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Glomerulonephritis v Pyelonephritis

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Pyelonephritis
Glomerulonephritis
Follows
streptococcal
a
mrodt
Descending
Inflammationsdestruction
->
Ad
body
backing
destruction
to
due
permeability
up in the
->
blood?protein
Manifestations
now enter urine
·
manifestations
(little urinel
hypervolemia/fluid
-
overload
chills
symptoms
back
pain
·
nIV
·
(blood in urine
burning
urgency
frequency
tachyprea
Hin
·
LADS
Creat/Bun
↑WBC
·
↑ ESR, CRP
(inflammation
I creat (Bun
·ESRICRP
U/A:
pos. Aso titer
·
fever,
·
WBC
·
or
Hallmark
·tachycardia
Labs
·
tenderness
CVA
·Flank
·
Hih, dyspnea, crackles, edema
cola.colored urine
·↑
organism:E.coli
↓ UOP
is a
·oliguria
kidney
Ascending
NGFR
->
infection
most
common
to
Fluid is
->
glomeruliactive
to
kidneys
the ureters;the
for the
glomeruli filter
ability
blood
There
to the
bladder, that spreads up
uil in the
infection strep-
leukocyte
UIA: proteinuria, hematuria, ↑ urine spec.
estuaries, nitrates,
WBC, bacteria
gravity
throatculture:group Astrep /pos.)
Diagnosis
Treatment
definitie:renal
biopsy
·
·
Treatment
Abx
to
treat
Antipyretics
Analgesics:opioids
infection
or
USAIDS
dialysis
·diuretics
man need
antihypertensives
for chronic
or
ADX
kidney transplant
corticosteroids
chronic:
underlying
correct
cause
such as
urinary obstruction, refluxor
nursing
care
monitor 1? Os
·
·
daily weights
↓
bladder
Abx
therapy
supportrenal function
protein, sodium,fluid intake
supportive
always
I
neurogenic
prolonged
care
ESRD
leads to
labs:*P creat
(4), 44
BUn
(100-200), 4K, dca,
Chronic: SIs:systemic edema, slurred speech, tremors, asterixis (flap wrists
RX:slow progression of d2: A protein dietsfluid restrictions, drug therapy manage symptoms
P
involuntarily),
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