Twin Cities District Dietetic Association Meeting November 9, 2010 Kim Bihm, RD, LD, CDE Mary Murphy, RN, MA, CWOCN Objectives Identify anatomy and physiology of skin Describe prevention strategies to reduce incidence of pressure ulcers Describe an interdisciplinary approach to prevention and treatment of pressure ulcers Define nutritional treatment modalities for wound healing. Why should we care? Complications to patients Lead to pressure ulcers Painful Infection Quality of Life Why should we care? Cost Hospitalizations Health care workers Skin Care Products Reduction in payment from regulatory bodies Incidence of Pressure Ulcers (PU) Data from the NPUAP Volume: 1-3 million people in US develop PU/year Mortality: 60,000 people die from PU complications/year Quality of Life: PU reduce quality of life due to pain, treatments, increased length of institutional stay, etc. Finances: Cost of treating PU ranges from 5-8.5 billion dollars/year Legal: 87% of verdicts from NH cases goes to Plaintiff Average award is $13.5 million Highest award is $312 million in one case! Clinical Practice Guidelines by NPUAP/EPUAP: Evidenced-Based Practice Best scientific research available Systemic review of literature Provides tools for best judgment Allows decision-making on more than “expert opinion” alone. DOES NOT dictate practice or replace clinical reasoning or judgment – it ENHANCES these! These are guidelines Policies are absolute An interdisciplinary approach to prevention and treatment of pressure ulcers • Hospital skin team – Registered Dietitian – Wound, Ostomy, Continence nursing – Occupational Therapy/Physical Therapy – Physicians – primary/specialty • Plastic surgery – RN staff – Respiratory Therapy – Education staff – Nursing Manager – Pharmacist Interdisciplinary Approach All disciplines need to assess for risk and put prevention interventions into place: Occupational Therapy Pressure Ulcer Protocol Nutrition C-collar inspection orders Nursing Physician High protein, high calorie diet with snacks and supplements Physical Therapy Wheelchair cushion pressure mapping Avoiding shear during transfers Cognitive screening Assistive Technology Speech Therapy Memory assessment Cognition Communication Assistive Technology Prevention: Risk Assessment Co-morbidities Previous PU Smoking hx Long OR time Long ED stays Critically ill – ICU= 4x more Wheelchairs Obese/thin Guidelines to Preventing Pressure Ulcers Combination of Risk Assessment + Skin Inspection + Clinical Judgment Reassess RISK Upon admission At regular frequency Change in condition Skin Inspections Head to toe inspection regularly Individualized plan of care Use Interdisciplinary Approach MD, Nutrition, PT/OT, Speech Therapy Skin Inspection Interdisciplinary Approach Risk Assessment Development of Prevention Strategies Anatomy and Physiology of Skin Largest organ of the body Weight: up to 15% of body weight – about 6 pounds Size: Average adult – 3000 square inches Receives 1/3 of body’s circulating blood volume Constantly exposed to changing environments Has capability to self-regenerate Skin Layers: Epidermis Outermost layer made of epidermal cells Thin and avascular Regenerates every 4-6 weeks Melanocytes reside in epidermis Melanin is pigment responsible for color of skin Skin Layers Dermis Thicker layer Contains: blood vessels hair follicles lymphatic vessels sebaceous glands sweat and scent glands nerve endings Skin Layer: Dermis •Collagen: •Major structural protein •Gives skin strength •Anchors dermis to hypodermis layer •Elastin: •Responsible for skin recoil and resiliency •Allows skin to stretch Skin Layers: Hypodermis Subcutaneous Tissue Composed of adipose and connective tissue Filled with major blood vessels, nerves and lymphatic vessels Attaches dermis to underlying structures Provides insulation and cushioning to body Acts as a ready reserve of energy Functions of Skin Body Image Maintenance of body form Appearance, attributes and expression Sensation Abundant nerve receptors in skin Touch Heat/Cold Pain Pressure Moisture Functions of Skin Regulation of body temperature 98.6 F / 37 C Thermoregulatory mechanisms: Circulation Blood vessels dilate to dissipate heat Blood vessels constrict to shunt heat to body organs Sweating 2-5 million sweat glands Functions of Skin Protection Safety against sunburn Melanin in the epidermal cells protects against ultraviolet light Metabolism Vitamin D formation Presence of sunlight This activates the metabolism of calcium and phosphate and minerals (important in bone formation) Functions of Skin Protection Barrier to germs and poisons Normal floral = Staph Aureus Diphtheroids Gram neg bacilli NOT Candida – That comes from GI tract Chemical defenses Sweat, oils, wax from skin glands contain lactic acid and fatty acid These acids make skin pH acidic to kill bacteria and fungi Functions of Skin Maintenance of water balance Prevents loss of water through evaporation <10% moisture – cells shrink = increase invasion of bacteria >30-40% moisture level = maceration Increased permeability Increased risk of injury from friction Theory of pH pH refers to management of acid or base levels Acidic is 0-6 Neutral is 7 Basic is 8-14 Rain is 5.6 Seawater is >7 Milk is <7 Gastric juices are acidic Saliva and blood are neutral Skin pH Skin pH is 4-6.8 with mean of 5.5 Depends on area of body Urine, stool, soap and frequent cleansing will increase pH to more basic levels Pooled urine changes pH to 7.1 – or alkaline shift = this contributes to overgrowth of bacteria Patients with fecal incontinence are 22x more likely to develop pressure ulcers Skin Changes Age-Related changes: Functions decline Epidermal/dermal junction flattens Decreases skin strength Increases risk for tearing Melanocytes shrink (decrease in volume) Increases sensitivity to sun Skin Changes Age-Related changes: Decreased sweat production Leads to increased dryness and flaking Nutrition changes Medications Guidelines to Preventing Pressure Ulcers Skin Inspections Checking all bony prominences Check under skin folds Check under medical devices Check where there is limited sensation Educate professional staff on skin conditions for early identification Technique for blanching response How to assess warmth, edema, and induration Set time frame for on-going inspections What are Pressure Ulcers? Pressure ulcer definition: A pressure ulcer is localized injury to the skin and/or underlying tissue usually over a bony prominence, as a result of pressure or pressure in combination with shear. Different from: Neuropathic ulcers Trauma injuries Arterial ulcers Venous ulcers Stage I Pressure Ulcers Intact skin with non- blanchable redness of a localized area- usually over a bony prominence. Stage II Pressure Ulcers Partial thickness loss of dermis presenting as a shallow open ulcer with a red pink wound bed, without slough. May also present as an intact or ruptured serum-filled blister. Stage III Pressure Ulcers Full thickness tissue loss. Subcutaneous fat may be visible but not bone, tendon, muscle. Slough may be present, but does not obscure the depth of tissue loss. May include undermining and tunneling Stage IV Pressure Ulcers Full thickness tissue loss with exposed bone, tendon or muscle. Slough/eschar may be present. Often includes undermining/tunneling. Unstageable Pressure Ulcers Full thickness tissue loss in which actual depth of ulcer is completely obscured by slough and/or eschar. Suspected Deep Tissue Injury Purple or maroon localized area of discolored intact skin or blood-filled blister due to damage of underlying soft tissue from pressure/ shear. Iatrogenic Damage: Pressure Injury from Medical Devices Assess for Risk by RN Braden Risk Assessment (by Barbara Braden) Reliable research based risk assessment tool Sensory Mobility Activity Friction/Shear Nutrition Moisture Risk due to Sensory Impairment Can they feel? Prevention: If they can’t feel – someone must look at skin!! Check under devices Check for proper fitting shoes and socks Need redistribution mattress Risk due to Mobility Impairment Can they move themselves? Prevention: Must be turned every 2 hours Must be trained in proper pressure relief Must have pillows elevated Risk due to Activity Can they walk? Are they bedfast? Chair fast? Prevention: Do they have a PT/OT consult? Do they have a proper fitting wheelchair cushion? Must have training in pressure relief Risk due to Friction and Shear Are they sliding in bed or wheelchair? Prevention: Watch transfers from w/c to bed If concerned, get PT/OT consult Manage spasticity Report concerns to MD Keep knee gatch up in bed to prevent sliding in bed SKIN INSPECTIONS: Bony Prominences To Check Support Surfaces How to make sense of the confusion???? What Do We Know- Evidence Pressure = Force/Area Pressure is caused by perpendicular force = Treatment = pressure redistribution Pressure redistribution = depth of pressure without bottoming out Shear is parallel force = Treatment = prevent sliding Features of Support Surfaces Air Fluidized A feature that provides pressure redistribution via a fluid-like medium created by forcing air through beads as characterized by immersion and envelopment Features of Support Surfaces Low Air Loss A feature that provides a flow of air to assist in managing the heat and humidity (microclimate) of the skin. Features of Support Surfaces Foam Elastic foam or Visco-elastic foam Features of Support Surfaces Gel A feature that is a solid, jelly-like material that can have properties ranging from soft and weak to hard and tough. It is a soft molding layer that contours around the shapes and bumps of the human body. Consider gel products for zone redistribution Features of Support Surfaces Alternating Pressure A feature that provides pressure redistribution via cyclic changes in loading and unloading as characterized by frequency, duration, amplitude and rate of change parameters. Repositioning – Evidence A Relieve/redistribute pressure 30 degree side lying is important Alternate positions Avoid shear Avoid lying on medical devices Avoid slouching in w/c – use footplates Avoid HOB elevation: HOB = shear/pressure Elevate heels Consider “zone” positioning changes Consider: Every layer on top of surface changes the surface support Think of chux/linen/briefs = change in performance of bed Wheelchair cushions Check w/cushion – pressure mapping Check chair position Back tilt w/ legs up Upright w/ foot rests Limit sitting time Risk due to Moisture Is their skin too moist? Prevention: Avoid plastic diapers Avoid extra pads that retain heat Skin barrier protection is critical Moisture Prevention Goals: Promote health of epidermis Maintain intact epidermal barrier Eliminate/minimize exposure to irritants Treat infection if present Create environment for healing damaged skin Prevention Strategies Keep skin clean, dry and protected Toileting program Structured bowel program Gentle cleansing-avoid mechanical irritation Balanced pH cleanser + moisturizer or humectants Soft cloth vs. wash cloth Pat dry Skin protectant/barrier Dimethicone Petrolatum Zinc Moisturizer - Emollient Products: Underpads Briefs/ underpads Needs to be highly absorptive Needs to quickly wick moisture away from patient Plastic/cloth absorptive products are occlusive Trap perspiration = increase heat/moisture = increase skin damage Typically made of 3 layers: Water-permeable cover next to skin Absorbent core (holds in moisture increasing heat) Water proof backing Look for product that “wicks” moisture away (polymer) Adhesive tabs seal and reseal as needed for easy inspection Durable – resist tearing Different sizes Breathable Products Consider containment products External catheters Indwelling Suprapubic catheters Intermittent Catheters Fecal pouches Fecal tubes Complex Process of Wound Healing Risk due to Nutrition Are they eating/drinking enough? Prevention: Need RD consult for any Braden score of 1 or 2 High protein diet Importance of snacks and supplements Good hydration Multivitamins/ minerals Labs: prealbumin Nutrition Screen for nutritional deficiencies Send nutrition consults Monitor for signs of dehydration – I/Os Monitor weight changes Highlight Braden Subcategory of Nutrition Prioritize protein intake Specific Recommendations Offer high protein supplements in addition to usual diet. Plan for supplement 60 minutes between meals Resource for Nutrition The Role of Nutrition in Pressure Ulcer Prevention and Treatment: National Pressure Ulcer Advisory Panel White Paper Patient/Caregiver Education Causes/ risk factors for PU development Ways to minimize risk: Regular inspections Prevent friction/shear Routinely turn/reposition Avoid use of rings, foam cut outs, donut-type devices Maintain adequate nutrition and fluid intake Monitor for weight loss, poor appetite Promptly report health care changes to providers TREATMENT of Pressure Ulcers: GOLD STANDARD of Wound Healing: Good signs of healing by 2 weeks 30% healing at 4 weeks Full closure at 12 weeks Guidelines to Treatment of Pressure Ulcers Principles of wound healing: Eliminate cause – moisture, pressure, shear , friction Wound cleansing Keep infection free Topical treatments Moist wound healing Protect periwound Refer as necessary for debridement Manage nutrition Repair of Skin Damage Repair of partial thickness skin damage Regeneration Damage is confined to epidermal and superficial dermal layers Epithelial cells will reproduce Trauma triggers inflammatory response Erythema, Edema, Serous exudate Epidermal resurfacing begins Day 7 - new blood vessels sprout Day 9- Collagen fibers are visible Collagen synthesis continues until about day 10-15 Repair of Skin Damage Repair of full thickness skin damage Scar formation Damage is deeper – to deeper dermal structures (hair follicles, sebaceous glands and sweat glands), subcutaneous tissue, muscle, tendons, ligaments, bone Damage is permanent. Healing is done by primary or secondary intention Primary intention – surgical closure Secondary intention –scar formation Repair of Skin Damage Scar formation process is complex with several phases: Hemostasis phase Clot formation Inflammatory phase Clean up phase Takes 3-4 days usually Proliferation phase Vascular integrity restored New connective tissue is growing Granulation tissue growth Wound contraction Maturation / Remodeling phase Wound Management Strategies Wound cleansing – Evidence C Cleanse wound and periwound with each dressing change Provide enough pressure to remove debris but not cause trauma (trauma = increase risk of infection) Product: Ok to use water/NS/ wound cleanser (reduces friction with surfactant) Ok to shower open wound Wound Management Strategies Manage wound infections Contamination Non-replicating organisms Colonized Bacteria in wound bed Organisms are attached and replicating Not affecting the environment Common organisms: staph and pseudomonas Critically colonized Wounds with more than 100,000 organisms will not heal Perpetual inflammatory phase Wound culture recommended at this point- 70% MRSA now Infection Invasion of the soft tissue Clinically ill Dose x virulence/ host response Wound Management Strategies Topical Agents Dakin’s solution – Sodium hypochiorite – 0.25% Good for gram - & + - best on staph Bleach w/ chlorine active ingredient Protect periwound skin with petroleum Acetic Acid – 0.5% Good for gram - & + - Best on pseudomonas Cadexomer Iodine Good for gram - & +, and anaerobes No resistance noted Effective in 48 hours Absorptive Limit to 2 weeks – risk of dermatitis Wound Management Strategies Topical Agents Silver Silver Sulfadiazine ointment Good for gram -, Klebsiella, Pseudomonas Seeing increased resistance to silver Honey-broad antimicrobial coverage Important to consider due to increase in resistance Good for gram -& +, pseudomonas, e-coli Change pH of wound tissue Don’t use if allergy to bee-stings Hydrophera blue- Broad antimicrobial coverage Polyvinyl alcohol sponge impregnated Methylene Blue and Gentian Violet Good for broad spectrum coverage- including MRSA and VRE Dressing is highly absorptive so good for highly exudative wounds Wound Management Strategies Topical antimicrobials – for tissue organisms Bactoban is resistant now Neomycin/Neosporin – NO NEO! = contact dermatitis Gentamycin = nephrotoxicity and resistance Bacitracin is ok still Good for gram + Resistance is rare Wound Management Strategies Systemic antibiotics for: Bacterimia/ sepsis Advancing cellulitis Osteomyelitis 95% of bone exposed is + for osteo Caution: MRSA is very virulent Cellulitis to pneumonia in 24-48 hours Wound Management Strategies Debride the pressure ulcer of devitalized tissue Debridement options: Surgical Conservative sharp Wound Management Strategies Debride the pressure ulcer of devitalized tissue Debridement options: High pressure fluid irrigation Ultrasonic Wound Management Strategies Debride the pressure ulcer of devitalized tissue Debridement options: Mechanical Autolytic Enzymatic Wound Management Strategies Debride the pressure ulcer of devitalized tissue Debridement options: Maggot Therapy Wound Management Strategies VAC Therapy Wound Management Strategies Electrical Stimulation Wound Management Strategies Hyperbaric Oxygen Therapy Wound Management Strategies Living Skin Equivalents Wound Management Strategies Flap Surgery – Umar Choudry, MD Pre-Surgery Preparation Interdisciplinary Teamwork !!!!! SCI Provider: Pre-op medical clearance Anticoagulant assessment Transfuse if Hgb < 8 Spasticity management Patient needs to lie straight for 3-4 weeks WOC Nurse: Prep wound – VAC therapy Pre-Surgery Preparation Nutrition Consult Assess nutrition needs Pre-albumin goal of >20 before surgery Zinc at least in normal range goal Supplemental vitamin C and multivitamin Speech Therapy Consult (if hx of dysphagia/aspiration) Assess risk of aspiration post-op May need to consider non-oral feeding alternative Pre-Surgery Preparation Physical Therapy and Occupational Therapy pre-op consults for baseline assessments Physical Therapy Transfers Seating/Positioning including pressure mapping ROM Tone Occupational Therapy ADLs Adaptive Equipment needs Functional transfers Seating/Positioning Post-op Course 3 weeks on Air Fluidized bed Week 4 Switch to low air loss bed Begin stretching Week 5-6 Sitting program Discharge week 6-7 VAMC -Mpls SCI Unit Outcomes: 2009 to present: 16 flap surgeries ZERO losses of the flap The Registered Dietitian’s Role in Wound Healing Pressure Ulcer Prevention • Global expert (NPUAP/EPUAP) consensus SUPPORTS nutritional assessment as part of a comprehensive interdisciplinary approach to preventing pressure ulcers! Pressure Ulcer Prevention Nutritional assessment can identify under nutrition, protein energy malnutrition, and unintentional weight loss (conditions that can contribute to the development of pressure ulcers or delay healing of pressure ulcers. NPUAP white paper 2009 Risk for Pressure Ulcers Unintentional weight loss Under nutrition Protein energy malnutrition Low BMI Inability to eat independently Cachexia Hyper metabolism Risk for Pressure Ulcers Diabetes Maintenance of proper glycemic control is vital to the healing process. Blood glucose may be influenced by non-nutritional factors such as illness, stress, infection, wounds, etc Risk for Pressure Ulcers Diabetes Calorie needs are increased to promote wound healing. The major fuel source for collagen synthesis is carbohydrates (~55% of calories should come from carbohydrates) Risk for Pressure Ulcers Diabetes Medications may need to be adjusted to accommodate increased carbohydrate intake. IV insulin drip may be used to control blood sugars post-op Under Nutrition Problems chewing and/or swallowing Decrease ability to feed self Decreased appetite Advanced age Unintentional weight loss Unintentional Weight Loss Can lead to: Impaired immune system Decreased serum albumin & prealbumin Decreased ambulation Weakness Development of pressure ulcers Non-healing pressure ulcers Loss of Lean Body Mass Defined as the mass of the body minus the fat that is metabolically active and accounts for ~75% of normal body wt. When <10% of LBM is lost, wound healing has priority for protein substrate When >10% of LBM is lost, the stimulus to restore LBM competes with the wound for protein When >20% of LBM is lost, correction of the LBM takes precedence and wound healing stops Medscape Today The Stress Response to Injury and Infection...: • The Wound Healing Process and the Stress Response Prevention: Risk Assessment Co-morbidities Diabetes Renal disease Immunosuppression Malnutrition Consultation of RD When to consult the Registered Dietitian: When patient is identified as: Being at risk for pressure ulcers Braden Risk Assessment score less than 19 Braden Risk Assessment- Nutritional sub-score of 1 or 2 Existing pressure ulcer Newly discovered pressure ulcer within 24 hours Worsening of a ulcer or with an ulcer not progressing through the normal stages of healing Consultation of RD Inadequate oral intake as shown by: • NPO status or clear liquid diet for >3 days • Eating <75% of meals for >3 days • Failure to consume nutritional supplements >3 days Difficulty chewing and/or swallowing Unable to eat independently Significant weight loss >5% in 30 days or >10% in 180 days Example of Nutrition Consult Template Braden Risk Assessment Nutrition Subcategory 1. Very Poor Never eats or completes meal 2 servings or < of protein Poor fluid intake 1/3 of any food offered or NPO or IV fluids/clear liquids for > 5 days NEEDS NUTRITION CONSULT Braden Risk Assessment Nutrition Subcategory 2. Probably Inadequate Eats ½ of any food offered Protein: 3 servings of meat or dairy daily Occasional intake of supplement or tube feeding or liquid diet less than requirements How often is TF turned off for activities? NEEDS NUTRITION CONSULT Braden Risk Assessment Nutrition Subcategory 3. Adequate Eats > 50% of most meals 4 servings of protein daily Occasionally refuses meal but takes supplement or tube feeding or TPN meets needs Braden Risk Assessment Nutrition Subcategory 4. Excellent Eats most meals and never refuses a meal Eats 4 or more servings of meat and dairy daily Doesn’t require supplements Nutrition Care Process Nutritional Assessment Data Food / Nutrition history Lab data, medical test and procedures Anthropometric measurements including weight history Physical examination findings (i.e. brittle nails, thinning hair, fragile & thin skin) Patient History Nutritional Assessment Data Estimating calories, protein, fluid, vitamin and minerals Adequacy of po intake (past and current) Barriers in meeting optimal nutrition -swallowing difficulties -chewing problems Cognitive deficits-ability to feed self Braden Risk Assessment scale, BMI, weight changes Individual goals and wishes of the patient Nutrition Diagnosis PES Statement ~60 nutrition diagnoses within 3 domains: 1- Clinical 2- Intake 3- Behavioral- Environmental Identifies a specific nutritional problem that the Registered Dietitian is responsible for treating ADA Nutrition Diagnosis and Intervention: Standardized Language for the NCP Example: Inadequate energy intake related to decreased appetite and dysphagia as shown by a significant wt loss of 6% in the past month and leaving >25% of meals uneaten for the past 4 days. Nutrition Interventions Specific to the nutrition diagnosis Interventions: • Strategies to positively change: • a nutrition-related behavior • environmental condition • health status for the patient ADA Nutrition Diagnosis and Intervention: Standardized Language for the NCP Nutrition Interventions Develop individualized interventions with the patient and the family Educate the patient and their family Liberalize the diet as much as possible Obtain food preferences Allow flexibility Discuss high protein/high calorie snacks/supplements with patients Nutrition Monitoring and Evaluation Monitoring, measuring , and analyzing patient outcomes relevant to the nutrition diagnosis, plan of care and goals Frequent follow up may be necessary when there is a change in condition or the wound is not healing Check in with WOC nurse frequently! Nutrition Monitoring & Evaluation Monitor po intake of meals, snacks, and supplements Monitor weight and weight changes Monitor nutritional labs but keep in mind that they may not always reflect the current nutritional status Biochemical Assessment Hepatic Proteins: Serum albumin Serum prealbumin Serum transferrin Hepatic Proteins and Nutrition Assessment; Journal of the American Dietetic Association 2004 Nutrition Labs: Albumin and Prealbumin Helpful Indicators of: Morbidity and mortality Systemic illness Help identify patients who may become malnourished Helpful for trending Careful of interpretation Negative acute phase reactant i.e. Increase illness = Decrease in lab values Decrease after surgery Decrease with infection, stress and inflammation Increases with dehydration Nutritional Requirements for Wound Healing National Pressure Ulcer Advisory Panel White Paper 2009 Calorie Requirements Use 30-35 calories/kg body weight as a guide Calorie needs may be higher in patients who are underweight or have had a significant weight loss Calorie needs may be higher in individuals with co-morbid medical conditions such as COPD, cancer, acute spinal cord injury, traumatic brain injury, hemodialysis, etc Calorie Requirements Weight loss should not be a goal in overweight or obese patients with pressure ulcers Caloric intake may need to be lowered in patients with chronic SCI who start to have an undesired weight gain. Weights must be monitored closely Protein Requirements Use 1.25-1.5 gm protein/kg body weight as a guide Protein needs may be greater than 1.5 gm/kg body weight if the patient has multiple pressure ulcers, larger stage 3 or 4 pressure ulcers, has pressure ulcers that are draining, or if lower protein levels are not promoting healing Protein Requirements Protein needs should be individualized using clinical judgment Ensure that adequate fluids are being provided or consumed and that renal function is preserved Caution should be exercised when determining protein needs in patient’s with impaired renal function and in the elderly Fluid Requirements 30-35 ml/kg body weight per day or 1 ml/calorie is usually adequate Fluid needs will be higher in patients with diarrhea, vomiting, profuse sweating, elevated temperature and/or in those experiencing considerable amounts of wound drainage or on VAC therapy Fluid Requirements Patients receiving higher amounts of protein may also need higher amounts of fluid . Those using air-fluidized beds may require an additional 10-15 ml per kg of body weight per day Monitor for signs symptoms of dehydration Fluid Requirements Interventions may need to be considered if fluid intake is inadequate (i.e. initiation of IV fluids, increase water flushes in patients receiving tube feedings) If fluid restriction is medically necessary, then a minimum of 1500 ml daily is suggested Vitamin and Mineral Requirements Other than a MVI, additional supplements or individual vitamin and minerals should only be recommend IF the patient is known to have a diet deficient in that vitamin or mineral and/or shows signs and symptoms of a clinical deficit Vitamin and Minerals Ascorbic Acid -enhances collagen production -increases formation of blood vessels -supports immune system Vitamin and Minerals Vitamin A -stimulate collagen production -enhances cell production Vitamin and Minerals Vitamin E -stabilizes cellular membranes Vitamin and Minerals Zinc -DNA and protein synthesis -Cellular production -Collagen formation If patient is known to have a diet insufficient in zinc, suggest supplementing with 40 mg of elemental zinc/day in divided doses for a 2-3 week period of time Vitamin and Minerals Arginine -stimulates protein production -supports immune function -stimulates collagen production At this time, safe maximum doses are not known More research is needed to verify what effects it has on healing of pressure ulcers Vitamin and Minerals Glutamine -decreases protein breakdown -supports immune function -stimulates cell growth and reproduction At this time supplemental use of arginine and glutamine is controversial and more research is needed. Supplementation is not recommended at this time (ADA Nutrition Care Manual 2009) Feeding Practices Provide assistance with meal set-up and feeding as needed Consult a Speech-Language Therapist and/or Occupational Therapist if a patient is found to have swallowing difficulties or problems self-feeding Feeding Practices Encourage patients to eat in a common patient dining area to promote socialization and allow for greater supervision of diet tolerance, food preferences, and assistance needs Provide therapeutic nutritional supplements, food fortifiers, and enhanced foods as appropriate. Supplements may be high calorie, high protein, and/or have some other component known to support or enhance wound healing Nutrition Support Consider alternate method of nutrition support if oral intake inadequate and if patient/family is agreeable. Consult Nutrition Support Team or Registered Dietitian (RD) for enteral or parenteral nutrition support recommendations. If the gut is working, the ideal route for feeding is enteral nutrition support. Education Educating the patient and family on the role of nutrition in wound healing is essential Give examples good sources of protein and how to incorporate them into their meals and snacks/supplements Provide written materials on nutrition and wound healing Nutrition is One Aspect of Care 134 In Conclusion Prevention is the best treatment Provide consistent, adequate nutrition Provide individual plan of care Liberalize diet as much as possible Educate patient, family, and staff Provide frequent follow up Working as a team is essential Questions Follow Up Please feel free to e-mail us if you would like additional information or resources: Kimberly.Bihm@va.gov Mary.Murphy5@va.gov References: Institute For Clinical Systems Improvement: Pressure Ulcer Prevention and Treatment Protocol, 2010 at ICSI.org Pressure Ulcer Prevention and Treatment Quick Reference Guide, 2009 – Developed by the NPUAP/EPUAP, npuap.org National Database of Nursing Quality Indicators (NDNQI) at nursingquality.org References Guideline for Prevention and Management of Pressure Ulcers – WOCN Clinical Practice Guideline Series American Dietetic Association-Nutrition Care Manual 2009 ADA Nutrition Diagnosis and Intervention: Standardized Language for the NCP The Role of Nutrition in Pressure Ulcer Prevention and Treatment: National Pressure Ulcer Advisory Panel White Paper 2009 http://www.npuap.org/Nutrition%20White%20Paper%20Website% 20Version.pdf