polio

advertisement

POLIOMYELITIES

Copyright 2005 Lippincott Williams & Wilkins

POLIOMYELITIES

Definition:

Acute viral infection of motor nuclei of CNS mainly motor nuclei of anterior horn cell of spinal cord and motor nuclei of the cranial nerve leading to lower motor neuron lesion (LMNL) of flaccid paralysis with normal sensation.

Poliomyelitis , often called polio or infantile paralysis , is an acute viral infectious disease spread from person to person, primarily via the fecal-oral route .The term derives from the Greek poliós ( πολιός), meaning "grey", myelós ( µυελός), referring to the " spinal cord ", and the suffix -itis , which denotes inflammation .

Types of Polio virus:

1- Brunhilde

2-Lansing

3- Lean infection of one virus give immunity to this type only

Copyright 2005 Lippincott Williams & Wilkins

Types of Vaccination:

1- Sabine:

Attenuated live polio virus Anticipated intramuscular

Dose------- 2,4,6 month poster dose 18 month preschool age 4 years

Advantage : Complete immunity with in 3 days

2- Salk:

• Killed virus attenuated oral or not supported by intestinal tract

• Advantages: effective, safe

• Dose: 2,4,6 month, 4 years

• Disadvantages: need repeated injection, delay in immunity takes 3 weeks to response & incomplete immunity

Stages:

1- Acute Stage; The first 2 Weeks

• Viral infection (vomiting, headache at the end of acute stage then paralysis appear in respiratory muscle and extremity)

Copyright 2005 Lippincott Williams & Wilkins

2Recovery Stage; From 2 weeks to 2 Years

• Called stage of functional recovery because:

• 1- Subside to edema

• 2- Improved neural plasticity

• 3- Hypertrophy of non affected muscle

Character paralysis:

• 1- LMNL 2- Flaccid

• 3- massive (affected more than one group)

• 4- Asymmetrical lesion

• 5- Patchy distribution

After 2 years recovery cannot occur because:

• 1- New formation of new motor end plate

• 2- Orientation of the axon is parallel to muscle

Chronic Stage: After 3 years

a- Muscle skletal deformity : Muscle weakness-----contracture……Deformity b- Respiratory affection:

 Affection in respiratory center in medulla lead to irregular breathing

 Affection in Spinal cord or nerves of respiration ------- respiratory muscles become weak

Copyright 2005 Lippincott Williams & Wilkins

Approximately 90% of polio infections

cause no symptoms at all

, affected individuals can exhibit a range of symptoms if the virus enters the

blood stream

.

1% of cases the virus enters the

central nervous system

, and destroying

motor neurons

, leading to

muscle weakness

and acute

flaccid paralysis

. Different types of paralysis may occur, depending on the nerves involved.

Spinal polio

is the most common form, characterized by asymmetric paralysis that most often involves the legs.

Bulbar polio leads to weakness of muscles innervated by

cranial nerves

. Bulbospinal polio is a combination of bulbar and spinal paralysis

Copyright 2005 Lippincott Williams & Wilkins

Classification of poliomyelitis:

• The term poliomyelitis is used to identify the disease caused by any of the three serotypes of poliovirus. Two basic patterns of polio infection are described:

• Minor illness which does not involve the central nervous system

(CNS), sometimes called abortive poliomyelitis .

• Major illness involving the CNS, which may be paralytic or nonparalytic

• In most people with a normal immune system , a poliovirus infection is asymptomatic . Rarely the infection produces minor symptoms; these may include upper respiratory tract infection ( sore throat and fever), gastrointestinal disturbances (nausea, vomiting, abdominal pain , constipation or, rarely, diarrhea), and influenza-like illness .

Copyright 2005 Lippincott Williams & Wilkins

The virus enters the central nervous system in about 3% of infections. Most patients with

CNS involvement develop non-paralytic aseptic meningitis , with symptoms of headache, neck, back, abdominal and extremity pain, fever, vomiting and irritability.

Approximately 1 in 1000 cases progress to paralytic disease, in which the muscles become weak, floppy and poorly controlled, and finally completely paralyzed; this condition is known as acute flaccid paralysis Depending on the site of paralysis, paralytic poliomyelitis is classified as spinal , bulbar , or bulbospinal

• Transmission

Poliomyelitis is highly contagious via the oral-oral (oropharyngeal source) and fecal-oral

(intestinal source) routes . It is seasonal in temperate climates , with peak transmission occurring in summer and autumn.

Risk Factors of polio include:

• immune deficiency , malnutrition , tonsillectomy , physical activity immediately following the onset of paralysis, skeletal muscle injury due to injection of vaccines or therapeutic agents, and pregnancy .

• Although the virus can cross the placenta during pregnancy, the fetus does not appear to be affected by either maternal infection or polio vaccination.

[ Maternal antibodies also cross the placenta , providing passive immunity that protects the infant from polio infection during the first few months of life .

Copyright 2005 Lippincott Williams & Wilkins

Physical Therapy Evaluation:

1- History:

Personal History

Past History

Present History

History of vaccination

2- Informal evaluation;

ALook to the child while entering the treatment room (if mother carrying him or not, walk alone, gait and type of brace

B- On the plinth, general conditions of the child(anemic , healthy)

C- Observe the chest from the anterior view(rate, pattern of breathing: shallow, irregular

D- Positions of the limb and leg length discrepancy

E- Skin condition(ulcer due to brace , scar due to tendon release

F-Spontaneous motility

G- Deformities:#

Acute Stage : Muscle imbalance , prolonged bed rest, inadequate P.T,

Shortening, Contracture Weight bearing on weak joints

Copyright 2005 Lippincott Williams & Wilkins

Chronic Stage: Deformities like:

Lower Limb

:

Hip--- Flexion, abduction, external rotation

Knee : Flexion deformity, genu valgus, genu varum, Genu

Recurvatum due to Ms imbalance between quadriceps and hamstring Ms

Ankle : equines deformity(plantar flexion), calcenous deformity(dorsiflexion)

Varus deformity(inversion), valgus deformity(eversion)

Talipus equino varus, Talipus equino valgus

Talipus calcenus varus, Talipus calcenus valgus

Quick Test :

Crock lying position to detect leg discrepancy:

If end of femur is forward --------- shortening of tibia

If end of femur upward------------ shortening of femur

Postural Fixation ; raise limb upward let it drop

Sudden drop limb ---------- hypotonia

Copyright 2005 Lippincott Williams & Wilkins

3Formal Evaluation;

Muscle Tone

Flexibility test

Muscle Test

ROM

Measurement

Postural assessment

Gait Analysis

A- Muscle tone:

By Observation: Supine----- Flaccid paralysis of limb due to hypotonia---------Frog like position(Abd, Ext Rotation hip, Plantar flex ankle).

Passive Movement Don’t forget to support distal part

Postural fixation raise limb then sudden drop : polio ----sudden Drop

BFlexibility Test: To test tightness of tendoachilis :

Supine lying with hip and knee flexion

Grasp : Thumb on sole of foot , Index on shaft of tibia, 4 fingers cubing on the heel

Copyright 2005 Lippincott Williams & Wilkins

From flexion knee make dorsiflexion ankle---------tightness of soleus

From extension knee make dorsiflexion ankle- Tightness in Gastrocniemus

Muscle Test:

Above 3 Years------------------ MMs Test

Below 3 years----------------Functional Ms test

Zero---------- No Contraction

Sub functional-----------Not complete ROM

Functional ------------------- complete ROM

Methods: a- Unfavorable position: Prone lying will facilitate neck trunk extension and by this way you can test neck and back Ms

Half prone for testing hip and knee flexors b- Reflexes according to age :

Positive supporting to test extensor of lower limb and plantar flexors

C- Toys put toy in direction that can facilitate movement d- spontaneous motility: uncontrolled movements e- Tactile stimulation( Scratching, Squeezing, tapping)

Hip:Tactile

Stimulation of hip flexors (iliopsoas): supine lying

Copyright 2005 Lippincott Williams & Wilkins

Grasp : Thumb on the sole of the foot, Index on shaft of tibia - 4 fingers cubing heel of the foot

Scratch: on inguinal ligament by followed passive movement until you feel that child can perform it actively

Gluteus Maximus : Half Prone lying with untested limb between my leg, Tested limb kept in flexion to isolate hamstring

Scratch on Gluteus Maximus followed by passive hip extension, or painful stimulus on ant aspect of thigh above just knee

Hip Adductors: Supine lying: Grasp One hand cubing heel of the foot other hand Scratch medial aspect of thigh followed by passive hip adduction by the hand which cup heel or painful stimulus on the greater trochanter

Knee: Tactile Stimulation: Knee flexors

Position : prone lying Knee slight flexion(10-15) to isolate gastrocnemius

Scratch by the hand which support pelvis on the back of the knee

Other hand which cup heel make passive knee flexion

Painful Stimuli on the ant aspect of distal part of tibia

To isolate biceps femoris put tested knee slight flexion with Ext Rotation

To isolate Semitendinosus put tested knee slight flexion with Med Rotation

Copyright 2005 Lippincott Williams & Wilkins

Knee extensors (Quadriceps):

Position : Supine lying tested limb in hip and Knee slight flexion Grasp : thumb above knee , index on lateral aspect of thigh, middle two fingers below knee from back for supporting, little finger to initate movement

Scratch by the hand which support pelvis on the Pelly of the muscle

Other hand which cup heel make passive knee flexion

Painful Stimuli on the post aspect of calcaneus

Ankle: Tibilais anterior

Tibilais posterior

Pronei(longus, brevis, tertus) in these 3 muscles must put knee in slight flexion(10-15) :

1-To isolate gastrocnemius

2- to prevent damage of collateral ligaments of knee

3- To prevent compression of post aspect of knee

Tactile stimulation to Tibilais anterior:

Position: supine lying with flexion knee , Thumb flex toes to isolate flexor hallicus, and digitorium , Index on ant aspect and medial of foot to maintain full inversion and plantar flexion, Middle finger palpat tendon of tibialis anterior, Ring and little finger on heel to perform passive dorsi flexion, Painful stimulus : On the sole of the foot

Copyright 2005 Lippincott Williams & Wilkins

• Tibialis Posterior:

Mainly inversion & assist in planter flexion

• Grasp: one hand catch lower end of tibia and other hand hold foot from ankle

• Scratch: on medial malleoli by little finger

• Peroneus longus---------eversion +plantar flexion Tendon Below Lat

Malleoli

• Peroneus brevis--------eversion +mid position of ankle Tendon Below Lat

Malleoli

• Peroneus Tartius -------eversion +dorsi flexion Tendon above Lat

Malleoli Painful stimulus on the medial aspect of foot

Copyright 2005 Lippincott Williams & Wilkins

Download