Measles is stated to be a helical symmetry
virus it results into the negative polarity of the nonsegmented Ribonucleic Acid (RNA). The virus has
15,894 nucleotides which encode 8 viral proteins
six-genes and at the same time it has an RNAbound RNA polymerase. The six genes mentioned
are nucleoprotein (N), phosphoprotein (P), matrix
(M), polymerase (L), hemagglutinin (H) and fusion
protein (F). Viral penetration to the RNA is via the
fusion of HN Glycoprotein in the host cell which
stimulates the fusion of the host cell membrane with
that of the virus. The matrix (m) just creates the
heart for the lipid coat in the fusion (Leung et al.,
2018). Polymerase encodes with the mRNAs for C
and V proteins which control or regulate the host
cell immune response during fusion forming the
patient’s sickness (Guseva et al., 2019; Leung et al.,
2018). Once the virus hits the cells of any tissue it
produces a mononuclear reaction which then
precipitates as an inflammation on the body of the
patient just like in our case the 6 year old Lucy
expressing multiple inflammatory foci on the body.
According to Krawiek and Hinson (2022), infection
starts after the hemagglutinin has bound in the
receptor cell position of the patient. The immune
system gets worse when the viral protein outside the
host cells utilizes the proteins C and V (Khan 2018).
The risk factors of Measles is that its
prolonged triage n the body of the host it develops
other complications such as severe pneumonia or
Student I.D Number: 3