CLINICAL SIGNS AND SYMPTOMS • Cardinal Symptoms – chest neck and/or arm pain or discomfort – Palpitation – Dyspnea – Syncope – Fatigue – Cough – Diaphoresis – Cyanosis – Edema and leg pain/claudication(vascu lar component) – 2 CHEST PAIN AND DISCOMFORT common presenting symptom of cardiovascular disease • cardiac or noncardiac in origin • radiate to the neck, jaw, upper trapezius muscle, upper back, shoulder, or arms *most common on left arm • follows the pattern of ulnar nerve distribution. • can be experienced in the somatic areas because the heart is supplied by the C3-T4 spinal segments • -Author/s (Year) 3 PALPITATION • presence of an irregular heartbeat, may also be referred to as arrhythmia or dysrhythmia, which may be caused by a relatively benign condition • a bump, pound, jump, flop, nutter, or racing sensation of the heart. • may include lightheadedness or syncope • as if the heart "skipped" a beat. -Author/s (Year) 4 DYSPNEA • also referred to as breathlessness or shortness of breath • Can vascular or pulmonary in origin • unexplained episodes of shortness of breath frequently accompany congestive heart failure (CHF). -Author/s (Year) 5 CARDIAC SYNCOPE • Cardiac syncope (fainting) or more mild lightheadedness can be caused by reduced oxygen delivery to the brain • Syncope that occurs without any warning period of lightheadedness, dizziness, or nausea may be a sign of heart valve or arrhythmia problems. -Author/s (Year) 6 EDEMA • Edema in the form of a 3-pound or greater weight gain or a gradual, continuous gain over several days that results in swelling of the ankles, abdomen, and hands combined with shortness of breath, fatigue, and dizziness may be red-flag symptoms of CHF. -Author/s (Year) 7 ANGINA • Acute pain in the chest, called angina pectoris, results from the imbalance between cardiac workload and oxygen supply to myocardial tissue. Angina is a symptom of obstructed or decreased blood supply to the heart muscle primarily from a condition called atherosclerosis. -Author/s (Year) 8 TYPES OF ANGINA PECTORIS • • Chronic Stable Angina – occurs at a predictable level of physical or emotional stress and responds promptly to rest or to nitroglycerin Resting Angina (Angina Decubitis)- occurs at rest in the supine position, pain is neither brought on by exercise nor relieved by rest. • Unstable Angina(Cresendo angina)- abrupt change in the intensity and frequency of symptoms or decreased threshold of stimulus, such as the onset of chest pain while at rest. – duration of these attacks is longer than the usual 1 to 5 minutes; they may last for up to 20 to 30 minutes. 9 TYPES OF ANGINA PECTORIS • • Nocturnal Angina- may awaken a person from sleep with the same sensation experienced during exertion. During sleep this exertion is usually caused by dreams. Atypical Angina- unusual symptoms (e.g., toothache or earache) related to physical or emotional exertion. These symptoms subside with rest or nitroglycerin • Prinzmetal's angina produces symptoms similar to those of typical angina but is caused by coronary artery spasm. These spasms periodically squeeze arteries shut and keep the blood from reaching the heart. 10 11 12 MYOCARDIAL INFARCTION 13 CONGESTIVE HEART FAILURE • • • • Dyspnea Orthopnea Cough Pulmonary edema • Cerebral hypoxia • Fatigue/Muscle crampingor weakness • Nocturia -Author/s (Year) 14 Heart Failure 15 CARDIAC VALVULAR DISEASE 16 RHEUMATIC FEVER 17 ENDOCARDITIS 18 MITRAL VALVE PROLAPSE 19 PHARMACOLOGIC MANAGEMENT • Diuretics OR "water pills“ – lower blood pressure by eliminating sodium and water and thus reducing the blood volume. • Beta-blockers – relax the blood vessels and the heart muscle by blocking the beta receptors on the sinoatrial node and myocardial cells, producing a decline in the force of contraction and a reduction in heart rate -Author/s (Year) 20 PHARMACOLOGIC MANAGEMENT • Alpha-1 blockers • Angiotensin-converting enzyme (ACE) inhibitors • Calcium channel blockers – lower the blood pressure by dilating blood vessels. – improve cardiac function in individuals with heart failure and are used for persons with diabetes or early kidney damage. – inhibit calcium from entering the blood vessel walls – Side effects may include swelling in the feet and ankles, orthostatic hypotension, headache, and nausea. -Author/s (Year) 21 CARDIAC REHAB POST-MI • Divided into 3 stages/Phases – Phase I (Acute Phase) • Phase Ib (Inpatient Rehabilitation Phase – Phase II (training Phase) – Phase III (Maintenance Phase) -Author/s (Year) 22 Acute Phase (Phase I) Education about cardiopulmonary risk factor modification is introduced at the time of acute hospitalization. • Post-MI heart rate increase with activity should be kept to within 20 beats per minute of baseline, and SBP kept within 20 mm Hg of baseline • A decrease of 10 mm Hg or more is indicative of further medical issues and exercise should be halted. • The target intensity at the end of the phase I program exercise is to a level of four METs • -Author/s (Year) 23 24 Inpatient Rehabilitation Phase (Phase IB) • The guidelines for exercise are the same as they are for patients in phase 1, but with a longer recovery period extending their hospitalized care to an acute or subacute rehabilitation setting before discharge. -Author/s (Year) 25 Training Phase (Phase II) • • • • • starts after a symptom limited full level ETT for patients with cardiac disease or a CPET for patients with complex pulmonary disease. allows for setting target heart rates and target exercise intensity from the exercise. three sessions per week for 8 to 12 weeks. Patients need to learn to begin exercise with a stretching session, then a warm-up session, a period of training exercise at designated intensity, followed by a cool-down period. The principles of specificity of training need to be remembered because training benefits generally are seen in the specific muscles exercised. -Author/s (Year) 26 Maintenance Phase (Phase III) • • The benefits of a phase 2 program can be lost in as little time as a few weeks if a patients ceases to exercise. Because of this, patient education of the importance of making exercise a part of their new health habits has to be emphasized and the patient needs to integrate exercise as a part of a healthy lifestyle. To maintain capacity, patients should perform moderate exercise at the target intensity learned in their rehabilitation program for at least 30 minutes three times a week. With lowlevel exercise, the frequency has to be increased to five times a week for maintenance of gains. -Author/s (Year) 27 -Author/s (Year) 28 Exercise Intensity prescribed by either HR or by subjective report, a rating of perceived exertion (RPE). Subjective ratings of intensity of exertion have been used to quantify effort during exercise. • A common aerobic exercise prescription based on HR is 70% to 85% of HRmax • -Author/s (Year) 29 Exercise Frequency • commonly prescribed three to five times per week. The patient should not experience increased fatigue as a result of exercise. If fatigue does occur, the frequency and/or intensity of exercise should be decreased. -Author/s (Year) 30 Exercise Duration • The goal of 30 to 40 minutes of aerobic exercise with an additional 5 to 10 minutes of warm-up and an adequate cool-down is appropriate. -Author/s (Year) 31 PHYSICAL THERAPY INTERVENTIONS • • • • Aerobic Exercise Strength Training Ventilatory Muscle Training Activity Pacing and Energy Conservation Technique 32