Supplemental Figure Legends
PATIENT 1
Figure 1. Baseline angiogram, BVS deployment and final result
The angiogram of patient 1 showed a long stenosis at the proximal - mid LAD and a stenosis at
the distal LAD. There was also a lesion at the proximal segment of the first diagonal branch which
was treated only with balloon dilatation. Two BVS were deployed at the proximal - mid LAD with
minimal overlapping (Absorb 3x28mm and Absorb 3.5x28mm) and another BVS (Absorb 2.5x18
mm) was deployed at the distal LAD. The final angiogram showed a good result.
Figure 2. Control angiogram and diagonal stenting
Patients 1 presented two months later with ACS. Because of persistent symptoms and minimal
ECG changes a new angiogram was performed. There was a good patency of the LAD scaffolds
but, in the absence other possible culprits, it was decided to address the chronic dissection with
TIMI 3 flow and no contrast stain in the diagonal branch that was previously treated only with
balloon dilatation. The intervention was performed with the use of two 18x2.25 mm Zotarolimus
eluting stents. A final good angiographic result was achieved.
PATIENT 2
Figure 3 Baseline angiogram
Patient 2 is a 54 years old lady affected with insulin dependent diabetes, hypertension and
dyslipidaemia. She was submitted to coronary angiogram for stable angina (CCS2). The drug
eluting stents implanted one year before in the proximal - mid LAD were patent (caudal view) but
there was a severe stenosis at the proximal LAD and a severe diffuse narrowing at the distal LAD
(cranial view). There was also a 90% stenosis involving a distal branch of the LCX.
Figure 4 Angiographic result after treatment
A 3.0x18 mm BVS was deployed at the proximal LAD while the distal LAD was treated with
only balloon dilatation. A DES was deployed to treat the lesion at the proximal segment of the
circumflex distal branch. The final angiogram showed a good result.
Figure 5 Nine months control angiogram
Patient 2 presented 9 months later with angina (CCS 3). The coronary angiogram showed a total
LAD occlusion at the distal segment of the LAD. There was also disease progression involving all
the distal braches of the LCX with total occlusion of the DES implanted 9 months before. The BVS
implanted at the proximal LAD was patent. The patient underwent CABG (LIMA to LAD, LSVG
to D1, LSVG to OM1, LSVG to distal RCA) in October 2013. At 1 months follow up she was
asymptomatic.