CARDIOVASCULAR
review cardiac
output & how
inotrophy:contractivity
chronotrophy
it
f(x)
=
want optimal stroke volume!
not
hitting on
types of
Atherosclerosis
-
excess
corrnaryarterdie
x
failure
lipids
eine
disease
cholesterol
know
that
types
4
1.LDL
2.
multiple components
-
equal
total cholesterol
plaque
VLVL-low density
3. HDL
-
"good"cholesterol;remove LDL
4. TG- stored infat cells
lab values remember / the
type
LDL oxidation
-damage not due to trauma butchemical
or
long term hypertension
-
damage Inside triggers monocytes
in
They
come
help me, I
mediators
broken, fix me!)
↳results in inflammation
and stage
↳D
fatty streak formations creating
-
3rd stage
foamy cells
-
foanty cells present
collagen cap pulling
nee
*
This is
where
into
capsule
you start seeing
sx
4
complication lesion
fibrous plaque creates thrombus
-
coagulation plaque
of
-anything distal
aspirin prevent
ne
will have oclussion
plaque formation;is "slicks; lets it gothru
xipidemia
1.
(AD
-
-
occlusive
microvascular disease
(seen more in women)
SX Often don't match traditional
stable anging
ID
↳
MIdSX
managed
*
around
stage 3
x
patterns
Acute coronary sx-heart attack
2. DAD
·
distal areas
(legs)
leg pain my ambulation
Drisk for complete obstruction
·can be dangerous
perfusion
↳no
risk factors
footof
amputation
Dyscipidemia
-askabout
family history
-Inherited autosomal
-
age
-
exposure
spesity/dlet
~
*
xanthomal (location?)
sores not healing in
specific areas
(not enough blood flow
In that area)
SMOKING
-
-
presents
phacnes
-
-
-
review ix & cues &How It
AICONOl
GOAL:
-
nursing process
fat &active
by
S
lifestyle changes
↑
activity
adequate/approp. fiber
tackle way fats are metabolizes
have a good balance
PREVEN
AYidemic
of death,
ex: cherrios
heart
attack
complications of hyperlipidemid*
4 across all body systems
-What are the clinical cues!
Aretnopaiy
DVT
inven
-
-
-
formation of clot limits blood flow
sudden on set
-
have
a
pain occuring w/ rest
4)
swelling
better pain relief when
walking
Intermittent claudication
-cues: cool, pale no swelling
L
Nursing
·
process
Assess & Action steps
1.
lipid labs
changes (ways to fall
3. family history
4. activity levels
2. diet
↑
5. Smoking/alcohol
VISIONIOng
7. BM1 cheight &
6.
weight)
advise fiber
ATORVASTATIN
-
a
know process&
CoQ1o-muscles
AlE:Keastained
-
that need energy
myalgial?)
urine
focusons
how to monitor
lower back pain
& What needs to
management
-
monitor
educate
=
take WI
AO
do not stop abruptly
rebound
will not immed, feel better
report
->
certainsx
eye exams is a
big thing
CI20STAIOL
contra:e failure, bleeding disorders
admini:only W/ H20
given eightdue to vasodilation
SIE:a*, rhinitis, cough
often
teaching:
takes
Waproti, er?
up to 3
mo to take effect
don't memorize mechanism
Mr
happen next
BP:systolic
-
diastolic
-
pressure against arteries
when heart contracts
rests
what BP means (the reading)
Isolated systolic HTN
(SPP>130/DBP (80)
-
big gap
blw the #'s)
clinical cues
HTN- "silent Killer:
x:
-headaches afrequent
A
pounding of chest /ears
-vision problems AAFalled eye
↳falling/bruising
-
exams
as a
result
nosebleeds
2D blast of
pressure
-
blood in urine
HTN caused by:
1.
Arterioles
2.
Venmoles
Hypertension
↳
too much pressure, volume
or both
3. I
4. Kidney
5.
RAAD
lymphatics
maintain BP
fever
sepsis
IPPUR
CIVcUl. blood volume
diabetes
the conditions
I
THI diuretics
advenergic antagonist
2, blockers
calcium channel blockers
working
on
pressure &volume
ARB
HCTZ
mild-moderate HTN
-
where?Location?
electrolyte fX?
monitoring?
METOPROLOL
a
⑦
t contract
$ HR
relaxrate
(-) inotrophy
Ifchronotrophy
↓
I
B,
conduction rate
⑭) testing
longer fill time
BP drops naturally
MONITOR
HR!
-
blocks now
high
HR
is(?)
SIE:INS(dizzy, drowsy, blurred vision), G1(NIV), peripheral edema
caution for those my asthma
liver dysfunction
or COPD
leave this med pic of erectile dysfunction
-people
really talk about se
PRAIOSIN
-
my ptonce they take it
monitor fall risk
& blocker
by sis response in arterioles
selective for a smooth muscle
allates both avery &
veins
1huge drop in BD
work
-
can be given
&
-
watchfor:
-first
dose
-
safety
phenomenon
orthostatic HTN
ido not
give
sitting down then
few hours do PT
night
report ptsx, tachycardia, safety concerns
esp.change output **
-
VALSARTIN
ARB
-
vasodilator
anglotension & from anglo I receptors
caution: pregnancy, kidney disease
blocks
DO NOT
ASPIRIN
TAKE
SXSAMe aS W/HTN
when
I
AIP/NE
Options
2 deff.
x & & blood vessels
vacation!!
=
ANGIOEDEMA
swelling in deep layers
know order a priority
7
skin
Aging & impact
Absorption (PR) small bowel
feeding tubes:changing
the
of
surface
sA
are a
changed
over time
know
metrition inare as
book
situations
in
lower absorption rate
kidneys
know
is a
baseline for toxicity
GFR:filters fluid by
big picture
-
examples
drugs
of
when certain
glomerulus
age
bad
lower #=
calculation not necessary
butknow
components?
100-125
=
normal
GFR