rit¥i'"i; .. u ""'"' ""' .,,..,..,._ ,,u.;r.,,.i .u 11w .-w1,,,.;....,i1 4'NIJ;,...i.1 ~...:··~' .Unu6Fy 24,31, 1'i)86, Volume 2!15 Copyr9ht 1986, >.m.11<;lUl Mt1dical A.uo.;iation Council Report 0 Alcohol and the Driver· Council on Scientific Affairs 0 • Scientific Investigations have produced 50 years of accumulated evidence showing a direct relationship between increasing blood alcohol concentration {BAC) In drivers and Increasing risk of a motor vehicle crash. There Is scientific consensus that alcohol causes deterioration of driving skills beginning at 0.05°/o BAC or oven lower, and progressively serious Impairment at higher BACs. Drivers aged 16 to 24 years have the highest representation of all age groups In alcohol-related road crashes; young drivers Involved In alcohol-related fatal crashes have lower average BACs than older drivers. Alcohol Impairs driving skills by its ef1ects on the central nervous system, acting like a general anesthetic. It renders slower and less efficient both information acquisition and information processing, making divided-attentio n tasks such as &leering and braking more difficult to carry out without error. The influence of alcohol on emotions and attitudes may be a crash risk factor related to driving style In addition to drivli:g skill Biologic variability among humans produces substan~lal dlf1erenc:;~s lfl a_lc_ohol influence and alcohol tolerance, making virtually useless any attempts to fix a "safe" drinking level for drivers. The American Medical Association supports a policy recommending (1) public education urging drivers not to drink, (2) adoption by all states of O.OSo/o BAC as per se illegal tor driving, (3) 21 years as the legal drinking age In all stales, (4) adoption by all states of administrative driver's license suspen.slon In driving-under-th e-Influence cases, and (5) encouragement for the automobile Industry to develop a safety module that thwarts operation ot a motor vehicle by an Intoxicated person. (JAMA 1986;255:522-5 27) TIIREE resolutions relating to alcohol and driving \Vere referred to the Board of 'frustees at the 198·1 Annual Meeting of the llouse of fJelegales. From lhEI Counl;it on Scientilic Allairs. Division ol Personal and Public Health Policy, American Medical As.:1oc1ahon. Chicago. AttpOtl A ol the Council on Scienlilic Allairs. adopled by lhEI House of Delegates ol the American Mt1d1cal Association ol Iha Annual Moelmg, June 'rhe 1-Juuse requested that a comprehensive report on alcohol and its effects he prepared for ·the 1985 Annual Meeting. Resolution 18 called for the American Medical AssociatioH (A!iiIA) to study methodology int.ended to deter the use of an autornohile by an intoxicated person. Resolution G·i asked the 1985. 0 This report is 1101 in1ended to be consl1uE1d or to serve as a standard al medical cam Shtndards or medical care are determined on the basi:s of all of the lacts and circumstances involved ir1 an indlll'idual ca!te and are sub1ocl lo change as sdenlllic knowledue an<1 technology advance and patti:rns of prachce uvotve This report refll1Cls lhe 11iews ol tho S<:Juntllic ~Wrutu1a as ul June 19U5 riepnnt 1aque~t3 to ~v1:s1on ol Pe1sonal and Public Hoallh Polic"t. Council on Scitinl!fic AUairs. A11•oncun Mu!l•Gal A3:-;oc1at1on. 5J5 N Ouurborn St. C/uc1t1,10. IL 60610 (John C. Bo1lhn, Pl10) 522 See also pi: 450 and 529. AMA to urge Arucricans to refrain front drivinl{ under the influence of alcohol, asked the A f\·1 A tu conduct an education can1pail,'."n nu this st1bject, an·d ask1•1I lhi• AMA lo su11port n1an- datory susp1·11si(1n of a JAMA, Jan 24/31, 1986-Vol 255, No 4 driv1~r's for one year for any conviction for a moving violation if any alcohol is found in the driver's blood. Hesolution 83 urged an A~1A study of recent legislation among the states on driving \Vhile impaired, \vith incorporation of the effective ele1nents into 1nodel legislation for distribution to the 1nembership. In addressing the concerns cited in the resolutions, re\•ie\vS were undertaken of current literature on (I) the relationship between blood alcohol levels and dr!ver impairment, (2) scientific issue·s regarding the reliability of 1nethods to test blood alcohol levels in drivers, and (3) alcohol-impaire d ddving counterrneasure s. Epidemiology ol Alcohol in Road Crashes Studies carried out in the United St.ates and other developed nations sinte the 1930s indicate a strong, direct relationship between increas .. ing blood alcohol concentration (BAC) in a n1otor vehicle driver and increasing risk of his involvement in a road crash. u A driver's relative risk of having a road crash sho\vS a dramatic rise as Members of the Council on Scient1lic Altairs include Iha following: John A. Beljan, MD. Philad<;1lphia; Geo1ge M. Bohigian, MO, St Louis; William D. Dolan. MD. Arlington, Va E Harvey Estes J1. MO, Durham, NC. lra R Friodland_<;1r, MD. Chicago; Ray W Qdlord, _Jr, MO. Clcvti_land, Chai_rman~_John H_ Moxley 111,-MD, eev~rly Hills: Calif, Vice· Chairman; Pelar t-f_ Sayrn, Boston. Medical Sludunt, William C. Scou, MD. Tucson, .loscµh H. Skom. MD. Chicago; R~ers J Sr~1•th. MO, Po1Uand, Ora: James B. Snow. Jr. MO, Philadelt)~••a. John C Ballin. PhD, Ch1Cal)O, Secrnlary: James L Breeling, 5taH Author license Alcohol-Council on Scientific Aflairs --- -r-:-.-- , I-'"· .1 45 70 0 40 ~ ~ -- 35 • 65 60 • ;-ao ~ - -~ f-- -- 55 il 0 25 50 i ~ 45 20 d: I I 40 35 I 30 / 5 1 0.00 -~ ~v 0.04 0.02 0.06 -- I 25 0_08 0. 12 0. 10 o. 16 0. 14 0.20 o. 18 Blood Alcohol Level, 'li Fig 1. -Relat ive probab ility of causin g crash rises wilh rising blood alcoho l levels. 15 0 0 JAMA, Jan 24131 , 1986 -Vol 255, No. 4 /, ,___ -- 10 the drive r's BAC increa ses (l•"'igs 1 and 2).'·' Alcoh ol-im paired drive rs are believed to be respo nsible for 25% to ::J5% of all crash es causin g seriou s injury and 6% of all crash es. In .single·vehicle crash es, 55% to 65% of fatall y injure d drive rs have BACs of 0.10% or greate r.) In most states of the Unite d States , a BAC of 0.10% is the legal defini tion of being under the influence of alcohol for drivin g-und er-the -influ ence (DUI) prose cution . Since 1960 the AMA has recom mend ed that a blood alcoh ol level of 0.10% be accep ted as prima facie evidence of being under lhe influence, a positi on that the (AJuncil on Scien tific Affai rs believ es shoul d be revise d to a lower BA C in light of scient ific evidence. Significant alcoh ol involve1nent in injury causi ng road crash es begins at a drive r BAC of 0.05%. Jn a recen t review, Johns tonJ conclu ded that lO~t.i of drive rs in crash es that cause property dama ge had BACs of 0.05% or great er and that 16% to :J8% of drive rs in injury -caus ing crash es had BA Cs of 0.05% or great er (Tabl e 1). Jn 1982, one in three perso ns killed in Austr alian road crash es and one in five injure d had a BAC of 0.05% or I • 20 ---- 5 1 < 10 I I 30 - --·· -- 50 ,,...-- 70 ,/ 90 110 / .. 130 150 - 170 190 200 Blood Alcohol Concentration, mg/dl Fig 2.-Ra lalive probab ility ol being respon sible for fatal crash rises wilh rising blood alcoho l concen tration s. more. 5 McDe rmott and Stron g' found that drive rs with alcohol levels of 0.05% or niore have a greatl y increa sed risk of being involved in a road crash causi ng injury or death . In the first three years of con11lulsory BAC testin g of adult road crash victims in Austr alia's Victo ria State , 27.1 % of 21,863 male drive r casua lties and 8.7% of 9,187 femal e drtvc r casualti es had DACs excee ding the Victoria legal lhnit of 0.05'lo. Soder strorn et al1 repor ted that of 413 road crash victim s with n1easurahle BACs at an emerg ency ruedical servic es cente r in Mary land, 91 had RACs of less than 0.10%. Alcohol involv emen t in crash es had been called an epidctnic, with little dimin ution in its propo rtions despi te heigh tening of puLlic consci1:,usncss by the activi ties of variou s co1nn1uni- ty group s and anti-d runk- drivin g campaigns.• Ravag es of the epide mic have been great er amon g the young. Fatal Accident Repo rting Syste m (FAR S) data show, from 1977 to 1981, a stead y increa se in the overa ll proportio n of measu reable blood alcohol levels in drive rs aged 16 to 25 years involved in fatal crash es.' The authors believed the data to be more repre senta tive of patter ns of alcohol use in that age group than impro vement in BAC testin g and repor ting. Drive rs aged 16 lo -19 ye:irs have . the highe st rate of alcohol-involved fatal crash es per unit of trave_~-~ _Epiden1iolcgic data from .F'ARS also indicate over a numb er of years that young er drive rs involv ed in fatal crash es have lower avera ge BACs than older drive rs. 1" Previo us reviews of biogra phica l variahle:i in alcohol- Alcoh ol-Cou ncil on Scient ific Affairs 523 115 Table 1.-lncidence of Blood Alcohol Concentra11o ns ol 50 mg I dl. by Crash Se1,1erily and Type ol Road User 0 150 125 Cra•h Severity,% Type ot Road Uaer Orivttr Paaitenger• Mo1orcydisl Podealtlan • ~ 13 Ptoperty Damage Injury Fatal ::::: 10 16-38 45-55 25-35 35 30 .. 165 25 22-25 19-25 • Percenlag<i shown is ol those older than • "- 100 ~ a: 75 50 14 Yt:LlfS. 25 related crashes furnished the same finding. The role of alcohol in crashes of tl'cnage drivers also is indicated in FARS data for 1981 showing that l\-vice as many with positive BACs \Vere involved in single-veh ide crashes as oppo~(~d to n1ultiple-vc hicle fatal crashes. A driver in a singlevehiclt! accident is presu1ned r·csponsible for his own crash. In the sarne data, five tirnes rnore male than ft>.rnale teenage drivers were lnvolved in single-vehi cle fatal accidents, bearing oul by trend if not by precise ratio another consistent biographic al find- 0 0 ing Analysis of 198:! l•'Al!S data showed that :la% (17,764) of all drivers in fatal road crashes that year \\'ere 16 to 2-1 years uld. Of that nu111bcr, 68'.1<) (G,83:1) were alcohol involved, con1pared \vith 26% in all olher age groups. Fatalities in road crashes involving drinking drivers aged 16 to 2,1 years numbered 7,784 in 1Hl:IB, of whurn 5l'Y" (J,~)02) were the drivers themselves . 11 A rnodel developed by Siin1isonu (Fig 3) shows the relative risk by age group of having a fatal crash if drivers were in1paired by BACs of 0.08'%, or greater. With the risk of a sober driver having- a fatal crash set at 1, the risk for in1paired 16- to 17-yc,tr-uld s is 165. Young drivers arc overrepres ented in crashes and also in alcoholinvolvcd crashes \vhen IlACs are low lo 1noderate. Overreµres entution 1na.v include cxiiosure (rniles driven} as a l"Olllponenl. {)vcrrepres entation at lo\\' BA Cs 1nay he a function of young-t•r drinkers having !t•ss alcohol tolerance than CXJJerience1l drinker::; and younger drivers having less cxperit•ru·e than older drivt~rs."' 524 MSL" 16-17 20-24 25-29 30-34 35.44 45.49 50+ Total Impairment Risk Fact°'s Fig 3.-Age group and risk ol fatal collision if impaired. The Effects of Alcohol \vith the drug. One effect of alcohol in road Dose/weig ht charts may not be crashes is its contrihuLio n to enhanceappropriat e guides to drinking behav1ncnt of injury in alcohol-in1paircd ior.11 Biologic variability of response victin1s. Prevention or liniitation of to alcohol has been den1onstra tcd trau1na is less likely in alcoholunder controlled experiznen tal condii1nµaircd .drivers because they are tions \vith both n1ale and female less likely to use seat belts. - ~ Consubjects: in single-dose drinking tests trary tc the pe;,111;..:· helicl in being the elapsed tiine f--orry c.n4 of drinking 11 too drunk to g~t hurt," 1110!·~ alcoholtu peak BAG varied from 14 to 138 irnpaired crash victiins suffer serious minutes in one group, and in a followinjury than i;olier victin1s.1.' Aleohol up study the sa1~e investigato r found con1plicates the physi.:ian's task of a 14-fold variation between aLsorptreating traurna: neurc>logic injury . tion times in different subjects. 19-w nuty be 1naskeJ hy drunkenne ss, and Women achieve higher peak BACs acute and/or chronic intoxicat.ion than men Y:hen given identical rnay Lie linked Lt> a consitlerab lc range weight-adj usted doses. of metabolic disturbanc es, as \vcll as A reviey.· of seven studies spanning to altered responses to anesthesia and a 50-year period (Table 2) indicates alterations in host defenses against that at BACs of up to 0.05%, 0% to' infection. 1 Expcri1nen tally controlled ioc;o of persons were considered to be injuries to laboratory anirnals result "drunk"; at levels of 0.05% to 0.10%, in lower survival rates for animals 149b to 68'.lh of persons were considfirst given alcohol and n1ore t!xtensive ered to be 'jdrunk"; and at levels of intracrania l hernorrhag e in alcohol0.10% to 0.15%, 83% to 97% were in1pairetl animals after experin1cn tal considered to he tlrunk. 11 penetration of brain tissue. 1. . ' 'fhe deleterious effect of alcohol at The influence of alcohol related to BAC levels of 0.05% to 0.06% is seen driving hchavior and driving skills is in pcrson8 performing curve-nego niediated through its effects on the tiating Hdriving" tasks under laboracentral nervous systen1, si1nilar to tory conditions. Verhaegen et aizi conthose of general anc•.;thetic. Alcohol cluded that at BACs between 0.05% in sn1all doses 1:1ay cause JH~rforrn­ and 0.06'.Y,,, perforrnanc.e in informa· ance of drivi11g--relatcd skills to fall lion processing and curve-nego tiation off; in n1otleratl! tu high a1nounls, skill deteriorate d in test subjects. alcohol dirnini:,;hes perfonuanc e Burns and Moskowitz 21 observed a across tlu~ lioard with gt•1H:ral i1npairJO';;, lo 15'Yr1 degree of in1pairn1ent at rnent of nervous function. Effects BACs of 0.05% tq 0.08% when subn1ay vary \vith p'.'.>ychologic.al profiles, jcets had to perform a divided-att en· tolerance to alcohol, and CX)jericncc tion task of tracking and reaction. JAMA, Jan 24.'31, 1986-Vol 255, No. 4 11 1 Alcohol-Co uncil on Scientilic Affairs ,. --- -:-:i Jt Table 2.-Relation Between Blood Alcohol Level and Drunkenness I % of P•r11on11 Found to Be Drunk 0 0.00- 0.051- 0.05° 0.10 19 0 0.1010.15 50 0.1510.20 0.201· 0.25 83 93 0.2510.30 98 0.301· 0.35 100 2 38 93 97 99 100 100 10 16 47 83 90 95 96 10 66 61 92 97 100 0 46 50 92 100 100 0 14 69 90 94 94 0.3510.40 Total Peraona 0.401 Examined lnYe11tlg11tora t 1,984 Wtdmark 221 Schwarz 195 IOO 950 93 100 1,000 Jetter ... . .. 1.712 Ander Sen 100 100 100 140 100 IOO 100 100 I b 133 r 9 Harger 111 "''• I 188 7 25 49 85 93 97 98 100 99 750 • 32 82 89 95 98 99 99 100 6,69.ot Hine 123 . .. 'Percent weigh! by volume (0.05% w/v=-50 rngtdl; 0.15% w/v= 150 1119/dl) ol blood alcohol. fNumbers under namas ol inveSll\l&lors are ralerenca citalions in reference 35. source ol Table 2 ar 0 1 A tl\vood et te:-;ted driver:; on closed-course conditions and using a nu1ltivariate analysis technique found that drivers with BACs of 0.06% to 0.ClH'X. exhibited n1ore variability than alcohol-free drivers in lane position, brake use, and steering controls. ln1pairment of judgrnent by alcohol \vas dernonslrate<l at BAC~ as low as 0.04 % when skilled bus drivers misjudged their ability to drive a vehicle through a space as much as 12 in narrower than the bus. H Johnston) states that many tests of alcohol use and skills relevant to driving show that both information acquisition and information processing are rendered slower ancl less efficient, and the ability to carry out a divided-attention task that requires intellectual time sharing is impaired. I le hypothesizes that \vhen impaired drivers enter curves, they devote so rnuch attention to the steering task that other perception of cues related to road curvature suffers, and they fail to reduee speed adequately. has been a revival of interest in saliva-alcohol testing. Laboratory rnethods for analysis of blood san1ples include (1) che1nical reduction of acid dichro1natc, (2) enzyn1atic oxidation by alcohol dehydrogenase with colorin1etric determi. nation of NAfHI (the reduced for1n of nicolinarnitlc - adenine dinucleoti1le) conversion, and (3) gas-li_quid chromatography. Choice of the mclhod used Ly a laboratory 1nay be influenced by the size and sophistication of the facility and the reliability of the rnethod for confirn1ation of roadside breath analyses, All three n1ethods have strengths ant! \veaknesses, but gas chromatol{raphy is the n1ost accurate and best suited for handling large nun1bers of san1ples. IL al~o has lhe advantage of sensitivity to other aliphatic alcohols or volatile toxins that a suspecl may have been ingesting with, or without, elhanol. ~ Breath analysis is by far the nlost comn1on rnethod of n1easuring BAC. The concentration of ethanol in one volume of blood is stated in inost textbooks anJ highway safely regulations to be equivalent to lhat in 2,100 volun1cs of alveolar air.~ llulio\vskii~ challenges the 2,100:1 conversion factor un the hasis of sopliistiti\ll'd chcniie~tl a11alyses of blood and breath alcohol. I le and O'Nt~ill plal'e the 1nean alcohol partition factor between blood and breath, in tht~ postahsorptive phase in h1•allhy adult n1ales, al approxi1natl'ly ~.:100:1, \vith a range of I.7~17:1 to ~.71;:1: I f(Jr !I.")':;. of a population of :1~1;1 1 Chemical Tests for SAC 0 Determination of BAC is made directly by che1nical testing of blood dra\vn fro1n the subject or indirectly by testing of expired Lrcath or urine. A1ore and rnore, breath-alcohol analy::3is is performed for the purpose of dl~terrnining the concentration of alcohol in hreath, rather than for atlernpted conversion to blood-alcohol eunl·enlrution. ()thcr fluids and tissut~s 1nay be tested bu! usually are not in the livin~ ~uhjt•1·t; r1•e1•11tly, lh1·n· JAMA, Jan 24131, 1986-VQ\ 2!:l0, No 1 4 healthy adult n1en and a range of 1,555:1 to 3,005:1for99.7%. Quantitative breath-alcohol analyzers are all currently factory calibrated to a conversion factor of 2,100:1 to 1neet oflicial guidelines of the National l:ligh- way Traffic Safety Administration. Dubowski questions whether the conversion of breath alcohol concentra- . tion. to BAC .should be retained for for~~1sic p.urpOS~S' ~nd recommends that breath alcohol coneentration alone be used-for statutory definition of DUL That the 2,100:1 ratio is too low has been raised as a possibility to explain why breath analysis values from one well-regarded instrument are on the average 10%, to 153 lower than alcohol concentration in blood samples taken at the san1e time_2<1 In a seven-1nonth trial in London or three types of breath-testing instrun1ents used in the United States, the breath-testing instruments tended to underread actual blood alcohol levels by 0 to 20 mg/dL in the RAC range of 0.05<Jo to 0.10%.)0 The US National Highway Traffic Safety Administration publishes rnodel specifications for the performanee, calibration, and testing of breath alcohol testing devices to ensure their reliilhility. DUI Countermeasures Strengthening of state DUI laws has lH·t!n a trend over the past several year~. State ltq.,dslalon; are apparently that a growing national n·,·oi~11izing Alcohol ·--Council on Scientific Allairs ·-----,- I 525 0 consensus against driving under the inlluence ruu!'lt be hacked by specific laws needed by police, courts, and licensing ag-encics to get alcoholi1npaircd drivers otf the road. The A MA in May 1982 issued to state and medical specialty societies a rlocun1ent titled "Drunk Driving La\vs" and urged state medical a~so­ ciations to consider seeking enact1ncnt of legislation to strengthen l)UI la\vs in their respective stales. 'l'hc AMA !louse of l)elagates at its l98J Annual Meeting (Resolution 95, A-8:1) rcafllrn1cd AMA policy to encourage each state 1nedical society to seek an<l support legislation lo raise the n1inin1utn drinking age Lo 21 years, and it urged all physicians to educate their patients about the dan- 0 0 gcr!i of alcohol abuse in general and operating a motor vehicle while under the influence of alcohol in particular. An1ong the tnore visible and easily identified strengthening of state l)Ul laws is the rcplacernent of "presumptive" Ly "per se" Ja,vs. The latter laws 1nake it illegal in and of itself to drive 'vilh a BAC over certain specified lin1its. In states with "illegal per se" ht\\'S, proof of driving under the influence of alcohol is automatic \Vhen a properly ad1ninistered test of the specified type shows the driver's BAC to Le over a specified limit. ~lost states with illegal per se la\vs set the BAC limit at 0.10%, but the range an\ong all such stale laws is front 0.08% lo 0.15%. A variation on the illegal per se la\\' is a l\vo-stcp la\v adopted in sou1e slatl!s: (1) illegal per se set at a specified BAC, and (2) presu111ption of driving under the influence set at a lo\'..·er BAC, requiring supporting evid~nce other than breath or blood test for prosecution. 'fhe Iiighway Users Fetleration rt•c01nn1ends an illegal per se law as one provision in any driving legislative package. Provisions incluJe (1) adrninistrative driver's lict'nse suspension, \Vhcrcliy the liccns(! of any tlrivcr arrested for driving under thl• inllul'f\t'.c is s11spc111led for a spt~cilied period, \vith harsh penalties itnposeJ for driving: while the license is suspended (the 1neasure is .ain1cd at the rcpe•1t offender); and (2) rl'Conling of all aleohol-relateJ arrc::;ts, a provision incant to identify repeat uffcndcn; and particularly those whose aJc(Jhol526 related arrc::.ts are frequently plea ()n three occasions, in 1972, 1978, hargaint•d to a lt!:-;~rr charge not and 1H8:~. large assctnhlic:i of North related lo alcohol. American investigators ranked huIn rcvie\ving: tht! recent records of rnan-related risk factors at or near control rneasures, \Valler 11 identified lhe top of DUI research needs. In t\vO as being associated with positive each instance, the group asked for results: (1) An ine!"ca~"-' in thP age at 1nultivariate st~dies that incorporate \vhir.h one can h.:! h,sucd a driver's hun1an-related variables of an attitulicense or can drink legally see1ns to dinal-personality nature and a longhave a positive effect in reducing the term research strategy coordinated nun1ber of alcohol-related crashes L:v thrc.. 1gh so1ne cen~ral organization. 11 16- and 17-year-olds. (~) License susYouthful drh·ini~ and drinking is an pension or revocation is the most area \vhere research on multicausalicost-effective countern1easure yet ty seemed urgently warranted to identified for reducing driving by investigators, in light of the peculiardrunk driving offenders. Arrest, trial, ly high risk of dealh and injury from and irnprison1nent are far n1ore alcohol-related crashes in this expensive in public servant tirne and group.u Some suggestive research public funds. Revocation of a license indicates that drinking and driving for driving under the influence may populations contain drinking/drivbe 1nandatory on conviction or n1ay ing/crash-prone subpopulations in occur ad1ninistratively upon evidence. whon1 the influence of alcohol on that the person co1nn1it tetl the ofe1notions and attitudes rnay be an fense. Waller noted that several important causative factor.n The ininvestigators have reported that one fluence of alcohol on an ernotionally third to l\vo thirds of persons with charged driving style may be as revolu~d lict~nses eontinuctl to drive irnportant as its influence on driving skill.JU while the revocation or suspension \Vas in effect but \Vere driving less Social and cultural factors that often and 1norc carefully; n1ultiple .influence the magnitude, characterisl)LJJ 1.1ffen..ler:> wh,~ ''·t·i·c sn~- 1 .enl!ed tics, and persis~encc of the drinking had Letter subsequent records than and driving problem arc not yet co1nparable convictees whose licenses defined. Whether sustained shifts in \Vere not suspended. sociai norms related ta rlrinking and The i1npact of per s1' legislation drivL1g can be brought about-as upon deterrence of alcohol-irnpaired they were in relation to littering, driving \Vas unclear in four reviews of smoking, and diet/fitness/heart disthe data, according to Waller. A diffiease-is a qaestion yet to be anculty often encountered was the swered_» inability of the revie\ver to separate Conclusions the effect of per sc laws fron1 that of other countcr1neasures instituted at 1. Alcohol causes deterioration of about the san1e tin1e in the san1e driving skills beginning at 0.05% states.u llAC (50 mg of ethanol per deciliter of Con1parison of n1~uit.1atory licensing blood) or even lower. Deterioration sanctions \vith education and rehabilprogresses r~pidly \\'ith rising BAC to itation IJrogran1s for [){JI offenders in serious impairment of driving skills four state::> dc1nonstratcd clear supeat BACs of 0.10% and above, accordriority of the licensing sanctioni;; in ing to scientific consensus. reducing DUI recidivism and suhse2. Drivers with BACs of 0.05% to t}UeJtt crash involve1ncnt. 11 • o.iory(J are significantly represented in road crash statistics. Research and Human-Rolatod 3. fJrivers aged 16 to 21 years have Risk Factors the highest rate of alcohol-involved !vtu !Lidi!ici pl i nury i II\ cstiga lions of fatai cra:;hes per rr::Je, ;vith lo\vcr driving and drinking are rare to nonaverage BACs than older drivers. existt~nl. lirlultiplt~ foci of research The Council on Scientific Affairs interest-eg, higlnvay and auto saferecom1nc11d::; that the: AMA (1) direct ty, phannaetilog-y, a!cobu~ and subpublic :riformation and education stance abuse, trau111a trt'at.nH~nt, legagainst any drinking by drivers and islation, anti rcgulatio11-·-have ten(lt•d encourage other organizations to do to OfH!rate \\'ithout strong linkages. the san1c; (2) adopt a position sup- JAMA. Jan 24131. 1986-Vol 255, No. 4 Alcohol- Council on Scientific Affairs I 6 wi.·. ( I ~. porting a 0.05% RAC as per se illegal for driving and urge incorporation that position into all state DUI laws; (3) reaftlrm the position supporting 21 years as the legal drinking age, strong penalties for providing alcohol or 0 to persons younger than 21 years, and stronger penalties for JJroviding alcohol to drivers younger than 21 years; (4) urge adoption by all states of an adn1inistrative suspension or revocation of driver license8 after f)l!I con- viction and mandalory revocation after a specifred number of repeat offenses; (5) encourage automobile industry efforts to develop a safety module that thwarts operation of a car by an intoxicated person. References I. Normnn I.CT: /(o(lf/ T1·f\tfic Ar.ci•frnl.s.: f.'pid1·· Cu11trol, u11d Preventia11, public health paper 12. Geneva, World llealth Organi2ation, l:Jti2. 2. McLean AJ, Holul.iowyci OT: Alcohol and the risk of accident involvement. Proceedings of the Eighth International ConCtirenee on Alcohol, Drugs and TraHlc Safety, Stockholm, June 15-19, mwltiuy, }!j80. 3. Perrine MW: Alcohol involvement in high- way crashlls: A review of the e1-1idemiolo11:ic cvi1lence. Clin Pla1>t Sury 1975;2:11-3-1. ·L Laurie W: Alcohol and the road toll. A1t'd J Au.st 1981;1:226-227. 5. Johnston Ht The role of alcohol in road rrashcs. E1yonomics 1982;25:941-9-16. 6. Mclkrmoll 1", Strong P: Compulsory blootl alcohol testing of road crash casualties in Victoria: The first three years. /ifo:d J Au:it 1978; 2:fil2-615. · 7. Soderstrom CA, DuPriest RW, l~nncr C, et al: Akohol and roadway trauma: Problems of diai.::nos!s anJ managcmt>nt. Am Suru 1!179; -l:-d~l-13li. 0 8. Fell JC: Tracking the alcohol in\'Olvement problem in U.S. highway crashes, in ProcL·edi11gs uf t/11· ;!7/Ji A1111mil Cunferenc11 of tlic Aml·ricun Ali,'.lociation /01· Automolive .hlf1dici11e. 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