Neurological Emergencies Coma, Seizures, Syncope, Stroke Temple College EMS Professions Coma State of unconsciousness from which patient cannot be aroused Coma Unconsciousness = Immediate Life Threat Loss of airway Aspiration Coma Management of ABC’s must come before investigation of cause Airway Open, clear, maintain If trauma present or no history available, immediately control C-spine Breathing Assess presence, adequacy High concentration O2 immediately on all patients with decreased LOC Assist if respiratory rate, tidal volume inadequate Circulation Pulses? Perfusion? After ABC’s stabilized. . . Quickly investigate cause DERM D = Depth of coma What does patient respond to? How does he respond? E = Eyes Pupils equal, dilated, constricted, Responsive to light? How? R = Respiratory pattern Rate? Unusually deep or shallow? Altered pattern? M = Motor Function Evidence of paralysis? Movement on stimulation? How? Vital Signs Shock? Increased ICP? Arrhythmias? Head to Toe Survey Injuries causing coma? Injuries caused by fall? What do the scene, bystanders tell you? Possible Causes Not enough oxygen Not enough sugar Not enough blood flow to deliver O2, sugar Direct brain injury Structural (trauma) Metabolic (toxins, infections, temperature) Possible Causes Alcohol Epilepsy Insulin Overdose Uremia (and other metabolic causes) Trauma Infection Psychiatric Stroke, syncope Management Secure airway Protective reflexes may be lost Immobilize spine unless absolutely certain injury not present Spinal injury not suspected - patient on left side Management High concentration O2 Assist ventilation as needed Monitor neurological/vital signs every 5 minutes Management Protect patient’s eyes on long transports (tape shut, moist pads) Patient may hear, understand even though unable to respond Treat, reassure accordingly Seizures Episodes of uncoordinated electrical activity in brain Signs/symptoms depend on area involved Epilepsy Tendency to have repeated episodes of seizure activity Seizure Types Grand mal (major motor) Petit mal (absence) Focal motor (simple partial) Psychomotor (complex partial) Grand Mal Seizure Aura Sensation coming before convulsion Patient may recognize as sign of impending seizure May help locate origin of seizure in brain Grand Mal Seizure Convulsion Loss of consciousness Tonic phase - rigidity Clonic phase - rhythmic jerking, incontinence, ineffective breathing Grand Mal Seizure Post-ictal Phase Exhaustion Drowsiness Headache Possible hemiparesis (Todd’s paralysis) Petit Mal Seizure Loss of consciousness No loss of postural tone More common in children Focal Motor Seizure Rhythmic jerking of limb, one side of body No loss of consciousness Psychomotor Seizure Loss of consciousness Sterotyped movements (automatisms) May look purposeful, but aren’t Lip smacking, movements of hands May be called in as “drunk”, “O.D.”, “psych patient” Generalized Seizure Management During seizure Remove from potential harm Do not forcibly restrain Roll on side Avoid putting anything in mouth Generalized Seizure Management After seizure ends Assess ABC’s Clear airway Most common cause of seizure deaths is post-ictal airway loss Generalized Seizure Management High concentration O2 - immediately!! Assist breathing if ventilation inadequate Generalized Seizure Management Obtain history/physical Trauma that could have caused, been caused by seizure Anti-seizure medications Neuro/vital signs every 5 minutes If patient ventilating adequately, transport on left side Seizures Anything that injures brain can cause seizures (AEIOU/TIPS) Do not assume seizures are due to idiopathic epilepsy until proven otherwise Status Epilepticus > 2 seizures without intervening conscious period Immediate Life Threat Management Secure airway Assist breathing with O2 Transport Request ALS intercept Syncope Fainting Sudden, temporary loss of consciousness Caused by lack of blood flow to brain Causes Stress, fright, pain (vasovagal syncope) Orthostatic hypotension (BP fall on standing) Decreased blood volume Increased size of vascular space Decreased cardiac output Prolonged forceful coughing Management ABCs Keep patient supine, elevate lower extremities Oxygen Assess underlying cause CVA Cerebrovascular Stroke accident CVA Damage of portion of brain due to interruption of blood supply Mechanisms Thrombosis Hemorrhage Embolism Thrombosis Blockage of vessel by thrombus Usually forms at area narrowed by atherosclerosis Typically in older persons Frequently occurs during sleep Hemorrhage Vessel ruptures Associated with hypertension, aneurysms of cerebral blood vessels Usually characterized by Sudden onset Severe signs, symptoms Embolism Blood clots, plaque fragments travel through vessel; lodge, block flow Often associated with: Atherosclerosis of carotids Chronic atrial fibrillation Signs/Symptoms Alterations in consciousness Altered affect Confusion Dizziness Coma Signs/Symptoms Localizing signs Paralysis Loss of sensation Loss of speech Unilateral blindness Loss of vision in half of visual field of both eyes Unequal pupils Signs/Symptoms Seizures Headache Stiff neck Transient Ischemic Attacks TIAs “Little strokes” Produce deficits that resolve completely in <24 hours Frequently precede CVA Management Assess ABC’s Protect airway High concentration O2 Vital signs every 5-10 minutes Note increased BP, irregular pulse Management Nothing by mouth Avoid rough handling Transport paralyzed side down Guard your conversation Patients who cannot speak may still understand! Management CVAs caused by thrombus, embolus may be reversible with thrombolytics (clot busters) Early recognition, rapid transport to appropriate facility is critical