et al.,
2015), which is promising given that this is the core feature of HD. However, the
same meta-analysis indicated that clutter and excess acquiring displayed more
muted responses to intervention. Of note, longer treatment and frequency of home
visits predicted better clinical outcomes. Taken as a whole, the current treatment
outcome literature on HD indicates that while overall evidence of CBT for HD is
positive, rates of clinically significant change tend to be modest, such that many
patients continue to exhibit clinically significant HD symptoms on termination
(Tolin et al., 2015).
Overview of Treatment Components
Individual CBT for hoarding disorder involves an average of six hour-long sessions,
depending on the client’s specific needs and range of symptoms (Muroff et al., 2014;
Steketee et al., 2010). Delivered in group format, CBT for hoarding can involve
between 12 and 20 sessions (Gilliam et al., 2011; Muroff et al., 2009, 2012), with
protocols for use in geriatric populations having an average length of 24 sessions
(Ayers et al., 2011, 2014. Manualized self-help and peer support groups are typically
15 weeks in length. For telehealth-based CBT for hoarding, which allows therapists
to provide treatment via technologies such as phone or video calls rather than in
person, the number of sessions is variable (Muroff et al., 2010).
Cognitive behavioral treatment begins with detailed assessment of HD symptoms, as well as relevant comorbidities and any safety concerns to aid in case
conceptualization and treatment planning (Muroff & Underwood, 2016). As discussed above, the assessment phase should also be used to begin mapping a
particular patient’s specific experiences onto the general CBT conceptualization of
hoarding, to allow for more specific treatment planning (see Figure 17.3 for a patient
example). During the initial treatment modules, the clinician provides psychoeducation on HD etiology and treatment with CBT, with patients setting realistic treatment goals. Next, patients learn skills for problem-solving, organizing, and making
decisions. The bulk of treatment focuses on the use of behavioral techniques and
cognitive interventions to reduce saving and acquiring behaviors and beliefs and to
increase sorting and organizational practices and discarding behaviors. The balance
between behavioral and cognitive techniques, as well as the order in which they are
introduced and used throughout treatment, may differ across clients, depending on
motivational factors, the rigidity of saving beliefs, and other considerations (e.g.,
safety concerns may prioritize behavioral interventions). Sessions can take place
exclusively within the therapist’s office, by having clients bring belongings into
session; however, home visits are typically helpful and sometimes necessary. In
351
352
Stamatis, Roddy, and Timpano
the event that sessions take place only in the office, it is recommended that clients
do not sort any of the objects prior to coming into session, in an effort to reproduce
a sorting experience as naturalistic as possible. For clients with greater difficulties
with acquiring, therapists may also want to incorporate nonacquiring “shopping”
exposures. These nonacquiring exposures involve trips to specific stores (e.g., bigbox stores) or events (e.g., yard sales), and involve exposure to objects without
actually collecting any items. Throughout treatment, therapists help clients to foster
alternative pleasurable activities (e.g., social activities) and utilize motivational
interviewing techniques to enhance treatment adherence. Treatment wraps up with
several relapse prevention sessions. Below, we describe the core treatment modules
implemented across all CBT for hoarding treatments (Timpano et al., 2016; Tolin
et al., 2015).
Key Interventions
Psychoeducation
Following assessment and case conceptualization, treatment starts with psychoeducation about HD, as well as comorbid symptoms as relevant to the individual client.
The therapist provides the client with an overview of the CBT model of HD,
integrating the client’s specific symptoms of difficulty discarding, clutter, and excessive acquiring, as well as distress and impairment, into the description. Risk and
vulnerability factors for HD are shared and discussed, which may yield additional
information relevant to treatment while helping the client to better understand the
relationships between the thoughts, emotions, and behaviors that have contributed
to their hoarding symptoms. Throughout this module, it is important to address
questions and elicit client feedback on extent to which the CBT model resonates
with the client’s experience of hoarding symptoms, as well as ways in which these
symptoms engender distress and impairment. By accruing concrete information
about client symptoms, the therapist may present the intervention plan in a manner
that directly addresses the client’s specific concerns. For example, when discussing
the course of HD, clients will often report that they noticed a propensity to save or
collect early in life, even though they may not have come in for treatment until much
later, when the clutter has become unmanageable or when social or housing concerns have arisen. The therapist may use this information in describing the CBT
triangle, pointing out that therapy will address the underlying cognitive factors (e.g.,
early beliefs about saving), while also targeting the problem behaviors (e.g., difficulty discarding) in order to reduce negative emotion and impairing clutter.
Motivational Interviewing (MI)
By integrating techniques from motivational interviewing (MI) during early sessions, the therapist targets common barriers to treatment (described in detail below)
that are likely to arise during later sessions. As early as the diagnostic feedback and
psychoeducation sessions, the therapist may adopt an ask-tell-ask-reflect style to
elicit “change talk” from the client. Key strategies from MI that aid in eliciting
CBT for Hoarding Disorder
motivational factors for a given client include reflective listening, asking open-ended
questions, and generating pros and cons to encourage behavioral change. For
example, “You just really love your belongings, and at the same time, reducing
clutter would allow you to have your grandchildren come over. What are some
other ways life would be different if you reduced the clutter in your home?” Clients
may create a decisional balance, and therapists can use change rulers to elicit
additional change talk (Miller & Rollnick, 2012). The use of metaphors and
examples, which can be personalized to the individual, can help to further assist
clients with HD in identifying motivational factors. Jointly, these techniques are
aimed at helping the patient consider their symptoms and the resulting consequences more objectively and to encourage change by having the patient selfgenerate both positive and negative facets and outcomes of their behavior and
how this may or may not line up with their treatment goals. Given that motivational
lapses are fairly common among clients with HD, therapists may pull from and
adapt MI strategies to hoarding as needed throughout the course of therapy.
Skills Training
Having agreed on a treatment plan with the client and established motivations for
behavioral change, the therapist and client move to the skills training modules. As
previously described, individuals with HD often exhibit and report deficits in
planning, organizing, and categorizing objects. Consequently, teaching clients concrete skills prior to beginning the behavioral components of treatment helps to
reduce potential barriers. The ultimate goal of skills training is for clinicians to
equip clients with specific skills that will be essential to successful sorting and
discarding exposures during the next phase of treatment. Through the core components of skills training, the therapist will teach problem-solving steps and discuss
the importance of planning prior to acting. For instance, clients learn the technique
of linking important items (e.g., keys, wallet) with appliances used frequently (e.g.,
coffeemaker). Clients also learn to prioritize important tasks and divide projects
into manageable steps, an important skill to make later discarding sessions more
manageable. Given the attention difficulties that many patients experience, regardless of whether they meet criteria for ADHD or not, it is often helpful to provide
skills to manage distractions. For example, clients may be assisted in setting up a
calendar system to manage appointments and daily activities, using timers to guide
sorting sessions, and creating staging areas to remain focused on sorting the specific
items at hand. During the skills training module, the therapist also introduces the
use of decision trees in dealing with objects, which involve establishing a hierarchy
of decisions (e.g., save or discard?), as well as stepwise contingencies based on those
decisions (e.g., If discard, what category does it belong to? Options: trash, recycling,
donations) and specific instructions to handle objects within each category (e.g., If
donation, place in donation bin and identify plan for removing donation bin at the
end of the sorting session). Additional skills covered in this module include identifying categories that will be used in later sorting, and establishing guidelines for the
353
354
Stamatis, Roddy, and Timpano
length of time items need to be saved (e.g., tax documents should be saved for seven
years and can then be discarded).
Behavioral Experiments and Exposures
Across the various CBT protocols for HD, the majority of sessions (at least 10) are
devoted to sorting and discarding exposures, as well as nonacquiring exposures for
clients who endorse acquiring symptoms (Ayers et al., 2011; Muroff et al., 2014;
Steketee & Frost, 2013). Although discarding and nonacquiring exposures share
many features with traditional exposures for anxiety disorders, conducting exposure
therapy with patients with HD also presents unique challenges. Often, discarding
exposures lead to discussions of distress tolerance to the uncomfortable and challenging process of making decisions about objects. Given these challenges, structure
is key to promoting productive exposure sessions, whether it is in the office, during
home visits, or while the clients are practicing sorting on their own. Prior to
beginning exposures, rules and guidelines discussed during the skills training
modules are refreshed, including the establishment of categories for sorting and
the “only handle it once” (OHIO) rule. It is critical to assist the client in creating
schedules for discarding sessions at home. The therapist will provide the client with
support in establishing a reminder system, which may include smartphones or an
easily carried paper-and-pencil planner, and in generally maximizing the feasibility
of the discarding plan. The client is encouraged to determine a final “home” for
each item sorted during a session, with an emphasis on saving enough time during
each exposure to ensure that all items have a proper home. Interim “staging areas”
can be created for use during exposure sessions, and the sorting area should be clear
and organized in order to facilitate following the OHIO rule. To provide additional
structure during sorting, clients may find it helpful to hang up a copy of the sorting
rules (see Table 17.1 for example), as well as a list of questions to help make
decisions about objects, which are introduced during the cognitive techniques
module (see below).
In order to promote success in discarding, the therapist must provide the patient
with significant preparation prior to beginning in-session sorting. The first component of exposures for HD involves providing the client with psychoeducation and
rationale. The therapist highlights the role of avoidance in maintaining hoarding
and anxiety symptoms, also teaching the client about the process of habituation. It is
important to make use of graphs and figures to emphasize that habituation is not a
linear process, but rather that we expect ups and downs within an overall downward
trend in anxiety. When the client has exhibited an understanding of the reasoning
behind exposure therapy and a willingness to engage with the intervention, the next
step is to create a hierarchy. The therapist aids the client in brainstorming a variety
of items and situations that increase in difficulty. It is generally useful to provide the
client with sample anchors. For example, “A zero on my hierarchy might be getting
rid of expired food, whereas a ten for me would be sorting through my grandfather’s
belongings after he recently passed away. What would a zero look like for you?
A ten?” The therapist will emphasize that exposure therapy starts with an item or
CBT for Hoarding Disorder
355
Table 17.1. Example sorting: Rules for Rona
Sorting papers
Sorting books
If dated pre-2000 ! discard
If tax related, keep anything since 2014
Newspaper/magazine clippings
Recipes ! save
Past travel related ! discard
Future travel related ! keep
Notes/handwritten lists ! Ron decide
Instructions/manuals ! keep
Documentation of art ! keep
Paid bills older than four years ! shred and
discard
Bank statements older than three years ! shred
and discard
Accounting books ! keep
Nature books
Picture books ! donate
Natural history books ! keep
Fiction books ! Ron sort
If read ! donate
First edition books ! keep
Children’s books ! daughter decides
Old (dated) textbooks ! donate
Sorting objects
If old (dated) and unused ! donate
Any art or artifacts ! keep
Related to travel ! keep in travel box
Photos ! keep
For other objects ! make best guess what is
useful for Ron
a
Ron and therapist developed these sorting rules collaboratively during session. He practiced sorting with therapist
and volunteer before using rules with family when they came to town.
situation that is level three in difficulty, and that over time more difficult situations
will be tackled. Clients will begin bringing items into session to practice discarding
exposure techniques with the therapist, who then assists the client in structuring athome exposure sessions. In session, the therapist will aid the client in asking
questions and referring to decision trees about objects to help make decisions about
the right “home” for a given item. Clients learn to rate their Subjective Units of
Distress (SUDs) every five minutes throughout an exposure session, an important
self-monitoring skill to understand emotional responses to decisions about objects,
and to highlight the natural reduction in distress through habituation over time. In
more advanced exposure sessions, clients may begin to set time goals for making
decisions about objects or set a priori goals for the proportion of objects to be
discarded from a given pile.
Nonacquiring exposures are typically conducted during later sessions as needed
and after a separate hierarchy has been established. To promote nonacquiring,
clients are encouraged to reflect on triggers of compulsive shopping or acquiring,
and to establish clear acquiring rules. Depending on the client’s acquiring tendencies, the therapist will work with the client to establish plans for nonacquiring
shopping exposures. Starting with a difficulty level of three on their nonacquiring
hierarchy, clients will go to a specific store or event with the goal of not purchasing
or picking up free items, rating their SUDs throughout. During the exposures,
clients learn to rate their SUDs over time, and to ask questions about objects that
will help inform their decision to acquire (see following section on cognitive
356
Stamatis, Roddy, and Timpano
techniques). Moreover, the therapist will work together with the client to identify
alternative sources of coping and pleasurable activities that the client can perform
instead of acquiring or when urges to acquire surface. Given the role of positive
emotionality in HD, it is critical to promote engagement in adaptive pleasurable
activities as the client reduces their acquiring.
Cognitive Techniques
The first step in introducing cognitive techniques is for the therapist and client to
identify cognitive errors and related core beliefs that influence current symptoms.
Clients are assigned behavioral monitoring exercises between sessions, which may
include a thought record specific to emotional experiences about objects (e.g.,
Steketee & Frost, 2013). During sessions, the therapist makes use of both general
cognitive strategies and others that are specifically targeted for hoarding. General
cognitive techniques include Socratic questioning and asking for evidence, using the
downward arrow technique to help identify core beliefs, focusing on perfectionism
as a continuum, considering advantages and disadvantages, and honing in on costs
of saving items and benefits of discarding (Steketee & Frost, 2013). The hoardingspecific strategies focus more directly on questions about keeping versus discarding
possessions (Ayers et al., 2011; Muroff et al., 2014; Steketee & Frost, 2013).
Questions such as “How many do I already have? How many would be enough?
Do I have a specific plan to use this item within a reasonable timeframe? Would
I die without it?” can be crucial in assisting patients in clarifying motivating factors
for discarding, as well as the next steps in a sorting exercise. In an additional
cognitive strategy, the therapist role-plays the client perspective, asking the client
to play the role of a third person questioning the therapist about decisions to keep
or discard items. As in the motivational interviewing module, metaphors and stories
serve to enhance the introduction of cognitive techniques. For example, while
helping clients to grapple with the subjectivity of “needs” versus “wants,” the
therapist may share a version of the following:
Let’s take the example of going to the beach. Is this a “want” or a “need”? [Client(s) will
say want]. Okay, it seems like a want – and maybe for me, right now, going to the beach is
a want rather than a need. But let’s say I’m very depressed and overworked, and I’ve
been having trouble taking pleasure in the activities I usually enjoy, such as going to the
beach. In this case, would taking some time to go to the beach feel more like a want or
a need for me? [Need]. The same thing applies to objects – that is, the same item could be
a want for one person and a need for another person, or even a want for one person at a
certain time in their life and a need for that same person at a different time in their life.
A final cognitive strategy that can be extremely powerful involves behavioral
experiments that test certain beliefs patients have related to objects, their ability to
cope without the objects, and their assumptions about what will happen should they
no longer own an object. For example, clients commonly assert the belief that they
would feel immense distress if they no longer had certain possessions. As a behavioral experiment, a client may test the strength of the distress they believe they
CBT for Hoarding Disorder
would feel living without one specific item. To do so, the client would give this
possession (e.g., a set of magazines from several years prior) to the therapist,
with the agreement that if after one month the client still feels they need the
object, it will be returned; otherwise, it will be discarded. The client would be
instructed to rate on a 1–10 scale the extent to which they feel distressed about
not having the object each week. Clients typically report minimal distress and often
forget that the therapist even has the item. By testing client beliefs using the
behavioral experiment framework, the therapist provides the client with concrete
data to illustrate the extent to which they overestimate the future negative impact of
discarding items.
Relapse Prevention
Toward the end of treatment, at least one session is dedicated to relapse prevention.
The broad goal of relapse prevention is to increase the likelihood that clients will
continue to engage in daily sorting and discarding exposures. During the relapse
prevention module, the client is prompted to reflect on progress gained in therapy
and to write down key skills learned. The definitions of cognitive strategies are
elicited, and clients are encouraged to identify which of these strategies they have
been using, and which they would like to make a stronger effort to implement
moving forward. In addition, the clinician reviews concepts relating to exposure
therapy, including avoidance and habituation, and the client identifies and problemsolves barriers to implementing exposures in their day-to-day life. Clients also
consider warning signs and behaviors of getting “stuck” in treatment, such as
missing a day of discarding.
Barriers and Obstacles
General Prognosis
In the context of treatment for OCD and related disorders, hoarding symptoms
have historically been associated with poorer responses to CBT and other interventions (Abramowitz et al., 2003; Bloch et al., 2014). Given that HD was added to the
DSM-5 as an independent diagnostic entity fairly recently (Mataix-Cols et al., 2010),
the literature on HD treatment remains relatively new and indicates only modest
support for intervention effects (Thompson et al., 2017). Aggregating the effects of
treatment trials of CBT for hoarding (Tolin et al., 2015), a meta-analysis indicated
that although CBT produced reliable decreases in HD symptoms, most patients
continued to endorse symptom levels in the clinically significant range after treatment. In terms of long-term effects, the maintenance of treatment gains has been
reported at six-month (O’Connor et al., 2018) and 12-month (Muroff et al., 2014)
follow-up assessments after group and individual CBT for hoarding, respectively.
However, given the chronicity of hoarding symptoms, longer-term investigations of
HD symptom course following treatment – for example, at two years posttreatment – are warranted.
357
358
Stamatis, Roddy, and Timpano
Treatment Barriers
While empirical evidence for HD treatment prognosis remains in its early stages, a
number of barriers and obstacles have been identified in the context of delivering
hoarding treatment to clients. Common factors that stand to stymie HD interventions include low motivation and insight, comorbid conditions, the involvement of
family and friends, limited resources, and legal problems. Contributing to poor
insight, perceptions of clutter may vary significantly across individuals, and individuals with HD often exhibit anosognosia, or a lack of awareness of the impact and
severity of their symptoms (Frost et al., 2010). A key strategy in combating this
obstacle, along with the rigid beliefs and defensiveness that similarly hinder treatment progress, is drawing from MI to increase awareness of the discrepancies
between the client’s current concerns and their goals and values. A related consideration is whether distress tolerance skills should be infused with the general treatment approach described above, to increase motivation and encourage full
engagement with the sorting exposures. There is growing research support for a
specific association between greater hoarding severity and a subjective sense that
one has difficulties handling distressing situations (Shaw et al., 2015; Timpano et al.,
2014), which may contribute to avoidance behaviors and poor coping responses.
These findings have led some newer treatment manuals to recommend directly
addressing emotional processes, with a particular focus on distress tolerance, in
therapy with patients with HD (Tolin et al., 2017).
Another issue that arises in treatment is the influence of comorbid conditions
across medical (e.g., pain, mobility issues) and psychological (e.g., social anxiety,
ADHD) domains. Beyond the individual, treatment interference may relate to the
misguided attempts to help by family members and friends. When loved ones become
involved in treatment, it is essential that they agree to follow the guidelines developed
by the therapist and client (Frost & Steketee, 1998). Conversely, limited social
support and other resources, along with higher levels of distrust (Weintraub et al.,
2018), can slow treatment progress. Finally, legal problems such as eviction, loss of
housing benefits, and competency questions may create significant obstacles during
hoarding treatment. Given the uniquely high likelihood of housing-related legal
issues in HD compared with other disorders, clinicians must work closely with an
interdisciplinary team (e.g., social workers, community members, and law enforcement) to provide the client with comprehensive care (Bratiotis & Woody, 2014).
Case Vignette: Treatment
At the start of treatment with Ron, psychoeducation about hoarding disorder and
the CBT model for hoarding were discussed. Ron initially reported high motivation
and capability for sorting and discarding objects. Therefore, the therapist assigned
Ron the homework of sorting objects for 20 minutes per day between sessions,
emphasizing the importance of following through with discarding or recycling items
that were sorted into those piles as well as abiding by the OHIO rule. However, it
quickly became apparent that Ron’s depression was interfering with his ability to
CBT for Hoarding Disorder
sort and discard independently. Ron reported difficulty getting started sorting,
related to his anhedonia and low motivation. The therapist addressed these
treatment-interfering behaviors in three ways. First, the therapist assisted Ron in
planning specific times to sort and setting alarms as reminders. Second, the therapist
introduced the idea of behavioral activation to help Ron improve his mood and
reduce interfering depressive symptoms. Ron identified walking more frequently
around his neighborhood as a behavioral strategy to increase his social interactions
and physical activity. Third, the therapist had Ron bring a banker box into session to
sort in session together as the first sorting exposure.
The therapist used this in-session exposure to gather important insights about
how Ron related to objects and was able to start to address these with the client.
First, Ron had several saving beliefs such as difficulty with items that he considered
“unique” and difficulty discarding items without knowing the item’s future home
(e.g., Would someone else read and enjoy this book, or would it end up in the
landfill?). Second, Ron struggled to make decisions about objects when not all
objects in that category were present (e.g., he could not sort CDs unless his entire
collection was present). In-session sorting was repeated several times so Ron and
the therapist could consider how emotions and memories connected to the objects.
Ron began to distinguish “good” reasons for saving from instances in which “HD
was putting up a roadblock” that prevented him from moving forward. Throughout
these sessions, the therapist approached Ron’s objects with open-minded curiosity.
Finally, Ron and his therapist discussed items within a particular category that he
considered meaningful; they ranked items along a gradient from very meaningful
(e.g., artwork he and his wife had collected during their travels) to somewhat
meaningful (e.g., a brochure from a museum he wants to visit).
Toward the end of treatment, Ron had made significant progress sorting and
discarding objects in his home; however, he had yet to sort items in his storage units,
and his daughter was coming to town to help with this process. The therapist aided
Ron in preparing for his daughter’s visit in several ways. First, Ron and his therapist
created a list of sorting rules collaboratively in session that he and his daughter would
use while sorting the storage units (Table 17.1). Second, in order to practice sorting,
the therapist and a volunteer from the outpatient clinic spent one therapy session
doing a practice sort with Ron at the storage unit with his permission. During this
session, Ron practiced explaining the sorting rules to the volunteer and allowed the
therapist and volunteer to sort alongside him using the sorting rules. Third, when
Ron’s daughter came to town, she joined Ron for a family therapy session. Ron led
the session, explaining the CBT model of hoarding to his daughter and sharing with
her the progress he had made in treatment. Finally, time in session was devoted to
collaboratively creating a relapse prevention plan with both Ron and his family.
Conclusion
Hoarding disorder, defined by difficulty discarding, excessive acquiring, and clutter,
represents a difficult condition to assess and treat. Identification of individuals
359
360
Stamatis, Roddy, and Timpano
suffering from HD is a critical public health issue, as HD can interfere with activities
of daily living and create safety or fire hazards in extreme cases. Detailed assessment of HD symptoms is critical to successful treatment; by considering which
features of HD are salient for a given client, therapists can better address individual
factors such as saving beliefs that may contribute to discarding roadblocks. Often
comorbid with other psychological disorders, HD can be especially challenging to
treat in the presence of interfering mood and affective symptoms, as well as other
issues such as information-processing impairments. However, there is evidence that
HD responds well to cognitive behavioral treatment in a variety of modalities
(individual, group, and community level). As demonstrated in the treatment overview and case vignette sections, the successful treatment of HD necessitates a
combination of cognitive techniques, such as addressing core beliefs around saving
and acquiring, as well as behavioral techniques, such as sorting exposures. Despite
certain challenges, treating clients with HD is incredibly rewarding clinical work. As
clients gain mastery of the techniques and see tangible evidence of their improvement by decreasing cutter and acquisition, they are often incredibly grateful for the
help and guidance of therapists and community partners.
Summary of Key Points
Hoarding symptoms are influenced by positive and negative reinforcement
systems, both of which constitute important treatment targets.
Although hoarding disorder remains a challenging condition to treat, cognitive
behavioral therapy may help reduce clutter, acquiring, and difficulty
discarding symptoms.
Cognitive behavioral therapy for hoarding disorder involves symptom assessment, psychoeducation, skills training, discarding exposures, and
cognitive restructuring.
When treating patients with hoarding disorder and their family members,
therapists will often collaborate with social workers and individuals in the
broader community.
Patience and flexibility are essential when working with patients who have
hoarding disorder.
REFERENCES
Abramowitz, J. S., Franklin, M. E., Schwartz, S. A., & Furr, J. M. (2003). Symptom presentation and outcome of cognitive-behavioral therapy for obsessive-compulsive disorder.
Journal of Consulting and Clinical Psychology, 71(6), 1049.
American Psychiatric Association. (2013). Diagnostic and statistical manual of mental disorders (5th ed.). American Psychiatric Publishing.
American Psychological Association. (2014). Guidelines for psychological practice with older
adults. The American Psychologist, 69(1), 34.
Ayers, C. R., Saxena, S., Espejo, E., Twamley, E. W., Granholm, E., & Wetherell, J. L.
(2014). Novel treatment for geriatric hoarding disorder: An open trial of cognitive
CBT for Hoarding Disorder
rehabilitation paired with behavior therapy. The American Journal of Geriatric
Psychiatry, 22(3), 248–252.
Ayers, C. R., Wetherell, J. L., Golshan, S., & Saxena, S. (2011). Cognitive-behavioral
therapy for geriatric compulsive hoarding. Behaviour Research and Therapy, 49(10),
689–694.
Bloch, M. H., Bartley, C. A., Zipperer, L., Jakubovski, E., Landeros-Weisenberger, A.,
Pittenger, C., & Leckman, J. F. (2014). Meta-analysis: Hoarding symptoms associated
with poor treatment outcome in obsessive-compulsive disorder. Molecular Psychiatry,
19(9), 1025.
Brakoulias, V., Eslick, G. D., & Starcevic, V. (2015). A meta-analysis of the response of
pathological hoarding to pharmacotherapy. Psychiatry Research, 229(1–2), 272–276.
Bratiotis, C. (2013). Community hoarding task forces: A comparative case study of five task
forces in the United States. Health & Social Care in the Community, 21(3), 245–253.
Bratiotis, C., Sorrentino Schmalisch, C., & Steketee, G. (2011). The hoarding handbook:
A Guide for human service professionals. Oxford University Press.
Bratiotis, C., & Woody, S. (2014). Community interventions. In R. O. Frost & G. Steketee
(Eds.), The Oxford handbook of hoarding and acquiring (p. 316). Oxford University
Press.
Chasson, G. S., Carpenter, A., Ewing, J., Gibby, B., & Lee, N. (2014). Empowering families
to help a loved one with hoarding disorder: Pilot study of family-as-motivators training.
Behaviour Research and Therapy, 63, 9–16.
Cromer, K. R., Schmidt, N. B., & Murphy, D. L. (2007). Do traumatic events influence the
clinical expression of compulsive hoarding? Behaviour Research and Therapy, 45(11),
2581–2592.
First, M. B., & Williams, J. B. (2016). SCID-5-CV: Structured clinical interview for DSM-5
disorders: Clinician version: American Psychiatric Association Publishing.
Frost, R., Kim, H.-J., Morris, C., Bloss, C., Murray-Close, M., & Steketee, G. (1998).
Hoarding, compulsive buying and reasons for saving. Behaviour Research and
Therapy, 36(7–8), 657–664.
Frost, R., Rosenfield, E., Steketee, G., & Tolin, D. (2013). An examination of excessive
acquisition in hoarding disorder. Journal of Obsessive-Compulsive and Related
Disorders, 2(3), 338–345.
Frost, R., Ruby, D., & Shuer, L. (2012). The buried in treasures workshop: Waitlist control
trial of facilitated support groups for hoarding. Behaviour Research and Therapy, 50(11),
661–667.
Frost, R., & Steketee, G. (1998). Hoarding: Clinical aspects and treatment strategies. In
M. Jenike, L. Baer, & W. E. Minichiello (Eds.), Obsessive compulsive disorders:
Practical management (3rd ed., pp. 533–554). Mosby.
Frost, R., Steketee, G., & Williams, L. (2000). Hoarding: A community health problem.
Health & Social Care in the Community, 8(4), 229–234.
(2002). Compulsive buying, compulsive hoarding, and obsessive-compulsive disorder.
Behavior Therapy, 33(2), 201–214.
Fr
Download