Erectile Dysfunction John Ewan Sandyford Glasgow Overview Epidemiology Anatomy and Physiology History Examination Investigations Treatment Definition of ED DSM-IV (American Psychiatric Association, 2000) Persistent or recurrent inability to attain, or to maintain until completion of the sexual activity, an adequate erection The disturbance causes marked distress or interpersonal difficulty The erectile dysfunction is not better accounted for by another Axis I disorder (other than a sexual dysfunction) and is not due exclusively to the direct physiological effects of a substance (e.g. a drug of abuse, a medication) or a general medical condition Epidemiology Massachusetts Male Aging Study, Feldman et al. J Urol 1994; 150:54-61 Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 7 Anatomy and Physiology of erection Reproduced from Carson C, Holmes S, Kirby R. Fast Facts- Erectile Dysfunction. Oxford: Health Press Limited; 2002: 8 Anatomy and Physiology of erection Parasympathetic nerves S2-4 mediate erection Sympathetic nerves T11-L2 control ejaculation and detumescence Smooth muscle relaxation – Nitric oxide diffuses into cavernosal smooth muscle cells, activates Guanylate cyclase converts guanosine triphosphate to cGMP resulting in smooth muscle relaxation. Effect of cGMP stopped by Phosphodiesterase type 5 which exists primarily in corpora cavernosa. Veno-occlusive Mechanism Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 :12 History Detailed description of problem, is it ED? Causative factors Sexual desire/libido Ejaculatory disorders Impact on quality of life and on relationship Expectations of treatment Clues differentiating psychogenic from organic causes Psychogenic – – – – – – – Sudden onset Situational Normal waking and nocturnal erections Normal erection with masturbation Relationship problems Life event Anxiety, fear, depression Organic – – – – – Gradual onset All situations Reduced or absent waking and nocturnal erections No erection with masturbation Penile pain Relationship issues Current relationship status Length of relationship Previous sexual partners and relationships Partner issues e.g. menopause/pain/cancer History Medical Surgical Psychiatric Medication Smoking Alcohol Recreational drug use Arteriogenic Cause of ED Hypertension Smoking Diabetes Hyperlipidaemia Peripheral vascular disease Blunt perineal or pelvic trauma Pelvic irradiation Neurogenic causes of ED Lesions of medial preoptic nucleus, paraventicular nucleus, hippocampus Spinal trauma Myelodisplasia (spina bifida) Pelvic surgery/radiotherapy Multiple sclerosis Intervertebral disc lesion Peripheral neuropathies – – – Alcohol Diabetes HIV Psychogenic and Psychiatric causes Anxiety Loss of attraction to partner Relationship difficulties Stress Depression Psychogenic ED Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 33 Endocrine causes of ED Hypogonadism – – – Low testosterone Raised SHBG Raised Prolactin Thyroid disease Drugs associated with ED Antihypertensives – – – Antidepressants – – – Thiazides B blockers Centrally acting drugs Tricyclics MAO inhibitors SSRI Anticholinergics – Atropine Antipsychotics – Anxiolytics – Phenothiazines Benzodiazepines Psychotropic drugs – – – – Alcohol Opiates Amphetamines Cocaine Examination Blood pressure Peripheral pulses, palpate for AAA Testes size and consistency Secondary sexual characteristics Penis for Peyronie’s plaques, phimosis ED and Coronary Artery Disease Generalised atherosclerosis Penile arteries smaller than coronary arteries ED pre-dates coronary artery disease Man with ED and no cardiac symptoms is a cardiac patient until proven otherwise Investigations Fasting glucose and lipids Morning testosterone and SHBG If testosterone is low or borderline repeat with Prolactin, FSH and LH Thyroid function PSA Specialised Investigations Vascular studies – – – Young patients with primary ED History of trauma e.g. penile fracture Patients unresponsive to medical therapies Treatment of ED General Measures Smoking cessation Reduce alcohol Weight loss Exercise Endocrine Disorders Hypogonadism Hyperthyroidism Hyperprolactinaemia Endocrinology referral Psychosexual therapy Even if cause of ED is physical the patient will develop psychosexual issues Performance anxiety Sensate focus exercises Relationship counselling Drugs for ED Oral agents – – Intra-cavernosal – Centrally acting dopamine-receptor agonist Apomorphine (discontinued in UK) Phosphodiesterase type 5 inhibitors Prostaglandin E1 Alprostadil Intra-urethral – Alprostadil PDE5 inhibitors Sildenafil (Viagra) 25mg, 50mg, 100mg – – – Tadalafil (Cialis) 10mg, 20mg – – – 30 minutes before sexual activity 36 hour window Absorption not affected by food Tadalafil (Cialis) 5mg – 1 hour before sexual activity 4-6 hour window Absorption delayed by fatty meal daily Vardenafil (Levitra) 5mg, 10mg, 20mg – – – 30-60 minutes before sexual activity 4-6 hour window Absorption delayed by fatty meal PDE5 Physiology Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 40 PDE5 Inhibitors Side Effects Facial flushing Headache Nasal congestion Dizziness Dyspepsia Visual disturbance (blue halo) Priapism Non-arteritic anterior ischaemic optic neuropathy PDE5 Contraindications Recent cardiovascular event Nitrates Hypotension Anatomical deformity – Angulation, cavernosal fibrosis, Peyronie’s Predisposition to prolonged erection – – – Sickle cell disease Multiple myeloma Leukaemia PDE5 Drug Interactions Nitrates – – – Cytochrome P450 inhibitors – – Glyceryl trinitrate, isosorbide mono or dinitrate Chest pain after taking Sildenafil/Vardenafil no nitrates 24 hours, Tadalafil no nitrates 48 hours Recreational amyl nitrate (Poppers) Protease inhibitors especially Ritonavir use very small dose Cimetidine, Ketoconazole, Erythromycin Alpha blockers Intracavernosal Injections Alprostadil (Caverject, Viridal) 5-40 mcg – – – – Independent of intact nervous system Manual dexterity, adequate vision, training Contraindicated: bleeding disorders, sickle cell anaemia, multiple myeloma, leukaemia Side effects: penoscrotal pain, haematoma, fibrosis at injection sites, priapism Papaverine, Phentolamine, Aviptadil (vaso-intestinal peptide) been used sole or with Alprostadil Intracavernosal Injections Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 53 Intraurethral Alprostadil (Muse) 125mg, 250mg, 500mg,1g – – – Pellet inserted with applicator Massage penis to aid absorption Side effects: Penile pain, dizziness, priapism rare Intraurethral Alprostadil Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 55 Vacuum Devices Blood trapped in intracorporal and extracorporal compartments of penis Constricting ring at base of penis Cyanosis, oedema, cold Pivots at base below ring Maximum time 30 minutes Vacuum devices Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 61 Penile Prostheses Semi-rigid rods 2 piece inflatable prosthesis 3 piece inflatable prosthesis with abdominal reservoir Risks – – – – Infection Destroys corpora cavernosa Erosion and extrusion Mechanical failure Penile Prosthesis Reproduced from Carson C,Holmes S,Kirby R. Fast Facts-Erectile Dysfunction. Oxford: Health Press Limited; 2002 : 66 NHS Prescription for ED Diabetes Multiple sclerosis Parkinson’s Disease Poliomyelitis Prostate cancer Prostatectomy incl TRP Radical pelvic surgery Severe pelvic injury Renal failure – – On dialysis Transplant Single gene neurological disease Spinal cord injury Spina bifida Receiving NHS Rx 14/9/1998 Severe distress Private Prescription Pharmacy costs vary Sildenafil 100mgX4 £25-£40 Pharmacy2U £25 Conclusions ED is a common problem Impact on patient and partner/s Overlap of psychological and physical May be initial presentation of diabetes or coronary artery disease Good range of safe and effective therapies If YOU don’t ask your patient may be too embarrassed to tell you