sample nursing notes
nursebossstore.com
nursebossstore
ww.pinterest.com/nursebossstore
nursebossessentials
nursebossstore.com
The Nursing Mega Bundle
Over 650 pages of nursing study guides
click here
nursebossstore.com
nursebossstore
ww.pinterest.com/nursebossstore
nursebossessentials
DISCLAIMER
Do not attempt to sell, copy or redistribute this material as Fiskvik
Boahemaa Antwi is the sole owner of this material. Kindly note that you
cannot share or reproduce this item. All study guides are protected by the
copyright law.
UPGRADE NOW AND PURCHASE
THE FULL VERSION. 600 PAGES
OF NURSING NOTES
CLICK HERE
PDF-DIGITAL ONLY
medical-surgical nursing
Treatment
Signs & Symptoms
Causes
Nursing Assessment
Pathophysiology
Expected
Findings
Nursing Interventions
Diagnostic Test
nursebossstore.com
nursebossstore.com
CARDIOVASCULAR DISORDERS
Coronary Artery Narrowing or occlusion of the coronary
arteries due to atherosclerosis.
Disease
Angina
Chest pain due to decreased myocardial
oxygenation.
Myocardial
Infarction
Myocardial tissue damage as a result of
oxygen deprivation
Heart failure
The inability of the heart muscle to pump
enough blood to meet the metabolic
demands of the body.
Cardiogenic
Shock
Decreased cardiac output and decreased
tissue perfusion.
Pericarditis
Pericarditis is an infection of the
pericardium.
Endocarditis
Inflammation and infection of the
endocardium
Myocarditis
Myocarditis is the inflammation of the heart
muscles (myocardium).
Cardiac
Tamponade
Cardiac tamponade decreases ventricular
filling and cardiac output. Due to pericardial
effusion.
Aortic
Aneurysm
Aortic aneurysm is an enlargement/dilation
of the aorta.
nursebossstore.com
RESPIRATORY DISORDERS
asthma
Smooth muscle constriction of the bronchi.
COPD- Chronic
Bronchitis
Overproduction of mucus due to
inflammatory response.
COPDEMPHYSEMA
Progressive respiratory disease
characterized by the enlargement of the
alveolar.
PLEURAL
EFFUSION
Accumulation of fluid in the pleural space.
HEMOTHORAX
Accumulation of blood in the pleural cavity.
PNEUMOTHORAX
Air leaks into pleural space. Causes impaired
lung expansion.
PNEUMONIA
Inflammation of the pulmonary tissue
caused by bacteria, fungi and viruses
pancreatic disorders
Pancreatitis
Inflammation of the pancreas. Digestive
enzymes starts digesting the pancreas.
Cholecystitis
Inflammation of the gallbladder.
Cholelithiasis
Most of cholelithiasis is caused by
cholesterol gallstones.
nursebossstore.com
GASTROINTESTINAL DISORDERS
Hiatal Hernia
Hiatal hernia occurs when a portion of the
stomach herniates through the diaphragm
and into the thorax.
GERD
A digestive disorder that occurs due to the
backflow of gastric content.
Gastritis
Gastritis is the inflammation of the gastric
mucosa.
Appendicitis
Inflammation of the vermiform appendix.
Peptic Ulcer
Disease
Ulceration that erodes the gastric or
duodenal mucosa.
Ulcerative
Colitis
Characterized by the ulceration and
inflammation of the colon and rectum.
Causes poor nutrient absorption.
Crohns Disease
Inflammation in the gastrointestinal tract
hepatic disorders
Cirrhosis
Cirrhosis is a chronic progressive disease of
the liver characterized by fibrosis
Portal
Hypertension
Increased pressure in the portal veins due to
obstruction of the portal blood flow.
Esophageal
Varices
Increased pressure from the portal vein
causes blood to flow into smaller veins in the
esophagus. Smaller veins may rupture.
nursebossstore.com
Genitourinary DISORDERS
Acute Kidney
Injury
Renal cell damage characterized by a
sudden deterioration in kidney function.
chronic kidney
disease
Slow, progressive and irreversible loss of
kidney function.(GFR <60mL/min).
Glomeruloneph
ritis
A group of renal diseases caused by
immunologic response that triggers the
inflammation of the glomerular tissue.
Nephrotic
Syndrome
Nephrotic syndrome is characterized by
proteinuria, leading to low protein levels in
the blood (hypoproteinemia).
Renal Calculi
Renal calculi is also known as kidney stones.
Urinary Tract
Infection
UTI is the infection/inflammation of any part
of the urinary system.
Pyelonephritis
Inflammation of the renal pelvis caused by
bacterial infection.
neurological DISORDERS
Head Injury
Trauma to the skull that causes brain
damage.
Stroke
Stroke is the loss of neurological functions
due to the lack of blood flow to the brain.
Seizures
Seizures is characterized by a sudden,
uncontrolled electrical disturbance in the
brain.
fractures
fractures
fractures
A NORMAL BONE
Impacted: a part
of the bone that
impact another
bone
Transverse:
straight across
the bone.
Greenstick: one
side of the bone
is broken, the
other side is
bent
Oblique: fracture
that run at an
angle across
Complete fracture:
complete break
through the bones
that separates
into two.
Spiral: fracture
that circles or
spirals around
the shaft.
Open fracture
(compound):
fracture with an
open wound.
Comminuted
break into more
than two
fragments.
Fissure fracture
are cracks in the
bone.
Closed fracture:
bone break
without open
wound in skin.
Compression:
one bone
compresses
another bone
coronary artery disease
nursebossstore.com
pathophysiology
CAD is caused by atherosclerosis (plaque formation) that
results in the narrowing or occlusion of one or more
coronary arteries.
Risk factor
1. Age, Gender
2. Family hx, HTN
3. High blood cholesterol level,
4. Diabetes,
5. Smoking,
6. Obesity
nursing interventions
1. Pain assessment, vital signs/ECG
2. Administer oxygen
3. Administer medications
4. Promote bed rest
5. Place client in a Semi-Fowler's
position.
Patient Education
1. Lifestyle modifications
2. Low-sodium and low-cholesterol
diet.
3. Stress management
signs & symptoms
1. Chest pain
2. Dyspnea/SOB
3. Fatigue
4. Dizziness
5. Syncope
6. Cough
7. Normal findings
during asymptomatic
period
diagnostic tests
Electrocardiography
-ST segment elevation, t-wave
inversion and abnormal Q
wave(MI)
2. Cardiac catheterization
-may show atherosclerotic lesions.
3. Blood lipids level would be
elevated.
Anatomy
Physiology
PATHOPHYSIOLOGY
1.Cardiac contractility: force and
velocity of contraction
2.Cardiac Output: the amount of
blood pumped by the ventricles
per minute.
3.Stroke volume: the volume of
blood discharged from the
ventricle with every contraction
4.Preload: ventricular stretch at
the end of diastole.
5.Afterload: the 'load' to which
the heart must pump against.
1.Heart failure (HF) occurs when the heart is unable to pump enough
blood to meet the body's demands.
2.Heart failure can affect the left or right side of the heart (left and
right HF)
3.Left sided heart failure is evident in the pulmonary system. Remember:
Left = Lungs
4.Right sided heart failure is evident in the systemic system
Note: in order to understand the signs and symptoms based on left sided and right sided heart
failure, you need to understand the flow of blood through the heart and body.
RISK FACTORS
1.CAD
2.MI
3.Myocarditis/Endocarditis
4.Diabetes
5.Hypertension
6.Abnormal heart valves
7.Cardiomyopathy
8.Congenital heart disease
SIGNS/SYMPTOMS
Right-sided HF
Edema of the extremities,
abdominal distention, JVD,
splenomegaly, hepatomegaly,
weight gain
Left-sided HF
Dyspnea, crackles, tachypnea,
pulmonary congestion, dry cough
nursebossstore.com
nursebossstore.com
pericarditis
Anatomy
Physiology
The pericardium can be
divided into:
The parietal pericardium is
the outer membrane.
The visceral pericardium is
the inner membrane.
Major functions of the
pericardium:
1. Protects the heart
2. Lubricates to reduce
friction (The pericardial
sac contains 5-20ml of
pericardial fluid)
PATHOPHYSIOLOGY
1. Pericarditis is the inflammation of the pericardium (mostly
with fluid accumulation)
2. Acute pericarditis: inflammation of the pericardium+ a
pericardial effusion. Symptoms develops quickly.
3. Subacute pericarditis: within weeks to months
4. Chronic pericarditis: pericarditis >6 months
5. Pericardial effusion z: fluid accumulation in the pericardium.
6. Complication: cardiac tamponade
RISK FACTORS
1. MI
2. Autoimmune diseases
3. Injury
4. Heart surgery
5. Bacterial, viral and
fungal infections
SIGNS/SYMPTOMS
Pain
Pain that radiates to the
left side of neck, shoulders
and back
Pain experienced during
inspiration
Pain experienced when in a
supine position
Fever
Fatigue
Pericardial friction rub (during
auscultation)
nursebossstore.com
angina
Anatomy
Physiology
LAYERS OF THE HEART
Epicardium: outermost
layer of the heart
Myocardium: middle
layer of the heart
Endocardium: innermost
layer of the heart
The myocardium is also
known as the heart
muscle.
It is responsible for the
involuntary contractions
and relaxation of the
heart.
PATHOPHYSIOLOGY
1. Angina is characterized by chest pain due to the imbalance of
myocardial oxygen demand and oxygen supply by the
coronary arteries.
2. Types of Angina:
a. Stable angina-occurs due to physical exertion. It has a
regular pattern. Pain relieved by rest.
b. Unstable angina- unexpected chest pain that increases in
severity, duration and occurrence (may occur at rest).
c. Variant angina- occurs due to coronary artery spasm.
Occurs at rest.
d. Intractable angina- chronic (recurrent angina)
e. Preinfarction angina- occurs before an MI
RISK FACTORS
1. Family history of
heart disease
2. Hypertension
3. High blood
cholesterol
4. Diabetes
5. Smoking
6. Obesity
SIGNS/SYMPTOMS
1. Pain
2. Dyspnea/SOB
3. Tachycardia
4. Palpitations
5. Dizziness
6. Syncope
7. Diaphoresis (Sweating)
8. Pallor
9. Elevated BP
nursebossstore.com
Myocardial Infarction
Anatomy
Physiology
LAYERS OF THE HEART
Epicardium: outermost
layer of the heart
Myocardium: middle
layer of the heart
Endocardium: innermost
layer of the heart
The myocardium is also
known as the heart
muscle.
It is responsible for the
involuntary contractions
and relaxation of the
heart.
PATHOPHYSIOLOGY
1. A prolonged and severe imbalance between myocardial
oxygen supply and demand causes myocardial infarction.
2. Myocardial infarction is mostly caused by coronary
atherosclerosis.
3. Acute MI + unstable angina = acute coronary syndrome.
4. Acute MI can be non ST segment elevation myocardial infarction
(NSTEMI) or ST segment elevation myocardial infarction
(STEMI).
RISK FACTORS
1. CAD
2. Atherosclerosis
3. High cholesterol level
4. Diabetes
5. Hypertension
6. Smoking
7. Stress
SIGNS/SYMPTOMS
1. Pain- crushing substernal
pain that radiates to the
left arm, jaw or back.
2. Dyspnea
3. Dysrhythmias
4. Pallor
5. Cyanosis
6. Diaphoresis
7. Anxiety
asthma
nursebossstore.com
pathophysiology
Chronic inflammatory disease of the airway.
Inflammation and hypersensitivity to a trigger
(stimuli). Smooth muscle constriction of the bronchi.
Intermittent airflow obstruction.
Risk factor
Allergies
Stress
Hormonal changes
signs & symptoms
Chest tightness
Wheezing
Shortness of breath
Cough
Restlessness
nursing interventions
Assess patient's respiratory rate,
depth and pattern
Monitor pulse ox
Monitor vital signs
Maintain patent airway
Administer O2 therapy as
prescribed
Administer medications as
ordered.
Patient Education
Medication regimen.
Identify and avoid triggers.
Long term management.
diagnostic tests
ABGs
Pulmonary function tests
Peak expiratory flow
Spirometry
Allergy test
Pulse oximetry
CBC
gerd
nursebossstore.com
pathophysiology
A digestive disorder that occurs due to the backflow of gastric
content.
Impaired or dysfunctional lower esophageal sphincter (LES)
causes regurgitation of stomach content into the esophagus.
Risk factor
Hiatal Hernia
Pregnancy
Pyloric surgery
Smoking
Obesity
Alcohol
Fatty foods
nursing interventions
Assess pain
Elevate head of bed (HOB)
Avoid eating 2 to 3 hours before bedtime
Avoid lying down after eating
Administer medications as ordered
Patient Education
Avoid alcohol, fatty foods, caffeine,
tobacco, and other irritants
Avoid eating 2 to 3 hours before bedtime
Avoid lying down after eating
Avoid NSAIDS and anticholinergics
Maintain healthy body weight (exercise)
signs & symptoms
Heart burn
Dysphagia
Regurgitation
Epigastric pain
Dyspepsia
(indigestion)
diagnostic tests
Upper endoscopy
Esophageal pH studies
Barium swallow
(esophagram)
STROKE
nursebossstore.com
pathophysiology
Stroke is the loss of neurological functions due to the lack of blood
flow to the brain.
Types
Ischemic Stroke, Hemorrhagic Stroke, Transient Ischemic Attack
Risk factor
TIA
Hypertension
Smoking
Atherosclerosis
Diabetes
High cholesterol
nursing interventions
Maintain patent airway
Administer 02
Administer tPA
Monitor VS-maintain BP @
150/100
Monitor LOC
Monitor for signs of increase ICP
Elevate HOB
Administer IV fluids
Insert Foley's catheter
Prevention of DVT
Assist with self care and ADLs
signs & symptoms
Drooping of face
One sided weakness
Slurred speech
Blurred vision
Agnosia
High BP
Unilateral neglect
Apraxia
diagnostic tests
CT scan
MRI
Electroencephalography
Carotid ultrasound
Cerebral arteriography
nursebossstore.com
ELECTROLYTE
pharmacology
MOA
Nursing Considerations
Indications
Contraindications
Patient Education
SIde/Adverse Effects
nursebossstore.com
nursebossstore.com
CARDIOVASCULAR DRUGS
DRUG CLASS
Thrombolytics
Antiplatelets
Anticoagulants
Cardiac
Glycosides
Thiazide
Diuretics
Loop Diuretics
MEDICATIONS
Tenecteplase
MECHANISM OF ACTION
Thrombolytic drugs dissolve clots by
activating plasminogen that forms plasmin.
ASPIRIN
Antiplatelet drugs prevent the aggregation or
adhesion of platelets.
HEPARIN
Anticoagulants interfere and prevent the
formation of clots by inhibiting factors in the
clotting cascade.
digoxin
Cardiac glycosides- increase cardiac
contractility/positive inotropic effect,
negative chronotropic effect
Chlorothiazide
Thiazide diuretics increase the excretion of Na
and water in the distal convoluted tubule.
Furosemide
Loop diuretics decrease the reabsorption of
sodium and chloride in the ascending Loop of
Henle.
PotassiumSparing Diuretics
Spironolactone
ACE Inhibitors
Enalapril
Potassium-sparing diuretics cause sodium
and water excretion in the distal tubule,
whilst promoting potassium retention (blocks
aldosterone receptors)
Angiotensin-Converting Enzyme Inhibitor
(ACE Inhibitors) prevents the conversion of
angiotensin I to angiotensin II which
prevents vasoconstriction.
nursebossstore.com
DRUG CLASS
ARBS
Calcium
Channel Blockers
Beta
Adrenergic Blockers
MEDICATIONS
Losartan
Nifedipine
MECHANISM OF ACTION
ARBs prevent aldosterone release and
peripheral vasoconstriction by selectively
blocking angiotensin II receptors.
Calcium channel blockers prevent calcium
ions movement across myocardial cell
membrane. This causes relaxation of
smooth muscle.
Atenolol,
Beta adrenergic blockers block the effect of
epinephrine at the receptor sites.
Adrenergic
Agonist
Epinephrine
Adrenergic agonist stimulates the adrenergic
receptors (both alpha or beta receptors) of
target organs.
Antianginal
Agents
Nitroglycerin
Nitrates are antianginal agents that relax
smooth muscles, resulting in vasodilation,
reduced preload (dilating veins) and afterload
(dilating arteries) and decreased myocardial
oxygen demand.
respiratory DRUGS
DRUG CLASS
MEDICATIONS
Inhaled
Anticholinergics
Atrovent
Antihistamines
Expectorants
MECHANISM OF ACTION
Inhaled anticholinergics prevent the binding of
acetylcholine (neurotransmitter) by blocking
muscarinic receptors.
This results in bronchodilation (relaxation of
smooth muscle in the bronchi).
Brompheniriamine Decreases bronchial secretions
Guaifenesin
Expectorants reduce the surface tension of
bronchial secretion and induce productive
cough to promote patent airway.
nursebossstore.com
DRUG CLASS
MEDICATIONS
Mucolytics
Acetylcysteine
MECHANISM OF ACTION
Mucolytics liquefy or thins respiratory
secretions (mucus) for airway clearance
(productive cough).
Decongestants cause vasoconstriction in the
Oxymetazoline upper respiratory system. This leads to
shrinking swollen mucous membrane and
reduced fluid secretion.
Decongestant
Benzonatate
Antitussives
Antitussives suppress the cough reflex by
directly acting on the cough control center in
the medulla.
Glucocorticoids are anti-inflammatory agents
Beclomethasone that decrease inflammatory response in the
airway.
Glucocorticoids
(Corticosteroids)
Sympathomimetic
Bronchodilators
Salmeterol
Sympathomimetic affects the beta-receptors
found in the bronchi which leads to the
relaxation of smooth muscle in the bronchi.
gastrointestinal DRUGS
DRUG CLASS
MEDICATIONS
Proton Pump
Inhibitor
Histamine (H2)
Receptor Antagonist
Antacid
MECHANISM OF ACTION
Omeprazole
Proton pump inhibitors suppress the
secretion of HCL in the stomach
Ranitidine
Histamine (H2) receptor antagonist blocks the
action of histamine, which produces HCL
secretion.
Calcium
carbonate
Antacid are alkaline compounds that
neutralizes acids and prevents the conversion
of pepsinogen to pepsin in the stomach.
nursebossstore.com
DRUG CLASS
Antiemetics
Laxatives
MEDICATIONS
Ondansetron
Psyllium
MECHANISM OF ACTION
Antiemetics suppress nausea and vomiting
by acting on the brain's control center to
stop the nerve impulse.
Laxatives promote bowel elimination.
ANTIBIOTICS
DRUG CLASS
MEDICATIONS
Penicillin
Penicillins
Cephalosporin
Cefazolin
Aminoglycosides
Tetracycline
MECHANISM OF ACTION
Penicillins inhibit bacterial cell wall synthesis.
Therefore, the bacteria's cell wall swells,
ruptures and dies.
Cephalosporins inhibit bacterial cell wall
synthesis.
gentamicin
Aminoglycosides inhibit bacteria protein
synthesis. They inhibit the translation of mRNA
to protein by irreversibly binding to bacteria
ribosome.
doxycycline
Tetracycline are broad-spectrum and inhibits
protein synthesis which causes the inability for
bacterial growth
Sulfonamides
sulfadiazine
Fluoroquinolones
levofloxacin
Sulfonamides inhibit the metabolic process
essential for the function and growth of the
bacterial cell.
Flouroquinolones interfere with DNA gryase
(an enzyme) needed by the bacteria for the
synthesis of DNA
nursebossstore.com
NEUROLOGICAL DRUGS
DRUG CLASS
NSAIDs
Salicylates
Acetaminophen
Opioid Analgesics
Anticholinesterases
MEDICATIONS
diclofenac
ASPIRIN
Acetaminophen
Morphine
Edrophonium
Dopaminergics
Apomorphine
Benzodiazepines
DiazepaM
Hydantoins
Phenytoin
MECHANISM OF ACTION
NSAIDs have anti-inflammatory, analgesic and
antipyretic properties. NSAIDs inhibits
prostaglandin synthesis
Salicylates inhibit synthesis of prostaglandin.
Salicylates have anti-inflammatory,
antipyretic and analgesic properties.
Acetaminophen inhibits prostaglandin
synthesis (limited to CNS and not periphery)
Suppresses pain impulses.
Used to treat muscle weakness in myasthenia
gravis. Anticholinesterases blocks
acetylcholine breakdown.
Dopaminergic drugs stimulate dopamine
receptors and increase dopamine
concentration.
Benzodiazepines are used to treat absence
seizures.
They enhance the effect of GABA
Blocks sodium channels and inhibits neurons
from firing to stabilize central nervous
system membrane
nursebossstore.com
-PINE
nursebossessentials
nursebossstore
Cephalosporin
CEF-, CEPH-
-OPRAZOLE
PPIs:
-CILLIN
Penicillin
-FLOXACIN
Quinolones:
Macrolides
-MYCIN
-TIDINE
H2 Receptor
Antagonists
GI
-PROFEN
NSAIDS
SULFONAMIDES
SULF-
-VIR
Antiviral
ANTIVIRAL
OPIOIDS
-DONE
PAIN
-THIAZIDE
Thiazide Diuretics
@nursebossessentials
ACE INHIBITORS LOOP DIURETICS
-SEMIDE
-PRIL
ANTIBIOTICS
Xantine
-PHYLLINE
@nursebossessentials
statinS
-STATIN
ARBS
-SARTAN
RESP
CC BLOCKERS
CARDIO
beta blockers
-LOL
CARDIOVASCULAR
suffixes +
prefixes
nursebossstore.com
nursebossstore.com
nursebossessentials
antidote
name:
FLUMAZENIL
name:
BENZODIAZEPINES
name:
PROTAMINE
antidote
antidote
name:
ATROPINE
antidote
nursebossstore
name:
DIGOXIN
medication
name:
INSULIN
medication
name:
DIGIBIND
antidote
name:
GLUCAGON
antidote
name:
name:
ACETAMINOPHEN ACETYLCYSTEINE
medication
antidote
name:
VIT K
name:
CHOLINERGICS
medication
name:
NALAXONE
antidote
medication
name:
HEPARIN
medication
name:
WAFARIN
medication
name:
OPIOIDS
medication
drug and
antidote
nursebossstore.com
nursing health assessment
NURSING ASSESSMENTnursebossstore.com
REVIEW
GENERAL SURVEY
1. Assess physical appearance,
mood, affect and grooming.
2. Assess orientation: Oriented to
Person, Place, Time and Situation.
3. Assess level of consciousness.
4. Assess speech.
Vital Signs
Pulse: 60-100 bpm
Blood Pressure Systolic: 120
Diastolic: 80
Respiratory Rate: 12-18 bpm
O2 Saturation: 95-100%
Temperature: 36.5-37.5 degrees C
HEAD AND FACE
1. Assess head size, shape,
symmetry.
2. Inspect and palpate head,
scalp
3. Palpate sinuses and TMJ
Face
1. Assess facial symmetry
2. Assess cranial nerve 7
Eyes/ Ears/ Nose
Inspect external eye structures,
conjunctiva and sclera.
Test cranial nerve III, IV, VI
PERRLA- Pupils are Equal,
Round, Reactive to Light and
Accommodation.
Pupil size: 3-5mm
Ears: Assess for redness, drainage.
Test cranial nerveVestibulocochlear
Nose: Assess shape, symmetry, size,
patency. Test cranial nerve I
Mouth
1. Inspect lip color, sores, gums,
tongue, teeth, soft and hard
palate, uvula
2. Test cranial nerve 9, 12 and 10
Neck
1. Palpate lymph node, carotid
artery, presence of goiter.
2. Auscultate for bruits.
3. Test cranial nerve 11
Lungs
1. Inspect symmetrical chest
movement
2. Palpate for pain and lumps
3. Percuss using the Z-block
method
4. Auscultate lung sounds
HEART
1. Auscultate heart sounds (Aortic,
Pumonic, Erb's Point, Tricuspid
and Mitral)
ABDOMEN
1. Inspect, Auscultate, Percuss,
Palpate
2. Inspect skin color, contour and
aortic pulsations.
3. Auscultate bowel sounds from
RLQ clockwise.
SKIN + EXTREMITIES
1. Assess and inspect skin, nails,
muscle strength, ROM,
curvature of spine.
2. Palpate pulses
nursebossstore.com
AUSCULTATION
nursebossstore.com
AUSCULTATION
nursebossstore.com
AUSCULTATION
nursebossstore.com
TRIADS
maternal and child
nursebossstore.com
gtpal
GTPAL: DESCRIBES PREGNANCY OUTCOMES
GRAVIDITY
TERM BIRTHS
PRETERM BIRTHS
ABORTIONS
1. Number of pregnancies
(twins and triplets are
counted as one)
2. Present pregnancy included.
1. The number born at term
(longer than 37 weeks of
gestation)
2. Twins and triplets are
counted as one.
20-37 weeks of gestation.
(Count twins and triplets as
one)
Includes alive and still birth
Less that 20 weeks of
gestation.
Count twins and triplets as one
also includes miscarriages
LIVING CHILDREN
current living children.
Count children individually
lochia
Lochia
nursebossessentials
description
Lochia rubra
Bright red.
Last for 1-3 days
Heavy flow
lochia SEROSA
Pink/Brown
Lasts: Day 4 to Day 10
Moderate-small
lochia alba
Whitish/yellow
Lasts: may last up to 6
weeks
Gradually
reduces/disappears
INFECTION/ WARNING SIGNS
Foul smelling or purulent
lochia
Fever
Abdominal
pain/tenderness
lab values
nursebossstore.com
nursebossessentials
nursebossstore
0U
/L
AL
P
20
-4
7-5
6U
/L
23s
ec
on
ds
IN
R
AL
T
TT
30
-4
0s
ec
HE
s
PA
RIN
aP
T
25
-3
5s
ec
s
PT
PT
1013
sec
on
ds
coagulation
1040
U/
L
AS
T
To
0.1
-1.2
ta
mg
l
/dL
Bil
ir
ub
in
g/d
L
Al
bu
3.4
mi
-5
n
.4
Liver
-10
0%
Sa
O2
95
%
mm
Hg
HC
O3
22
-2
6
00
mm
Hg
Pa
O2
80
-1
45
mm
Hg
Pa
CO
35
2
-
7.4
5
pH
7.3
5-
abgs
ta
s
gn
e
l
95 lor
-10
5 m ide
Eq
/L
ch
ci
911 m um
g/d
L
ca
si
1.5
um
-2
.5 m
Eq
/L
ma
si
3.5
um
-5
.0 m
Eq
/L
po
s
135 odi
u
-14
5m m
Eq
/L
electrolytes
t
gr
av
1.0
10it
y
1.0
30
Sp
ec
ifi
c
R
90
-12
0
GF
in
O.6
in
-1.2
e
mg
/dL
ea
Cr
BU
7-2
N
0m
g/d
L
renal
C
a
t
150
,00 el
ce 0-4 et
s
lls/ 00
mc ,00
L 0
Pl
B(
14M)
18
g/d
L
Hg
B(
12F
16
g/d )
L
Hg
4.5
-5
.5
RB
W
4,5
B
00
-11 C
,00
0
cbc
lab values
nursebossstore.com
nursebossstore.com
To
ta
l
nursebossessentials
AL
P
AL
T
AS
T
Bil
ir
ub
in
Al
bu
mi
n
Liver
IN
R
Sa
O2
HC
O3
TT
T
Pa
CO
2
pH
abgs
Pa
O2
aP
PT
PT
coagulation
ch
de
iu
m
ri
lc
iu
m
iu
m
es
lo
ca
gn
ss
di
um
ta
ma
po
so
electrolytes
R
gr
av
it
y
GF
ti
ni
ne
Sp
ec
ifi
c
ea
Cr
N
BU
renal
Pl
at
el
et
s
M)
F)
B(
B(
Hg
Hg
C
BC
RB
W
cbc
lab values TEMPLATE
nursebossstore.com
nursebossstore
meds
meds
pharmacy
Carbamazepine:
4-12 mg/L
nursebossstore.com
ab
ov
e
di
ab
et
6.5
ic
%&
:
pr
edi
ab
et
5.7
ic
-6
.4%
di non
ab
4- etic
5.6
%
hba1c
m
pharmacy
pharmacy
Digoxin:
0.5-2 ng/mL
Theophylline
10-20 mcg/mL
pharmacy
PhenobarbitaL
15-40 mcg/mL
pharmacy
pharmacy
Phenytoin
10-20mg/L
Lidocaine
1.5-5mg/L
nursebossessentials
Di
me
ma
70
-10
p
0m
mH
g
<0.
50
D-
i
18.
524
.9
bm
515
mm
Hg
ic
p
r
on
1545
ia
U/
dL
Am
other
Tr
ig
ly
ce
ri
<15
de
0m
s
g/d
L
>60
L
mg
/dL
HD
L
<10
0m
g/d
L
LD
Ch
ol
toest
<20
ta er
0m
l ol
g/d
L
Lipid
l
KMB
3%
-5
%
CP
-M
03n
B
g/m
L
CK
ob
085
in
ng
/m
L
og
My
on
0in
0.4
ng
I
/m
L
Tr
op
Cardiac
nursebossstore.com
lab values
Lithium
0.8-1.5 mmol/L
pharmacy
nursebossstore
KCP
Tr
ig
ly
ce
ri
de
s
L
HD
L
pharmacy
Digoxin:
pharmacy
Carbamazepine:
nursebossstore.com
D-
ma
p
Di
me
i
di
ab
et
ic
:
di non
ab
et
ic
pr
edi
ab
et
ic
bm
ic
p
mo
ni
a
r
LD
Ch
ol
toest
ta er
l ol
Am
My
og
CK
lo
-M
B
bin
I
MB
lab values
in
on
Tr
op
Lipid
meds
meds
hba1c
other
Cardiac
nursebossstore.com
pharmacy
Theophylline
pharmacy
PhenobarbitaL
pharmacy
pharmacy
Phenytoin
Lidocaine
nursebossessentials
pharmacy
Lithium
nursebossstore
nursebossstore.com
Isotonic
IV
SOLUTION
0.9% Normal
Saline
TYPE
0.9%
NaCl
Monitor closely for
hypervolemia, especially
with heart failure or renal
failure.
LR
Burns
GI tract fluid
losses
Traumas
Should not be used if serum
pH is greater than 7.5
because it will worsen
alkalosis.
D5W
Patients with
hypernatremia
Used to treat
hypoglycemia
Starts as isotonic and then
changes to hypotonic when
dextrose is metabolized
Fluid and
electrolyte
replenishment
and caloric
supply
5% Dextrose and
Lactated Ringer’s
5% Dextrose
in Water
Lactated
Ringer’s
Hypotonic
10% Dextrose
in Water
Hypertonic
(D10W)
5% Dextrose in
0.45% Saline
Hypertonic
D5LR
D50.45%
NaCl
0.45% Saline
Hypertonic
CONSIDERATIONS
Severe vomiting +
Diarrhea
Blood transfusion
To replace Na + Cl
Isotonic
Isotonic
USES
0.45%
NaCl
Caloric supply
Monitor signs of
hypervolemia
Monitor signs of
hypervolemia
Maintenance
fluid
Monitor signs of
hypervolemia
Used to treat
intracellular
dehydration
Fluid replacement
among patients
with hypovolemia
Avoid in patients with
trauma, risk for increased
ICP or burns.
Monitor for hypotension
reviews!
nursebossstore.com