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Biology Discussion Forum: Answers to Mystery of the Toxic Flea Dip
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THURSDAY, FEBRUARY 24, 2005
Answers to Mystery of the Toxic Flea Dip
Hey Class,
Here are the answers to the flea dip mystery....don't worry if your answers
don't exactly match, as long as you have the main idea. Remember, this
wasn't a major assignment. Treat this like the chapter questions you usaully
do for Mr. Mascarin. Don't hesitate to ask any questions either in class or
through this forum.
PART 1
What could have been in the flea-dip that killed the girl?
The rotenone killed the girl.
Queen
Lambrini
buzyfriends
sweet_dr33ms
How could a product that is normally harmless to humans and pets
have killed the girl?
The girl must have had an unusual exposure to or received an abnormally
high dose of the flea-dip. She may have swallowed a large quantity of it.
hypn0tik
Anonymous
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PART 2:
Give the autopsy report and recalling your knowledge about the
functions of cellular organelles, what general function of the cell did
the Fleacide probably affect?
The massive cell death in the kidney, lungs, and thymus, and heart indicate
that damage occured at the cellular level. More specially, the damage to the
mitochondria suggests that aerobic respiration was disrupted.
Cellular Respiration
Questions
PART 3:
WELCOME
What cellular process was impared by the Fleacide?
The normal levels of acetyl-Co A indicate that glycolysis is functioning
normally. The absesnce of ATP from the mitochondira suggests that either
the Kreb Cycle or the electron transport chain were impareid
PART 4:
Given this new information, what specific cellular process do you
think was affected by teh Fleacide?
The low levels of NAD+ and high levels of NADH indicate that the electron
transport chain was disrupted. Specially, NADH is not releasing its H.
A product sold in health food stores called Inada contains NAD, what
if you administered this to the girl. Could you save her? Why or why
not?
Probably not. The low level of NAD+ in the heart cells is merely a symptom
of the poisoning. The real problem is that rotenone prevents the transfer of
electrons from NADH to teh electron transport chain . Thus, adding NAD+ to
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3/1/2021
Biology Discussion Forum: Answers to Mystery of the Toxic Flea Dip
the cells may increase the pool of NADH, but it will not restore the function
of the electron transport chain if NADH still cannot release its electron to
make ATPs.
Would artifical respiratioin or oxygenation save the girl? Why or why
not?
Probably not. The real problem is that rotenone prevents the transfer of
electrons from NADH to the electron transport chain. A greater concentration
of oxygen does not solve the defect in the electron transport chain. Oxygen
does not "make" NADH release H. It only accepts the electrons after it has
been released.
Hope this helps.
Ms Yeung
posted by MsYeung @ 8:04 PM
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