Nursing
Cheat Sheets
76 Cheat Sheets for
Nursing Students
NRSNG
Jon Haws RN
Sandra Haws RD
Copyright © 2018 by NRSNG, LLC
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Table of contents
Introduction
Cardiac
Blood Pressure Values
R v L Heart Failure
Types of Cardiomyopathy
12 Lead MI Locations
Angina
Heart Blocks
Cardiac Auscultation
Chest Pain Identification
H’s and T’s of ACLS
Coronary Circulation
Types of Aneurysms
Fundamentals
Patient Mobility
Chest Tube Care
Pathophysiology Concept Map
Head to Toe Assessment
Pressure Ulcers
Types of Lines
Medical Spanish
Therapeutic Diets
IV Sites and Considerations
Patient Safety
Colostomy Care
Abdominal Pain
Ulcerative Colitis Vs. Crohn’s Disease
Chronic Kidney Disease Symptoms
Types of Viral Hepatitis
Hematologic/Oncology/Immunology
Types of Anemia
Integumentary
Burn Staging
Skin Cancer
Skin Lesions
Labs
Lab Value Skeletons
Lab Value for Clinical
Blood Compatibility Chart
ABG Rome Flowchart
Cardiac Biomarkers
IV Colors and Gauges
ABG Analysis
Fluids and Electrolytes
Endocrine/Metabolic
Endocrine Study Guide Chart
Addison’s Vs. Cushing’s
Hyper Vs. Hypothyroidism
Musculoskeletal
Fracture Management
Mental Health
Stroke Symptoms by Location
Neuro Dysfunction by Pupil Assessment
Routine Neuro Assessments
Ob
Newborn Assessment
Labor
Clinical Assistant – Brain Sheet
Pediatric Burn Chart
Erikson’s Stages of Psychosocial Development
Congenital Heart Defects
Pharmacology
Crystalloid IV Solutions
Drug Card
Nervous System Pharmacology
Dopamine Vs Dobutamine
Beta Blockers
Common ICU Drips
Medication Antidotes
Insulin Cheat Sheet
Common Antihypertensive Drugs
Antidepressant Cheat Sheet
Immunization Schedule
Antibiotic Cheat Sheet
Answering Pharmacology Questions
Therapeutic Drug Levels
Antidysrrhythmic Meds and Action Potential Chart
Respiratory
Hierarchy of O2 Delivery Systems
Lung Sounds
Gas Exchange
Asthma Medications
Artificial Airways Decision Tree
Ventilator Alarms
Chest Tube Management
Introduction
My journey into nursing was a long one, but I have found it to be a truly rewarding career
that allows me to make a difference and have ample family time. I am confident that you
will achieve your goals. The fact that you are seeing additional resources to improve your
understanding speaks volumes to your dedication.
This book is intended to provide you with a quick reference to some of the most needed
and most used information for nursing students.
This is not a complete guide to nursing but a simple, compact, and quick reference to
some of the most important information.
Happy Nursing!
Jon Haws RN
For colored images of Cheat Sheets for Nurses download
the Kindle version of the book, which is available at no
additional cost with each physical purchase of the book.
10 Common Ekg Heart Rhythms
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Fibrillation
Atrial Flutter
Supraventricular
Tachycardia
Premature Atrial
Contraction
Premature Ventricular
Contraction
Ventricular Tachycardia
Ventricular Fibrillation
[NRSNG Academy Lesson: EKG Waveforms]
Hemodynamic Values
Methods To Elevate Parameter
Methods To Decrease Parameter
Cardiac Output
Blood Pressure
CO=HR*SV (4-8L/min)
CO*SVR
Heart Rate
X
60-100 bpm
Cardiac Output
CO=HR*SV (4-8L/min)
Treat cause, parasympatholytic
(Atropine), sympathomimetic
(Epinephrine), pacemaker
Treat cause, antidysrhythmics,
Vagal electrical therapy
Preload
Contractility
PAOP, CVP
Cardiac glycosides,
Sympathomimetics
Fluids, blood
Venous vasodilators,
diuretics ace inhibitors,
ARBs
Beta blockers, Ca
channel blockers
Afterload
SVR
Vasopressors
Arterial vasodilators,
ACE inhibitors ARBs,
IABP
Key Hemodynamic Values (With Equations)
Cardiac Output (CO)
HR x SV
4-8 L/min
Cardiac Index (CI)
CO/BSA
2.5-4 L/min/m2
Central Venous Pressure (CVP)
2-6 mmHg
Mean Arterial Pressure (MAP)
SBP+(2xDBP)/3
70-100 mmHg
Stroke Volume (SV)
EDV - ESV
60-120 ml/beat
Stroke Volume Index (SVI)
SV/BSA
30-65 ml/m2/beat
Pulmonary Artery Occlusion Pressure (PAOP)
Systemic Vascular Resistance (SVR)
8-12 mmHg
[MAP-RAP) x 80]/CI
Central Venous Oxygen Saturation (ScvO2)
Oxygen Delivery (DO2)
800-1400 dynes/sec/cm-5
65-85%
CO x CaO2 x 10
[NRSNG Academy Lesson: Preload and Afterload]
900-1100 ml/min
Blood Pressure Values
BLOOD PRESSURE VALUES
New 2017 AHA guidelines have eliminated pre-hypertensionand lowered the
threshold for the diagnosis of hypertension to allow for earlier intervention.
Blood pressure categories in the new guideline are:
Normal:
Less than 120/80 mm Hg;
Elevated:
Systolic between 120-12 and diastolic less than 80;
Stage 1:
Systolic between 130-13 or diastolic between 80-8 ;
Stage 2:
Systolic at least 140 or diastolic at least 0 mm Hg;
Hypertensive crisis Systolicover 180 and/or diastolic over 120,with patients
needing prompt changes in medication if there are no
other indications of problems, or immediate
hospitalization if there are signs of organ damage.
CATEGORY
Systolic mm Hg
Normal
Diastolic mm Hg
<120
<80
Elevated
120-129
<80
Stage 1
130-139
80-89
Stage 2
>140
>90
Hypertensive
Crisis
>180
>120
SOURCE:
http //www.acc.org/latest-in-cardiology/articles/201 /11/08/11/4 /mon-5pm-bp-guideline-aha-201
[NRSNG Academy Lesson: Hypertension]
[NRSNG Academy Lesson: Hypertension]
R v L Heart Failure
[NRSNG Academy Lesson: Heart Failure]
Types of Cardiomyopathy
[NRSNG Academy Lesson: Cardiomyopathy]
12 Lead MI Locations
[NRSNG Academy Lesson: Myocardial Infarction]
Angina
ANGINA
Chest pain resulting from inadequate blood flow to heart muscle. Most common cause is
coronary artery disease (CAD). Other causes include anemia, heart failure, abnormal rhythms.
STABLE
UNSTABLE
VARIANT
ONSET
Exertion/Stress
Rest/Exertion/Stress
Typically at Rest
PREDICTABLE
Predictable
Unpredictable
Unpredictable
Up to 30 min
Varies
May Resolve with Nitro
May Resolve with Nitro
DURATION
RESOLUTION
5 min
Rest or Nitro
ASSESSMENT
DIAGNOSIS
Pain
EKG
Dyspnea
Stress Test
Pallor
Cardiac Biomarkers
Sweating
Cardiac Cath
Tachycardia
INTERVENTIONS
PAIN provide rest and administer nitro as prescribed
O2 provide supplemental oxygen to the patient
12 LEAD EKG obtain 12 lead ekg
REST maintain bed rest to reduce O2 demands
ASSESS assess vital signs and pain
Syncope
HTN
[NRSNG Academy Lesson: Angina]
[NRSNG Academy Lesson: Angina]
Heart Blocks
HEART BLOCKS
FAILURE OF THE HEART’S NATURAL PACEMAKER DUE TO OBSTRUCTION
(“BLOCK”) IN THE ELECTRICAL CONDUCTION SYSTEM OF THE HEART.
Relationship of P waves to QRSs
All P waves are
followed by a
QRS but PR is >0.20
First-Degree
AV block
Progressive
lengthening of
PR until a
P is not
followed by
a QRS, then
repeated
Seconddegree
AV block,
Type I
Every other
P wave is not
conducted (2:1)
PR interval of
conducted P wave
is consistent
QRS
< 0.12
QRS is
0.12 or >
Only one
P wave in
a row is not
conducted
More than
one P wave
in a row
is not
conducted
QRS
< 0.12
Seconddegree
AV block,
Type I
Seconddegree
AV block,
Type II
Seconddegree
AV block,
Type II
High
grade
AV block
Complete
heart block
with
junctional
escape
rhythm
1°
Benign but
can progress
Significance
Treatment
No P waves are
followed by
(associated with)
QRSs (i.e.,
AV dissociation)
Some P waves are
not followed by QRSs
Observation,
d/c digitalis use
2° Type I
2° Type II
QRS is
0.12 or >
Complete
heart block
with
ventricular
escape
rhythm
3°
Block at AV node, Block at Bundle of
usually transient,
His, occurs with
does not usually anterior MI, often
progress
progresses to
complete block
Ventricular
asystole in absence
of escape beat
Close monitoring,
d/c digitalis use,
treat if patient
is symptomatic
Pacemaker,
atropine, monitor
for hypoperfusion
Atropine,
transcutaneous
or transvenous
pacemaker
[NRSNG Academy Lesson: 1st Degree AV Heart Block]
[NRSNG Academy Lesson: 1st Degree AV Heart Block]
Cardiac Auscultation
[NRSNG Academy Lesson: Heart Sounds]
Chest Pain Identification
CHEST PAIN IDENTIFICATION
CAUSE
PROVOCATION
- Ex
- Stress
- Cold
ANGINA
- Smoking
QUALITY
- Heavy
pressure
- Tightness
- Dull ache
REGION
- Substernal
- Radia
to jaw,
arms,
neck,
abdomen
SEVERITY
- Mild to
severe
TIMING
- Gradual or
sudden
onset
- <5 min but
may last
up to
15min
SIGNS/SYMPTOMS
- Tachycardia
TREATMENT
- Rest
- Dyspnea
- Oxygen
- N/V
- Nitro
- Diaphoresis
- Calcium
Channel
Blocker
- Anxiety
- ST-T wave
changes
- Stress
- Lifestyle
change
- Similar to
angina
- Pressure
on chest
- Clinched
fist over
chest
ACUTE MI
- Substernal
- Radia
to jaw,
arms, neck,
abdomen
- No
symptoms
to severe
- Sudden
onset
- >30min up
to 2 hours
- Tachycardia
- MONA
- Dyspnea
- Fibrinol
or
percutaneous
coronary
interven
- N/V
- Diaphoresis
- Anxiety
- Impending doom
- T wave inversion,
ST eleva
- S4
- Venous
stasis
PULMONARY
EMBOLISM
- Hyperco
agulability
- Vascular
injury
- Sharp
- Shoo
- Deep
- Worsened
with
inspira
- Substernal
or lateral
chest
- Mild to
severe
- Sudden
onset
minutes to
hours
- Radiates to
shoulder
and neck
- Tachycardia
- Narco
- Tachypnea
- High
Fowler’s
- Dyspnea
- Anxiety
- Chest
Splin
- Hemoptysis
- Thrombol
- Fever
- Chest
trauma
- Excessive
l
volume or
PEEP with
PNEUMOTHORAX mechanical
ven a
- Sharp
tearing
- Exacer
bated by
breathing
- Lateral
chest with
to
radia
shoulder,
arms, back
- Mild to
severe
- Sudden
onset
hours to
days
- Tachypnea
- Narco
- Tachycardia
- Chest tube
- JVD
- Anxiety
- Diminished
breath sounds
- Tracheal
devia
- Bleb
- Hyperresonance
- Dyspnea
[NRSNG Academy Lesson: Angina]
[NRSNG Academy Lesson: Angina]
H’s and T’s of ACLs
H’S AND T’S OF ACLS
A mnemonic used to aid in remembering the possible causes of cardiac arrest. A variety of disease
processes can lead to a cardiac arrest; however, they usually boil down to one or more of the "Hs and Ts".
H’s of ACLS
Causes
Signs
T’s of ACLS
Treatment
Hypovolemia
HR
Narrow QRS
Blood loss
Fluid administration
Fluid challenge
Hypoxia
HR
Cyanosis
Patent airway
Ventilate
Hydrogen
Ion (Acidosis)
Hyper/Hypokalemia
Hypothermia
ABG (Low pH)
Low amplitude QRS
Sodium bicarbonate
Ventilate
Flat T waves with U
wave (hypo) or
Peaked
T waves with wide
QRS (hyper)
Ventilate (metabolic)
Sodium bicarbonate
(respiratory)
Calcium Gluconate
Insulin and D50
Albuterol
Hypothermia
Warming measures
Causes
Toxins
Signs
Prolonged QT
Treatment
Antidote Based on
overdose
Tamponade
(Cardiac)
HR
Narrow QRS
JVD
Mu ed heart
Pericardiocentesis
Thoracotomy
Tension
Pneumothorax
HR
Narrow QRS
Unequal breaths
Tracheal deviation
Decompression
Chest tube
Thrombosis
(coronary or
pulmonary)
EKG alteration
Chest pain
Narrow QRS
SOB
Embolectomy
Fibrinolytics
Anticoagulants
Angioplasty
Stent
CABG
H’s
Hypovolemia: A lack of circulating body fluids, principally blood volume. This is usually (though not exclusively) caused
by some form of bleeding, anaphylaxis, or pregnancy with gravid uterus.
Hypoxia: A lack of oxygen delivery to the heart, brain and other vital organs. Rapid assessment of airway patency and
respiratory effort must be performed.
Hydrogen Ion: An abnormal pH in the body as a result of lactic acidosis which occurs in prolonged hypoxia and in
severe infection, diabetic ketoacidosis, renal failure causing uremia, or ingestion of toxic agents or overdose of
pharmacological agents.
Hyper/Hypokalemia:
Both excess and inadequate potassium can be life-threatening.
Hypothermia: A low core body temperature, defined clinically as a temperature of less than 35 degrees Celsius
(95 degrees Fahrenheit).
T’s
Toxins: Toxin ingestion is one of the most common causes of cardiac arrest. Prolonged QT is a common sign.
Tamponade: Blood or other fluids building up in the pericardium can put pressure on the heart so that it is not able to
beat
Tension Pneumothorax: The build-up of air into one of the pleural cavities, which causes a mediastinal shift. When this
happens, the great vessels (particularly the superior vena cava) become kinked, which limits blood return to the heart
Thrombosis: Hemodynamically significant pulmonary emboli are generally massive and typically fatal.
Text from Wikipedia.org used on CC license
[NRSNG Academy Lesson: Ventricular Tachycardia]
[NRSNG Academy Lesson: Ventricular Tachycardia]
Coronary Circulation
[NRSNG Academy Lesson: Coronary Circulation]
Types of Aneurysms
TYPES OF ANEURYSMS
An Aneurysm is an outpouching of the vessel wall due to weakened vessel muscle
layers. This is usually caused by hypertension. Blood ow becomes turbulent at the
sit of the aneurysm, pu ng it at high risk for rupture.
By Nichole Weaver, © NRSNG, LLC.
FUSIFORM
SACCULAR
DISSECTING
FALSE
Fusiform
aneurysms
surround the full
circumference of
the vessel. This is
common in the
Abdominal Aorta.
Saccular
aneurysms pouch
out from one side
of the vessel.
Also called “Berry
Aneurysms”.
Dissection occurs
when the inner
layer of the
vessel is torn
away. Blood ows
between vessel
layers instead of
forward.
False aneurysms
appear on scans,
but are actually a
blood clot
formed outside
the vessel. Blood
ow is not
impeded.
[NRSNG Academy Lesson: Aortic Aneurysm]
Patient Mobility
PATIENT MOBILITY
GENERAL HOME SAFETY GUIDELINES
Use nonslip mats, grab bars, and raised toilet seats
CRUTCHES
Standing up: scoot to front of chair, hold both crutches in hand of injured side, push
up on good foot.
Stairs:
Up
Down
Unaffected/strong leg first
Affected/weak leg and
crutches second
Affected/weak leg and
crutches first
Unaffected/strong leg second
WALKERS
ng
How to ambulate
Types
Push forward about 6” (or whatever is
Advance weak leg, then strong leg
walker
advance the walker and weak leg
together at once, then move the
stronger leg forward
able to push the walker
forward
CANES
Use on STRONG SIDE
Elbow should be flexed slightly
Tip of nearest point of the cane should be about 6” in front of and 6” to the side
of that strong leg
How to ambulate
Advance the cane about 1 foot forward (or
Types
Straight or regular
Tripod
Quad
Move the WEAK SIDE while the cane and
Then move the strong leg ahead, while
the weaker leg is supported by the cane
Sources American Academy of Orthopaedic Surgeons. How to Use C
rutches, Canes, and Walkers-OrthoInfo - AAOS.
.org/topic.cfm topic=a00181
[NRSNG Academy Lesson: Fall and Injury Prevention]
Chest Tube Care
[NRSNG Academy Lesson: Chest Tube Management]
Pathophysiology Concept Map
PATHOPHYSIOLOGY CONCEPT MAP
Risk Factors
Sign And Symptoms
Potential
Complications
DISEASE PROCESS
Pathophysiology (Definition / etiology chronicity and prognosis)
Medical intervention, Labs and diagnostic studies
Nursing Diagnosis
Nursing Interventions
Head to Toe Assessment
[NRSNG Academy Lesson: Head to Toe Nursing Assessment]
Pressure Ulcers
[NRSNG Academy Lesson: Complications of Immobility]
Types of Lines
[NRSNG Academy Lesson: Preparing an IV Infusion]
Medical spanish
MEDICAL SPANISH
BASIC ASSESSMENT
ESSENTIAL WORDS
My name is _______ . I am your nurse.
Me llamo _______. S oy su enferme ra (o).
Please
Por favor
What is your nam e?
¿Cómo se llam a?
Thank you
Gracias
How are you feeling?
¿Cómo se sie nte?
Never
Nunca
Date
Fecha
Sign ature
Firma
Good- bye
Adiós
Where
Donde
What is the d ate?
¿A qué fecha e stamos?
Where are you?
¿Dónde e stá usted?
How old are you?
¿Cuá ntos añ os tiene?
Where do you live?
¿Dónde vive usted?
Are you having pai n? Where?
¿Tiene do lor? Dond e?
PHYSICAL ASSESSMENT
MEALTIMES
Brea kfast
Desayuno
Lunch
Almuer zo
Dinner
Cena
ESSENTIAL ITEMS
Blank et
Manta
Brush
Cepillo
Gown
Bata
Lotion
Loción
Pillow
Almohada
Shampoo
Champú
Shaving cream
Crema de afeitar
Sheet
Sábana
Soap
Jabón
Towel
Toalla
Do you have_________?
¿Tiene usted ________?
Have you ever had______?
Ha tenido alguna vez ________?
Itching
Picazón
Sores
Llagas
Edema/swelling
Edema/hinchazón
Pain
Dolor
Chest pain
Dolor de pecho
Nausea
Náuseas
Vomiting
Vómitos
POSITIONING
Lean backward
Recuéstese
Lean forward
Inclínese hacia adelante
Lie down
Acuéstese
Sit down
Siéntese
Stand up
Póngase de pie
MEDICAL SPANISH
ANATOMY
Skin
Piel
Chest
Pecho
Lungs
Pulmones
Eyes
Ojos
Heart
Corazón
Kidneys
Riñones
Pulse
Pulso
Arm
Brazo
Leg
Pierna
BASIC NURSING PROCEDURES
I'm going to take your ________.
Voy a medirle _______.
Vital signs
Los signos vitales
Blood pressure
La presión sanguínea
Pulse
El pulso
Temperature
La temperatura
I'm going to take a blood sample.
Voy a tomarle una muestra de sangre.
Can you provide a urine specimen?
¿Puede darnos un espécimen de orina?
DIAGNOSTIC TESTS
Biopsy
Biopsia
Blood test
Análisis de la sangre
Blood culture
Cultivo de la sangre
CT scan
Tomogra a computarizada
Ultrasound
Ultrasonido
X-ray
Radiogra a
Therapeutic Diets
THERAPEUTIC DIETS
NAME
CONSIDERATIONS
NPO
Nothing by mouth
(nil per os)
CLEAR
LIQUID
FULL
LIQUID
PUREED
DIET
transparent to light and liquid at body temperature
broth
hard candy
gelatin
popsicles
water
coffee
fruit juice
tea
clear and opaque liquid foods at body temperature
all clear liquid items
ice cream
sherbet
breakfast drinks
fat free & 1% milk
pudding
thin hot cereals (cream of wheat)
foods that require no chewing
all full liquid items
mashed potatoes
MECHANICAL
foods that require less chewing
tender fruits and vegetables
chopped, ground, & pureed foods
tender meats
LOW RESIDUE/
LOW FIBER
limit fiber intake to 10g a day
white rice
HIGH RESIDUE/
HIGH FIBER
high fiber intake 20-35g a day
fruits and vegetables
CONSISTENT
CARB
limited starches, juice, fruit, milk, and sugars
CARDIAC
restrict fat and sodium intake
SODIUMRESTRICTED
limit sodium intake to set amount (4
DIET
white bread
refined cereals and pastas
whole-grain products
control carbohydrate intake
(diabetic diet)
g,
3g , 2g , 1g , 500mg)
avoid canned, frozen, boxed, smoked, salted foods
[NRSNG Academy Lesson: Nutrition]
IV Sites and Considerations
[NRSNG Academy Lesson: Preparing an IV Infusion]
Patient
Safety
PATIENT SAFETY
FIRE SAFETY
RACE
RESTRAINT SAFETY
PASS
R: Rescue patients in danger
P: Pull the fire extinguisher pin
Use the least restrictive method possible
A: Activate the fire alarm
A: Aim at the base of the fire
Order for restraints needs to be renewed within set time frame
C: Confine the fire
S: Squeeze the handle
Never ordered PRN
E Extinguish the fire
S: Sweep extinguisher from side
to side
Assess skin integrity, neurovascular, and circulatory status every 30m
Remove restraint every 2 hours to check pressure areas
FALL SAFETY
STANDARD PRECAUTIONS
CONTACT PRECAUTIONS
Asses for risk factors
Hand hygiene before and after every patient contact
DISEASES
PROTECTION
Bed in low and locked position
Use PPE when risk of body fluid exposure
Norovirus
Private room
Bed alarm as needed
Use and dispose of sharps safely
Rotavirus
1:1 monitoring
Clostridium difficile
Gown and gloves when
in patient room
Clean all shared patient equipment
Draining wounds
Use of aseptic technique
MDROs
Dispose of all waste and linen safely
AIRBORNE PRECAUTIONS
DISEASES
PROTECTION
Hand hygiene on exit,
Soap and water for C. Diff
DROPLET PRECAUTIONS
DISEASES
PROTECTION
Measles
Single negative pressure room
Influenza
Pneumonia
Private room
Chickenpox
6-12 air exchanges per hour
Meningitis
Sepsis
Surgical mask within 3 feet of
patient
TB
Wear respirator or mask
Mumps
Pertussis
Mask must be worn by client when
leaving room
Rubella
Patient must wear a mask when
leaving room
Colostomy Care
[NRSNG Academy Lesson: Diverticulosis – Diverticulitis]
Abdominal
Pain
ABDOMINAL PAIN
9 Regions of the Abdomen
Gallstones
Cholecystitis
Stomach ulcer
Duodenal ulcer
Hepatits
Heartburn
Indi estion
Hiatal hernia
Epigastric hernia
Stomach ulcer
Duodenal ulcer
Hepatitis
Dyspepsia
astritis
Stomach ulcer
Pancreatitis
Kidney stones
Kidney infection
IBD
Consti ation
Umbilical hernia
Early a endicitis
Stomach ulcer
IBD
Pancreatitis
Kidney stones
Kidney infection
IBD
Consti ation
A endicitis
Inguinal Hernia
IBD
Pelvic Pain
Consti ation
Bladder infection
Prostatitis
Diverticulitis
IBD
Inguinal hernia
Pelvic pain
Consti ation
IBD
Pelvic pain
Inguinal hernia
CRITICAL POINT:
When assessing the abdomen the correct assessment order is:
Inspect, Auscultate, Percuss, Palpate
Ulcerative Colitis Vs. Crohn’s
Disease
[NRSNG Academy Lesson: Inflammatory Bowel Disease]
Chronic kidney Disease symptoms
CHRONIC KIDNEY DISEASE SYMPTOMS
By Nichole Weaver, © NRSNG, LLC.
Chronic Kidney Disease involves symptoms associated with LOSS of normal kidney unctions such as
Excretion of waste products
Production of urine
Electrolyte balance
Acid-base balance
Fluid volume regulation
Erythropoietin - creation of RBCs
[NRSNG Academy Lesson: Chronic Kidney Disease]
[NRSNG Academy Lesson: Chronic Kidney Disease]
Types of Viral Hepatitis
TYPES OF VIRAL HEPATITIS
VIRAL TYPE
TRANSMISSION
PREVENTION
Hepatitis A Virus
(HAV)**
Fecal - Oral
Hand Hygiene
Safe Food Handling
Vaccine
Safe Sex Practices
Handwashing
Needle Safety
Blood Screening
Vaccine
Hepatitis B Virus
(HBV)**
Blood - Body Fluids
Hepatitis C Virus
(HCV)**
Blood
Hepatitis D Virus
(HDV)
Blood - Body Fluids
Same as HBV
Fecal - Oral
Same as HAV
Hepatitis E Virus
(HEV)
[NRSNG Academy Lesson Hepatitis]
[NRSNG Academy Lesson: Hepatitis]
Handwashing
Needle Safety
Blood Screening
Types of Anemia
TYPES OF ANEMIA
Anemia involves a decreased oxygen carrying capacity of the blood due to a
change in the amount, size, or shape of the red blood cell or the amount of
hemoglobin available for binding oxygen to the cell.
Anemia of Blood Loss
Hemorrhage
COLOR
SIZE
SHAPE
Normochromic
Normocytic
Normal
Normochromic
Normocytic
Normal
Hypochromic
Normocytic
Normal
Normochromic
Macrocytic
Enlarged
Normochromic
Microcytic
Abnormal
A lastic Anemia
Leukemia
Medications
Iron-De ciency Anemia
Poor iron intake
Chronic blood loss
Pernicious Anemia
Poor Vitamin B12 intake
Sickle Cell Anemia
Genetics
Triggers (cold, infection)
[NRSNG Academy Lesson: Anemia]
[NRSNG Academy Lesson: Anemia]
Burn staging
BURN STAGING
First Degree
Second Degree
Reddened,
painful, intact
skin
Partial
Thickness,
broken skin,
pain, pink/red,
blisters
By The original uploader was
Snickerdo at English Wikipedia Transferred from en.wikipedia to
Commons., CC BY-SA 3.0,
h ps //commons.wikimedia.org/w/in
dex.php?curid=3358773
Third Degree
Fourth Degree
Full thickness,
often painless,
white/black
eschar
Muscle and/or
bone exposed.
Common in
electrical
burns
By Cli ord Sheckter, Arhana
Cha opadhyay, John Paro and Yvonne
Karanas - Direct source. Full paper., CC
BY 4.0,
h ps //commons.wikimedia.org/w/in
dex.php?curid=68491398
By goga312. Original uploader was
Goga312 at ru.wikipedia - Transferred
from ru.wikipedia(Original te t
), CC BY-SA 3.0,
h ps //commons.wikimedia.org/w/in
dex.php?curid=7771672
[NRSNG Academy Lesson: Burn Injuries]
[NRSNG Academy Lesson: Burn Injuries]
skin Cancer
[NRSNG Academy Lesson: Skin Cancer]
skin Lesions
SKIN LESIONS
Macule and Patch
A macule is a at area of hyperpigmentation, usually < 10mm.
A Patch is a larger macule (usually > 10mm).
By Madhero88 - Own work, CC BY-SA
, h ps //commons.wikimedia.org/w/index.php?curid=14546457
Papule and Plaque
A papule is a well-de ned raised area with no visible ui ,
usually < 10 mm.
A plaque is a large papule or group of them, usually > 10 mm,
or a large raised plateau-like lesion
By Madhero88 - Own work, CC BY-SA
, h ps //commons.wikimedia.org/w/index.php?curid=14546485
Nodules
A nodule is similar to a papule - raised area with no uid - but
is much deeper in the dermis than a papule.
By Madhero88 - Own work, CC BY-SA
, h ps //commons.wikimedia.org/w/index.php?curid=14546471
Vesicles and Bulla
A vesicle is a small, well-de ned raised area lled with uid,
usually <10mm.
A Bulla is a large vesicle, usually >10mm.
Both are also known as blisters.
By Madhero88 - Own work, CC BY-SA
, h ps //commons.wikimedia.org/w/index.php?curid=14546567
Fissures, Erosions, and Ulcers
A ssure is a crack in the skin that is usually narrow but deep.
Erosions involve full loss of the epidermis in a de ned area.
Ulcers involve loss of the epidermis and some or all of the dermis.
By Madhero88 - Own work, CC BY-SA
, h ps //commons.wikimedia.org/w/index.php?curid=14546561
[NRSNG Academy Lesson: Integumentary Important Points]
[NRSNG Academy Lesson: Integumentary Important Points]
Lab Value skeletons
LAB VALUE SKELETONS
Complete Blood Count (CBC)
Liver Enzymes
Hgb
T. Bili
D. Bili
WBC
PLT
Hct
AST
ALT
ALK Phos
Arterial Blood Gas (ABG)
pH
PaCO2
PaO2
HCO3
BE
Basic Metabolic Panel (BMP or CHEM-7) and CHEM-10
Na
Cl
K
HCO3
BUN
Ca
Glu
Mg
Cr
Phos
Bleeding
Times
Liver Profile
Ca
TP
AST
LDH
PO4
Alb
ALT
ALP
Bili
PT
[NRSNG Academy Lesson: Shorthand Lab Values]
[NRSNG Academy Lesson: Shorthand Lab Values]
PTT
INR
Lab Value for Clinical
[NRSNG Academy Lesson: Lab Panels]
Blood Compatibility Chart
BLOOD COMPATIBILITY CHART
DONOR BLOOD TYPE
O-
O+
B-
B+
A-
A+
AB-
PATIENT BLOOD TYPE
AB+
ABA+
AB+
BO+
O-
[NRSNG Academy Lesson: Sickle Cell Anemia]
[NRSNG Academy Lesson: Sickle Cell Anemia]
AB+
ABg Rome Flowchart
ABG ROME FLOWCHART
pH
HIGH
LOW
Alkalosis
Acidosis
HIGH
PaCO2
Respiratory
Acidosis
LOW
HCO3
LOW
PaCO2
HIGH
HCO3
Metabolic
Acidosis
Respiratory
Alkalosis
Metabolic
Alkalosis
[NRSNG Academy Lesson: ABG Labs]
[NRSNG Academy Lesson: ABG Labs]
Cardiac Biomarkers
MYOGLOBIN
CARDIAC BIOMARKERS
5-70 ng/mL
Rise
Peak
Return
1-4 hours
6-12 hours
1-2 days
TROPONIN I
CK-MB
<2.40 ng/mL
Rise
6-10 hours
12-24 hours
Peak
2-3 days
Return
<0.035 ng/mL
Rise
Peak
4-6 hours
18 hours
Return
1-2 weeks
LDH1
88-230 U/L
Rise
8-12 hours
72 hours
Peak
1-2 weeks
Return
[NRSNG Academy Lesson: Dysrhythmias]
[NRSNG Academy Lesson: Dysrhythmias]
IV Colors
and gauges
IV THERAPY
USES
FLOW RATE LENGTH SIZE
(mm)
(ml/min)
COLOR
IV COLORS AND GAUGES
14G 16G 18G 20G
22G 24G
45
45
32
32
25
19
240
180
90
60
36
20
Trauma
Rapid Blood
Surgery
Rapid Blood
Surgery
Routine
Blood
Surgery
Routine
Blood
Small Veins
Peds
Routine
Peds
Important Points:
Check facility protocols. In general blood should not be given in a
catheter <20G. Keep in mind the a smaller gauge # means a larger
IV catheter. Most adult patients will need an 18G or 20G. Always
consider what fluids the patient will be receiving before determining
size.
[NRSNG Academy Lesson: Preparing an IV Infusion]
[NRSNG Academy Lesson: Preparing an IV Infusion]
ABg Analysis
ABG ANALYSIS
Normal ABG Values
pH
DISORDER
7.35-7.45
PaCO 2
35-45 mmHg
HCO3-
22-26 mEq/L
PaO2
80-100 mmHg
CAUSES
ASSESSMENT FINDINGS
TREATMENTS
Respiratory Acidosis
pH < 7.35; PaCO2 > 45
Hypoventilation
-CNS depression
-Pulmonary edema
-Respiratory arrest
-Airway obstruction
-Bradycardia
-Hypotension
-Confusion
-Somnolence
-Increase RR
-Reposition patient
-Maintain patent airway
-Mechanical ventilation
- Rate
- Vt
Respiratory Alkalosis
pH > 7.45; PaCO2 < 35
Hyperventilation
-Excessive
mechanical ventilation
-Anxiety
-Fever
-Pneumothorax
-Tachycardia
-Palpitations
-Anxiety
-Seizures
-Perspiration/
diaphoresis
-Decrease RR
-Administer sedatives
-Rebreather mask
-Mechanical ventilation
- RR
- Sedation
- Vt
Metabolic Acidosis
pH < 7.35; HCO3 < 22
Acid Gain
-Shock
-Ketoacidosis
-Renal failure
Bicarbonate loss
-Diarrhea
-Bile drainage
-Nausea/vomiting
-Malaise
-Tachypnea
-Hypotension
-Confusion
-Improve oxygenation
-Treat Cause
-DKA
-Diarrhea
-Renal failure
Metabolic Alkalosis
pH > 7.45; HCO3 > 26
Acid Loss
-Vomiting
-Potassium loss
(diuretic use)
-Hyperaldosteronism
- Cushing's
- Steroids
- Bicarbonate
gain
- Nausea/vomiting/
diarrhea
-Confusion
-Seizures
-Tetany
-Administer buffer
-Treat cause
[NRSNG Academy Lesson: ABG Labs]
[NRSNG Academy Lesson: ABG Labs]
Fluids and Electrolytes
[NRSNG Academy Lesson: Potassium – K]
Endocrine study guide Chart
ENDOCRINE STUDY GUIDE CHART
UNDER PRODUCTION
SYNDROME
OVER PRODUCTION
SYNDROME
HORMONE
GLAND
GH
Anterio r Pituitary
ADH
Posterio r Pituitary
Diabetes Insipidus
SIADH
T3,T4
Thyroid
Myxedema Coma
Graves
PTH
Parathyroid
Hypopa
Hyperparathyroid
rathyroid
Hyperpa
Hypoparathyroid
rathyroid
Cortisol
Adrenal
Addisons
Cushings
Insulin
Panc reas
Diabetes Mellitus
Acromega ly
[NRSNG Academy Lesson: Addisons Disease]
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NRSNG.com
[NRSNG Academy Lesson: Addisons Disease]
Addison’s Vs. Cushing’s
[NRSNG Academy Lesson: Addisons Disease]
Hyper Vs. Hypothyroidism
HYPER VS. HYPOTHYROIDISM
Body System
Hypothyroidism
Metabolic
Hypometabolic
Hypoglycemia
Cold intolerance
Weight gain / Edema
Hypermetabolic
Temperature
Heat Intolerance
Weight Loss
Bradycardia
Hypotension
Anemia
Tachycardia
Hypertension
Palpitations
Neurological
Lethargy / Fatigue
Weakness
Muscle aches
Paresthesias
Hyperactive reflexes
Hand tremor
Emotional instability
Agitation
Integumentary
Dry skin
Loss of body hair
Fine, thin hair
Constipation
Goiter
Exophthalmos
Goiter
Cardiovascular
Other
Hyperthyroidism
T3, T4
Hormone Levels
T3, T4
Free T4
Free T4
TSH
TSH
[NRSNG Academy Lesson: Hyperthyroidism]
[NRSNG Academy Lesson: Hyperthyroidism]
Fracture Management
FRACTURE MANAGEMENT
Strain
excessive stretching of muscle
Sprain
excessive stretching of ligament
TREATMENT
ICE Rest, Ice, Compression, Elevation
TYPES OF FRACTURES
By OpenStax College - Anatomy & Physiology, Connexions Website.
h p //cnx.org/content/col11496/1.6/, Sep 7, 2015., CC BY 4.0,
|
h ps //commons.wikimedia.org/w/index.php?curid=30127535
TRACTION
Buck’s Traction
Skeletal Traction
force applied to splint
pin through bone to hold weight
Force in opposite direction
Realign & immobilize fracture
Traction weights:
Hang freely
Do not remove without order
Support weight when moving patient
MONITOR FOR COMPLICATIONS
Fat Embolism
Compartment Syndrome
[NRSNG Academy Lesson: Fractures]
[NRSNG Academy Lesson: Fractures]
Stroke Symptoms by Location
[NRSNG Academy Lesson: Assessment]
Neuro Dysfunction by
Pupil Assessment
[NRSNG Academy Lesson: Routine Neuro Assessments]
Routine Neuro Assessments
ROUTINE NEURO ASSESSMENTS
Pupils Equal, Round, and Reactive to Light and Accommodation
(PERRLA) + Size in mm
LEVELS OF CONSCIOUSNESS
Normal
A&O x 4, Alert
Confused
A&O x <3, unable to answer
Delirious
Confused and agitated
Somnolent
Excessively sleepy or drowsy
Obtunded
Awake, but slow or no response to surroundings
Stuporous
Sleep-like, no spontaneous activity, withdraws to pain
Coma
SCORE
NO response to stimuli, unable to arouse
GLASGOW COMA SCALE
2
4
3
1
5
6
-
-
Eyes
No opening
Open to pain
Open to voice
Open
spontaneously
Verbal
No response
Incomprehensible
sounds
Inappropriate
words
Disoriented
Oriented
-
Motor
No response
Abnormal
Extension
Abnormal
Flexion
Withdraws
to Pain
Localizes
to Pain
Follows
Commands
MUSCLE STRENGTH
SCORE
ABILITY
0
No muscle contraction
1
Muscle twitch
2
Movement without gravity
3
Movement against gravity
4
Movement against resistance
5
Full Strength
[NRSNG Academy Lesson: Routine Neuro Assessments]
[NRSNG Academy Lesson: Routine Neuro Assessments]
Newborn Assessment
[NRSNG Academy Lesson: Initial Care of the Newborn]
Labor
LABOR
STAGES OF LABOR
First Stage
Second Stage
acement an
ilation of cervi
pulsion of fetus
ree sta es latent, active,
an transition
Mot er is tal ative an
ea er in latent p ase,
becomin tire , restless,
an ious as labor intensi es
an contractions become
stron er
Pus in sta e
Third Stage
eparation of placenta
pulsion of placenta
Mot er as intense
concentration on pus in
it contractions may fall
asleep bet een contractions
Mot er is relieve after
birt of ne born mot er
is usually very tire
Fourth Stage
P ysical recovery
r after e pulsion
of placenta
Mot er is tire , but is ea er
to become ac uainte
it
er ne born
FETAL POSITIONS
Verte Positions
ROA (right occipitoanterior)
O
left occipitoanterior
ROP ri
t occipitoposterior
ace Positions
RMA (right mentoanterior)
M
left mentoanterior
RMP ri
reech Positions
left sacroanterior
P left sacroposterior
Other
Brow
Shoulder
t mentoposterior
OP left occipitoposterior
ROT (right occipitotransverse)
O
left occipitotransverse )
FETAL MONITORING
VEAL – CHOP
V
E
A
L
VARIABLE DECELERATION
EARLY DECELERATION
ACCELERATION
LATE ACCELERATION
C
H
O
P
CORD COMPRESSION
HEAD COMPRESSION
OKAY!
PLACENTAL INSUFFICIENCY
[NRSNG[NRSNG
Academy
Mechanisms
AcademyLesson:
Lesson: Mechanisms
of Labor] of Labor]
Clinical Assistant – Brain sheet
CLINICAL ASSISTANT - BRAIN SHEET
Date:
Patient Initials
Floor:
Room Number:
Reason for hospitalization:
Focused Ass ess me nt:
Assess me nt Notes:
Consultations/ Tests:
Name
Patient Med ications:
Reason
Considera ons
[NRSNG Academy Lesson: Documentation]
[NRSNG Academy Lesson: Documentation]
Time
CLINICAL ASSISTANT - BRAIN SHEET
Normal L ab Values
Na
K
Cl
CO2
BUN
Creat
pH
135-148
3.5-5.3
100-112
23-29
5.0 - 25.0
0.5 - 1.7
7.35-7.45
WBC
RBC male
RBC female
Hgb male
Hgb female
Hct male
Hct female
3.6-9.2
4.39-5.58
3.70-5.14
13.7-17.3
12-15.5
39-49
35-46
Platelet
Albumin
Ca
PT
aPTT
INR
Billirubin
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
Patient Vitals
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
Time
Pulse
Pulse Ox
Respirations
BP
Temp
Pain
140-400
3.5-5.0
8.3-10.3
10.4-12.2
24-33
2.0-3.0
0.0-1.0
“Nurses Dispense
Comfort, Compassion,
and Caring Without
Even a Prescription.”
Val Saintsbury
Intake & Output
IV Site Assess me nt/Fluid/Rate
Tasks/ Notes
Calculations
Things to Research/I mpr ove
[NRSNG Academy Lesson: Documentation]
Pediatric Burn Chart
PEDIATRIC BURN CHART
BASED ON LUND BROWDER CHART
A
A
1
1
2
13
2
2
1
1 1/4
1 1/4
1
1 1/4
1 1/4
2
1
B
B
C
C
BIRTH
A: 1/2 of Head
9 1/2
B: 1/2 of Thigh
2 3/4
C: 1/2 of Leg
2 1/2
C
C
1 3/4
1
1
1 1/4
2 1/2 2 1/2
B
B
6 1/2
3 1/4
4
2 1/2
2 3/4
AGE 15 YR
ADULT
4 1/2
3 1/2
4 1/2
4 3/4
3 1/4
3 1/2
2
1 1/2
1 1/4
AGE 5 YR
8 1/2
13
2
2
AGE 1 YR
1 3/4
1
1 1/2
B
B
A
1 1/2
1 1/4
2
B
A
13
2
AREA
1
1
2
13
1
1
1 1/2
B
1 1/4
C
1 3/4
C
1 3/4
AREA
AGE 10 YR
A: 1/2 of Head
5 1/2
B: 1/2 of Thigh
4 1/2
C: 1/2 of Leg
3
C
1 3/4
C
1 3/4
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[NRSNG Academy Lesson: Burn Injuries]
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NRSNG.com
[NRSNG Academy Lesson: Burn Injuries]
Erikson’s stages of Psychosocial
ERIKSON’S STAGES
Development
OF PSYCHOSOCIAL
DEVELOPMENT
AGE
STAGES
CHARACTERISTICS
Infancy (birth to 18 months)
Trust vs Mistrust
Development of trust based on
caregivers
Early childhood (18 mo - 3yr)
Autonomy vs Shame and Doubt
Development of sense of
personal control
Preschool (3-5yr)
Initiative vs Guilt
Development of sense of
purpose and directive
School age (6-11yr)
Industry vs Inferiority
Development of pride in accom plishments
Adolescence (12-18yr)
Identity vs Role Confusion
Exploration of independence and
development of self
Early adulthood (18-40yr)
Intimacy vs Isolation
Development of personal
relationships and love
Adulthood (40-65yr)
Generativity vs Stagnation
Fulfilling goals and building
career and family
Older adult (65yr-death)
Integrity vs Despair
Looking back on life with accep tance
[NRSNG Academy Lesson: Theories of Growth and Development]
[NRSNG Academy Lesson: Theories of Growth and Development]
Congenital Heart Defects
CONGENITAL HEART DEFECTS
CYANOSIS
NO
Vascularity
Increased
Vascularity
Normal
Aortic Stenosis
Pulmonic Stenosis
oarctation of the Aorta
L Atrium
Enlarged
YES
YES
efect
Transposition of the
reat Arteries (T A)
Truncus Arteriosus
TAPV
Tricuspid Atresia
Tingle Ventricle
Cardiac
Enlargement
YES
Ebstein’s Anomaly
Pulmonic Atresia
Tricuspid Atresia
Aorta
Enlarged
Patent uctus
Arteriosus (P A)
Vascularity
Decreased
NO
Atrial Septal
YES
Vascularity
Increased
NO
Tetralogy
of allot
NO
Ventricular Septal
efect
NRSNG.com
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- “Tools
Academy
andLesson:
Confidence
Congenital
to Succeed
Heart in
Defects]
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[NRSNG Academy Lesson: Congenital Heart Defects]
Crystalloid IV Solutions
CRYSTALLOID IV SOLUTIONS
IVF
Content
Tonicity
Osmolality
(mOsm/L)
Uses
D5W
- 50 g/L glucose
- 170 Kcals/L
- no electrolytes
Isotonic
252
- treat hypernatremia, replace water loss
- free water (helps renal excretion of solutes)
- used to administer medications
D10W
- 100 g/L glucose
- 340 Kcals/L
- no electrolytes
Hypertonic
505
- free water only
154
- maintenance solution, but doesn’t
replace other daily electrolytes
- free water and NaCl
- replace hypotonic fluid loss
- can cause IVF overload if
infused too rapidly
Isotonic
308
- used for postoperative fluids
- increase IVF and replace ECF
fluid losses
- NaCl in higher concentration
then blood levels
- no free water
- can cause IVF overload
- only solution that can be
administered with blood products
Hypertonic
1026
- 0.45% saline
½NS
- 77 mMol/L of Na+ and Cl
- no electrolytes
Hypotonic
-
- 0.9% saline
NS
- 154 mMol/L of Na+ and Cl - no calories
- 3.0% saline
3%NS
D5-¼NS
- 513 mMol/L of Na+ and Cl-
- cerebral edema
- 0.225% saline
- 50 g/L glucose
- Provides NaCl and free water
- 170 kcals/L
- 38.5 mMol/L of Na+ and Cl
Isotonic
330
- maintenance solution, but doesn’t
replace other daily electrolytes
- free water and NaCl
- 50 g/L glucose
Hypertonic
- 170 kcals/L
- 77 mMol/L of Na+ and Cl
406
- 50 g/L glucose
Hypertonic
- 170 kcals/L
- 154 mMol/L of Na+ and Cl -
- replace hypotonic fluid loss
- can cause IVF overload if infused
too rapidly
-
- 0.9% saline
D5-NS
- treatment of hypernatremia
- replace hypotonic fluid loss
-
- 0.45% saline
D5-½NS
- administer cautiously, slowly
treatment for symptomatic
hyponatremia
560
- increase IVF and replace ECF
fluid losses
- used for postoperative fluids
- NaCl in higher concentration
then blood levels
- no free water
- can cause IVF overload
[NRSNG Academy Lesson: Preparing an IV Infusion]
[NRSNG Academy Lesson: Preparing an IV Infusion]
Drug Card
DRUG CARD
Generic Name
Pharmacologic Class
Trade Name
Therapeutic Class
_
Action
_
Reason Given (Disease States)
_
Nursing Process
Pre-Administration Assessment
Post Administration Evaluation
Nursing Considerations
Other
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
______________________________________________________________________________________________________________________________
[NRSNG Academy Lesson Essential NCLEX Meds by Class]
[NRSNG Academy Lesson: Essential NCLEX Meds by Class]
Nervous system Pharmacology
NERVOUS SYSTEM PHARMACOLOGY
Nervous System
CNS
Brain
PNS
Spinal
Cord
Motor
Neurons
Autonomic
Nervous System
Sympathetic
Nervous System
(Adrenergic)
“Fight or Flight”
Primarily Norepinephrine (Adrenaline)
Alpha Receptors: vessels
Beta1-Receptors: heart
Beta2-Receptors: bronchial/vascular
smooth muscle
Dopaminergic: renal/mesenteric artery
Sensory
Neurons
Somatic
Nervous System
Parasympathetic
Nervous System
(Cholinergic)
“Rest and Digest”
Primarily ACh
Muscarinic Receptors
Nicotinic Receptors
MED CLASSES/EXAMPLE MEDS
ADRENERGIC AGONIST (SYMPATHOMIMETIC):
Mimics the effects of the SNS. (dobutamine,
dopamine, epinephrine, norepinephrine,
phenylephrine, albuterol, isoproterenol,
salmeterol).
CHOLINERGIC AGONISTS
(PARASYMPATHOMIMETIC): Mimic the effects
of the PNS. (donepezil, bethanechol,
pyridostigmine).
ADRENERGIC BLOCKING AGENTS
(SYMPATHOLYTIC): Block the effects of the SNS.
(beta-blockers, amiodarone, tamsulosin, doxazosin,
phentola mine).
ANTICHOLINERGIC AGENTS
(PARASYMPATHOLYTIC): Block the effects
of the PNS. (atropine, scopolamin, ipratropium,
trospium).
[NRSNG Academy Lesson: Autonomic Nervous System]
[NRSNG Academy Lesson: Autonomic Nervous System]
Dopamine Vs Dobutamine
DOPAMINE VS DOBUTAMINE
DOPAMINE
DOBUTAMINE
Vasopressor
Inotrope Only
Alpha 1 effects leading to
Primarily exhibits Beta 1 effects
to aid in increasing CO
Action
ects
Beta 1 effects leading to an
increase in HR at lower doses.
Increased perfusion to the
kidneys at 5 mcg/kg/min.
Effective to
BP in distributive
shocks (septic, anaphylactic) as
it will contribute to
SVR
through vasoconstriction
Usage
Drug
CO
**Dobutamine
Dopamine
MAP
SVR
Phenylephrine
PAOP
Generally given to
CO. Used
in HF and Cardiogenic shock.
Drug of choice for
CO as it does not
cause
HR at lower doses leading to
less myocardial oxygen demand
SVR
HR
same or
CO
Vasopressin
Norepinephrine
Epinephrine
Dopamine
Dobutamine
SVR
Check us out on YouTube
CO
View Jon's Books on Amazon.com
Find our Podcast on iTunes and Stitcher: Search "NRSNG"
[NRSNG Academy Lesson: Cardiogenic Shock]
[NRSNG Academy Lesson: Cardiogenic Shock]
Beta Blockers
BETA BLOCKERS
Sympathetic Nervous System - ight or light
o SNS stim ulates recep tors throug hout the body to create "fight or flight response"
o Recep tors
Alpha - ves sels
Beta 1 - Hea rt
Beta 2 - Lungs
eta
Receptors
o When stim ulated by SNS they cause:
Increase Cardiac Output
Increase HR in SA node (Chronotropic effect)
Increase atrial contractility (Inotropic effect)
Increase conduction and autom aticity of AV node
Increase conduction and autom aticity of ventricles
oal of eta loc er Therapy
o Goal is to block stim ulation of B1 receptors in heart = HR
ommon
- loc ers
o Metoprolol
o Esm olol
o Propranolol
Side
ects of
- loc ers
o Bradycardia
o
Blood Pres sure
o Bronchoconstriction
o Blood sug ar abnorm alities
[NRSNG Academy Lesson: Disease Specific Medications]
Common ICU Drips
[NRSNG Academy Lesson: Vasopressin]
Medication Antidotes
MEDICATION ANTIDOTES
Med ication
Acetam inophen
Anticholinesterase
Anticholinergics
Benzodiazepines
Beta -Blockers
Ca Channel Blockers
Coumadin
Digo xin
Dopam ine
Heroin
Heparin
Iron
Malignant Hyperthermia
Methotrexate
Narcotics
Potassium
Tricyclic Antide pressants
Antidote
acetylcysteine, mucomyst
atropine, pralidoxine
physos tigmine
Romazicon (flumaze nil)
glucago n, epinephrine
Ca Chloride, glucag on
phy tonadione, vitam in K
Digibind
Rigitine
Narcan (naloxone)
protamine sulfate
d eferoxamine
dantrolene
leucovorin calcium
Narcan (nalxone)
Insulin, Bicarb, albut erol, Kayexa late
physos tigimine, Bicarb
[NRSNG Academy Lesson: 6 Rights of Medication Administration]
[NRSNG Academy Lesson: 6 Rights of Medication Administration]
Insulin Cheat sheet
INSULIN CHEAT SHEET
Intermediate
Acting
Pre- Mixed NPH
w/ Reg ular
ONSET
PEAK
DURATION
15m
30-90m
3-5h
Apidra
Insulin glulisine
15m
30-90m
3-5h
Humalog
Insulin lispro
15m
30-90m
3-5h
Humulin R
Regular
30-60m
2-4h
5-8h
Novolin R
Regular
30-60m
2-4h
5-8h
Humulin N
NPH
1-3h
8h
12-16h
Novolin N
NPH
1-3h
8h
12-16h
Humulin 70/30
70%NPH and 30% Reg
30-60m
varies
10-16h
Novolin 70/30
70%NPH and 30% Reg
30-60m
varies
10-16h
Humulin 50/50
50%NPH and 50% Reg
30-60m
varies
10-16h
MIXING INSULIN
REGULAR
Short-Acting
GENERIC NAME
Insulin aspart
REGULAR
Rapid-Acting
BRAND NAME
NovoLog
NPH
TYPE
NPH
1) Withdraw enough air equal to the total amount of insulin.
2) Inject the air into the NPH without touching the insulin.
3) Inject remaining air into the regular insulin then withdraw
the regular dosage.
4) Withdraw the NPH dosage.
[NRSNG Academy Lesson: Diabetes Management]
[NRSNG Academy Lesson: Diabetes Management]
Common Antihypertensive Drugs
[NRSNG Academy Lesson: ACE Inhibitors]
Antidepressant Cheat Sheet
[NRSNG Academy Lesson: Antidepressants]
Immunization Schedule
IMMUNIZATION SCHEDULE
BABY (months)
BIRTH
HepB
1
CHILD (years)
2
4
6
HepB
12
15
18
19-23
2-3
4-6
HepB
RV
RV
RV
TDaP
TDaP
TDaP
Hib
Hib
Hib
Hib
PCV
PCV
PCV
PCV
IPV
IPV
TDaP
TDaP
IPV
IPV
Anual In uen a (Yearly)
MMR
MMR
Varicella
Varicella
HepA (2
ose series)
[NRSNG Academy Lesson: Rubeola – Measles]
[NRSNG Academy Lesson: Rubeola – Measles]
Antibiotic Cheat Sheet
ANTIBIOTIC CHEAT SHEET
ANTIBIOTIC MOA
In ibition of
nucleic aci synthesis
In ibition of
protein synthesis
In ibition of
cell wall synthesis
Disruption of cell
membrane function
ell wall
loc pathways an
in ibit metabolism
ell
membrane
NA
olic acid
Ribosome
ow and
here Various Antibiotics
ram Negative
or
ram Positive
uter membrane
ipoproteins
Peptidoglycan
Periplasmic
space
utoplasmic
membrane
ipopolysaccharides
Porin
Protein
Gram + and -
Gram +
Penicillins
(Amoxicillin)
Gram + (Strep, Syphillis)
Disrupts synth of peptidoglycan
Tetracyclines
(tetracycline, doxycycline)
Broad spectrum (Gram +/-, atypicals)
Inhibit protein synth
Sulfonamides
(TMP-SMZ)
UTIs
Inhibit DNA synth
Macrolides
(azythromycin, erythromycin)
Gram + (URI’s, Strep, Staph)
Inhibits protein synth
Cephalosporins
Disrupts synth of peptidoglycan
1st gen Gram + (Keflex)
2nd gen Gram - Gram + (Cefzil)
3rd gen Gram - Gram + Pseudomonas (cefdinir)
4th gen Pseudomonas (Cefepime)
5th gen MRSA (Ceftobiprole)
Carbapenems
(meropenem)
Broad spectrum
Disrupts synth of peptidoglycan
Fluoroquinolones
(Ci ro o acin e o o acin)
Broad spectrum
Inhibit DNA synth
Metronidazole
(Flagyl)
Anaerobes, protozoa
Disrupts DNA
Lincosamides (clindamycin)
Step, Staph
Inhibit protein synth
Gram Aminoglycosides
(streptomycin, tobramycin, gentamicin)
Gram - Psuedomonas - TB
Inhibit protein synth
[NRSNG Academy Lesson Penicillin and Cephlosporins]
[NRSNG Academy Lesson: Penicillin and Cephlosporins]
Answering Pharmacology
Questions QUESTIONS
ANSWERING PHARMACOLOGY
12 Points to Answering Pharmacology Questions
1. Patient Safety
The NCLEX®is concerned about if you will be a SAFE nurse. Always think about what
option will lead to your patient being safe. You can automatically exclude options that
will put your patient in harm.
2. Focus on Side
ects
Learn the top 3 side effects with major medication classes. If you know the class and the
major side effects associated with that class you greatly increase your chances of
answering correctly.
3. ABCs
Airway, Breathing, Circulation. The ABCswill never go away. Focus on the nursing
process and the ABCswith each and every question including side effects.
4. Prefi es and Su
es
Learn the most common prefixes and suffixes. This will cut down your total study time
tremendously.
5. Look for Patient Clues
Does the question provide information about the patients original diagnosis? Use
general clues in the question about the patients, their history, and their condition. These
clues will guide you to the medications they will be taking.
6. General Patient Reaction
Look for clues in the patients reactions. For example if the patient reports dizziness, this
is a clue that you should assess blood pressure. Use your assessment skills to answer
pharmacology questions.
7. Generic
Only generic names will be used on the actual NCLEX®. Although these names can be a
bit harder to pronounce, they will provide clues (prefix/suffix) into the type of
medication it is which will guide you in choosing the correct answer.
8. Random, Random, Random
Regardless of how much you study . . . you will get that insanely random medication
that no one has ever heard of. In this case just take a deep breath, relax, and use your
nursing judgment, critical thinking, and think Patient Safety.
[NRSNG Academy Lesson: 12 Points to Answering Pharmacology Questions]
[NRSNG Academy Lesson: 12 Points to Answering Pharmacology Questions]
ANSWERING
PHARMACOLOGY
QUESTIONS
Answering
Pharmacology
Questions
9. Medical Diagnosis
Does the question identify a medical diagnosis If you have a working medical
diagnosis, use your knowledge to determine what signs and symptoms the
patient will have, what medications they will require to manage those symptoms,
and what are the main side effects of those medications.
10. Freebies
If you are already familiar with the medication . . . simply use your knowledge,
the nursing process, and critical thinking to answer the question.
11. Med Classes
Learn to recognize common side effects with major medication classes and the
appropriate nursing intervention for each of these side effects.
12. Why is the Medication Given?
Why is the medication being given. Try to identify a relationship between the
medication and the patients diagnosis. If you have the underlying diagnosis you can
generally identify what medication will be given for that condition.
[NRSNG Academy Lesson: 12 Points to Answering Pharmacology Questions]
[NRSNG Academy Lesson: 12 Points to Answering Pharmacology Questions]
Therapeutic
Drug LEVELS
Levels
THERAPEUTIC DRUG
DRUG
THERAPEUTIC LEVEL
Digoxin
0.8-2 ng/mL
Lithium
0.8-1.2 mEq/L
Theophylline
10-20 mcg/mL
Phenytoin
10-20 mcg/L
OTHERS
Vancomycin
Trough 10-20 mcg/L
Salicylate
150-300 mg/L
Carbamazepine
5-12 mcg/L
Gentamicin
5-10 mcg/mL
Phenobarbital
10-40 mcg/mL
Procainamide
4-10 mcg/mL
Amitriptyline
120-150 ng/mL
[NRSNG Academy Lesson: Therapeutic Drug Levels]
[NRSNG Academy Lesson: Therapeutic Drug Levels]
ANTIDYSRRHYTHMIC MEDS AND ACTION
Antidysrrhythmic
Meds and Action
POTENTIAL CHART
Antiarrhythmic AChart
ents
Potential
K
Cl
Ca
I
Class IV
Ca Channel Blocker
Verapamil
Diltiazem
1
2
K
0
a I
Class I
Class II
Beta Blocker
Na Channel Blocker
1a: Procainamide
1b: Lidocaine
1c: Proprafenone
3
Class III
K Channel Blocker
Amiodarone
Sotalol
Propranolol
Metoprolol
4
4
This chart represents the cardiac action potential (first image) with the electrical conduction of the heart EKG.
The EKG is representative of what is occuring during each phase of the cardiac action potential.
Along the cardiac action potential you will see what is occuring with the ions.
Below the ion activity you will note what antiarrhythmic medications will have an effect during that phase of
the action potential.
[NRSNG Academy Lesson: Calcium Channel Blockers]
[NRSNG Academy Lesson: Calcium Channel Blockers]
Hierarchy of O2 Delivery systems
HIERARCHY OF O2 DELIVERY SYSTEMS
METHOD
Nasal Cannula
1 lpm = 24%
2 lpm = 28%
3 lpm = 32%
4 lpm = 36%
5 lpm = 40%
6 lpm = 44%
Simple Face Mask
5 lpm = 40%
6 lpm = 45-50%
7 lpm = 50-55%
8 lpm = 55-60%
Non-rebreather Mask
6 lpm = 60%
7 lpm = 70%
8 lpm = 80%
9 lpm = 90%
10 lpm = close to 100
Terms to Know:
Pressure support:
Preset inspiratory support level. When the pt initiates
a breath, this positive pressure flows to assist the pts
spontaneous breaths.
2
PEEP ( ositive end-expiatory pressure):
Maintenance of pressure above atmospheric at end
expiration.
Auto-PEEP:
Trapping of gas in the lung caused by insufficient
expiatory time (breath stacking). Increases risk of
barotrauma.
Venturi Mask
4 lpm = 24-28%
8 lpm = 35-40%
12 lpm = 50%
PIP (peak inspiratory pressure):
Airway pressure at the peak of inspiration.
Trach Collar
21- 0 at 10L
Tidal Volume (Vt):
The volume of air expired with each breath
T-Piece
21-100 with flow rate at 2.5 times minute ventilation
CPAP
Positive airway pressure during spontaneous breaths
Bi-PAP
Positive pressure during spontaneous breaths and
preset pressure to be maintained during expiration
SIMV
Preset Vt and f. Circuit remains open between
mandatory breaths so pt can take additional breaths.
Ventilator doesn’t cycle during spontaneous breaths
so Vt varies. Mandatory breaths synchronized so they
do not occur during spontaneous breaths.
Respiratory Rate (f):
The number of breaths per minute, may be greater
than preset frequency, but not less.
Minute ventilation (Ve):
Vt X f; volume of air expired per minute.
PaCO2 (35-45 mm Hg):
Amount of CO2 dissolved in arterial blood. Partial
pressure of arterial CO2.
SaO2 (95-100%):
Percentage of oxygenated hemoglobin in arterial
blood. Indirectly measured via SpO2 (pulse ox).
PaO2 (80-100 mm Hg):
Amount of oxygen dissolved in blood plasma.
Bi-PAP
Preset Vt and f and inspiratory effort required to assist
spontaneous breaths. Delivers control breaths. Cycles
additionally if pt inspiratory effort is adequate.
Same Vt delivered for spontaneous breaths.
[NRSNG Academy Lesson Hierarchy of O2 Delivery]
[NRSNG Academy Lesson: Hierarchy of O2 Delivery]
Lung sounds
[NRSNG Academy Lesson: Lung Sounds]
gas Exchange
GAS EXCHANGE
By helix84 (en:Image:Alveoli.jpg) [GFDL (h p //www.gnu.org/copyleft/fdl.html), CC-BY-SA-3.0
(h p //creativecommons.org/licenses/by-sa/3.0/) or CC BY 2.5 (h p //creativecommons.org/licenses/by/2.5)], via Wikimedia Commons
Gas exchange occurs in the alveoli t e functional unit of the lungs.
Deoxygenated blood enters the capillaries surrounding the alveoli, O2 enters the
bloodstream and CO2 exits into the alveoli to be exhaled.
Causes of Poor Gas Exchange
Priority Interventions
Atelectasis
Re-in ate alveoli
Hyper or Hypoventilation
Treat cause to restore normal
Poor Air ow (airway swelling or
breathing pa ern
bronchoconstriction)
Give meds to open airways
Pulmonary Edema
Diuretics to decrease uid in lungs
Pulmonary Embolism
Thrombolytic or Thrombectomy
Vasoconstriction
Vasodilators
Low blood volume
Replace lost blood volume
[NRSNG Academy Lesson: Gas Exchange]
[NRSNG Academy Lesson: Gas Exchange]
Asthma Medications
[NRSNG Academy Lesson: Asthma]
Artificial Airways Decision Tree
ARTIFICIAL AIRWAYS DECISION TREE
Use this decision tree to determine which of the four arti cial airways
is most appropriate for your patient’s situation
Conscious
Clears own
secretions
Apply oxygen
as needed
Can’t clear own
secretions
E ective respiratory
e ort
Ine ective
respiratory e ort
Nasopharyngeal
Airway + Suction
Requires ventilation
Endotracheal Tube
Unconscious
Respiratory e ort,
unprotected airway
No respiratory e ort
If head tilt, chin lift or
jaw thrust ine ective
No contraindication
to intubation
Tracheal obstruction
or damage
Oropharyngeal Airway
+ Bag/Valve/Mask
Endotracheal Tube
Tracheotomy
[NRSNG Academy Lesson: Artificial Airways]
Ventilator Alarms
[NRSNG Academy Lesson: Vent Alarms]
Chest Tube Management
CHEST TUBE MANAGEMENT
INDICATIONS FOR A CHEST TUBE:
Drain uid, blood, or air
Pleural e
on
Hemothorax
Pneumothorax
Establish negative pressure
Facilitate lung e pansion
By British Columbia Institute of Technology (BCIT). Download t is book
for free at h p //open.bccampus.ca h ps //opente tbc.ca/clinicalskills/chapter/10-7-chest drainage-syst
ems/, CC BY-SA 4.0,
h ps //commons.wikimedia.org/w/index.p p?curid=66770951
PRIORITY NURSING ASSESSMENTS (TWO AA’S)
Tidaling -
uid should uctuate with respirations
Water seal - there should be su ient water in the water seal chamber
Output - color, character, and quanti y of output - measured hourly at rst, then every 4-8 hours
per policy
Air leak - conti ous bubbling in the water seal chamber indicates an air leak - this should be
troubleshooted immediately
Ability to breathe - always assess the patient’s lung sounds and respiratory e ort
SpO
is the patient oxygenati g?
SAFETY CONSIDERATIONS
Avoid dependent loops
Never strip or clamp tubing
Ensure collection chamber stays upri ht
Assess insertion site & dressing for bleeding or drainage
Accidental removal - cover wit
sided occlusive dressing
[NRSNG Academy Lesson: Chest Tube Management]
[NRSNG Academy Lesson: Chest Tube Management]
Notes
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Notes
83
Notes
84
Notes
85
Notes
86
Notes
87
Notes
88
Notes
89
Notes
90