se
Disea
Pathophysiology
↳
heart
R
cannot
L
Hf
-
-
0
HTN
pulmonary
causes
anything
,
congestion
perfusion
+
fluid
→
up from lungs
Rv → systemic circulation
→
°
W
lack Of perfusion to
muscle
__
'
acute cause of Hf : lack of perfusion
to part of
muscle → cells die = Hf
HF
✓
lower
extremely
hepatq splenomegaly
Cliver + spleen enlargement
0
-
from
fatigue
o
nocturia
°
Wt
0
gain
es
help
w/ ventilation
→
edema from
coughs csputum]
congestion → body
to
get rid
,
HTN → LHF
→
fluid
→
give meds
to help evaluate
→
measure
fluid
in
/ at
-
are
~
nursing
care
~
~
after Mt
to show
,
not to
,
doesn't
percussion
body
-
i
release
we
do
→
produce that
Hf
is a chronic condition
serious
complications
Abbs blocks
0
NS
that i can
"
→
)
"
tells
work
~
bp
,
so
anything
in these
that
can
lead
Pt
workload
on
the
Or more
in
the
Pt education
0
°
report
SOB
,
pushed
out into
→
,
4
→
→
°
Thora centesis
Peri edema
.
,
fluid
to
overcome
an
0
Na> →
"
of force the L
(amount
produce
the co needed
we do not
the tissues
) But
fye
chronic Hf
,
doesn't
-
respond
to
implant helps w/ contraction
last resort
Heart transplant
around
at risk
an
A fib
I WK
cough
,
nausea
L
,
abd retention , PND
→
causing
enlargement
liver enzymes
of liver
→
→
elevation of
liver impalement
→
liver failure
=
SOB
o
inserted ,
fluid drained a t
needle
renal
\
failure
cardiac muscle doesn't
its supposed
for Afibcatrialfibrilation)
(
CUA )
swooshed around in atrium
clot to break off travel to
to
→
head
renal failure
contract the
Kidneys dont get
Percussion they need
→
increase in fluid
risk factor for stroke
blood gets
Hepatomegaly
→
.
3 -5lb
of extra
volume excess CFVE)
space →
lungs
-
.
)
" Ventricular assist device
"" "☐
-
system
Dysrythmias
'
pumping
prevents complication
/
gets rid
treatment anymore
to
11010MGBP)
heart chambers
get enlarged due to
excersise
3lb / 2d
,
fluid
→
perfuse to
want
fluid gets
pleural
accumulates around
~
wgt gain
Dizzy
+
to
(
fluid backed Pinto
lungs
Pink frothy sputum
°
control
medication
Fye
&
HTN
has to
body Give
with because
vasoconstriction
to
& force of contr
Diuretics
In after load
catacholltminl release
→
low sodium diet
-
=
CHZO follows
mostly from ANA diet
Pleural effusion
°
L
TX :
0
efficiently
co
Possible complications
Stresses the pt system ( infection)
to
cardiac fibers
this
not want
to these
KBP and blood volume
do not want
THR n Bp ← we
more
wears out
(t ) inotrope
law C. When
Can not contract
→
°
~
not
overdo
flu / pneumonia vaccination
o
,
force of contraction
A
muscle
→
need more blood volume:)
l
of percussion
cardiac rehab / excersise
o
tell
the sympathetic
meds
those
long CHTNJ
to & renal
BP
HF NCO
Organs
put
req for
ace inhibit
& HR
lack
.
Hgb
frank Starling
L
brings
?
they retaining fluid
admin meds
o
leads
→
me some blood
daily weight
0
bad the HF is
u
~
stretched for too
the RAAJC kidneys
-
( activates
progress
Platters?
)
Digoxin ( cardiac glycoside
L
Beta blockers
o
"
0
for HF
TBP tfluid retention Bot !
leads to RHF
-
treatment
ARDS
+
angiotensin , aldosterone
\
excess
:
efficiently
activates
I 310 'S
"
Determines how
cpgc
o
retention
w/ Hf
→
+
fluid
Hf NCO
PMP
→
#I
Ace inhibitors
o
(
control BP
l
panel)
kidney function
to body
chem marker heart releases
When its being overly stretched
Medications
( & BP &
Nursing interventions
& patient teaching
o
metabolic
liver /
BNP ( b type naturetic peptide]
✓
running on treadmill chemically induced
tachycardia to monitor for symptoms
retaining fluid
elevate HOB w/ pillows
to
Hf
+
electrolytes
-
dyspnea (PND)
cause of
l
Stress Test
nocturnal
pink frothy sputum
e s fluid in
lungs pulmonary
#1
Cmp ( complete
-
0
0
o
,
°
°
paroxysmal
es
~
valves
ejection fraction C. 1. of how well LX contracts]
goes into
to see problem
-
SVC
into
cars ; catheter
coronary arteries
the chamber sizes +
on
angiogram
( if
pt has
Cardiogram
-
more info
)
pain ( CP
o
"" "
C back
°
their conduction system?
.
Jugular venous distention
0
echo
(
Chest
NC
backed up fluid via
Ekg
Chows
SOB
0
0
Diagnostics
0
up
0
fluid collecting in abdomen
L
backing
it came from
lungs
ascities
°
starts
from where
The
failure
Heart
fluid
→
peripheral edema
0
muscle
heart muscle wears
→
out
fluid backs
of the
myocardial infarction
°
coronary artery disease
0
→
Clinical Manifestations I
back up in
myopathy
-
disease
Needed for the extra
co
RV
6
muscle
out
to for the
harder
pumps
inadequate
fluid from lungs
=
cardio
o
lover time wears
increase blood V01Me
-
that
HF
-
caused by HTN
o
pulmonary pathology :
or
chronic HTN
o
Most common !
°
Caused by L HF : fluid
from lungs backs up to RV
°
Risk factors
efficiently
pump
failure
Heart
to work
way
the
properly