Chapter 4 Injury Mechanism and Classification of Injury Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomic Foundations • Anatomic position Anatomic position • Joint movement – Sagittal plane – Frontal plane – Transverse plane • Directional terms • Movement Terms Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Mechanism of Injury • Mechanism of Injury (MOI): How an injury occurs • Components used to analyze MOI: – Application of force – Tissue type • Severity of force Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Force • Force: a push or pull acting on a body (e.g., gravity, friction) • Force acting on a body causes: – Acceleration – Deformation • Factors that determine injury: – Magnitude of force – Material properties of tissues involved Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Force (cont’d) • Small load = elastic response • Large load = plastic response • Yield point = load exceeds the ultimate failure point of the tissue resulting in mechanical failure • Anisotropic = material is stronger in resisting force from certain directions than others Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Mechanical Forces - Injury • Compression Mechanisms of injury – Force that crushes tissues • Tension – Force that pulls and stretches tissues • Shearing – Force that moves across the parallel design of the fibers Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Stress • Stress = Force x Surface area affected • Same force over a large area vs. a small area can have very different results Stress Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Injury Types • Acute Injury – Single force – Characterized by a definitive moment of onset – Force = macrotrauma • Chronic Injury – Repeated forces – Characterized by becoming more problematic over time (Gradual onset over time) – Forces = microtrauma Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Check for Understanding! Movements in the sagittal plane include flexion, extension, abduction, and adduction. A. True B. False Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Check for Understanding! Which of the following is a correctly matched pair of terms? (Select all that apply) A. Adduction – movement away from the midline of the body B. Flexion – decreasing an angle C. Extension – increasing an angle D. Plantar flexion – movement of the forefoot toward the shin Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Check for Understanding! When tissues sustain a force, what are the primary factors that determine the occurrence of an injury? (Select all that apply) A. The magnitude of the force B. The direction of the force C. The material properties of the involved tissues D. The length of time the force is applied Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Check for Understanding! What are the three primary mechanical forces that produce injury? Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Soft Tissue • Collagen – Primary component of skin, tendon, ligaments – Protein substance strong in resisting tensile forces – Wavy configuration that allows for an elastic type deformation or stretch but, otherwise, is inelastic Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Soft Tissue (cont’d) Collagen fibers • Elastin – Protein substance in connective tissue – Adds elasticity Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Skin • Epidermis – Multiple layers • Dermis – Loose, multidirectional arrangement of collagen fibers Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Skin Injury Classification Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Skin Wounds • Blisters – Accumulation of fluid between epidermis and dermis – Caused by repeated application of shear in one or more directions • Skin bruises – Accumulation of blood within skin – Results from compression sustained during a blow Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Muscles • Produce skeletal movement and maintain postural alignment • Viscoelastic – Extensibility – Elasticity Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Muscle tissue Muscle (cont’d) • Irritability: ability to respond to a stimulus – Electrochemical – nerve impulse – Mechanical – external blow • Contractility: ability to develop tension – Isometric – Concentric – Eccentric Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Tendons • Muscle to bone • Dense connective tissue with unidirectional bundles of collagen & some elastin • Collagen – parallel arrangement – Helps in resisting high, unidirectional tension loads from the attached muscle • 2X as strong as muscle it serves • Yield point 5-8% in length Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Tendons (cont’d) Collagen arrangements in tendon and ligament tissue Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Contusions • MOI: compression • Can be both deep and superficial • Must be cautious and aware of more severe injuries associated with repeated blows • S&S: – Onset - acute – Ecchymosis: if superficial – Hematoma – Restrictions in ROM – Pain – localized – Swelling – Associated nerve compression Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification for Contusions Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Strains • Damage to muscle or tendon – Key factor: magnitude of force and structure's cross-sectional area • MOI: – Abnormally high tensile force • Most common site for tears: near the musculotendinous junction Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Strains <table 4.4, classifications of strains> Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Muscle Cramps and Spasms • Involuntary muscle contraction • Cramp: – Biochemical imbalance (dehydration) associated with muscle fatigue – Painful – Types • Clonic – alternating contraction/relaxation • Tonic – constant Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Muscle Cramps and Spasms (cont’d) • Spasm: – Reflex action caused by: • Biochemical imbalance or • Mechanical blow to nerve or muscle Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Myositis and Fasciitis • MOI: repeated movements irritate the tissues • Myositis: – Inflammation of muscle tissue (e.g., shin splints) • Fasciitis: – Inflammation of the fascia (e.g., plantar fasciitis) Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Tendinitis and Tenosynovitis • Tendinitis: inflammation of a tendon – Related to aging and degenerative changes – S&S: pain and swelling with tendon movement • Tenosynovitis: inflammation of the tendon sheath – Acute: rapid onset, crepitus, local swelling – Chronic: same as acute, thickened tendon, nodule formation in sheath Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Myositis Ossificans • Mineral deposits in muscle associated with prolonged chronic inflammation – Ectopic calcification – Common site: quadriceps • Calcific tendinitis: mineral deposits in the tendon Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Overuse Injuries • Results from repetitive use • Factors: – Intrinsic – Extrinsic Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Overuse Injuries (cont’d) • Classification – Stage 1: pain after activity only – Stage 2: pain during activity, does not restrict performance – Stage 3: pain during activity, restricts performance – Stage 4: chronic unremitting pain, even at rest Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Considerations of Joints • Articulation of two bones • Classified by structure and function • Structure – Cartilaginous – Fibrous – Synovial Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Considerations of Joints (cont’d) • Function: based on the amount of movement allowed – Synarthoses – Amphiarthroses – Diarthroses Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Diarthrodial Joints • Components – Articular cartilage – Joint (synovial) cavity – Articular capsule – Synovial fluid – Reinforcing ligaments • Intrinsic or Extrinsic Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Diarthrodial Joints (cont’d) Joint components Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Articular Cartilage • Ends of bones covered by hyaline cartilage…solid type of connective tissue • More resistant to deformation than fibrous connective tissue and more resilient than bone • No blood supply; nourished by synovial fluid Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Joint Cavity • Filled with synovial fluid Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Articular Capsule • Cuff of fibrous tissue – Primarily bundles of collagen • Primary function: hold bones together • Inner layer: synovial membrane – Produces synovial fluid that lubricates the joint. Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Synovial Fluid • Functions – Lubricate joint – Reduce friction – Nourish joint Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Ligaments • Bone to bone – Intrinsic – Extrinsic • Maintain anatomical integrity and structural alignment • Collagen and elastin intermixed (contain elastin – more elastic than tendons) – Viscoelastic Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Ligaments (cont’d) • Resists large tensile loads along the long axis of the ligament and smaller loads from other directions – static stabilizers • Fail in fast loading situations • Strongest in their middle and weakest at their ends • Healing process – slow due to a limited blood supply Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Diarthrodial Joints • Plane • Condyloid • Hinge • Saddle • Pivot • Ball-and-socket Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Injury to the Ligament • Compromises the ability of the ligament to stabilize the joint • MOI: – High tensile force • S&S: – Pain; point tenderness; swelling; loss of function; instability Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Sprains <table 4.5, classification of sprains> Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Dislocations and Subluxations • Joint forced beyond normal limits • MOI: tension • Increased susceptibility for chronic or recurrent dislocations • S&S: – Pain – Swelling – Point tenderness – Deformity – Loss of limb function Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Osteoarthritis • Degeneration of articular cartilage • S&S: – Pain – Limited movement • No definitive cause; rather, several contributing factors Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bursitis • Inflammation of bursa • S&S: • Acute or chronic – Localized swelling • MOI: – Point tenderness – Compression – Warm to touch Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Soft Tissue Injury Check for Understanding! The discoloration or swelling outside a joint in the surrounding soft tissue is termed: A. Bruising B. Ecchymosis C. Edema D. Effusion Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Soft Tissue Injury Check for Understanding! The ability of a muscle to be stretched or increased in length is termed: A. Contractility B. Elasticity C. Plasticity D. Extensibility Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Soft Tissue Injury Check for Understanding! Joint capsules are fluid-filled sacs that serve to reduce friction in the tissues surrounding the joints. A. True B. False Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Soft Tissue Injury Check for Understanding! Which of the following statements is true? (Select all that apply) A. A tear of a ligament is referred to as a sprain. B. A muscle spasm is brought on by a biochemical imbalance, sometimes associated with muscle fatigue. C. Overuse injuries are more often attributed to intrinsic rather than extrinsic factors. D. The onset of bursitis can be acute or chronic. Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Soft Tissue Injury Check for Understanding! Strains and sprains that produce moderate discomfort, tenderness, swelling, ecchymosis, detectable joint instability, and/or muscle weakness are categorized as: A. 1st degree B. 2nd degree C. 3rd degree D. Severe Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Bone • Primary constituents: – Calcium carbonate – Calcium phosphate – Collagen – Water Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Bone (cont’d) Bone macrostructure • Structure: – Diaphysis – Epiphysis – Membranes • Periosteum – Medullary cavity – Apophysis Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Bone (cont’d) • Bone growth: – Longitudinal • Continues until epiphysis closes – Diameter • Continues to grow throughout life – New bone formed via the periosteum; bone is resorbed around the medullary cavity • Osteoblasts: form new bone • Osteoclasts: resorb bone Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Bone (cont’d) Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical properties of Bone (cont’d) • Composition – Cortical • Compact bone tissue of high density (low porosity) • Outside • Can withstand greater stress but less strain – Cancellous • Bone tissue of low density (high porosity) • Inside • Can tolerate more strain Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Injury Classifications Bone injury mechanisms Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Injury Classifications (cont’d) • Fracture: Disruption in the continuity of bone • S&S: – Rapid swelling – Ecchymosis – Deformity or shortening of the limb – Precise point tenderness – Grating or crepitus – Guarding or disability Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Injury Classifications (cont’d) • Type of fracture dependent upon: – Force applied – The health and maturity of the bone at the time of injury • Bone susceptible to: – Compression, tension, shear, bending, and torsion Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Types of Fractures Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Stress Fracture • MOI: repeated lower magnitude forces • Can become worse over time • Begins as a small disruption in the outer layers of cortical bone and ending as complete cortical fracture with possible displacement Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Osteopenia • Reduced bone mineral density • Predisposes individual to fracture – Particularly stress fractures • Possible causes: – Amenorrhea, hormonal factors, dietary insufficiencies Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Epiphyseal Injuries • Classifications – Injury to growth plate could result in alteration in normal growth – Acute injury Epiphyseal injuries • Types I-V Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Epiphyseal Injuries (cont’d) • Osteochondrosis – Disruption of blood supply to epiphysis – Idiopathic – Causing necrosis and possible deformity – Example: Legg-Calvé-Perthes disease Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Classification of Epiphyseal Injuries (cont’d) • Apophysitis – Osteochondrosis of apophysis – Idiopathic or traumatic avulsion fracture – Example: • Sever’s disease • Osgood-Schlatter disease Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! In a comminuted fracture, the bone fragments into several pieces. A. True B. False Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! Osteopenia is a condition: A. That is exclusive to an older adult population B. That predisposes an individual to stress fractures C. That only involves females D. That inhibits longitudinal bone growth Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! Epiphyseal injuries can include damage to the: (select all that apply) A. Epiphyseal plate B. Ligaments C. Articular cartilage D. The apophysis Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Nerves • Nervous System – CNS: • Brain • Spinal cord – PNS: • 12 pairs of cranial nerves • 31 pairs of spinal nerves, along with their branches Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Anatomical Properties of Nerves (cont’d) Spinal nerves • Spinal nerves – Roots • Posterior – afferent • Anterior – efferent Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Nerve Injury Classifications • MOI: Tensile or compression force • Neurapraxia (grade 1) – Localized conduction block: temporary loss of sensation and/or motor – Resolves within days to a few weeks Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Nerve Injury Classifications (cont’d) • Axonotmesis (grade 2) – Significant motor and mild sensory deficits – Lasts at least 2 weeks • Neurotmesis (grade 3) – Motor and sensory deficit – Lasts up to 1 year Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Nerve Injury Classifications (cont’d) • Compression: – More complex; dependent upon: • Force magnitude and duration • Direct or indirect • Nerve injuries result in a variety of afferent symptoms – Hyperesthesia – Hypoesthesia – Paresthesia Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Nerve Injury Classifications (cont’d) • Neuralgia – Chronic pain along nerve course • Healing: if completely severed, healing does not occur Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins The Neurological Basis of Pain • Sources – Somatic, visceral, and psychogenic • Nociceptors: produce pain sensation – Mechanosensitive: initiate pain by acute trauma – Chemosensitive: causes persistent pain in chronic injuries and the early stages of healing Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins The Neurological Basis of Pain (cont’d) • Fibers transmitting pain – A fibers – C fibers – T cells • Gate control theory of pain Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins The Neurological Basis of Pain (cont’d) • Factors than mediate pain – Brain production of opioid peptides and endorphins – Cognitive and affective filters • Referred pain – Pain perceived at a location remote from the site actually causing the pain • Radiating pain – Pain felt both at its source and along a nerve Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! The posterior branches are the afferent (sensory) nerves that transmit information from sensory receptors in the skin, tendons, ligaments, and muscles to the central nervous system. A. True B. False Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! ___________ is perceived at a location remote from the site of the tissues actually causing the pain. A. Radiating pain B. Cognitive pain C. Acute pain D. Referred pain Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins Bone Tissue Injury Check for Understanding! Grade II nerve injuries that produce significant motor and mild sensory deficits that last at least two weeks are termed: A. Neurapraxia B. Axonotmesis C. Neurotmesis Copyright © 2011 Wolters Kluwer Health | Lippincott Williams & Wilkins