Emergency Evaluation of the Dyspneic Patient Dr. Didem Ay Emergency Medicine Goals • • • • • Definitions Emergency Department Evaluation Respiratory Assessment Treatment Etiology • Dyspnea: – Sensation of breathlessness or inadequate breathing – Most common complaint of patients with cardiopulmonary diseases. • Dyspnea - common complaint/symptom – Defined as“shortness of breath” or “breathlessness” – “abnormal/uncomfortable breathing” – “not getting enough air” • Multiple etiologies – 2/3 of cases - cardiac or pulmonary etiology Terms • Tachypnea: Rapid breathing • Ortopnea: Dyspnea in the recumbent position • Paroxysmal nocturnal dyspnea: Orthopnea that awakens the patient from sleep Terms • Trepopnea: Dyspnea associated with only one of several recumbent positions • Platypnea: The opposite of orthopnea (dyspnea in the upright position) • Hyperpnea: Hyperventilation and is defined as minute ventilation in excess of metabolic demand • Hypoxia: Insufficient delivery of oxygen to the tissues • Hypoxemia: Abnormally low arterial oxygen tension. (PaO2) < 60mmHg or arteriel oxygen saturation (SaO2) < 90% Oxyhemoglobin Dissociation Curve Attention • Psychogenic dyspnea should be diagnosed after exclusion of organic causes Emergency Department Evaluation • There is no one specific cause of dyspnea and no single specific treatment • Treatment varies according to patient’s condition – – – – chief complaint history exam laboratory & study results Respiration: Inspiration and expiration to provide sufficient tissue oxygenation Respiratory distress: Unnatural, uncomfortable, distressing inspiration and expiration causing tissue hypoxia Clinically hypoxia, cyanosis, hypercapnia occur Respiratory Assesment • Primary evaluation: Goal is to eliminate life threatening causes • Secondary evaluation: Detailed Respiratory Assesment • Primary – Normal: Spontaneous, comfortable, painless, regular respiration: 12-20/min (in adults) – Look, listen and feel for breathing – Wheezing, stridor? – Consciousness? – Talking? – Paradoxal chest movement?(flail chest) Look, Listen, Feel Respiratory Assesment • Primary – Respiratory distress – Head-tilt, chin lift or jaw trust – Open airway – Reasses breathing – If breathing present, start oxygen – If breathing is not present start artificial ventilation Baş geriye, alt çene öne yukarıya Aspiration Airways 28 33 34 35 Respiratory Assesment • Secondary – History – Physical examination – Chest film History • Age, past medical condition • Associated symptoms (Fever, cough, sputum, angina, pretibial oedema) • Timing: acuity and duration (Spontaneous/sudden onset, dyspnea on effort, orthopnea, PND) • Severity • Past medical history • Smoking, drugs (OKS, HRT), trauma, immobilization, malignancy Signs and Symptoms Serious respiratory distress : 1. Clinical: 1. 2. 3. 4. Tachypnea (RR> 35/min), apnea Cyanosis Retractions Agitation, inability to talk, unconscioussness, coma 5. Rales, wheezes 2. SO2< %90, PaO2<60 mmHg, PCO2>45 mmHg • • • • • • • • Physical Examination Head to toe Vital signs Consciousness Skin color (paleness, diaphoresis, cyanosis, erythema, urticaria) Retractions (intercostal, suprasternal, abdominal) Clubbing Auscultation Signs of heart failure Laboratory • • • • • • • Pulse oxymetry, arterial blood gases Complete blood count Chest X-ray, lateral neck X-ray ECG monitorization Echocardiography Biochemical parameters If needed: CT, ventilation-perfusion scintigraphy Pulse Oxymetry • Rapid, widely available, noninvasive means of assessment in most clinical situations– insensitive (may be normal in acute dyspnea) • The % of Oxygen saturation does not always correspond to PaO2 • The hemoglobin desaturation curve can be shifted depending on the pH, temperature or arterial carbon monoxide or carbon dioxide levels Arterial Blood Gases • Commonly used to evaluate acute dyspnea • Can provide information about altered pH, hypercapnia, hypocapnia or hypoxemia • Normal ABGs do not exclude cardiac/pulmonary diseases as cause of dyspnea – Remember- ABGs may be normal even in cases of acute dyspnea - ABGs do not evaluate breathing Arterial Blood Gases Normal values – PO2 – PCO2 – Sat O2 – pH – P(A-a) O2 – HCO3 – Base Excess 75-100 mmHg 35-45 mmHg 95-100 % 7.35-7.45 12-20 mmHg 22-26 mEg/l +or-2 Alveolar-Arterial Oxygen Partial Pressure Gradient • A-a O2 gradient measures how well alveolar oxygen is transferred from the lungs to the circulation • P(A-a) O2 = 149 – PaCO2 / 0.8 - PaO2 • N = 2.5 + age X 0.21 (+/-11) • Any parenchymal disease in lungs? • Following measure Respiratory Arrest • • • • • • • • • • Acute myocardial infarction Stroke Foreign body obstruction Drowning Electrical injury Intoxication Excess narcotics Trauma (Tension pneumotorax) Suffocation Severe metabolic acidosis Differential Diagnosis • Four general categories – Cardiac – Pulmonary – Mixed cardiac or pulmonary – Non-cardiac or non-pulmonary Pulmonary Etiology • • • • • • • COPD Asthma Pulmonary thromboembolism Restrictive Lung Disorders Hereditary Lung Disorders Pneumonia Pneumothorax Cardiac Etiology • • • • • • • • CHF CAD MI (recent or past history) Cardiomyopathy Valvular dysfunction Left ventricular hypertrophy Pericarditis Arrhythmias Mixed Cardiac/Pulmonary Etiology • COPD with pulmonary HTN and/or cor pulmonale • Chronic pulmonary emboli • Pleural effusion Noncardiac or Nonpulmonary Etiology • • • • • • Metabolic conditions (e.g. acidosis) Pain Trauma Neuromuscular disorders Functional (anxiety,panic disorders, hyperventilation) Chemical exposure Classic Presentations • COPD: Hyperinflation, diminished breath sounds, wheezes, use of accessory muscles of respiration • Pulmonary Edema: JVD, diffuse rales, cardiac gallop, peripheral edema • Upper airway obstruction: Inability to speak, inspiratory wheeze, diminished breath sounds Worry!! • Confusion, agitation, loss of consciousness • Diaphoresis, cyanosis, bradycardia, hypertension Goals of treatment • Etiology treatment! • PaO2 > 60 must be • SO2 > 90 must be Havayolu Kardiyak Akc Plevra GDuvarı Vasküler Nöromus küler Çeşitli HY kitle LV yetm Astım Pnx PE SVO Anemi Yb cisim MI KOAH Pl eff. Hava emb Frenik sinir paralizi Met asidoz Anjioödem Perikardit / P tamponad Pnömoni Pl yapışıklık Yağ emb Guillain-Barre Syn. Şok HY stenozu HT kriz P ödem G Duv yaralanmaları Amn sıvı emb Botulizm Düşük kard out-put Broşektazi Aritmi P kontüzyon Abd distansiyon Pulm HT Nöropati Hipoxi Trakeomalazi Miyokardit Atalektazi Kifoskolyoz Veno-okluziv hastalık miyopati CO intox KMP Alveolit Pectus excavatum Sickle-cell MetHb İntrakard şant P fibrozis Gebelik Vaskülit Ateş, hiper/hipotiroi di, LV çıkış obst ARDS AV fistül psikiyatrik Kapak bzk sarkoidoz